Relevant bibliographies by topics / Glaucoma

Academic literature on the topic 'Glaucoma'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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Journal articles on the topic "Glaucoma"

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Engin, Kaya N., Cemil Yılmazlı, Günay Engin, and Levent Bilgiç. "Results of Combined Cyclectomy/Trabeculectomy Procedure Compared with Ahmed Glaucoma Valve Implant in Neovascular Glaucoma Cases." ISRN Ophthalmology 2011 (December 8, 2011): 1–6. http://dx.doi.org/10.5402/2011/680827.

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Purpose. Cyclectomy/trabeculectomy and Ahmed glaucoma valve implant are operations suggested for refractory glaucomas. In this study, we have evaluated the outcomes that we observed with these two techniques in neovascular glaucoma patients. Material and Methods. Thirty-five eyes with neovascular glaucomas were included in this study. Ahmed Glaucoma valve (group A) was applied to ten eyes, while cyclectomy/trabeculectomy was applied to 25 eyes (group C/T). Vision, intraocular pressure and complications were evaluated at the end of the first week and after 6 and 12 months. Results. Vision preservations were 80% and 92%, and success rates in reducing intraocular pressure were 50% and 72% for Ahmed glaucoma valve and cyclectomy/trabeculectomy groups, respectively. None of the differences in complication rates was found to be statistically significant. Conclusions. In the surgical management of neovascular glaucoma, cyclectomy/trabeculectomy was shown to be an alternative to Ahmed glaucoma valve operation.
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McDowell, Colleen M., and Michael G. Anderson. "Toward a Genetic Understanding of Glaucoma—Breakthroughs and Challenges from Studies of Exfoliation Glaucoma." US Ophthalmic Review 04, no. 01 (2011): 23. http://dx.doi.org/10.17925/usor.2011.04.01.23.

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Glaucoma is a complex group of diseases causing progressive degeneration of the optic nerve. Despite strong evidence of hereditary contributions to glaucoma, relatively few genetic mutations associated with glaucoma risk have thus far been identified. Glaucoma likely represents a complex genetic trait influenced by multiple loci and many confounding factors, increasing the challenge in identifying glaucoma’s hereditary basis. In this article, a brief history of the search for glaucoma-causing mutations is presented, highlighting recent breakthroughs that have identified lysyl oxidase-like 1 (LOXL1) as a major genetic risk factor contributing to exfoliative glaucoma. In considering challenges that remain ahead, our central tenet is that the search for glaucoma’s genetic elements will continue to require experimental ingenuity and synergistic approaches involving animal models that render the complexity of glaucoma more tractable.
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Calugaru, Dan. "Intraocular pressure modifications in patients with acute central/hemicentral retinal vein occlusions." International Journal of Ophthalmology 14, no. 6 (June 18, 2021): 931–35. http://dx.doi.org/10.18240/ijo.2021.06.20.

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Intraocular pressure (IOP) modifications in patients with acute central/hemicentral retinal vein occlusions (RVOs) consist in IOP reductions and increases. The IOP reduction is due to a transitional hyposecretory phase of the aqueous humor, that increases gradually until 3mo after the venous occlusion onset, and then finally disappears after month 4th. The IOP increases lead to the ocular hypertension and glaucoma. The possible pathogenetic correlations between ocular hypertension/glaucoma and acute central/hemicentral RVOs have been classified into three groups: 1) the venous occlusion precedes the ocular hypertension/glaucoma causing neovascular glaucoma and secondary angle-closure glaucoma without rubeosis; 2) the ocular hypertension and the glaucoma precede the venous occlusion and favor its appearance (ocular hypertension, primary angle-closure, primary angle-closure glaucoma, and open angle glaucomas); and 3) the venous occlusion and the ocular hypertension/glaucoma are mostly age dependent appearances due to common vascular and collagen alterations, lacking a causal connection between the 2 conditions.
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Díaz, Esperanza Gutiérrez, Enrique Mencía Gutiérrez, and Pilar Tejada Palacios. "The Role of Glaucoma Drainage Devices in Paediatric Glaucoma." European Ophthalmic Review 06, no. 04 (2012): 208. http://dx.doi.org/10.17925/eor.2012.06.04.208.

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Glaucoma drainage devices may be indicated in glaucoma associated to complex congenital anomalies or dysgenesis of the anterior segment, secondary glaucomas such as those developed after congenital cataract surgery, severe primary congenital glaucoma after failed angle surgery and in eyes with severe conjunctival scarring. They can be combined with several other procedures, such as cataract extraction, intraocular lens implantation, keratoplasty or pars plana vitrectomy. This procedure may achieve a sustained and prolonged intraocular pressure reduction in children, with early post-operative success as high as 90 %, although it decreases over time, to 40–60 % survival at 4–6 years using the Kaplan-Meier analysis and most children need to reinitiate glaucoma treatment postoperatively. We must bear in mind that complications may occur, especially tube malposition and exposure, and multiple repeated surgeries may be necessary. The advantages and complications should be weighed in each individual case.
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Zia, Rashid. "editorial." Pakistan Journal of Ophthalmology 35, no. 4 (January 23, 2020): 215–16. http://dx.doi.org/10.36351/pjo.v35i4.988.

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Glaucoma, is a group of conditions characterized by optic disc cupping and visual field defects. Evaluation, staging and monitoring of glaucoma requires a series of functional tests which is a time consuming process. So far, Standard Automated Perimetry (SAP) is recognized as a reference standard for all the functional testing1. Glaucoma may present with a structural or a functional change. Therefore, the correct test strategy for diagnosis is vital to prevent overlooking the onset of glaucoma2.
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Abeshi, Andi, Francesca Fanelli, Tommaso Beccari, Munis Dundar, Lucia Ziccardi, and Matteo Bertelli. "Genetic testing for Mendelian glaucoma." EuroBiotech Journal 1, s1 (October 27, 2017): 70–73. http://dx.doi.org/10.24190/issn2564-615x/2017/s1.22.

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Abstract We studied the scientific literature and disease guidelines in order to summarize the clinical utility of genetic testing for Mendelian glaucomas, a large heterogeneous group of inherited disorders, classified according to age of onset as congenital glaucoma, juvenile glaucoma and age-related glaucoma. Variations in the TEK, MYOC, ASB10, NTF4, OPA1, WDR36 and OPTN genes are inherited in an autosomal dominant manner and variations in the CYP1B1 and LTBP2 genes have autosomal recessive inheritance. The prevalence of congenital glaucoma is estimated at 1-9 per 100 000, that of juvenile glaucoma at 1 per 50 000, while there is insufficient data to establish the prevalence of age-related glaucoma. Clinical diagnosis is based on clinical findings, age of onset, family history, ophthalmological examination, intraocular pressure, gonioscopy and fundoscopy. The genetic test is useful for confirming diagnosis, and for differential diagnosis, couple risk assessment and access to clinical trials.
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Castro, Ana Flávia Nogueira, and Lívia Oliveira Delgado Mota. "Uma abordagem geral do Glaucoma: revisão de literatura." Revista Eletrônica Acervo Médico 20 (November 4, 2022): e11136. http://dx.doi.org/10.25248/reamed.e11136.2022.

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Objetivo: Analisar as características do Glaucoma. Revisão bibliográfica: O glaucoma é um grupo de doenças cuja característica comum é a degeneração progressiva do nervo óptico, com perda de células ganglionares da retina, afinamento da camada de fibras nervosas da retina e aumento da escavação do disco óptico. Os sintomas visuais costumam ser uma característica tardia do glaucoma, quando a doença já está muito avançada. Dentre os muitos tipos de glaucoma, os mais frequentes são os glaucomas primários de ângulo aberto (GPAA) e de ângulo fechado (GPAF). Estima-se que 4,5 milhões ou mais de 12% de todos os casos de cegueira em todo o mundo foram resultado de glaucoma. Considerações finais: O Glaucoma é uma doença que cursa com redução da acuidade visual e, consequentemente, declínio da qualidade de vida dos pacientes. Embora o glaucoma progrida, geralmente, de forma lenta, é a causa mais comum de cegueira evitável irreversível no mundo. Dessa forma, é fundamental a conscientização acerca da doença a fim de propiciar o diagnóstico de forma mais precoce possível com tratamento adequado instituído pelo oftalmologista a fim de evitar a progressão do glaucoma.
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Loayza-Gamboa, Waldo. "Glaucomas secundarios." Diagnóstico 62, no. 1 (April 23, 2023): e437. http://dx.doi.org/10.33734/diagnostico.v62i1.437.

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Los glaucomas secundarios (GS) son un grupo de glaucomas causados por factores sistémicos y/o desórdenes oculares que producen hipertensión ocular y posterior daño del nervio óptico. Su etiología es muy variada, entre ellas: trauma ocular, uveíticos, pseudoexfoliativos, inducidas por cirugía ocular, retinopatia diabética, inducidas por el cristalino, relacionadas a medicamentos, principalmente corticoides, tumores, entre otros. Se estima que son alrededor de 6 millones de personas que tienen GS comparado con los 67 millones de personas con glaucoma primario alrededor del mundo. Desde el punto de vista epidemiológico y de intervención médica los más importantes son: los glaucomas secundarios a pseudoexfoliación, los glaucomas neovasculares y los glaucomas uveíticos. Realizamos una revisión actualizada, con los últimos reportes epidemiológicos y avances en genética de estas tres tipo de glaucoma secundario presentando las últimas modalidades de tratamiento farmacológico y quirúrgico.
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Sarwade, Prakash Pralhad, Madhav Kumar Mishra, Jyoti, Harpreet Kaur, C. Madhavi Latha, S. Hameedullah Sherief, Kavita Narayan Gaisamudre (Sarwade), Rapborlang Khongshei, and Parmar Hemantbhai Natubhai. "A Detailed Study of Glaucoma in Adults, Its Pathogenesis, Diagnosis and Management." Journal for Research in Applied Sciences and Biotechnology 3, no. 3 (July 21, 2024): 223–30. http://dx.doi.org/10.55544/jrasb.3.3.34.

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As the second most prevalent cause of blindness in the world, after cataracts, glaucoma diagnosis and treatment are crucial topics for general physicians to know. Glaucomas can often be categorised based on three key factors: the extent to which the anterior chamber angle is open or closed, the intensity of the onset (acute or chronic), and the underlying cause (primary or secondary). Most cases of glaucoma are primary, indicating the absence of any comorbidities. Nevertheless, secondary glaucomas may arise from a range of visual illnesses as their root cause. Individuals who are susceptible to developing chronic glaucoma should undergo regular eye examinations in order to detect the condition at an early stage and prevent the progressive deterioration of vision that may occur prior to diagnosis. Glaucoma leads to impairments in both central and peripheral vision fields due to damage to the optic nerve and the retinal nerve fibre layer. All current treatments, including drugs, lasers, and procedures, aim to reduce intraocular pressure (IOP) because it is the only factor that can be changed and is crucial in the progression of the condition. Pharmacotherapy is the usual first-line treatment, although its effectiveness is constrained by noncompliance, adverse effects, and cost. While laser and surgical methods offer the ability to effectively lower intraocular pressure (IOP) for extended periods and at a lower cost compared to medicine, they come with increased risks during the process and a higher likelihood of treatment not being successful. Various modern minimally invasive glaucoma operations have replaced conventional incisional therapies because they offer improved safety and reduced efficacy decreases. While the long-term success of minimally invasive glaucoma surgeries still needs to be determined by large-scale randomised trials, these methods have greatly transformed the surgical treatment of glaucoma.
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Hudock, Stephen A. "The Diagnosis and Treatment of Glaucoma." Guthrie Journal 64, no. 1 (March 1995): 4–8. http://dx.doi.org/10.3138/guthrie.64.1.004.

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Although the last 40 years have brought revolutionary changes in our understanding of the glaucomas and in our ability to diagnose and to treat them, the cause still eludes us, as does a cure. Since the most common forms of glaucoma are asymptomatic, we understand better than ever that routine eye examinations are critically important for diagnosing the glaucomas in their early stages, when they can be treated more successfully.
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Dissertations / Theses on the topic "Glaucoma"

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Baker, H. "Glaucoma awareness." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/16272/.

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This thesis investigates three different aspects of glaucoma awareness using both quantitative and qualitative methods. Patient Awareness: This qualitative study looked at patients perceptions of glaucoma. Participants (N=28) reported low levels of awareness of glaucoma prior to their diagnosis and assumed that symptoms were the ‘normal’ deterioration of eyesight. As symptoms have a gradual onset, participants had learnt to cope with diminishing sight ability. Findings suggested health promotion a priority to increase public awareness of the existence and symptoms of glaucoma among those at high risk. Current public awareness: This study looked to document public awareness and knowledge of glaucoma. The study used health knowledge questionnaires in three different populations: 1. nationally representative sample of 1009 people 2. telephone Interviews – 500 Isle of Wight, 226 Ealing 3. face-to-face interviews – 300 Ealing Between 71-93% of those interviewed by telephone had heard of glaucoma. However, only 23% of those interviewed face-to-face in Ealing reported having heard of glaucoma. We found a relatively high level of awareness and knowledge of glaucoma in the general UK population but identified at least one pocket of poor knowledge in a specific subpopulation. Can we change awareness? This study investigated whether a public health campaign could increase awareness and change help-seeking behaviour with respect to ocular health with residents in Southall, Ealing aged 60+. The health knowledge questionnaire from the previous study was used to assess the campaign. The health campaign comprised of four components. 1. Television 2. Local Press 3. Local Radio 4. Places of worship The results showed a significant increase in the number of people who had heard of Glaucoma rising from 22% to 53%. Before the intervention most people had heard about glaucoma from their GP, friend or relative. After intervention the majority (69%) had heard of glaucoma from the radio. This study showed a significant increase in awareness from using different kinds of media and showed radio to be the most effective in the target community. Although the campaign raised awareness it did not show a change in help seeking behaviour.
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Sharma, A. "Glaucoma care." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10041509/.

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What is the problem? The number of people coming to the hospital eye departments is likely to increase in the future, as a result of an ageing population, increased optometric case finding and raised public awareness. This fact coupled with the increased economic pressures in health-care financing, and the relative shortage of ophthalmologists in the United Kingdom is going to put significant strain on ophthalmology provision. As a result of these issues, there has been a drive by the government to move eye care into the community and to have more primary care involvement. A variety of alternative models have been proposed for patient care in the community. An important part of assessing these models is to investigate their relative cost effectiveness as well as safety, capacity and patient acceptance. What are the current models? There have been many shared care models that have been proposed. These have included the Community and Hospital Allied Network Glaucoma Evaluation Scheme (CHANGES), the Peterborough Scheme, East Devon Scheme, Waltham Forest Scheme and the Nottingham Scheme. One of the main schemes was the Bristol Shared Care Scheme. This scheme was shown not to be cost effective. It did show that community optometrist’s measurements were of comparable accuracy to those made in the hospital. The annual cost per patient follow-up by a community optometrist was £68.98-£108.98 compared to £14.50-£59.95 in the hospital. The main reason for the cost difference was due to a variation in the patient recall interval between the community and hospital. The second reason was due to the re-referral of patients back from the community clinics to the hospital clinics. What was our contribution? We developed an Integrated Glaucoma Care Model. This involved training and accrediting community optometrists to run Moorfields glaucoma clinics in their Optometric practices whilst alternating attending glaucoma clinics in the hospital. Our results showed that it was more costly to run the community based glaucoma clinics compared to hospital based clinics. These were the same findings as in the Bristol shared care model. The main reasons for the higher costs in the community were due to the large overhead costs of running the glaucoma scheme in the community optometric practices as well as fewer patients being seen in the community compared to the hospital. The community optometrists involved in our scheme were in general found to be competent, efficient and safe. The patient perspectives of our model were overall positive with a large majority of patients happy to be seen in the community again. What were our recommendations? Our main recommendation was to evolve our model to run the shared care scheme within the hospital setting to avoid the high rental costs of the optometric practices. This model is being successfully run at Bristol Eye Hospital where there is a complete shared care department involving optometrists. This type of model could utilise hospital optometrists but could also have accredited community optometrists attending the hospital and participating in such schemes. A second possibility could be to run these shared care schemes in hospital satellite settings or mobile units. An example of this is the Newmedica model. There is a clear requirement for cost effectiveness evaluation of such schemes along with an assessment of safety, capacity and patient acceptance before any conclusions can be reached.
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Lester, Karen Leah. "Reverse engineering glaucoma." Thesis, University of Liverpool, 2018. http://livrepository.liverpool.ac.uk/3022786/.

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Primary open angle glaucoma (POAG) is a leading cause of irreversible blindness worldwide, and the only modifiable risk factor is intraocular pressure (IOP). Glaucoma is a complex disease with specific endophenotypes, and disease pathogenesis is likely to involve multiple pathways linking genetic and environmental interactions. Growth factors present in the aqueous humour in POAG increase outflow resistance and elevate IOP. TGF-β2 alters ECM production and turnover in the trabecular meshwork (TM) and has been shown in numerous studies to play a role in the pathogenesis of POAG. No current pharmacological interventions target the deleterious effects of TGF-β2 of the TM which produced elevated IOP. In addition to TGF-β another well characterised glaucoma stimulus is corticosteroids. Corticosteroids are used in ophthalmology to decrease inflammation and preserve ocular function. However side effects including cataract, enhanced infection, and glaucoma are associated with their use. Small, naturally occurring regulatory genes, micro RNAs (miRNAs), target many genes downstream of TGF-β2 and are expressed in response to corticosteroids. The current work set out to identify key differentially expressed genes by RNA-Seq in the human trabecular meshwork (TM) in response to two glaucoma stimuli; TGF-β2 and dexamethasone; and investigate the ability of miRNAs to manipulate gene expression within the TM to reduce pathological insults central to glaucoma. Investigating the influence of TGF-β2 on gene expression in primary human TM cells demonstrated that the majority of the significantly differentially expressed genes were involved in extracellular matrix remodelling and actin cytoskeletal re-organisation likely via the RhoA signalling pathway. The influence of dexamethasone on gene expression in primary human TM cells identified genes involved in extracellular matrix remodelling and genes required for glucocorticoid receptor nuclear translocation. Differentially expressed miRNAs in healthy and glaucomatous human TM cells were identified by a miRNA microarray. Manipulation of validated mRNA targets by identified miRNAs indicated a complex regulatory network and in vitro functional analyses further identified regulation of actin cytoskeleton remodelling via miRNA inhibition. The findings of this study indicate that TGF-β2 and dexamethasone have significant effects on extracellular matrix remodelling and actin cytoskeletal re-organisation in human TM cells. The RNA-Seq and miRNA array have identified potential novel therapeutic targets for glaucoma.
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Ritland, Jon Ståle. "Primary Open-Angle Glaucoma & Exfoliative Glaucoma : Survival, Comorbidity and Genetics." Doctoral thesis, Norwegian University of Science and Technology, Department of Cancer Research and Molecular Medicine, 2008. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-2226.

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Lee, Simon. "Visual monitoring of glaucoma." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.291080.

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Theodossiades, Julia Elizabeth. "Glaucoma detection by optometrists." Thesis, University College London (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414489.

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Bernardes, Joana Roque. "Tratamento do glaucoma canino." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2008. http://hdl.handle.net/10400.5/865.

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Dissertação de Mestrado Integrado em Medicina Veterinária
A realização deste trabalho teve como objectivo o estudo das diferentes opções de tratamento do glaucoma canino, assim como, a busca da melhor opção para cada caso. Este trabalho baseou-se numa extensa pesquisa bibliográfica e no estudo de vários casos clínicos. Ao longo deste trabalho mencionei as várias opções médicas e cirúrgicas de tratamento do glaucoma canino. Além disso, referi o possível recurso a medicinas alternativas como a acupunctura. Existe um vasto campo de escolha no que se refere ao tratamento médico do glaucoma, com várias estratégias possíveis a seguir e muitos medicamentos a eleger. A eleição do tratamento baseia-se nas indicações, contra-indicações, efeitos secundários dos vários medicamentos e as possibilidades económicas do proprietário. O factor económico é, muitas vezes, um factor de peso na eleição do tratamento. Além deste vasto leque de opções que já são utilizadas, existe ainda um grande número em estudo que, num futuro próximo, poderão ser utilizadas para auxiliar no combate a esta afecção. Quando as opções médicas falham, dispomos ainda de várias técnicas cirúrgicas que podem ser efectuadas para resolução deste problema.
ABSTRACT - The goals of this work were to study the different options in the treatment of the canine glaucoma, as well as to search the best treatment option to each case. The study bases are a large amount of bibliography research and the study of several clinical cases. Through all this work I’ve mentioned the several medical and surgical options in the treatment of the canine glaucoma. Beyond that, I’ve mentioned the possible use of alternative medicines like acupuncture. There’s a large variety in what refers to the medical treatment of the canine glaucoma, with a big variety of possible strategies to choose and lot’s of medicines to use. The selection of the treatment has bases in the indications, adverse effects and secondary effects of the various medicines and the economical possibilities of the owner. The economical factor is, most of the times, very important in the treatment option to choose. In spite of all the options that are already in use, there are several options that still are being studied and that in a near future may be able to be used in the treatment of this disease. When the medical options fail, there still are several surgical options that can be used to solve this problem.
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Strouthidis, N. G. "Measuring progression in glaucoma." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1446208/.

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Background: Primary open angle glaucoma is characterised by progressive optic neuropathy associated with characteristic visual field loss. The ability to measure disease progression is of vital importance in the management of patients with glaucoma. Conventionally, disease progression has been monitored using static automated perimetry. Recently, devices which image the optic nerve head quantifiably have been introduced. This thesis sets out to compare structural and functional progression in ocular hypertensive subjects followed longitudinally using novel progression algorithms. Plan of research: The investigations may be considered in three parts. Firstly, the factors affecting the test-retest variability of the Heidelberg Retina Tomograph (HRT) are identified and methods to improve repeatability are investigated. Secondly, novel HRT trend and event analyses, based on the findings of the test-retest studies, are compared with established field progression techniques in ocular hypertensive and control subjects. Thirdly, a previously described novel spatial filter is assessed in terms of its impact on the monitoring of visual field progression and in terms of its agreement with a previously described 'structural' map. Results: Rim area was identified as the most repeatable HRT parameter its variability can be improved by using the 320pm reference plane and by using only good quality images. Agreement as regards structural and functional progression was poor, regardless of the estimated specificities of the algorithms used or technique adopted. The novel spatial filter appeared to confer some advantage in terms of specificity, comparable to the effect of confirmatory testing. The functional relationship between test-points, characterised by the filter, correlated well with the expected structural pattern. Clinical significance: The poor agreement suggests that the monitoring of both structure and function is essential to provide the best chance of detecting progression at all stages of the disease. Spatial filtering techniques may provide some additional benefit in the monitoring of progression, particularly once structural data are incorporated.
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Bernier, Sophie. "Épidémiologie génétique du glaucome primaire à angle ouvert : étude de deux mutations du gène TIGR obsrvées chez deux familles de l'est du Québec /." Thèse, Ste-Foy : Chicoutimi : Université Laval ;. Université du Québec à Chicoutimi, 1999. http://theses.uqac.ca.

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Gal, Michelle Rose. "A novel glaucoma drainage valve." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0015/MQ58723.pdf.

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Books on the topic "Glaucoma"

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Glaucoma. Oxford: Oxford University Press, 2012.

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Grehn, Franz, and Robert Stamper, eds. Glaucoma. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-69475-5.

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Ichhpujani, Parul, ed. Glaucoma. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8457-8.

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Jakobs, Tatjana C., ed. Glaucoma. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7407-8.

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Grehn, Franz, and Robert Stamper. Glaucoma. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18633-2.

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National Institutes of Health (U.S.), ed. Glaucoma. Bethesda, Md: National Institutes of Health, 1988.

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E, Cairns J., ed. Glaucoma. London: Grune & Stratton, 1986.

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E, Cairns J., ed. Glaucoma. London: Grune & Stratton, 1986.

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Glaucoma. 2nd ed. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2012.

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Gilbert, Smolin, and Friedlaender Mitchell H, eds. Glaucoma. Boston: Little, Brown, 1990.

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Book chapters on the topic "Glaucoma"

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Lee, Richard K. "Glaucomas: Pseudoexfoliation Glaucoma." In Pearls of Glaucoma Management, 337–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_44.

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Shuba, Lesya, and Young H. Kwon. "Glaucomas: Uveitic Glaucoma." In Pearls of Glaucoma Management, 371–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_48.

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Wenick, Adam S., and Alan L. Robin. "Glaucomas: Neovascular Glaucoma." In Pearls of Glaucoma Management, 379–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_49.

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Lee, Richard K. "Glaucomas: Pseudoexfoliation Glaucoma." In Pearls of Glaucoma Management, 411–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49042-6_44.

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Shuba, Lesya, and Young H. Kwon. "Glaucomas: Uveitic Glaucoma." In Pearls of Glaucoma Management, 447–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49042-6_48.

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Wenick, Adam S., and Alan L. Robin. "Glaucomas: Neovascular Glaucoma." In Pearls of Glaucoma Management, 457–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49042-6_49.

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de Moraes, Carlos Gustavo V., and Remo Susanna. "Glaucomas: Pigment Dispersion Glaucoma and Angle Recession Glaucoma." In Pearls of Glaucoma Management, 345–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_45.

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Sugiyama, Kazuhisa, and Yoshiaki Kitazawa. "Glaucomas: Managing Normal-Tension Glaucoma." In Pearls of Glaucoma Management, 331–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_43.

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Giaconi, JoAnn A. "Glaucomas: Glaucoma and the Cornea." In Pearls of Glaucoma Management, 363–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68240-0_47.

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Sugiyama, Kazuhisa, and Yoshiaki Kitazawa. "Glaucomas: Managing Normal-Tension Glaucoma." In Pearls of Glaucoma Management, 403–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49042-6_43.

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Conference papers on the topic "Glaucoma"

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Ethier, C. Ross, Richie Abel, E. A. Sander, John G. Flanagan, and Michael Girard. "Next-Generation Techniques for Analysis of Lamina Cribrosa Microstructure." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80523.

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Glaucoma describes a group of potentially blinding ocular disorders, afflicting c. 60 million people worldwide. Of these, c. 8 million are bilaterally blind, estimated to increase to 11 million by 2020. The central event in glaucoma is slow and irreversible damage of retinal ganglion cells, responsible for carrying visual information from the retina to the brain (Figure 1). Intraocular pressure (IOP) is a risk factor for glaucoma1–4, and significant, sustained IOP reduction is unequivocally beneficial in the clinical management of glaucoma patients2, 3, 5. Unfortunately, we do not understand how elevated IOP leads to the loss of retinal ganglion cells.
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Gupta, Krati, Anshul Thakur, Michael Goldbaum, and Siamak Yousefi. "Glaucoma Precognition: Recognizing Preclinical Visual Functional Signs of Glaucoma." In 2020 IEEE/CVF Conference on Computer Vision and Pattern Recognition Workshops (CVPRW). IEEE, 2020. http://dx.doi.org/10.1109/cvprw50498.2020.00518.

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Lovasik, John V., Hélène Kergoat, Pierre Forcier, Robert Wojciechowski, and Etty Bitton. "Topographic and Hemodynamic Changes in the Optic Nerve Head in Humans Subsequent to Experimental Modulation of the Intraocular Pressure." In Vision Science and its Applications. Washington, D.C.: Optica Publishing Group, 1995. http://dx.doi.org/10.1364/vsia.1995.sua3.

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The attrition of nerve fibers in the peripapillary area and the optic nerve head (ONH) in glaucoma has been attributed to mechanical compression of, or aberrant blood flow to the ganglion cell axons comprising the optic nerve1. Recently, greater attention has been focussed on the vascular factors in glaucoma2. Much of the delay in understanding the pathophysiology of glaucoma has been the absence of clinically usable instruments for reliably quantifying subtle structural changes in the ONH, and blood flow in the optic nerve. Fortunately, Petrig and Riva recently demonstrated the applicability of laser doppler principles for measuring blood flow in the ONH in man3, and confocal imaging principles have been adapted for computer reconstruction of serial laser scanned planes of the ophthalmoscopically visible ONH to provide highly accurate and reliable three-dimensional images4 for fundamental/clinical research.
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Shankar, Smitha, and Michael Austin. "MEMS glaucoma monitoring device." In The 14th International Symposium on: Smart Structures and Materials & Nondestructive Evaluation and Health Monitoring, edited by Vijay K. Varadan. SPIE, 2007. http://dx.doi.org/10.1117/12.716519.

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Ko, Match Wai Lun. "Translational Biomechanics in Glaucoma." In Advances in Memristor Circuits and Bioinspired Systems (WSCAS-2015). Singapore: Research Publishing Services, 2015. http://dx.doi.org/10.3850/978-981-09-4424-7_work2.

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Bullimore, Mark A., Joanne M. Wood, and Kirk Swenson. "Motion Perception in Glaucoma." In Noninvasive Assessment of the Visual System. Washington, D.C.: Optica Publishing Group, 1992. http://dx.doi.org/10.1364/navs.1992.sub1.

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The early detection of optic nerve damage is a prerequisite for effective therapy in primary open-angle glaucoma (Kass et al., 1990). An alarmingly large proportion of retinal ganglion cells may be damaged, however, before visual field defects are observed with conventional manual or automated perimetry (Quigley et al., 1982; Quigley et al., 1989). This implies that either there is widespread redundancy within the retina such that substantial cell death can occur without loss of visual performance or that our current clinical procedures do not test the function of the cells that are damaged in early glaucoma. Recent studies lead us to believe that the latter of these two hypotheses may be correct.
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Girard, Michaël J. A., Lyn Zimmo, Edward T. White, Jean Martial Mari, C. Ross Ethier, and Nicholas G. Strouthidis. "Towards a Biomechanically-Based Diagnosis for Glaucoma: In Vivo Deformation Mapping of the Human Optic Nerve Head." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80557.

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Glaucoma is the most common cause of irreversible blindness. It is associated with a progressive loss of cells within the optic nerve head (ONH) at the back of the eye. Glaucoma remains incurable and its exact causes are not well understood. It was once thought to occur only in eyes with elevated pressure (i.e., intraocular pressure or IOP) and to date, lowering IOP is the only clinical treatment proven to be beneficial for slowing the progression of glaucoma. However, the success rate of such therapy is only 50%. Multiple lines of evidence now indicate that IOP is not the only important risk factor in the disease. For instance, while some patients develop glaucoma at elevated IOP (high-tension glaucoma), some develop glaucoma at normal IOP levels (normal-tension glaucoma), and some others with elevated IOP do not develop glaucoma at all.
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Rajabi, Shadi, Craig A. Simmons, and C. Ross Ethier. "Design of Experiments for Exposure of Astrocytes to Elevated Hydrostatic Pressure and Hypoxia." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192558.

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Glaucoma, a chronic optic neuropathy, is the second most common cause of blindness, affecting 67 million people worldwide. The damage in glaucoma occurs at the optic nerve head (ONH), where the axons of the retinal ganglion cells leave the eye posteriorly. Glaucoma is frequently associated with elevated intraocular pressure (IOP), and visual field loss can be prevented by significant lowering of IOP. Hence, the role of pressure in glaucoma is important. Unfortunately, the mechanism by which pressure leads to vision loss in glaucoma is very poorly understood.
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Puangarom, S., A. Twinvitoo, S. Sangchocanonta, A. Munthuli, P. Phienphanich, R. Itthipanichpong, K. Ratanawongphaibul, et al. "3-LbNets: Tri-Labeling Deep Convolutional Neural Network for the Automated Screening of Glaucoma, Glaucoma Suspect, and No Glaucoma in Fundus Images." In 2023 45th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2023. http://dx.doi.org/10.1109/embc40787.2023.10340102.

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Mohammad, Suraya, and D. T. Morris. "Texture analysis for glaucoma classification." In 2015 International Conference on BioSignal Analysis, Processing and Systems (ICBAPS). IEEE, 2015. http://dx.doi.org/10.1109/icbaps.2015.7292226.

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Reports on the topic "Glaucoma"

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Dakov, Nikolai, Stanislava Kostova, and Ivan Tanev. Selective Laser Trabeculoplasty in Primary Open-angle Glaucoma and Pseudoexfoliation Glaucoma – Efficiency and Correlated Parameters. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, February 2018. http://dx.doi.org/10.7546/crabs.2018.02.17.

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Dakov, Nikolai, Stanislava Kostova, and Ivan Tanev. Selective Laser Trabeculoplasty in Primary Open-angle Glaucoma and Pseudoexfoliation Glaucoma – Efficiency and Correlated Parameters. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, February 2018. http://dx.doi.org/10.7546/grabs2018.2.17.

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Xu, Genghuan, Guihua Xu, Xiaoyi Wang, Wei Wang, Juanjuan Wang, Lili Chen, and Zilin Chen. Association of Matrix Metalloproteinases-9 Polymorphisms with Glaucoma: a Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0079.

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Jiang, Fei, and Huarong Wu. The Value of Machine Learning in Glaucoma Diagnosis: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2024. http://dx.doi.org/10.37766/inplasy2024.6.0010.

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Zhou, Rouxi. Laser trabeculoplasty for open-angle glaucoma: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0031.

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Skutnik, Bolesh J. Diode Pumped Frequency Doubled ND:Yag Laser for the Treatment of Glaucoma and Retinal Disease. Fort Belvoir, VA: Defense Technical Information Center, October 2002. http://dx.doi.org/10.21236/ada408804.

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Skutnik, Bolesh J. Diode Pumped Frequency Doubled ND:YAG Laser for the Treatment of Glaucoma and Retinal Disease. Fort Belvoir, VA: Defense Technical Information Center, November 2002. http://dx.doi.org/10.21236/ada409052.

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Liu, Guoying, Shao Dongping, and Zhao Liuning. A meta-analysis of laser peripheral iridectomy in the treatment of acute angle-closure glaucoma. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2021. http://dx.doi.org/10.37766/inplasy2021.8.0051.

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Li, Xin. Efficacy of Paul drainage device in the treatment of glaucoma : A single-arm Meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2024. http://dx.doi.org/10.37766/inplasy2024.5.0013.

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Zhang, Jian. Can neuroprotection effectively manage primary open-angle glaucoma? a protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0107.

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