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1

Goulbourne, P. Andrew. Evidence for the role of fimbriae in the adhesion of bacteroides gingivalis to actinomyces viscosus. Ottawa: National Library of Canada, 1990.

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2

Loomer, Peter Michael. The direct effects of Porphyromonas Gingivalis 2561 on bone formation and mineral resorption in vitro. [Toronto: University of Toronto, Faculty of Dentistry], 1997.

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3

Sun, Frank. Identification of Porphyromonas (Bacteroides) Gingivalis outer membrane proteins that bind to and degrade human matrix proteins. [Toronto: Faculty of Dentistry, University of Toronto, 1992.

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4

Gingival. Palencia, España: Menoscuarto Ediciones, 2012.

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5

Kasaj, Adrian, ed. Gingival Recession Management. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70719-8.

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6

Ruiz, Jose-Luis. Supra-Gingival Minimally Invasive Dentistry. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118976449.

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7

Chou, Debra Hsin-I. TNF-r̀egulation of phagocytosis in human gingival fibroblasts. Ottawa: National Library of Canada, 1995.

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8

Chou, Debra Hsin-I. TNF-[alpha] regulation of phagocytosis in human gingival fibroblasts. [Toronto: Faculty of Dentistry, University of Toronto], 1995.

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9

Issa, Roger. The effect of benzydamine HCL on human plaque and gingival tissues. [Toronto]: Faculty of Dentistry, University of Toronto, 1986.

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10

Andlin-Sobocki, Anna. Gingival recession, keratinized and attached gingiva in anterior teeth of children. Umeå: [University of Umeå], 1993.

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11

Apse, Peter. Clinical and laboratory protocol for microbiological investigation of the gingival sulcus. [Toronto: Faculty of Dentistry, University of Toronto, 1987.

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12

Tessier, Jean-François. Relationship between periodontal probing velocity and gingival inflammation in human subjects. [Toronto: Faculty of Dentistry, University of Toronto], 1992.

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13

Dahllöf, Goran. Phenytoin-induced gingival overgrowth in epileptic children: A clinical, histological and biochemical study. Stockholm: Department of Pedodontics, School of Dentistry, Karolinska Institutet, 1986.

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14

Teng, Andy Y. T. Gingival crevicular fluid gelatinase and its relationship to periodontal disease in human subjects. [Toronto: Faculty of Dentistry, University of Toronto], 1991.

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15

Németh, Endre. Responses of gingival fibroblast and endothelial cell populations to experimentally-inducedinflammation in monkeys. Ottawa: National Library of Canada, 1993.

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16

Németh, Endre. Responses of gingival fibroblast and endothelial cell populations to experimentally-induced inflammation in monkeys. [Toronto: Faculty of Dentistry, University of Toronto], 1993.

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17

Yang, Po Fong. Filamentous actin disruption and diminished inositol phosphate response in gingival fibroblasts caused by Treponema denticola. [Toronto: University of Toronto, Faculty of Dentistry], 1998.

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18

Battikhi, Tulin. Treponema Denticola outer membrane extract enhances the phagocytosis of collagen coated-beads by human gingival fibroblasts. [Toronto: University of Toronto, Faculty of Dentistry], 1998.

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19

Karim, Minaz. Controlled force measurements of gingival attachment level made with the Toronto automated probe using electronic guidance. [Toronto: Faculty of Dentistry, University of Toronto], 1989.

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20

Saadoun, Andre P. Esthetic soft tissue management of teeth and implants. Chichester, West Sussex: John Wiley & Sons, 2013.

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21

Hatfield, Densie. Tooth & gum care. Springhouse, Pa: Springhouse Corp., 1986.

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22

Gottlow, Jan. New attachment formation by guided tissue regeneration. Göteborg, Sweden: University of Göteborg, Faculty of Odontology, Dept. of Periodontology, 1986.

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23

Silvestri, Livia. The regulation of intracellular calcium ion concentration and mitochondrial function by cyclosporin A: a putative mechanism for the pathogenesis of gingival overgrowth. [Toronto: Faculty of Dentistry, University of Toronto], 2000.

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24

Wouters, Francis Roger. Objective methods of recording gingivitis and marginal periodontitis in man: A laser, radiography- and computer-based study of gingival swelling and interproximal alveolar bone levels. Stockholm: Kongl. Carolinska Medico Chirurgiska Institutet, 1988.

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25

Reversing Gum Disease Naturally. New York: John Wiley & Sons, Ltd., 2003.

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26

N, Shah Haroun, Mayrand D, and Genco Robert J, eds. Biology of the species Porphyromonas gingivalis. Boca Raton: CRC Press, 1993.

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27

Papaioannou, W. The Adhesion of Porphyromonas Gingivalis and Periodontitis. Leuven University Press, 1998.

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28

Andreou, Vana. Resveratrol mediated reversal of aryl hydrocarbon and porphyromonas gingivalis lipopolysaccharide induced inhibition of osteogenesis in vitro. 2002.

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29

Immunization with Porphyromonas Gingivalis Cysteine Protease: Effects on Experimental Gingivitis and Ligature-Induced Periodontitis in Macaca Fascicularis. Storming Media, 1997.

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30

Schwarz-Faulkner, Susanne *. The development and application of an "in vitro" assay to quantitate the adherence of "Bacteroides gingivalis" to "Actinomyces viscosus" and saliva-coated hydroxyapatite. 1988.

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31

J, Genco Robert, ed. Molecular pathogenesis of periodontaldisease. Washington, D.C: ASM Press, 1994.

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32

J, Genco Robert, ed. Molecular pathogenesis of periodontal disease. Washington, D.C: ASM Press, 1994.

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33

Lafaurie, Gloria Inés. Gingival Tissue and Pregnancy. INTECH Open Access Publisher, 2011.

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34

Park, Jae Hyun. Clinical Considerations of Open Gingival Embrasures. INTECH Open Access Publisher, 2012.

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35

Kasaj, Adrian. Gingival Recession Management: A Clinical Manual. Springer, 2018.

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36

Kasaj, Adrian. Gingival Recession Management: A Clinical Manual. Springer, 2018.

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37

Wilson, John W., and Lynn L. Estes. Gastrointestinal Infections. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199797783.003.0113.

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•Ludwig angina: Acute soft tissue infection usually of dental origin; spreads rapidly and is bilateral; involves submandibular and sublingual spaces and can spread to the neck; may include respiratory obstruction from edema•Acute necrotizing ulcerative gingivitis (eg, Vincent angina, trench mouth): Mixed bacterial infection with gingival ulcerations and gingival breakdown, usually due to poor dental hygiene...
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38

Panagakos, Fotinos, ed. Gingival Diseases - Their Aetiology, Prevention and Treatment. InTech, 2011. http://dx.doi.org/10.5772/877.

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39

Eddin Omar Al Ostwani, Alaa, ed. Gingival Disease - A Professional Approach for Treatment and Prevention. IntechOpen, 2019. http://dx.doi.org/10.5772/intechopen.73822.

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40

Periodontal and Gingival Health and Diseases: Children, Adolescents and Young Adults. Informa Healthcare, 2001.

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41

Ruiz, Jose Luis. Supra-Gingival Minimally Invasive Dentistry: A Healthier Approach to Esthetic Restorations. Wiley & Sons, Incorporated, John, 2017.

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42

Ruiz, Jose Luis. Supra-Gingival Minimally Invasive Dentistry: A Healthier Approach to Esthetic Restorations. Wiley & Sons, Incorporated, John, 2017.

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43

Enrique, Bimstein, ed. Periodontal and gingival health and diseases: Children, adolescents, and young adults. London: Martin Dunitz, 2001.

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44

Saadoun, Andre P. Esthetic Soft Tissue Management of Teeth and Implants. Wiley & Sons, Incorporated, John, 2012.

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45

Saadoun, Andre P. Esthetic Soft Tissue Management of Teeth and Implants. Wiley & Sons, Incorporated, John, 2012.

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46

Saadoun, Andre P. Esthetic Soft Tissue Management of Teeth and Implants. Wiley & Sons, Incorporated, John, 2012.

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47

Sybert, Virginia P. Disorders of Epidermal Appendages. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195397666.003.0003.

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Hair – Alopecias – Loose Anagen Hair – Male Pattern Baldness – Marie Unna Syndrome – Hirsutism – Gingival Fibromatosis and Hypertrichosis – Hypertrichosis Lanuginosa Congenita – Leprechaunism – Localized Hypertrichosis – Polycystic Ovarian Disease – Hair Shaft Abnormalities, Isolated – Monilethrix – Pili Annulati – Pili Torti – Pili Trianguli Et Canaliculi – Trichorrhexis Invaginata – Trichorrhexis Nodosa – Woolly Hair – Hair Shaft Abnormalities, Syndromic – Menkes Disease – Trichodentoosseous Syndrome – Trichorhinophalangeal Syndrome – Trichothiodystrophy – Nails – Nail Disorders, Isolated – Congenital Malalignment of the Great Toenails – Familial Dystrophic Shedding of the Nails – Leukonychia – Twenty-Nail Dystrophy – Nail Disorders, Syndromic – Nail-Patella Syndrome – Onychotrichodysplasia and Neutropenia – Pachyonychia Congenita – Sweat Glands – Hidradenitis Suppurativa – Hyperhidrosis – Multiple Syringomas – Sebaceous Glands – Eruptive Vellus Hair Cysts – Familial Dyskeratotic Comedones – Oral-Facial-Digital Syndrome Type I – Steatocystoma Multiplex – Ectodermal Dysplasia Syndromes – AEC Syndrome – Clouston Syndrome – EEC Syndrome – Focal Facial Ectodermal Dysplasia – GAPO Syndrome – Hypohidrotic Ectodermal Dysplasia – Tooth and Nail Syndrome
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48

MacDonald, Kevin G. An assessment of interproximal crestal bone topography and gingival papilla morphology adjacent to sintered porous-surfaced dental implants. 2006.

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49

Sybert, Virginia P. Disorders of Epidermal Appendages. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276478.003.0003.

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Chapter 3 starts by covering conditions of the hair, including Alopecias (Loose Anagen Hair, Male Pattern Baldness, and Marie Unna Syndrome), Hirsutism (Gingival Fibromatosis and Hypertrichosis, Hypertrichosis Lanuginosa Congenita, Leprechaunism, and Localized Hypertrichosis), and Hair Shaft Abnormalities (including Monilethrix, Pili Annulati, Pili Torti, Pili Trianguli Et Canaliculi, Trichorrhexis Invaginata, Trichorrhexis Nodosa, Woolly Hair, Menkes Disease, Trichodentoosseous Syndrome, Trichorhinophalangeal Syndrome, and Trichothiodystrophy). It then covers conditions of the nails, including Congenital Malalignment of the Great Toenails, Familial Dystrophic Shedding of the Nails, Leukonychia, Twenty-Nail Dystrophy, Nail-Patella Syndrome, Onychotrichodysplasia and Neutropenia, and Pachyonychia Congenita). Conditions of the Sweat Glands (Hidradenitis Suppurativa, Hyperhidrosis, and Multiple Syringomas), Sebaceous Glands (Eruptive Vellus Hair Cysts, Familial Dyskeratotic Comedones, Oral-Facial-Digital Syndrome Type I, and Steatocystoma Multiplex), and Ectodermal Dysplasia Syndromes (AEC Syndrome, Clouston Syndrome, EEC Syndrome, Focal Facial Ectodermal Dysplasia, GAPO Syndrome, Hypohidrotic Ectodermal Dysplasia, and Tooth and Nail Syndrome) are also covered. Each condition is discussed in detail, including dermatologic features, associated anomalies, histopathology, basic defect, treatment, mode of inheritance, prenatal diagnosis, and differential diagnosis.
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50

Senzon, Sandra. Reversing Gum Disease Naturally: A Holistic Home Care Program. Wiley & Sons, Incorporated, John, 2007.

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