Relevant bibliographies by topics / GEWE

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'GEWE'

Author: Grafiati
Published: 11 September 2023

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Journal articles on the topic "GEWE"

1

Lorenz, Friederike. "Gewe(r)bliche Ware." kma - Klinik Management aktuell 11, no. 09 (September 2006): 16–19. http://dx.doi.org/10.1055/s-0036-1573889.

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Eigentlich sollte das Gesetz nur an EU-Standards angeglichen werden. Doch das Bundesgesundheitsministerium schießt ohne Not über das Ziel hinaus. Hornhäute und Herzklappen sind künftig Arzneimitteln rechtlich gleichgestellt. Die Krankenhäuser sind die Dummen, sie müssen eine technische Ausrüstung wie Pharma­hersteller zur Gewebeaufbewahrung bereitstellen.
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Rab, M. Abdur, and Jasmin Akhter. "Sine-Function Method In The Soliton Solution Of Nonlinear Partial Differential Equations." GANIT: Journal of Bangladesh Mathematical Society 32 (February 4, 2013): 55–60. http://dx.doi.org/10.3329/ganit.v32i0.13647.

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In this paper we establish a traveling wave solution for nonlinear partial differential equations using sine-function method. The method is used to obtain the exact solutions for three different types of nonlinear partial differential equations like general equal width wave equation (GEWE), general regularized long wave equation (GRLW), general Korteweg-de Vries equation(GKDV) which are the important soliton equations DOI: http://dx.doi.org/10.3329/ganit.v32i0.13647 GANIT J. Bangladesh Math. Soc. (ISSN 1606-3694) 32 (2012) 55-60
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Agarwala, Ajita, and Rishu Chaujar. "Noise analysis of gate electrode work function engineered recessed channel (GEWE-RC) MOSFET." Journal of Physics: Conference Series 367 (May 21, 2012): 012013. http://dx.doi.org/10.1088/1742-6596/367/1/012013.

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Obermayer-Pietsch, B., and M. Ulbing. "MicroRNAs in der Osteologie." Osteologie 24, no. 04 (2015): 219–24. http://dx.doi.org/10.1055/s-0037-1622069.

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ZusammenfassungMicroRNAs (miRNAs) sind einzelsträngige, nichtkodierende RNA-Elemente, die die Umsetzung von Genen zu Proteinen über die Transkription und Translation beeinflussen können. Durch ihre Bindung an mRNA greifen miRNAs als Regulatoren in die verschiedensten Signalwege im Organismus ein. Dabei beeinflussen miRNAs unterschiedlichste biologische Prozesse, darunter Zelldifferenzierung, Proliferation oder Organ-/Gewe-beentwicklung und sind auch in der Knochenhomöostase wichtig. Sie spielen eine Rolle bei Knochenformation, -resorption, -remodelling und Knochenzelldifferenzierung. Damit sind unterschiedliche Expressionen von miRNA ein wichtiger Pathomechanismus bei osteologischen Erkrankungen wie der Osteoporose, der renalen Osteodystrophie oder von Knochenmalignomen. In dieser Übersichts arbeit werden miRNAs definiert und charakterisiert sowie deren Rollen in der Osteologie und bei osteologischen Erkrankungen dargestellt.
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Gupta, Neha, and Rishu Chaujar. "Quantum analysis based extraction of frequency dependent intrinsic and extrinsic parameters for GEWE-SiNW MOSFET." Journal of Computational Electronics 16, no. 1 (January 24, 2017): 61–73. http://dx.doi.org/10.1007/s10825-016-0949-4.

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Gupta, Neha, Ajay Kumar, and Rishu Chaujar. "Oxide bound impact on hot-carrier degradation for gate electrode workfunction engineered (GEWE) silicon nanowire MOSFET." Microsystem Technologies 22, no. 11 (May 7, 2015): 2655–64. http://dx.doi.org/10.1007/s00542-015-2557-9.

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Chaujar, Rishu, Ravneet Kaur, Manoj Saxena, Mridula Gupta, and R. S. Gupta. "Design considerations and impact of technological parametric variations on RF/microwave performance of GEWE-RC MOSFET." Microwave and Optical Technology Letters 52, no. 3 (January 8, 2010): 652–57. http://dx.doi.org/10.1002/mop.25008.

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Gupta, Neha, Ajay Kumar, and Rishu Chaujar. "Impact of device parameter variation on RF performance of gate electrode workfunction engineered (GEWE)-silicon nanowire (SiNW) MOSFET." Journal of Computational Electronics 14, no. 3 (June 18, 2015): 798–810. http://dx.doi.org/10.1007/s10825-015-0715-z.

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Gupta, N., and R. Chaujar. "Investigation of temperature variations on analog/RF and linearity performance of stacked gate GEWE-SiNW MOSFET for improved device reliability." Microelectronics Reliability 64 (September 2016): 235–41. http://dx.doi.org/10.1016/j.microrel.2016.07.095.

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Gupta, Neha, and Rishu Chaujar. "Optimization of high-k and gate metal workfunction for improved analog and intermodulation performance of Gate Stack (GS)-GEWE-SiNW MOSFET." Superlattices and Microstructures 97 (September 2016): 630–41. http://dx.doi.org/10.1016/j.spmi.2016.07.021.

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Dissertations / Theses on the topic "GEWE"

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Gewe, Susanne [Verfasser], and KLAUS-MICHAEL [Akademischer Betreuer] BRAUMANN. "Gesundheitsbildung im Sport an der Berufsschule : Untersuchung, Entwicklung und praxisbezogene Überprüfung eines Curriculumbausteins im Fach Sport am Beispiel von Medizinischen und Zahnmedizinischen Fachangestellten in der Ausbildung / Susanne Gewe. Betreuer: Klaus-Michael Braumann." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2014. http://d-nb.info/1046460242/34.

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2

Vasanwala, Farha Huseini. "Gene manipulations for cancer gene therapy." Diss., The University of Arizona, 2002. http://hdl.handle.net/10150/289776.

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Tumor cells can be modified with cytokine genes such as the Interleukin-2 (IL-2) gene. The levels of IL-2 expressed are critical for successful treatment. We have tried to achieve higher levels of IL-2 than those currently available by conventional plasmids. Use of a transcriptional activator, e.g; the tat gene along with the HIV promoter driving the IL-2 gene, greatly increased IL-2 levels compared to widely used cytomegalovirus (CMV) driven plasmids. Control of the tat gene with an inducible promoter, i.e; the human HSP70B promoter, permitted control of gene expression. The inducibility of the HSP70B promoter by heat, γ-radiation and geldanamycin (a chemotherapeutic drug) allowed for a combinatorial approach to cancer treatment with hyperthermia, radiation therapy and chemotherapy. Also a brief heat treatment of 10 min at 42°C of target cells increased plasmid uptake, and higher levels of gene expression could be achieved. Another arm of immunotherapy is adoptive therapy with Tumor Infiltrating Lymphocytes (TILs). Insufficient numbers of tumor-specific T-cells limit the success of TIL therapy. An alternative approach to overcome this limitation is to transfer tumor-specific T cell receptor (TCR) into peripheral T-cells, redirecting their specificity to the tumor cell. To prove the feasibility of this technique, T-cell receptors were identified and cloned from hybridomas specific for the tumor cell line, MO5. A three domain single chain T-cell receptor was also constructed from the tumor-specific TCR genes to investigate the ability of a single chain T-cell receptor to activate T-cells. The CD3ζ chain was linked to the single chain to allow signal transduction upon antigen recognition by the TCR. The full length and the single chain TCR were cloned into a retroviral vector and transfected into mouse and human T cell lines. Cell surface expression of the chains were detected by flow cytometry. Functionality of the transfected TCR chains was assessed by IL-2 secretion on co-culture of the tumor cell line MO5 and the transfected T-cells. The two different approaches described here, i.e; higher levels of IL-2 for IL-2 gene therapy and specific redirection of T-cells can potentially greatly enhance the success rate of cancer treatment.
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Bashiardes, Evy. "Gene polymorphisms, gene expression and atherosclerotic plaques." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420882.

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Payne, Katie Emma. "β₃ integrin gene polymorphisms and gene regulation." Thesis, University of Leeds, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413197.

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Lodhi, Saad Salman Khan. "Unraveling gene gene interactions in rheumatoid arthritis." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-19908.

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Rheumatoid arthritis (RA) is a systematic autoimmune disorder characterized by a persistent joint inflammation. A subset of HLA-DRB1 alleles known as shared epitope (SE) are the strongest genetic risk factors to develop anti-citrullinated protein antibody positive (ACPA-positive) RA. A strong enrichment of interactions exists between ACPA-positive RA-associated genetic variants and HLA-DRB1 SE alleles in disease development. Pathway analysis was performed to investigate how the interactions between risk variants (SNPs) with HLA-DRB1 from a previous study related to ACPA-positive RA. Gene-gene interactions analysis was performed between non-HLA risk variants and HLA-DRB1 SE alleles in SRQ biobank (SRQb) case-control cohort. We also evaluated whether the reported gene-gene interactions from a previous study relate to methotrexate (MTX) response for RA patients, at three and six months of follow-up in EIRA study. Interaction analysis based on an additive model was performed to understand the combined effect of two risk factors in the disease and treatment response. Two out of three genes from pathway analysis that were RXRA and NR3C1, pointed to ACPA-positive RA related important pathways including vitamin D receptor (VDR) pathway and adipocytokine signaling pathway. The replication analysis in SRQ-case-control study showed 2.627% of the evaluated SNPs insignificant additive interaction with HLA-DRB1 SE alleles. No interactions were significant in relation to the response to MTX monotherapy after 3 and 6 months follow-up. This project provides new insights into the gene-gene interactions in the study of ACPA-positive RA and suggests candidate genes for future functional studies.
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Smith, Erin N. "Gene-environment interaction in yeast gene expression /." Thesis, Connect to this title online; UW restricted, 2008. http://hdl.handle.net/1773/5025.

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Li, Wei. "Analyzing Gene Expression Data in Terms of Gene Sets: Gene Set Enrichment Analysis." Digital Archive @ GSU, 2009. http://digitalarchive.gsu.edu/math_theses/79.

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The DNA microarray biotechnology simultaneously monitors the expression of thousands of genes and aims to identify genes that are differently expressed under different conditions. From the statistical point of view, it can be restated as identify genes strongly associated with the response or covariant of interest. The Gene Set Enrichment Analysis (GSEA) method is one method which focuses the analysis at the functional related gene sets level instead of single genes. It helps biologists to interpret the DNA microarray data by their previous biological knowledge of the genes in a gene set. GSEA has been shown to efficiently identify gene sets containing known disease-related genes in the real experiments. Here we want to evaluate the statistical power of this method by simulation studies. The results show that the the power of GSEA is good enough to identify the gene sets highly associated with the response or covariant of interest.
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Carmo, Ana Paula Santos do. "Estudo da distribuiÃÃo da frequÃncia de genÃtipos de Helicobacter pylori em lesÃes gÃstricas." Universidade Federal do CearÃ, 2011. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=9557.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior
A bactÃria Helicobacter pylori, à um agente etiolÃgico bem estabelecido para o desenvolvimento de lesÃes gÃstricas como gastrite, Ãlcera pÃptica, metaplasia e doenÃas malignas, com alta incidÃncia de infecÃÃo em todo mundo, porÃm cerca de 80% dos indivÃduos infectados permanecem assintomÃticos e apenas uma minoria desenvolve doenÃas a ela relacionadas. Estudos tÃm sido realizados na tentativa de identificar a relaÃÃo de genes determinantes da patogenicidade de H. pylori, entretanto, atà o momento apenas os genes cagA e o alelo de vacA s1m1 sÃo considerados marcadores de virulÃncia para o desenvolvimento de lesÃes gÃstricas mais graves. A bactÃria H. pylori possui uma alta variabilidade genÃtica, sendo que um dos mecanismos propostos para o desenvolvimento de lesÃo seria atravÃs da inflamaÃÃo. DiferenÃas na intensidade de respostas inflamatÃrias poderiam ser decorrentes do perfil genotÃpico da cepa infectante. Recentes trabalhos apontam a importÃncia dos genes cagE, virB11 de H. pylori, em cÃncer gÃstrico. Entretanto, estudos em lesÃes gÃstricas sÃo restritos. Assim, o objetivo deste trabalho foi determinar o perfil genotÃpico das cepas de H. pylori quanto a presenÃa dos genes de virulÃncia cagA, cagE, virB11, vacA e flaA, circulantes no estado do Cearà em 201 casos de lesÃes gÃstricas de diferentes gravidades, coletadas de pacientes dispÃpticos atendidos em trÃs hospitais de Fortaleza-CE. A detecÃÃo de H. pylori foi feita atravÃs da amplificaÃÃo do gene ureC, os genes estudados por amplificaÃÃo de fragmentos especÃficos, usando a tÃcnica de PCR, foram observados em gel de agarose 1% e poliacrilamida a 6% e 8%. Neste estudo houve um predomÃnio do sexo feminino 59% (119/201), principalmente na faixa etÃria (15-44) 30% (61/201), alta taxa de infecÃÃo por H. pylori 97,5% (196/201). A gastrite crÃnica ativa (GCA) foi a lesÃo gÃstrica mais frequente (55,7%;112/201), associada a pacientes na faixa etÃria de 15-44 (36,6% : 41/112) opondo-se à metaplasia intestinal em que, a frequÃncia dos casos aumentou com a idade sendo maior em pacientes mais velhos 46% (16/35), o que concorda com a literatura. O Ãnico caso de displasia ocorreu numa paciente > 65 anos. As lesÃes gÃstricas foram predominantemente localizadas no antro. O gene cagA foi mais frequente na gastrite crÃnica ativa (GCA), estando tambÃm em alta frequÃncia na gastrite atrÃfica (GA) e metaplasia intestinal (MI). Na GCA foi observado tambÃm alta frequÃncia dos genes cagE, virB11, vacAm1 e flaA, com diferenÃa estatÃstica, quando comparada com a gastrite crÃnica inativa (GCI) (cagA - p=0,007 cagE- p=0,000, virB11 - p=0,005, vacAm1- p=0,047 e flaA - p=0,001), sendo que os genes virB11 e flaA foram mais frequentes na GCA do que na Ãlcera. Os alelos s1 e m1 de vacA , bem como a combinaÃÃo s1m1 foram os mais frequentes nas lesÃes gÃstricas. Os casos H. pylori positivos foram agrupados de acordo com a presenÃa dos genes estudados, levando em consideraÃÃo a presenÃa do alelo s1 e os genes da ilha. Nessas anÃlises foi observada maior frequÃncia de cepas contendo os genes vacA s1 e [Ia (cagA+, cagE+ e virB11+ ) + Ib (cagA+ e virB11+; cagE+ e virB11+) ; 47,7% ] na gastrite crÃnica ativa em relaÃÃo a gastrite crÃnica inativa (GCI), esta com maior frequÃncia de cepas dos grupos [Ic (cagA+ ou cagE+ ou virB11+ ou cagA+ e cagE+) +Id (ureC+); 62%]. Adicionalmente, maior frequÃncia de cepas pertencentes aos grupos Ia e Ib foi observada na metaplasia intestinal incompleta, (59%), enquanto que na metaplasia intestinal completa uma maior frequencia de cepas pertencentes aos grupos Ic + Id, (78%) (p=0,027). Todos os casos de gastrite atrÃfica e Ãlcera eram do grupo I, e um Ãnico caso de displasia pertencia ao grupo Ia. Esses dados evidenciam uma associaÃÃo de cepas com genÃtipos mais virulentos em lesÃes com potencialidade de malignizaÃÃo.
A bactÃria Helicobacter pylori à um agente etiolÃgico bem estabelecido para o desenvolvimento de lesÃes gÃstricas como gastrite, Ãlcera pÃptica, metaplasia e doenÃas malignas, com alta incidÃncia de infecÃÃo todo mundo, porÃm cerca de 80% dos indivÃduos infectados permanecem assintomÃticos e apenas uma minoria desenvolve doenÃas a ela relacionadas. Estudos tem sido realizados na tentativa de identificar a relaÃÃo de genes determinantes da patogenicidade de H. pylori, entretanto, atà o momento apenas os genes cagA e o alelo de vacA s1m1 sÃo considerados marcadores de virulÃncia para o desenvolvimento de lesÃes gÃstricas mais graves. A bactÃria H. pylori possui uma alta variabilidade genÃtica sendo que um dos mecanismos propostos para o desenvolvimento de lesÃo seria atravÃs da inflamaÃÃo. DiferenÃas na intensidade de respostas inflamatÃrias poderiam ser decorrentes do perfil genotÃpico da cepa infectante. Recentes estudos apontam a importÃncia dos genes cagE, virB11 de H. pylori, em cÃncer gÃstrico. Entretanto, estudos em lesÃes gÃstricas sÃo restritos. Assim, o objetivo deste trabalho foi determinar o perfil genotÃpico das cepas de H. pylori quanto a presenÃa dos genes de virulÃncia cagA, cagE, virB11, vacA e flaA, circulantes no estado do Cearà em 201 casos de lesÃes gÃstricas de diferentes gravidades, coletadas de pacientes dispÃpticos atendidos em trÃs hospitais de Fortaleza-CE. A detecÃÃo de H. pylori foi feita atravÃs da amplificaÃÃo do gene ureC, e os genes estudados por amplificaÃÃo de fragmentos especÃficos, usando a tÃcnica de PCR, e foram observados em gel de agarose 1% e poliacrilamida a 6% e 8%. Nesse estudo houve um predomÃnio do sexo feminino 59% (119/201), principalmente na faixa etÃria (15-44) 30% (61/201), e alta taxa de infecÃÃo por H. pylori 97,5% (196/201). A gastrite crÃnica ativa (GCA), foi a lesÃo gÃstrica mais frequente (55,7%;112/201), associada a pacientes na faixa etÃria de 15-44 (36,6% : 41/112) opondo-se à metaplasia intestinal onde a frequÃncia dos casos aumentou com a idade sendo maior em pacientes mais velhos 46% (16/35), o que concorda com a literatura. O Ãnico caso de displasia ocorreu numa paciente > 65 anos. As lesÃes gÃstricas foram predominantemente localizadas no antro. O gene cagA foi mais frequente na gastrite crÃnica ativa (GCA), estando tambÃm em alta frequÃncia na gastrite atrÃfica (GA) e metaplasia intestinal (MI). Na GCA foi observado tambÃm alta freqÃÃncias dos genes cagE, virB11, vacAm1 e flaA com diferenÃa estatÃstica quando comparada com a gastrite crÃnica inativa (GCI) (cagA - p=0,007 cagE- p=0,000, virB11 - p=0,005, vacAm1- p=0,047 e flaA - p=0,001), sendo que os genes virB11 e flaA foram mais frequentes na GCA que na Ãlcera. Os alelos s1 e m1 de vacA , bem como a combinaÃÃo s1m1 foram os mais frequentes nas lesÃes gÃstricas. Os casos H. pylori positivos foram agrupados de acordo com a presenÃa dos genes estudados, levando em consideraÃÃo a presenÃa do alelo s1 e os genes da ilha. Nestas anÃlises foi observada maior frequÃncia de cepas contendo os genes vacA s1 e [Ia (cagA+, cagE+ e virB11+ ) + Ib (cagA+ e virB11+; cagE+ e virB11+) ; 47,7% ] na gastrite crÃnica ativa em relaÃÃo a gastrite crÃnica inativa (GCI), esta Ãltima com maior frequÃncia de cepas dos grupos [Ic (cagA+ ou cagE+ ou virB11+ ou cagA+ e cagE+) +Id (ureC+); 62%]. Adicionalmente, maior frequÃncia de cepas pertencentes aos grupos Ia e Ib foi observada na metaplasia intestinal incompleta, (59%), enquanto que na metaplasia intestinal completa uma maior frequencia de cepas pertencentes aos grupos Ic + Id, (78%) (p=0,027). Todos os casos de gastrite atrÃfica e Ãlcera eram do grupo I, e o Ãnico caso de displasia pertencia ao grupo Ia. Esses dados evidenciam uma associaÃÃo de cepas com genÃtipos mais virulentos em lesÃes com potencialidade de malignizaÃÃo.
The bacterium Helicobacter pylori is a well-established etiological factor in the development of gastric lesions such as gastritis, peptic ulcers, metaplasia and malignancy. The incidence of infection by this pathogen is high worldwide, but about 80% of infected individuals remain asymptomatic, and only a minority develops related diseases. Many studies have been conducted in an attempt to identify the involvement of genes in determining the pathogenicity of H. pylori, but so far, only the genes cagA and vacA allele s1m1 are considered virulence markers for the development of the more severe gastric lesions. H. pylori bacteria have a high genetic variability and one of the proposed mechanisms for lesion development is through inflammation. Differences in the intensity of inflammatory responses could be due to the genotypic profile of the infecting strain. Recent studies indicate the importance of the genes cagE and virB11, but mostly involving gastric cancer, while their role in gastric lesions is limited. The objective of this study was to determine the genetic subtypes of H. pylori strains and the presence of the virulence genes cagA, cagE, virB11 and flaA genes and vacA alleles, circulating in Ceara state, Brazil. Samples were collected from 201 cases of gastric lesions of varying severity, in dyspeptic patients treated at three hospitals in Fortaleza, Ceara State. The detection of H. pylori was performed using ureC gene amplification by PCR, and the detection of the genes studied was carried out by amplification of gene-specific fragments separated in 1% agarose gels. The sample was predominantly female (59%, 119/201) and mainly in the age group 15-44 years old (30%, 61/201), and had a high rate of H. pylori infection (97.5%, 196/201). Active chronic gastritis (ACG) was the most common gastric lesion (55.7%, 112/201), associated with patients aged 15-44 (36.6%, 41/112), unlike intestinal metaplasia, in which the frequency of cases increased with age, being higher in older patients (46%, 16/35), which agrees with the literature. A single case of dysplasia occurred in a patient > 65 years. The lesions were predominantly located in the gastric antrum and 30% of the cases had lesions located in the body and antrum simultaneously. The cagA gene was more frequent in ACG, showing a statistically significant correlation (r = 0.220, p = 0.007); it also showed a high frequency in atrophic gastritis and in intestinal metaplasia. In ACG, a high frequency of the genes cagE, virB11, flaA and vacAm1 was also statistically associated when compared to chronic inactive gastritis (ICG) (cagA - p = 0.007, cagE - p = 0.000, virB11 - p = 0.005, vacAm1 p = 0.047 and flaA - p = 0.001), and the genes virB11 and flaA were more frequent in ACG than in ulcer. The vacA alleles s1 and m1, and the combination s1m1, were the most frequent ones in gastric lesions. Considering the genotypes of H. pylori grouped by the presence of the genes studied, we observed a higher frequency of the most virulent strains in the ACG group (Ia + Ib, 47.7%) when compared to ICG, the latter showing a higher frequency of less virulent strains (Ic + Id, 62%). Additionally, a higher frequency of more virulent strains, belonging to groups Ia and Ib, was observed in incomplete intestinal metaplasia (59%), while in complete intestinal metaplasia an increased frequency of less virulent strains, belonging to the groups Ic + Id (78%) (p = 0.027), was found. All cases of atrophic gastritis and ulcer were in group I, and the single case of dysplasia belonged to group Ia (high virulence). These data indicate the important role of more virulent strains in potential malignant lesions.
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Nastase, Mannila Maria. "Fibrinogen and susceptibility to myocardial infarction : role of gene-gene and gene-environment interactions /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-672-7/.

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Rück, Andreas. "Myocardial gene therapy and gene expression in angina pectoris /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-648-4/.

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Books on the topic "GEWE"

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Gede. Beijing Shi: Zhongguo he ping chu ban she, 1996.

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Gene. Spain: Pocket Books, 2005.

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Karasu, Bilge. Gece. Istanbul: Metis Yayinlari, 1992.

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Masashi, Sada. Gege. Tōkyō: Gentōsha, 2002.

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Carolyn, Cook. Gene. Grantsville, MD: Hobby House Press, 1998.

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Gene. Gene. Milano: Editoriale Giorgio Mondadori, 2020.

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Carolyn, Cook. Gene. 3rd ed. Grantsville, Md: Hobby House Press, 2001.

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Karasu, Bilge. Gece. İstanbul: İletişim Yayınları, 1985.

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Long, Wu, ed. Geye. Taibei Shi: Yi shu tu shu gong si, 1999.

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Schaffer, David V., and Weichang Zhou, eds. Gene Therapy and Gene Delivery Systems. Berlin/Heidelberg: Springer-Verlag, 2005. http://dx.doi.org/10.1007/11542766.

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Book chapters on the topic "GEWE"

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Moore, Sarah R. "Gene-Gene Interactions." In Encyclopedia of Personality and Individual Differences, 1757–59. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_1472.

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Galik, Elizabeth, Shin Fukudo, Yukari Tanaka, Yori Gidron, Tavis S. Campbell, Jillian A. Johnson, Kristin A. Zernicke, et al. "Gene-Gene Interaction." In Encyclopedia of Behavioral Medicine, 841–42. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_690.

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Jiang, Rong. "Gene-Gene Interaction." In Encyclopedia of Behavioral Medicine, 925–26. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_690.

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Zheng, Gang, Yaning Yang, Xiaofeng Zhu, and Robert C. Elston. "Gene-Gene Interactions." In Analysis of Genetic Association Studies, 235–56. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-2245-7_8.

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Moore, Sarah R. "Gene-Gene Interactions." In Encyclopedia of Personality and Individual Differences, 1–3. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_1472-1.

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DeWan, Andrew T. "Gene-Gene and Gene-Environment Interactions." In Methods in Molecular Biology, 89–110. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7868-7_7.

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Ivanov, V. I. "“Good Gene” /“Bad Gene”." In Genes and Resistance to Disease, 159–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56947-0_14.

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Dardick, Christopher D., and James N. Culver. "Gene-for-Gene Interactions." In Molecular Biology of Plant Viruses, 211–24. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5063-1_10.

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Palmer, Rohan, and Martin Hahn. "Gene." In Encyclopedia of Clinical Neuropsychology, 1549–50. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1858.

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Palmer, Rohan, and Martin Hahn. "Gene." In Encyclopedia of Clinical Neuropsychology, 1135–36. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1858.

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Conference papers on the topic "GEWE"

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Chaujar, R., R. Kaur, M. Saxena, M. Gupta, and R. S. Gupta. "GEWE-RC MOSFET: High performance RF solution to CMOS technology." In 2008 Asia Pacific Microwave Conference. IEEE, 2008. http://dx.doi.org/10.1109/apmc.2008.4958185.

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Gupta, Neha, and Rishu Chaujar. "Implications of transport models on the analog performance of Gate Electrode Workfunction Engineered (GEWE) Silicon Nanowire MOSFET." In 2014 2nd International Conference on Devices, Circuits and Systems (ICDCS). IEEE, 2014. http://dx.doi.org/10.1109/icdcsyst.2014.6926154.

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Gupta, Neha, Ajay Kumar, and Rishu Chaujar. "Effect of dielectric engineering on analog and linearity performance of gate electrode workfunction engineered (GEWE) silicon nanowire MOSFET." In 2015 IEEE 15th International Conference on Nanotechnology (IEEE-NANO). IEEE, 2015. http://dx.doi.org/10.1109/nano.2015.7388768.

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Chaujar, R., R. Kaur, M. Saxena, M. Gupta, and R. S. Gupta. "GEWE-RC MOSFET: A solution to CMOS technology for RFIC design based on the concept of intercept point." In 2008 International Conference on Recent Advances in Microwave Theory and Applications (MICROWAVE). IEEE, 2008. http://dx.doi.org/10.1109/amta.2008.4762978.

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"6th Belgian Brain Congress: The gene-environment tango in the healthy and diseased brain." In 6th Belgian Brain Congress: The gene-environment tango in the healthy and diseased brain. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-978-5.

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Li, Ling. "Mitochondrial fostering: the mitochondrial geme may play a role in plant orphan gene evolution." In ASPB PLANT BIOLOGY 2020. USA: ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1332405.

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Kojima, T., M. Tanimoto, T. Kamiya, Y. Obata, K. Kurachi, and H. Saito. "ANALYSIS OF FACTOR IX GENE IN NORMAL SUBJECTS AND HEMOPHILIA B PATIENTS IN JAPAN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644077.

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We have examined DNA samples from 25 hemophilia B patients (21 B- patients, 2 BR patients and 2 B+ patients) and 51 normal subjects with molecular probes (pHFIX and 2 genomic fragments). By structural gene analysis, 4 out of 7 patients who developed anti-factor IX antibodies were detected to have gross factor IX gene deletion. Although these four patients showed normal pattern of HPRT gene detected by pCDHPRT, the gene deletions were found to expand more than 34kb including with entire factor IX exons. Quantitative Southern blot analysis of factor IX gene of the patient's family members indicated that the gene deletion was inherited in one family, establishing the carrier status of 2 aunts, 2 cousins and one sister. The 'de novo' mutation of factor IX gene was also established in 2 families. Three patients with anti-factor IX antibodies and 17 patients without antibody to factor IX had normal pattern of factor IX gene by several restriction enzyme digestions. Analysis of factor IX gene of three patients with anti-factor IX antibodies and two B+ patients are now underway to detect the unique gene defects which may be responsible for the disease Phenotypes. Common RFLPs in factor IX gene were studied in normal Japanese subjects. More than 80 X chromosomes were analysed with BamHI, Ddel, MspI, TaqI or XmnI digestion, followed by hybridization with pHFIX. RFLPs produced by these enzymes were found to be uncommon or possibly absent in normal Japanese subjects. These results imply that racial differences in the frequency of gene polymorphisms should be seriously considered before initiating the gene counseling by the genetic probes.
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Tsakirpaloglou, Nikolaos. "Optimizing a high-throughput gene editing pipeline at the Texas A&M Crop Geme Editing Lab." In ASPB PLANT BIOLOGY 2020. USA: ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1053060.

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Yasin, Jeshima. "Geme wide stress responsive gene network and miR target identification from an orphan legume Horsegram (Macrotyloma uniflorum)." In ASPB PLANT BIOLOGY 2020. USA: ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1332446.

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Polstein, Lauren R., and Charles A. Gersbach. "Photoregulated Gene Expression in Human Cells With Light-Inducible Engineered Transcription Factors." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80573.

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Systems for controlling gene expression in mammalian cells have a wide range of applications in medicine, biotechnology and basic science. An ideal gene regulatory system would allow for precise and specific control over the magnitude and kinetics of gene expression in space and time, while also exerting minimal influence on other genes and cellular components. Several gene regulatory systems have been developed in which orthogonal transcription machinery from prokaryotes or insects has been imported into mammalian cells and used to control the expression of a specific gene. Despite the transformative impact of these systems in biomedical and biological research, several limitations of these technologies restrict the scope of possible applications. For example, gene expression in these systems is controlled by a freely diffusible small molecule, such as an antibiotic or steroid. Consequently, it is not possible to achieve spatial control over gene expression within cell culture, tissues, or whole organisms. This is in contrast to natural mechanisms of biological regulation in which spatial control is critical, such as developmental patterning and tissue morphogenesis. Second, dynamic gene regulation requires the removal of these small molecules, which may be slow, laborious, and/or impractical for a particular application. To overcome these limitations, we have engineered an optogenetic system in which the magnitude of gene expression in human cells can be finely tuned by photoregulated synthetic transcription factors.
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Reports on the topic "GEWE"

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Marshall, Dana R., Olufemi J. Adegoke, and Wei Zheng. Gene-Gene and Gene-Environment Interactions in the Etiology of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2007. http://dx.doi.org/10.21236/ada472397.

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Wolstenhome, D. R. The plant mitochondrial mat-r gene/nad1 gene complex. Office of Scientific and Technical Information (OSTI), December 1996. http://dx.doi.org/10.2172/763987.

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Ding, Yi. Adenovirus-Mediated p202 Gene Transfer in Breast Cancer Gene Therapy. Fort Belvoir, VA: Defense Technical Information Center, May 2005. http://dx.doi.org/10.21236/ada442740.

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Wolstenholme, D. R. The plant mitochondrial mat-r gene/nad1 gene complex. Progress report. Office of Scientific and Technical Information (OSTI), June 1994. http://dx.doi.org/10.2172/10159855.

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Sun, Jielin, Jianfeng Xu, and Siqun L. Zheng. Systematic Search for Gene-Gene Interaction Effect on Prostate Cancer Risk. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada564269.

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Sun, Jielin, Jianfeng Xu, and Siqun L. Zheng. Systematic Search for Gene-Gene Interaction Effect on Prostate Cancer Risk. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada593732.

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Einstein, J. R., R. J. Mural, X. Guan, and E. C. Uberbacher. Computer-based construction of gene models using the GRAIL Gene Assembly Program. Office of Scientific and Technical Information (OSTI), September 1992. http://dx.doi.org/10.2172/7160076.

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Yang, Ning-Sun. A Novel Gene Gun-Mediated IL-12 Gene Therapy for Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, October 2000. http://dx.doi.org/10.21236/ada394902.

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Kuchka, M. R. Post transcriptional regulation of chloroplast gene expression by nuclear encoded gene products. Office of Scientific and Technical Information (OSTI), January 1992. http://dx.doi.org/10.2172/7309627.

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Kuchka, M. R. Post transcriptional regulation of chloroplast gene expression by nuclear encoded gene products. Office of Scientific and Technical Information (OSTI), January 1992. http://dx.doi.org/10.2172/5268747.

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