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Journal articles on the topic 'Genetic regulation'

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Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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1

Humphries, S. E. "Genetic Regulation of Fibrinogen." European Heart Journal 16, suppl A (March 2, 1995): 16–20. http://dx.doi.org/10.1093/eurheartj/16.suppl_a.16.

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2

Depuydt, Christophe E., Adel Zalata, Christian R. de Potter, John van Emmelo, and Frank H. Comhaire. "Genetic regulation of gametogensis." Molecular Human Reproduction 2, no. 1 (1996): 2–8. http://dx.doi.org/10.1093/molehr/2.1.2.

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3

Breslow, Jan L. "Genetic regulation of apolipoproteins." American Heart Journal 113, no. 2 (February 1987): 422–27. http://dx.doi.org/10.1016/0002-8703(87)90608-9.

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4

Osiewacz, Heinz D. "Genetic regulation of aging." Journal of Molecular Medicine 75, no. 10 (October 13, 1997): 715–27. http://dx.doi.org/10.1007/s001090050158.

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5

Evans, Barbara J. "Economic Regulation of Next-Generation Sequencing." Journal of Law, Medicine & Ethics 42, S1 (2014): 51–66. http://dx.doi.org/10.1111/jlme.12162.

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The genetic testing industry is in a period of potentially major structural change driven by several factors. These include weaker patent protections after Association for Molecular Pathology v. Myriad Genetics (the “Myriad decision”) and Mayo Collaborative Services v. Prometheus Laboratories, Inc.; a continuing shift from single-gene tests to genome-scale sequencing; and a set of February 2014 amendments to the Clinical Laboratory Improvement Amendments of 1988 (CLIA) regulations and the Health Insurance Portability and Accountability Act (HIPAA) Privacy Rule. This article explores the nature of these changes and why they strain existing regulatory frameworks for protecting patients, research subjects, and other consumers who receive genetic testing.Oversight of genetic testing has, at least to date, had two major thrusts: (1) privacy and ethical protections and (2) traditional consumer health and safety regulations. Examples of the first are the Genetic Information Nondiscrimination Act and the HIPAA Privacy Rule, which after 2013 amendments expressly protects genetic privacy as well as other medical privacy.
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6

Jiang, Guanglong, Jill L. Reiter, Chuanpeng Dong, Yue Wang, Fang Fang, Zhaoyang Jiang, and Yunlong Liu. "Genetic Regulation of Human isomiR Biogenesis." Cancers 15, no. 17 (September 4, 2023): 4411. http://dx.doi.org/10.3390/cancers15174411.

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MicroRNAs play a critical role in regulating gene expression post-transcriptionally. Variations in mature microRNA sequences, known as isomiRs, arise from imprecise cleavage and nucleotide substitution or addition. These isomiRs can target different mRNAs or compete with their canonical counterparts, thereby expanding the scope of miRNA post-transcriptional regulation. Our study investigated the relationship between cis-acting single-nucleotide polymorphisms (SNPs) in precursor miRNA regions and isomiR composition, represented by the ratio of a specific 5′-isomiR subtype to all isomiRs identified for a particular mature miRNA. Significant associations between 95 SNP–isomiR pairs were identified. Of note, rs6505162 was significantly associated with both the 5′-extension of hsa-miR-423-3p and the 5′-trimming of hsa-miR-423-5p. Comparison of breast cancer and normal samples revealed that the expression of both isomiRs was significantly higher in tumors than in normal tissues. This study sheds light on the genetic regulation of isomiR maturation and advances our understanding of post-transcriptional regulation by microRNAs.
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7

Motta-Murguia, Lourdes, and Garbiñe Saruwatari-Zavala. "Mexican Regulation of Biobanks." Journal of Law, Medicine & Ethics 44, no. 1 (2016): 58–67. http://dx.doi.org/10.1177/1073110516644199.

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Biobank-based research in Mexico is mostly governed by research and data protection laws. There is no direct mention of biobanks in either statutory or regulatory law besides a requirement that the Federal Ministry of Health and a Mexican institution devoted to scientific research approve the transfer of biological materials outside of Mexico for population genetics research purposes. Such requirements are the basis of Genomic Sovereignty in Mexico, but such requirements have not prevented international collaboration. In addition, Mexican law singles out genetic research in informed consent provisions, but it does not specify whether all biobank-based research is genetic research. In order to facilitate international collaboration on biobank-based research, Mexico should directly address biobanking in its laws, building on the research framework and data protection framework already in place.
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8

Meeks, J. J., and E. M. Schaeffer. "Genetic Regulation of Prostate Development." Journal of Andrology 32, no. 3 (October 7, 2010): 210–17. http://dx.doi.org/10.2164/jandrol.110.011577.

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9

Yue, Shanna, Philip Whalen, and Youn Hee Jee. "Genetic regulation of linear growth." Annals of Pediatric Endocrinology & Metabolism 24, no. 1 (March 31, 2019): 2–14. http://dx.doi.org/10.6065/apem.2019.24.1.2.

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10

Howard, Sasha R. "Genetic regulation in pubertal delay." Journal of Molecular Endocrinology 63, no. 3 (October 2019): R37—R49. http://dx.doi.org/10.1530/jme-19-0130.

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Delayed puberty represents the clinical presentation of a final common pathway for many different pathological mechanisms. In the majority of patients presenting with significantly delayed puberty, there is a clear family history of delayed or disturbed puberty, and pubertal timing is known to be a trait with strong heritability. Thus, genetic factors clearly play a key role in determining the timing of puberty, and mutations in certain genes are recognised as responsible for delayed or absent puberty in a minority of patients. Through the identification of causal genetic defects such as these we have been able to learn a great deal about the pathogenesis of disrupted puberty and its genetic regulation. Firstly, deficiency in key genes that govern the development of the gonadotropin-releasing hormone system during fetal development may result in a spectrum of conditions ranging from isolated delayed puberty to absent puberty with anosmia. Secondly, a balance of inhibitory and excitatory signals, acting upstream of GnRH secretion, are vital for the correct timing of puberty. These act to repress the hypothalamic–pituitary–gonadal axis during mid-childhood and allow it to reactivate at puberty, and alterations in this equilibrium can cause delayed (or precocious) puberty. Thirdly, disturbances of energy metabolism inputs to the kisspeptin–GnRH system may also lead to late onset of puberty associated with changes in body mass.
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11

Lettre, Guillaume. "Genetic regulation of adult stature." Current Opinion in Pediatrics 21, no. 4 (August 2009): 515–22. http://dx.doi.org/10.1097/mop.0b013e32832c6dce.

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12

Palca, Joseph. "Genetic manipulation: Living outside regulation." Nature 324, no. 6094 (November 1986): 202. http://dx.doi.org/10.1038/324202a0.

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13

TAKEUCHI, TAKUJI, and HIROAKI YAMAMOTO. "Genetic Regulation of Melanocyte Differentiation." Pigment Cell Research 1, s1 (July 1988): 32–37. http://dx.doi.org/10.1111/j.1600-0749.1988.tb00792.x.

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14

Lauc, Gordan, Igor Rudan, Harry Campbell, and Pauline M. Rudd. "Complex genetic regulation of proteinglycosylation." Mol. BioSyst. 6, no. 2 (2010): 329–35. http://dx.doi.org/10.1039/b910377e.

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15

ZENG, BaoSheng, YongPing HUANG, AnJiang TAN, ShuQing CHEN, and Jun XU. "Genetic Regulation of Insect Populations." SCIENTIA SINICA Vitae 43, no. 12 (December 1, 2013): 1098–104. http://dx.doi.org/10.1360/052013-315.

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16

Behringer, R. R., J. J. Rasweiler, C. H. Chen, and C. J. Cretekos. "Genetic Regulation of Mammalian Diversity." Cold Spring Harbor Symposia on Quantitative Biology 74 (January 1, 2009): 297–302. http://dx.doi.org/10.1101/sqb.2009.74.035.

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17

Wang, Bing, Steven M. Smith, and Jiayang Li. "Genetic Regulation of Shoot Architecture." Annual Review of Plant Biology 69, no. 1 (April 29, 2018): 437–68. http://dx.doi.org/10.1146/annurev-arplant-042817-040422.

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18

Wang, Vincent Y., and Huda Y. Zoghbi. "Genetic regulation of cerebellar development." Nature Reviews Neuroscience 2, no. 7 (July 2001): 484–91. http://dx.doi.org/10.1038/35081558.

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19

Gillam, Mary P., and Peter Kopp. "Genetic regulation of thyroid development." Current Opinion in Pediatrics 13, no. 4 (August 2001): 358–63. http://dx.doi.org/10.1097/00008480-200108000-00013.

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20

Jones, L. C. "Genetic regulation of endothelial function." Heart 91, no. 10 (October 1, 2005): 1275–77. http://dx.doi.org/10.1136/hrt.2005.061325.

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21

Sims, Thomas L. "Genetic regulation of self‐incompatibility." Critical Reviews in Plant Sciences 12, no. 1-2 (January 1993): 129–67. http://dx.doi.org/10.1080/07352689309382359.

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22

Smith, Michael W. "Genetic regulation of enterocyte differentiation." Proceedings of the Nutrition Society 52, no. 2 (August 1993): 293–300. http://dx.doi.org/10.1079/pns19930065.

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23

Hsu, Hui-Chen, Lina Li, Huang-Ge Zhang, and John D. Mountz. "Genetic regulation of thymic involution." Mechanisms of Ageing and Development 126, no. 1 (January 2005): 87–97. http://dx.doi.org/10.1016/j.mad.2004.09.016.

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24

Sims, T. L. "Genetic Regulation of Self-Incompatibility." Critical Reviews in Plant Sciences 12, no. 1 (1993): 129. http://dx.doi.org/10.1080/713608043.

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25

Wyllie, Andrew H. "The genetic regulation of apoptosis." Current Opinion in Genetics & Development 5, no. 1 (February 1995): 97–104. http://dx.doi.org/10.1016/s0959-437x(95)90060-8.

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26

DiRita, V. J., and J. J. Mekalanos. "Genetic Regulation of Bacterial Virulence." Annual Review of Genetics 23, no. 1 (December 1989): 455–82. http://dx.doi.org/10.1146/annurev.ge.23.120189.002323.

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27

Waldron, Denise. "Bimodal regulation of genetic competence." Nature Reviews Genetics 16, no. 8 (July 17, 2015): 439. http://dx.doi.org/10.1038/nrg3987.

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28

Kitao, Yuko, Yasuaki Sadanaga, and Takeru Ishikawa. "Genetic Regulation of Allergic Rhinitis." Pediatrics International 29, no. 5 (October 1987): 654–57. http://dx.doi.org/10.1111/j.1442-200x.1987.tb00354.x.

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29

Aleksunes, Lauren M. "Genetic regulation of drug transporters." Drug Metabolism and Pharmacokinetics 33, no. 1 (January 2018): S10. http://dx.doi.org/10.1016/j.dmpk.2017.11.048.

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30

Vijayraghavan, Usha. "Genetic regulation of flower development." Journal of Biosciences 21, no. 3 (May 1996): 379–95. http://dx.doi.org/10.1007/bf02703096.

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31

Skodbo, Sara. "Enrolling genetic technology in regulation." Focaal 2005, no. 46 (December 1, 2005): 91–106. http://dx.doi.org/10.3167/092012906780786825.

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This article addresses the need to overcome theoretical weaknesses of both technologically and socially deterministic accounts of technological development. Technology does not simply 'impact' on local contexts, but nor does it act as a tabula rasa, subject to the free attribution of meaning by local social actors. Expanding on theoretical developments in the anthropology of art (Gell 1998) and gender and technology (Strathern 1988, 1999, 2001), the essay seeks to explore genetic technology as a social agent and as a technological 'index'. Examining a case of genetic technology regulation and innovation in Norway, the article argues that technology is best understood as an agent that is engaged with on an affective basis by those who interact with it.
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32

Minton, Kirsty. "Genetic regulation of peripheral tolerance." Nature Reviews Immunology 12, no. 3 (February 24, 2012): 151. http://dx.doi.org/10.1038/nri3182.

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33

Knobler, R. L., J. Marini, M. Perreault, and F. D. Lublin. "GENETIC REGULATION OF EAE SUSCEPTIBILITY." Journal of Neuropathology and Experimental Neurology 49, no. 3 (May 1990): 288. http://dx.doi.org/10.1097/00005072-199005000-00087.

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34

Pekhov, A. P., V. P. Shchipkov, and K. S. Krivskaya. "Genetic regulation of plasmid transfer." Bulletin of Experimental Biology and Medicine 120, no. 1 (July 1995): 651–57. http://dx.doi.org/10.1007/bf02444651.

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35

Gattineni, Jyothsna, and Michel Baum. "Genetic disorders of phosphate regulation." Pediatric Nephrology 27, no. 9 (February 14, 2012): 1477–87. http://dx.doi.org/10.1007/s00467-012-2103-2.

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36

Evans, James P., and Michael S. Watson. "Genetic Testing and FDA Regulation." JAMA 313, no. 7 (February 17, 2015): 669. http://dx.doi.org/10.1001/jama.2014.18145.

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37

Tunggal, Patrick, Neil Smyth, Mats Paulsson, and Mark-Christoph Ott. "Laminins: Structure and genetic regulation." Microscopy Research and Technique 51, no. 3 (2000): 214–27. http://dx.doi.org/10.1002/1097-0029(20001101)51:3<214::aid-jemt2>3.0.co;2-j.

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38

Kallio, Pekka J., Jorma J. Palvimo, and Olli A. Jänne. "Genetic regulation of androgen action." Prostate 29, S6 (1996): 45–51. http://dx.doi.org/10.1002/(sici)1097-0045(1996)6+<45::aid-pros9>3.0.co;2-j.

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39

Wang, Wenjie, Xinle Liang, Yudong Li, Pinmei Wang, and Nancy P. Keller. "Genetic Regulation of Mycotoxin Biosynthesis." Journal of Fungi 9, no. 1 (December 22, 2022): 21. http://dx.doi.org/10.3390/jof9010021.

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Mycotoxin contamination in food poses health hazards to humans. Current methods of controlling mycotoxins still have limitations and more effective approaches are needed. During the past decades of years, variable environmental factors have been tested for their influence on mycotoxin production leading to elucidation of a complex regulatory network involved in mycotoxin biosynthesis. These regulators are putative targets for screening molecules that could inhibit mycotoxin synthesis. Here, we summarize the regulatory mechanisms of hierarchical regulators, including pathway-specific regulators, global regulators and epigenetic regulators, on the production of the most critical mycotoxins (aflatoxins, patulin, citrinin, trichothecenes and fumonisins). Future studies on regulation of mycotoxins will provide valuable knowledge for exploring novel methods to inhibit mycotoxin biosynthesis in a more efficient way.
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40

Arych, Mykhailo, Khrystyna Shchubelka, Walter Wolfsberger, and Taras Oleksyk. "DOES UKRAINE NEED A SPECIFIC REGULATION RELATED TO THE APPLYING OF GENETIC INFORMATION FOR RISK ASSESSMENT IN INSURANCE?" Fìnansi Ukraïni 2024, no. 2 (May 17, 2024): 85–100. http://dx.doi.org/10.33763/finukr2024.02.085.

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Introduction. The article examines the specifics and role of genetic information for insurance risk assessment in the life insurance market in Ukraine. Problem Statement. The insurance market developments of different countries are increasingly characterized by the adoption of specific regulations regarding the features and conditions of use of genetic information. Therefore, the issue of regulating the rights and obligations of all participants in insurance relations regarding the use of such information of future owners of insurance policies for underwriting in insurance requires a comprehensive solution taking into account the interests of all parties. The purpose of the research is to evaluate the necessity of application of specific regulations on the insurance market, specifically in the field of using genetic data for insurance purposes. Methods. The sources of materials were scientific publications, analytical studies, as well as legislation in the field of regulation of the use of genetic information for the assessment of insurance risks. The research paper used the following empirical methods, such as analysis, synthesis, grouping, description, comparison, theoretical generalization. Results. The results show that currently some risks exist in Ukraine: firstly, the genetic discrimination, since most life insurance companies are interested in the genetic information of policyholders, and can request it from any third parties: therefore, there is a possibility of using it to assess insurance risk; secondly, an information asymmetry, which is a consequence of greater awareness of insurance companies about the insurance risks than that of the policyholders. After all, policyholders may not inform the insurance company about all the genetic data (for example, the results of genetic studies) that describe their genetic predisposition to future changes in health. Conclusions. This study substantiated factors which confirm the relevance of introducing legislative regulation regarding the use of genetic information (including the results of genetic analyzes) for underwriting in insurance.
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41

Bowen, Deborah J., Kathryn M. Battuello, and Monique Raats. "Marketing Genetic Tests: Empowerment or Snake Oil?" Health Education & Behavior 32, no. 5 (October 2005): 676–85. http://dx.doi.org/10.1177/1090198105278825.

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Genetic tests are currently being offered to the general public with little oversight and regulation as to which tests are allowed to be sold clinically and little control over the marketing and promotion of sales and use. This article provides discussion and data to indicate that the general public holds high opinions of genetic testing and that current media outlets for public education on genetic testing are not adequate to increase accurate knowledge of genetics. The authors argue that more regulation is needed to control and correct this problem in the United States.
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42

Estermann, Martin Andres, Andrew Thomas Major, and Craig Allen Smith. "Genetic Regulation of Avian Testis Development." Genes 12, no. 9 (September 21, 2021): 1459. http://dx.doi.org/10.3390/genes12091459.

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As in other vertebrates, avian testes are the site of spermatogenesis and androgen production. The paired testes of birds differentiate during embryogenesis, first marked by the development of pre-Sertoli cells in the gonadal primordium and their condensation into seminiferous cords. Germ cells become enclosed in these cords and enter mitotic arrest, while steroidogenic Leydig cells subsequently differentiate around the cords. This review describes our current understanding of avian testis development at the cell biology and genetic levels. Most of this knowledge has come from studies on the chicken embryo, though other species are increasingly being examined. In chicken, testis development is governed by the Z-chromosome-linked DMRT1 gene, which directly or indirectly activates the male factors, HEMGN, SOX9 and AMH. Recent single cell RNA-seq has defined cell lineage specification during chicken testis development, while comparative studies point to deep conservation of avian testis formation. Lastly, we identify areas of future research on the genetics of avian testis development.
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43

McGrath, Isabelle M., Sally Mortlock, and Grant W. Montgomery. "Genetic Regulation of Physiological Reproductive Lifespan and Female Fertility." International Journal of Molecular Sciences 22, no. 5 (March 4, 2021): 2556. http://dx.doi.org/10.3390/ijms22052556.

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There is substantial genetic variation for common traits associated with reproductive lifespan and for common diseases influencing female fertility. Progress in high-throughput sequencing and genome-wide association studies (GWAS) have transformed our understanding of common genetic risk factors for complex traits and diseases influencing reproductive lifespan and fertility. The data emerging from GWAS demonstrate the utility of genetics to explain epidemiological observations, revealing shared biological pathways linking puberty timing, fertility, reproductive ageing and health outcomes. The observations also identify unique genetic risk factors specific to different reproductive diseases impacting on female fertility. Sequencing in patients with primary ovarian insufficiency (POI) have identified mutations in a large number of genes while GWAS have revealed shared genetic risk factors for POI and ovarian ageing. Studies on age at menopause implicate DNA damage/repair genes with implications for follicle health and ageing. In addition to the discovery of individual genes and pathways, the increasingly powerful studies on common genetic risk factors help interpret the underlying relationships and direction of causation in the regulation of reproductive lifespan, fertility and related traits.
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44

Walton, E. F., R. M. Wu, R. J. Schaffer, K. Thodey, B. J. Janssen, and A. C. Richardson. "GENETIC REGULATION OF BUDBREAK IN KIWIFRUIT." Acta Horticulturae, no. 753 (October 2007): 561–66. http://dx.doi.org/10.17660/actahortic.2007.753.74.

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45

Varkonyi-Gasic, E., R. Wu, S. Moss, and R. P. Hellens. "GENETIC REGULATION OF FLOWERING IN KIWIFRUIT." Acta Horticulturae, no. 913 (November 2011): 221–27. http://dx.doi.org/10.17660/actahortic.2011.913.28.

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46

Xu, Daichao, Chengyu Zou, and Junying Yuan. "Genetic Regulation of RIPK1 and Necroptosis." Annual Review of Genetics 55, no. 1 (November 23, 2021): 235–63. http://dx.doi.org/10.1146/annurev-genet-071719-022748.

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The receptor-interacting protein kinase 1 (RIPK1) is recognized as a master upstream regulator that controls cell survival and inflammatory signaling as well as multiple cell death pathways, including apoptosis and necroptosis. The activation of RIPK1 kinase is extensively modulated by ubiquitination and phosphorylation, which are mediated by multiple factors that also control the activation of the NF-κB pathway. We discuss current findings regarding the genetic modulation of RIPK1 that controls its activation and interaction with downstream mediators, such as caspase-8 and RIPK3, to promote apoptosis and necroptosis. We also address genetic autoinflammatory human conditions that involve abnormal activation of RIPK1. Leveraging these new genetic and mechanistic insights, we postulate how an improved understanding of RIPK1 biology may support the development of therapeutics that target RIPK1 for the treatment of human inflammatory and neurodegenerative diseases.
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47

Kobayashi, Kazuo, Noriko Yamagata, Takashi Katsura, Sachiko Sugihara, Ikuyo Sato, Keita Kasahara, Terumi Takahashi, and Takeshi Yoshida. "Genetic regulation of tuberculous granulomatous inflammation." Ensho 12, no. 2 (1992): 147–53. http://dx.doi.org/10.2492/jsir1981.12.147.

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48

Rizzoti, Karine. "Genetic regulation of murine pituitary development." Journal of Molecular Endocrinology 54, no. 2 (January 13, 2015): R55—R73. http://dx.doi.org/10.1530/jme-14-0237.

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Significant progress has been made recently in unravelling the embryonic events leading to pituitary morphogenesis, bothin vivoandin vitro. This includes dissection of the molecular mechanisms controlling patterning of the ventral diencephalon that regulate formation of the pituitary anlagen or Rathke's pouch. There is also a better characterisation of processes that underlie maintenance of pituitary progenitors, specification of endocrine lineages and the three-dimensional organisation of newly differentiated endocrine cells. Furthermore, a population of adult pituitary stem cells (SCs), originating from embryonic progenitors, have been described and shown to have not only regenerative potential, but also the capacity to induce tumour formation. Finally, the successful recapitulationin vitroof embryonic events leading to generation of endocrine cells from embryonic SCs, and their subsequent transplantation, represents exciting advances towards the use of regenerative medicine to treat endocrine deficits. In this review, an up-to-date description of pituitary morphogenesis will be provided and discussed with particular reference to pituitary SC studies.
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49

Zagozewski, J. L., Q. Zhang, and D. D. Eisenstat. "Genetic regulation of vertebrate eye development." Clinical Genetics 86, no. 5 (September 25, 2014): 453–60. http://dx.doi.org/10.1111/cge.12493.

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50

SASSA, SHIGERU, HIROYOSHI FUJITA, and OSAMU SUGITA. "Genetic Regulation of the Heme Pathway." Annals of the New York Academy of Sciences 514, no. 1 Mechanisms of (December 1987): 15–22. http://dx.doi.org/10.1111/j.1749-6632.1987.tb48756.x.

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