Books: 'Gene dependency'

1

Moore, David S. The Dependent Gene: The Fallacy of "Nature vs. Nurture". New York: A W. H. Freeman book, Times Books, Henry Holt and Co., 2002.

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2

1960-, Moore David Scott. The dependent gene: The fallacy of "nature vs. nurture". New York: Henry Holt, 2003.

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3

Azmi, Peter B. Bacterially-derived DNA elements from the gene GPT can block enhancer-dependent transcriptional activation of an adjacent gene in a position-dependent manner. Ottawa: National Library of Canada, 2002.

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4

Worthinhton, Jenny. Radiation-controlled gene expression: A novel approach to oxygenation-dependent radiotherapy. [s.l: The Author], 2000.

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5

D, Takezawa, and United States. National Aeronautics and Space Administration., eds. Calcium-dependent protein kinase genes in corn roots. [Washington, DC: National Aeronautics and Space Administration, 1996.

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6

D, Takezawa, and United States. National Aeronautics and Space Administration., eds. Calcium-dependent protein kinase genes in corn roots. [Washington, DC: National Aeronautics and Space Administration, 1996.

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7

D, Takezawa, and United States. National Aeronautics and Space Administration., eds. Calcium-dependent protein kinase genes in corn roots. [Washington, DC: National Aeronautics and Space Administration, 1996.

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8

Lightfoot, Maria Eugenia Vidal. Studies of the calcium dependence of prolactin gene expression in GH[inferior]3 cells. Birmingham: University of Birmingham, 1990.

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9

Sprowl, Jason A. Identification of PKCa-dependent genes associated with growth arrest in Saccharomyces cerevisiae. Sudbury, Ont: Laurentian University, 2004.

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10

D, Takezawa, Poovaiah B. W, and United States. National Aeronautics and Space Administration., eds. Chimeric plant calcium/calmodulin-dependent protein kinase gene with a neural visinin-like calcium-binding domain. [Washington, DC: National Aeronautics and Space Administration, 1995.

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11

Gross, Gerhard, and Thomas Häupl. Stem cell-dependent therapies: Mesenchymal stem cells in chronic inflammatory disorders. Berlin: De Gruyter, 2013.

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12

C, Clarke B., Partridge Linda, and Royal Society (Great Britain), eds. Frequency-dependent selection: Proceedings of Royal Society discussion meeting held on 24 and 25 June 1987. London: Royal Society, 1988.

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13

Royal Society (Great Britain). Discussion Meeting. Frequency-dependent selection: Proceedings of a Royal Society Discussion Meeting held on 24 and 25 June 1987. London: The Society, 1988.

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14

Thompson, Miles. Mutation screening of dopamine and serotonin candidate genes in Tourette's syndrome and alcohol-dependent patients. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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15

Grove, Jane Isobel. Escherichia coli genes essential for formate-dependent nitrite reduction, cytochrome C biosynthesis and periplasmic nitrate reduction. Birmingham: University of Birmingham, 1996.

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16

Tisdale, Sarah. RNA-Dependent Control of Histone Gene Expression by the Spinal Muscular Atrophy Protein SMN: Mechanisms and Role in Motor Neuron Disease. [New York, N.Y.?]: [publisher not identified], 2015.

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17

Oelbaum, Raymond Stuart. An analysis of four candidate genes for non-insulin-dependent diabetes using restriction fragment length polymorphism markers. Manchester: University of Manchester, 1994.

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18

Bessai, Ivana Soric. The effect of tubercidin on the induction of AP-1 and NF-[kappa]B dependent genes by interleukin-1. Ottawa: National Library of Canada, 2000.

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19

Adamović, Tatjana. Identifiction and characterization of cancer genes in hormone-dependent tumors: Molecular genetic analysis in rat models of endometrial and mammary cancer. [Göteborg]: Dept. of Cell and Molecular Biology-Genetics, Lundberg Insitutute, Göteborgs University, 2006.

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20

Peakman, Timothy Charles. The regulation of the promoters of two genes involved in the NaDh-dependent nitrite reduction reaction in anaerobic cultures of Escherichia Coli K12. Birmingham: University of Birmingham, 1988.

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21

B, Kastan M., and Imperial Cancer Research Fund (Great Britain), eds. Checkpoint controls and cancer. Plainview, NY: Cold Spring Harbor Laboratory Press, 1997.

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22

Stocker, Claire. Type 2 diabetes: Methods and protocols. New York: Humana Press, 2009.

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23

G, Parker Malcolm, ed. Growth regulation by nuclear hormone receptors. Plainview, N.Y: Cold Spring Harbor Laboratory Press, 1992.

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24

Berenson, Gerald S. Evolution of Cardio-Metabolic Risk from Birth to Middle Age: The Bogalusa Heart Study. Dordrecht: Springer Science+Business Media B.V., 2011.

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25

Moore, David S., and Richard A. Griggs. Psychology; a Concise Introduction + the Dependent Gene. Worth Publishers, 2005.

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26

Gaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.

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Abstract:

Nervous system alterations can occur at any stage of prenatal or postnatal development. Any of these derangements, whether environmental or genetic, will affect electrical transmission, causing electroencephalogram (EEG) alteration and possibly epilepsy. Genetic insults may be multisystemic (for example, neurocutaneous syndromes) or affect only the brain. Gene mutations account for inborn errors of metabolism, channelopathies, brain malformations, and impaired synaptogenesis. Inborn errors of metabolism cause seizures and EEG abnormalities through a variety of mechanisms, including disrupted energy metabolism (mitochondrial disorders, glucose transporter defect), neuronal toxicity (amino and organic acidopathies), impaired neuronal function (lysosomal and peroxisomal disorders), alteration of neurotransmitter systems (nonketotic hyperglycinemia), and vitamin and co-factor dependency (pyridoxine-dependent seizures). Environmental causes of perinatal brain injury often result in motor or intellectual impairment (cerebral palsy). Multiple proposed etiologies exist for autism, many focusing on synaptic development. This chapter reviews the EEG findings associated with this myriad of pathologies occurring in childhood.

27

Clarke, B. C., and Linda Partridge. Frequency-dependent Selection. The Royal Society, 1988.

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28

Moore, David S. The Dependent Gene: The Fallacy of "Nature vs. Nurture". Owl Books, 2003.

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29

Segawa, M. Age-Related Dopamine-Dependent Disorders: International Symposium on Age-Related Monoamine-Dependent Disorders and Their Modulation by Gene and Gend (Monographs in Clinical Neuroscience). S. Karger AG (Switzerland), 1995.

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30

Calcium-dependent protein kinase genes in corn roots. [Washington, DC: National Aeronautics and Space Administration, 1996.

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31

Stallings, Michael C., Ian R. Gizer, and Kelly C. Young-Wolff. Genetic Epidemiology and Molecular Genetics. Edited by Kenneth J. Sher. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199381678.013.002.

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The tools of genetic epidemiology—family, adoption, and twin studies—show convincingly that substance use behavior and substance use disorders are influenced by both genetic and familial and extrafamilial environmental factors. Environmental factors appear to play a more influential role in the early stages of substance use, whereas genetic factors become more important in the development of problem use and substance use disorder. Moreover, some genetic effects are likely conditional on conducive environments; research employing both behavior genetic approaches and measured genes point to important gene–environment interactions that promote substance use and dependence. Consequently, a full understanding of the addiction process requires investigating substance use behavior within its comorbid context. The identification of specific genetic mechanisms underlying these heritable influences is elusive. These findings have prompted the development of new strategies for testing the joint effect of multiple genetic variants in gene-based or gene pathway analyses.

32

Litvack, Michael. Gene expression profiling of a CpG-ODN-dependent antiviral system in RAW 264.7 cells. 2005.

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33

Litvack, Michael. Gene expression profiling of a CpG-ODN-dependent antiviral system in RAW 264.7 cells. 2005.

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34

Kuznetsova, Tatiana. Context-dependency Of Renin-angiotensin-aldosterone System Genes In Relation To Left Ventricular Structure. Leuven Univ Pr, 2004.

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35

Alves, Ines Teles, Jan Trapman, and Guido Jenster. Molecular biology of prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0059.

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Prostate cancer is a heterogeneous disease that arises through the acquisition of key malignant hallmarks. At the molecular level, prostate tumours are dependent upon the androgen receptor pathway, which affects cell function, growth, and behaviour through downstream androgen-regulated genes. Prostate cancer requires this activity and manipulates the AR pathway to maintain signalling. For example, mutation of the AR (to bind ligands other than androgens) or amplification/duplication of the AR allows signalling to continue in the absence of testosterone. Around 50% of prostate cancers have a gene fusion between the androgen-regulated component of the TMPRSS2 gene and a transcription factor (e.g. ETS family members ERG and ETV1). This results in aberrant androgen stimulated cell growth. Current research is using molecular knowledge to identify biomarkers, such as PCA3, and new therapies, such as enzalutamide or abiraterone acetate.

36

Roth, Jeffrey Stephen. Isolation and expression of a cDNA clone encoding the catalytic subunit of the rat cAMP-dependent protein kinase. 1987.

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37

(Editor), Antonio Giordano, and Gaetano Romano (Editor), eds. Cell Cycle Control and Dysregulation Protocols (Methods in Molecular Biology). Humana Press, 2004.

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38

Khachatryan, Armen. Targeting of calcitonin gene-related peptide expression to the pancreatic beta cells prevents insulin-dependent diabetes mellitus in non-obese diabetic mice. 1996.

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39

Økland, Jorunn. Death and the Maiden. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198722618.003.0002.

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In a comparative study of the manifesto genre, the chapter selects three manifestos that from the outset shares common traits, including glorification of death and violence, a preoccupation with gender, the ‘laying bare’ of future events, all from a marginal position in the present. The three are the biblical Book of Revelation, Solanas’s SCUM Manifesto, and Breivik’s 2083: A European Declaration of Independence. The modern manifestos demonstrate genre similarities and a continuity in themes, preoccupations compared to Revelation, but postulating direct dependence would mean going too far. Instead, the chapter demonstrates how the misogynist ideology of Revelation lives on in the clockwork of the genre it helped to create, long after any detailed knowledge of the text itself is forgotten.

40

Vaheri, Antti, James N. Mills, Christina F. Spiropoulou, and Brian Hjelle. Hantaviruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0035.

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Hantaviruses (genus Hantavirus, family Bunyaviridae) are rodent- and insectivore-borne zoonotic viruses. Several hantaviruses are human pathogens, some with 10-35% mortality, and cause two diseases: hemorrhagic fever with renal syndrome (HFRS) in Eurasia, and hantavirus cardiopulmonary syndrome (HCPS) in the Americas. Hantaviruses are enveloped and have a three-segmented, single-stranded, negative-sense RNA genome. The L gene encodes an RNA-dependent RNA polymerase, the M gene encodes two glycoproteins (Gn and Gc), and the S gene encodes a nucleocapsid protein. In addition, the S genes of some hantaviruses have an NSs open reading frame that can act as an interferon antagonist. Similarities between phylogenies have suggested ancient codivergence of the viruses and their hosts to many authors, but increasing evidence for frequent, recent host switching and local adaptation has led to questioning of this model. Infected rodents establish persistent infections with little or no effect on the host. Humans are infected from aerosols of rodent excreta, direct contact of broken skin or mucous membranes with infectious virus, or rodent bite. One hantavirus, Andes virus, is unique in that it is known to be transmitted from person-to-person. HFRS and HCPS, although primarily affecting kidneys and lungs, respectively, share a number of clinical features, such as capillary leakage, TNF-, and thrombocytopenia; notably, hemorrhages and alterations in renal function also occur in HCPS and cardiac and pulmonary involvement are not rare in HFRS. Of the four structural proteins, both in humoral and cellular immunity, the nucleocapsid protein appears to be the principal immunogen. Cytotoxic T-lymphocyte responses are seen in both HFRS and HCPS and may be important for both protective immunity and pathogenesis. Diagnosis is mainly based on detection of IgM antibodies although viral RNA (vRNA) may be readily, although not invariably, detected in blood, urine and saliva. For sero/genotyping neutralization tests/RNA sequencing are required. Formalin-inactivated vaccines have been widely used in China and Korea but not outside Asia. Hantaviruses are prime examples of emerging and re-emerging infections and, given the limited number of rodents and insectivores thus far studied, it is likely that many new hantaviruses will be detected in the near future.

41

Campbell, Joseph W. The Order and the Other. University Press of Mississippi, 2019. http://dx.doi.org/10.14325/mississippi/9781496824721.001.0001.

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The Order and the Other is a call to reexamine the relationship between dystopian literature and science fiction by thinking about the work that each genre does on and for the reader. The author believes that this is especially necessary in regards to dystopian literature intended for adolescents. Now that the cultural boom of YA Dystopian texts is over, this book attempts to understand that boom by placing dystopian works into the larger context of belonging to literary history of dystopian works. It attempts to help readers see how surveillance and power form the way that not only the characters within the films or books think about themselves, but also how it shapes the readers, as well. It also helps show that the surveillance culture and state that we see within such texts is not dependent on science fiction genre structures to exist. Finally, the book examines the most recent efforts to understand the genre and suggests ways inquiry into the genre might go forward.

42

Barañano, Kristin W. Angelman Syndrome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0055.

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Angelman syndrome (AS) is a severe neurodevelopmental disorder caused by maternal deficiency of the epigenetically imprinted gene UBE3A. It is characterized by severe developmental delay, an ataxic gait disorder, an apparent happy demeanor with frequent smiling or laughing, and severe expressive language impairments. Understanding the neurobiology of AS has focused on understanding how UBE3A is regulated by neuronal activity, as well as the targets of its ubiquitin E3 ligase activity. This has led to a model of the role of UBE3A in the regulation of experience-dependent sculpting of synaptic circuits. At this time, treatment is largely supportive, but efforts directed toward reversing the epigenetic silencing machinery may lead to improved synaptic function in AS patients.

43

Perillán, José G. Science Between Myth and History. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780198864967.001.0001.

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Scientists regularly employ historical narrative as a rhetorical tool in their communication of science, yet there’s been little reflection on its effects within scientific communities and beyond.Â Science Between Myth and History begins to unravel these threads of influence. The stories scientists tell are not just poorly researched scholarly histories, they are myth-histories, a chimeric genre that bridges distinct narrative modes. This study goes beyond polarizing questions about who owns the history of science and establishes a common ground from which to better understand the messy and lasting legacy of the stories scientists tell. It aims to stimulate vigorous conversation among science practitioners, scholars, and communicators. Scientific myth-histories undoubtedly deliver value, coherence, and inspiration to their communities. They are tools used to broker scientific consensus, resolve controversies, and navigate power dynamics. Yet beyond the explicit intent and rationale behind their use, these narratives tend to have great rhetorical power and social agency that bear unintended consequences. This book unpacks the concept of myth-history and explores four case studies in which scientist storytellers use their narratives to teach, build consensus, and inform the broader public. From geo-politically informed quantum interpretation debates to high-stakes gene-editing patent disputes, these case studies illustrate the implications of storytelling in science. Science Between Myth and HistoryÂ calls on scientists not to eschew writing about their history, but to take more account of the stories they tell and the image of science they project. In this time of eroding common ground, when many find themselves dependent on, yet distrustful of scientific research, this book interrogates the effects of mismatched, dissonant portraits of science.

44

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Vitamin B7 (biotin) in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0011.

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Biotin is a water-soluble B vitamin (vitamin B7) which acts as a coenzyme to carboxylases and has roles in gluconeogenesis, fatty acid synthesis, and amino acid catabolism. Reduced activity of biotin-dependent enzymes (acetyl-CoA carboxylase I and II, and propionyl-CoA carboxylase) alters lipid metabolism and may impair synthesis of polyunsaturated fatty acids and prostaglandins; in addition, biotin has effects on gene expression by binding covalently to histones. Deficiency can be caused by prolonged consumption of egg whites, which contain the biotin-binding protein avidin. Smoking accelerates the degradation of biotin, which can result in marginal biotin deficiency. The effects of deficiency include disruption of immune function and lipid metabolism, with some evidence of teratogenicity in animals. Dietary deficiency is unlikely, although high consumption of egg whites should be avoided in pregnancy.

45

Ng, Dominic S. Familial Apolipoprotein A-I Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0036.

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Apolipoprotein (apo) A-I is the key structural protein of high-density lipoprotein (HDL) and is necessary for sustaining the circulating level of HDL. It has also been studied extensively for its role in mediating many of the antiatherosclerotic and antithrombotic properties of HDL. More than 50 naturally occurring mutations and variants have been described, and they usually result in marked HDL deficiency in a gene-dose dependent manner. However, the propensity to develop accelerated coronary heart disease (CHD) is highly heterogeneous. Mutations resulting in inability to synthesize apo A-I tend to be associated with early CHD, while mutations resulting in structurally altered apo A-I are generally not associated. Furthermore, a number of apo A-I variants, for example apo A-I Iowa or apo A-I Helsinki, have been linked to amyloidosis, resulting in potentially serious morbid complications.

46

Freer, Scott. American Disaster Movies of the 1970s. Bloomsbury Publishing Inc, 2023. http://dx.doi.org/10.5040/9781501336867.

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The 1970s witnessed the ‘golden age’ of American disaster movies. This book defines the generic characteristics, such as ensemble acting and visual spectacle to place this dystopian genre within its immediate American context: the end of the civil rights and countercultural era, the Watergate crisis, and the defeat in Vietnam. Many of the themes dramatized by films such as Airport (1970) and The Towering Inferno (1974) anticipate present-day debates such as irresponsible and corrupt corporate capitalism, misplaced faith in science and technology, ‘eco-collapse’, political ‘terrorist’ threats, natural disasters, threatened and displaced populations, unwarranted suffering, plus other ‘monstrosities of modernity’. In American Disaster Movies of the 1970s, Scott Freer examines the 1970s’ cultural phenomenon of the disaster movie genre, introducing a historicist perspective by placing the film genre within the immediate context of the 1970s American high modernity whilst engaging with relevant philosophical, theological and eco-critical ideas on evil, determinism, and human nature. The book contextualizes the 1970s disaster cycle by drawing on the longer cultural history of modernist reactions to modern anxieties including the widespread dependence on technology and corporate power. Freer explores the aesthetic theories of modern tragedy (i.e. the hubris of human fate and the aesthetic pleasure of exaggerated horror/terror on screen) as a means of explaining the power of disaster movies as a form of ethical criticism.

47

Humphreys, S. C. Kinship in Ancient Athens. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198788249.001.0001.

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The book covers Athenian kinship from Drakon and Solon to Menander (with some references to later developments). It uses a wide range of sources: epigraphic, literary/forensic, and archaeological. It provides an ethnographic ‘thick description’ of Athenians’ interaction with their kin in all contexts: legal relations (adoption, guardianship, marriage, inheritance, disputes in and out of court); economic interaction (property, economic independence/dependence of sons in relation to fathers); training in specialist skills (doctors, actors, artists), loans, guarantees, etc.; rituals (naming, rites de passage, funerals and commemoration, dedications, cultic associations); war (military commands, organization of land and sea forces); and political contexts, both informal (hetaireiai) and formal (Assembly, Council). Volume II deals with corporate groups recruited by patrifiliation: tribes and trittyes (both pre-Kleisthenic and Kleisthenic), phratries, genê, and demes. The section on the demes stresses variety rather than common features, and provides up-to-date information on location and prosopography.

48

Allen, Lydia Ross. Dominant negative serum response factor transgene expression affects the regulation of activity-dependent early response genes coding for cell signaling proteins, transcription factors, structural proteins, and enzymes in primary neuronal cultures. 2004.

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49

Plastics in Automotive Engineering PIAE EUROPE. VDI Verlag, 2019. http://dx.doi.org/10.51202/9783181023433.

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Der VDI-Bericht ist ausschließlich als PDF erschienen! / Sie möchten gerne mehr erfahren? Inhalt Plastic components: future requirements Zukünftige Anforderungen an Kunststoffbauteile Volvo Cars recycled plastics strategy – Kick-starting with a recycled-plastics demo car 1 S. Tostar, Volvo Car Group, Gothenburg, Sweden Sustainable materials for the interior parts 5 Nachhaltige Materialien für das Interieur 17 C. Schütz, L. Lewerdomski, E. Körner, C. Winkelmann, Volkswagen AG, Wolfsburg Interior-Trends/Trends im Fahrzeuginnenraum Antimicrobial treatment of textiles and decorative materials for passenger transport and car-sharing concepts 29 Antimikrobielle Ausstattung von Textilien und Dekormaterialien für Personentransport und Carsharing Konzepte 43 M. Schneider, A. Cordella, car i.t.a. GmbH & Co. KG, Kirkel-Limbach Simulation/Simulation Elasto-viscoplastic temperature-dependent material model for a talc-filled PP/PE copolymer 57 Elasto-viskoplastisches temperaturabhängi...

50

Kirchman, David L. Community structure of microbes in natural environments. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789406.003.0004.

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Community structure refers to the taxonomic types of microbes and their relative abundance in an environment. This chapter focuses on bacteria with a few words about fungi; protists and viruses are discussed in Chapters 9 and 10. Traditional methods for identifying microbes rely on biochemical testing of phenotype observable in the laboratory. Even for cultivated microbes and larger organisms, the traditional, phenotype approach has been replaced by comparing sequences of specific genes, those for 16S rRNA (archaea and bacteria) or 18S rRNA (microbial eukaryotes). Cultivation-independent approaches based on 16S rRNA gene sequencing have revealed that natural microbial communities have a few abundant types and many rare ones. These organisms differ substantially from those that can be grown in the laboratory using cultivation-dependent approaches. The abundant types of microbes found in soils, freshwater lakes, and oceans all differ. Once thought to be confined to extreme habitats, Archaea are now known to occur everywhere, but are particularly abundant in the deep ocean, where they make up as much as 50% of the total microbial abundance. Dispersal of bacteria and other small microbes is thought to be easy, leading to the Bass Becking hypothesis that “everything is everywhere, but the environment selects.” Among several factors known to affect community structure, salinity and temperature are very important, as is pH especially in soils. In addition to bottom-up factors, both top-down factors, grazing and viral lysis, also shape community structure. According to the Kill the Winner hypothesis, viruses select for fast-growing types, allowing slower growing defensive specialists to survive. Cultivation-independent approaches indicate that fungi are more diverse than previously appreciated, but they are less diverse than bacteria, especially in aquatic habitats. The community structure of fungi is affected by many of the same factors shaping bacterial community structure, but the dispersal of fungi is more limited than that of bacteria. The chapter ends with a discussion about the relationship between community structure and biogeochemical processes. The value of community structure information varies with the process and the degree of metabolic redundancy among the community members for the process.

Books on the topic 'Gene dependency'

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