Academic literature on the topic 'GDNF'
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Journal articles on the topic "GDNF"
Doretto, Lucas B., Arno J. Butzge, Rafael T. Nakajima, Emanuel R. M. Martinez, Beatriz Marques de Souza, Maira da Silva Rodrigues, Ivana F. Rosa, et al. "Gdnf Acts as a Germ Cell-Derived Growth Factor and Regulates the Zebrafish Germ Stem Cell Niche in Autocrine- and Paracrine-Dependent Manners." Cells 11, no. 8 (April 11, 2022): 1295. http://dx.doi.org/10.3390/cells11081295.
Full textMoreau, Evelyne, José Vilar, Martine Lelièvre-Pégorier, Claudie Merlet-Bénichou, and Thierry Gilbert. "Regulation of c-ret expression by retinoic acid in rat metanephros: implication in nephron mass control." American Journal of Physiology-Renal Physiology 275, no. 6 (December 1, 1998): F938—F945. http://dx.doi.org/10.1152/ajprenal.1998.275.6.f938.
Full textMarsh, Debbie J., Zimu Zheng, Andrew Arnold, Scott D. Andrew, Diana Learoyd, Andrea Frilling, Paul Komminoth, et al. "Mutation Analysis of Glial Cell Line-Derived Neurotrophic Factor, a Ligand for an RET/Coreceptor Complex, in Multiple Endocrine Neoplasia Type 2 and Sporadic Neuroendocrine Tumors." Journal of Clinical Endocrinology & Metabolism 82, no. 9 (September 1, 1997): 3025–28. http://dx.doi.org/10.1210/jcem.82.9.4197.
Full textWalton, Kevin M. "GDNF." Molecular Neurobiology 19, no. 1 (February 1999): 43–59. http://dx.doi.org/10.1007/bf02741377.
Full textTao, Le, Wenting Ma, Liu Wu, Mingyi Xu, Yanqin Yang, Wei Zhang, Wenjun Sha, et al. "Glial cell line-derived neurotrophic factor (GDNF) mediates hepatic stellate cell activation via ALK5/Smad signalling." Gut 68, no. 12 (June 6, 2019): 2214–27. http://dx.doi.org/10.1136/gutjnl-2018-317872.
Full textShi, Haikun, Daniel Patschan, Gunnar P. H. Dietz, Mathias Bähr, Matthew Plotkin, and Michael S. Goligorsky. "Glial cell line-derived neurotrophic growth factor increases motility and survival of cultured mesenchymal stem cells and ameliorates acute kidney injury." American Journal of Physiology-Renal Physiology 294, no. 1 (January 2008): F229—F235. http://dx.doi.org/10.1152/ajprenal.00386.2007.
Full textMagill, Christina K., Amy M. Moore, Ying Yan, Alice Y. Tong, Matthew R. MacEwan, Andrew Yee, Ayato Hayashi, et al. "The differential effects of pathway- versus target-derived glial cell line–derived neurotrophic factor on peripheral nerve regeneration." Journal of Neurosurgery 113, no. 1 (July 2010): 102–9. http://dx.doi.org/10.3171/2009.10.jns091092.
Full textPopsueva, Anna, Dmitry Poteryaev, Elena Arighi, Xiaojuan Meng, Alexandre Angers-Loustau, David Kaplan, Mart Saarma, and Hannu Sariola. "GDNF promotes tubulogenesis of GFRα1-expressing MDCK cells by Src-mediated phosphorylation of Met receptor tyrosine kinase." Journal of Cell Biology 161, no. 1 (April 7, 2003): 119–29. http://dx.doi.org/10.1083/jcb.200212174.
Full textCintrón-Colón, Alberto F., Gabriel Almeida-Alves, Alicia M. Boynton, and John M. Spitsbergen. "GDNF synthesis, signaling, and retrograde transport in motor neurons." Cell and Tissue Research 382, no. 1 (September 8, 2020): 47–56. http://dx.doi.org/10.1007/s00441-020-03287-6.
Full textZaman, V., Z. Li, L. Middaugh, S. Ramamoorthy, B. Rohrer, M. E. Nelson, A. C. Tomac, B. J. Hoffer, G. A. Gerhardt, and A. Ch Granholm. "The Noradrenergic System of Aged GDNF Heterozygous Mice." Cell Transplantation 12, no. 3 (April 2003): 291–303. http://dx.doi.org/10.3727/000000003108746740.
Full textDissertations / Theses on the topic "GDNF"
Ivanchuk, Stacey M. "Expression of RET, GDNF and GDNFR-Ã in human development and disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq20655.pdf.
Full textDHALIWAL, PARVIN. "PARKINSON'S GDNF THERAPY AND OXIDATIVE STRESS." Thesis, The University of Arizona, 2008. http://hdl.handle.net/10150/190438.
Full textRoxo, Tiago Filipe Dias Santos. "Efeito anti-inflamatório do GDNF: qual a sua contribuição para a neuroprotecção dopaminérgica?" Master's thesis, Universidade da Beira Interior, 2013. http://hdl.handle.net/10400.6/1625.
Full textAs microglias são os macrófagos residentes do Sistema Nervoso Central e actuam como a principal forma de defesa imunitária. Podem assumir um estado denominado activado, tendo a sua capacidade fagocítica aumentada e produzindo espécies reactivas de oxigénio, com o propósito de proteger as células do Sistema Nervoso Central. No entanto, este estado activado tem sido também relacionado com um processo neurodegenerativo. Aumentos dos níveis de interleucinas e citocinas têm sido descritos em doenças neurodegenerativas, nomeadamente na doença de Parkinson, onde a perda de neurónios dopaminérgicos tem sido associada a uma excessiva activação microglial. Factores solúveis libertados pelos astrócitos mostraram ser capazes de modular a reactividade microglial. Destes factores, o factor derivado de uma linha de células da glia (GDNF) destacou-se pela sua capacidade em proteger os neurónios dopaminérgicos, tanto in vitro como in vivo. Alguns estudos têm também demonstrado uma acção anti-inflamatória do GDNF, mediada pelo receptor GFR1, sugerindo que possa existir uma relação entre estes dois efeitos. No entanto, até ao momento, não foi ainda demonstrada uma relação de causaefeito entre eles. Assim, este trabalho tem como objectivo elucidar a importância do controlo da reactividade microglial pelo GDNF na sobrevivência dos neurónios dopaminérgicos. A estratégia principal será impedir a acção do GDNF especificamente na microglia, através do silenciamento do receptor GFR1, e avaliar o efeito deste silenciamento na acção neuroprotectora do GDNF após aplicação de um estímulo inflamatório. A expressão de GFR1 em culturas primárias de microglia do mesencéfalo ventral e numa linha celular de microglia N9 foi confirmada por imunocitoquímica e Western Blot. O silenciamento do receptor GFR1 na linha celular de microglia N9 foi alcançado com sucesso e resultados preliminares sugerem que o silenciamento deste receptor em culturas primárias de microglia é também possível. A exposição de co-culturas de microglia N9 e culturas mistas de neurónios e glia do mesencéfalo ventral a diferentes concentrações de LPS induziu a morte selectiva de neurónios dopaminérgicos. Paralelamente, foi possível observar um aumento da reactividade microglial. Experiências adicionais serão necessárias para atingir o objectivo principal deste trabalho. No entanto, estes resultados servirão de base para, em futuras experiências, elucidar a relevância do efeito anti-inflamatório do GDNF na neuroprotecção dopaminérgica.
Trupp, Miles. "Neurotrophic signalling by GDNF and its receptors /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980602trup.
Full textFink, Erin Nicole. "GM1 signaling through the GDNF receptor complex." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1198013799.
Full textChermenina, Maria. "GDNF and alpha-synuclein in nigrostriatal degeneration." Doctoral thesis, Umeå universitet, Histologi med cellbiologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-91811.
Full textOliveira, Julieta Conceição Mendes Borges. "GDNF e GPER: novas ferramentas no controlo da neuroinflamação?" Master's thesis, Universidade da Beira Interior, 2013. http://hdl.handle.net/10400.6/1628.
Full textMicroglial cells, the macrophages resident in the central nervous system, are responsible for the innate immune response. When moderately activated, these cells perform vital functions such as phagocytosing dead cells and removing cell debris and toxins. However, a persistent activation of these cells may result in deregulation of its activity. They can become reactive and contribute to neuronal death. Increasing evidences suggests that inflammation and oxidative stress mediated by reactive microglia play a key role in the progression of various neurodegenerative diseases such as Parkinson's disease. Both the glial cell line derived neurotrophic factor (GDNF) and estrogens are reported to play a role in this process and to control excessive activation of microglia. A previous study from our group that used a primary culture of ventral midbrain microglia showed that GDNF present in medium conditioned by astrocytes can inhibit microglial reactivity induced by Zymozan A. The first objective of the present study was to verify if the presence of neurons, injured or not, could influence this anti-inflammatory effect exerted by GDNF. Using the same culture we found that media conditioned by both astrocytes and neurons was no longer capable of inhibiting NO production on LPS-stimulated microglia. The different effects of the two media may be related to the fact that the media conditioned by cultures of neurons and astrocytes presented lower levels of GDNF as compared with media conditioned only by astrocytes. On the other hand, studies using primary cultures and microglia cell lines demonstrated the ability of estrogen to attenuate parameters of microglial activation such as phagocytic activity, production of reactive oxygen and nitrogen, as well as other factors inflammatory cascade. The ability of estrogens to regulate estrogen receptors α and β present in microglia was previously described. However, more recently a transmembrane estrogen receptor, the G-protein coupled estrogen receptor (GPER) was identified. The objective of the second part of the present work was to clarify the involvement of GPER in the control microglial reactivity mediated by estradiol. Using the N9 microglial cell line, an agonist and an antagonist of GPER receptor, we found that GPER activation promoted the migration of microglial cells and significantly reduced the various parameters of microglial reactivity evaluated. Taken together these results suggest that GPER can be an important therapeutic target for neurodegenerative and neuroinflammatory diseases, especially in males, for whom estrogen therapy is not feasible.
Fonseca, Ana Paula da Silva. "Contribuição do GDNF para a neuroprotecção exercida pelo estrogénio." Master's thesis, Universidade da Beira Interior, 2010. http://hdl.handle.net/10400.6/799.
Full textParkinson´s disease is the second most common neurodegenerative disorder after Alzheimer and is mainly characterized by a progressive and selective depletion of dopamine neurons in the Substantia Nigra. Numerous studies have reported a greater prevalence and incidence of PD in men than in women. Studies involving estrogen treatment of ovariectomised rodents attribute this largely to the neuroprotective effets of estrogen. However, a neuroprotective role for physiologic levels of circulating estrogen in females is less clear. Estrogens have also been shown to regulate the expression of neurotrophic factors, like glial cell line-derived neurotrophic factor (GDNF), which might mediate their neuroprotective effects. GDNF produces neuroprotective and regenerative effects in the nigrostriatal pathways, acting as a potent survival factor for dopaminergic neurons that degenerate in Parkinson’s disease. In order to clarify the role of endogenous levels of estrogens in protecting the nigrostriatal pathway, we used the 6-hydroxydopamine (6-OHDA) model of Parkinson’s disease and tested how the removal of ovaries in fertile females interferes with extent of the dopaminergic lesion induced by 6-OHDA. Female Wistar rats were ovariectomised and 3 weeks after the surgery the animals were stereotaxically injected in the striatum with 6-OHDA. The extent of the lesion was assessed by counting the cells expressing the dopaminergic marker tyrosine hydroxylase by imunohistochemistry and also the expression levels of this protein by Western blot in both the Substantia Nigra and the striatum. The plasma levels of estradiol were also quantified. To determine if there was a relationship between estradiol levels, the expression of GDNF and the extent of the dopaminergic lesion, we also studied the expression of the neurotrophic factor GDNF. Our findings strongly suggest that endogenously produced estrogens and GDNF are associated with increased levels of striatal tyrosine hydroxylase, a marker of dopaminergic cell survival.
Wartiovaara, Kirmo. "GDNF and p75 neurotrophin receptor in development and disease." Helsinki : University of Helsinki, 1999. http://ethesis.helsinki.fi/julkaisut/laa/biola/vk/wartiovaara/.
Full textÅkerud, Peter. "GDNF family ligands and neural stem cells in Parkinson's disease /." Stockholm : [Karolinska Univ. Press], 2001. http://diss.kib.ki.se/2001/91-7349-042-3/.
Full textBooks on the topic "GDNF"
David, Dellafiora, and Saltram House, eds. GDN PL7. [Plympton?]: National Trust, 1991.
Find full textDecèze, Dominique. Haute tension à EDF-GDF. Paris: Gawsewitch, 2005.
Find full textUniversitas Indonesia. Pusat Penelitian Pranata Pembangunan., ed. Evaluasi pelaksanaan GDN di DKI Jakarta. [Jakarta]: Kerja sama Pusat Penelitian Pranata Pembangunan, UI dengan Direktorat Sosial Politik, DKI Jakarta, 1997.
Find full textPierre-Eric, Tixier, and Mauchamp Nelly, eds. EDF-GDF: Une entreprise publique en mutation. Paris: La Découverte, 2000.
Find full textWeber, Uwe. Der Grenzüberschreitende Datenfluss (GDF): Ein neues Phänomen der internationalen Kommunikation. München: R. Fischer, 1993.
Find full textGDF-Suez, Arcelor, EADS, Pechiney: Les dossiers noirs de la droite. Paris: Gawsewitch, 2007.
Find full textMcPhee, Jennifer. ID-1 and GDF-8 as negative regulators of skeletal muscle mass. Sudbury, Ont: Laurentian University, Behavioural Neuroscience Program, 1998.
Find full textMarcel, Goldberg, Leclerc Annette, and Bugel Isabelle, eds. Cohorte GAZEL, 20000 volontaires d'EDF-GDF pour la recherche médicale: Bilan 1989-1993. Paris: Editions INSERM, 1994.
Find full textGermany) Group Decision and Negotiation (Conference) (7th 2006 Karlsruhe. Group Decision and Negotiation (GDN) 2006: International conference, Karlsruhe, Germany, June 25-28, 2006 : proceedings. Edited by Seifert Stefan 1971 editor and Weinhardt Christof editor. Karlsruhe: Universitätsverlag Karlsruhe, 2006.
Find full textCondijts, Joan. GDF-Suez, le dossier secret de la fusion: Enquête dans les coulisses du capitalisme à la française. Paris: Michalon, 2008.
Find full textBook chapters on the topic "GDNF"
Peterziel, H., and J. Strelau. "GDNF and Related Proteins." In Handbook of Neurochemistry and Molecular Neurobiology, 69–91. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-30381-9_4.
Full textKiuchi, K., and H. Xiao. "GDNF-Induced Expression of Tyrosine Hydroxylase." In Catecholamine Research, 115–18. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4757-3538-3_24.
Full textRossi, Jari, and Matti S. Airaksinen. "GDNF Family Signalling in Exocrine Tissues: Distinct Roles for GDNF and Neurturin in Parasympathetic Neuron Development." In Advances in Experimental Medicine and Biology, 19–26. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0717-8_2.
Full textUnsicker, K., C. Suter-Crazzolara, and K. Krieglstein. "Neurotrophic Roles of GDNF and Related Factors." In Neurotrophic Factors, 189–224. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59920-0_8.
Full textRosenblad, Carl, Deniz Kirik, and Anders Björklund. "Effects of GDNF on Nigrostriatal Dopamine Neurons." In Advances in Behavioral Biology, 117–30. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0179-4_12.
Full textBankiewicz, Krystof, Waldy San Sebastian, Lluis Samaranch, and John Forsayeth. "GDNF and AADC Gene Therapy for Parkinson’s Disease." In Translational Neuroscience, 65–88. Boston, MA: Springer US, 2016. http://dx.doi.org/10.1007/978-1-4899-7654-3_4.
Full textBarua, Sonia, and Yashwant V. Pathak. "Unilateral Ex Vivo Gene Therapy by GDNF in Neurodegenerative Diseases." In Gene Delivery Systems, 155–61. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003186069-9.
Full textSariola, Hannu, and Tiina Immonen. "GDNF Maintains Mouse Spermatogonial Stem Cells In Vivo and In Vitro." In Methods in Molecular Biology™, 127–35. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-214-8_9.
Full textNitta, Atsumi, Rina Murai, Keiko Maruyama, and Shoei Furukawa. "FK506 Protects Dopaminergic Degeneration Through Induction of GDNF in Rodent Brains." In Mapping the Progress of Alzheimer’s and Parkinson’s Disease, 463–67. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-0-306-47593-1_79.
Full textCass, Wayne A., Cecilia M. Kearns, and Don M. Gash. "Protective and Regenerative Properties of GDNF in the Central Nervous System." In Neuroprotective Signal Transduction, 145–61. Totowa, NJ: Humana Press, 1998. http://dx.doi.org/10.1007/978-1-59259-475-7_8.
Full textConference papers on the topic "GDNF"
Torres Ortega, Pablo Vicente, Cristian Smerdou, Elisa Garbayo, and María J. Blanco Prieto. "Sustained GDNF delivery via PLGA nanoparticles." In The 1st International Electronic Conference on Pharmaceutics. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/iecp2020-08799.
Full textMarquardt, Laura M., and Shelly E. Sakiyama-Elbert. "Effect of GDNF on schwann cell differentiation and interaction with neurons in vitro." In 2014 40th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2014. http://dx.doi.org/10.1109/nebec.2014.6972867.
Full textFeng, R., L. Tao, W. Ma, L. Wu, E. Seki, C. Liu, and S. Dooley. "Glial cell line-derived neurotrophic factor (GDNF) mediates hepatic stellate cell activation via ALK5/Smad signaling." In Viszeralmedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1695260.
Full textBhoj, Vijay G., Selene Nunez-Cruz, Kenneth Zhou, Dimitrios Arhontoulis, Michael Feldman, Keith Mansfield, Haiyong Peng, Christoph Rader, Don L. Siegel, and Michael C. Milone. "Abstract 2295: GDNF family receptor alpha 4 (GFRa4)-targeted adoptive T-cell immunotherapy for medullary thyroid carcinoma." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-2295.
Full textFerrante, Catherine A., Nikki DeAngelis, and Raluca Verona. "Abstract 4048: GFRα1 is required for GDNF-induced viability, migration, and signaling through RET in breast cancer cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4048.
Full textGardaneh, Mossa, and Sahar Shojaei. "Abstract 969: The anti-apoptotic impact of neurotrophic factor GDNF on breast cancer cells pre-treated with trastuzumab." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-969.
Full textFan, Ching-Hsiang, Chien-Yu Ting, En-Ling Chang, Hao-Li Liu, Hong-Lin Chan, You-Yin Chen, and Chih-Kuang Yeh. "Ultrasound-triggered and targeted gene delivery by using cationic microbubbles to enhance GDNF gene transfection in a rat Parkinson's disease model." In 2015 IEEE International Ultrasonics Symposium (IUS). IEEE, 2015. http://dx.doi.org/10.1109/ultsym.2015.0204.
Full textMorandi, Andrea, Lesley-Ann Martin, Qiong Gao, Alan Mackay, David Robertson, Marketa Zvelebil, Mitch Dowsett, Ivan Plaza-Menacho, and Clare M. Isacke. "Abstract 4761: Glial cell derived neurotrophic factor (GDNF)-RET signaling as a target in aromatase inhibitor resistant ER-positive breast cancers." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4761.
Full textAsker, Hasan. "The Effects of Cinnamon Extract Supplementation on Immunolocalization of Glial Cell Line Derived Neurotrophic Factor (GDNF) in Testis of Diabetic Rats." In 15th International Congress of Histochemistry and Cytochemistry. Istanbul: LookUs Scientific, 2017. http://dx.doi.org/10.5505/2017ichc.pp-89.
Full textCavel, Oren, Richard J. Wong, Olga Shomron, Moran Amit, and Ziv Gil. "Abstract 1529: Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RET tyrosine kinase receptor." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-1529.
Full textReports on the topic "GDNF"
Svendsen, Clive N. Regulated GDNF Delivery In Vivo using Neural Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada436921.
Full textSvendsen, Clive N. Regulated GDNF Delivery in Vivo Using Neural Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada428919.
Full textSvendsen, Clive. Regulated GDNF Delivery in Vivo Using Neural Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2007. http://dx.doi.org/10.21236/ada471929.
Full textSvendsen, Clive, and Genevieve Gowing. Muscle-Derived GDNF: A Gene Therapeutic Approach for Preserving Motor Neuron Function in ALS. Fort Belvoir, VA: Defense Technical Information Center, August 2015. http://dx.doi.org/10.21236/ada621394.
Full textHoke, Ahmet, and Hai-Quan Mao. Use of GDNF-Releasing Nanofiber Nerve Guide Conduits for the Repair of Conus Medullaris/Cauda Equina Injury in the Nonhuman Primate. Fort Belvoir, VA: Defense Technical Information Center, December 2013. http://dx.doi.org/10.21236/ada613645.
Full textHoke, Ahmet, and Hai-Quan Mao. Use of GDNF-Releasing Nanofiber Nerve Guide Conduits for the Repair of Conus Medullaris/Cauda Equina Injury in the Nonhuman Primate. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada581474.
Full textChriste, Kari. Use of GDNF-Releasing Nanofiber Nerve Guide Conduits for the Repair of Conus Medullaris/Cauda Equina Injury in the Nonhuman Primate. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada599060.
Full textChriste, Kari, Leif Havton, and Ahmet Hoke. Use of GDNF-Releasing Nanofiber Nerve Guide Conduits for the Repair of Conus Medullaris/Cauda Equina Injury in the Non-Human Primate. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada581480.
Full textBlanchat, Thomas K., Patrick Dennis Brady, Dann A. Jernigan, Anay Josephine Luketa, Mark R. Nissen, Carlos Lopez, Nancy Vermillion, and Marion Michael Hightower. Cost estimate for a proposed GDF Suez LNG testing program. Office of Scientific and Technical Information (OSTI), February 2014. http://dx.doi.org/10.2172/1204098.
Full textZhou, Zhongwei, Hongli Liu, Huixiang Ju, Hongmei Chen, Li Li, Hao Jin, and Mingzhong Sun. Circulating GDF-15 in relation to the progression and prognosis of chronic kidney disease: A systematic review and dose-response meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0076.
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