Academic literature on the topic 'Gcb-Dlbcl'
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Journal articles on the topic "Gcb-Dlbcl":
Pukiat, Sulada, Nuttapong Ngamphaiboon, Pooja Advani, Julio Chavez, George Deeb, Anjana Elefante, and Francisco J. Hernandez-Ilizaliturri. "BCL-2 Expression at the Time of Diagnosis Affects the Clinical Outcome of Patients with Germinal Center and Non-Germinal Center Diffuse Large B-Cell Lymphoma (DLBCL) Treated with Chemo-Immunotherpy." Blood 116, no. 21 (November 19, 2010): 3130. http://dx.doi.org/10.1182/blood.v116.21.3130.3130.
Tarius, Jenifer Marsela, Hermawan Istiadi, Ika Pawitra Miranti, and Intan Rahmania Eka Dini. "THE CORRELATION BETWEEN CELL OF ORIGIN SUBTYPE WITH OVERALL SURVIVAL OF DIFFUSE LARGE B-CELL LYMPHOMA PATIENTS IN KARIADI GENERAL HOSPITAL SEMARANG." DIPONEGORO MEDICAL JOURNAL (JURNAL KEDOKTERAN DIPONEGORO) 9, no. 3 (May 12, 2020): 252–58. http://dx.doi.org/10.14710/dmj.v9i3.27504.
Gandhi, Shipra, Vishala T. Neppalli, George Deeb, Myron S. Czuczman, and Francisco J. Hernandez-Ilizaliturri. "Distinct CD30 Expression Patterns In Germinal Center B-Cell (GCB) and Non-GCB Diffuse Large B-Cell Lymphoma (DLBCL)." Blood 122, no. 21 (November 15, 2013): 5064. http://dx.doi.org/10.1182/blood.v122.21.5064.5064.
Hayama, Miyuki, Masataka Okamoto, Yuki Hagiwara, Ken Tanae, Mika Kohri, Naoki Takahashi, Tadashi Yoshino, Koichi Ohshima, and Nozomi Niitsu. "Clinical Significance of Immunohistochemical Markers of Diffuse Large B-Cell Lymphoma In the Rituximab Era." Blood 116, no. 21 (November 19, 2010): 1800. http://dx.doi.org/10.1182/blood.v116.21.1800.1800.
Kharchenko, Yevgeniya, Tatyana Semiglazova, Anna Artemeva, Galina Kireeva, I. Polyatskin, Ilya Zyuzgin, Larisa Filatova, Yuliya Chudinovskikh, Margarita Motalkina, and Yuliya Oleynik. "PROGNOSTIC IMPACT OF IMMUNOHISTOCHEMICAL AND MOLECULAR GENETIC MARKERS IN DIFFUSE LARGE B-CELL LYMPHOMA." Problems in oncology 66, no. 1 (January 1, 2020): 79–89. http://dx.doi.org/10.37469/0507-3758-2020-66-1-79-89.
Adhi Pangarsa, Eko, Desta Nur Ewika Ardini, Daniel Rizky, Kevin Tandarto, Hermawan Istiadi, Dik Puspasari, Budi Setiawan, et al. "The association of Hypoxia-Inducible Factor-2α (HIF-2α) overexpression score with Germinal Center B-Cell Like (GCB) and Non-Germinal Center B-Cell Like (Non-GCB) subtypes of Diffuse Large B-cell Lymphoma (DLBCL)." Bali Medical Journal 12, no. 3 (August 22, 2023): 2456–62. http://dx.doi.org/10.15562/bmj.v12i3.4521.
Mishima, Yuko, Masahiro Yokoyama, Noriko Nishimura, Kyoko Ueda, Tadahiro Gunji, Hideaki Nitta, Yoshiharu Kusano, et al. "R-CHOP Therapy Cannot Overcome CD5 Positive Non-GCB Subtype of DLBCL." Blood 126, no. 23 (December 3, 2015): 1507. http://dx.doi.org/10.1182/blood.v126.23.1507.1507.
Chavez, Julio, Mark Walsh, Francisco J. Hernandez-Ilizaliturri, Anjana Elefante, and Myron S. Czuczman. "Classification of Newly Diagnosed Diffuse Large B-Cell Lymphoma (DLBCL) According to the Han's Criteria Defines Two Groups of Patients with Different Clinical Outcomes Following Systemic Rituximab-Multi Agent Anthracycline-Based Therapy." Blood 114, no. 22 (November 20, 2009): 623. http://dx.doi.org/10.1182/blood.v114.22.623.623.
Van Meerten, Tom, Renee Bouwstra, Yuan He, de Boer Janneke, Hilde Kooistra, Rudolf Fehrmann, Emanuele Ammatuna, Gerwin Huls, and Edwin Bremer. "CD47 Expression Defines the Efficacy of Rituximab in Non-Germinal Center B-Cell (non-GCB) Diffuse Large B-Cell Lymphoma (DLBCL)." Blood 132, Supplement 1 (November 29, 2018): 2852. http://dx.doi.org/10.1182/blood-2018-99-114561.
Niitsu, Nozomi, Naoki Takahashi, Tadashi Yoshino, Masataka Okamoto, and Shigeo Nakamura. "Prognostic Significance of EBV Association in Diffuse Large B-Cell Lymphoma in the Rituximab Era." Blood 126, no. 23 (December 3, 2015): 3911. http://dx.doi.org/10.1182/blood.v126.23.3911.3911.
Dissertations / Theses on the topic "Gcb-Dlbcl":
Prévaud, Léa. "Rôle de la sous-unité c-Rel NFkB dans les Lymphômes B Diffus à Grandes Cellules du Centre Germinatif (GCB-DLBCLs) : établissement d'un modèle murin préclinique." Electronic Thesis or Diss., Limoges, 2023. http://www.theses.fr/2023LIMO0108.
The transcription factor Rel/NF-kB includes 5 subunits (SU), p50, p52, c-Rel, RelA and RelB which associate into dimers. NF-κB is at the heart of the ontogeny of mature B lymphocytes in the germinal centers (GC) for c-Rel and RelB and during plasma cell differentiation for RelA. Diffuse large cell lymphoma (DLBCL) represent more than 80% of aggressive B-cell lymphomas. We have published that NF-kB SUs must be taken into account differentially, such that RelB is a marker of poor prognosis, RelA is the SU of the ABC molecular subtype (activated B cell) and cRel that of GCB (germinal center B cell)-DLBCL with a novel clean transcriptomic signature. This project consists of understanding mechanistically how c-Rel induces the transformation of a GC B lymphocyte. We have established a new mouse model of c-Rel overexpression (with YFP) in some CG B lymphocytes (tdTomato-AID-Creert2) and are testing the clonal emergence of a tumor. The originality of this inducible model relies in the fact that it makes it possible to follow the competition between the B of the GCs on expressed c-Rel (tdTomato and YFP) compared to their normal counterpart (tdTomato)
Wu, Yen-Fei, and 吳彥霏. "The Role of Galectin-9 in Germinal Centre B-cell-like Diffuse Large B Cell Lymphoma (GCB-DLBCL)." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/72125881113790583302.
國立臺灣大學
免疫學研究所
104
Galectin-9, a tandem-repeat type galectin, was first identified in patients with a nodular sclerosis type of Hodgkin’s disease. Previous studies demonstrated various regulatory roles of galectin-9 in the immune responses. However, role of galectin-9 in cancer biology remains elusive. Here, we found galectin-9 expression in germinal centre B-cell-like diffuse large B cell lymphoma (GCB DLBCLs) was up-regulated amongst germinal center lymphomas. To determine the role of elevated galectin-9 expression in vivo, we used NOD/SCID xenograft model of GCB DLBCLs. Strikingly, we observed a dramatic reduction of tumor volume and weight in mice receiving galectin-9 knockdown GCB DLBCL cells. Interestingly, knockdown of galectin-9 did not affect the tumorigenicity in NOD scid gamma (NSG) xenograft model, which implied natural killer (NK) cells might be responsible for the reduced tumor size in NOD/SCID mice. Thus, depletion of NK cells was achieved by anti-asGM1 antibody in NOD/SCID mice. As a result, we found partially restored tumorigenicity in NOD/SCID mice engrafted with galectin-9 knockdown GCB DLBCLs after NK cell depletion. Moreover, tumor-infiltrating NK cells were significantly increased in galectin-9-depleted tumors in NOD/SCID mice. In term of molecular mechanisms, knockdown of galectin-9 induces neither apoptosis nor cell cycle arrest in GCB DLBCLs. On the other hand, we found that the level of c-Jun, a regulator in tumor microenvironment, was decreased in galectin-9 knockdown GCB DLBCL tumors. These data suggested that galectin-9 derived from GCB-DLBCL might cause the immune escape through inhibiting NK cell activity.
Book chapters on the topic "Gcb-Dlbcl":
Abraham Jacob, Linu, and Animesh Gupta. "DLBCL Subtypes and Prognosis Based on Immunophenotyping." In Lymphoma - Recent Advances [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.109216.
Conference papers on the topic "Gcb-Dlbcl":
Koning, Marvyn T., Rudolf Übelhart, Arjen H. G. Cleven, Willem H. Zoutman, Sander A. J. van der Zeeuw, Philip Kluin, Marieke Griffioen, et al. "Abstract LB-012: Autonomous, antigen-independent B-cell receptor signalling as a novel pathogenetic mechanism in non-GCB DLBCL." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-lb-012.
Maerevoet, Marie, Jason Westin, Catherine Thieblemont, Josee Zijlstra, Brian T. Hill, Fatima De La Cruz Vicente, Sylvain Choquet, et al. "Abstract CT132: A Phase 2b randomized study of selinexor in patients with relapsed/refractory Diffuse Large B-Cell Lymphoma (DLBCL) demonstrates durable responses in both GCB & Non-GCB subtypes." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-ct132.
Lin, Xiaoyu, Xiaoli Huang, Aparna Sarthy, Terry Magoc, Daniel Albert, Lloyd Lam, Tamar Uziel, et al. "Abstract 4706: ABBV-075 exhibits robust in vitro and in vivo activities against the ABC and GCB subtypes of DLBCL." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4706.
Kawagishi, Aki, Hiroki Irie, Yoshio Ogino, Hideya Komatani, and Teruhiro Utsugi. "Abstract A274: Novel SYK inhibitors have demonstrated potent antiproliferative effects in both ABC- and GCB-DLBCL cell lines via suppression of multiple pathways downstream of the B-cell receptor." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Oct 19-23, 2013; Boston, MA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.targ-13-a274.