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1

Group, Diagram, ed. Collins gem patience card games. HarperCollins, 1996.

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2

Herbelin, Yehonathan. Gam ani hayiti sham: I was there. Kineret, 2019.

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3

Elʻazar, Mosheh ben Shelomoh. Sefer Maʻaneh rakh: Bo yevoʼar ekh yitnaheg ha-adam ... she-lo yikhʻos ṿeshe-lo yaḳpid ṿe-lo yitraʻem ... ṿe-gam ... ʻal ezeh ha-ofanim mutar li-kheʻos ... ṿe-gam yevoʼar ... kol darkhe ha-teshuvah she-mefuzarim ben ha-sefarim ... Sifre Or ha-ḥayim, 2000.

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4

Health policy in Britain: The politics and organisation of the National Health Service. 2nd ed. Macmillan, 1985.

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5

Health policy in Britain: The politics and organisation of the National Health Service. 3rd ed. Macmillan, 1992.

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6

Health policy in Britain: The politics and organisation of the National Health Service. 3rd ed. Macmillan, 1992.

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7

Cui, Zhao, Neil Turner, and Ming-hui Zhao. Antiglomerular basement membrane disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0073_update_001.

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Cyclophosphamide and plasma exchange are the standard of care in rapidly progressive glomerulonephritis or lung haemorrhage caused by antiglomerular basement membrane (anti-GBM) disease, and it is unusual to encounter patients at earlier stages. Steroids are universally used in addition. There is some evidence that plasma exchange may not be a critical part of treatment at an earlier stage. There is no more than anecdotal evidence for other therapies. Slower-onset therapies such as antibodies to B cells are rarely appropriate. If untreated, patients with severe anti-GBM disease will not recove
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8

Cui, Zhao, Neil Turner, and Ming-hui Zhao. Alport post-transplant antiglomerular basement membrane disease. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0075.

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Alport antiglomerular basement membrane (anti-GBM) disease is a rare example of disease caused by allo-sensitization after renal transplantation, first described in 1992. Because the recipient lacks a specific glomerular basement membrane (GBM) protein, they can become sensitized to the normal molecule present in the GBM of the donor kidney. The disease is restricted to the allograft. Interestingly severe disease arises from this only arises rarely, certainly less than 1 in 20, probably closer to 1 in 50. It characteristically causes late graft loss in a first transplant with accelerated tempo
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9

Cattran, Daniel C., and Heather N. Reich. Membranous glomerulonephritis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0064_update_001.

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It has been clear for several decades from comparison with the rodent model disease Heymann nephritis that membranous glomerulonephritis (MGN) is an immune condition in which antibodies, usually autoantibodies, bind to targets on the surface of podocytes. However, the antigen in Heymann nephritis, megalin, is not present on human podocytes. The first potential antigen was identified by studying rare examples of maternal alloimmunization, leading to congenital membranous nephropathy in the infant caused by antibodies to neutral endopeptidase. More recently, the target of autoantibody formation
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10

Group, Diagram, and Group Diagram. Patience Card Games (Collins Gem). HarperCollins Publishers, 1996.

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11

Group, Diagram, and Group Diagram. Patience Card Games (Collins Gem). HarperCollins Publishers, 1996.

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12

Hellerstein, David J., and Ron B. Aviram. Supportive Psychotherapy and Case Management. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199997510.003.0018.

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Psychotherapy of borderline personality disorder (BPD) has often focused on specialized and putatively more effective treatments such as dialectical behavioral therapy. In recent years, accumulating evidence suggests that many BPD patients benefit equally well from well-structured but less specialized care designed to meet their needs. Supportive psychotherapy (SPT) and various forms of clinical management, such as structured clinical management (SCM) and general psychiatric management (GPM), have been successfully adapted for treatment of BPD. This chapter describes the theoretical background
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13

Sanghani, Rupa Mehta, and Kim Allan Williams. Radionuclide Angiography. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0005.

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This chapter discusses the technique and use of radionuclide angiography with planar and tomographic imaging. Planar techniques have given way to tomographic imaging in recent years. An overview of RNA is given, including technical issues such as radiopharmaceutical administration, and performance aspects including image acquisition and data interpretation. First-pass RNA (FPRNA), gated planar equilibrium RNA (ERNA) and gated tomographic equilibrium blood pool imaging (GBP-SPECT) are discussed in detail. The use of RNA in select patient populations, including coronary artery disease, valvular
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14

Filippi, Massimo, and Maria A. Rocca. Multiple Sclerosis: White Matter versus Gray Matter Involvement (The Cause of Disability in MS). Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0083.

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The classic view of multiple sclerosis (MS) as a chronic, inflammatory-demyelinating condition affecting solely the white matter (WM) of the central nervous system (CNS) has been challenged by the demonstration, from pathologic and magnetic resonance imaging (MRI) studies, of an extensive and diffuse involvement of the gray matter (GM). This observation has driven the application of modern MR technology and methods of analysis to quantify the extent and distribution of damage to the different compartments of the CNS, with the ultimate goal of improving our understanding of the factors associat
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15

Venet, Fabienne, and Alain Lepape. Immunoparesis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0313.

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In parallel with an exaggerated pro-inflammatory response, critically-ill patients develop an immunosuppressive phase, termed immunoparesis/immunoparalysis or immune reprogramming. Innate and adaptive immune responses are affected. In particular, impaired neutrophil recruitment to injury sites and abnormal accumulation in remote sites; monocyte deactivation with preferential anti-inflammatory cytokine production and altered antigen presentation capacity; and a dramatic lymphopenia associated with major induction of apoptosis, functional, and phenotypic alterations have been described. The inte
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16

Cassiman, David, Pascal Laforêt, and Fanny Mochel. Glycogen Storage Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0001.

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Glucose is the body’s major energy source, and carbohydrate serves as fuel—particularly during high-intensity exercise that requires rapid energy release. A deficiency of any of the enzymes involved in the catabolism of glycogen to glucose may cause symptoms, with hypoglycemia and exercise intolerance as the most common presentations. Glycogen storage disorders (GSD) affect muscle, liver, and brain. The most common GSDs affecting muscle are GSD II (Pompe disease) and GSD V (McArdle disease). GSDs affecting mainly the liver are GSD I, III, IV, VI, IX, XI. Most liver-GSDs present during infancy,
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17

Ver, Maria R., and Tammy S. Fouse. Vertical Banded Gastroplasty. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0036.

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Vertical banded gastroplasty (VBG) is the construction of a small vertical pouch using a linear stapler, with addition of a Marlex band placed through a window formed by a circular stapler. Compared to gastric bypass, VBG maintains a more normal anatomy of the upper gastrointestinal tract. However, due to poor long-term outcomes and a high percentage of patients requiring revisional surgery, most bariatric surgeons have abandoned VBG as a primary bariatric procedure. Early complications include acute gastric distention and gastric leaks. Late complications include mesh migration or erosion, st
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18

Karmali, Mohamed A., and Jan M. Sargeant. Verocytotoxin-producing Escherichia coli (VTEC) infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0008.

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Verocytotoxin (VT)-producing Escherichia coli (VTEC), also known as Shiga toxin producing E. coli (STEC), are zoonotic agents, which cause a potentially fatal illness whose clinical spectrum includes diarrhoea, haemorrhagic colitis, and the haemolytic uraemic syndrome (HUS). VTEC are of serious public health concern because of their association with large outbreaks and with HUS, which is the leading cause of acute renal failure in children. Although over 200 different OH serotypes of VTEC have been associated with human illness, the vast majority of reported outbreaks and sporadic cases of VTE
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19

Ham, Christopher. Health Policy in Britain: The Politics and Organisation of the National Health Service; Fifth Edition (Public Policy and Politics). 5th ed. Palgrave Macmillan, 2004.

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20

Ham, Christopher. Health Policy in Britain: The Politics and Organization of the National Health Service (Studies in Social Policy). 2nd ed. Baywood Pub Co, 1986.

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21

Ham, Christopher. Health Policy in Britain: The Politics and Organisation of the National Health Service; Fifth Edition (Public Policy and Politics). 5th ed. Palgrave Macmillan, 2004.

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22

Ham, Christopher. Health Policy in Britain: The Politics and Organization of the National Health Service. Taylor & Francis Group, 2019.

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