Relevant bibliographies by topics / Gastriti

Academic literature on the topic 'Gastriti'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Gastriti"

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Güney, Güven, Yılmaz Baş, Murat Kekilli, and Emre Demir. "Is History of Adenotonsillectomy a Protective Factor for Helicobacter pylori Gastritis?" Flora the Journal of Infectious Diseases and Clinical Microbiology 24, no. 1 (March 1, 2019): 27–31. http://dx.doi.org/10.5578/flora.67193.

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Lupu, V. V., Gabriela Păduraru, Ancuţa Ignat, Eliza Tighici-Saizu, Claudia Olaru, Nicoleta Gimiga, Angelica Cristina Marin, Cornelia Săvescu, Ioana Florea, and Marin Burlea. "GASTRITELE ŞI HELICOBACTER PYLORI LA COPIL – DIAGNOSTICUL ENDOSCOPIC PRIMAR ŞI SECUNDAR." Romanian Journal of Pediatrics 64, no. 3 (September 30, 2015): 326–29. http://dx.doi.org/10.37897/rjp.2015.3.19.

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Infecţia cu Helicobacter pylori (H. pylori) este o problemă comună şi semnificativă de sănătate publică. Obiectiv. Determinarea prevalenţei infecţiei cu H. pylori la copiii cu simptomatologie sugestivă şi conturarea cazurilor în funcţie de tipul şi felul gastritei prin efectuarea de endoscopii digestive superioare. Material şi metodă. Studiu retrospectiv realizat pe o perioadă de 5 ani pe 1.269 de copii cu simptomatologie gastrică, la care s-au efectuat endoscopii cu biopsie, stabilindu-se rata de infecţie cu H. pylori prin examen direct sau testul ureazei. Rezultate. Frecvenţa H. pylori în cazul gastritei acute a fost semnificativ mai mică (34,78%) comparativ cu frecvenţa acesteia în cazul gastritei cronice (54,94%). În cadrul celor 10 tipuri de gastrită, cele mai frecvente sunt formele purpurice (43,66%), nodular purpurică (25,93%) şi nodular antrală (15,84%), la polul opus situându-se gastrita atrofi că şi hipertrofică. Asocierile cele mai frecvente decelate endoscopic sunt cu esofagita gr I în 51,6%, duodenita în 45,07% şi forma purpurică în 24,74%. Concluzii. Infecţia cu H. pylori este cel mai frecvent asociată gastritei cronice (54,94%). Identificarea precoce a infecţiei este esenţială pentru eradicarea bacteriei şi prevenirea apariţiei diferitelor tipuri de gastrită identifi cate endoscopic.
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KUTLAR, Mehmet Can, Gülser DOĞAN, Lara GÜLKAYA, Aynur Sema DERİNYOL, Kübra Nur YEĞİN, Fatma TEKİN, Melis Nur ÖZDEMİR, et al. "Bir Kanıta Dayalı Tıp Uygulaması: Helicobacter Pylori Gastriti Tedavisinde Uygun Yaklaşım Nasıl Olmalı?" Yüksek İhtisas Üniversitesi Sağlık Bilimleri Dergisi 3, no. 1 (April 29, 2022): 6–14. http://dx.doi.org/10.51261/yiu.2022.00042.

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ABSTRACT Introduction: Evidence-Based Medicine Program, which is a part of Phase III education program of Yüksek İhtisas University Faculty of Medicine, was held between 12.10.2021 and 16.11.2021 in six divided sessions, as a total of 13 hours. Material and Method: Our study group students, together with the facilitator lecturer, divided the work among ourselves for the problems of the case chosen and obtained literature information to find out the solution by using the evidence pyramid. Results: By working on the information obtained in the practice of evidence-based medicine, the students developed solutions for the problems of a patient diagnosed with Helicobacter pylori gastritis, who was given traditional triple therapy (clarithromycin, amoxicillin, lansoprazole) and had problems during the treatment. Conclusion: It was thought that the patient was not treated appropriately. Quadruple bismuth or dual amoxicillin therapy has been suggested. Proton pump inhibitor should be given bid and the patient should comply with the 14-day treatment period. In addition to drug treatment, probiotic supplement (Lactobacillus derivatives) may also be recommended to the patient. Keywords: Evidence-based medicine, Helicobacter pylori, gastritis, treatment, dietary supplement
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Franic, Teo V., Louise M. Judd, Nhung V. Nguyen, Linda C. Samuelson, Kate L. Loveland, Andy S. Giraud, Paul A. Gleeson, and Ian R. van Driel. "Growth factors associated with gastric mucosal hypertrophy in autoimmune gastritis." American Journal of Physiology-Gastrointestinal and Liver Physiology 287, no. 4 (October 2004): G910—G918. http://dx.doi.org/10.1152/ajpgi.00469.2003.

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A prominent pathological feature of murine autoimmune gastritis is a pronounced mucosal hypertrophy. Here, we examined factors that may be responsible for inducing this hypertrophy. Because gastrin is known to be both an inducer of gastric mucosal cell proliferation and is elevated in autoimmune gastritis, mice deficient in gastrin were thymectomised at day 3 and assessed for autoimmune gastritis. Gastrin-deficient mice showed all the characteristic features of murine autoimmune gastritis, including gastric unit hypertrophy due to hyperproliferation and accumulation of immature epithelial cells, decreases in the number of zymogenic and parietal cells, and autoantibodies to the gastric H+/K+-ATPase. Hence, gastrin is not required for either the establishment of chronic gastritis or development of the typical pathological features of this disease. We also examined mRNA levels of a number of gastric mucosal growth factors in RNA samples from mice with hypertrophic autoimmune gastritis. Members of the Reg family, RegIIIβ and RegIIIγ, were greatly elevated in mice with hypertrophic gastritis, whereas RegI and amphiregulin (an EGF receptor ligand) were more modestly and/or inconsistently induced. These data demonstrate that induction of gastric mitogenic factors, such as members of the Reg family, can be achieved in inflammatory situations by gastrin-independent pathways. Members of the Reg family, in particular RegIIIβ and RegIIIγ, are good candidates to be involved in inducing the mucosal hyperproliferation in autoimmune gastritis. These findings are likely to be of relevance to other gastric inflammatory conditions.
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Sablina, Anastasiya O., Sergej S. Aleksanin, and Oleg A. Sablin. "Anti-parietal cell antibodies seroconversion in patients with autoimmune atrophic gastritis: a prospective study." HERALD of North-Western State Medical University named after I.I. Mechnikov 12, no. 1 (May 28, 2020): 71–78. http://dx.doi.org/10.17816/mechnikov202012171-78.

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The purpose of the study was to evaluate the atrophic changes of body and antrum gastric mucosa, the occurrence of Helicobacter pylori infection and the possibility of seroconversion in patients with autoimmune gastritis throughout 10 years. Material and methods. 203 Chernobyl nuclear power plant accident recovery workers were included in the prospective study. Blood levels of anti-parietal cell antibodies, basal gastrin-17, pepsinogens I and II were evaluated in all the patients to diagnose autoimmune gastritis and to assess gastric mucosa non-invasively. Results. Anti-parietal cell antibodies were found in 34.5% of the patients. Eradication rates were low (32.850.0%) in the patients with atrophy of gastric mucosa in the first 3 years of observation. Statistically significant decrease in pepsinogen I and gastrin-17 serum levels was observed in the patients with H. pylori-associated autoimmune gastritis throughout first 46 years. In the next 710 years pepsinogen I and gastrin-17 serum levels were increasing possibly due to positive effect of H. pylori eradication therapy. Successful eradication leads to disappearance of anti-parietal cell antibodies in 33.4% of the patients by the 10th year of the observation. Conclusion. The obtained results show that H. pylori eradication therapy is effective in reducing atrophic changes of gastric mucosa in the patients with autoimmune gastritis. Against the background of successful treatment the levels of pepsinogen I and gastrin-17, the markers of body and antrum gastric mucosa atrophy, were increasing. In the patients with autoimmune gastritis but without H. pylori infection the following trend was not noticed.
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Elsebaey, Mohamed A., Mohamed A. Tawfik, Samah A. Elshweikh, Manal Saad Negm, Mohammed H. Elnaggar, Ghada Mahmoud Alghazaly, and Sherief Abd-Elsalam. "Impact of Helicobacter pylori Infection on Gastric Variceal Bleeding among Patients with Liver Cirrhosis." Gastroenterology Research and Practice 2019 (February 10, 2019): 1–6. http://dx.doi.org/10.1155/2019/6529420.

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Background and Aims. Currently, it is well known that Helicobacter pylori- (H. pylori-) related peptic ulcer is one of the main causes of nonvariceal bleeding in cirrhotic patients. However, there is a lack of data to identify the exact effect ofH. pyloriinfection on variceal bleeding. This study was conducted to identify the impact ofH. pyloriinfection on gastric variceal bleeding in cirrhotic patients.Patients and Methods. 76 cirrhotic patients with gastric varices were included in this prospective study and divided into 2 groups: nonbleeding gastric varices (32 patients) and bleeding gastric varices (44 patients). The fasting serum gastrin level was measured. Mucosal biopsies from the gastric body and antrum were examined to determine the patterns of gastritis and the presence ofH. pylori.Results. The frequency ofH. pyloriinfection in the studied patients was 59.2%. There were significant differences between both groups regarding liver decompensation (P=0.001), red color sign over gastric varices (P=0.0011), prevalence ofH. pyloriinfection (P=0.0049), histological patterns of gastritis (P=0.0069), and serum gastrin level (P=0.0200). By multivariate analysis, Child C cirrhosis, red color sign over gastric varices, andH. pylori-induced follicular gastritis were independent risk factors for bleeding from gastric varices.Conclusion.H. pylori-induced follicular gastritis is considered as an additional risk factor for bleeding from gastric varices.
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Feldman, Mark, Byron Cryer, and Edward Lee. "Effects of Helicobacter pylorigastritis on gastric secretion in healthy human beings." American Journal of Physiology-Gastrointestinal and Liver Physiology 274, no. 6 (June 1, 1998): G1011—G1017. http://dx.doi.org/10.1152/ajpgi.1998.274.6.g1011.

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Helicobacter pylori gastritis is common, but effects on gastric secretion are not well understood. We measured basal and pentagastrin-stimulated gastric acidity, pepsin activity, and fluid output, as well as serum gastrin concentrations and H. pylori antibody levels, before and after treatment of H. pylori gastritis in 28 men and women. Subjects were studied before and 1 and 3 mo after a course of bismuth, metronidazole, and tetracycline. Elimination of H. pylori gastritis, accomplished in 14 subjects, increased basal and pentagastrin-stimulated gastric acidity (by 15 meq/l) and basal acid output significantly (by 2.1 meq/h 1 mo after therapy). Elimination of H. pylori had an opposite effect on pepsin secretion, significantly decreasing pepsin output by 30%. Elimination of H. pylori significantly reduced nonparietal fluid output by 35%, without affecting fluid output from parietal cells. Serum gastrin and H. pylori antibody levels declined significantly after elimination of H. pylori. None of these changes was observed in 14 subjects whose H. pylori gastritis was resistant to antimicrobial therapy. In summary, eradication of H. pylori infection increases gastric acidity by reducing nonparietal gastric secretion from peptic and other cells.
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Varro, Andrea, and Joy ES Ardill. "Gastrin: an analytical review." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 40, no. 5 (September 1, 2003): 472–80. http://dx.doi.org/10.1258/000456303322326380.

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The gastric hormone gastrin is produced in multiple forms that vary in their biological properties. In this analytical review, the strategies available for the assay of different gastrins in plasma are considered. Except for research purposes, it is seldom necessary or even desirable to employ assays that are specific for an individual molecular form of gastrin. Instead, routine clinical assays of plasma gastrin should ideally react equally with the sulphated and unsulphated forms of the COOH-terminally amidated peptides, the most important of which are peptides of 17 and 34 amino acid residues (i.e. G17 and G34), and should not react with the related hormone cholecystokinin. Methods appropriate for the use of such assays are reviewed. These assays are important in the diagnosis of gastrinoma. Although it is recognized that circulating concentrations of gastrin are elevated in other conditions, including chronic atrophic gastritis, Helicobacter pylori infection and long-term administration of proton pump inhibitors, it is not clear whether gastrin radioimmunoassay is important for the clinical management in these conditions.
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Sequenza, M. J., M. R. Loi, F. Londrino, P. Sale, S. Andrulli, A. Noce, O. Durante, et al. "Sicurezza nella scelta dell'Inibitore di Pompa Protonica nel nefropatico cronico." Giornale di Clinica Nefrologica e Dialisi 24, no. 3 (January 26, 2018): 11–15. http://dx.doi.org/10.33393/gcnd.2012.1152.

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Il paziente nefropatico cronico facilmente presenta alterazioni morfologiche e funzionali dell'apparato gastroenterico. I segni più comuni e precoci nella sindrome uremica cronica sono rappresentati dai disturbi gastrointestinali. Da alcuni decenni abbiamo a disposizione dei farmaci con potente azione inibente la secrezione acida gastrica: gli inibitori di pompa protonica (IPP) hanno una struttura chimica affine, uno stesso meccanismo d'azione e sono molto importanti per il trattamento delle patologie acido correlate, per l'eradicazione dell'Helicobacter Pylori, per la prevenzione e la cura della gastropatia da farmaci antinfiammatori non steroidei (FANS). Somministriamo ai nostri pazienti questa classe di farmaci, con terapie che continuano nel tempo, nonostante la risoluzione della malattia (gastroprotezione). Ma gli IPP possono essere utilizzati indistintamente nei nefropatici cronici oppure sarebbe utile conoscere il profilo del farmaco per una corretta scelta? In questo articolo si argomenta che i loro effetti collaterali non sono molto rilevanti e sono abbastanza simili: il loro impiego nel lungo termine è sicuro. La potenza e l'efficacia dei vari IPP, dall'analisi comparativa dei vari trial clinici, risulta essere molto simile sulla base dei milligrammi di sostanza utilizzata. L'unica eccezione illustrata in questo lavoro è rappresentata da 6 pazienti in emodialisi, trattati con lansoprazolo (15 mg), che presentavano gastriti e ulcere peptiche complicate da gravi episodi di ematemesi e melena con conseguente anemia. Tutti gli IPP hanno dimostrato un'efficacia clinica sovrapponibile, tuttavia vanno valutati di volta in volta i vantaggi (relativi) di ciascun IPP. I criteri di scelta di un IPP sembrano basati, principalmente sulle indicazioni autorizzate, sulle formulazioni disponibili, sul profilo di sicurezza del farmaco.
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Antico, Antonio, Marilina Tampoia, Danilo Villalta, Elio Tonutti, Renato Tozzoli, and Nicola Bizzaro. "Clinical Usefulness of the Serological Gastric Biopsy for the Diagnosis of Chronic Autoimmune Gastritis." Clinical and Developmental Immunology 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/520970.

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Aim. To assess the predictive value for chronic autoimmune gastritis (AIG) of the combined assay of anti-parietal-cell antibodies (PCA), anti-intrinsic-factor antibodies (IFA), anti-Helicobacter pylori(Hp) antibodies, and measurement of blood gastrin.Methods. We studied 181 consecutive patients with anemia, due to iron deficiency resistant to oral replacement therapy or to vitamin B12 deficiency.Results. 83 patients (45.8%) tested positive for PCA and underwent gastroscopy with multiple gastric biopsies. On the basis of the histological diagnosis, PCA-positive patients were divided into 4 groups: (1) 30 patients with chronic atrophic gastritis; they had high concentrations of PCA and gastrin and no detectable IFA; (2) 14 subjects with metaplastic gastric atrophy; they had high PCA, IFA, and gastrin; (3) 18 patients with nonspecific lymphocytic inflammation with increased PCA, normal gastrin levels, and absence of IFA; (4) 21 patients with multifocal atrophic gastritis with “borderline” PCA, normal gastrin, absence of IFA and presence of anti-Hp in 100% of the cases.Conclusions. The assay of four serological markers proved particularly effective in the diagnostic classification of gastritis and highly correlated with the histological profile. As such, this laboratory diagnostic profile may be considered an authentic “serological biopsy.”
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Dissertations / Theses on the topic "Gastriti"

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PIAZZA, STEFANO. "THE EFFECT OF TANNINS FROM CASTANEA SATIVA MILL. ON INFLAMMATION INDUCED BY HELICOBACTER PYLORI IN GASTRIC EPITHELIAL CELLS." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/800834.

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La terapia di eradicazione contro Helicobacter pylori (H. pylori) comporta la riduzione dei livelli di IL-8 nella mucosa gastrica e il controllo del rischio di cancro e ulcera. L’approccio terapeutico corrente è minacciato dall’antibiotico resistenza. Pertanto, la scoperta di composti vegetali di origine dietetica con effetto anti-infiammatorio e anti-batterico potrebbe contribuire al controllo della gastrite e migliorare il profilo di sicurezza della terapia. Le piante contenenti tannini, inclusi gli ellagitannini, hanno già dimostrato effetti gastro-protettivi in studi preclinici, ma richiedono ulteriore validazione farmacologica. Castanea sativa Mill. (castagno) rappresenta una fonte di tannini utilizzata tradizionalmente contro disturbi gastro-intestinali e respiratori. Gli estratti di C. sativa Mill. sono ammessi come ingredienti nutraceutici in diversi paesi europei sulla base del loro impiego tradizionale; d’altra parte, la loro composizione fitochimica e la loro attività biologica sono scarsamente studiate. Ciò comporta un limite per la documentazione di indicazioni salutistiche (health claims). L’obiettivo del seguente progetto di dottorato è stato di caratterizzare estratti idroalcolici ottenuti da frutti e foglie di C. sativa Mill., con un’attenzione particolare alla valutazione della composizione in tannini. In parallelo, gli estratti sono stati studiati come potenziali candidati contro la gastrite ed H. pylori, attraverso un approccio multidisciplinare che ha compreso esperimenti in vitro di tipo farmacologico e microbiologico. Nell’ottica di una possibile applicazione nutraceutica, il lavoro ha considerato l’importanza dell’origine botanica degli estratti e ha valutato l’impatto della digestione gastrica sulla stabilità dei composti bioattivi. I risultati conseguiti sugli estratti del frutto (la castagna) sono stati pubblicati dal gruppo di ricerca (Sangiovanni and Piazza et al. 2018): in breve, seguendo un approccio bio-guidato, gli estratti idroalcolici di frutti da 5 varietà di castagno sono stati utilizzati nel modello di rilascio di IL-8 indotta da TNFα in cellule epiteliali gastriche (AGS). Due delle varietà (Venegon e Verdesa) sono emerse per la loro attività inibitoria promettente (IC50 minore di 10 μg/mL) e di conseguenza sono state caratterizzate per il loro contenuto polifenolico. L’analisi HPLC-MS ha rilevato la presenza di tannini condensati (proantocianidine oligomeriche) negli estratti ottenuti dalle componenti esterne della castagna (pericarpo ed episperma), i quali hanno correlato con l’inibizione di IL-8 (IC50s minori di 0.5 μg/mL); al contrario, la parte edibile del frutto (l’endosperma) è risultato privo di proantocianidine e attività biologica. I tannini idrolizzabili tipici della corteccia di castagno, ovvero gallotannini ed ellagitannini, sono risultati assenti nel frutto. In accordo con la letteratura riguardante i tannini, gli estratti hanno mantenuto la loro attività inibitoria in seguito alla simulazione in vitro della digestione gastrica. Inoltre, l’attività biologica è stata modestamente intaccata da brevi trattamenti termici (fino a 100°C per 30 min), suggerendo la possibilità di recuperare le parti esterne per l’arricchimento di una farina di castagne bioattiva. Di conseguenza, una farina arricchita di episperma è stata preparata rispettando la composizione originale della castagna, quindi è stata estratta e valutata per l’inibizione di IL-8 (IC50 = 16.35 μg/mL), rivelando una potenziale applicazione nutraceutica. Seguendo un approccio simile sono stati studiati gli estratti idroalcolici delle foglie appartenenti alle varietà più promettenti (Venegon e Verdesa). Diversamente dal frutto, nella foglia sono stati riconosciuti gli ellagitannini castalagina e vescalagina (1-2%), i quali hanno stimolato l’approfondimento di un potenziale effetto contro H. pylori. In particolare, sono stati valutati l’effetto anti-batterico e anti-infiammatorio in un modello di co-coltura di cellule epiteliali gastriche infettate con il batterio (cellule tumorali AGS a confronto con cellule non-tumorali GES-1). Le foglie hanno inibito il rilascio di IL-8 con IC50 inferiori a 30 μg/mL in entrambi i modelli cellulari e hanno inibito la crescita batterica a 100 μg/mL. La simulazione della digestione gastrica ha moderatamente inficiato gli effetti biologici (2 volte) e il contenuto in fenoli totali, suggerendo la loro probabile stabilità a livello gastrico in seguito ad assunzione orale. Per gli ellagitannini castalagina e vescalagina è stato dimostrato lo stesso effetto inibitorio (IC50 di IL-8 minore di 16 μM; MIC = 25 μM), ma a concentrazioni che non spiegano completamente l’effetto dell’estratto originale; di conseguenza, la sinergia con altri polifenoli potrebbe giocare un ruolo. Il lavoro sperimentale ha posto le prime basi per il recupero a scopo nutraceutico di scarti agricoli della coltura del castagno, quali le parti esterne del frutto e le foglie, sottolineando il ruolo dei tannini per la loro attività biologica.
The eradication therapy against Helicobacter pylori (H. pylori) accounts for the reduction of IL-8 levels in gastric mucosa and the related risk of gastric cancer and ulcer onset. However, the current therapeutic approach has being challenged by antibiotic resistance. The discovery of natural compounds from edible plants with anti-inflammatory and anti-bacterial activities may contribute to the control of the disease and improve the safety profile of eradication. Plants producing tannins, including ellagitannins, have already shown pre-clinical gastroprotective evidences, but pharmacological validations are still required. Accordingly, Castanea sativa Mill. (sweet chestnut) is a well-known source of tannins, traditionally used for gastro-intestinal and respiratory diseases. On the base of the traditional indication, extracts from sweet chestnut are currently allowed as botanical ingredients by several European countries; however, their phytochemical composition and biological activity are poorly investigated, thus limiting the substantiation of health claims. The aim of the phD project was to characterize hydroalcoholic extracts from fruit and leaves of sweet chestnut, with a particular attention to tannin composition. In parallel, extracts were evaluated as potential candidates against gastritis and H. pylori through a multidisciplinary approach, including in vitro pharmacological and microbiological experiments. In the view of nutraceutical application, the work took into account the importance of the botanical origin of the extracts and evaluated the impact of gastric digestion on the stability of natural compounds. The results obtained on the fruit (chestnut) have been published by the research group (Sangiovanni and Piazza et al. 2018): in brief, following a bio-guided approach, hydroalcoholic extracts from chestnut of five different varieties were screened for their ability to impair TNFα-induced IL-8 release in human gastric epithelial cells (AGS). Two varieties (Venegon and Verdesa) emerged for their promising inhibitory activity (IC50s less than 10 μg/mL) and further characterized for their polyphenolic content. HPLC-MS analysis revealed the presence of condensed tannins (oligomeric proanthocyanidins) in extracts from the outer part of chestnut (pericarp and episperm), which correlated with IL-8 impairment (IC50s less than 0.5 μg/mL); on the contrary, the edible part (endosperm) was devoid of proanthocyanidins and inactive. Of note, hydrolysable tannins typical of the bark of sweet chestnut, namely gallotannins and ellagitannins, were absent in chestnut. In line with the literature on tannins, extracts maintained their inhibitory activity after the simulation of gastric digestion. Moreover, the bioactivity was modestly affected by the application of brief thermal treatments (till 100 °C for 30 min), thus suggesting the possibility of recovering chestnut shell for the enrichment of a bioactive chestnut flour. Consequently, an episperm-fortified flour was prepared respecting the original composition of chestnut, then extracted and assayed again for IL-8 impairment (IC50 = 16.35 μg/mL), thus demonstrating the potential application of chestnut by- products in nutraceuticals. Following a similar approach, hydroalcoholic extracts from leaf belonging to the most promising varieties (Venegon and Verdesa) were investigated. Contrarily to the fruit, the ellagitannins castalagin and vescalagin were revealed (1-2%) in leaf, thus prompting further studies on the anti-H. pylori effect. In particular, the antibacterial and anti-inflammatory activity was evaluated in a co-culture model of gastric epithelial cells infected with the bacteria (tumoral AGS in comparison with non-tumoral GES- 1). Leaf extracts inhibited IL-8 release in both the infected cells with IC50s lower than 30 μg/mL and impaired the bacterial growth at 100 μg/mL. After simulated gastric digestion, these bioactivities were moderately impaired (2 folds); in paralleled, a slight decrease in total phenols content was observed, thus suggesting their possible stability at gastric level after oral consumption. Ellagitannins castalagin and vescalagin exhibited the same inhibitory effects (IC50 on IL-8 less than 16 μM; MIC = 25 μM), but at concentrations unable to completely explain the effect of the whole extract; therefore, the synergy with other polyphenols could play a role. The experimental work sustained for the first time the potential anti-gastritis application of by- products from sweet chestnut, such as chestnut shell and leaf, pointing out the role of tannins for the biological activity.
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Duarte, Ana Rita Ribeirinho. "Pesquisa de helicobacter spp. em felinos." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2009. http://hdl.handle.net/10400.5/1335.

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Dissertação de Mestrado Integrado em Medicina Veterinária
Esta dissertação resultou da realização de um estágio curricular de seis meses no Hospital Veterinário de São Bento. Temas como Gastroenterologia e Microbiologia estavam presentes diariamente no trabalho de clínica. A gastrite pode ocorrer como processo agudo ou crónico. Uma das principais formas de gastrite crónica é a linfoplasmocítica, na qual se incluem diversas etiologias, nomeadamente bactérias como Helicobacter spp. O Helicobacter pylori é conhecido extensamente no mundo de Medicina Humana, uma vez que, desde a sua descoberta, foi associado a gastrites severas, ulceração gastroduodenal e neoplasias gástricas. Uma das lesões mais características provocadas por Helicobacter pylori é a gastrite atrófica, associada em elevado grau a neoplasia gástrica. Esta bactéria já foi identificada em gatos, provocando também doença. Existem outras espécies de bactérias do género Helicobacter infectantes do estômago de gatos e de cães, como Helicobacter felis, Helicobacter bizzozeronii, Helicobacter salomonis e Helicobacter heilmannii, numa prevalência de cerca de 90 a 100 % e 67 a 100 %, respectivamente. Porém a sua associação a patologia gástrica é controversa, surgindo lesões histológicas apenas em alguns casos. Tal como em humanos, é comum a formação de folículos linfóides. Os meios de diagnóstico de infecção por H. pylori em humanos têm evoluído, no entanto a análise histopatológica continua a ser um dos métodos mais fiáveis, que dá também acesso a informação sobre lesão gástrica. O tratamento utilizado inclui normalmente antibioterapia combinada tripla, sendo eficaz na redução de lesões histopatológicas, mas na maioria dos casos não erradica eficazmente o microrganismo. Ao longo do estágio foi desenvolvido um estudo que incluía a realização do estudo macroscópico do estômago de gatos escolhidos de modo aleatório, citologia da mucosa gástrica, teste rápido de urease e análise histopatológica das amostras de biopsia em colaboração com o laboratório de Anatomia Patológica da Faculdade de Medicina Veterinária. Em onze gatos foi revelada uma prevalência nula de microrganismos e foram encontrados cinco casos de gastrite crónica atrófica fúndica, com lesões de hiperplasia folicular em dois deles e focos esporádicos linfóides nos outros três. No entanto alguns dos animais tiveram reacção positiva ao teste de urease, entre os quais dois também tinham lesões de gastrite atrófica. Estes achados histopatológicos são importantes, uma vez que são compatíveis com a teoria de que estes animais domésticos podem ser um bom modelo de estudo para as gastrites humanas ou mesmo potenciais reservatórios de microrganismos tipo Helicobacter spp., que infectam humanos.
ABSTRACT - Helicobacter spp. Detection in Felines - This dissertation results of a six month traineeship in Hospital Veterinário de São Bento. Clinical gastroenterology and microbiology presentions in felines are a common problem in Veterinary Medicine. Particulary, gastritis can be present in its acute or chronic form. One of the most common forms of chronic gastritis is lymphoplasmacytic, where several aetiology agents can be involved, namely Helicobacter spp. bacteria. Helicobacter pylori is a well studied bacteria in Human Medicine, since it has been associated with different gastric lesions, such as severe gastritis, gastroduodenal ulceration and gastric cancer. Atrophic gastritis is related as a common feature in H. pylori infection, showing a strong relation with the onset of gastric cancer. H. pylori was also identified in stomach cats and relationed with gastric disease. Other non H. pylori-Helicobacter are known to infect cat and dog, namely Helicobacter felis, Helicobacter bizzozeronii, Helicobacter salomonis e “Helicobacter heilmannii”, being their prevalence 90 to 100 % and 67 to 100 %, respectively. Nevertheless, its relationship with gastric pathology is not well clarified and histopathogical lesions appear only in few subjects. As in humans, the appearance of lymphoid follicles was a common found. Diagnostic tests for detection of H. pylori have evoluted, but histopathological analysis is still the “golden standart” to determine the presence of the bacteria and to identify lesions in gastric mucosa. Therapeutic protocols are composed by a combination of three antibiotics, leading to the clearance of gastric mucosa lesions, but not to the complete eradication of the bacteria. In the present work a trial was made for detection of Helicobacter spp. in eleven cats, chosen randomly. The methods chosen were: macroscopic evaluation of the gastric mucosa, urease test, brush cytology and histopathological analysis, in collaboration with the Laboratório de Anatomia Patológica da Faculdade de Medicina Veterinária. The tests revealed a 0 % prevalence of microorganisms and atrophic gastritis in five cats, lesions of follicular hyperplasia in two of them and lymphoid follicles in three subjects. Nevertheless, some cats had positive reactions to urease test, two of which had also atrophic gastritis. These findings contribute to the idea that cats can represent a good model disease of human gastritis and be potentials reservatories for Helicobacter spp.
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Wen, Sicheng. "The mucosal immune response against Helicobacter pylori infection /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-033-6/.

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Ivanauskas, Audrius. "Ankstyvos skrandžio vėžio bei ikivėžinių būklių diagnostikos galimybių įvertinimas." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2008. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2008~D_20080319_133515-70953.

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Visame pasaulyje skrandžio vėžys yra didelė problema, kasmet diagnozuojama apie 875000 naujų atvejų ir 645000 miršta nuo šios ligos. Lietuvos vėžio registro duomenimis, 2005 m. Lietuvoje sergamumas skrandžio vėžiu buvo 35,0 vyrų ir 22,1 moterų tarpe 100000 gyventojų. Šis susirgimas vis dar dažniausiai diagnozuojamas vėlyvose stadijose, kuomet radikali operacija jau nėra galima. Skrandžio vėžio vystymasis yra kompleksinis ir šiuo metu dar pilnai neišaiškintas procesas. Nauji tyrimai patvirtino, kad epigenetinė pažaida – TPEF/HPP1 geno metilinimas yra dažnas reiškinys tulžies ir šlapimo pūsles, kolorektalinio vėžio atvejais. Vis dėlto, nebuvo pakankamai duomenų apie TPEF/HPP1 geno metilinimo reikšmę skrandžio kancerogenezėje. Atliktame tyrime nustatyta, kad TPEF/HPP1 geno metilinimas gali būti ankstyvas šio pažeidimo požymis. Taip pat galima daryti prielaidą, kad TPEF/HPP1 genas yra skrandžio naviką slopinantis genas. Kitas svarbus skrandžio vėžio rizikos faktorius yra H. pylori sąlygotas atrofinis gastritas. Tyrimo metu nustatytas didelis vyresnių nei 55 m. dispepsija sergančių pacientų infekuotumas H. pylori. Statistiškai patikimo skirtumo tarp atrofijos ir žarninės metaplazijos dažnumo dispepsija sergantiems pacientams Taivanyje, Lietuvoje, Latvijoje negauta. Buvo nustatyta stipri koreliacija tarp skrandžio atrofijos ir žarninės metaplazijos. Klinikinėje praktikoje atrofinis gastritas patvirtinamas histologiškai (pagal 1994 m. Hiūstone modifikuotą Sidnėjaus klasifikaciją)... [toliau žr. visą tekstą]
INTRODUCTION Gastric cancer is rampant in many countries around the world and it accounts for approximately 875000 new cases and 645000 deaths annually [Jemal A et al, 2004]. While overall incidence of gastric cancer is falling, in many countries of the world it remains one the most frequent causes of cancer related deaths. According to GLOBOCAN age-standardized cancer incidence data, in Germany 15.1 males and 8.8 females per 100.000 persons developed gastric cancer in 2002, in Lithuania – 25.3 males and 13.0 females, in Latvia – 24.6 males and 11.1 females, respectively. According to the data of Lithuanian Cancer Registry, gastric cancer incidence was 35.0 in male, 22.1 in female per 100.000 persons in 2005, 30.4 in male, 17.8 in female in 2004, respectively [Kurtinaitis J, 2004]. At present, primary or secondary prevention is likely to be the most effective means of reducing the incidence and mortality from this disease. However, to be successful, this strategy depends upon knowledge of the etiological factors and pathogenetic mechanisms involved in gastric carcinogenesis. Helicobacter pylori (H. pylori) has been categorized as a group I carcinogen by the International Agency for Research on Cancer and World Health Organization (WHO) in 1994. Development of gastric cancer is a complex and poorly understood process. It is clear that besides chronic gastritis caused by H. pylori, dietary factors, high salt and nitrate intake, smoking and, possibly, alcohol consumption are... [to full text]
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Araújo, José Carlos Ribeiro de. "Influência do refluxo duodenogástrico nas alterações histológicas da mucosa gástrica em ratos infectados por Helicobacter pylori: modelo experimental." Universidade do Estado do Rio de Janeiro, 2013. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=6343.

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O Helicobacter pylori, é tido como o principal fator de risco para o carcinoma gástrico. Diferentes estudos experimentais em animais procuram relacionar essa carcinogênese a outros fatores carcinógenos sem sucesso. Neste estudo, procurou-se avaliar-se em ratos, se há correlação entre o refluxo duodenogástrico, o Helicobacter pylori e o desenvolvimento do câncer gástrico ou de seus precursores. Para tal, realizou-se nos três grupos de ratos (n de dez por grupo) as técnicas de: piloroplastia precedida de infecção, gastrectomia subtotal precedida de infecção e um grupo no qual foi praticada apenas a infecção. Apois seis meses, analisou-se as alterações da mucosa, comparando-se os três grupos. As alterações da mucosa pesquisadas foram as seguintes: gastrites, metaplasias, displasias e neoplasias epiteliais. Ao término do estudo, foi encontrado, no grupo submetido a piloroplastia precedida de infecção um alto percentual de alterações epiteliais. Conclui-se que, no rato, a operação de piloroplastia, levou ao maior desenvolvimento da população do Helicobacter pylori, que se relaciona com as lesões pré- malignas e o adenocarcinoma gástrico.
Helicobacter pylori is considered the main risk factor for gastric carcinoma. Different experimental studies in animals seek to relate this carcinogenesis to other carcinogenic factors without success. This study sought to evaluate in rats, if there was a correlation between duodenogastric reflux, Helicobacter pylori and the development of gastric cancer or its precursors. To this end, it was carried out in three groups of rats the techniques: pyloroplasty, subtotal gastrectomy and only infection. After six months, the changes in mucosa were analyzed comparing the three groups. The mucosal changes reseached were: gastritis, metaplasia, dysplasia and epithelial neoplasms. At the end of the study, was found in the group that underwent pyloroplasty a high percentage of epithelial alterations and these correlated with the population of Helicobacter pylori. It is concluded that in the rat, the operation of pyloroplasty led to increased colonization of the population of Helicobacter pylori and is related with the development of benign lesions and gastric cancer.
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Sun, Yi-Qian. "Experimental Helicobacter pylori infection in an animal model : gastric microflora, morpho-functional development, mucosal barrier function, and effects of antioxidants in Mongolian gerbils /." Linköping : Univ, 2004. http://www.bibl.liu.se/liupubl/disp/disp2004/med876s.pdf.

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Petersson, Fredrik. "Chronic gastritis in a sample of the general population : Helicobacter pylori infection, metaplastic transformation, epithelial proliferation, p53- and p21 expression and antral mucosal gastrin content with reference to gastric carcinoma development /." Linköping : Univ, 2004. http://www.bibl.liu.se/liupubl/disp/disp2004/med855s.pdf.

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Gagnemo-Persson, Rebecca. "Interaction between calcium, calciotropic hormones and the gastrin-ECL-cell axis." Lund : Dept. of Pharmacology, Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/39777196.html.

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Tschudi, Michèle E. "Prevalence of gastritis and colonization with gastric Helicobacter spp. in dogs /." [S.l.] : [s.n.], 1998. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Motta, Cicero Roberio AraÃjo. "PrevalÃncia de lesÃes precursoras do cÃncer gÃstrico e do Helicobacter pylori em familiares de pacientes com cÃncer gÃstrico." Universidade Federal do CearÃ, 2004. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1267.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior
A infecÃÃo pelo Helicobacter pylori acomete mais da metade da populaÃÃo mundial, sendo esta bactÃria reconhecida como carcinÃgeno do grupo I pela OrganizaÃÃo Mundial de SaÃde-OMS. Familiares em primeiro grau de pacientes com cÃncer gÃstrico tÃm um maior risco de desenvolver cÃncer gÃstrico. Avaliamos a prevalÃncia de lesÃes precursoras do cÃncer gÃstrico e do Helicobacter pylori nos familiares em primeiro grau de pacientes com cÃncer gÃstrico, quando comparado a controles sem histÃria familiar. Cento e quatro familiares foram recrutados à partir de 40 casos de cÃncer gÃstrico tipo nÃo-cÃrdia e foram comparados com cento e dezoito controles, nÃo havendo diferenÃas estatisticamente significantes entre os dois grupos com relaÃÃo a idade, sexo, tabagismo, etilismo e condiÃÃes socioeconÃmicas garantindo a homogeneidade da amostra. Todos os pacientes foram submetidos a avaliaÃÃo endoscÃpica e biÃpsias seguindo o protocolo de Sydney. A anÃlise histopatolÃgica foi realizada pÃr um Ãnico patologista experiente e mascarado quanto a origem das amostras. Ainda que a prevalÃncia da atrofia e da metaplasia intestinal tenha ocorrida de forma similar nos dois grupos, a associaÃÃo destas lesÃes foi mais encontrada nos familiares que nos controles (p=0,021). A metaplasia intestinal tipo incompleta foi mais significante nos familiares (p=0,001), assim como a displasia (p=0,025). O padrÃo de gastrite encontrado nos familiares foi o de pangastrite associada a presenÃa de folÃculos linfÃides, padrÃo este jà definido como o de fenotÃpico de maior risco para a carcinogÃnese gÃstrica. NÃo houve diferenÃa estatisticamente significante entre os dois grupos com relaÃÃo a prevalÃncia do H. pylori , porÃm a topografia da infecÃÃo envolvendo antro e corpo foi maior nos familiares (p=0,001). De acordo com os resultados obtidos neste estudo, encontramos que familiares de pacientes com cÃncer gÃstrico tÃm uma maior prevalÃncia de alteraÃÃes histopatolÃgicas, estando estas alteraÃÃes confinadas a presenÃa do Helicobacter pylori
Infection by Helicobacter pylori, a bacterial species classified by WHO as being carcinogenic (group I) affects more than half the world population. First-degree relatives to patients with gastric cancer are at increased risk of developing gastric cancer. The present study evaluated the prevalence of precursor lesions of gastric cancer and infection by Helicobacter pylori in first-degree relatives to patients with gastric cancer as compared to controls with no family history of gastric cancer. One hundred four first-degree relatives to 40 patients with noncardiac gastric cancer were enrolled in the study and compared to 108 controls. The groups were statistically homogenous in terms of age. All patients were submitted to endoscopic evaluation and biopsy as described in the Sydney protocol. The histopathological analysis was carried out by a single, experienced pathologist blinded to the origin of the samples. Although the prevalence of atrophy and intestinal metaplasia was similar for the two groups, association with these lesions was more common among relatives than controls (p=0.021). Incomplete intestinal metaplasia was also more significant among relatives (p=0.001), as was displasia (p=0.025). The group of relatives presented a pattern of pangastritis associated with lymphoid follicles characteristic of increased risk for gastric carcinogenesis. There was no statistically significant difference between the groups with regard to the prevalence of H. pylori, though infection involving body and antrum was more prevalent among relatives (p=0.001). Our findings suggest that relatives to patients with gastric cancer present a greater prevalence of histopathological changes associated with the presence of H. pylori
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Books on the topic "Gastriti"

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H, Walsh John, ed. Gastrin. New York: Raven Press, 1993.

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International Symposium on Gastrin and Cholecystokinin (1987 Cap d'Agde, France). Gastrin and cholecystokinin--chemistry, physiology, and pharmacology: Proceedings of the International Symposium on Gastrin and Cholecystokinin, le Cap d'Agde (Hérhault), France, 7-11 September 1987. Edited by Bali Jean-Pierre and Martinez Jean. Amsterdam: Excerpta Medica, 1987.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. Gastritis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8.

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A, Kozol Robert, ed. Gastritis. Boca Raton: CRC Press, 1993.

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Piracha, Kashif. Chronic Atrophic Gastritis. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09872-7.

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Desai, H. G. Gastritis: Indian perspective. Mumbai: Vakils, Feffer and Simons Pvt. Ltd., 2008.

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Gastritis diet: The ultimate guide to curing stomach pains and relief from indigestion. Columbia, South Carolina: Kavshi Publishing, 2016.

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R, Holt Peter, and Russell Robert 1941-, eds. Chronic gastritis and hypochlorhydria in the elderly. Boca Raton: CRC Press, 1993.

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La gastrite di Platone. 2nd ed. Palermo: Sellerio, 1998.

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Cheli, Rodolfo. Gastritis: A critical review. Berlin: Springer-Verlag, 1987.

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Book chapters on the topic "Gastriti"

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Lee, Sang Kil. "Gastritis and Gastric Ulcers." In Clinical Gastrointestinal Endoscopy, 117–41. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-4995-8_8.

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Lee, Sang Kil. "Gastritis and Gastric Ulcers." In Clinical Gastrointestinal Endoscopy, 99–122. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35626-1_8.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Gastric Ulcer." In Gastritis, 183–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_13.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Definition of Gastritis." In Gastritis, 3–11. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_1.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Symptomatology." In Gastritis, 159–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_10.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Diagnosis." In Gastritis, 168–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_11.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Therapy." In Gastritis, 177–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_12.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Duodenal Ulcer." In Gastritis, 188–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_14.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Gastritis and Chronic Duodenitis." In Gastritis, 192–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_15.

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Cheli, Rodolfo, Alessandro Perasso, and Attilio Giacosa. "Chronic Atrophic Gastritis and Cancer." In Gastritis, 194–205. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71845-8_16.

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Conference papers on the topic "Gastriti"

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Djuria, Rachmawati Felani. "Factors Relating to Knowledge about Gastritical Swamedcation among Students at Pondok Hidayatussalikin Pangkalpinang." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.02.05.

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Background: Gastritis was one of the diseases that could be self-treatment handling. Usually gastritis was found middle age in many of student. This is because the student choose a task that many lectures and activites on school, not yet eat. One of the school in Pangkalpinang is Pondok Pesantren Hidayatussalikin. The purposes of this study is to know the factors that related to knowledge about self-treatment gastritis at Pondok Pesantren Hidayatussalikin Pangkalpinang. Subjects and Method: This was a cross-sectional study conducted at Pondok Pesantren Hidayatussalikin Pangkalpinang. A total of 130 students were enrolled in this study. The dependent variable was knowledge about self-treatment gastritis. The independen variables were interest, experience, and information resources. Results: 125 students had knowledge about self-treatment gastritis self-treatment gastritis (96.3%), 95 students (73.6%) had gastritis, and 69 students (53.6%) got information about self-treatment gastritis from one dan two media. 88 respondents (67.7%) had good level of knowledge about self-treatment gastritis. There was no statistically significant relationship between interest, experience, and information resources on the level of knowledge about self-treatment gastritis Conclusion: Knowledge about self-treatment gastritis is not associate by interest, experience, and information resources. Keywords: self-treatment gastritis, knowledge, interest, experience, information resource Correspondence: Rachmawati Felani Djuria. School of Health Polytechnics, Ministry of Health, Pangkalpinang. Jl. Telaga Biru I, Desa Padang Baru, Kecamatan Pangkalan Baru, Bangka Tengah, Bangka Belitung Province. Email: felanDJ87@gmail.com. Mobile: 081995596959. DOI: https://doi.org/10.26911/the7thicph.02.05
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Li, Zhangyong, Na Hu, Chaoshi Ren, Shengrong Liu, Chunlun Liu, and Xiaojie Fang. "Detection and Evaluation of Gastric Motility for Erosive Gastritis Patients." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE 2009). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162601.

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Barbosa, Laís Andrade, and Thaeny Lawane Purificação Ribeiro. "INFLUÊNCIA DO MANEJO DE DESMAME SOBRE A OCORRÊNCIA DE GASTRITE EM POTROS." In I Congresso Brasileiro Online de Práticas Veterinárias: Uma abordagem para animais de grande porte e produção Animal. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/granvet-11.

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Introdução: Os erros de manejo durante a separação da égua e potro podem ocasionar estresse severo e distúrbios que afetam a saúde digestiva do potro, levando a inflamações na mucosa conhecidas como gastrites. Deste modo, a indústria de suplementos alimentares se tornou destaque utilizando técnicas de manejo nutricional para apoiar a saúde gástrica, sendo de grande interesse para médicos veterinários e proprietários de cavalos. Objetivos: Avaliar a ocorrência de gastrite em potros por duas técnicas de desmame, o desenvolvimento e o efeito do uso de suplemento alimentar preventivo. Material e métodos: Foram utilizados 16 potros, machos e fêmeas, com 5 meses e peso corpóreo entre 230 e 260 kg. Os potros e as éguas foram colocados em piquete coletivo durante o período lactante das éguas. As éguas e os potros receberam a matéria seca de acordo com as exigências nutricionais da categoria. Um grupo de potros foi alojado em baias de forma individual e o restante em piquete coletivo. Após o desmame, os potros da baia foram transferidos ao piquete. O projeto utilizado foi o de blocos casualizados, sendo dois métodos de desmame e utilização ou não do suplemento alimentar. Foram avaliados o escore de lesão da mucosa gástrica dos potros e a intensidade por meio de gastroscopia. Resultados: Os resultados do teste de Tukey observaram maior diminuição da incidência de lesões dos potros desmamados em piquete em relação aos desmamados em baia, em número de lesões gástricas e sua intensidade. Os dados demonstraram a influência do manejo de desmame na intensidade de lesões. Conclusão: Conclui-se que a presença de gastrite pode ser observada antes do desmame, sendo que a incidência foi influenciada pelo manejo. Não houve impacto no desenvolvimento dos potros, e a utilização de suplemento alimentar não apresentou efeitos.
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Togo, Ren, Kenta Ishihara, Takahiro Ogawa, and Miki Haseyama. "Anonymous Gastritis Image Generation via Adversarial Learning from Gastric X-Ray Images." In 2018 25th IEEE International Conference on Image Processing (ICIP). IEEE, 2018. http://dx.doi.org/10.1109/icip.2018.8451019.

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Gao, Yu-Lan, and Yue-Han Zhang. "Gastric cancer prevention targeted on risk assessment: the Kyoto Classification of Gastritis." In 2021 11th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2021. http://dx.doi.org/10.1109/itme53901.2021.00074.

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Salem, Mahala, and Richam Gaze Hajar. "ASSOCIAÇÃO DA GASTRITE CRÔNICA COM HELICOBACTER PYLORI." In I Congresso On-line Nacional de Histologia e Embriologia Humana. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/rems/3206.

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Introdução: A gastrite crônica é principalmente causada pelo hipercloridrismo no piloro por infecção pelo Helicobacter pylori. Essa bactéria é um bacilo flagelado espiralado que se localiza sobre o epitélio da área antro-piloro. Nessa região, altera a fisiologia local e inibe o feedback negativo, inativando as células D e induzindo a hipercloridria. A virulência do H. pilory é baseada em 4 fundamentos: Flagelos: mobilidade no muco; Urease: eleva o pH gástrico; Adesinas: aderência às células foveolares (glândulas gástricas) – presente na biópsia e fator determinante para o diagnóstico e Toxinas: CagA (úlcera e câncer associado – metaplasia intestinal). Objetivos: O presente estudo visa verificar os fatores associados com o diagnóstico de gastrite crônica e a presença do H. pylori e demonstrar a importância da prevenção e tratamento da doença. Material e métodos: Este trabalho trata-se de uma revisão bibliográfica, que visa demonstração de características sobre manifestação clínica da gastrite associada ao H. pylori baseada em estudos passados. Para a realização do estudo, foram utilizados livros e artigos encontrados em sites acadêmicos como Google acadêmico e Scielo. Resultado: O Helicobacter pylori causa gastrite ao estimular a produção de citocinas pró-inflamatórias e destruir diretamente as células epiteliais. A gastrite é dividida em duas categorias, aguda e crônica, ambas com diferentes graus de descamação da superfície epitelial gástrica e diferentes graus de infiltração inflamatória. Na fase aguda, pode haver casos leves sem grandes alterações inflamatórias, enquanto em casos mais graves a inflamação pode ter sangramento adicional ou mesmo erosões da mucosa. A inflamação crônica da mucosa causada pelo Helicobacter pylori reduz a secreção ácida, levando à hipoacidez, além de reduzir a secreção de pepsina, contribuindo na proliferação bacteriana e na persistência da doença, aumentando o risco de mutações genômicas. Conclusão: A gastrite é uma patologia caracterizada por alterações histológicas na mucosa gástrica, na qual se observa infiltração de células inflamatórias. É a lesão mais comum que acomete o estômago. Pode-se concluir que múltiplas etiologias interagem para o aparecimento da gastrite crônica. Em palavras derradeiras, deve-se manter uma nutrição adequada ao longo da vida como método profilático contra o aparecimento de gastrite e o Helicobacter pylori.
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Honing, Judith, E. Dieninyte, MO Donovan, LAA Brosens, RC Fitzgerald, BLAM Weusten, and M. Di Pietro. "OFR-5 Surveillance and endoscopic recognition of gastric intestinal metaplasia and atrophic gastritis." In Abstracts of the BSG Annual Meeting, 8–12 November 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-bsg.22.

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Kanai, Misaki, Ren Togo, Takahiro Ogawa, and Miki Haseyama. "Fine-tuning of Pre-trained DCNN for Gastritis Detection from Gastric X-ray Images." In 2019 IEEE 1st Global Conference on Life Sciences and Technologies (LifeTech). IEEE, 2019. http://dx.doi.org/10.1109/lifetech.2019.8884069.

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Matsumoto, Masanao, Naoki Saito, Takahiro Ogawa, and Miki Haseyama. "Chronic Gastritis Detection from Gastric X-ray Images via Deep Autoencoding Gaussian Mixture Models." In 2019 IEEE 1st Global Conference on Life Sciences and Technologies (LifeTech). IEEE, 2019. http://dx.doi.org/10.1109/lifetech.2019.8884074.

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Togo, Ren, Kenta Ishihara, Takahiro Ogawa, and Miki Haseyama. "Anonymous Image Data Generation from Gastric X-Ray Images for Improving Gastritis Recognition Performance." In 2018 IEEE International Conference on Consumer Electronics-Taiwan (ICCE-TW). IEEE, 2018. http://dx.doi.org/10.1109/icce-china.2018.8448715.

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Reports on the topic "Gastriti"

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Chen, Chuyan, Yi Yang, Peng Li, and Haiyi Hu. Incidence of gastric neoplasms arising from autoimmune metaplastic atrophic gastritis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0021.

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Dubois, Andre. Radioprotectants and Gastric Function. Fort Belvoir, VA: Defense Technical Information Center, January 1990. http://dx.doi.org/10.21236/ada227447.

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Meng, Yan, Junren Ma, Xin Zhou, and Wei Fu. The difference of prognosis between gastric carcinoma with neuroendocrine components and pure gastric neuroendocrine carcinoma compared with gastric carcinoma: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0016.

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Parmar, Chetan. Mini (One Anastomosis) Gastric Bypass. Touch Surgery Publications, March 2019. http://dx.doi.org/10.18556/touchsurgery/2016.s0158.

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Parmar, Chetan. Mini (One Anastomosis) Gastric Bypass. Touch Surgery Simulations, March 2019. http://dx.doi.org/10.18556/touchsurgery/2019.s0158.

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Deng, Chun, Zhenyu Zhang, Zhi Guo, Hengduo Qi, Yang Liu, Haimin Xiao, and Xiaojun Li. Assessment of intraoperative use of indocyanine green fluorescence imaging on the number of lymph node dissection during minimally invasive gastrectomy: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0062.

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Review question / Objective: Whether is indocyanine green fluorescence imaging-guided lymphadenectomy feasible to improve the number of lymph node dissections during radical gastrectomy in patients with gastric cancer undergoing curative resection? Condition being studied: Gastric cancer was the sixth most common malignant tumor and the fourth leading cause of cancer-related death in the world. Radical lymphadenectomy was a standard procedure in radical gastrectomy for gastric cancer. The retrieval of more lymph nodes was beneficial for improving the accuracy of tumor staging and the long-term survival of patients with gastric cancer. Indocyanine green(ICG) near-infrared fluorescent imaging has been found to provide surgeons with effective visualization of the lymphatic anatomy. As a new surgical navigation technique, ICG near-infrared fluorescent imaging was a hot spot and had already demonstrated promising results in the localization of lymph nodes during surgery in patients with breast cancer, non–small cell lung cancer, and gastric cancer. In addition, ICG had increasingly been reported in the localization of tumor, lymph node dissection, and the evaluation of anastomotic blood supply during radical gastrectomy for gastric cancer. However, it remained unclear whether ICG fluorescence imaging would assist surgeons in performing safe and sufficient lymphadenectomy.
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Yingjun, Nie, Wang Chengyulin, and Wu Qiaofeng. Moxibustion for the treatment of chronic gastritis: A systematic review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0080.

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Atladottir, Dr Hjördis Osk, and Dr Niels Kim Schønemann. Broncho-gastric fistula complicating mechanical ventilation. The Association of Anaesthetists of Great Britain and Ireland, December 2016. http://dx.doi.org/10.21466/ac.bfcmvac.2016.

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Purkayastha, Sanjay. Laparoscopic Roux-en-Y Gastric Bypass. Touch Surgery Simulations, 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0099.

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López-Valverde, Nansi, Antonio López-Valverde, Ana Suarez, Bruno Macedo de Sousa, and Juan Manuel Aragoneses. Association of gastric infection and periodontal disease through Helicobacter pylori as a common denominator: A systematic review and meta-analysi. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0097.

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Review question / Objective: Is gastric helicobacter pylori infection related to periodontal diseases? Condition being studied: Therefore, the aim of this systematic review and meta-analysis was to identify and analyze clinical studies to determine the direct correlation between Helicobacter Pylori gastric infection andPeriodontal Disease. Study designs to be included: Clinical studies that provided data on Helicobacter Pylori infection in both the stomach and oral cavity, confirmed by polymerase chain reaction (PCR), rapid urease test (RUT) or enzyme-linked immunosorbent assay (ELISA). Clinical studies that associated PD with Helicobacter Pylori. The diagnosis of PD was confirmed ac-cording to the diagnostic criteria in periodontology.
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