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1

Buzsáki, G. Abstracts of papers presented at the 2006 meeting on the GABAergic system: December 6-December 9, 2006. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 2006.

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2

G, Bowery N., and Nisticò Giuseppe, eds. GABA: Basic research and clinical applications. Rome: Pythagora Press, 1989.

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3

Patel, Sunit Mohanlal. The effects of the inhibitory amino acid gamma-aminobutyric acid (GABA) on food intake in the rat. Portsmouth: University of Portsmouth, 2003.

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4

MacGregor, Kennaway Byron. Does Gamma-aminobutyric acid function as a plant resistance mechanism against phytophagous insect activity? St. Catharines, Ont: Brock University, Dept. of Biological Sciences, 2000.

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5

Sahley, Billie Jay. GABA: The anxiety amino acid. San Antonio, Tex: Pain & Stress Center, 1998.

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6

M, Birkner Katherine, ed. GABA: The anxiety amino acid. 2nd ed. San Antonio, Tex: Pain & Stress Publications, 2001.

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7

Sahley, Billie Jay. GABA: The anxiety amino acid. 3rd ed. San Antonio, Tex: Pain & Stress Publications, 2003.

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8

J, Enna S., and Bowery N. G, eds. The GABA receptors. 2nd ed. Totowa, N.J: Humana Press, 1997.

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9

Giorgio, Racagni, Donoso Alfredo O, and International Symposium on GABA in Endocrine Function (1985 : Buenos Aires, Argentina), eds. GABA and endocrine function. New York: Raven Press, 1986.

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10

G, Bowery N., Bittiger H, Olpe H. -R, and International GABAB̳ Symposium (1st : 1989 : King's College), eds. GABAB̳ receptors in mammalian function. Chichester, West Sussex, England: Wiley, 1990.

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11

Ellis, G. P., and G. B. West. Progress in medicinal chemistry. Amsterdam: Elsevier, 1985.

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12

1945-, Björklund Anders, and Hökfelt Tomas, eds. GABA and neuropeptides in the CNS. Amsterdam: Elsevier, 1985.

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13

Taupin, Philippe. The hippocampus: Neurotransmission and plasticity in the nervous system. New York: Nova Biomedical Books, 2007.

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14

1931-, Tanaka Chikako, and Bowery N. G, eds. GABA: Receptors, transporters, and metabolism. Basel: Birkhäuser Verlag, 1996.

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15

G, Bartholini, Lloyd K. G, Morselli Paolo Lucio, World Health Organization, and International Congress of Pharmacology (9th : 1984 : London, England), eds. GABA and mood disorders: Experimental and clinical research. New York: Raven Press, 1986.

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16

G, Avanzini, ed. Neurotransmitters in epilepsy. New York: Elsevier, 1992.

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17

Jerome, Engel, ed. Molecular neurobiology of epilepsy. Amsterdam: Elsevier, 1992.

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18

Godfrey, Tunnicliff, and Raess B. U, eds. GABA mechanisms in epilepsy. New York: Wiley-Liss, 1991.

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19

W, Olsen Richard, and Venter J. Craig, eds. Benzodiazepine/GABA receptors and chloride channels: Structural and functional properties. New York: A.R. Liss, 1986.

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20

Ranney, Mize R., Marc R. E, and Sillito A. M, eds. GABA in the retina and central visual system. Amsterdam: Elsevier, 1992.

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21

1946-, Stephens David N., ed. Anxiolytic [Beta]-carbolines: From molecular biology to the clinic. Berlin: Springer-Verlag, 1993.

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22

Letters), Alfred Benzon Symposium (32nd 1991 Royal Danish Academy of Science and. Drug research related to neuroactive amino acids: Proceedings of the Alfred Benzon Symposium 32 held at the premises of the Royal Danish Academy of Sciences and Letters, Copenhagen, June 16-20, 1991. Copenhagen: Munksgaard, 1992.

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23

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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24

Gamma-Aminobutyric Acid - Neuropsychiatric and Therapeutic Implications [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.92940.

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25

Vlainic, Josipa, and Maja Jazvinscak Jembrek. Gamma-Aminobutyric Acid: Biosynthesis, Medicinal Uses and Health Effects. Nova Science Publishers, Incorporated, 2014.

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26

Elias, Alan. Gamma-Aminobutyric Acid in the Regulation of Hormone Secretion. Praeger, 1985.

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27

Gamma-aminobutyric acid in the regulation of hormone secretion. New York: Praeger, 1985.

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28

Volkova, Yulia, Tatyana Tihonova, and Igor Zavarzin. Modern trends in the development of gamma-aminobutyric acid type A receptor modulators. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m3034.978-5-317-06826-4.

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Γ-Aminobutyric acid type A receptors (GABAARs) are among the vital biological targets that regulate the key processes of the central nervous system and human mental health: learning and memory formation processes, wakefulness and sleep. Modulators of GABAARs are widely used as anticonvulsants, anesthetics, antidepressants, sleeping medicine and anti-anxiety drugs. This monograph is the first systematic analysis of the literature data on the search for novel GABAARs-specific modulators of synthetic origin in the last ten years. The monograph is addressed to a wide range of specialists in the fields of medicinal chemistry and chemistry of biologically active compounds.
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29

(Editor), S. J. Enna, J. Thomas August (Editor), and Ferid Murad (Editor), eds. GABA, Volume 54 (Advances in Pharmacology). Academic Press, 2006.

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30

(Editor), S. J. Enna, J. Thomas August (Editor), and Ferid Murad (Editor), eds. GABA, Volume 54 (Advances in Pharmacology). Academic Press, 2006.

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31

Murad, Ferid, J. Thomas August, and S. J. Enna. Gaba. Elsevier Science & Technology Books, 2006.

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32

Sahley, Billie Jay. GABA: The Anxiety Amino Acid. 2nd ed. Pain & Stress Publications, 2001.

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33

Krogsgaard-Larsen, Povl, Jan Egebjerg, and Arne Schousboe. Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. Taylor & Francis Group, 2001.

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34

Kohn, Patricia. The effect of Thiopentone and Librium on the efflux of gamma aminobutyric acid from slices of rat cerebral cortex. 1989.

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35

(Editor), Dianna A. Redburn, and Arne Schousboe (Editor), eds. Neurotrophic Activity of Gaba During Development (Neurology and Neurobiology, V. 32). Wiley-Liss, 1987.

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36

Hu, Ming. The Effects of chronic ethanol exposure on presynaptic cholinergic receptor modulation of (3)H-GABA release in rat hippocampus. 1999.

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37

Möhler, Hanns. Pharmacology of GABA and Glycine Neurotransmission. Springer Berlin / Heidelberg, 2012.

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38

Nutt, David J., and Liam J. Nestor. The GABA system and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0008.

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Research points to the potential role of gamma-aminobutyric acid (GABA) in substance addiction. GABA is the major inhibitory neurotransmitter in the brain. Disturbances in the GABA system may predate substance abuse and addiction, whereby its efficacy to modulate other neurotransmitter systems (e.g. dopamine) strongly implicated in substance addiction behaviours is impaired. There are a number of addictive substances that boost GABA functioning, however, such as alcohol and benzodiazepines. Medications that boost the availability of GABA or mimic its effects at receptors may possess some clinical potential in treating addiction, but also have abuse liability.
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39

Jung, Bruce John. The Effects of repeated cocaine on striatal GABAergic neurotransmission in Sprague-Dawley rats. 2000.

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40

(Editor), Jan Egebjerg, Arne Schousboe (Editor), and Povl Krogsgaard-Larsen (Editor), eds. Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology (The Taylor & Francis Series in Pharmaceutical Sciences). CRC, 2001.

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41

Krogsgaard-Larsen, Povl, Jan Egebjerg, and Arne Schousboe. Glutamate and GABA Receptors and Transporters: Structure, Function and Pharmacology. Taylor & Francis Group, 2001.

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42

Krogsgaard-Larsen, Povl, Jan Egebjerg, and Arne Schousboe. Glutamate and Gaba Receptors and Transporters: Structure, Function and Pharmacology. Taylor & Francis Group, 2000.

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43

Pharmacology of GABA and Glycine Neurotransmission. Springer, 2001.

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44

Möhler, Hanns. Pharmacology of GABA and Glycine Neurotransmission. Springer London, Limited, 2012.

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45

Dienel, Samuel J., and David A. Lewis. Cellular Mechanisms of Psychotic Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0018.

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Cognitive dysfunction in schizophrenia, including disturbances in working memory, is a core feature of the illness and the best predictor of long-term functional outcome. Working memory relies on neural network oscillations in the prefrontal cortex. Gamma-aminobutyric acid (GABA) neurons in the prefrontal cortex, which are crucial for this oscillatory activity, exhibit a number of alterations in individuals diagnosed with schizophrenia. These GABA neuron disturbances may be secondary to upstream alterations in excitatory pyramidal cells in the prefrontal cortex. Together, these findings suggest both a neural substrate for working memory impairments in schizophrenia and therapeutic targets for improving functional outcomes in this patient population.
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46

(Editor), Chikako Tanaka, and Norman G. Bowery (Editor), eds. Gaba : Receptors, Transporters and Metabolism (Advances in Pharmacological Sciences) (Advances in Pharmacological Sciences). Birkhauser, 1996.

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47

Wolters, Alexander, Ulf Ziemann, and Reiner Benecke. The cortical silent period. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0010.

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The cortical silent period (cSP) refers to an interruption of voluntary muscle contraction by transcranial stimulation of the contralateral motor cortex. This article summarizes the physiology of cSP and gives guidelines as to how the cSP should be recorded and analysed, and refers to useful clinical applications. It describes the mechanisms at the spinal and supraspinal level can account for the cSP. It is currently thought that the cSP is mediated by gamma-aminobutyric acid receptors. cSP measurements are easy to obtain but require a standardized protocol to allow useful interpretation. Conditioning electrical stimulation of cutaneous nerves shortens the cSP. A conditioning transcranial magnetic stimulation pulse also affects cSP duration. The cSP duration is influenced by pathological conditions. cSP is an attractive probe to assess motor cortical inhibition in health and disease.
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48

Bowery, Norman G. Gaba: Receptors, Transporters and Metabolism. Island Press, 1995.

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49

Bolam, J. P., J. M. Tepper, and E. D. Abercrombie. GABA and the Basal Ganglia. Elsevier Science & Technology Books, 2007.

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50

(Editor), J. M. Tepper, E. D. Abercrombie (Editor), and J. P. Bolam (Editor), eds. GABA and the Basal Ganglia, Volume 160 (Progress in Brain Research) (Progress in Brain Research). Elsevier Science, 2007.

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