Academic literature on the topic 'Gamma-aminobutyric acid'

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Journal articles on the topic "Gamma-aminobutyric acid"

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Roberts, Eugene. "Gamma-aminobutyric acid." Scholarpedia 2, no. 10 (2007): 3356. http://dx.doi.org/10.4249/scholarpedia.3356.

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Habibuddin, M., S. Sen, M. Pal, and S. P. Pal. "N-Valproyl gamma aminobutyric acid." Behavioural Pharmacology 6, SUPPLEMENT 1 (May 1995): 95. http://dx.doi.org/10.1097/00008877-199505001-00111.

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Le Vo, Tam Dinh, and Soon-Ho Hong. "Optimization of gamma-Aminobutyric Acid Bioconversion by Recombinant Escherichia coli." KSBB Journal 27, no. 2 (April 30, 2012): 127–30. http://dx.doi.org/10.7841/ksbbj.2012.27.2.127.

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Girard, C. L., J. R. Seoane, and J. J. Matte. "Topographic studies of the effects of microinjections of muscimol on the hypothalamic control of feed intake in sheep." Canadian Journal of Physiology and Pharmacology 64, no. 4 (April 1, 1986): 406–10. http://dx.doi.org/10.1139/y86-065.

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Ten sheep were used to define the anatomical basis for the feeding systems sensitive to gamma-aminobutyric acid, by using intrahypothalamic microinjections of the gamma-aminobutyric acid agonist, muscimol. In satiated sheep, 1 μL of muscimol (0.5 nmol/μL) elicited feeding when injected into paraventricular, ventromedial, and anterior hypothalamic areas. Similar injections into 39 sites tested in 6-h fasted sheep failed to decrease feed intake. The data suggest that neurons sensitive to gamma-aminobutyric acid in medial hypothalamus may be involved in the initiation of feeding.
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Ho, I. K., and Beth Hoskins. "NEUROTOXICANTS AND GAMMA-AMINOBUTYRIC ACID RECEPTORS." Journal of Toxicological Sciences 15, SupplementIV (1990): 3–13. http://dx.doi.org/10.2131/jts.15.supplementiv_3.

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MacNaughton, WK, and A. Krantis. "Gamma-aminobutyric acid-induced chloride secretion." Gastroenterology 112, no. 2 (February 1997): 672–73. http://dx.doi.org/10.1053/gast.1997.v112.agast970672.

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Hardcastle, J., and PT Hardcastle. "gamma-Aminobutyric acid and intestinal secretion." Gastroenterology 111, no. 4 (October 1996): 1163–64. http://dx.doi.org/10.1016/s0016-5085(96)70096-3.

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Minuk, Gerald Y. "Gamma-Aminobutyric Acid and the Liver." Digestive Diseases 11, no. 1 (1993): 45–54. http://dx.doi.org/10.1159/000171400.

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Olsen, R. W. "Absinthe and gamma -aminobutyric acid receptors." Proceedings of the National Academy of Sciences 97, no. 9 (April 25, 2000): 4417–18. http://dx.doi.org/10.1073/pnas.97.9.4417.

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Belokrylov, G. A., and I. V. Molchanova. "Immunostimulating property of gamma-aminobutyric acid." Bulletin of Experimental Biology and Medicine 104, no. 3 (September 1987): 1284–86. http://dx.doi.org/10.1007/bf00842017.

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Dissertations / Theses on the topic "Gamma-aminobutyric acid"

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Robinson, Timothy Neil. "Gamma-aminobutyric acid-mediated neurotransmission in insects." Thesis, University of Bath, 1986. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.760552.

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Aanesen, Arthur. "Gaba and human spermatozoa : characterization and regulation of gaba transport proteins /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980925aane.

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Obrocea, Gabriela Valeria. "Ionic mechanisms of anoxia, potential role for gamma-aminobutyric acid." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0027/NQ36789.pdf.

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Wong, Amy NL 1980. "Reduced levels of brain gamma-aminobutyric acid in patients with multiple sclerosis." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98521.

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Proton magnetic resonance spectroscopy was used to measure relative GABA concentration in the brains of patients with MS. It was hypothesized that microglial activation in MS cortex and presumed associated stripping of GABAergic synapses would be associated with a lowering of GABA concentration.
Twelve patients with MS (six relapsing remitting, six secondary progressive) and five healthy control subjects were examined using a GABA-optimized STEAM sequence on a scanner operating at 3.0 tesla. A volume of 2 cm x 3 cm x 3 cm including posterior cingulate and occipital cortex was chosen. GABA/Cr was reduced by approximately 50% in patients with MS with respect to healthy control subjects. This was true for relapsing remitting patients with short duration disease, as well as secondary progressive patients with longstanding disease. NA/Cr, Ino/Cr and GABA/NA were decreased, while Glu/Cr was increased in patients with respect to normal controls.
These observations are consistent with a specific effect of MS on GABAergic interneurons. The fact that this occurs early in the disease suggests that a reduction in GABAergic inhibition may initially be part of a neuroprotective response.
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Dominguez, Jennifer Estrella. "The impact of pregnancy and childbirth on gamma-aminobutyric acid neuronal function." [New Haven, Conn. : s.n.], 2008. http://ymtdl.med.yale.edu/theses/available/etd-12022008-105710/.

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Moore, Melissa Dawn. "The role of the gamma-aminobutyric acid type A receptor in learning and memory." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1779835251&sid=27&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Lewis, John F. "The design and synthesis of sulphur heterocycles with potential as #gamma#- aminobutyric acid antagonists." Thesis, Staffordshire University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.314152.

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Hüser, Anke Christine [Verfasser], and Ulf-Ingo [Gutachter] Flügge. "Gamma-aminobutyric acid Metabolism in Arabidopsis thaliana / Anke Christine Hüser ; Gutachter: Ulf-Ingo Flügge." Köln : Universitäts- und Stadtbibliothek Köln, 2009. http://d-nb.info/116531312X/34.

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Rottkamp, Catherine Anne-Marie. "The role of Hox cofactors in vertebrate spinal coed development." Cleveland, Ohio : Case Western Reserve University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=case1194575822.

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Eroglu, Ezgi. "Association Between Gamma Aminobutyric Acid (gaba) Type B Receptors Gene Polymorphisms And Idiopathic Generalized Epilepsy." Master's thesis, METU, 2012. http://etd.lib.metu.edu.tr/upload/12614056/index.pdf.

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Epilepsy is neurological disorder affecting 0.5 to 1% of the population all around the world. It is characterized by the seizures, which are the sudden alterations of behavior due to a temporary change in electrical functioning of the brain. Idiopathic generalized epilepsy (IGE) accounts for one-fifth of all the other epilepsy types, and several gene mutations were identified as the causes of IGE. In general, voltage-gated and ligand-gated ion channel mutations are linked with seizure formation. Gamma amino butyric acid (GABA), the most important inhibitory neurotransmitter of the central nervous system, and its receptors are commonly mentioned in the pathophysiology of epilepsies. Decrease in the inhibitory effect of GABA in neurons causes epileptic discharges resulting in seizure development. The study population consisted of a total of 176 idiopathic generalized epilepsy (IGE) patients, 83 subjects having psychogenic non-epileptic seizures (PNES), 86 non-epileptic control subjects from Turkey. Total blood samples were obtained from Gü
lhane Military Medical Academy Hospital Neurology Department, Ankara. There was no statistically difference between the patient and control groups in terms of age. Genomic DNA isolations were performed and genotyping of G1465A and C59T polymorphisms of GABAB1 gene
rs1999501, rs967932, rs3780428 and rs944688 polymorphisms of GABAB2 gene were determined by PCR-RFLP technique. In this study, GABAB1 G1465A polymorphic allele was not observed in Turkish population. For GABAB1 C59T polymorphism, polymorphic allele frequencies were found as 0.097 in IGE patients
0.072 in PNES subjects and 0.105 in non-epileptic control subjects. No significant difference is identified for C59T polymorphism in all three groups. Four SNPs of GABAB2 were studied
rs967932 was found to increase the risk of IGE 3.6-fold (P=0.031) compared to PNES subjects, polymorphic allele frequencies were found as 0.060 in IGE patients
0.018 in PNES subjects and 0.035 in non-epileptic control subjects. For rs1999501 polymorphism, polymorphic allele frequencies were found as 0.077 in IGE patients
0.048 in PNES subjects and 0.093 in non-epileptic control subjects. For rs3780428 polymorphism, polymorphic allele frequencies were found as 0.267 in IGE patients
0.235 in PNES subjects and 0.256 in non-epileptic control subjects. For rs944688 polymorphism, polymorphic allele frequencies were found as 0.196 in IGE patients
0.260 in PNES subjects and 0.227 in non-epileptic control subjects. No significant difference was identified for rs1999501, rs3780428 and rs944688 polymorphisms among IGE patients, PNES subjects and non-epileptic control groups. IGE risk was 6.54-fold higher for subjects having combined GA genotype for rs967932 and GG genotype for rs3780428 when compared with PNES subjects (P=0.042). The combination of CC genotype for rs1999501, GG genotype for rs967932 and TT genotype for rs944688 had around 9-fold protective effect against IGE when both compared with PNES subjects (P=0.038) and non-epileptic control subjects (P=0.041).
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Books on the topic "Gamma-aminobutyric acid"

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Buzsáki, G. Abstracts of papers presented at the 2006 meeting on the GABAergic system: December 6-December 9, 2006. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 2006.

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G, Bowery N., and Nisticò Giuseppe, eds. GABA: Basic research and clinical applications. Rome: Pythagora Press, 1989.

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Patel, Sunit Mohanlal. The effects of the inhibitory amino acid gamma-aminobutyric acid (GABA) on food intake in the rat. Portsmouth: University of Portsmouth, 2003.

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MacGregor, Kennaway Byron. Does Gamma-aminobutyric acid function as a plant resistance mechanism against phytophagous insect activity? St. Catharines, Ont: Brock University, Dept. of Biological Sciences, 2000.

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Sahley, Billie Jay. GABA: The anxiety amino acid. San Antonio, Tex: Pain & Stress Center, 1998.

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M, Birkner Katherine, ed. GABA: The anxiety amino acid. 2nd ed. San Antonio, Tex: Pain & Stress Publications, 2001.

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Sahley, Billie Jay. GABA: The anxiety amino acid. 3rd ed. San Antonio, Tex: Pain & Stress Publications, 2003.

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J, Enna S., and Bowery N. G, eds. The GABA receptors. 2nd ed. Totowa, N.J: Humana Press, 1997.

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Giorgio, Racagni, Donoso Alfredo O, and International Symposium on GABA in Endocrine Function (1985 : Buenos Aires, Argentina), eds. GABA and endocrine function. New York: Raven Press, 1986.

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G, Bowery N., Bittiger H, Olpe H. -R, and International GABAB̳ Symposium (1st : 1989 : King's College), eds. GABAB̳ receptors in mammalian function. Chichester, West Sussex, England: Wiley, 1990.

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Book chapters on the topic "Gamma-aminobutyric acid"

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Jiang, Bo, Yuanxin Fu, and Tao Zhang. "Gamma-Aminobutyric Acid." In Bioactive Proteins and Peptides as Functional Foods and Nutraceuticals, 121–33. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9780813811048.ch9.

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Galik, Elizabeth. "Gamma-Aminobutyric Acid (GABA)." In Encyclopedia of Behavioral Medicine, 911. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1404.

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Galik, Elizabeth, Shin Fukudo, Yukari Tanaka, Yori Gidron, Tavis S. Campbell, Jillian A. Johnson, Kristin A. Zernicke, et al. "Gamma-Aminobutyric Acid (GABA)." In Encyclopedia of Behavioral Medicine, 829. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1404.

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Sapse, Anne-Marie. "Gamma-Aminobutyric Acid (GABA)." In Molecular Orbital Calculations for Amino Acids and Peptides, 27–40. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1354-3_3.

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Lummis, S. C. R., and I. L. Martin. "Convulsants and Gamma-Aminobutyric Acid Receptors." In Selective Neurotoxicity, 507–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-85117-9_14.

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Schousboe, A., B. Belhage, E. Meier, R. Hammerschlag, and G. H. Hansen. "Gamma-Aminobutyric Acid as a Neurotrophic Agent." In Amino Acid Availability and Brain Function in Health and Disease, 449–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73175-4_41.

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Alho, H., I. Hanbauer, A. Guidotti, and E. Costa. "Gamma-Aminobutyric Acid (GABA) Signaling in the Adrenal Medulla." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia, 61–66. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_11.

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Shi, Feng, Yalan Ni, and Nannan Wang. "Metabolism and Biotechnological Production of Gamma-Aminobutyric Acid (GABA)." In Industrial Biotechnology of Vitamins, Biopigments, and Antioxidants, 445–68. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2016. http://dx.doi.org/10.1002/9783527681754.ch16.

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Jones, Mathew. "Gamma-Aminobutyric Acid Type-A (GABA-A) Receptors, Kinetic Models." In Encyclopedia of Computational Neuroscience, 1258–61. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_348.

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Jones, Mathew. "Gamma-Aminobutyric Acid Type-A (GABA-A) Receptors, Kinetic Models." In Encyclopedia of Computational Neuroscience, 1–4. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7320-6_348-1.

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Conference papers on the topic "Gamma-aminobutyric acid"

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Tan, Chuanxin, Xiaohui Yan, Xiaoguang Wu, Chang Zhu, Mengxing Gou, and Xuejun Liu. "Research Progress of Gamma-Aminobutyric Acid(GABA)." In 2017 6th International Conference on Energy, Environment and Sustainable Development (ICEESD 2017). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/iceesd-17.2017.114.

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Mahayothee, Busarakorn, Supaporn Klaykruayat, Marcus Nagle, and Joachim Müller. "Effect of temperature and mode of drying on bioactive compounds and quality of germinated parboiled rice." In 21st International Drying Symposium. Valencia: Universitat Politècnica València, 2018. http://dx.doi.org/10.4995/ids2018.2018.7366.

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Germinated parboiled rice (GPR) is recognized as a functional food because it is rich in bioactive compounds, especially gamma-aminobutyric acid (GABA). GPR was produced by soaking, incubating, steaming, and then drying using a high-precision hot air dryer. The results indicated that air flow mode and drying temperature had significant effects on the quality of GPR. Drying at higher temperatures and shorter times conserved GABA content. Using through-flow mode decreased drying time and prevented color change. However, a slightly lower percentage of head rice yield was observed. Moreover, using through-flow mode negatively affected the hardness loss after cooking.Keywords: Germinated parboiled rice; Drying mode; Gamma-aminobutyric acid; Head rice yield
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Lopes, Isabela Santos, Murilo Montenegro Garrigós, and Lilia Coronato Courrol. "Metallic Nanoparticles Functionalized with Aminolevulinic Acid and Gamma-Aminobutyric Acid: Applications in Medicine and Agriculture." In 2023 International Conference on Optical MEMS and Nanophotonics (OMN) and SBFoton International Optics and Photonics Conference (SBFoton IOPC). IEEE, 2023. http://dx.doi.org/10.1109/omn/sbfotoniopc58971.2023.10230941.

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Chen, Weiqing, and Xiaofeng Wang. "Effect of Cultivating Conditions on gamma-Aminobutyric Acid Production by Lactobacillus brevis." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5516078.

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Wang, Luke Y. J., Rong Tong, and Daniel S. Kohane. "Synthesis and proton NMR spectroscopy of intra-vesicular gamma-aminobutyric acid (GABA)." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609695.

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Lopes, Isabela S., Vitor G. Vital, Suzan P. Vasconclellos, and Lilia C. Courrol. "The synergistic effects of gamma-aminobutyric acid silver nanoparticles and light in antimicrobial photodynamic therapy." In Nanophotonics X, edited by David L. Andrews, Angus J. Bain, and Antonio Ambrosio. SPIE, 2024. http://dx.doi.org/10.1117/12.3017632.

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Rusdan, Ilzamha Hadijah, Gusti Ayuning Kukilo, and Sonny Witanto. "Identification of gamma-aminobutyric acid (GABA) and dietary fiber of Indonesian local germinated brown rice (GBR)." In INTERNATIONAL CONFERENCE ON BIOINFORMATICS AND NANO-MEDICINE FROM NATURAL RESOURCES FOR BIOMEDICAL RESEARCH: 3rd Annual Scientific Meeting for Biomedical Sciences. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5109988.

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Dampf, Sara J., and Timothy M. Korter. "Low-frequency Vibrational Spectroscopy of $\gamma$-Aminobutyric Acid Derivatives: GABA Hydrochloride and $\beta$-Phenyl-GABA Hydrochloride." In 2020 45th International Conference on Infrared, Millimeter and Terahertz Waves (IRMMW-THz). IEEE, 2020. http://dx.doi.org/10.1109/irmmw-thz46771.2020.9370844.

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Wu, Weijuan. "Abstract 3309: Castration-resistant prostate cancer (CRPC) growth is mediated through gamma-aminobutyric acid a receptor (GABAAR)." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3309.

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Santos Lopes, Isabela, and Lilia Coronato Courrol. "Evaluation of sunflower seed priming with gamma-aminobutyric acid-capped silver nanoparticles produced by the photoreduction method." In Photosensitive Materials and their Applications III, edited by Robert R. McLeod, Yasuo Tomita, and Inmaculada Pascual Villalobos. SPIE, 2024. http://dx.doi.org/10.1117/12.3017479.

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Reports on the topic "Gamma-aminobutyric acid"

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Koh, Wee Yin, Babak Rasti, Xiao Xian Lim, and Wan June Tan. GABA DACHO: Dark chocolate enriched with gamma-aminobutyric acid (GABA) and inulin as prebiotic. Peeref, June 2023. http://dx.doi.org/10.54985/peeref.2306p1335975.

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Ron, Eliora, and Eugene Eugene Nester. Global functional genomics of plant cell transformation by agrobacterium. United States Department of Agriculture, March 2009. http://dx.doi.org/10.32747/2009.7695860.bard.

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The aim of this study was to carry out a global functional genomics analysis of plant cell transformation by Agrobacterium in order to define and characterize the physiology of Agrobacterium in the acidic environment of a wounded plant. We planed to study the proteome and transcriptome of Agrobacterium in response to a change in pH, from 7.2 to 5.5 and identify genes and circuits directly involved in this change. Bacteria-plant interactions involve a large number of global regulatory systems, which are essential for protection against new stressful conditions. The interaction of bacteria with their hosts has been previously studied by genetic-physiological methods. We wanted to make use of the new capabilities to study these interactions on a global scale, using transcription analysis (transcriptomics, microarrays) and proteomics (2D gel electrophoresis and mass spectrometry). The results provided extensive data on the functional genomics under conditions that partially mimic plant infection and – in addition - revealed some surprising and significant data. Thus, we identified the genes whose expression is modulated when Agrobacterium is grown under the acidic conditions found in the rhizosphere (pH 5.5), an essential environmental factor in Agrobacterium – plant interactions essential for induction of the virulence program by plant signal molecules. Among the 45 genes whose expression was significantly elevated, of special interest is the two-component chromosomally encoded system, ChvG/I which is involved in regulating acid inducible genes. A second exciting system under acid and ChvG/Icontrol is a secretion system for proteins, T6SS, encoded by 14 genes which appears to be important for Rhizobium leguminosarum nodule formation and nitrogen fixation and for virulence of Agrobacterium. The proteome analysis revealed that gamma aminobutyric acid (GABA), a metabolite secreted by wounded plants, induces the synthesis of an Agrobacterium lactonase which degrades the quorum sensing signal, N-acyl homoserine lactone (AHL), resulting in attenuation of virulence. In addition, through a transcriptomic analysis of Agrobacterium growing at the pH of the rhizosphere (pH=5.5), we demonstrated that salicylic acid (SA) a well-studied plant signal molecule important in plant defense, attenuates Agrobacterium virulence in two distinct ways - by down regulating the synthesis of the virulence (vir) genes required for the processing and transfer of the T-DNA and by inducing the same lactonase, which in turn degrades the AHL. Thus, GABA and SA with different molecular structures, induce the expression of these same genes. The identification of genes whose expression is modulated by conditions that mimic plant infection, as well as the identification of regulatory molecules that help control the early stages of infection, advance our understanding of this complex bacterial-plant interaction and has immediate potential applications to modify it. We expect that the data generated by our research will be used to develop novel strategies for the control of crown gall disease. Moreover, these results will also provide the basis for future biotechnological approaches that will use genetic manipulations to improve bacterial-plant interactions, leading to more efficient DNA transfer to recalcitrant plants and robust symbiosis. These advances will, in turn, contribute to plant protection by introducing genes for resistance against other bacteria, pests and environmental stress.
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