Books on the topic 'Fungal'

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1

Jennings, D. H. Fungal biology: Understanding the fungal lifestyle. Oxford: BIOS Scientific Publishers, 1996.

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2

G, Lysek, ed. Fungal biology: Understanding the fungal lifestyle. 2nd ed. Oxford, UK: BIOS, 1999.

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3

Fungal morphogenesis. Cambridge: Cambridge University Press, 1998.

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4

Kumar, Sachin, Pratibha Dheeran, Mohammad Taherzadeh, and Samir Khanal, eds. Fungal Biorefineries. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90379-8.

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5

O'Connor, Louise, and Barry Glynn, eds. Fungal Diagnostics. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-257-5.

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6

Kronstad, J. W., ed. Fungal Pathology. Dordrecht: Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-015-9546-9.

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7

Richardson, Malcolm D., and David W. Warnock. Fungal Infection. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118321492.

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8

Drouhet, Edouard, Garry T. Cole, Louis de Repentigny, Jean-Paul Latgé, and Bertrand Dupont, eds. Fungal Antigens. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0773-0.

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9

Hock, Bertold, ed. Fungal Associations. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30826-0.

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10

Hock, Bertold, ed. Fungal Associations. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-662-07334-6.

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11

Deacon, Jim. Fungal Biology. Malden, MA USA: Blackwell Publishing Ltd., 2005. http://dx.doi.org/10.1002/9781118685068.

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12

Richardson, Malcolm D., and David W. Warnock, eds. Fungal Infection. Oxford, UK: Blackwell Publishing Ltd, 2003. http://dx.doi.org/10.1002/9780470755259.

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13

Xu, Jin-Rong, and Burton H. Bluhm, eds. Fungal Genomics. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-040-9.

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14

Gupta, Vijai Kumar, Robert L. Mach, and S. Sreenivasaprasad, eds. Fungal Biomolecules. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118958308.

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15

Dix, Neville J., and John Webster. Fungal Ecology. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0693-1.

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16

Nowrousian, Minou, ed. Fungal Genomics. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-45218-5.

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17

Szaniszlo, Paul J., and James L. Harris, eds. Fungal Dimorphism. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4982-2.

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18

Fidel, Paul L., and Gary B. Huffnagle, eds. Fungal Immunology. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/b107411.

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19

Brown, Alistair J. P., ed. Fungal Genomics. Berlin/Heidelberg: Springer-Verlag, 2006. http://dx.doi.org/10.1007/3-540-30809-1.

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20

de Vries, Ronald P., Adrian Tsang, and Igor V. Grigoriev, eds. Fungal Genomics. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7804-5.

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21

Prasad, Ram, ed. Fungal Nanotechnology. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-68424-6.

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22

Mérillon, Jean-Michel, and Kishan Gopal Ramawat, eds. Fungal Metabolites. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19456-1.

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23

Dhusia, Kalyani, Kalpana Raja, and Pramod Ramteke, eds. Fungal Siderophores. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-53077-8.

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24

K, Arora Dilip, Elander Richard P, and Mukerji K. G, eds. Fungal biotechnology. New York, N.Y: Marcel Dekker, 1992.

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25

Griffin, David H. Fungal physiology. 2nd ed. New York: Wiley-Liss, 1994.

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26

P, Jones Michael, ed. Fungal diseases. Philadelphia: W.B. Saunders, 2003.

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27

T, Anke, ed. Fungal biotechnology. London: Chapman & Hall, 1997.

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28

L, Mandell Gerald, Diamond Richard D. 1942-, and Current Medicine Inc, eds. Fungal infections. Philadelphia: Current Medicine, 2000.

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29

Hudson, H. J. Fungal biology. London: Edward Arnold, 1986.

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30

Borman, Andrew M. Fungal taxonomy and nomenclature. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0002.

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This chapter summarizes historical and modern approaches to fungal taxonomy, the current taxonomic standing of medically important fungi, and the implications for fungal nomenclature following the recent Amsterdam Declaration on Fungal Nomenclature, which prohibits dual nomenclature. Fungi comprise an entire kingdom, containing an estimated 1–10 million species. Traditionally, fungal identification was based on examination of morphological and phenotypic features, including the type of sexual spores they form, and method of formation, and structural features of their asexual spores. Thus, many fungi have been described and named independently several times based on either their sexual or asexual stages, resulting in a single genetic entity having multiple names. Recent molecular approaches to fungal identification have led to profound changes in fungal nomenclature and taxonomy. Certain phyla have now been disbanded, cryptic species have been identified via molecular approaches, and long-recognized species have been transferred to new genera based on genotypic comparisons.
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31

Drake, Sarah, and Jonathan Sandoe. Fungal cardiovascular infections. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0021.

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Fungal cardiovascular disease can broadly be divided into four groups: infective endocarditis (including implantable cardiac electronic devices), mycotic aneurysms, vascular graft infections, and intravascular catheter-related infections. These conditions are rare but are associated with significant morbidity and mortality, which may be in excess of 80% in certain groups of patients. Candida spp. and Aspergillus spp. account for the majority of these infections, but rare fungi may also be involved, particularly in infective endocarditis, where they are responsible for approximately 25% of cases. This chapter will cover the epidemiology, causative fungi, clinical features, diagnosis, management, and prevention of these four fungal cardiovascular conditions.
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32

Odds, Frank C. Pathogenesis of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0008.

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The pathogenesis of fungal disease involves an interplay between fungal virulence factors and host immune responses. Most fungal pathogens are opportunists that preferentially invade hosts with immune defects, but the fact that relative pathogenicity varies between fungal species (and even between different strains within a species) is evidence that fungi have evolved multiple, different molecular virulence factors. Experiments in which genes encoding putative virulence attributes are specifically disrupted and the resulting mutants are tested for virulence in a range of vertebrate and invertebrate hosts have identified or confirmed many gene products as significant for the pathogenesis of various types of fungal disease. These include factors determining fungal shape in vivo, biofilm formation, and a plethora of surface components, including adhesins and hydrolytic enzymes. This chapter provides an overview of fungal virulence attributes.
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33

Barton, Richard. Serology of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0042.

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Examination of serum and other body fluids for the presence of antibodies to fungi, or the direct detection of the fungal antigens themselves, can play an important role in the diagnosis of fungal disease. Various methods have been applied, though currently the most commonly used is some form of enzyme-linked immunosorbent assay. Antigen detection has become a standard method for diagnosing cryptococcosis and can play a key role in detecting aspergillosis, and to a lesser extent candidiasis, depending on the underlying disease. Antibody testing is routine for many fungal diseases, including coccidioidomycosis, histoplasmosis, and many forms of aspergillosis. Beta-D-glucan is a generic fungal antigen found in the cell walls of many fungi, and detection of BDG is a test which many find useful when screening the sera of at-risk patients. Increasingly, physicians and scientists are looking to serodiagnostic tests not only to diagnose, but also to monitor treatment outcomes.
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34

Dambuza, Ivy M., Jeanette Wagener, Gordon D. Brown, and Neil A. R. Gow. Immunology of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0009.

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Advances in modern medicine, such as organ transplantations and the appearance of HIV (human immunodeficiency virus), have significantly increased the patient cohort at risk of developing chronic superficial and life-threatening invasive fungal infections. To tackle this major healthcare problem, there is an urgent need to understand immunity against fungal infections for the purposes of vaccine design or immune-mediated interventions. In this chapter, we give an overview of the components of the innate and adaptive immune system and how they contribute to host defence against fungi. The various cell types contributing to fungal recognition and the subsequent stimulation of phagocytosis, the activation of inflammatory and B- and T-cell responses, and fungal clearance are discussed using the major fungal pathogens as model systems.
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35

Jennings, Prof D. Fungal Biology: Understanding the Fungal Lifestyle. Garland Science, 1999.

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36

Jennings, D. H., and G. Lysek. Fungal Biology: Understanding the Fungal Lifestyle. Bios Scientific Pub Ltd, 1999.

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37

Tunnicliffe, Georgia, and Matthew Wise. Pulmonary fungal infections. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0007.

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Pulmonary fungal infections remain relatively uncommon, although they are increasingly diagnosed as a consequence of a growing population of immunocompromised individuals, foreign travel, and improved diagnostic tools. Groups who were not previously thought to be at significant risk of invasive disease are also being recognized. The increasing incidence of fungal lung disease as a consequence of changing patient demographics means that clinicians will encounter cases in outpatient clinics, medical admission departments, and the intensive care unit with increasing frequency. As international travel increases, so too will presentations of endemic mycoses to respiratory physicians practising in the United Kingdom. Many fungi, such as Aspergillus species, are ubiquitous and can cause a spectrum of pulmonary disorders from colonization, leading to hypersensitivity reactions, to invasive disease with high mortality rates. This chapter considers commonly encountered fungi and how diseases associated with them may present.
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38

Matthews, Philippa C. Fungal infections. Edited by Philippa C. Matthews. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737773.003.0011.

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This chapter consists of short notes, diagrams, maps, and tables to summarize fungal infections that are significant causes of disease in the tropics and subtropics, with a primary focus on dimorphic fungi (Histoplasma, Blastomyces, Coccidioides, Paracoccidioides, and Penicillium species). The chapter also includes cryptococcal infection and Madura foot. For ease of reference, each topic is broken down into sections, including classification, epidemiology, microbiology, pathophysiology, clinical syndromes, diagnosis, treatment and prevention.
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39

Dighton, John, James F. White, and Peter Oudemans. Fungal Community. Taylor & Francis Group, 2010.

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40

Lalithakumari, D. Fungal Protoplast. Taylor & Francis Group, 2019.

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41

Dighton, John, and James F. White. Fungal Community. Taylor & Francis Group, 2021.

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42

Cleverley, Joanne. The imaging of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0041.

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The imaging of fungal infection is diverse and often non-specific with multiple abnormalities commonly identified, frequently with more than one organ involved. By correlating the clinical information, which should include patient immune status, pre-existing chronic disease, and potential exposure to endemic fungi, and using this information with an awareness of the radiographic findings of fungal infection, a potential diagnosis can be ascertained. In this chapter, the imaging of fungal infection is discussed, concentrating on the various imaging modalities available, their role, and the major organs involved, highlighting any distinguishing radiographic findings, which may help in the search for a definitive diagnosis.
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43

Mycoremediation: Fungal Bioremediation. Wiley-Interscience, 2006.

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44

Singh, Harbhajan. Mycoremediation: Fungal Bioremediation. Wiley & Sons, Incorporated, John, 2006.

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45

Gadd, G. M., N. A. R. Gow, and G. D. Robson. Fungal Colony. Cambridge University Press, 2010.

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46

Ramawat, Kishan Gopal, and Jean-Michel Mérillon. Fungal Metabolites. Springer, 2017.

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47

Hock, Bertold. Fungal Associations. Springer, 2012.

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48

Buck, Kenneth William. Fungal Virology. Taylor & Francis Group, 2018.

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49

Kemper, Carol A., and Stanley C. Deresinski. Fungal arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0106.

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Fungal infections of the musculoskeletal system are uncommon and diagnosis is often delayed. Infection is more common in the immunocompromised patient. The most important infections are due to candida species, Histoplasmosis capsulatum, Blastomycosis dermatiditis, and Coccidioides immitis. Amphotericin B remains the initial therapeutic agent of choice for many serious fungal infections, especially for those who are severely immunosuppressed, have life-threatening or central nervous system disease, or who have failed azole therapy.
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50

Todd, Stacy, and Nick Beeching. Fungal infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0315.

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Fungi, comprising yeasts, moulds, and higher fungi, have a worldwide distribution and are uncommon causes of disease in healthy individuals. However, over the last 20 years, invasive fungal disease (IFD) has become an increasing cause of morbidity and mortality. This is probably due to the increasing numbers of patients with underlying host conditions, which predispose to opportunistic IFD (e.g. transplant and anti-tumour necrosis factor immunosuppression, HIV, or chronic lung disease), and to increased recognition of endemic IFD (e.g. histoplasmosis), which cause disease in both immunocompetent and immunocompromised hosts in selected geographic locations. Diagnosis of IFD remains a challenge. Symptoms are often non-specific, and a definite diagnosis requires invasive sampling with appropriate laboratory testing of these samples. Non-invasive tests are being developed, but their positive and negative predictive values still need validation. Diagnostic criteria (‘proven, probable, and possible’) established primarily for use in research and clinical trials can also prove useful in clinical environments. However, the most important step in identifying patients with IFD is to consider the diagnosis in those at risk. This chapter will focus on the commonest causes of IFD (Candida spp., Aspergillus spp., Cryptococcus spp., and histoplasmosis).
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