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Journal articles on the topic 'Frontotemporal dementia'

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Author: Grafiati

Published: 4 June 2021

Last updated: 7 July 2024

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1

Snowden, Julie S., David Neary, and David M. A. Mann. "Frontotemporal dementia." British Journal of Psychiatry 180, no. 2 (February 2002): 140–43. http://dx.doi.org/10.1192/bjp.180.2.140.

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BackgroundFrontotemporal dementia accounts for up to 20% of cases of dementia in the presenium, yet remains poorly recognised. Diagnostic criteria have been devised to aid clinical diagnosis.AimsTo provide an overview of clinical and pathological characteristics of frontotemporal dementia and its nosological status.MethodsThe review summarises consensus diagnostic criteria for frontotemporal dementia and draws on the authors'clinical experience of 300 frontotemporal dementia cases, and pathological experience of 50 autopsied cases.ResultsFrontotemporal dementia is characterised by pronounced changes in affect and personal and social conduct. Some patients also develop motor neuron disease. Mutations in the tau gene account for some but not all familial cases of frontotemporal dementia.ConclusionsFrontotemporal dementia is a focal form of dementia, which is clinically and pathologically distinct from other dementias. It represents an important model for understanding the functions of the frontotemporal lobes.

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2

Dacka, Michał, Mikołaj Porzak, Karol Bochyński, Konrad Białogłowski, Paulina Dąbrowska, Michał Żuber, Katarzyna Ciuba, Katarzyna Molenda, Filip Borodziuk, and Barbara Borodziuk. "Frontotemporal Dementia: A Clinical Review." Journal of Education, Health and Sport 59 (February 14, 2024): 235–46. http://dx.doi.org/10.12775/jehs.2024.59.015.

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Frontotemporal dementia is a disease in which atrophic changes occur in the frontal lobes and frontal temporal lobes of the brain. Frontotemporal dementias are a clinically, neuroanatomically and pathologically diverse group of diseases that collectively constitute an important cause of young-onset dementia. The most common form of frontotemporal dementia is the so-called behavioral variant of frontotemporal dementia. Underlying these pathological changes is the degeneration of nerve cells (i.e. neurons), which occurs through the accumulation of abnormal proteins inside them. Therefore, the review of current studies in the subject of Frontotemporal dementia was conducted in order to access possible risk factors and new ways of management and treatment of this complex disease.

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3

Rusina, Robert, Radoslav Mmatěj, Zsolt Cséfalvay, Jiří Keller, Vanda Franková, and Martin Vyhnálek. "Frontotemporal dementia." Česká a slovenská neurologie a neurochirurgie 84/117, no. 1 (February 28, 2021): 9–29. http://dx.doi.org/10.48095/cccsnn20219.

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4

Swartz, J. Randolph, Bruce L. Miller, Ira M. Lesser, and Amy L. Darby. "Frontotemporal Dementia." Journal of Clinical Psychiatry 58, no. 5 (May 15, 1997): 212–17. http://dx.doi.org/10.4088/jcp.v58n0506.

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5

Fedorova, Yana B. "Frontotemporal dementia." Psychiatry 77 (2018): 60–85. http://dx.doi.org/10.30629/2618-6667-2018-77-60-85.

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6

Marshall, Katherine, and Deborah Hale. "Frontotemporal Dementia." Home Healthcare Now 40, no. 4 (July 2022): 223. http://dx.doi.org/10.1097/nhh.0000000000001092.

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7

Rosness, Tor Atle, Knut Engedal, and Zeina Chemali. "Frontotemporal Dementia." Journal of Geriatric Psychiatry and Neurology 29, no. 5 (August 8, 2016): 271–80. http://dx.doi.org/10.1177/0891988716654986.

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8

Graff-Radford, Neill, and Bryan Woodruff. "Frontotemporal Dementia." Seminars in Neurology 27, no. 1 (February 2007): 048–57. http://dx.doi.org/10.1055/s-2006-956755.

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9

Warren, J. D., J. D. Rohrer, and M. N. Rossor. "Frontotemporal dementia." BMJ 347, aug12 3 (August 6, 2013): f4827. http://dx.doi.org/10.1136/bmj.f4827.

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10

Miller, Bruce, and David Perry. "Frontotemporal Dementia." Seminars in Neurology 33, no. 04 (November 14, 2013): 336–41. http://dx.doi.org/10.1055/s-0033-1359316.

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11

Kertesz, Andrew. "Frontotemporal Dementia." Cognitive and Behavioral Neurology 21, no. 3 (September 2008): 127–33. http://dx.doi.org/10.1097/wnn.0b013e31818a8c66.

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12

Grossman, Murray. "Frontotemporal Dementia." Alzheimer Disease & Associated Disorders 19, Supplement 1 (October 2005): S1—S2. http://dx.doi.org/10.1097/01.wad.0000183087.07315.a7.

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13

Knibb, Jonathan A., Christopher M. Kipps, and John R. Hodges. "Frontotemporal dementia." Current Opinion in Neurology 19, no. 6 (December 2006): 565–71. http://dx.doi.org/10.1097/01.wco.0000247606.57567.41.

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14

Nurrenbrock, Mary. "Frontotemporal Dementia." Neurology Now 5, no. 5 (September 2009): 8. http://dx.doi.org/10.1097/01.nnn.0000361349.38521.2e.

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15

&NA;. "Frontotemporal Dementia." Neurology Now 5, no. 5 (September 2009): 8. http://dx.doi.org/10.1097/01.nnn.0000361350.38521.f8.

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16

Graham, Andrew, and John R. Hodges. "Frontotemporal dementia." Psychiatry 4, no. 1 (January 2005): 55–58. http://dx.doi.org/10.1383/psyt.4.1.55.58313.

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17

Seeley, William W., Juan Zhou, and Eun-Joo Kim. "Frontotemporal Dementia." Neuroscientist 18, no. 4 (June 13, 2011): 373–85. http://dx.doi.org/10.1177/1073858411410354.

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18

Neary, David, Julie Snowden, and David Mann. "Frontotemporal dementia." Lancet Neurology 4, no. 11 (November 2005): 771–80. http://dx.doi.org/10.1016/s1474-4422(05)70223-4.

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19

Kumar-Singh, Samir, and Marc Cruts. "Frontotemporal dementia." Lancet Neurology 7, no. 8 (August 2008): 681. http://dx.doi.org/10.1016/s1474-4422(08)70160-1.

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20

Rosen, Howard J., Jeannie Lengenfelder, and Bruce Miller. "FRONTOTEMPORAL DEMENTIA." Neurologic Clinics 18, no. 4 (November 2000): 979–92. http://dx.doi.org/10.1016/s0733-8619(05)70235-8.

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21

Honig, L. S. "Frontotemporal Dementia." Science of Aging Knowledge Environment 2003, no. 13 (April 2, 2003): 1dn—1. http://dx.doi.org/10.1126/sageke.2003.13.dn1.

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22

Viskontas, Indre, and Bruce Miller. "FRONTOTEMPORAL DEMENTIA." CONTINUUM: Lifelong Learning in Neurology 13 (April 2007): 87–108. http://dx.doi.org/10.1212/01.con.0000267237.15122.52.

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23

Graff-Radford, Neill, and Bryan Woodruff. "FRONTOTEMPORAL DEMENTIA." CONTINUUM: Lifelong Learning in Neurology 10 (February 2004): 58–80. http://dx.doi.org/10.1212/01.con.0000293547.16812.60.

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24

Kertesz, Andrew, and David G. Munoz. "Frontotemporal dementia." Medical Clinics of North America 86, no. 3 (May 2002): 501–18. http://dx.doi.org/10.1016/s0025-7125(02)00011-1.

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25

Bang, Jee, Salvatore Spina, and Bruce L. Miller. "Frontotemporal dementia." Lancet 386, no. 10004 (October 2015): 1672–82. http://dx.doi.org/10.1016/s0140-6736(15)00461-4.

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26

Mariani, C., S. Defendi, E. Mailland, and S. Pomati. "Frontotemporal dementia." Neurological Sciences 27, S1 (March 2006): s35—s36. http://dx.doi.org/10.1007/s10072-006-0544-8.

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27

Kelley, Roger E., and Ramy El-Khoury. "Frontotemporal Dementia." Neurologic Clinics 34, no. 1 (February 2016): 171–81. http://dx.doi.org/10.1016/j.ncl.2015.08.007.

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28

Olney, Nicholas T., Salvatore Spina, and Bruce L. Miller. "Frontotemporal Dementia." Neurologic Clinics 35, no. 2 (May 2017): 339–74. http://dx.doi.org/10.1016/j.ncl.2017.01.008.

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29

Younes, Kyan, and Bruce L. Miller. "Frontotemporal Dementia." Psychiatric Clinics of North America 43, no. 2 (June 2020): 331–44. http://dx.doi.org/10.1016/j.psc.2020.02.006.

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30

Goldman, Jill S., Jennifer M. Farmer, Vivianna M. Van Deerlin, Kirk C. Wilhelmsen, Bruce L. Miller, and Murray Grossman. "Frontotemporal Dementia:." Neurologist 10, no. 5 (September 2004): 227–34. http://dx.doi.org/10.1097/01.nrl.0000138735.48533.26.

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31

Graham, Andrew, and John R. Hodges. "Frontotemporal dementia." Psychiatry 7, no. 1 (January 2008): 24–28. http://dx.doi.org/10.1016/j.mppsy.2007.11.008.

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32

Roberson, Erik D. "Frontotemporal dementia." Current Neurology and Neuroscience Reports 6, no. 6 (November 2006): 481–89. http://dx.doi.org/10.1007/s11910-006-0050-7.

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33

Medina-Rioja, Raul, Gina Gonzalez-Calderon, and Mario Masellis. "Frontotemporal dementia." Canadian Medical Association Journal 195, no. 48 (December 10, 2023): E1660. http://dx.doi.org/10.1503/cmaj.230407.

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34

Sivasathiaseelan, Harri, Charles Marshall, Jennifer Agustus, Elia Benhamou, Rebecca Bond, Janneke van Leeuwen, Chris Hardy, Jonathan Rohrer, and Jason Warren. "Frontotemporal Dementia: A Clinical Review." Seminars in Neurology 39, no. 02 (March 29, 2019): 251–63. http://dx.doi.org/10.1055/s-0039-1683379.

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AbstractFrontotemporal dementias are a clinically, neuroanatomically, and pathologically diverse group of diseases that collectively constitute an important cause of young-onset dementia. Clinically, frontotemporal dementias characteristically strike capacities that define us as individuals, presenting broadly as disorders of social behavior or language. Neurobiologically, these diseases can be regarded as “molecular nexopathies,” a paradigm for selective targeting and destruction of brain networks by pathogenic proteins. Mutations in three major genes collectively account for a substantial proportion of behavioral presentations, with far-reaching implications for the lives of families but also potential opportunities for presymptomatic diagnosis and intervention. Predicting molecular pathology from clinical and radiological phenotypes remains challenging; however, certain patterns have been identified, and genetically mediated forms of frontotemporal dementia have spearheaded this enterprise. Here we present a clinical roadmap for diagnosis and assessment of the frontotemporal dementias, motivated by our emerging understanding of the mechanisms by which pathogenic protein effects at the cellular level translate to abnormal neural network physiology and ultimately, complex clinical symptoms. We conclude by outlining principles of management and prospects for disease modification.

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35

Fonseca, L., and Á. Machado. "Suicide in frontotemporal dementia." European Psychiatry 26, S2 (March 2011): 487. http://dx.doi.org/10.1016/s0924-9338(11)72194-0.

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IntroductionBehavioral and personality changes are the core symptoms of frontotemporal dementia. Suicide and suicide attempts have been reported in demented patients.Clinical caseWe present a case of an 80 years-old-male patient, with a suicide attempt at the age of 76 as the presentation symptom of FTD.Clincal studyThere are few studies of suicide or selfharm in frontotemporal dementia where such behavior might be expected to be more common. We are conducting a clinical study in FTD patients about the relation between FTD and suicide. The results of such study will be presented and discussed.DiscussionTo our knowledge, there are no reliable data or reports about suicide in FTD patients. Also, we didn’t find any case report of a suicide attempt as the first presentation symptom of FTD. We discuss the known data about this issue considering our clinical study and report.

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36

Massano, João, Miguel Leão, Carolina Garrett, and On behalf of Grupo de Neurogenética do Centro Hospitalar São João. "Investigação de Etiologia Genética nas Demências Neurodegenerativas: Recomendações do Grupo de Neurogenética do Centro Hospitalar São João." Acta Médica Portuguesa 29, no. 10 (October 31, 2016): 675. http://dx.doi.org/10.20344/amp.7583.

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In the past few years several gene mutations have been identified as causative of the most frequent neurodegenerative dementias (Alzheimer disease and frontotemporal dementia). These advances, along with the complex phenotype-genotype relationships and the costs associated with genetic testing, have often made it difficult for clinicians to decide with regard to a rational plan for the investigation of the genetic etiology of the degenerative dementias. The Centro Hospitalar São João Neurogenetics Group, a multidisciplinary team of Neurologists and Geneticists with special interest in neurogenetic disorders, devised consensus recommendations for the investigation of the genetic etiology of Alzheimer disease and frontotemporal dementia in clinical practice, based on international consensus documents (currently containing partly outdated information) and published scientific evidence on this topic. Alzheimerdisease may be caused by mutations in PSEN1, PSEN2 and APP. APOE genotyping is not recommended for the diagnostic or genetic counseling purposes in Alzheimer disease. Frontotemporal dementia may be caused by mutations in several genes such as c9orf72, PGRN, MAPT, TBK1, VCP, SQSTM1, and UBQLN2. This paper pragmatically approaches the process of genetic diagnosis in Alzheimer disease and frontotemporal dementia, with specific recommendations for both disorders.

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37

Nazarko, Linda. "Dementia 6. Frontotemporal dementia: diagnosis, treatment and medical management." British Journal of Healthcare Assistants 14, no. 1 (January 2, 2020): 36–43. http://dx.doi.org/10.12968/bjha.2020.14.1.36.

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Frontotemporal dementia is a rare form of dementia and around 2% of people with dementia have frontotemporal dementia ( Warren et al, 2013 ). This article, the sixth in a series, explains about the pathophysiology and clinical features of frontotemporal dementia and how it is diagnosed, treated and managed.

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38

Schoder, Delphine, Didier Hannequin, Olivier Martinaud, Gaëlle Opolczynski, Lucie Guyant-Maréchal, Isabelle Le Ber, and Dominique Campion. "Morbid risk for schizophrenia in first-degree relatives of people with frontotemporal dementia." British Journal of Psychiatry 197, no. 1 (July 2010): 28–35. http://dx.doi.org/10.1192/bjp.bp.109.068981.

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BackgroundFamilial co-occurrence of frontotemporal dementia and schizophrenia has never been investigated.AimsTo test the hypothesis that frontotemporal dementia and schizophrenia might have a common aetiology in some families in which both syndromes coexist (mixed families).MethodThe morbid risk for schizophrenia, calculated in first-degree relatives of 100 frontotemporal dementia probands, was compared with that calculated in first-degree relatives of 100 Alzheimer's disease probands. In mixed families, sequencing analysis of known frontotemporal dementia genes and detailed phenotype characterisation of individuals with frontotemporal dementia and schizophrenia were performed.ResultsThe morbid risk for schizophrenia was significantly higher in relatives of frontotemporal dementia probands (1.35, s.e. = 0.45) than in relatives of Alzheimer's disease probands (0.32, s.e. = 0.22). Ten mixed families were characterised. In three of them a frontotemporal dementia causal mutation was identified that was present in individuals with schizophrenia. Several specific clinical features were noted in people with schizophrenia and frontotemporal dementia in mixed families.ConclusionsCo-occurrence of schizophrenia and frontotemporal dementia could indicate, in some families, a common aetiology for both conditions.

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39

Miller, Bruce L. "Frontotemporal Dementia and Semantic Dementia." Alzheimer Disease & Associated Disorders 21, no. 4 (October 2007): S19—S22. http://dx.doi.org/10.1097/wad.0b013e31815c0f7a.

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40

Wilhelmsen, Kirk C. "Frontotemporal Dementia Genetics." Journal of Geriatric Psychiatry and Neurology 11, no. 2 (July 1998): 55–60. http://dx.doi.org/10.1177/089198879801100203.

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Frontotemporal dementia (FTD) is the most common syndrome in which the focus of neurodegeneration is the frontal lobes. FTD is frequently familial. It is also often due to a susceptibility locus on chromosome 17q21-22. Some 17q21-22-linked families have mutations in the tau gene and most have microscopically visible aggregates of hyperphosphorylated tau. Demonstrating that mutations in tau can produce neurodegeneration will necessitate a reassessment of the role of tau in the pathogenesis of the many diseases in which tau biology is disrupted.

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41

Borroni, Barbara, Marta A. Manes, Antonella Alberici, Maura Cosseddu, Pia Bernasconi, Silvana Archetti, Lorenzo Pinelli, Roberto Gasparotti, and Alessandro Padovani. "Autoimmune Frontotemporal Dementia." Alzheimer Disease & Associated Disorders 31, no. 3 (2017): 259–62. http://dx.doi.org/10.1097/wad.0000000000000180.

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42

Molano, J. "FRONTOTEMPORAL DEMENTIA SYNDROMES." Neurology 72, no. 19 (May 11, 2009): 1711. http://dx.doi.org/10.1212/wnl.0b013e3181a56016.

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43

Josephs, K. A. "Frontotemporal dementia syndromes." Journal of Neurology, Neurosurgery & Psychiatry 79, no. 5 (May 1, 2008): 614. http://dx.doi.org/10.1136/jnnp.2007.139188.

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44

Nazarko, Linda. "Frontotemporal dementia: diagnosis, treatment and medical management." British Journal of Neuroscience Nursing 16, no. 2 (April 2, 2020): 61–68. http://dx.doi.org/10.12968/bjnn.2020.16.2.61.

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Frontotemporal dementia is a rare form of dementia and around 2% of people with dementia have frontotemporal dementia ( Warren et al, 2013 ). This article explains the pathophysiology and clinical features of frontotemporal dementia and how it is diagnosed, treated and managed.

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45

Funayama, Michitaka, Asuka Nakajima, Shin Kurose, and Taketo Takata. "Putative Alcohol-Related Dementia as an Early Manifestation of Right Temporal Variant of Frontotemporal Dementia." Journal of Alzheimer's Disease 83, no. 2 (September 14, 2021): 531–37. http://dx.doi.org/10.3233/jad-210501.

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Diagnosis of frontotemporal dementia is challenging in the early stages. Various psychiatric and neurological diseases are misdiagnosed as frontotemporal dementia and vice versa. Here we present a case with right temporal variant of frontotemporal dementia who presented with alcohol dependency and remarkable behavioral symptoms and was first misdiagnosed as having alcohol-related dementia. He then revealed symptoms related to right temporal variant of frontotemporal dementia, such as prosopagnosia, difficulty recognizing his housemates, loss of empathy, ritualistic behaviors, and difficulty finding and comprehending words. Retrospectively, his alcohol dependency itself was considered an early manifestation of right temporal variant of frontotemporal dementia.

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46

Orsini, Marco, Marcos RG de Freitas, Paulo Cezar Vieira, Carlos Henrique Melo Reis, Gabriela Vieira, and Antônio Marcos da Silva Catharino. "Glioblastoma multiforme mimicking frontotemporal dementia: A case report." International Journal of Case Reports and Images 15, no. 1 (January 30, 2024): 12–16. http://dx.doi.org/10.5348/101436z01mo2024cr.

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Glioblastoma (GBM) is the most common and aggressive primary brain tumor in adults. In this case a 57-year-old male patient had cognitive decline, visual dysfunction, and memory loss. He is not able to interact during the medical consultation. The brain Magnetic Resonance Imaging (MRI) showed an infiltrative and expansive lesion compressing the third ventricle and extending to midbrain to the left. The anatomopathological report concluded the diagnosis of Grade IV glioblastoma and the patient will undergo to surgical intervention.

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47

Fedorova, Ya B. "Evolution of Concepts of Differential Diagnostics in Frontotemporal Dementia." Psikhiatriya 21, no. 7 (January 31, 2024): 76–96. http://dx.doi.org/10.30629/2618-6667-2023-21-7-76-96.

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Background: frontotemporal dementia (FTD) is a group of neurodegenerative diseases, with onset usually in presenile age, the clinical picture is manifested by behavioral disorders and relatively intact cognitive features in the initial disease. In the early stages of FTD, it is difficult to differentiate this type from other dementias or other mental diseases. The aim was to analyse recent scientific publications on the problem of differential diagnostics of frontotemporal dementia. Material and methods: using the keywords “frontotemporal dementia”, “frontotemporal lobar degeneration”, “differential diagnosis of frontotemporal dementia”, “behavioral variant of frontotemporal dementia”, selected and analyze publications for the last two decades. Results: the behavioral variant of FTD (bv-FTD) is the most common form of FTD, accounting for 50% of all cases of FTD, and especially in cases with early onset. Predominantly, this variant of FTD presents diagnostic difficulties, due to the limited accuracy of neuroimaging examinations and the lack of specific biomarkers. The clinical symptoms of bv-FTD are characterized by considerable overlap with symptoms of neurodegenerative diseases and mental diseases, such as schizophrenia, bipolar affective disorder, obsessive-compulsive disorder, and personality disorders. Conclusion: the diagnosis of FTD at the initial stage of the disease is problematic and difficult, the sensitivity and specificity of almost all diagnostic methods increase as the disease progresses. This literature review highlights some of the diagnostic methods that can be used in suspected cases of FTD and informs about the differential diagnostics recommendations that have been developed to improve the accuracy of FTD diagnosis.

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48

Martins, Leonardo Tafarello, Ivan Abdalla Teixeira, Jerson Laks, and Valeska Marinho. "Recognizing Late Onset Frontotemporal Dementia with the DAPHNE scale: A case report." Dementia & Neuropsychologia 12, no. 1 (March 2018): 75–79. http://dx.doi.org/10.1590/1980-57642018dn12-010011.

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ABSTRACT Frontotemporal dementias are classically described as early onset dementias with personality and behavioral changes, however, late onset forms can also be found. Considering the paucity of information about late onset behavioral variant frontotemporal dementia and its challenging diagnosis, we present a case report of an 85-year-old woman with behavioral changes and slow progression to dementia who was first diagnosed as having bipolar disorder and then Alzheimer's disease. The Daphne scale provided a structured means to improve clinical diagnosis, also supported by characteristic features on MRI and SPECT, while CSF biomarkers ruled out atypical Alzheimer's disease.

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49

Velakoulis, D., M. Walterfang, R. Mocellin, C. Pantelis, and C. McLean. "Frontotemporal dementia presenting as schizophrenia-like psychosis in young people: clinicopathological series and review of cases." British Journal of Psychiatry 194, no. 4 (April 2009): 298–305. http://dx.doi.org/10.1192/bjp.bp.108.057034.

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BackgroundFew studies have investigated the relationship between schizophrenia and frontotemporal dementia.AimsTo investigate this relationship through a clinicopathological investigation of young-onset frontotemporal dementia and a review of the case literature.MethodCases of young-onset frontotemporal dementia were identified within the local brain bank. The clinical course and pathological findings were collated. For the literature review, cases of frontotemporal dementia identified through Medline were selected according to defined criteria. The demographic, clinical, pathological and genetic characteristics of cases presenting with a psychotic illness were identified.ResultsIn the case series, 5 of 17 patients with frontotemporal dementia had presented with a psychotic illness (schizophrenia/schizoaffective disordern=4, bipolar disordern=1) an average of 5 years prior to the dementia diagnosis. Patients with schizophrenia exhibited changes consistent with TDP-43 and ubiquitin-positive frontotemporal dementia. In the cases review, a third of patients aged 30 years or under and a quarter of those aged 40 years or under had been diagnosed with psychosis at presentation.ConclusionsPatients with young-onset frontotemporal dementia may be diagnosed with a psychotic illness years before the dementia diagnosis is made. These findings have implications for clinicians and for our further understanding of the neurobiology of psychotic illness.

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50

O’brien, John T. "Commentary on: psychotic symptoms in frontotemporal dementia: a diagnostic dilemma?" International Psychogeriatrics 27, no. 4 (March 2, 2015): 529–30. http://dx.doi.org/10.1017/s1041610214002920.

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Frontotemporal dementia is clearly a very important condition, not only because alongside Alzheimer's disease, vascular dementia, and Lewy body dementia it is one of the four most common causes of dementia, but because its broad symptom profile and younger age of onset makes it a particularly challenging condition to diagnose and manage. In recent years, great progress has been made in understanding genetic and pathological underpinnings of frontotemporal dementias, and in characterizing specific pathological causes. In particular, three major subtypes can be delineated: those associated pathologically with tau; transactive responsive DNA binding protein (TDP-43); or fused in sarcoma (FUS). Of these, tau and TDP-43 are by far the two most common subtypes of frontotemporal dementia. In addition, there have been important genetic advances and several autosomal dominant mutations have been described, for example associated with MAPT, progranulin, and C9ORF72. However, despite these important advances regarding pathophysiological heterogeneity, relatively little is known about the frequency, nature, and correlates of psychotic symptoms in the disorder. It is therefore very timely that in this month's Paper of the Month, Waldo and colleagues (Waldo et al., 2015) describe a very detailed description of a cohort of 97 consecutive subjects seen in Lund who received a neuropathological diagnosis of frontotemporal lobar degeneration.

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