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Books on the topic 'Frontotemporal dementia'

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Author: Grafiati
Published: 4 June 2021
Last updated: 7 July 2024

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1

F, Lebert, and Pasquier F, eds. Frontotemporal dementia. Dordrecht: ICG Publications, 1996.

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2

R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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3

R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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4

Dickerson, Bradford C., ed. Hodges' Frontotemporal Dementia. Cambridge: Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781316091586.

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5

Hodges, John R., ed. Frontotemporal Dementia Syndromes. Cambridge: Cambridge University Press, 2007. http://dx.doi.org/10.1017/cbo9781316135457.

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6

Roberson, Erik D., ed. Alzheimer's Disease and Frontotemporal Dementia. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-744-0.

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7

Alzheimer's disease and frontotemporal dementia: Methods and protocols. New York: Humana Press, 2011.

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8

Frontotemporal disorders: Information for patients, families, and caregivers. Bethesda, Md.]: National Institutes of Health, National Institute on Aging, 2010.

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9

Language, interaction and frontotemporal dementia: Reverse engineering the social mind. London: Equinox Pub., 2010.

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10

W, Mates Andrea, Mikesell Lisa, and Smith Michael Sean, eds. Language, interaction and frontotemporal dementia: Reverse engineering the social mind. Oakville, CT: Equinox Pub. Ltd., 2010.

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11

Ghetti, Bernardino, Emanuele Buratti, Bradley Boeve, and Rosa Rademakers, eds. Frontotemporal Dementias. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-51140-1.

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12

Griffiths, Helen L. Communication disorder in dementia due to frontotemporal cerebral atrophy: A linguistic and cognitive analysis : a comparative study with Alzheimer's disease. Manchester: University of Manchester, 1996.

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13

Deramecourt, Vincent, Florence Lebert, and Florence Pasquier. Frontotemporal dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0036.

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Frontotemporal dementia (FTD) is the second most common form of dementia in persons younger than 65 years after Alzheimer’s disease. The FTD spectrum is characterized by clinical, molecular and genetic heterogeneity. Core features of FTD are behavioural and language manifestations and the clinical spectrum of FTD currently includes a behavioural variant, progressive nonfluent aphasia and semantic dementia. The most common behavioural features are disinhibition, apathy, loss of empathy, hyperorality and perseveration. Neuroimaging usually demonstrates focal atrophy and hypometabolism in the anterior part of the frontal and temporal lobes. A careful history and neuropsychological examination, and judicious use of neuroimaging, can help distinguish FTD from other common forms of dementia, especially Alzheimer’s disease, vascular dementia, and dementia with Lewy bodies. Although no specific pharmacological treatments for FTD exists, symptom management with serotonin reuptake inhibitors and non pharmacological interventions have been shown to be beneficial.
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14

Miller, Bruce L. Frontotemporal Dementia. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780195380491.001.0001.

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Frontotemporal Dementia provides an in-depth look at the history, various types, genetics, neuropathology and psychosocial aspects of one of the most common but least understood causes of dementia, frontotemporal lobar degeneration, from one of the world's leading centers for the study of dementia. Aided by the latest research in diagnosis, mechanism and treatment, this resource captures the rich and quickly changing landscape of a devastating neurodegenerative disease, and offers up-to-date clinical advice for patient care. Frontotemporal dementia, in particular, raises psychological and philosophical questions about the nature of self, free will, emotion, art and behavior - important topics for practitioners and families to appreciate as they care for the sufferer. It includes case studies, photographs and figures from the leaders in the field and personal communication from the researchers driving these developments.
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15

Frontotemporal Dementia. Oxford University Press, Incorporated, 2014.

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16

Evans, Lauren, Ellen Steinbart, NetCE, and CE Resource. Frontotemporal Dementia. CE Resource, Incorporated, 2021.

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17

Frontotemporal Dementia. Oxford University Press, Incorporated, 2014.

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18

Hodges, John R. Frontotemporal Dementia Syndromes. University of Cambridge ESOL Examinations, 2016.

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19

Hodges, John R. Frontotemporal Dementia Syndromes. Cambridge University Press, 2011.

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20

Frontotemporal Dementia Syndromes. Cambridge University Press, 2007.

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21

Dickerson, Bradford C. Hodges' Frontotemporal Dementia. Cambridge University Press, 2015.

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22

Hodges' Frontotemporal Dementia. Cambridge University Press, 2016.

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23

Dickerson, Bradford C. Hodges' Frontotemporal Dementia. Cambridge University Press, 2016.

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24

Brookes, Alpha. Dementia Signs : Frontotemporal Dementia: Dementia Meaning. Independently Published, 2021.

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25

Briggs, John, Jerry Beller, Beller Health, and Brain Research. Frontotemporal Related Dementias: Behavioral Variant Frontotemporal Dementia and Progressive Supranuclear Palsy. Independently Published, 2020.

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26

Beller, Jerry, and Beller Health. BvFTD Behavioral Variant Dementia: Frontotemporal Dementia. Independently Published, 2019.

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27

Beller, Jerry, and Beller Health. 14 Dementia Types: Alzheimers Lewy Body Dementia Frontotemporal Dementia Vascular Dementia Huntington's Disease Other Dementias. Independently Published, 2019.

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28

Beller, Jerry, and Beller Health. 2019 Dementia Overview: Alzheimers , Lewy Body Dementia , Frontotemporal Dementia , Vascular Dementia , Huntington's Disease , Other Dementias. Independently Published, 2019.

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29

Kaplan, Tamara, and Tracey Milligan. Dementia 1: Defining Dementia, Alzheimer’s Disease and Frontotemporal Dementia (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0009.

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The video in this chapter explores dementia, and focuses on definitions of dementia, Alzheimer’s disease and Frontotemporal Dementia. Dementia is defined as a cognitive decline in one or more cognitive domains including memory, language, attention, visuospatial processing and social behavior. Two hallmark pathologic features of Alzheimer’s disease (AD) are plaques, which are formed from amyloid and neurofibrillary tangles, which involve tau, whereas symptoms of Frontotemporal Dementia (FTD) may include behavioral changes, apathy and disinhibition and ritualistic or repetitive behaviors. Language may also be affected, and this can be a presenting symptom.
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30

Beller, Jerry, and Beller Health. 2019 Primary Progressive Aphasia: Frontotemporal Dementia. Independently Published, 2019.

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31

Beller, Jerry, and Beller Health. FTD Dementia : Frontotemporal Dementia: Behavioral Variant Primary Progressive Aphasia. Independently Published, 2019.

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32

Roberson, Erik D. Alzheimer's Disease and Frontotemporal Dementia: Methods and Protocols. Humana Press, 2016.

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33

Graff-Radford, Jonathan, and Keith A. Josephs. Frontotemporal Lobar Degeneration. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0018.

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Frontotemporal dementia (FTD) is an uncommon but important form of degenerative disease characterized by clinical syndromes that result from degeneration of the frontal and temporal lobes. FTD is divided based on clinical presentation into behavioral variant FTD (bvFTD), semantic dementia, and progressive nonfluent/agrammatic aphasia. Several recent studies have advanced our knowledge of the genetics of FTD, with the three most common FTD genes being microtubule-associated protein tau (MAPT) and progranulin (GRN), and a noncoding repeat expansion in C9ORF72. Tau and TDP-43 are the most common pathologies associated with FTD. No pharmacological therapies are currently approved for use in FTD.
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34

When Love Meets Dementia: Frontotemporal Degeneration and the Family. McFarland & Company, Incorporated Publishers, 2019.

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35

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Dementia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0011.

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This chapter discusses the clinical features and evidence-based pharmacological management of dementia disorders (Alzheimer’s disease (AD), vascular dementia, dementia with Lewy bodies (DLB), Parkinson’s disease dementia (PDD), frontotemporal dementia (FTD), mixed dementia, and mild cognitive impairment (MCI)).
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36

Houlden, Henry, Alan Edward Renton, and Francesca Luisa Conforti, eds. Multifaceted Genes in Amyotrophic Lateral Sclerosis-Frontotemporal Dementia. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-904-2.

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37

Piggott, Margaret Ann. Neurochemical pathology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0007.

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This chapter considers the neurodegenerative disorders Alzheimer’s disease (AD), Lewy body dementias (dementia with Lewy bodies (DLB) and Parkinson’s disease dementia(PDD)), frontotemporal dementia (FTD); and also vascular dementia (VaD) which results from cerebrovascular disease. These different conditions, which give rise to dementia syndromes, each have distinct neurochemical pathologies, with important implications for treatment. As increased age is the common risk factor generally associated with dementing illnesses, neurochemical changes are set in the context of the changes which occur during ageing. A detailed understanding of the neurotransmitter function in each condition can lead to rational drug design and treatment strategies appropriate for each group of patients. Neurochemical pathology in transmitter systems underlying clinical features of these disorders are reviewed.
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38

Gan, Li. Cellular Mechanisms of Dementia. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0054.

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Neurodegenerative dementias, including Alzheimer’s disease (AD), Parkinson’s disease (PD) and Frontotemporal dementia (FTD), pose enormous challenges for our aging society. Genetic and mechanistic studies have revealed common molecular and cellular pathways, including imbalanced proteostasis and aberrant innate immune responses. Key pathogens in AD, PD, and FTD accumulate and spread from one brain region to another, resulting in network dysfunction and cognitive decline. These diseases are multifactorial, caused by interactions among multiple genetic, epigenetic, and environmental factors and pathways. Combination therapies that target multiple pathways may also be needed to stop or delay the dementing conditions in neurodegenerative dementias.
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39

Amyotrophic Lateral Sclerosis And The Frontotemporal Dementias. Oxford University Press, USA, 2012.

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40

Strong, Michael J. Amyotrophic Lateral Sclerosis and the Frontotemporal Dementias. Oxford University Press, 2012.

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41

Zahn, Roland, and Alistair Burns. Dementia disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198779803.003.0001.

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This chapter provides a brief overview of the different forms of dementia syndromes and provides a simple algorithm for initial differential diagnosis. Rapidly progressive dementias have to be excluded which require specific investigations to detect Creutzfeldt–Jakob as well as inflammatory and autoimmune diseases. A lead symptom-based approach in patients with slowly progressive cognitive and behavioural impairments without neurological symptoms is applied: progressive and primary impairments in recent memory are characteristic of typical Alzheimer’s dementia, primary behavioural changes point to the behavioural variant of frontotemporal dementia, primary impairments of language or speech are distinctive for progressive aphasias, fluctuating impairments of attention are a hallmark of Lewy body dementia, whereas primary visuospatial impairments suggest a posterior cortical atrophy. The chapter further discusses updated vascular dementia guidelines and DSM-5 revisions of defining dementia. Current diagnostic criteria for the different dementias are referenced and the role of neuroimaging is illustrated.
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42

Jr, Moncivais Manuel Manny. Just Squeeze My Hand: A Caregiver's Experience with Frontotemporal Dementia. Christian Faith Publishing, 2021.

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43

Language, Interaction, and Frontotemporal Dementia: Reverse Engineering the Social Mind. Equinox Publishing (Indonesia), 2013.

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44

Jr, Moncivais Manuel Manny. Just Squeeze My Hand: A Caregiver's Experience with Frontotemporal Dementia. Christian Faith Publishing, 2021.

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45

Utianski, Rene L. Primary Progressive Aphasia and Other Frontotemporal Dementias: Diagnosis and Treatment of Associated Communication Disorders. Plural Publishing, Incorporated, 2019.

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46

Burrell, James R., and John R. Hodges. Dementia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0010.

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Cognitive neurology has exploded over the last century, and especially over the last 20 years. From the distinction of dementia as a pathological entity, rather than just ‘normal’ ageing, to more sophisticated sub-classification of dementia syndromes, much has been learned, though great challenges remain. From an incredible array of worthy research studies, ten landmark papers in the field of dementia are presented in this chapter. With regard to Alzheimer’s disease, the following are discussed: the initial description of the disease, both clinically and pathologically; the development of meaningful clinical assessment measures; the early clinical manifestations and genetic causes; the precursors to symptomatic treatment; the use of neuroimaging to identify amyloid pathology in vivo; and the staging of Alzheimer’s pathology. The clinical features and genetic causes of frontotemporal dementia, an important non-Alzheimer’s primary dementia syndrome seen especially in younger patients, are also discussed.
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47

Hodges, John R. Delirium and Dementia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198749189.003.0002.

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Delirium and dementia affect one or more cognitive domains with a distributed neural basis—attention, memory, and executive function—in some instances accompanied by more focal cognitive deficits. Patients with one, or both, of these conditions constitute the commonest presentation in behavioural neurology and in geriatric psychiatry. This chapter first describes the core characteristics and causes of delirium. This is followed by a description of the major causes of dementia notably, Alzheimer’s disease, frontotemporal dementia, Huntington’s disease, dementia with Lewy bodies, and progressive supranuclear palsy. These are contrasted with pseudodementia, with mention of the causes of rapidly progressive dementia, and differential diagnosis of delirium and dementia.
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48

Frontotemporal Dementias: 4th International Conference, Lund, Sweden, April 24-26 2003 (Special Issue Dementia and Geriatric Cognitive Disorders 2004). Not Avail, 2004.

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49

Briggs, John, Jerry Beller, Beller Health, and Brain Research. BvFTD Behavioral Variant Frontotemporal Dementia: Guide for Doctors, Nurses, Patients, Families, and Caregivers. Independently Published, 2020.

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50

Beller, Jerry, and Beller Health. Dementia Types, Risk Factors, and Symptoms: Alzheimer's Disease Vascular Lewy Body Frontotemporal Huntington's Normal Pressure Hydrocephalus Wernicke Korsakoff Dementias. Independently Published, 2019.

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