Relevant bibliographies by topics / Frontotemporal dementia

Academic literature on the topic 'Frontotemporal dementia'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 July 2024

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Journal articles on the topic "Frontotemporal dementia"

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Snowden, Julie S., David Neary, and David M. A. Mann. "Frontotemporal dementia." British Journal of Psychiatry 180, no. 2 (February 2002): 140–43. http://dx.doi.org/10.1192/bjp.180.2.140.

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BackgroundFrontotemporal dementia accounts for up to 20% of cases of dementia in the presenium, yet remains poorly recognised. Diagnostic criteria have been devised to aid clinical diagnosis.AimsTo provide an overview of clinical and pathological characteristics of frontotemporal dementia and its nosological status.MethodsThe review summarises consensus diagnostic criteria for frontotemporal dementia and draws on the authors'clinical experience of 300 frontotemporal dementia cases, and pathological experience of 50 autopsied cases.ResultsFrontotemporal dementia is characterised by pronounced changes in affect and personal and social conduct. Some patients also develop motor neuron disease. Mutations in the tau gene account for some but not all familial cases of frontotemporal dementia.ConclusionsFrontotemporal dementia is a focal form of dementia, which is clinically and pathologically distinct from other dementias. It represents an important model for understanding the functions of the frontotemporal lobes.
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Dacka, Michał, Mikołaj Porzak, Karol Bochyński, Konrad Białogłowski, Paulina Dąbrowska, Michał Żuber, Katarzyna Ciuba, Katarzyna Molenda, Filip Borodziuk, and Barbara Borodziuk. "Frontotemporal Dementia: A Clinical Review." Journal of Education, Health and Sport 59 (February 14, 2024): 235–46. http://dx.doi.org/10.12775/jehs.2024.59.015.

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Frontotemporal dementia is a disease in which atrophic changes occur in the frontal lobes and frontal temporal lobes of the brain. Frontotemporal dementias are a clinically, neuroanatomically and pathologically diverse group of diseases that collectively constitute an important cause of young-onset dementia. The most common form of frontotemporal dementia is the so-called behavioral variant of frontotemporal dementia. Underlying these pathological changes is the degeneration of nerve cells (i.e. neurons), which occurs through the accumulation of abnormal proteins inside them. Therefore, the review of current studies in the subject of Frontotemporal dementia was conducted in order to access possible risk factors and new ways of management and treatment of this complex disease.
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Rusina, Robert, Radoslav Mmatěj, Zsolt Cséfalvay, Jiří Keller, Vanda Franková, and Martin Vyhnálek. "Frontotemporal dementia." Česká a slovenská neurologie a neurochirurgie 84/117, no. 1 (February 28, 2021): 9–29. http://dx.doi.org/10.48095/cccsnn20219.

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Swartz, J. Randolph, Bruce L. Miller, Ira M. Lesser, and Amy L. Darby. "Frontotemporal Dementia." Journal of Clinical Psychiatry 58, no. 5 (May 15, 1997): 212–17. http://dx.doi.org/10.4088/jcp.v58n0506.

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Fedorova, Yana B. "Frontotemporal dementia." Psychiatry 77 (2018): 60–85. http://dx.doi.org/10.30629/2618-6667-2018-77-60-85.

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Marshall, Katherine, and Deborah Hale. "Frontotemporal Dementia." Home Healthcare Now 40, no. 4 (July 2022): 223. http://dx.doi.org/10.1097/nhh.0000000000001092.

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Rosness, Tor Atle, Knut Engedal, and Zeina Chemali. "Frontotemporal Dementia." Journal of Geriatric Psychiatry and Neurology 29, no. 5 (August 8, 2016): 271–80. http://dx.doi.org/10.1177/0891988716654986.

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Graff-Radford, Neill, and Bryan Woodruff. "Frontotemporal Dementia." Seminars in Neurology 27, no. 1 (February 2007): 048–57. http://dx.doi.org/10.1055/s-2006-956755.

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Warren, J. D., J. D. Rohrer, and M. N. Rossor. "Frontotemporal dementia." BMJ 347, aug12 3 (August 6, 2013): f4827. http://dx.doi.org/10.1136/bmj.f4827.

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Miller, Bruce, and David Perry. "Frontotemporal Dementia." Seminars in Neurology 33, no. 04 (November 14, 2013): 336–41. http://dx.doi.org/10.1055/s-0033-1359316.

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Dissertations / Theses on the topic "Frontotemporal dementia"

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Sampson, Elizabeth Lesley. "Longitudinal studies in frontotemporal dementia." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406422.

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Rascovsky, Katya. "Neuropsychological aspects of frontotemporal dementia /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3167837.

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Graham, A. J. "Learning and memory in frontotemporal dementia." Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.599590.

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Yancopoulou, Despina. "Neuropathological and genetic studies on frontotemporal dementia." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.614974.

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Orr, Miranda Ethel. "Mouse and stem cell models of frontotemporal dementia." Diss., Montana State University, 2012. http://etd.lib.montana.edu/etd/2012/orr/OrrM0512.pdf.

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Alzheimer's disease (AD) is the most prevalent brain disease in the United States, and an escalating health concern. AD patient brains acquire hallmark protein aggregates, referred to as senile A beta plaques and neurofibrillary tangles (NFTs), that coincide with brain cell loss and dementia. A subset of AD patients carry mutant genes. Our understanding of AD largely relies on model systems that express these gene variants. Mice engineered to express AD-mutant human genes develop A beta plaques, but fail to develop the tau-containing NFTs or cell loss. Mutant tau variants are required to induce NFTs and neuronal loss in mice, but AD patients carry normal tau genes. The inability of mouse tau to become a pathogenic protein in the presence of AD-mutant gene variants, and the general insufficiency of the current systems to recapitulate AD, inspired the research described here. To determine if species differences between mouse and human tau inhibit the progression of AD in mice, I utilized a well-characterized mouse model of a related disease, frontotemporal dementia (FTD). FTD mice carry mutant human tau and develop NFTs and cell loss. I ablated mouse tau in FTD mice and looked for signs of more severe pathology. I compared the FTD mice, with and without mouse tau, to FTD mice with and without wild type human tau to investigate potential tau species-specific differences. My studies indicated that wild type tau, mouse or human, dampened the pathological effects of FTD tau implying a general, not mouse-specific, effect of normal tau protein. Our data suggest that unknown factors, distinct from endogenous mouse tau, contribute to the inability of mice to model AD. The recent interest in patient-specific stem cell (SC) models to study disease necessitates a thorough evaluation of their ability to recapitulate key characteristics of disease, reproducibility, and longevity. I generated and characterized brain SC cultures from FTD fetal mice and compared them to those generated from mice with normal human tau. Significant genotype associated differences were discovered in the SC system and later verified in adult mice to reinforce the potential of patient-specific SC models to study disease.
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Rizzu, Patrizia. "Mutations in frontotemporal dementia linking tau to neurodegeneration." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2002. http://hdl.handle.net/1765/12093.

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Urwin, H. N. "Cellular models of CHMP2B mutations in frontotemporal dementia." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19564/.

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Mutations in the charged multivesicular body protein 2B gene (CHMP2B) cause frontotemporal dementia termed frontotemporal dementia linked to chromosome 3 (FTD-3) in a large Danish pedigree and also in an unrelated Belgian familial FTD patient. Genetic analyses on the Danish pedigree and the role of CHMP2B mutations in frontotemporal dementia are reported. The clinicopathological spectrum of FTD-3 and other FTD subtypes is also described. CHMP2B is a component of the endosomal sorting complex required for transport (ESCRT-III), which is required for formation and function of the multivesicular body (MVB), an endosomal structure that fuses with the lysosome to degrade endocytosed proteins. Cellular models of CHMP2B mutations showed an enlarged late endosomal phenotype and an abnormal pattern of ubiquitination. Functional studies demonstrated a specific disruption of endosome-lysosome fusion but not sorting of endocytosed receptors by the MVB. Investigations into the mechanism of impaired fusion suggested impaired recruitment of the GTPase Rab7, known to be necessary for vesicular fusion, onto endosomes in CHMP2B mutant cells. Studies of patient tissue revealed a novel endosomal pathology in CHMP2B mutation-positive patient brains and also abnormal endosomes in patient fibroblasts. These data indicate that defects in endosomal fusion events can lead to neurodegeneration and suggest a potential pathogenic mechanism for CHMP2B mutations.
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Suhonen, N. M. (Noora Maria). "Cognitive and behavioral characteristics of frontotemporal lobar degeneration." Doctoral thesis, Oulun yliopisto, 2017. http://urn.fi/urn:isbn:9789526216102.

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Abstract Frontotemporal lobar degeneration (FTLD) is the second commonest cause of dementia after Alzheimer’s disease (AD) in patients <65 years. Its most frequent clinical subtype is behavioral variant frontotemporal dementia (bvFTD) characterized by behavioral change and executive deficits. FTLD also encompasses two variants of primary progressive aphasia (PPA) characterized by language deficits. The majority of familial FTLD cases are linked to the C9ORF72 expansion mutation. As both cognitive and behavioral changes are core diagnostic features of FTLD, neuropsychological assessment is vital. However, neuropsychological literature is inconclusive regarding the most functional measures for detecting FTLD. Current knowledge on the cognitive profile of patients with the C9ORF72 expansion is scarce. The aims of this thesis were threefold: (1) to identify the cognitive measures that optimally serve the differential diagnosis of FTLD, (2) to characterize the neuropsychological profile of C9ORF72 expansion; and (3) to examine the utility of the Modified Frontal Behavioral Inventory (FBI-mod) in differentiating FTLD, AD, and mild cognitive impairment (MCI). The participants comprised FTLD, AD, and MCI patients diagnosed in the University Hospitals of Oulu and Kuopio. The patients underwent a detailed neuropsychological assessment including the CERAD neuropsychological battery (CERAD-NB) and the FBI-mod. While bvFTD was characterized by verbal fluency, working memory, and verbal comprehension deficits relative to AD, AD was associated with greater episodic memory impairments. The poorer delayed recall in AD was further evident on the memory tests of the CERAD-NB; however, its overall utility in the differentiation between FTLD and AD was limited. The C9ORF72 expansion carriers showed more severe executive deficits than non-carriers. The C9ORF72 expansion may further be associated with slowly progressing FTLD. On the FBI-mod, bvFTD was linked to amplified behavioral symptoms relative to AD, MCI, and PPA. Findings highlight the importance of incorporating a broad cognitive battery in the neuropsychological evaluation of FTLD. Though the clinical phenotype of C9ORF72 expansion appears broad, executive impairment likely is a core feature of bvFTD patients with the expansion. The use of the FBI-mod is recommended as a structured measure for behavioral symptoms of bvFTD
Tiivistelmä Otsa-ohimolohkorappeumat on Alzheimerin taudin (AT) jälkeen yleisin työikäisten dementiaa aiheuttava sairausryhmä. Sen yleisin alamuoto on otsalohkodementia, jonka ensioireita ovat käyttäytymisen muutokset ja toiminnanohjauksen ongelmat. Sairausryhmään kuuluu myös kaksi kielellisin oirein ilmenevää alatyyppiä. C9ORF72-toistojaksomutaation on todettu selittävän suurimman osan perinnöllisistä tapauksista. Kognitiivisten ja käyttäytymiseen liittyvien muutosten arvioiminen on keskeinen osa taudin diagnostiikkaa. Tutkimustiedon perusteella on epäselvää, mitkä neuropsykologiset menetelmät soveltuvat parhaiten otsa-ohimolohkorappeumien tunnistamiseen. Tieto C9ORF72-mutaation kantajien kognitiivisesta profiilista on niukkaa. Tutkimuksen tavoitteena oli löytää neuropsykologisia menetelmiä, joista on hyötyä otsa-ohimolohkorappeumien erotusdiagnostiikassa ja selvittää C9ORF72-mutaation kantajien neuropsykologisia erityispiirteitä. Lisäksi haluttiin tutkia käytösoireita kartoittavan FBI-mod -läheiskyselyn hyödyllisyyttä otsa-ohimolohkorappeumien, AT:n ja lievän kognitiivisen heikentymän (MCI) erottamisessa. Aineisto koostui Oulun ja Kuopion yliopistosairaaloissa diagnosoiduista otsa-ohimolohkorappeuma-, AT- ja MCI-potilaista, joille oli tehty CERAD-tehtäväsarja, laaja neuropsykologinen tutkimus sekä FBI-mod. Otsalohkodementiaa sairastavat suoriutuivat AT-potilaita heikommin sanasujuvuutta, työmuistia ja kielellistä käsityskykyä arvioivissa tehtävissä, kun taas tapahtumamuisti oli heikompi AT:a sairastavilla. Myös CERAD-tehtäväsarjassa AT-potilaat suoriutuivat heikommin viivästetyn mieleenpalautuksen tehtävissä, mutta kokonaisuutena tehtäväsarjan kyky erotella otsa-ohimolohkorappeumaa ja AT:a sairastavat oli rajallinen. C9ORF72-mutaation kantajilla toiminnanohjauksen ongelmat olivat vaikeampia kuin ei-kantajilla. Lisäksi havaittiin, että C9ORF72-mutaatioon liittyvä sairaus voi edetä hyvin hitaasti. FBI-mod erotteli hyvin otsalohkodementiaa sairastavat AT- ja MCI-potilaista sekä otsa-ohimolohkorappeumien kielellistä muotoa sairastavista. Tulokset korostavat laajan neuropsykologisen tutkimuksen merkitystä otsa-ohimolohkorappeumien diagnostiikassa. Vaikka C9ORF72-mutaation kliininen kuva on vaihteleva, ovat toiminnanohjauksen ongelmat keskeinen osa taudinkuvaa. FBI-mod -kyselyn käyttö on suositeltavaa otsalohkodementiaan liittyvien käytösoireiden strukturoidussa arvioinnissa
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Haussmann, Robert, Marek Wysocki, Moritz D. Brandt, Andreas Hermann, and Markus Donix. "MAPT mutation associated with frontotemporal dementia and parkinsonism (FTDP-17)." Cambridge University Press, 2017. https://tud.qucosa.de/id/qucosa%3A70705.

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We present a 56-year-old patient suffering from frontotemporal dementia with parkinsonism linked to chromosome 17 (FTDP-17). The history included a three-generation pedigree and the patient was found to be a mutation carrier. The diagnosis was hindered by late appearance of the hypokinetic movement disorder. For clinicians, it is important to consider rare neurodegenerative disease variants in early-onset familial dementia syndromes with behavioral, cognitive, and motor symptoms
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Bradley, Paul. "The family experience of frontotemporal dementia : a qualitative study." Thesis, University of Birmingham, 2009. http://etheses.bham.ac.uk//id/eprint/403/.

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Overview This thesis is submitted in partial fulfilment for the requirements of the degree of Doctor of Clinical Psychology at the School of Psychology, University of Birmingham. It comprises of a research and a clinical volume. Volume I Volume I is the research component of the thesis. It consists of two papers, the first of which is a review of the literature that uses ‘Theory of Mind’ (ToM) tasks with people with frontal-variant frontotemporal dementia (fvFTD). All the research identified is systematically appraised in terms of the methodology and the quality of the published reports. The evidence indicates that ToM is impaired in people with fvFTD; however more robust findings were evident from research which comprehensively measured neuropsychological functioning and used established and well known ToM tests. More recent research has diversified into exploring other aspects of social cognition, such as emotional processing and empathy, and their relationships with ToM. The nominated journal for this review paper is ‘Neuropsychologia’. The second paper is a qualitative research project that explores the experiences of family members of people living with fvFTD. The research questions were: How does the development of fvFTD in a working age person affect the family experience of living with that person, and how might mental health services respond to the needs of those family members? Individual in-depth interviews were carried out with six relatives (including partners, a sibling, and an adult child), and Interpretative Phenomenological Analysis (IPA) was used to analyse the data resulting in the emergence of four main themes. The findings demonstrate how family caregivers of people with fvFTD have to contend with specific behavioural challenges and personality changes associated with the condition. The study also indicates that knowledge about fvFTD is lacking in both carers and professionals alike, causing uncertainty and long periods waiting for a diagnosis, which adds to the burden of care for these people. Services need to be developed to cater for specific individual needs and awareness needs to be raised in all health care services. The nominated journal for this research paper is ‘Dementia: The International Journal of Social Research and Practice’. Volume II Volume II is the clinical component of the thesis, which consists of five clinical practice reports (CPRs) that describe and evaluate clinical work carried out during clinical placements throughout the training course. The first CPR ‘Psychological Models’ formulates the case of an 18-year-old woman with anxiety symptoms from a systemic and a cognitive perspective. The second CPR ‘Small Scale Service-Related Research Project’ is a qualitative evaluation of a drop-in service for young people leaving care. The third CPR ‘Single Case Experimental Design’ evaluates the intervention designed to support a woman with a moderate learning disability and behaviour that challenged services. The fourth CPR ‘Case Study’ details the neuropsychological assessment of an 81-year-old man with memory problems. The fifth CPR was presented orally and it describes the use of Cognitive Analytic Therapy with a woman presenting with anxiety following treatment for breast cancer. The abstract is included here only.
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Books on the topic "Frontotemporal dementia"

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F, Lebert, and Pasquier F, eds. Frontotemporal dementia. Dordrecht: ICG Publications, 1996.

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R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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R, Hodges John, ed. Frontotemporal dementia syndromes. Cambridge: Cambridge University Press, 2007.

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Dickerson, Bradford C., ed. Hodges' Frontotemporal Dementia. Cambridge: Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781316091586.

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Hodges, John R., ed. Frontotemporal Dementia Syndromes. Cambridge: Cambridge University Press, 2007. http://dx.doi.org/10.1017/cbo9781316135457.

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Roberson, Erik D., ed. Alzheimer's Disease and Frontotemporal Dementia. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-744-0.

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Alzheimer's disease and frontotemporal dementia: Methods and protocols. New York: Humana Press, 2011.

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Frontotemporal disorders: Information for patients, families, and caregivers. Bethesda, Md.]: National Institutes of Health, National Institute on Aging, 2010.

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Language, interaction and frontotemporal dementia: Reverse engineering the social mind. London: Equinox Pub., 2010.

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W, Mates Andrea, Mikesell Lisa, and Smith Michael Sean, eds. Language, interaction and frontotemporal dementia: Reverse engineering the social mind. Oakville, CT: Equinox Pub. Ltd., 2010.

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Book chapters on the topic "Frontotemporal dementia"

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Wider, Christian W., Barbara Jasinska-Myga, Takuya Konno, and Zbigniew K. Wszolek. "Frontotemporal Dementia." In Neurodegeneration, 115–25. Oxford, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118661895.ch12.

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Pasquier, Florence, Thibaud Lebouvier, and Florence Lebert. "Frontotemporal Dementia." In Neuropsychiatric Symptoms of Cognitive Impairment and Dementia, 279–302. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39138-0_13.

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Doherty, Colin P., Siobhan Hutchinson, Sharon Abrahams, and Robert F. Coen. "Frontotemporal Dementia." In Neurodegenerative Disorders, 115–42. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-011-3_6.

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Perry, David C., and Howard J. Rosen. "Frontotemporal dementia." In Non-Alzheimer's and Atypical Dementia, 49–63. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118797662.ch5.

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Léger, Gabriel C. "Frontotemporal Dementia." In Neuro-Geriatrics, 103–24. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56484-5_9.

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Boxer, Adam L. "Frontotemporal Dementia." In The Handbook of Alzheimer's Disease and Other Dementias, 145–78. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444344110.ch4.

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Rascovsky, Katya, and Diana L. Matallana. "Frontotemporal dementia." In International Neurology, 153–56. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118777329.ch43.

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Gustafson, L., and A. Brun. "Frontotemporal Dementia." In Dementias, 151–71. Milano: Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2149-5_7.

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Franceschi, Ana M. "Frontotemporal Dementia." In PET/MR Imaging, 327–28. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65106-4_132.

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Subasree, R., and Suvarna Alladi. "Frontotemporal Dementia." In Case-based Approach to Common Neurological Disorders, 257–64. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-8676-7_29.

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Conference papers on the topic "Frontotemporal dementia"

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MILLER, BRUCE L. "FRONTOTEMPORAL DEMENTIA (FTD): INSIGHTS INTO WISDOM AND ALTRUISM." In Proceedings of the 45th Session of the International Seminars on Nuclear War and Planetary Emergencies. WORLD SCIENTIFIC, 2013. http://dx.doi.org/10.1142/9789814531788_0035.

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Souza, Leonardo de, Maxime Bertoux, Luciano Mariano, Elisa Resende, Antônio Lúcio Teixeira, Francisco Cardoso, Sarah Camargos, Vítor Tumas, and Paulo Caramelli. "MENTALIZING IN FRONTOTEMPORAL DEMENTIA AND PROGRESSIVE SUPRANUCLEAR PALSY." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda015.

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Background: Mentalizing and emotion recognition are impaired in behavioral variant frontotemporal dementia (bvFTD). It is not clear whether these abilities are disturbed in progressive supranuclear palsy (PSP). Objective: To investigate social cognition (SC) between bvFTD and PSP. The neural basis of SC in PSP and bvFTD groups were also investigated by neuroimaging. Methods: Data from the notification sheet were collected and patients were classified according to current clinical and pathological criteria. Results: Groups did not differ on age, schooling and sex. Compared to controls, bvFTD and PSP patients had reduced scores in all tests of SC. bvFTD and PSP did not differ on measures of SC. PSP and bvFTD had cerebral atrophy in critical regions for SC. The cortical correlates of emotion recognition overlapped in bvFTD and PSP, correlating with frontal medial cortex, insula and limbic structures. PSP and bvFTD patients also displayed similar patterns of brain correlations (anterior temporal lobes) for social norms. The neural correlates of mentalizing were associated with frontal and temporal poles bilaterally, in both bvFTD and PSP. Conclusion:PSP patients exhibit impairment in mentalizing. PSP and bvFTD share clinical, cognitive and neuroimaging features.
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Dias, Jéssica Azevedo, Matheus de Serpa Vale, Raquel Penido Oliveira, and Thaís Helen Rezende Pio. "Alzheimer’s: what is the difference between or frontotemporal dementia." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.544.

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Introduction: In the study of neurodegenerative diseases, Alzheimer’s disease (AD) has been classically considered with a typical presentation of cognitive symptoms and neuroanatomical changes. However, there are clinical phenotypes of AD whose neurobiological bases are similar to frontotemporal dementia (FTD). In this sense, the heterogeneity of these pictures leads to inaccurate evaluation and diagnosis processes, due to the scant knowledge about their neurocognitive symptoms. The early stages of Alzheimer’s-type dementia are classically characterized by memory impairment, whereas behavioral and personality changes appear in the early stages of FTD. However, in clinical practice, the differential diagnosis is difficult. Objectives: The objective of this systematic review is to establish neuropsychological characteristics and similarities in patients with AD and FTD, identifying key elements for their differential diagnosis, through clinical and imaging exams, with the objective of enhancing the clinical management of the patient. Methods: A bibliographic survey was carried out in SciELO and PubMed databases and indexers, using the terms “frontotemporal dementia”, “alzheimer’s” and “neuropsychology”, in Portuguese and English. Six updated articles were then selected, considering their adequacy to the objective of the work. Results: Evidence suggests that there are important differences in cognitive domains such as language (eg, verbal fluency), memory, social cognition, executive functioning, and behavior; These aspects should be considered fundamental in any process of neuropsychological evaluation and diagnosis. Conclusion: There are linguistic aspects that promise to be powerful biomarkers for the differential diagnosis between AD and FTD, namely semantic and phonemic verbal fluency and semantic-grammatical alterations, which may be fundamental in differentiating the diseases leading to an adequate conduct for each patient.
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Ramos, Júlia Xavier, Bruno Zacarias, Breno Barbosa, and Simone Brandão. "18-FDG PET ANALYSYS FOR DEMENTIA DIAGNOSIS- BASELINE RESULTS FROM A REFERENCE CENTER IN RECIFE, BRAZIL." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda061.

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Background: Positrons emission tomography associated with computed tomography- PET/CT using the 18 F-fluorodeoxyglucose is a well-established exam for the medical evaluation of dementia, mainly helping in differential diagnosis to determine the specific type of dementia. Objectives: To describe the role of the PET/CT in the differential diagnosis of dementia in patients. Methods: a single-center, descriptive and records-based analysis of patients with Dementia evaluated in a clinic of Neurology at Recife and referred to PET/ CT due to diagnosis uncertainty, between 2020-2021. Results: 29 patients were included. The mean age was 65 years-old and 62% were female. Alzheimer’s dementia was the main diagnostic hypothesis (41.3%). PET/CT was suggestive of Alzheimer’s in 24%, Frontotemporal dementia in 21% and Lewy Bodies Dementia in 17% of patients. PET/CT results disagreed from clinical hypothesis in 21% o and in 10% it was inconclusive. In 38% it corroborated the clinical suspicion. Conclusions: in this sample the use of PET/CT FDG contributed to improve diagnostic accuracy in a significant subset of patients, mostly in the scenery of diagnostic uncertainty or atypical syndromes such as earlyonset dementias. A larger sample size and the continuation of this research will give us more information in the near future.
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Kumari, R. S., P. S. Mathuranath, T. Varghese, and C. Kesavdas. "Segmentation of mr brain images using FCM technique in frontotemporal dementia." In IET Chennai 3rd International Conference on Sustainable Energy and Intelligent Systems (SEISCON 2012). Institution of Engineering and Technology, 2012. http://dx.doi.org/10.1049/cp.2012.2191.

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Jaeger, Antônio, Eduarda Carreira, Natália Gama, Paulo Caramelli, and Leonardo Souza. "CONTROLLED EPISODIC RETRIEVAL IN BEHAVIORAL VARIANT FRONTOTEMPORAL DEMENTIA AND ALZHEIMER DISEASE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda012.

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Background: Recent studies showed that episodic memory is impaired in behavioral variant of Frontotemporal dementia (bvFTD), but in contrast to Alzheimer disease (AD) it is assumed to be caused primarily by deficits in executive control. Objective: Our goal was to probe this possibility by testing bvFTD and AD patients in a source memory task which manipulated executive control. Methods: We assessed 14 healthy controls (HC), 20 bvFTD patients, and 18 AD patients in a source memory task for spatial location in which objects were first seen at the left or right side of the screen, and at test in the center of the screen, when participants were asked to indicate in which side of the screen each object was studied. Importantly, at test, predictive arrow cues (66.7% valid/33.3% invalid) indicated the likely prior location of each object. Results: BvFTD and AD patients showed indistinguishable overall memory performances, although both showed significantly poorer performances than HC. Furthermore, although both HC and bvFTD participants had their memory judgments affected by cueing, showing poorer memory accuracy after invalid than after valid cues, AD patients showed equivalent performance for both cue types. Conclusion: The current findings support the notion that episodic memory is impaired in bvFTD, and suggests that such impairment can be as severe as in AD. The cause of this impairment, however, was not related to the executive dysfunctions manipulated in the current source memory task, but rather to further mechanisms in the bvFTD memory deficits.
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Arca, Vitor, Pedro Albuquerque, Victor Correia, Amanda Pires, Hugo Araújo, Luziany Araújo, and Breno Barbosa. "RIGHT VS. LEFT TEMPORAL LOBE SEMIOLOGY IN DEMENTIA: LESSONS FROM TWO CASES WITH FOCAL FRONTOTEMPORAL DEMENTIA SYNDROMES." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda100.

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Background: Case 1: a 59-year old man presented to our service with 4 years of progressive cognitive and behavioral symptoms. He became forgetful and experienced difficulties managing his payments. After 4 years he could no longer recognise his relatives. Cognitive assessment showed a mini-mental status examination of 17/30. MRI and SPECT revealed respectively focal atrophy and hipoperfusion of the frontal regions and anterior right temporal lobe. Case 2: a 72-year-old woman was brought to evaluation with a 5-years history of progressive language and behavioral deterioration. Her family reported early speech errors and behavioral changes, with a marked aggressiveness, ritualistic behaviors and hyperorality. Cognitive evaluation revealed a MMSE of 6/30 mainly due to a relatively fluent afasia. Brain MRI showed asymmetric cerebral atrophy, more prominent in the anterior left temporal lobe. Objective: N/H Methods: N/H Results: N/H Conclusion: We describe two cases of suspected frontotemporal dementia (FTD) syndromes. The left ATL may receive proportionately more input from the lexical and phonological centers subserving word processing. The right ATL may receive more input from right-lateralized emotion processing hubs. Focal atrophy of the left anterior temporal lobe has been associated with the semantic type of primary progressive aphasia evolving to semantic dementia. In contrast, focal atrophy of the right temporal lobe has recently been described as a controversial entity reported as the right temporal variant of FTD.
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SPILLANTINI, MARIA GRAZIA. "TAU GENE MUTATIONS IN FRONTOTEMPORAL DEMENTIA AND PARKINSONISM LINKED TO CHROMOSOME 17." In Proceedings of the International Seminar on Nuclear War and Planetary Emergencies — 27th Session. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812705150_0071.

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Horn, Jean-francois, Marie-odile Habert, Alain Giron, and Bernard Fertil. "ALZHEIMER'S DISEASE AND FRONTOTEMPORAL DEMENTIA DIFFERENTIAL AUTOMATIC DIAGNOSIS BASED ON SPECT IMAGES." In 2007 4th IEEE International Symposium on Biomedical Imaging: From Nano to Macro. IEEE, 2007. http://dx.doi.org/10.1109/isbi.2007.357107.

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Mary Clare, McKenna, Hutchinson Siobhan, and Bede Peter. "Cerebellar involvement in frontotemporal dementia: the infratentorial imaging signatures of FTD phenotypes." In Association of British Neurologists: Annual Meeting Abstracts 2023. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jnnp-2023-abn.78.

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Reports on the topic "Frontotemporal dementia"

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Wei, Huijie, Xin Mu, Yu Li, Hua Lei, De Yang, Tian Li, and Junwei Ren. Meta-analysis of the association between CHCHD10 Pro34Ser variant and the risk of frontotemporal dementia. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2021. http://dx.doi.org/10.37766/inplasy2021.5.0090.

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