Journal articles: 'Footrot'

1

Morgan, Kenton. "Footrot." In Practice 9, no. 4 (July 1987): 124–29. http://dx.doi.org/10.1136/inpract.9.4.124.

APA, Harvard, Vancouver, ISO, and other styles

2

Scott, Phil. "Ovine footrot." Livestock 17, no. 3 (May 2012): 37–40. http://dx.doi.org/10.1111/j.2044-3870.2012.00114.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

ABBOTT, KA, and JR EGERTON. "Eradication of footrot of lesser clinical severity (intermediate footrot)." Australian Veterinary Journal 81, no. 11 (November 2003): 688–93. http://dx.doi.org/10.1111/j.1751-0813.2003.tb12541.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Gaden, C. A., B. F. Cheetham, E. Hall, G. Green, and M. E. Katz. "Producer-initiated field research leads to a new diagnostic test for footrot." Animal Production Science 53, no. 8 (2013): 610. http://dx.doi.org/10.1071/an11175.

Full text

Abstract:

The Cicerone Project was formed in 1998 to address problems faced by wool producers. In the New England area, the issue of suspected false positive diagnoses of virulent footrot, which can be a significant cause of economic loss to individual producers, was investigated. In New South Wales, footrot diagnosis is primarily a field diagnosis supported by the gelatin gel laboratory test. The principal causative agent of footrot is Dichelobacter nodosus. If the gelatin gel test finds strains of D. nodosus to be thermostable (gel stable), a finding of virulent footrot is likely and quarantine of the affected property follows. However, livestock producers and inspectors reported that there were a considerable number of cases where laboratory tests found strains to be stable but these strains did not cause virulent footrot in the field. Preliminary results using DNA markers associated with virulent footrot showed that one of these markers, intA, was absent in gel stable, field benign strains but present in all strains tested which caused field virulent footrot. A trial conducted at Uralla, New South Wales, demonstrated conclusively that there were strains of D. nodosus which were stable in the gelatin gel test but did not cause virulent footrot in the field. All of these strains were negative in the intA DNA test. These results were confirmed in a second field trial at Molong, New South Wales. These trials were instrumental in establishing that the gelatin gel test at times gave results inconsistent with the clinical expression of footrot, potentially leading to a false positive diagnosis of virulent footrot. Subsequent research led to confirmation of the intA test, which is now available as an additional tool for footrot diagnosis.

APA, Harvard, Vancouver, ISO, and other styles

5

Angell, Joseph William, Dai H. Grove-White, and Jennifer S. Duncan. "Sheep and farm level factors associated with footrot: a longitudinal repeated cross-sectional study of sheep on six farms in the UK." Veterinary Record 182, no. 10 (January 23, 2018): 293. http://dx.doi.org/10.1136/vr.104553.

Full text

Abstract:

Footrot is an ovine foot disease of infectious origin and a cause of serious welfare and economic compromise in affected animals and flocks. The development of footrot in sheep is associated with the infectious agent Dichelobacter nodosus, which may invade as a primary pathogen, but the risk of disease is increased following damage to the interdigital skin of the foot. In this study, we used data from six farms in North Wales collected between June 2012 and October 2013 to model the dynamic changes of footrot prevalence over time and investigate the association of footrot with multiple farm, management, environmental and sheep factors. Footrot prevalence varied widely within and between farms and overall varied with season with an increase in prevalence shown in late summer and again in the spring. In addition, sheep were more likely to have footrot when the flock size was larger, when grazing poached pasture or when grazing a longer sward, and yearling sheep were less likely to have footrot when compared with lambs and adult sheep. These data may be helpful for advising farmers of likely environmental events, risk groups and management practices that may increase the probability of sheep developing footrot.

APA, Harvard, Vancouver, ISO, and other styles

6

Dhungyel, Om, James Hunter, and Richard Whittington. "Footrot vaccines and vaccination." Vaccine 32, no. 26 (May 2014): 3139–46. http://dx.doi.org/10.1016/j.vaccine.2014.04.006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Egerton, John. "Aetiology of ovine footrot." Preventive Veterinary Medicine 117, no. 1 (November 2014): 313. http://dx.doi.org/10.1016/j.prevetmed.2014.08.019.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Agbaje, Michael, Catrin S. Rutland, Grazieli Maboni, Adam Blanchard, Melissa Bexon, Ceri Stewart, Michael A. Jones, and Sabine Totemeyer. "Novel inflammatory cell infiltration scoring system to investigate healthy and footrot affected ovine interdigital skin." PeerJ 6 (July 2, 2018): e5097. http://dx.doi.org/10.7717/peerj.5097.

Full text

Abstract:

Ovine footrot is a degenerative disease of sheep feet leading to the separation of hoof-horn from the underlying skin and lameness. This study quantitatively examined histological features of the ovine interdigital skin as well as their relationship with pro-inflammatory cytokine (IL-1β) and virulent Dichelobacter nodosus in footrot. From 55 healthy and 30 footrot ovine feet, parallel biopsies (one fixed for histology) were collected post-slaughter and analysed for lesions and histopathological analysis using haematoxylin and eosin and Periodic Acid-Schiff. Histological lesions were similar in both conditions while inflammatory scores mirror IL-1β expression levels. Increased inflammatory score corresponded with high virulent D. nodosus load and was significant (p < 0.0001) in footrot feet with an inflammatory score of 3 compared to scores 1 and 2. In addition, in contrast to healthy tissues, localisation of eubacterial load extended beyond follicular depths in footrot samples. The novel inflammatory cell infiltration scoring system in this study may be used to grade inflammatory response in the ovine feet and demonstrated an association between severity of inflammatory response and increased virulent D. nodosus load.

APA, Harvard, Vancouver, ISO, and other styles

9

Skerman, TM, DL Johnson, DW Kane, and JN Clarke. "Clinical footscald and footrot in a New Zealand Romney flock: phenotypic and genetic parameters." Australian Journal of Agricultural Research 39, no. 5 (1988): 907. http://dx.doi.org/10.1071/ar9880907.

Full text

Abstract:

Phenotypic and genetic parameters for clinical footscald and footrot were investigated in a pedigree flock of New Zealand Romney sheep inspected during outbreaks over six years. At inspections, each sheep's overall status with regard to footscald and footrot was summarized on a combined severity scale (Rating). Sheep were also categorized in terms of two binomial variables specifying the presence or absence of footscald or footrot (FI) and the presence or absence of footrot only (FR). A trinomial variable specified no infection, infection limited to footscald, or footrot with or without footscald (FSR), all variables being derived from the Rating value. Notwithstanding marked annual variations in prevalence of FI, F1 prevalence increased with age among ewes, and a higher prevalence of FI among male than female lambs was noted. Birth rank, birth date and dam's age had no significant effect on Rating in lambs. Repeatability of ewe Rating over successive annual inspections was low. Heritabilities of Rating, FI, FR and FSR, estimated by half-sib analyses were 0.14, 0.28, 0.17 and 0.25, respectively. Heritability of Rating estimated from dam-offspring regression was 0.12. Production traits did not appear to be genetically associated with liability to footrot. Selection of the top 5% of progenytested sires for breeding could be expected to reduce the prevalence of FI by approximately 45% per generation.

APA, Harvard, Vancouver, ISO, and other styles

10

Raadsma, H. W., and O. P. Dhungyel. "A review of footrot in sheep: New approaches for control of virulent footrot." Livestock Science 156, no. 1-3 (September 2013): 115–25. http://dx.doi.org/10.1016/j.livsci.2013.06.011.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Lacombe-Antoneli, Angela, S. Píriz, and S. Vadillo. "In vitro antimicrobial susceptibility of anaerobic bacteria isolated from caprine footrot." Acta Veterinaria Hungarica 55, no. 1 (March 1, 2007): 11–20. http://dx.doi.org/10.1556/avet.55.2007.1.2.

Full text

Abstract:

The in vitro antimicrobial susceptibility of Gram-negative anaerobic bacilli commonly isolated from footrot in goats was studied. A total of 97 isolates belonging to the genera Dichelobacter, Fusobacterium, Prevotella, Porphyromonas and Bacteroides, obtained from clinical cases of footrot in south-western Spain between March 2000 and May 2001, were tested against 25 antimicrobial agents comprising β-lactams, aminoglycosides, macrolides, chloramphenicol, quinolones, lincosamides, sulphonamides and tetracyclines in order to optimise antibiotic treatment of this disease in goats. β-lactams, tetracyclines and metronidazole displayed the highest in vitro efficacy against the species involved in the pathogenesis of footrot.

APA, Harvard, Vancouver, ISO, and other styles

12

Qureshi, Sabia, Shakil A. Wani, Shaheen Farooq, Zahid Kashoo, Basharat Bhat, Mohammad Isfaqul Hussain, Aasim Habib, et al. "Genome sequence of Dichelobacter nodosus JKS-07B isolate from J&K, India associated with virulent footrot of sheep." Science Progress 104, no. 4 (October 2021): 003685042110576. http://dx.doi.org/10.1177/00368504211057678.

Full text

Abstract:

Introduction: Virulent footrot of sheep caused by Dichelobacter nodosus is associated with tremendous economic losses due to recurrent treatment costs and increased culling rates. This organism being a fastidious anaerobe is difficult to isolate on ordinary media that does not support its growth. The D. nodosus serogroup B isolate described in the present study has been used in the preparation of the whole-cell killed vaccine against footrot in India. D. nodosus serogroup B is the predominant serogroup involved in virulent footrot (lesion score 4) in India as well as in many sheep-rearing countries of the globe. Methods: Genomic DNA was extracted using wizard Genomic DNA purification kit. The whole genome of the D. nodosus strain B was sequenced using an Illumina HiSeq 2500 platform and annotated according to functional gene categories. Annotations were performed using in-house developed Perl scripts using Nr/Nt database, uniprot, Pfam, KEGG, Panther DB, and GO database. Result: The assembled genome size is 1.311,533 Mb and GC content is 44.38. A total of 1215 protein-coding genes, 44tRNA and 7 rRNA were identified. The genome shows 98.63% sequence homology with the reference genome. However, 21 new genes have been identified in this genome. The information will provide insights into the various genes and regulators necessary for D. nodosus growth and survival. Discussion: The genome information of this serogroup B of D. nodosus isolate involved in 85–90% cases of virulent footrot of sheep in India provides further insights for improvement of the killed vaccine (B serogroup) developed recently in India. For the development of an efficacious vaccine against virulent footrot, it is essential to know the serological diversity as well as the virulent status of the strains of the D. nodosus. This serogroup isolate is a potential vaccine candidate to mitigate ovine footrot in India as the majority of virulent footrot cases belong to serogroup B of D. nodosus.

APA, Harvard, Vancouver, ISO, and other styles

13

Nieuwhof, G. J., and S. C. Bishop. "Costs of the major endemic diseases of sheep in Great Britain and the potential benefits of reduction in disease impact." Animal Science 81, no. 1 (August 2005): 23–29. http://dx.doi.org/10.1079/asc41010023.

Full text

Abstract:

AbstractThe costs of three major endemic sheep diseases in Great Britain, gastro-intestinal (GI) parasites, footrot and scab, were estimated and compared with costs for other diseases from another study. Disease costs include lost performance, preventive measures and treatment of affected animals. The most costly disease, of those studied, for the British sheep industry is infestation with GI parasites, with estimated annual costs of £84 million. Annual costs for the other two diseases are £24 million for footrot and £8 million for sheep scab. This compares with literature estimates of £20 million for Chlamydial abortions and £12 million for toxoplasmosis. For sheep scab most costs are for preventive measures, therefore, short of eradication, a reduction in incidence will have a limited effect on costs. For GI parasites, costs are linearly related to the severity of the infestation and a reduction of the disease will have a proportional effect on the costs to the industry. For footrot about half the costs are for preventive measures, the other half is for lost production and treatment. A reduction in the incidence of footrot has a proportional effect on the £10 million associated with loss of production and treatment of infected animals. It is concluded that gastro-intestinal parasites and footrot are two sheep diseases in Britain for which a reduction of severity or incidence will have a large impact on costs of production.

APA, Harvard, Vancouver, ISO, and other styles

14

O'MEARA, T. J., J. R. EGERTON, and H. W. RAADSMA. "Recombinant vaccines against ovine footrot." Immunology and Cell Biology 71, no. 5 (October 1993): 473–88. http://dx.doi.org/10.1038/icb.1993.53.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Han, Xiaoyan, Ruth M. Kennan, and Julian I. Rood. "Serine proteases and ovine footrot." Microbiology Australia 34, no. 1 (2013): 37. http://dx.doi.org/10.1071/ma13012.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Kennan, Ruth M., Xiaoyan Han, Corrine J. Porter, and Julian I. Rood. "The pathogenesis of ovine footrot." Veterinary Microbiology 153, no. 1-2 (November 2011): 59–66. http://dx.doi.org/10.1016/j.vetmic.2011.04.005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Allworth, M. B. "Challenges in ovine footrot control." Small Ruminant Research 118, no. 1-3 (May 2014): 110–13. http://dx.doi.org/10.1016/j.smallrumres.2013.12.007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Zheng, Jiasan, Shi Shu, Cheng Xia, Chuang Xu, Hongyou Zhang, and Hongbin Wang. "2-DE-MS based proteomic investigation of dairy cows with footrot." Journal of Veterinary Research 60, no. 1 (March 1, 2016): 63–69. http://dx.doi.org/10.1515/jvetres-2016-0010.

Full text

Abstract:

Abstract Introduction: The differentially expressed proteins between healthy cows and those with footrot were identified to explore changes in protein profiles associated with the disease. Material and Methods: Out of 36 cows selected for the experiment, 18 footrot-affected cows were included in the treatment group (group T) and 18 unaffected cows were included in the control group (group C). Plasma samples from groups T and C were subjected to two-dimensional electrophoresis analysis and differentially expressed proteins were identified by matrix-assisted laser desorption/ionisation tandem time-of-flight mass spectrometry. Bioinformatics, including gene ontology analysis and pathway analysis, was used for analysing all proteins. Results: Out of 63 spots identified by 2DE, 33 were selected for mass spectrum analysis, which identified 11 differentially expressed proteins in 26 spots. Footrot led to changes in profiles in plasma proteins that were classified to the pathway of inflammatory response, complement, and blood coagulation, among others. Conclusion: This study provides evidence of the defence mechanisms of cows with footrot to explore strategies for treatment.

APA, Harvard, Vancouver, ISO, and other styles

19

Duncan, J. S., D. Grove-White, E. Moks, D. Carroll, J. W. Oultram, C. J. Phythian, and H. W. Williams. "Impact of footrot vaccination and antibiotic therapy on footrot and contagious ovine digital dermatitis." Veterinary Record 170, no. 18 (January 21, 2012): 462. http://dx.doi.org/10.1136/vr.100363.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Schwartzkopf-Genswein, Karen, Wiolene Montanari Nordi, Desiree Gellatly, Daniela M. Meléndez, Timothy Schwinghamer, Sonia Marti, Kelly Anklam, Joyce Van Donkersgoed, Kathy Parker, and Dorte Dopfer. "PSIII-14 Infrared thermography as a tool to detect inflammation in feedlot lambs with footrot." Journal of Animal Science 97, Supplement_3 (December 2019): 185–86. http://dx.doi.org/10.1093/jas/skz258.383.

Full text

Abstract:

Abstract Infrared thermography (IRT) has been used as a non-invasive tool to detect inflammatory processes associated with disease in livestock. The aim of this study was to evaluate IRT as a tool to compare healthy and footrot (FR) affected hooves in feedlot lambs with varying degrees of lameness over two seasons. A total of 106 lame lambs with footrot from a feedlot in Alberta were individually categorized according to a 3-point locomotion scale [1 = mild (n = 7), 2 = moderate (n = 46) and 3 = severe lameness (n = 53)] during the summer (n = 39) and fall (n = 68) of 2018. All lambs were physically examined once by two experienced observers to determine if the lamb had footrot. IRT images of the interdigital space were used to obtain the maximum hoof temperature (MHT) of both FR affected as well as healthy (CT) hooves within the same animal. Generalized linear mixed models (SAS PROC GLIMMIX) were performed separately for each season and diagnosis and included locomotion score as a fixed effect and ambient temperature as a co-variate. Predicted means were compared using the limits at 95% of confidence. Overall, greater MHT (P < 0.05) were observed for FR affected compared to unaffected hooves for lambs categorized as moderately and severely lame, within each season. However, no differences (P > 0.05) in MHT were observed for lambs categorized as mildly lame, likely due to the small number of lambs having a locomotion score of 1. Under the conditions of this study, thermal images can be effectively used as a tool to distinguish footrot affected hooves in feedlot lambs with moderate and severe lameness. Further studies should be conducted with more lambs with a locomotion score of 1 to assess the relationship between mild lameness, IRT, and footrot diagnosis.

APA, Harvard, Vancouver, ISO, and other styles

21

Etebu, Ebimieowei, and A. Mark Osborn. "A Review of Indicators of Healthy Agricultural Soils with Pea Footrot Disease Suppression Potentials." Sustainable Agriculture Research 1, no. 2 (July 23, 2012): 235. http://dx.doi.org/10.5539/sar.v1n2p235.

Full text

Abstract:

The quality of a soil is often viewed in relation to its ability to suppress plant disease and enhance agricultural productivity. A soil is considered suppressive when, in spite of favourable conditions for disease incidence and development, a pathogen cannot become established, or establishes but produces no disease, or establishes and produces disease for a short time and then declines. The interplay of biotic and abiotic factors has long been known to assert disease suppressive capabilities or otherwise. However, the multi-functionality of soil makes the identification of a single property as a general indicator of soil health an uphill task. In this paper, therefore, some indicators of soil health important to agriculture are reviewed with emphasis on pea footrot disease suppression potentials. Findings show that footrot disease due to Nectria haematococca (anamorph Fusarium solani f.sp pisi) is a globally, economically important disease of peas, and an initial inoculum density of ? 100 pathogenic forms of N. haematococca cells would produce an appreciable level of pea footrot disease depending on the relative amount of phosphorus, carbon and nitrogen present in soil. It would be desirable to confirm pea footrot disease models obtained from pot experiments with results from field experiments.

APA, Harvard, Vancouver, ISO, and other styles

22

Fesseha, Haben. "Ovine Footrot and Its Clinical Management." Veterinary Medicine: Research and Reports Volume 12 (May 2021): 95–99. http://dx.doi.org/10.2147/vmrr.s306207.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Hurtado, M. A., S. Piriz, J. Valle, R. Jimenez, and S. Vadillo. "Aetiology of ovine footrot in Spain." Veterinary Record 142, no. 3 (January 17, 1998): 60–63. http://dx.doi.org/10.1136/vr.142.3.60.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

HINTON, DG. "Eradication of actively spreading ovine footrot." Australian Veterinary Journal 68, no. 3 (March 1991): 118–19. http://dx.doi.org/10.1111/j.1751-0813.1991.tb00774.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

EGERTON, JR. "Eradication of actively spreading ovine footrot." Australian Veterinary Journal 68, no. 8 (August 1991): 283. http://dx.doi.org/10.1111/j.1751-0813.1991.tb03250.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Winter, A. C. "Footrot control and eradication (elimination) strategies." Small Ruminant Research 86, no. 1-3 (October 2009): 90–93. http://dx.doi.org/10.1016/j.smallrumres.2009.09.026.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Vatn, Synnøve. "Enhanced possibilities for eradicating virulent footrot." Veterinary Journal 217 (November 2016): 1–2. http://dx.doi.org/10.1016/j.tvjl.2016.09.001.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Whittington, R. J., and P. J. Nicholls. "Grading the lesions of ovine footrot." Research in Veterinary Science 58, no. 1 (January 1995): 26–34. http://dx.doi.org/10.1016/0034-5288(95)90084-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Lewis, C. J. "Footrot and interdigital dermatitis in sheep." Veterinary Record 158, no. 2 (January 14, 2006): 71–72. http://dx.doi.org/10.1136/vr.158.2.71-b.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Wassink, G. J., L. E. Green, L. J. Moore, and R. Grogono-Thomas. "Footrot and interdigital dermatitis in sheep." Veterinary Record 158, no. 2 (January 14, 2006): 72. http://dx.doi.org/10.1136/vr.158.2.72.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Conington, J., B. Hosie, G. J. Nieuwhof, S. C. Bishop, and L. Bünger. "Breeding for resistance to footrot – the use of hoof lesion scoring to quantify footrot in sheep." Veterinary Research Communications 32, no. 8 (May 14, 2008): 583–89. http://dx.doi.org/10.1007/s11259-008-9062-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Haq, MM, MM Rahman, S. Rana, M. Khatun, and K. Samaddar. "Prevalence of ovine footrot in Brahmaputra Chars (shoals) in Mymensingh." Bangladesh Veterinarian 31, no. 2 (May 9, 2016): 74–83. http://dx.doi.org/10.3329/bvet.v31i2.27688.

Full text

Abstract:

A retrospective cohort study was conducted from July 2012 to August 2013 to determine prevalence of ovine footrot in chars (Shoals) of Mymensingh Sadar Upazila, Bangladesh, centred in Paranganj Union Parishad. A field survey used a prepared questionnaire and investigation of farm management. The prevalence of ovine footrot was 4.2% in the study group of 687 sheep, in 106 farms. The prevalence differed according to farm type and housing system. Prevalence of ovine footrot was higher (9.5%) in farms where sheep and other livestock were housed under the same roof. Prophylactic medication and footbath practice were absent. Common care and management practices were sheep bath, house and floor cleaning, deworming, and therapeutic medication. Highest prevalence (7.0%) was recorded in farms where prophylactic medication and deworming was irregular, and antiseptics were not used in sheep bathing and cleaning of sheep house and floor. The results suggest that improvement of the housing environment and management practices are required to control footrot.Bangl. vet. 2014. Vol. 31, No. 2, 74-83

APA, Harvard, Vancouver, ISO, and other styles

33

GHIMIRE, S. C., and J. R. EGERTON. "PCR–RFLP of outer membrane proteins gene of Dichelobacter nodosus: a new tool in the epidemiology of footrot." Epidemiology and Infection 122, no. 3 (June 1999): 521–28. http://dx.doi.org/10.1017/s0950268899002290.

Full text

Abstract:

Currently only phenotypic epidemiological markers, serogrouping and virulence testing of Dichelobacter nodosus, are available for investigating footrot outbreaks in small ruminants. These methods have limitations in tracing the source of infection. In this study, a genotypic marker, PCR–RFLP of outer membrane protein gene, was used to characterize D. nodosus. The technique was evaluated in a controlled experiment involving two strains of bacteria. PCR–RFLP was found to be highly specific in differentiating isolates obtained from recipient animals infected with different strains. Subsequently, this technique was used to characterize isolates obtained from field cases of footrot in Nepal. A total of 11 patterns was recognized among 66 Nepalese D. nodosus isolates representing four different serogroups. PCR–RFLP also discriminated isolates with similar phenotypic characteristics. However, all isolates which, phenotypically, were virulent were represented by only two patterns irrespective of their serogroups. It is suggested that PCR–RFLP described here could be a useful epidemiological marker in the study of footrot.

APA, Harvard, Vancouver, ISO, and other styles

34

Han, Jiaqi, Jing Dong, Wenhui Yang, Yunze Xu, Qiming Xing, Yuelei Chen, and Lin Li. "Detection of Fusobacterium necrophorum and Dichelobacter nodosus from cow footrot in the Heilongjiang Province, China." Acta Veterinaria Brno 88, no. 2 (2019): 165–68. http://dx.doi.org/10.2754/avb201988020165.

Full text

Abstract:

Cow footrot in the Heilongjiang Province, northeast China is a problem resulting in lost production in agriculture. In this study, 200 swab samples from footrot lesions of naturally infected cows with odorous exudative inflammation and keratinous hoof separation at 10 farms were examined in the period from May 2016 to May 2017. Twenty cows from each farm were taken for sampling. The samples were examined for detectingthe presence of Dichelobacter nodosus (D. nodosus) and Fusobacterium necrophorum (F. necrophorum). Such detection was carried out using polymerase chain reaction (PCR). The PCR primers were designed to identify the lktA gene, which encodes a leukotoxin unique to F. necrophorum, and the fimA gene of D. nodosus. Of the 200 samples, 111 (55.5%) revealed the presence of F. necrophorum and 11 (5.5%) exhibited D. nodosus. The frequent finding of F. necrophorum in cow farms of Heilongjiang province, northeast China is noteworthy. The possibility of F. necrophorum and D. nodosus infection should be an important concern when controlling cow footrot in China.

APA, Harvard, Vancouver, ISO, and other styles

35

Zanolari, Patrik, Salome Dürr, Jörg Jores, Adrian Steiner, and Peter Kuhnert. "Ovine footrot: A review of current knowledge." Veterinary Journal 271 (May 2021): 105647. http://dx.doi.org/10.1016/j.tvjl.2021.105647.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Lewis, R. D., H. H. Meyer, J. L. Gradin, and A. W. Smith. "Effectiveness of Vaccination in Controlling Ovine Footrot." Journal of Animal Science 67, no. 5 (1989): 1160. http://dx.doi.org/10.2527/jas1989.6751160x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Gurung, R. B., P. Tshering, O. P. Dhungyel, and J. R. Egerton. "Distribution and prevalence of footrot in Bhutan." Veterinary Journal 171, no. 2 (March 2006): 346–51. http://dx.doi.org/10.1016/j.tvjl.2004.11.012.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Whittington, R. J., V. F. Saunders, and E. K. Moses. "Antigens for serological diagnosis of ovine footrot." Veterinary Microbiology 54, no. 3-4 (March 1997): 255–74. http://dx.doi.org/10.1016/s0378-1135(96)01284-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

WHITTINGTON, RJ, and DJ MARSHALL. "An anamnestic serological test for ovine footrot." Australian Veterinary Journal 67, no. 5 (May 1990): 157–59. http://dx.doi.org/10.1111/j.1751-0813.1990.tb07744.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Maboni, G., S. Frosth, A. Aspán, and S. Tötemeyer. "Ovine footrot: new insights into bacterial colonisation." Veterinary Record 179, no. 9 (June 17, 2016): 228. http://dx.doi.org/10.1136/vr.103610.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

ABBOTT, KA, and JR EGERTON. "Effect of climatic region on the clinical expression of footrot of lesser clinical severity (intermediate footrot) in sheep." Australian Veterinary Journal 81, no. 12 (December 2003): 756–62. http://dx.doi.org/10.1111/j.1751-0813.2003.tb14609.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Schwartzkopf-Genswein, Karen S., Wiolene M. Nordi, Désirée Gellatly, Daniela M. Meléndez, Timothy D. Schwinghamer, Sonia Marti, Kelly S. Anklam, Dorte Dopfer, and Joyce Van Donkersgoed. "PSV-1 A prospective longitudinal study of the incidence and risk factors associated with footrot in feedlot lambs in Southern Alberta." Journal of Animal Science 98, Supplement_4 (November 3, 2020): 227. http://dx.doi.org/10.1093/jas/skaa278.417.

Full text

Abstract:

Abstract Lameness in sheep caused by footrot (FR) is a significant health, welfare, and economic concern worldwide. To date, no studies have documented the incidence of FR or associated risk factors in feedlot lambs in Alberta. The objectives of this study were to determine 1) FR incidence and 2) animal, managerial and environmental risk factors associated with FR in one Southern Alberta, lamb feedlot. Assessments were conducted biweekly (average of 10 pens per visit) by 2 experienced observers. A total of 73,150 lambs were assessed between October 2017 and March 2019. All lame lambs were scored according to a 3-point mobility scale (1 = mild, 2 = moderate, and 3 = severe lameness) and physically examined to diagnose the cause of lameness. Risk factors associated with FR were documented and included gender, days on feed (DOF), diet composition, and season. Multivariable regression models (SAS PROC GLIMMIX) were used to determine significant risk factors. A total of 473 lambs were identified as lame, 107 of which were diagnosed with FR (incidence of 22.6%). Footrot affected lambs had greater mobility scores (≥ 2; P < 0.0001) than all other lame diagnoses. Footrot was 4.40 and 0.10 times more likely (P < 0.0001) in female and wether than ram lambs, and 0.60 and 0.23 times more likely (P < 0.0001) in fall and summer than winter and spring seasons. The odds of being diagnosed with FR increased for each additional DOF and each unit increase of barley in the diet (P = 0.0268), while the odds decreased (P = 0.0016) for each additional unit of supplement in the diet. Based on our findings, footrot is an issue to lambs in Alberta, and further studies are still necessary to understand the risk factors associated with potential strategy for mitigating FR in feedlot lambs.

APA, Harvard, Vancouver, ISO, and other styles

43

Gelasakis, Kalogianni, and Bossis. "Aetiology, Risk Factors, Diagnosis and Control of Foot-Related Lameness in Dairy Sheep." Animals 9, no. 8 (July 31, 2019): 509. http://dx.doi.org/10.3390/ani9080509.

Full text

Abstract:

During the last twenty years, considerable research efforts have recognized the consequences of foot‐related lameness primarily in cattle, and meat and wool sheep. Despite the lack of extensive epidemiological studies, field observations and isolated research reports in dairy sheep have suggested that the problem might be more severe in semi-intensive and intensive farming systems. Footrot, contagious ovine digital dermatitis, ovine interdigital dermatitis, white line disease, and pedal joint abscess are the most common causes of foot-related lameness. Dichelobacter nodosus, Fusobacterium necrophorum, Treponema spp., and Actinomyces pyogenes are the most significant foot-related lameness-associated pathogens. Despite a documented hereditary predisposition, environmental factors are the most important in determining the occurrence of foot-related lameness. Moist and warm environment, increased parity and milk yield, inappropriate housing conditions and infrastructures, inadequate hygiene status, imbalanced nutrition, and insufficient foot care are the most critical risk factors. Furthermore, a foot-lameness control plan should include targeted implementation of claw trimming and footbathing, evidence-based planning of hygiene measures in preventive veterinary practices (i.e., antibiotic administration, vaccinations against footrot), selective breeding to footrot resistance, and, most importantly, the continuous training of farming personnel. Controlling foot-lameness in dairy sheep is critical in determining the well-being of animals, and strongly affects the farm’s profitability and sustainability.

APA, Harvard, Vancouver, ISO, and other styles

44

Storms, Julia, Anna Wirth, Danae Vasiliadis, Isabelle Brodard, Antje Hamann-Thölken, Christina Ambros, Udo Moog, Jörg Jores, Peter Kuhnert, and Ottmar Distl. "Prevalence of Dichelobacter nodosus and Ovine Footrot in German Sheep Flocks." Animals 11, no. 4 (April 12, 2021): 1102. http://dx.doi.org/10.3390/ani11041102.

Full text

Abstract:

The bacterium Dichelobacter nodosus (D. nodosus) is the causative agent of ovine footrot. The aim of this field study was to determine the prevalence of D. nodosus in German sheep flocks. The sheep owners participated voluntarily in the study. More than 9000 sheep from 207 flocks were screened for footrot scores using a Footrot Scoring System from 0 to 5 and sampling each sheep using one interdigital swab for all four feet of the sheep. The detection and discrimination between benign and virulent strains was done employing a real-time PCR. Our results showed a mean prevalence of 42.93% of D. nodosus in German sheep on an animal level. Underrunning of hoof horn on at least one foot (Scores 3-5) was detected in 567 sheep (6.13%). Sheep with four clinically healthy feet were found through visual inspection in 47.85% of all animals included in this study. In total, 1117 swabs from sheep with four clinically healthy feet tested positive for D. nodosus. In 90.35% of the positive swabs, virulent D. nodosus were detected. Benign D. nodosus were detected in 4.74% of the D. nodosus-positive swabs while 4.91% tested positive for both, benign and virulent D. nodosus. In 59 flocks D. nodosus were not detected and in 115 flocks only virulent D. nodosus were found while seven flocks tested positive for benign strains.

APA, Harvard, Vancouver, ISO, and other styles

45

Ozgen, Ediz Kagan, Seyda Cengiz, Mustafa Ulucan, Zafer Okumus, Asli Kortel, Hüseyin Erdem, and Huseyin Gurkan Sarac. "Isolation and identification of Dichelobacter nodosus and Fusobacterium necrophorum using the polymerase chain reaction method in sheep with footrot." Acta Veterinaria Brno 84, no. 2 (2015): 97–104. http://dx.doi.org/10.2754/avb201584020097.

Full text

Abstract:

Footrot is an important infectious disease of small ruminants leading to severe economical losses. The aim of the present study was to determine isolation and identification rates ofDichelobacter nodosusandFusobacterium necrophorumin the culture techniques and reveal the specificity and sensitivity of the culture technique based on the polymerase chain reaction (PCR) method in sheep with footrot. Dry swabs and swabs with Amies medium from 83 sheep were subjected to PCR and culture analyses. In dry swabs, 4 samples were positive forF. necrophorumand all were negative forD. nodosus. Colonies in Eugon and Fusobacterium selective agars from swabs with Amies medium were evaluated. Polymerase chain reaction analysis was conducted on macroscopically and microscopically unidentified samples. The positivity rate was 55.4% forD. nodosusand 69.8% forF. necrophorumin cultures from Fusobacterium selective agars. The positivity rate forD. nodosusin Fusobacterium selective agars was higher than that in Eugon agar. Performing PCR and culture methods increased positivity as compared to performing them alone. In comparison with the PCR method, culturing in Fusobacterium selective agars had moderate sensitivity and low specificity forD. nodosus(71.7 and 28.7%) andF. necrophorum(61.3 and 80.0%), respectively. In conclusion, Fusobacterium selective agar (without antibiotics) for isolation and identification ofD. nodosusis superior to Eugon agar.Fusobacterium necrophorumshould also be considered as a provoking agent for footrot in small ruminants. The PCR method on culture increases elucidation of definitive aetiology.

APA, Harvard, Vancouver, ISO, and other styles

46

Riofrio, A. C., J. C. Lopez, J. C. Moyano, R. Quinteros, P. R. Marini, and M. L. Fischman. "Strawberry Footrot in a Blackbellysheepin the Ecuador Amazonia." Sustainable Agriculture Research 5, no. 4 (September 20, 2016): 56. http://dx.doi.org/10.5539/sar.v5n4p56.

Full text

Abstract:

Dermatophilosis is an acute or cronic disease of the epidermis characterized by inflammatory, exudative and bleeding lesions with scabs, caused by the bacterium Dermathophiluscongolensis. It affects bovine, ovine, equine, cattle, goats, wild animals and also humans when it is considered aminor zoonosis. This work is about a case caused by Dermathophiluscongolensisin a sheep from Ecuadorian Amazonia. The affected sheep presented claudication with non itchy lesions in its right front limb, bleeding and painful at first. An exudate culture was taken to the laboratory. The samples were processed for a bacteriological diagnosis. Based on the clinical symptoms together with the lab results, Dermatophiluscongolensis was diagnosed. It was treated with BenzathinPenicilin 100,000 UI, each dosis of 10,000 UI / kg PV, every 48 hours, in 5 consecutive applications combined with Tolfenamic Acid 8gr. In doses of 2mg / kg, that is to say 1 ml / 40 Kg PV every 48 hours. 10 days after the beginning of the treatment, the claudication and the pain lessened and the animal tried to set its limb on the ground.

APA, Harvard, Vancouver, ISO, and other styles

47

Clifton, Rachel, and Laura Green. "Pathogenesis of ovine footrot disease: a complex picture." Veterinary Record 179, no. 9 (September 1, 2016): 225–27. http://dx.doi.org/10.1136/vr.i4554.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Green, Laura, and Rachel Clifton. "Diagnosing and managing footrot in sheep: an update." In Practice 40, no. 1 (December 22, 2017): 17–26. http://dx.doi.org/10.1136/inp.j4575.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

McGILLIVERY, DJ, JJ WEBBER, and LA TOOHEY. "A role for DNA characterisation in ovine footrot." Australian Veterinary Journal 66, no. 4 (April 1989): 106–8. http://dx.doi.org/10.1111/j.1751-0813.1989.tb09759.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Abbott, K. A., and C. J. Lewis. "Current approaches to the management of ovine footrot." Veterinary Journal 169, no. 1 (January 2005): 28–41. http://dx.doi.org/10.1016/j.tvjl.2004.05.008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Journal articles on the topic 'Footrot'

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles