Relevant bibliographies by topics / Foot and mouth disease

Academic literature on the topic 'Foot and mouth disease'

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Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Foot and mouth disease"

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Kabele, Pavel, Martina Mojhová, and Dita Smíšková. "Hand-foot-mouth disease in puerperium." Česká gynekologie 87, no. 1 (February 22, 2022): 47–49. http://dx.doi.org/10.48095/cccg202247.

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Our case report describes a case of an otherwise predominantly childhood disease in a young adult woman with a good socioeconomic background who developed pruritic exanthema on the 2nd day after spontaneous delivery. The aim of the paper is to characterize the disease and to describe the possible risks for mother and child according to the available literature, as well as complications not only in puerperium but also during pregnancy. Key words: hand – foot – mouth disease – exanthema – gravidity – postpartum period – Coxsackie
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Upadhyay, Kavita. "Hand, Foot and Mouth Disease - A Short Case Report." Indian Journal of Youth & Adolescent Health 9, no. 1 (February 28, 2022): 18–19. http://dx.doi.org/10.24321/2349.2880.202203.

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Hand, foot and mouth disease, which was once considered a disease of cattle, has been emerging as a common human childhood disease in the last few years but is rare in adults. It is a viral disease characterised by a brief febrile illness and typical vesicular rashes. In rare cases, patients may also develop neurological complications. This report describes a case of hand, foot and mouth disease, presented with typical clinical features in the central Indian region.
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Elsayed Elmeligy, Elsayed Ebrahime. "Foot and Mouth Disease." SOJ Veterinary Sciences 3, no. 4 (August 4, 2017): 1–2. http://dx.doi.org/10.15226/2381-2907/3/4/00138.

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Grubman, Marvin J., and Barry Baxt. "Foot-and-Mouth Disease." Clinical Microbiology Reviews 17, no. 2 (April 2004): 465–93. http://dx.doi.org/10.1128/cmr.17.2.465-493.2004.

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SUMMARY Foot-and-mouth disease (FMD) is a highly contagious disease of cloven-hoofed animals. The disease was initially described in the 16th century and was the first animal pathogen identified as a virus. Recent FMD outbreaks in developed countries and their significant economic impact have increased the concern of governments worldwide. This review describes the reemergence of FMD in developed countries that had been disease free for many years and the effect that this has had on disease control strategies. The etiologic agent, FMD virus (FMDV), a member of the Picornaviridae family, is examined in detail at the genetic, structural, and biochemical levels and in terms of its antigenic diversity. The virus replication cycle, including virus-receptor interactions as well as unique aspects of virus translation and shutoff of host macromolecular synthesis, is discussed. This information has been the basis for the development of improved protocols to rapidly identify disease outbreaks, to differentiate vaccinated from infected animals, and to begin to identify and test novel vaccine candidates. Furthermore, this knowledge, coupled with the ability to manipulate FMDV genomes at the molecular level, has provided the framework for examination of disease pathogenesis and the development of a more complete understanding of the virus and host factors involved.
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SALLADAY, SUSAN A. "Foot-in-mouth disease." Nursing 35, no. 4 (April 2005): 67. http://dx.doi.org/10.1097/00152193-200504000-00048.

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Davies, Gareth. "Foot and mouth disease." Research in Veterinary Science 73, no. 3 (December 2002): 195–99. http://dx.doi.org/10.1016/s0034-5288(02)00105-4.

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Lubroth, Juan. "Foot-and-mouth disease." Veterinary Clinics of North America: Food Animal Practice 18, no. 3 (November 2002): 475–99. http://dx.doi.org/10.1016/s0749-0720(02)00036-1.

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Shaila, M. S. "Eradication of foot-and-mouth disease: a foot in mouth proposition." Journal of Biosciences 26, no. 2 (June 2001): 125–26. http://dx.doi.org/10.1007/bf02703634.

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Rani, J. Emy Jancy. "Hand, foot and mouth disease." Asian Journal of Nursing Education and Research 10, no. 4 (2020): 495–98. http://dx.doi.org/10.5958/2349-2996.2020.00106.8.

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Kushner, D., and BD Caldwell. "Hand-foot-and-mouth disease." Journal of the American Podiatric Medical Association 86, no. 6 (June 1, 1996): 257–59. http://dx.doi.org/10.7547/87507315-86-6-257.

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Hand-foot-and-mouth disease is a highly contagious disease most often seen in children during the summer. It is caused most commonly by the virus coxsackie A16, but other enteroviruses have been implicated. It presents with low grade fever, and a vesicular eruption on the hands, feet, and mouth. More serious manifestations are seen less commonly. Diagnosis is most often clinical and treatment is symptomatic in nature. The infection in a male adult is presented.
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Dissertations / Theses on the topic "Foot and mouth disease"

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Nayak, Arabinda. "Foot and mouth disease virus RNA replication." Thesis, University of Surrey, 2005. http://epubs.surrey.ac.uk/842873/.

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Infection of susceptible cells with foot and mouth disease virus (FMDV) results in multiplication of the RNA genome and assembly of mature virions. The entire process of genome replication is completed in a few hours and encompasses many intracellular events. Like other picornaviruses, FMDV uses a peptide primed RNA replication mechanism. The factors that are required to uridylylate each of the three FMDV VPg peptides and the role of the FMDV cis-acting replication element (cre) or 3B Uridylylation Site (bus) in VPg uridylylation have been determined. The native N-terminus of the FMDV 3Dpol enzyme is a pre-requisite for VPg uridylylation in vitro and the effects of mutations in the RNA template are consistent with a slide-back mechanism. The role of the poly(A) tail in uridylylating VPg was insignificant using full-length FMDV RNA transcripts suggesting the possibility of an alternative mechanism of VPg incorporation into negative strand RNA. The optimal RNA sequences required for VPg uridylylation were found to be within the 5' non-coding region (NCR). Furthermore, the results also showed evidence for RNA-RNA interactions between distinct structures from within the 5' NCR that influence VPg uridylylation. The polymerase precursor 3CDpro is also a prerequisite for uridylylation of each of the FMDV VPg peptides. However BCpro alone can substitute for 3 CD, but is much less efficient. It also appeared that the overall charge of the VPg peptides determines their recognition by the FMDV 3Dpol. The RNA binding activity of the 3C was found to be required for its stimulatory effects on VPg uridylylation. Unlike the poliovirus cloverleaf, the FMDV S-fragment (at the 5' end of the genome) does not interact with the FMDV 3CD precursor protein; however it binds specifically to a cellular factor p48. The crude replication complexes (CRCs) isolated from FMDV-infected cells were found to synthesize viral RNA very efficiently and an in vitro RNA replication system developed using these CRCs can be used to study the complete RNA replication events of FMDV.
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Foster-Cuevas, Mildred. "Immunodeterminants of foot-and-mouth disease virus." Thesis, University of Hertfordshire, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338562.

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Puig, Arturo. "Lipopeptide vaccines against foot and mouth disease." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428103.

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Cottam, Eleanor Myfanwy. "Micro-evolution of foot-and-mouth disease virus." Thesis, Connect to e-thesis. Move to record for print version, 2008. http://theses.gla.ac.uk/92/.

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Thesis (Ph.D.) - University of Glasgow, 2008.
Ph.D. thesis submitted to the Division of Biochemistry and Molecular Biology, Institute of Biomedical and Life Sciences, University of Glasgow, 2008. Includes bibliographical references. Print version also available.
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Lea, Susan Mary. "Structural studies on foot-and-mouth disease virus." Thesis, University of Oxford, 1993. http://ora.ox.ac.uk/objects/uuid:438dc0ae-b899-40fd-84dc-03d3fc1a537f.

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Foot-and-mouth disease viruses (FMDVs) constitute the aphthovirus genus of the Picornaviridae. The structures of Oi subtype viruses OiK and G67 have been solved and comparisons reveal the structural basis of monoclonal antibody escape mutations in G67. Escape mutations are seen to occur at surface-exposed residues and to provoke structural changes limited to the altered side chains. Comparisons of the structures of O1 and O1BFS (Acharya et al., Nature 337, 709-716 (1989)) suggest that changes occurring 'in-the-field' in response to polyclonal antibody pressure may be subtly different from mutations produced by monoclonal antibody pressure in vitro. Field mutations are seen to alter less exposed residues and to have more far-reaching structural effects than the in vitro, monoclonal provoked mutations. Crystals of G67 are seen to be 'intimately twinned', the data possessing extra symmetry due to a mis-packing of the crystals. A protocol, based on current real-space averaging procedures with a novel constraint imposed, has been used successfully to deconvolute these data. This method might be more generally applied to deconvolute the wavelength overlaps that occur when using the Laue method. The structures of C-S8cl and mutant SD6-6 have been solved at a resolution of 3.5Å. These structures enable comparisons between members of different FMDV serotypes to be made for the first time, namely: serotype 0 (O1BFS) and serotype C (C-S8cl). Flexibility of the Arg-Gly-Asp containing G-H loop of VP1 is seen to be amongst the most conserved structural features. This loop is implicated in receptor binding and possible roles for the observed flexibility are discussed. The CS8cl structure also reveals more detail in previously disordered regions of the capsid, namely: the N-terminal residues of VP2 and potential myristate density under the 5-fold axis of the virion. Analysis of structures from the Protein Data Bank reveals different patterns of amino acid use in proteins involved in the two halves of the immune recognition event i.e. immunoglobulins and viruses. These patterns seem to be based not only on the characteristics of the most used amino acids but also on characteristics of the nucleotide codons used to code for them.
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Edacheril, Mathew. "Assessment of herd immunity to foot-and-mouth disease." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.314315.

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Mahdi, Ali Jafar. "Foot and mouth disease in Iraq: strategy and control." Kansas State University, 2010. http://hdl.handle.net/2097/4620.

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Master of Science
Department of Diagnostic Medicine/Pathobiology
Gary A. Anderson
Foot-and-mouth disease (FMD) is a highly infectious viral disease of cattle, pigs, sheep, goats, buffalo, and artiodactyl wildlife species. Foot-and-mouth disease virus is endemic and periodic devastating epidemics have occurred and caused heavy economic losses in Iraq for a long time. The first official cases of FMD were recorded in 1937, while the first record of a specific FMD serotype in Iraq was serotype A in 1952. Other serotypes have been reported since then; serotypes O, SAT-1 and Asia1 were recorded in 1957, 1962, and 1975, respectively. Veterinary Services in Iraq has been severely weakened over the past two decades, and its infrastructure has been devastated as a consequence of previous political conflicts, wars and international sanctions. The breakdown of Veterinary Services led to the disruption of disease control strategies, collapse of disease surveillance and monitoring, and weakening of response systems. The destruction of the Al-Dora FMD laboratories for diagnosis and vaccine production by the United Nation in 1996, and the restrictions placed on the importation of vaccines have strongly affected the FMD control program. A severe epidemic of FMD occurred in Iraq in 1998, affecting 2.5 million ruminants and causing heavy losses in newly born animals. It is estimated to have killed about 550,000 animals. The outbreak was due to the serotype O1 Middle East strain which has affected large and small ruminants. In 2009, Iraq was severely affected by new serotype A (subtype A Iran 05). The major efforts of Veterinary Services in Iraq have been directed towards control of FMD by vaccination strategies. Two types of vaccine have been used, trivalent vaccine (O, A 22, and Asia 1) for cattle and buffalo and monovalent vaccine (O Manisa) for sheep and goats. Vaccination has been implemented once yearly on a voluntary basis. Sometimes other limited control measures have accompanied vaccination, which include quarantine, movement control, focused vaccination, disinfection, and public awareness programs. The FMD control program in Iraq has been confronted by many challenges: deficits in FMD surveillance and emergency preparedness, limited diagnostic capabilities, difficulties in restricting animal movement, and lack and irregular supply of appropriate vaccines.
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Будаєва, І. В., and Г. О. Ревенко. "Hand-foot-and-mouth disease (HFMD) у Дніпропетровському регіоні." Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/64722.

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Ентеровірусний везикулярний стоматит з екзантемою (Hand-Foot-and-Mouth Disease (HFMD)) - захворювання, що перебігає з ураженням шкіри кистей і стоп, слизової оболонки порожнини рота. Ентеровіруси мають тропність до нервової та серцево-судинної системи.
Clinical and epidemiological particularities of HFMD in children are presented. Considering cardiotropic features enteroviruses ECG and CFK-MB are evaluated in 14 children. Signs of myocarditis are revealed.
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Ramanoon, Siti. "The epidemiology of foot and mouth disease in Malaysia." Thesis, Ramanoon, Siti (2016) The epidemiology of foot and mouth disease in Malaysia. PhD thesis, Murdoch University, 2016. https://researchrepository.murdoch.edu.au/id/eprint/32386/.

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The objectives of the present study were to determine the prevalence of foot and mouth disease (FMD) and the serotypes of foot and mouth disease virus (FMDV) in Malaysia; to describe the temporal and spatial distribution of FMD in Malaysia; to evaluate the risk of the introduction of FMD to Malaysia; to evaluate the effectiveness of mitigation strategies adopted in Malaysia during outbreaks of FMD; and ultimately to give recommendations on FMD control to the Malaysia-Thailand-Myanmar (MTM) Tri-state Commission and Zoning Working Groups. The first documented outbreak of FMD in Peninsular Malaysia was in the 1860s and, although there have been periods where no outbreaks have been reported, the disease is now endemic in Peninsular Malaysia. Serotypes A, O and Asia 1 have been involved in the outbreaks of FMD in Peninsular Malaysia. In contrast the states of Sabah and Sarawak, located on Borneo have never had a reported outbreak of FMD. The virus strains involved in outbreaks in Peninsular Malaysia are closely related to those in southern Thailand and outbreaks have occurred in both countries at similar times. A total of 622 outbreaks of FMD were reported between 2001 and 2011 in Peninsular Malaysia. Serotype O was responsible for 92% of the 253 outbreaks serotyped and A in 8%. The number of outbreaks of FMD differed significantly between years (χ2=621, P<0.01, df=10), month (χ2=621, P<0.01, df=11) and states (χ2=621, P<0.01, df=10). The highest number of outbreaks (110) occurred in 2009, while the lowest number (23) was in 2007. The monthly outbreak pattern of FMD showed a significant increase during the northeast monsoon season (November to March). It is hypothesised that the seasonal increase in outbreaks was related to animal movement to meet the demand for fresh meat for cultural events held during those months. Outbreaks of FMD in Peninsular Malaysia were most prevalent in Kedah (n=95). There was a significant correlation between the number of outbreaks and the average population size of cattle (r=0.731, P=0.007), buffalo (r=0.625, P=0.03) and goats (r=0.652, P=0.021). Cattle were involved in most outbreaks (87%). The overall prevalence of clinical disease was highest in cattle (15.4%), followed by buffalo (9.9%), goats (6.8%), sheep (6.6%) and pigs (6.5%). Cattle (odds ratio, OR=2.6, 95% CI 2.5, 2.8) and buffalo (OR=1.6, 95% CI 1.4, 1.8) were significantly more likely to be reported with clinical signs of disease than pigs. The case-fatality rate in cattle was 0.2% and goats 0.5%. The main sources of outbreaks were hypothesised to be the introduction of new animals or the illegal movement of animals (66% of outbreaks). A combination of control measures, including ring vaccination, animal movement management and quarantine, were implemented during outbreaks. The animal seroprevalence of FMD by the NSP test in cattle, buffalo, goats and sheep were 24.2% (95% CI: 23.8, 24.6), 52.7% (95% CI: 50.5, 55), 11.8% (95% CI: 11, 12.6) and 9.5% (95% CI: 6.9, 12.6), respectively. These findings indicate natural infection and provide evidence that the virus was circulating in the livestock population. Males were 2.2 (OR 95% CI: 2.04, 2.3) times more likely to be NSP positive than were females. Cattle belonging to the Murrah breed (OR=1.6, 95% CI: 1.3, 2.0) and dairy breeds (OR=1.3, 95% CI: 1.2, 1.5) were more likely to be seropositive than were meat breeds. The whole peninsula was infected with FMD (range of point prevalences 6 to 37% in Kuala Lumpur and Kelantan, respectively). In cattle, the overall seroprevalence based on the liquid phase blocking ELISA titre (LPBET) (positive titre >45) for type O (n=3025) was 74% (95% CI: 72, 76) and 52% (95% CI: 50, 53) of animals had protective titres (titre > 90). The findings indicate that, on average, the protective levels of antibody to type O in cattle were below the recommended level. In imported cattle (n=3295), more than 90% were positive on the LPBE percentage inhibition (PI) to type O and 49% were NSP positive indicating that they had been vaccinated and also exposed to a natural infection. This finding highlights that infected imported cattle may be an important source of FMD outbreaks, thus livestock consignments should be closely monitored. Based on the results of the LPBET adequate protective levels were present to serotype O in buffalo but not in goats, sheep or pigs. The results from the simulation study for the risk of introduction of FMD via importation of live cattle from Thailand showed that there is almost a 100% probability that there will be at least one infectious animal that is capable of transmitting infection being accepted for importation into Peninsular Malaysia in any given year. The estimated total number of outbreaks was 26 per year (range: 10-182; 95% CI: 1, 27) and the probability of outbreaks following effective contact was 92%. This indicates that importation of live cattle from Thailand is a strong factor contributing to the likelihood of outbreaks in Peninsular Malaysia. Furthermore, Malaysia is at continuous risk as long as the importation of live animals continues from infected countries. To reduce risk, interventions, including pre-arrival testing, vaccination and improved farm biosecurity, should be adopted on farms, irrespective if the new animals are from another country or from within Peninsular Malaysia. In conclusion, FMD is endemic in Peninsular Malaysia and movement of animals plays a major role in the spread of FMD. The protective level of immunity induced by vaccination was below the recommended level. Imported live cattle from Thailand could be infected with FMD and a potential source for introducing new strains of virus to Peninsular Malaysia. Therefore, studies should be conducted to trace the farms of origin of these imported cattle. The findings from this study could be used to improve the existing control strategy for FMD in Peninsular Malaysia thus ultimately underpinning the MTM FMD Campaign.
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Howes, Emma Louise. "Investigating the foot-and-mouth disease virus 3A protein." Thesis, University of St Andrews, 2018. http://hdl.handle.net/10023/15521.

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Foot-and-Mouth Disease Virus (FMDV) is a globally important pathogen responsible for causing Foot-and-Mouth Disease (FMD) in wildlife and domestic livestock species and has significant economic impacts. FMD is difficult to control due to its highly infectious nature, wide diversity of host species and the existence of multiple serotypes; therefore, understanding the processes of FMDV infection and viral RNA replication are key to the development of improved diagnostics and vaccines. This thesis investigates the potential roles of the FMDV 3A non-structural protein using a combination of sub-genomic replicons, recombinant viruses and proteomics techniques. The picornavirus 3A protein has previously been linked with roles in replication complex formation, virulence and determining viral host range. This thesis presents findings showing that a naturally occurring deletion in 3A had differing effects on replication in cells lines derived from different natural hosts thereby supporting the conclusion that 3A has an important role in viral host range. Proteomic (immunoprecipitation and mass spectroscopy) investigations were carried out to identify potential cellular interaction partners of FMDV 3A, and the impact on infection and replication of reducing expression of two selected cellular proteins Rab7L1 and TBC1D20 was investigated. The 3A protein of FMDV was shown to include a conserved FFAT motif (which bind the ER resident protein VAP) in its N terminal domain. A role for this motif was also investigated with the results suggesting that the 3A FFAT motif is important for efficient viral replication. Finally, the potential role of 3A to act as the donor of 3B during replication was investigated. Key findings from experiments using FMDV replicons and recombinant viruses showed that full-length P3 and the processing intermediate 3ABBB are not required for viral RNA replication suggesting that the preferred donor of 3B for uridylation is likely a 3BC containing precursor protein.
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Books on the topic "Foot and mouth disease"

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J, Rowlands David, ed. Foot-and-mouth disease. Amsterdam: Elsevier, 2003.

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van der Zijpp, A. J., M. J. E. Braker, C. H. A. M. Eilers, H. Kieft, T. A. Vogelzang, and S. J. Oosting. Foot and Mouth Disease. The Netherlands: Wageningen Academic Publishers, 2004. http://dx.doi.org/10.3920/978-90-8686-530-7.

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International Office of Epizootics. Foot and Mouth Disease Commission Conference. Foot and mouth disease. Paris: Office Intrernational des Epizooties, 1987.

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J, Rowlands D., ed. Foot-and-mouth disease. Amsterdam: Elsevier, 2003.

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Barclay, Christopher. Foot and mouth disease. London: House of Commons Library, 2001.

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J, Rowlands D., ed. Foot-and-mouth disease. Amsterdam: Elsevier, 2003.

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W, Compans Richard, ed. Foot-and-mouth disease. Berlin: Springer, 2005.

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Mahy, Brian W. J., ed. Foot-and-Mouth Disease Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/b138628.

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Francisco, Sobrino, and Domingo Esteban, eds. Foot and mouth disease: Current perspectives. Wymondham, Norfolk, England: Horizon Bioscience, 2004.

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Trades Union Congress. Economic and Social Affairs Department. Foot and mouth and benefits. [London]: TUC ESAD, 2001.

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Book chapters on the topic "Foot and mouth disease"

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Ruiz, Vanesa, and Andrés Wigdorovitz. "Foot-and-mouth Disease." In Prospects of Plant-Based Vaccines in Veterinary Medicine, 311–43. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90137-4_15.

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Lu, Puxuan, and BoPing Zhou. "Hand-Foot-Mouth Disease." In Radiology of Infectious Diseases: Volume 1, 127–55. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9882-2_17.

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Grubman, Marvin J., Luis L. Rodriguez, and Teresa de los Santos. "Foot-and-Mouth Disease." In The Picornaviruses, 397–410. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816698.ch25.

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Sebhatu, Tesfaalem Tekleghiorghis. "Foot-and-Mouth Disease." In Transboundary Animal Diseases in Sahelian Africa and Connected Regions, 207–31. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25385-1_11.

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van der Wal, Jacqueline E. "Hand-Foot-and-Mouth Disease." In Encyclopedia of Pathology, 1–2. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28845-1_727-1.

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Jing, Yuan, Huang Wen-Xian, Zeng Hong-Wu, Li Jian-Ming, Ou Shan-Xing, Gou Ji-zhou, Yang Guang, Zheng Guang-Ping, Shan Wan-Shui, and Lou Ming-Wu. "Hand-Foot-and-Mouth Disease." In Diagnostic Imaging of Emerging Infectious Diseases, 153–68. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-017-7363-8_7.

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Reich, Danya, Corinna Eleni Psomadakis, and Bobby Buka. "Hand, Foot, and Mouth Disease." In Top 50 Dermatology Case Studies for Primary Care, 321–29. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-18627-6_48.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Hand-Foot-and-Mouth Disease." In Encyclopedia of Molecular Mechanisms of Disease, 771. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_3375.

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van der Wal, Jacqueline E. "Hand-Foot-and-Mouth Disease." In Encyclopedia of Soil Science, 208–9. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-3-319-28085-1_727.

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Bauer, K. "Foot-and-mouth disease as zoonosis." In Viral Zoonoses and Food of Animal Origin, 95–97. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6534-8_9.

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Conference papers on the topic "Foot and mouth disease"

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J Oates, Briony. "Foot and Mouth Disease: Informing the Community?" In 2002 Informing Science + IT Education Conference. Informing Science Institute, 2002. http://dx.doi.org/10.28945/2550.

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The 2001 foot and mouth disease (FMD) outbreak in the UK had a significant impact on the economic and social wellbeing of rural communities. This paper examines the FMD pages of four local government websites in Northern England: Cumbria, Durham, Northumberland and North Yorkshire County Councils. Each county was badly affected by FMD. The contents of the FMD webpages are analysed and compared: which audiences were addressed, what information was provided or omitted, and how well the audiences’ needs were met. The study shows the breadth of audience types and information that could have been included, but no site covered all the necessary angles. Furthermore, the websites did little to address the psychological problems arising from FMD or to enhance participation and democracy in their local communities. By examining how the councils informed those affected, lessons can be learnt which are relevant to any future disruption to a community.
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Khammadov, N. I., A. I. Khamidullina, K. V. Usoltsev, and T. Kh Faizov. "GENETIC POLYMORPHISMS OF FOOT AND MOUTH DISEASE VIRUS." In Molecular Diagnostics and Biosafety. Federal Budget Institute of Science 'Central Research Institute for Epidemiology', 2020. http://dx.doi.org/10.36233/978-5-9900432-9-9-247.

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Aryan, Mohammad Farhad, Worarat Krathu, Chonlameth Arpnikanondt, and Boonrat Tassaneetrithep. "Image Recognition for Detecting Hand Foot and Mouth Disease." In IAIT2020: The 11th International Conference on Advances in Information Technology. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3406601.3406640.

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Wang, Jiaojiao, Jinglu Chen, Quannan Zu, Zhidong Cao, Saike He, and Daniel Dajun Zeng. "Healthcare-seeking behavior study on Beijing Hand-Foot-Mouth Disease Patients." In 2019 IEEE International Conference on Intelligence and Security Informatics (ISI). IEEE, 2019. http://dx.doi.org/10.1109/isi.2019.8823283.

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Abe, Maiku. "Risk-Sharing Model Of Foot-and-Mouth Disease Outbreak in Japan." In 2019 IEEE 8th Global Conference on Consumer Electronics (GCCE). IEEE, 2019. http://dx.doi.org/10.1109/gcce46687.2019.9015512.

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Pongsumpun, Puntani, and Napasool Wongvanich. "Age Structural Model of the Hand Foot Mouth Disease in Thailand." In 2018 2nd European Conference on Electrical Engineering and Computer Science (EECS). IEEE, 2018. http://dx.doi.org/10.1109/eecs.2018.00033.

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Longinotti, G., G. Ybarra, P. Lloret, C. Moina, A. Ciochinni, D. R. Serantes, L. Malatto, M. Roberti, S. Tropea, and L. Fraigi. "Diagnosis of foot-and-mouth disease by electrochemical enzyme-linked immunoassay." In 2010 32nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC 2010). IEEE, 2010. http://dx.doi.org/10.1109/iembs.2010.5626230.

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Matsushima, Masatomo, and Hironori Matsushima. "Infectious pattern of foot-and-mouth disease and the modified SIR model." In CENTRAL EUROPEAN SYMPOSIUM ON THERMOPHYSICS 2019 (CEST). AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5114538.

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Zhang, Pengli, Yingzhu Wei, and Tian Tang. "Transmission pattern and climatic effects as for the hand-foot-mouth disease." In 2021 International Conference on Public Health and Data Science (ICPHDS). IEEE, 2021. http://dx.doi.org/10.1109/icphds53608.2021.00061.

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Carvalho, Luiz Max F., Leonardo Bacelar Lima Santos, Paulo E. P. Burke, Marcos Quiles, and Waldemir de Castro Silveira. "A geographically-aware complex network approach for foot-and-mouth disease phylodynamics." In 6th International Conference on Nonlinear Science and Complexity. São José dos Campos, Brazil: INPE Instituto Nacional de Pesquisas Espaciais, 2016. http://dx.doi.org/10.20906/cps/nsc2016-0047.

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Reports on the topic "Foot and mouth disease"

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Hullinger, P. New England Foot and Mouth Disease Tabletop Exercise. Office of Scientific and Technical Information (OSTI), September 2008. http://dx.doi.org/10.2172/945849.

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Grubman, Marvin J., Yehuda Stram, Peter W. Mason, and Hagai Yadin. Development of an Empty Viral Capsid Vaccine against Foot and Mouth Disease. United States Department of Agriculture, August 1995. http://dx.doi.org/10.32747/1995.7570568.bard.

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Foot-and-mouth disease (FMD), a highly infectious viral disease of cloven-hoofed animals, is economically the most important disease of domestic animals. Although inactivated FMD vaccines have been succesfully used as part of comprehensive eradication programs in Western Europe, there are a number of concerns about their safety. In this proposal, we have attempted to develop a new generation of FMD vaccines that addresses these concerns. Specifically we have cloned the region of the viral genome coding for the structural proteins and the proteinase responsible for processing of the structural protein precursor into both a DNA vector and a replication-deficient human adenovirus. We have demonstrated the induction of an FMDV-specific immune response and a neutralizing antibody response with the DNA vectors in mice, but preliminary potency and efficacy studies in swine are variable. However, the adenovirus vector induces a significant and long-lived neutralizing antibody response in mice and most importantly a neutralizing and protective response in swine. These results suggest that the empty capsid approach is a potential alternative to the current vaccination strategy.
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M., BLAKE. Reflections on the Foot-and-Mouth Disease Epidemic of 2001: an Irish Perspective. O.I.E (World Organisation for Animal Health), April 2021. http://dx.doi.org/10.20506/bull.2021.nf.3165.

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This year marks the 20-year anniversary of the foot-and-mouth disease (FMD) epidemic, which originated in the United Kingdom (UK) in February 2001, and subsequently spread to Ireland, the Netherlands and France.
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Kostova-Vassilevska, T. On The Use Of Models To Assess Foot-And-Mouth Disease Transmission And Control. Office of Scientific and Technical Information (OSTI), July 2004. http://dx.doi.org/10.2172/15014467.

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C., MIDDLEMISS. Reflections on the Foot-and-Mouth Disease Epidemic of 2001: a United Kingdom Perspective. O.I.E (World Organisation for Animal Health), April 2021. http://dx.doi.org/10.20506/bull.2021.nf.3166.

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Thanda Kyaw, Ai. Socio-Economic Impacts of Foot and Mouth Disease Among Cattle Farmers in Sagaing and Mandalay Areas, Myanmar. O.I.E (World Organisation for Animal Health), May 2014. http://dx.doi.org/10.20506/standz.2784.

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The World Organisation for Animal Health (OIE) Sub-Regional Representation for South East Asia (OIE SRR-SEA) implemented the Stop Transboundary Animal Diseases and Zoonoses (STANDZ) Programme funded by AusAID to strengthen the veterinary services and effectively manage the control and eradication of foot and mouth disease (FMD) in Cambodia, Lao PDR and Myanmar. The purpose of the study is to understand how FMD outbreaks impact smallholder farmers, both men and women, at the household and village level and how control and eradication of FMD would benefit them. Specific aims are to estimate the direct and indirect socio-economic costs associated with the outbreaks of FMD as well as of the measures taken by farmers to deal with such outbreaks and to identify issues that contributed to the socio-economic impacts of FMD outbreaks and opportunities to reduce them.
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Mwebe, Robert, Chester Kalinda, Ekwaro A. Obuku, Eve Namisango, Alison A. Kinengyere, Moses Ocan, Ann Nanteza, Savino Biryomumaisho, and Lawrence Mugisha. Epidemiology and effectiveness of interventions for Foot and Mouth Disease in Africa: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0039.

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Review question / Objective: What is the epidemiology and effectiveness of control measures for foot and mouth disease in African countries?’ PICOS: Description of elements Population/ problem/Setting: Artiodactyla (cloven ungulates), domestic (cattle, sheep, goats, and pigs), camels and wildlife (buffaloes, deer, antelope, wild pigs, elephant, giraffe, and camelids) affected by Foot and Mouth Disease (FMD) or Hoof and Mouth Disease (HMD) caused by the Foot and Mouth Disease Virus (FMDV) in Africa. Intervention: Prevention measures: vaccination, ‘biosafety and biosecurity’, sensitization of the public. Control measures: quarantine, movement control, closure of markets and stock routes, mouth swabbing of animals with infected materials (old technique that is no long applicable), culling, mass slaughter, stamping out and any other interventions or control measures generally accepted by the ‘community of practice’ of animal health practitioners. Comparator: areas that did not have any control activities for FMD, in head-to-head comparisons in the same study. Outcome: epidemiological outcomes: incidence, prevalence, patterns or trends, clinical symptoms, and risk factors. Effectiveness outcomes: success, and usefulness of the interventions measured as averted deaths, illness and infections, and costs associated with the interventions (cost–effectiveness). Study design: epidemiological designs include cohort design for incidence, cross sectional for prevalence and case-control for clinical symptoms and risk factors. Interventional designs include randomized controlled trials, cluster randomized trials, quasi-experimental designs – controlled before and after, interrupted time series, [regression discontinuity design, difference-in-difference, and propensity score matching]. Timelines: 1900 – 2022.
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McLeod, Ross. Costs of FMD in SE Asia and economic benefits of the Southeast Asia Foot and Mouth Disease Campaign. O.I.E (World Organisation for Animal Health), July 2013. http://dx.doi.org/10.20506/standz.2780.

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Becker, Yechiel, D. M. Moore, and Hagai Yadin. Diagnosis of Foot and Mouth Disease Virus by Cloning, Sequencing and Monoclonal Antibodies to VP1 of Israeli Serotypes. United States Department of Agriculture, September 1987. http://dx.doi.org/10.32747/1987.7568079.bard.

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Frieson, Kate Grace. A Gender Assessment of SEACFMD 2020: A Roadmap to Prevent, Control and Eradicate foot and mouth disease (by 2020) in Southeast Asia and China. O.I.E (World Organisation for Animal Health), December 2013. http://dx.doi.org/10.20506/standz.2785.

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This gender assessment of SEACFMD 2020: A Roadmap to Prevent, Control and Eradicate foot and mouth disease (by 2020) in Southeast Asia and China, responds to the requirement of AusAID that all strategies affecting human health, food security and poverty alleviation incorporate a gender perspective as women are not often included in the technical and community based aspects of programs relating to animal health and disease control. Gender roles and responsibilities affect women’s and men’s ability and incentive to participate in FMD roadmap activities, and can potentially lead to different project impacts for men and women.
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