Academic literature on the topic 'Foetal mouse'
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Journal articles on the topic "Foetal mouse"
Meek, Jennifer, and Eileen D. Adamson. "Transferrin in foetal and adult mouse tissues: synthesis, storage and secretion." Development 86, no. 1 (April 1, 1985): 205–18. http://dx.doi.org/10.1242/dev.86.1.205.
Full textCeredig, R. "Mouse foetal thymus development." Research in Immunology 141, no. 3 (1990): 286–89. http://dx.doi.org/10.1016/0923-2494(90)90125-i.
Full textBro-Rasmussen, Frede, and O. Henriksen. "Foetal Erythropoiesis in the Mouse." Scandinavian Journal of Haematology 1, no. 1 (April 24, 2009): 26–37. http://dx.doi.org/10.1111/j.1600-0609.1964.tb00003.x.
Full textKershaw, T. R., and J. D. Sinden. "Survival of Foetal Neural Tissue from the H-2Kb-tsA58 Transgenic Mouse Grafted to Adult Mouse Brain." Cell Transplantation 2, no. 3 (May 1993): 215–22. http://dx.doi.org/10.1177/096368979300200305.
Full textReiksson, Margareta. "SALICYLATE-INDUCED FOETAL HAEMORRHAGE IN TWO MOUSE STRAINS." Acta Pathologica Microbiologica Scandinavica 76, no. 2 (August 18, 2009): 164–70. http://dx.doi.org/10.1111/j.1699-0463.1969.tb03247.x.
Full textCarroll, Mark, and Margaret M. Bird. "Glycoprotein expression in foetal and adult mouse cerebellum." Comparative Biochemistry and Physiology Part B: Comparative Biochemistry 95, no. 4 (January 1990): 855–60. http://dx.doi.org/10.1016/0305-0491(90)90328-q.
Full textMcCloghry, CE, DE Hollis, A. Foldes, AJ Rintoul, P. Baker, JD Vaughan, CA Maxwell, JP Kennedy, and PC Wynn. "The effects of exogenous melatonin and prolactin on wool follicle development in ovine foetal skin grafts." Australian Journal of Agricultural Research 44, no. 5 (1993): 993. http://dx.doi.org/10.1071/ar9930993.
Full textAirey, Chris J., Phoebe J. Smith, Katie Restall, Stephanie J. Marfy‐Smith, Tom P. Fleming, and Sandrine Willaime‐Morawek. "ISDN2014_0241: Maternal undernutrition affects neurogenesis in the foetal mouse brain." International Journal of Developmental Neuroscience 47, Part_A (December 2015): 72. http://dx.doi.org/10.1016/j.ijdevneu.2015.04.197.
Full textKovarik, Jiri, Maria Koulmanda, and Thomas E. Mandel. "The role of cytokines during rejection of foetal pig and foetal mouse pancreas grafts in nonobese diabetic mice." Transplant Immunology 5, no. 4 (December 1997): 307–14. http://dx.doi.org/10.1016/s0966-3274(97)80014-7.
Full textSAIDA, Kaname, Naoko KOMETANI, Tsuyoshi UCHIDE, and Youji MITSUI. "Sequence analysis and expression of the mouse full-length vasoactive intestinal contractor/endothelin-2 gene (EDN2): comparison with the endothelin-1 gene (EDN1)." Clinical Science 103, s2002 (September 1, 2002): 84S—89S. http://dx.doi.org/10.1042/cs103s084s.
Full textDissertations / Theses on the topic "Foetal mouse"
Morris, L. "Expression of surface molecules on mouse foetal macrophages." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235069.
Full textZhao, Wanfeng. "Development and differentiation of oesophageal muscle in mouse." Thesis, Royal Veterinary College (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367759.
Full textNorth, J. "The effects of soluble factors on thymocyte differentiation in the foetal mouse thymus." Thesis, University of Newcastle Upon Tyne, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375161.
Full textTollet, Cecilia Jenny. "The origin and early development of the intrinsic innervation in the foetal mouse lung." University of Western Australia. School of Biomedical and Chemical Sciences, 2003. http://theses.library.uwa.edu.au/adt-WU2004.0060.
Full textShaw, Michael Jan. "Foetal forebrain development in the trisomy 16 mouse : a possible animal model of Down's syndrome." Thesis, St George's, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325345.
Full textCarey, Luke Charles. "Maternal ethanol exposure in the metallothionein+/+ mouse is associated with reduced foetal zinc and increased abnormalities /." Title page and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09SB/09sbc274.pdf.
Full textButruille, Laura. "Altération de la croissance fœtale et programmation métabolique : étude de l’implication des Rho-kinases et du système apelinergique chez les rongeurs." Thesis, Lille 2, 2013. http://www.theses.fr/2013LIL2S015/document.
Full textDuring the last decade, many epidemiological studies have shown that adult chronic metabolic (obesity, diabetes) and cardiovascular diseases may be determined, at least in part, during pregnancy through alterations of intrauterine environment. The “fetal programming” hypothesis implies that disturbances of the fetal development (intra uterine growth restriction – IUGR or macrosomia) increase the vulnerability to develop these pathologies in adulthood. To gain more insight into the mechanisms implicated in fetal programming, we used two experimental models of rodents (rat, mouse) and evaluated first the effect of an inhibition of the Rho-kinase pathway in utero on fetal growth and postnatal development in rats. In another study performed in mice, we aimed to assess the expression of apelin and its receptor APJ in obese and glucose intolerant mice fed with a high fat diet. Using data of this preliminary study, we speculated that this signaling system may be targeted during the pregnancy of obese mothers and could be implicated into the physiopathological consequences that may affect the fetoplacental unit. We demonstrated that pregnant rats treated by L-NAME, a NO synthase inhibitor (50 mg/day) were hypertensive and that their newborns presented a dramatic IUGR. Maternal treatment with the vasodilator Fasudil (10 mg/day) restored a normal maternal blood pressure and remarkably alleviated the fetal growth of L-NAME newborns. In adults, L-NAME male rats developed mild metabolic pathologies whereas rats exposed in utero to Fasudil presented an overweight, with hyperphagia and glucose intolerance. In obese and glucose intolerant mice fed with a high fat diet, we showed that apelin gene expression was altered in several organs (liver, kidney and adipose tissue) without any variation of apelin plasma concentration. Further studies are currently performed in our laboratory to unravel the expression of the apelin/APJ pathway in pregnant obese mice and their offsprings
Roux, Marine. "Etude du rôle des gènes HOX dans le développement du cœur chez la souris." Thesis, Aix-Marseille, 2013. http://www.theses.fr/2013AIXM5086.
Full textHox genes are known to be involved in the establishment of cell position and identity along the anterior-posterior axis in embryos and could act as key downstream effectors of retinoic acid during heart development. In situ hybridization experiments show that Hoxb1, Hoxa1 and Hoxa3 define sub-domains within the second heart field (SHF). Our genetic lineage analysis reveals the contribution of Hoxb1+ cardiac progenitors to the atria and to the inferior wall of the outflow tract (OFT), which then gives rise to the myocardium at the base of the pulmonary trunk. Interestingly, Hoxa1+ progenitors contribute to the distal part of the OFT suggesting that these anterior Hox genes could play a role in its proximo-distal patterning. No cardiac anomalies had been reported so far in Hoxb1 mutant mice. However, our detailed study shows that mutant fetuses exhibit OFT misalignment and ventricular septal defects associated or not with ventricular wall and epicardium anomalies. Using a marker of the sub-pulmonary myocardium, we observe an abnormal contribution of SHF cells in Hoxb1-/- hearts. This defect is the consequence of the dysregulation of the signaling pathways controlling SHF regulation. Accordingly, those embryos exhibit a shorter OFT. The study of Hoxa1 mutant embryos reveals pharyngeal arch arteries patterning defects causing anomalies of the aortic arch and right subclavian artery at fetal stages. Using compound mutants, we show an increase in the penetrance and severity of these defects, suggesting a synergistic interaction between Hoxa1 and Hoxb1 during aortic arch patterning. Together, these data support a crucial role for anterior Hox genes in cardiac development
Porchet, Nicolas. "Role of signaling pathays in cell-fate specification in the early mouse embryo." Thesis, Université de Paris (2019-....), 2019. http://www.theses.fr/2019UNIP7096.
Full textDuring the early mouse embryogenesis, cell-fate specification events result in the formation of the pre-implantation blastocyst. Those events are mainly regulated by the action of signaling cascades activated upon fixation of the signaling molecules at the cell membrane. The activity of these signaling pathways allow the transcriptional regulation of a specific pool of genes responsible for cell-fate decisions and the formation of tissues. Here, I am interested in the roles of both ACTIVIN/NODAL and βCATENIN signaling pathways in the specification of cell identities during the maturation of the mouse blastocyst
Book chapters on the topic "Foetal mouse"
Coppen, Steven R., Riyaz A. Kaba, Deborah Halliday, Emmanuel Dupont, Jeremy N. Skepper, Suzy Elneil, and Nicholas J. Severs. "Comparison of connexin expression patterns in the developing mouse heart and human foetal heart." In Cardiac Cell Biology, 121–27. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4757-4712-6_16.
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