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Books on the topic 'Flaviviruses'

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Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

M, Chambers Thomas, ed. The flaviviruses. Oxford: Academic, 2004.

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2

M, Chambers Thomas, ed. The flaviviruses. Oxford: Academic, 2004.

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3

Sondra, Schlesinger, and Schlesinger Milton J, eds. The Togaviridae and Flaviviridae. New York: Plenum Press, 1986.

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4

Shi, Pei-Yong. Molecular virology and control of flaviviruses. Norfolk, UK: Caister Academic Press, 2012.

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5

Gregory, Bock, Goode Jamie, Novartis Foundation, and Novartis Institute for Tropical Diseases., eds. New treatment strategies for dengue and other flaviviral diseases. Chichester: John Wiley & Sons, 2006.

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6

Chambers, Thomas J. The Flaviviruses: Detection, Diagnosis and Vaccine Development. Burlington: Elsevier, 2003.

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7

M, Chambers Thomas, Monath Aaron J, Maramorosch Karl, Murphy Frederick A, and Shatkin Aaron J, eds. Advances in virus research. Amsterdam: Elsevier, 2004.

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8

M, Chambers Thomas, Monath Aaron J, Maramorosch Karl, Murphy Frederick A, and Shatkin Aaron J, eds. Advances in virus research. Amsterdam: Oxford, 2003.

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9

Schlesinger, Milton J., and Sondra Schlesinger. Togaviridae and Flaviviridae. Springer, 2012.

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10

Maramorosch, Karl, Frederick A. Murphy, Thomas P. Monath, Aaron J. Shatkin, and Thomas J. Chambers. Flaviviruses: Pathogenesis and Immunity. Elsevier Science & Technology Books, 2003.

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11

Gideon science GIDEON science team. Less-Common Flaviviruses: Global Status. Gideon Informatics, Incorporated, 2023.

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12

Maramorosch, Karl, Frederick A. Murphy, Thomas P. Monath, Aaron J. Shatkin, and Thomas J. Chambers. Flaviviruses: Structure, Replication and Evolution. Elsevier Science & Technology Books, 2003.

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13

Berger, Stephen, and Inc Gideon Informatics. Less-Common Flaviviruses: Global Status. Gideon Informatics, Incorporated, 2021.

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14

Berger, Stephen, and Inc Gideon Informatics. Less-Common Flaviviruses: Global Status. Gideon Informatics, Incorporated, 2022.

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15

Berger, Stephen, and Inc Gideon Informatics. Less-Common Flaviviruses: Global Status. Gideon Informatics, Incorporated, 2019.

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16

(Editor), Thomas J. Chambers, Thomas P. Monath (Editor), Karl Maramorosch (Series Editor), Aaron J. Shatkin (Series Editor), and Frederick A. Murphy (Series Editor), eds. The Flaviviruses: Structure, Replication and Evolution, Volume 59 (Advances in Virus Research). Academic Press, 2003.

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17

(Editor), Thomas J. Chambers, Thomas P. Monath (Editor), Karl Maramorosch (Series Editor), Aaron J. Shatkin (Series Editor), and Frederick A. Murphy (Series Editor), eds. The Flaviviruses: Pathogenesis and Immunity, Volume 60 (Advances in Virus Research). Academic Press, 2003.

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18

Schlesinger, Milton J., and Sondra Schlesinger. Togaviridae and Flaviviridae. Springer London, Limited, 2013.

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19

Zika and Other Neglected and Emerging Flaviviruses. Elsevier, 2021. http://dx.doi.org/10.1016/c2020-0-01482-9.

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20

The Flaviviruses: Detection, Diagnosis, and Vaccine Development. Elsevier, 2003. http://dx.doi.org/10.1016/s0065-3527(00)x0008-5.

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21

The Flaviviruses: Detection, Diagnosis and Vaccine Development, Volume 61 (Advances in Virus Research). Academic Press, 2003.

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22

Beltz, Lisa A. Zika and Other Neglected and Emerging Flaviviruses: The Continuing Threat to Human Health. Elsevier, 2021.

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23

Ruzek, Daniel, ed. Flavivirus Encephalitis. InTech, 2011. http://dx.doi.org/10.5772/847.

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24

Tkachev, Sergey, ed. Non-Flavivirus Encephalitis. InTech, 2011. http://dx.doi.org/10.5772/1740.

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25

Velásquez Serra, Glenda. Garrapatas: Vectores de Flavivirus. CIDEPRO EDITORIAL, 2019. http://dx.doi.org/10.29018/978-9942-823-09-0.

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26

Advances in Flavivirus Research. MDPI, 2017. http://dx.doi.org/10.3390/books978-3-03842-487-1.

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27

Molecular Biology of Flavivirus. Taylor & Francis, 2006.

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28

Keshav, Satish, and Palak Trivedi. Viral hepatitis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectively. These viruses are all hepatotrophic, in that the liver is the primary site of infection. HAV, HBV, and HEV are usually acute, self-limiting infections that may, nonetheless, cause morbidity and, in the case of HEV, fatality. However, HBV and, more so, HCV can cause chronic carriage of the virus over many years, as well as the development of chronic hepatitis. HDV is only pathogenic in conjunction with HBV. After recovery from acute infection with HAV, individuals have long-lasting immunity against further infection. The same holds true for the majority of individuals with acute HBV infection. There seems to be little natural immunity to HCV infection, and a significant proportion of cases result in chronic hepatitis. Immunity to HEV is not long-lasting, and repeated infections are possible. Many other viruses can cause hepatitis, of which cytomegalovirus, herpes simplex virus, Epstein–Barr virus, and flaviviruses such as dengue and yellow fever are the most important. The liver, however, is not their primary site of replication or cellular damage.

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29

Nuttall, Patricia A. Tick-borne encephalitides. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0044.

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Tick-borne encephalitides are caused by three different viruses transmitted by ticks and belonging to the Flaviviridae virus family: tick-borne encephalitis virus (Far Eastern, Siberian, and European subtypes), louping ill virus, and Powassan virus (including deer tick virus). These viruses cause encephalitis affecting humans in Eurasia and North America. In nature, they are maintained in transmission cycles involving Ixodes tick species and small or medium-sized wild mammals. The tick-borne flavivirus group is one of the most intensely studied groups of tick-borne pathogens.

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30

Monath, Thomas P., and J. Erin Staples. Yellow fever. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0045.

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Yellow fever is an acute mosquito-borne flavivirus infection characterized in its full-blown form by fever, jaundice, albuminuria, and haemorrhage. Two forms are distinguished: urban yellow fever in which the virus is spread from person to person by peridomestic Aedes aegypti mosquitoes and jungle (sylvan) yellow fever transmitted by tree-hole breeding mosquitoes between non-human primates and sometimes humans. Yellow fever is endemic and epidemic in tropical areas of the Americas and Africa but has never appeared in Asia or the Pacific region. Prevention and control are effected principally through yellow fever vaccination.

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31

Finn, Matthew. West Nile Virus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0053.

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West Nile virus (WNV) is a single-stranded RNA virus of the Flavivirus family that is transmitted via a mosquito vector, typically causing fever and capable of causing meningoencephalitis. Although mortality is low, it can lead to debilitating neuroinvasive disease in some patients. WNV is a leading cause of domestically-acquired arboviral disease and most commonly occurs in late August and early September. Consider WNV in otherwise unexplained cases of meningitis or encephalitis. Initial testing should consist of cerebrospinal fluid (CSF) analysis and West Nile immunoglobulin M enzyme-linked immunosorbent assay in serum and/or CSF. WNV is a nationally notifiable disease. Prevention remains the key to controlling this disease. Reducing the breeding grounds of the Culex mosquito and using insect repellant to prevent bites are two important strategies.

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32

Казачинская, Е. И. ВИРУС ДЕНГЕ. Академическое изд-во «Гео», 2021. http://dx.doi.org/10.21782/b978-5-6043022-6-2.

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The review is devoted to the analysis of literature data on the history research of dengue fever, the discovery of the etiological infectious agent of this disease-dengue virus and its serotypes. A taxonomic overview of the }lavivirus family, genome organization, structure and function of viral proteins, mosquito species-viral vectors and virus transmission cycles, theories of its origin are presented. As well as the evolution, characteristics and epidemiology of viral serotypes, cellular receptors for dengue virus penetration, pathogenicity for human and factors for the development of severe disease, induced immunity, applied methods and markers for diagnosis, principles of disease treatment and drug development (more information about monoclonal antibodies-potential therapeutic drugs), vaccine options and their effectiveness are considered. The book is intended for students, graduate students, employees of research institutions and universities, as well as doctors involved in the study of }laviviruses and the problem of differential diagnosis of flavivirus infections.

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33

Mesquita, Emersom C., and Fernando A. Bozza. Diagnosis and management of viral haemorrhagic fevers in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0293.

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In a globalized scenario where widespread international travel allows viral agents to migrate from endemic to non-endemic areas, health care providers and critical care specialists must be able to readily recognize a suspected case of viral haemorrhagic fever (VHF). Early suspicion is pivotal for improving patient outcome and to ensure that appropriate biosafety measures be applied. VHFs are acute febrile illnesses marked by coagulation disorders and organ specific syndromes. VHFs represent a great medical challenge because diseases are associated with a high mortality rate and many VHFs have the potential for person-to-person transmission (Filoviruses, Arenavioruses, and Bunyaviroses). Dengue is the most frequent haemorrhagic viral disease and re-emergent infection in the world and, due to its public health relevance, severe dengue will receive special attention in this chapter. The diagnosis of VHFs is made by detecting specific antibodies, viral antigens (ELISA) and viral nucleic acid (RT-PCR) on blood samples. Supportive care is the cornerstone in the treatment of VHFs. Ribavirin should be started as soon as a case of VHF is suspected and discontinued if a diagnosis of Filovirus or Flavivirus infection is established. Adjunctive antimicrobial therapy is usually implemented to treat co-existing or secondary infections. Antimalarial treatment should also be initiated if a malaria test (thick blood films) is not quickly available and/or reliable and patients travel history is compatible. It is always recommended to apply appropriate biosafety measures and notify local infection control unit and state and national authorities.

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34

Depa, Larisse, Larissa Depa, Crhisllane Vasconcelos, Vagner Fonseca, and Diego Frias. Estudo do uso de códons nos vírus da Dengue, Zika e Chikungunya com foco em terapia por inibição seletiva de tRNAs contra arboviroses. Edited by Diego Mariano. Alfahelix, 2021. http://dx.doi.org/10.51780/978-6-5992753-3-3.

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O vírus da dengue (DENV), o vírus da Zika (ZIKV) e o vírus da chikungunya (CHIKV) são espécies que apresentam relevância clínica para a saúde pública. Porém, ainda não existe um tratamento específico ou vacina disponível para esses arbovírus. Nesse contexto, é fundamental encontrar novos alvos terapêuticos que possam auxiliar estratégias e tratamentos mais eficientes. A metodologia de codon usage tem demonstrado bons resultados para encontrar alvos para terapias que visam inibidores de tradução. Este estudo buscou analisar o uso de códons e o equilíbrio entre a abundância relativa dos RNAs transportadores (tRNAs) para encontrar alvos terapêuticos que irão estimular novas alternativas de tratamento para infecções causadas pelos DENV, ZIKV e CHIKV. Para tanto, foi replicada uma estratégia computacional, assumindo uma terapia hipotética de inibição seletiva de tRNA (Selective Transport RNA Inhibition Therapy - STRIT), onde foi estabelecido um índice de potencial terapêutico (T-score) para encontrar potenciais espécies de tRNA que poderiam ser inibidas seletivamente para atenuar a replicação viral na célula hospedeira. Foram identificados os cinco códons com maior frequência relativa vírus/hospedeiro (mais relevantes para o vírus) nas seis espécies de arbovírus, notando que todos terminam com purinas A ou G. Os códons GGA (Glicina), AGA (Arginina) e ATA (Isoleucina) são relevantes em todos os flavivirus (ZIKV, DENV-1, DENV-2, DENV-3, DENV-4), mas não no alphavirus CHIKV, onde os códons ACG (Treonina) e CCG (Prolina) são os mais relevantes. Posteriormente, selecionando os cinco códons com maiores T-score nas seis espécies virais (30 códons em total) encontramos apenas 11 códons diferentes, todos terminados com A ou G. Agrupados segundo o nucleotídeo na primeira posição do códon estes 11 códons são: (AGA, ACA, ATA, ACG), (GGA, GCA, GTA, GCG), (CTA, CCG) e (TGG). No agrupamento, notamos outro fato intrigante: que 10 dos 11 códons mais bem ranqueados por T-score, terminam com GA, CA, TA ou CG. Nosso método identificou as espécies de tRNA (através da identificação do códon cognato com maior T-score), cuja inibição funcional por qualquer método específico a anticódon, poderia ter potenciais efeitos terapêuticos em células infectadas pelo vírus da Dengue, Zika e Chikungunya causando a inibição da tradução das proteínas do vírus sem ter um efeito deletério na sobrevivência das células hospedeiras durante o período da infeção. A predominância absoluta dos nucleotídeos A e G na terceira posição dos 11 códons com maior T-score, que por sua vez indica uma preferência dos arbovírus por 11 espécies de tRNA com C ou T na primeira posição do anticódon, abre um novo espaço de pesquisa na interação vírus-hospedeiro.

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35

Virally infected cells. New York: Plenum, 1989.

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