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Books on the topic 'Fibrosis'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 November 2024

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1

Rittié, Laure, ed. Fibrosis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8.

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2

David, Evered, Whelan Julie, Ciba Foundation, and Symposium on Fibrosis (1984 : Ciba Foundation), eds. Fibrosis. London: Pitman, 1985.

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3

Symposium on Fibrosis (1984 : London), ed. Fibrosis. London: Pitman, 1985.

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4

Symposium, Ciba Foundation. Fibrosis. London: Pitman, 1985.

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5

Varga, John, David A. Brenner, and Sem H. Phan, eds. Fibrosis Research. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1592599400.

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6

Silverstein, Alvin. Cystic fibrosis. New York: F. Watts, 1994.

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7

Davis, Stephanie Duggins, Margaret Rosenfeld, and James Chmiel, eds. Cystic Fibrosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7.

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8

Moss, Richard B., ed. Cystic Fibrosis. Totowa, NJ: Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-0475-6.

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9

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-117-8.

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10

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-120-8.

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11

Gold, Susan Dudley. Cystic fibrosis. New York: Crestwood House, 2000.

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12

B, Davis Pamela, ed. Cystic fibrosis. New York: M. Dekker, 1993.

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13

1951-, Goodfellow P., ed. Cystic fibrosis. Oxford: Oxford University Press, 1989.

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14

B, Fiel Stanley, ed. Cystic fibrosis. Philadelphia: Saunders, 1998.

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15

E, Hodson Margaret, Geddes Duncan M, and Bush, Andrew, 1954 Apr. 24-, eds. Cystic fibrosis. 3rd ed. London: Hodder Arnold, 2007.

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16

Jacqueline, Langwith, ed. Cystic fibrosis. Detroit: Greenhaven Press, 2009.

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17

Giddings, Sharon. Cystic fibrosis. New York: Chelsea House, 2009.

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18

Bjorklund, Ruth. Cystic fibrosis. New York: Marshall Cavendish Benchmark, 2009.

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19

S, Valiathan M., Somers Krishna, Kartha C. Chandrasekharan, and Symposium on Endomyocardial Fibrosis (1991 : Trivandrum, India), eds. Endomyocardial fibrosis. Delhi: Oxford University Press, 1993.

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20

Mandolfo, Anna. Cystic fibrosis. Southampton: Dulphar Laboratories, 1991.

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21

O, Warner J., ed. Cystic fibrosis. Edinburgh: Churchill Livingstone, 1992.

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22

M, Geddes Duncan, and Hodson Margaret E, eds. Cystic fibrosis. London: Chapman & Hall Medical, 1995.

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23

Posadas, A. Salcedo, and Rosa María Girón Moreno. Fibrosis quística. Majadahonda (Madrid): Ergón, 2005.

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24

Rosaler, Maxine. Cystic fibrosis. New York: The Rosen Pub. Group, 2007.

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25

J, David T., Goeting Nicola L. M, and Duphar Medical Relations, eds. Cystic fibrosis. Southampton: Duphar Medical Relations, 1991.

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26

Capewell, Gillian. Cystic fibrosis. London: Office of Health Economics, 1986.

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27

Gillian, Capewell, and Association of the British Pharmaceutical Industry. Office of Health Economics., eds. Cystic fibrosis. London: Office of Health Economics, 1986.

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28

Dennis, Shale, ed. Cystic fibrosis. London: BMJ Publishing Group, 1996.

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29

Lewis, Allen Julian, Rubenstein Ronald, and Panitch Howard B, eds. Cystic fibrosis. New York, NY: Informa Healthcare USA, 2010.

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30

1949-, Phan Sem Hin, and Thrall Roger S. 1947-, eds. Pulmonary fibrosis. New York: Marcel Dekker, 1995.

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31

Meyer, Keith C., and Steven D. Nathan, eds. Idiopathic Pulmonary Fibrosis. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99975-3.

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32

Meyer, Keith C., and Steven D. Nathan, eds. Idiopathic Pulmonary Fibrosis. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-682-5.

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33

Willis, Monte S., Cecelia C. Yates, and Jonathan C. Schisler, eds. Fibrosis in Disease. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98143-7.

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34

Nakamura, Hiroyuki, and Kazutetsu Aoshiba, eds. Idiopathic Pulmonary Fibrosis. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55582-7.

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35

Hodson, Margaret, Duncan Geddes, and Andrew Bush. Cystic Fibrosis. 3rd ed. A Hodder Arnold Publication, 2007.

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36

Hodson, Margaret, Duncan Geddes, and Andrew Bush. Cystic Fibrosis. Taylor & Francis Group, 2007.

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37

Evered, David, and Julie Whelan. Fibrosis. Wiley & Sons, Incorporated, John, 2009.

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38

Staff, CIBA Foundation Symposium. Fibrosis. Wiley & Sons, Limited, John, 2008.

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39

Korn, Joseph. Fibrosis. John Wiley and Sons Ltd, 2007.

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40

Webb, A. K. Cystic Fibrosis. European Respiratory Society, 2006.

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41

Spilsbury, Richard. Cystic Fibrosis. Rosen Publishing Group, 2018.

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42

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Cystic fibrosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0021.

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Gastrointestinal manifestations 156Management of gastrointestinal symptoms in children with CF 158Nutrition in CF 158Nutritional management 159Vitamins 160The incidence of cystic fibrosis (CF) is around 1 in 2500. Cases are diagnosed as a consequence of population screening or high-risk screening, or following presentation with clinical symptoms typical of the disorder. The basic defect is in the CFTR (cystic fibrosis transmembrane conductance regulator) protein which codes for a cyclic adenosine monophosphate-regulated chloride transporter in epithelial cells of exocrine organs. This is involved in salt and water balance across epithelial surfaces. The gene is on chromosome 7. There are multiple known mutations, the most common being ...

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43

Yaqoob, Muhammad M., Katherine Bennett-Richards, and Islam Junaid. Retroperitoneal fibrosis. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0357.

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Retroperitoneal fibrosis (RPF) is a rare but multifaceted disease which encompasses a range of conditions characterized by the presence of a fibro-inflammatory tissue, which usually surrounds the abdominal aorta, iliac arteries, and extends into the retroperitoneum to entrap ureters with resultant unilateral or bilateral obstruction, usually at the junction between the middle and lower thirds of the ureter. The condition is progressive: initially, the fibrous tissue is fairly cellular, later becoming relatively acellular. The mechanism by which obstruction occurs is probably due to loss of peristalsis. A histological diagnosis should be obtained if at all possible, and laparotomy is required in order to obtain a sufficiently large sample to differentiate between idiopathic and secondary causes of RPF. Treatment of idiopathic RPF is by corticosteroids in the first instance with ureteric stents or ureterolysis initially and requires regular monitoring.

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44

Webb, A. K., and F. A. Ratjen, eds. Cystic Fibrosis. European Respiratory Society Journals Ltd, 2006. http://dx.doi.org/10.1183/1025448x.erm3506.

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45

Mall, Marcus A., and J. Stuart Elborn, eds. Cystic Fibrosis. European Respiratory Society, 2014. http://dx.doi.org/10.1183/1025448x.erm6414.

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46

Razzaque, M. S., and T. Taguchi, eds. Renal Fibrosis. S. Karger AG, 2003. http://dx.doi.org/10.1159/isbn.978-3-318-00964-4.

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47

Bright-Thomas, Rowland J., and Andrew M. Jones. Cystic fibrosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0132.

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Cystic fibrosis is the most common lethal autosomal recessive disorder in Caucasians. There is no known survival advantage of the heterozygote carrier state. Chronic progressive pulmonary infection and bronchiectasis are the major causes of morbidity and mortality. The disease affects all ductal systems where the basic defect is manifest, including the pancreas, gastrointestinal tract, sinuses, hepatobiliary system, and male reproductive system, and has significant effects on nutrition and growth.

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48

Horsley, Alex, Steve Cunningham, and Alistair Innes, eds. Cystic Fibrosis. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199582709.001.0001.

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49

Martin, David, and Junzheng Wu. Cystic Fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0021.

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Cystic fibrosis (CF) is an inherited chronic disease that affects about 30,000 children and adults in the United States and 70,000 worldwide. CF is the most common fatal inherited disorder affecting Caucasians in the United States. While its presentation can vary in severity, the most common clinical manifestations are progressive lung damage and chronic digestive problems due to exocrine gland dysfunction and the production of thick viscous mucus. Careful perioperative management is important to avoid respiratory complications.

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50

Tinch, Brian, David Martin, and Junzheng Wu. Cystic Fibrosis. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0018.

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Cystic fibrosis is an inherited disorder. The diagnosis should be suspected in an infant who has meconium ileus or infants presenting to the operating room with volvulus. Cystic fibrosis is characterized by frequent mucous plugging in the respiratory tract which may manifest as wheezing and frequent intermittent flare-ups of respiratory decompensation. Optimization of the affected child’s respiratory status prior to elective surgery is mandatory to prevent difficulty with intraoperative ventilation. While the laryngeal mask airway may be used for short procedures, the use of an endotracheal tube facilitates suctioning of the frequently inspissated secretions that accompany cystic fibrosis in order to optimize ventilation.

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