Relevant bibliographies by topics / Fetuses

Academic literature on the topic 'Fetuses'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Fetuses"

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Yousif Mahmood, Mohammad, Nooruldeen Yaseen Khudhair, and Yaseen Mahmood Rasheed. "Heifers and multiparous cows are affected by Dystocia and its implications on the viability of the pregnancy." Bionatura 8, no. 1 (March 15, 2023): 1–5. http://dx.doi.org/10.21931/rb/2023.08.01.82.

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Dystocia considers one of the most common obstetrical problems in cattle, especially in heifers; the current study achieved to demonstrate Dystocia's impact on the viability of the fetus in heifers and cows. The study involved 15 multiparous cows aged 3-7 years and 10 heifers; these animals suffered abnormality calving. The study showed Dystocia higher significantly (P≤0.01) in 15 (60%) multiparous cows than 10 (40%) heifers; also, the rate of difficult male birth was greater 7 (70%) than in difficult female birth were 3 (30%) in heifer animals. Heifer had difficult male birth with the anterior presentation of the fetus was 5 (71.42%), higher than with posterior presentation fetus 2 (28.58%). One heifer cow (20%) had difficult male birth with flexion of the elbow joint in the anterior presentation alive fetus, whilst the heifer cows had difficult male birth with flexion of the elbow joint dead fetus were 2 (40%) with significant difference towards dead fetus at(P≤0.01). The number of heifers that had a problematic female birth with anterior presentation and flexion of the shoulder joint of an alive fetus was 1 (33.34%). In contrast, one heifer (33.33%) with a transverse presentation of a female dead fetus and one heifer (33.33%) had difficult female birth with posterior presentation and incomplete extension of hind limbs alive fetuses with a significant difference towards dead fetuses at (P≤0.05). The number of multiparous cows that had difficult male births was 8 (53.34%), and those that had difficult female births were (7) (46.66%). The multiparous cows have difficult male births with anterior and flexion of elbow joints, with a down deviation of head alive fetuses and back head, live fetuses were (37.5%), (12.5%) and (12.5%) respectively, with significant differences among these categories towards alive fetuses at (P≤0.01). The number of multiparous cows had difficult female birth with anterior presentation alive fetus, and uterine inertia was 1 (14.28%), whereas the multiparous cows had difficult female birth with anterior presentation and down deviation of fetus's head was 4 (57.14%) (3 alive fetus+1dead fetuses). With a significant difference among these categories towards live fetuses at (P≤0.01). The study concluded that Dystocia is a severe joint event in cows; the flexion of the elbow joint and head-down deviation in the anterior presentation of the fetus are common types of Dystocia, whilst the more common types of Dystocia are an incomplete extension of hind limbs in the birth canal in the posterior presentation, with variable effects of these types of Dystocia on fetal viability. Keywords: Calving abnormality, Dystocia, Fetal viability, Dairy cows
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Nuzhat, Ayesha. "Anatomy of Inferior Mesenteric Artery in Fetuses." Scientifica 2016 (2016): 1–5. http://dx.doi.org/10.1155/2016/5846578.

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Aim. To analyze Inferior Mesenteric Artery in fetuses through its site of origin, length, diameter, and variation of its branches.Method. 100 fetuses were collected from various hospitals in Warangal at Kakatiya Medical College in Andhra Pradesh, India, and were divided into two groups, group I (second-trimester fetuses) and group II (third-trimester fetuses), followed by dissection.Result.(1) Site of Origin. In group I fetuses, origin of Inferior Mesenteric Artery was at third lumbar vertebra in 33 out of 34 fetuses (97.2%). In one fetus it was at first lumbar vertebra, 2.8%. In all group II fetuses, origin of Inferior Mesenteric Artery was at third lumbar vertebra.(2) Length. In group I fetuses it ranged between 18 and 30 mm, average being 24 mm except in one fetus where it was 48 mm. In group II fetuses the length ranged from 30 to 34 mm, average being 32 mm.(3) Diameter. In group I fetuses it ranged from 0.5 to 1 mm, and in group II fetuses it ranged from 1 to 2 mm, average being 1.5 mm.(4) Branches. Out of 34 fetuses of group I, 4 fetuses showed variation. In one fetus left colic artery was arising from abdominal aorta, 2.9%. In 3 fetuses, Inferior Mesenteric Artery was giving a branch to left kidney, 8.8%. Out of 66 fetuses in group II, 64 had normal branching. In one fetus left renal artery was arising from Inferior Mesenteric Artery, 1.5%, and in another fetus one accessory renal artery was arising from Inferior Mesenteric Artery and entering the lower pole of left kidney.Conclusion. Formation, course, and branching pattern of an artery depend on development and origin of organs to attain the actual adult position.
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Bessarabov, V. I. "Multiple fetuses in the fetus." Russian Journal of Pediatric Surgery 27, no. 3 (August 9, 2023): 226–29. http://dx.doi.org/10.55308/1560-9510-2023-27-3-226-229.

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In the observation, a clinical case "fetus in fetus" in a newborn boy is described. The pathology was revealed before surgery. During the surgery, the second fetus was found in the same place, in retroperitoneal space. The multiplicity of "fetuses in fetus" has not yet been described by anyone yet, though some researchers express their opinion that such pathology is a possible option.
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Simpson, John M., Robert W. Yates, and Gurleen K. Sharland. "Irregular heart rate in the fetus—not always benign." Cardiology in the Young 6, no. 1 (January 1996): 28–31. http://dx.doi.org/10.1017/s1047951100003206.

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SummaryOne hundred-ninety-four fetuses with irregular heart rates were seen over a five-year period at a tertiary center for fetal cardiology. The median gestation at referral was 31 weeks, with a range from 19 to 41 weeks. Of these fetuses, 157 had extrasystoles of either atrial or ventricular origin. Blocked atrial ectopic beats had led to slow ventricular rates (80–110 beats per minute) in 37 fetuses. The structure of the heart was normal in all except two fetuses. Postnatal outcome was known for 165 of the fetuses. Of these, 157 (95%) had an uneventful antenatal and postnatal course. Tachyarrhythmias developed in eight fetuses (5%) in either the prenatal (n=4) or postnatal (n=4) period. Five of 37 fetuses with blocked atrial ectopic beats (13%) developed a tachyarrhythmia. No fetus developed hydrops, and all infants survived. All cases had required treatment with antiarrhythmic drugs. The occurrence of an irregular heart rhythm in the fetus, therefore, is not always benign. Fetuses with blocked atrial ectopic beats require particularly close monitoring.
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Keeling, Jean W., and Inger Kjaer. "Cervical Ribs: Useful Marker of Monosomy X in Fetal Hydrops." Pediatric and Developmental Pathology 2, no. 2 (March 1999): 119–23. http://dx.doi.org/10.1007/s100249900099.

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Cervical ribs were observed in six hydropic fetuses with 45X karyotype. To test the usefulness of this observation in the macerated hydropic fetus where chromosome culture is problematic, a group of 36 hydropic fetuses was examined. Cases were chosen to include fetuses with several karyotypic and pathological abnormalities known to be associated with fetal hydrops. Whole-body anteroposterior radiographs were evaluated without knowledge of the fetal karyotype or pathological findings. Twenty-five fetuses had an abnormal karyotype, seven had a normal karyotype and in four culture failed. In the last group, the number of X, 21 and 18 chromosomes per nucleus was estimated using FISH. Radiographic analysis demonstrated that among the 16 fetuses with 45,X karyotype or a single copy of X and female phenotype, 12 had a pair of cervical ribs. Three other fetuses had a single cervical rib. Only one fetus had no cervical ribs. The last fetus had tubular hypoplasia of the aortic arch and persistent mesocolon. Twelve of the sixteen 45,X fetuses had tubular hypoplasia of the aortic arch. Seven had other cardiovascular anomalies, five had renal anomalies, and five had anomalies of intestinal rotation. Cervical rib appears to be more common than other frequently recorded associations of 45,X. It is a useful and easily demonstrated mark in the evaluation of the macerated hydropic fetus.
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Kosheleva, N. G., and L. B. Zubjitskua. "Pregnancy outcomes, immunomorphologic condition of placenta after arv infection of pregnant woman. Prevention. Treatment." Journal of obstetrics and women's diseases 54, no. 3 (November 1, 2005): 12–18. http://dx.doi.org/10.17816/jowd81944.

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53 pregnancy outcomes for fetuses and newborns, immunomorphologic condition of placentae and organs of 29 stillborn fetuses were studied. Antigens of viruses A (H1N1 and H2N2), B, RC, AD were detected in placenta and organs of stillborn fetuses. Fixed immune complex, containing C3 complement and IgM, IgA, IgG, was observed in nearly 100 % in placenta and rather rarely in fetus organs. Virus antigens were the same in placenta and fetus organs.
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Picchiassi, Elena, Gian Carlo Di Renzo, Federica Tarquini, Vittorio Bini, Michela Centra, Luana Pennacchi, Fabiana Galeone, and Giuliana Coata. "The Potential Usefulness of Free Fetal DNA in Maternal Blood for Prenatal Fetal Gender Determination in Multiple Pregnancies." Twin Research and Human Genetics 15, no. 2 (March 28, 2012): 143–48. http://dx.doi.org/10.1375/twin.15.2.143.

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We applied a noninvasive prenatal test for the determination of fetal gender in multiple pregnancies by using free fetal DNA circulating in maternal blood in order to evaluate whether the quantification of male DNA could distinguish the fetal gender and the number of male and female fetuses in multiple pregnancies. We enrolled consecutively 44 women with twin pregnancies between 11–14 weeks of gestation. Peripheral maternal blood was collected, and genomic DNA was extracted from maternal plasma and analyzed for the multicopy DYS 14 sequence by using real-time PCR to quantify male DNA. Results showed that male DNA concentration was significantly higher in twin pregnancies with at least one male fetus, compared to twin pregnancies with only female fetuses. Comparing male DNA concentration in pregnancies with two male fetuses versus pregnancies with one female fetus and one male fetus, we did not obtain a significant difference between the two groups due to a slight overlapping of the range values. Therefore, our test correctly predicted fetal gender, distinguishing twin pregnancies with at least one male fetus from twin pregnancies with only female fetuses, with a diagnostic accuracy of 100%. For distinguishing pregnancies with two male fetuses from pregnancies with both female and male fetuses, a diagnostic accuracy of 76.1% was achieved.
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Alverson, Janet, Katherine I. O'Rourke, and Timothy V. Baszler. "PrPSc accumulation in fetal cotyledons of scrapie-resistant lambs is influenced by fetus location in the uterus." Journal of General Virology 87, no. 4 (April 1, 2006): 1035–41. http://dx.doi.org/10.1099/vir.0.81418-0.

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Placentae from scrapie-infected ewes have been shown to accumulate PrPSc when the genotype of the fetus is of a susceptible genotype (VRQ/VRQ, ARQ/VRQ or ARQ/ARQ). Cotyledons from fetuses of genotypes ARR/ARR, ARQ/ARR and ARQ/VRR have previously been shown to be resistant to PrPSc accumulation. By using ewes from a naturally infected scrapie flock, cotyledons from fetuses of multiple births of different genotypes were examined. PrPSc was detected in fetal cotyledons of genotype ARQ/ARQ, but not in cotyledons from their dizygotic twin of genotype ARQ/ARR. This confirms earlier reports of single fetuses of these genotypes, but is the first description of such a finding in twin fetuses, one of each genotype. It is also demonstrated that cotyledons from sibling fetuses of genotypes ARQ/VRQ and ARQ/ARQ have different patterns of PrPSc accumulation depending on whether the dam is of genotype ARQ/ARQ or ARQ/VRQ. Lastly, it is shown that cotyledons from fetuses with resistant genotypes are weakly positive for PrPSc when they have shared the same pregnant uterine horn with a fetus of a susceptible genotype with cotyledons positive for the detection of PrPSc. Additionally, a PCR product for the Sry gene, a product specific to males, was found in cotyledons from female fetuses that had shared a uterine horn with a male fetus. This indicates that some sharing of fetal blood occurs between placentomes and fetuses residing in the same uterine horn, which can result in PrPSc accumulation in cotyledons with resistant genotypes.
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Nonaka, Kazuaki, Yasunori Sasaki, Yoshihisa Watanabe, Ken-ichi Yanagita, and Minoru Nakata. "Effects of Fetus Weight, Dam Strain, Dam Weight, and Litter Size on the Craniofacial Morphogenesis of CL/Fr Mouse Fetuses Affected with Cleft Lip and Palate." Cleft Palate-Craniofacial Journal 34, no. 4 (July 1997): 325–30. http://dx.doi.org/10.1597/1545-1569_1997_034_0325_eofwds_2.3.co_2.

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Objective: This study examined the factors related to the morphogenesis of the craniofacial complex of the CL/Fr mouse fetus affected with CLP based on the findings of a lateral cephalogram. Design: Embryo transfer experiments were performed to determine the effect of the fetus weight, dam strain, dam weight, and litter size on the intra-uterine craniofacial morphogenesis of CL/Fr mouse fetuses. On the 18th gestational day, each pregnant dam that had received CL/Fr mouse embryos was laparotomized to remove the transferred fetuses that had developed in the uteri of the cleft lip and palate (CLP)-susceptible CL/Fr strain dam and the CLP-resistant C57BL strain dam. A cephalometric observation of the craniofacial morphology of each fetus was subsequently performed. Results: Based on a multiple regression analysis, the standardized partial regression coefficients of the affected fetus weight, the dam weight, and the litter size on the maxillary size of the affected CL/Fr fetus were 0.71 (p < .01), 0.03, and −0.07. According to a least-squares analysis of variance, the dam strain effect in addition to the effect of the affected fetus weight on the maxillary size and the cranial size of the affected fetuses was significant (p < .01 for cranial size, p < .05 for maxillary size) and close to a significant level (p = .09) for the mandibular size of the affected fetuses. The adjusted maxillary size and cranial size after statistically eliminating the effects of the affected fetus weight, dam weight, and lifter size on each original craniofacial size of the affected fetuses that had developed in the CL/Er dam strain were also significantly smaller than those of the affected fetuses that had developed in the C57BL dam strain. Conclusions: The present results indicate that the craniofacial growth of the CL/Fr mouse fetus affected with CLP increased in proportion to the fetus weight. The dam strain effect, in addition to the effect of the affected fetus weight, could thus not be ignored when the etiology of the spontaneous CLP was examined, while the uterine environment, provided by the CL/Fr strain dam, retarded the intra-uterine craniofacial growth of the affected fetuses. It was therefore concluded that the dam strain effect, as well as the effect of the affected fetus weight, both play an important role on the craniofacial morphogenesis of the CL/Fr strain of the affected fetuses that developed in both strain dams.
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Lakhdir, Farahaba R., Haiyan Tong, and Charles E. Wood. "Baroreceptor and prostanoid control of fetal renal cortical blood flow and plasma renin activity." Reproduction, Fertility and Development 13, no. 3 (2001): 119. http://dx.doi.org/10.1071/rd00101.

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Renal function in the fetus is important for maintenance of fetal fluid and electrolyte balance. This study was performed to test the role of prostaglandins and their interaction with arterial baroreceptors and chemoreceptors in the control of renal cortical blood flow during hypotension produced by vena caval obstruction in late-gestation fetal sheep. We studied 18 time-dated, chronically catheterized, fetal sheep (124–136 days gestation). Fetuses were either studied intact (n = 11) or sinoaortic denervated (n = 7), and each fetus was studied twice, with and without pretreatment with indomethacin (0.2 mg kg –1 , i.v.). Each fetus was subjected to hypotension caused by vena caval obstruction for 10 min. Before hypotension, renal cortical blood flow was higher in the vehicle-treated sinoaortic denervated fetuses than in vehicle-treated intact fetuses. The increased renal cortical blood flow observed in the sinoaortic denervated fetuses was counteracted by indomethacin, so that the difference between sinoaortic denervated and intact fetuses was eliminated after indomethacin treatment. Hypotension decreased renal blood flow equally in all groups. Plasma renin activity was increased in response to hypotension in the intact fetuses, but not in the sinoaortic denervated fetuses. Indomethacin treatment, by itself, did not alter plasma renin activity. It is concluded that both arterial baroreceptors and prostanoids influence renal blood flow. Further, renin secretion is influenced by arterial baroreceptors and chemoreceptors and there is no apparent modulatory effect of prostanoids on the baroreflex control of renin secretion.
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Dissertations / Theses on the topic "Fetuses"

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Proniaiev, D. V. "Fetuses anatomy of the ovarian." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18456.

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Kashperuk-Karpiuk, I. S. "Fetuses anatomy of the buccal region." Thesis, БДМУ, 2022. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/19320.

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Lavriv, I. P. "Fetuses anatomy of the parotid gland structure." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18449.

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Hedlund, Sebastian. "Expression of B-adrenergic receptors in chicken fetuses." Thesis, Linköping University, The Department of Physics, Chemistry and Biology, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-9819.

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Chicken fetuses exposed to chronic hypoxia suffer from growth retardation and

induces an overall sympathetic activity, including elevation of the concentration

of circulating catecholamines. Simultaneously, hypoxic fetuses display a

lowered β-adrenoreceptor (βAR) density in myocardial tissue. In vertebrates,

β1AR and β2AR are the most important signalling pathways for acute elevation

of cardiac performance. The aim of this study was to see how chronic hypoxia

affects the level of messenger RNA (mRNA) for the β1AR in the fetal chicken

heart at different developmental ages. The broiler chicken is a suitable model

organism for studying the progression of heart failure because the fast growth

rate requires a large increase in blood perfusion at the end of fetal development.

The β1AR sequence of the broiler chicken is 1587 bp and located on

chromosome 6. When running a PCR for quantification of the sequence,

primers for almost the whole sequence failed (1404 bp) and so did primers of

1193 bp; instead primers of 692 bp of the sequence were used and made

quantification possible. Similar results were obtained from both the heart and

liver of day 15 fetal chickens. The PCR product was cloned into a TOPO vector

and sent for sequencing, to enable the making of a probe for a northern blot

analysis of the mRNA in the fetal chicken hearts.

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Kryvetsky, I. V. "Anatomy of the spinal column in the fetuses." Thesis, БДМУ, 2022. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/19322.

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Korchynska, N. S. "Morphogenesis of the maxilla of the human fetuses." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17579.

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Lavriv, L. P. "Anatomy of the parotid gland structure in human fetuses." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17570.

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Orós, López Daniel. "Perinatal and neurodevelopmental outcome of late-onset growth restricted fetuses." Doctoral thesis, Universitat de Barcelona, 2010. http://hdl.handle.net/10803/2504.

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DE LA TESIS:

"Resultado perinatal y del neurodesarrollo en fetos con retraso de crecimiento intrauterino de instauración tardía"

TEXTO:

El retraso de crecimiento intrauterino es una de las patologías más graves del desarrollo fetal, asociándose con un incremento la mortalidad intrauterina, mortalidad perinatal y prematuridad, siendo un conocido factor de riesgo para el desarrollo de déficits neurológicos durante la infancia y la adolescencia. Se considera "pequeños" a todos los fetos con un peso por debajo del percentil 10 para su edad gestacional y sexo. Pero no todos los fetos "pequeños" son verdaderos retrasos de crecimiento. La búsqueda de variables clínicas que nos ayuden a diferenciar los fetos "pequeños normales" de los "retrasos de crecimiento intrauterino" (RCIU) ha sido uno de los focos más activos de investigación en medicina fetal durante los últimos 20 años.

El aumento de las resistencias vasculares placentarias, expresado por la elevación del índice de pulsatilidad de la arteria umbilical (AU), es el criterio diagnóstico más aceptado. La introducción del Doppler de la AU ha demostrado mejorar el resultado y reducir la mortalidad perinatal. Actualmente se asume que los fetos con un peso por debajo del percentil 10 y un aumento de las resistencias vasculares placentarias son RCIU, siendo los fetos con una resistencia vascular placentaria normal, fetos pequeños normales, a los que denominamos "pequeños para edad gestacional" (PEG).

Sin embargo, recientes publicaciones han puesto en duda el valor de la arteria umbilical para definir cuando un feto pequeño tiene bajo riesgo, encontrando que los fetos PEG también presentan resultado perinatal subóptimo, así como una mayor incidencia de un amplio espectro de alteraciones sutiles del desarrollo cerebral que se pueden expresar como alteraciones del comportamiento, desordenes neuromusculares, problemas en el aprendizaje y alteraciones de la conducta.

Los estudios incluidos en este proyecto son parte de una línea de investigación sobre la circulación cerebral de los fetos con retraso de crecimiento, y su capacidad de predicción de daños neurológicos.

El primer proyecto tiene por objeto determinar las tendencias longitudinales y tipo de cambio de los índices de pulsatilidad Doppler de la arteria cerebral uterina, umbilical y cerebral media en fetos PEG inicio tardío desde el diagnóstico hasta el parto.

El objetivo del segundo proyecto fue evaluar el desarrollo neuroconductual neonatal de fetos RCIU nacidos a término sin insuficiencia placentaria. Muchos estudios han encontrado asociaciones entre los fetos con RCIU precoz y el desarrollo del neurocomportamiento, sensorial y disfunciones cognitivas. Resultados a largo plazo de los bebés prematuros con RCIU ha revelado un perfil específico de las dificultades neurocognitivas con pobre funcionamiento ejecutivo, falta de flexibilidad y de creatividad, así como problemas del leguaje. Algunos estudios han relacionado estas dificultades en la infancia con trastornos de conducta ya presentes en el período neonatal, un momento en que las influencias ambientales son todavía mínimos. Algunos estudios también han informado a largo plazo de las desventajas cognitivas los niños con RCIU de instauración tardía, pero no hay información sobre el desarrollo neuroconductual de los bebés nacidos a término con RCIU sin insuficiencia placentaria.

El tercer proyecto fue dirigido para analizar si la investigación Doppler de la ACA es superior a la investigación Doppler de la arteria cerebral media en la predicción de resultados perinatales adversos en fetos PEG sin insuficiencia placentaria. Diversos estudios en fetos RCIU han demostrado una redistribución regional de suministro de sangre en el cerebro, que contribuye a la jerarquía regional en el deterioro del cerebro, haciendo que ciertas áreas más susceptibles que otras a la hipoxia. El lóbulo frontal del cerebro, se abastece principalmente por la ACA, es una de estas estructuras muy sensibles en los niños crónicamente hipóxicos. El estudio de ésta arteria podría ser superior a los parámetros estándar que se utiliza para detectar la redistribución del cerebro, la ACM, para la detección de los fetos en una fase temprana de la hipoxia cerebral.

Teniendo en cuenta lo anteriormente expuesto, nuestras hipótesis de trabajo serán:

a) Hipótesis conceptual

· Un porcentaje de fetos con retraso de crecimiento de aparición tardía, con función placentaria normal, han estado expuestos a hipoxia leve en el útero.

b) Hipótesis secundarias

· El seguimiento longitudinal de fetos con retraso de crecimiento de aparición tardía demuestra que los índices de pulsatilidad Doppler de la arteria cerebral anterior (ACA), la arteria cerebral media (ACM) y la relación cerebro-placentaria
(CPR) presentan modificaciones antes y de forma más frecuentes que la arteria umbilical (AU) materna y de las arterias uterinas (AUT).

· Los fetos con retraso de crecimiento de aparición tardía con función placentaria normal, tienen peores resultados perinatales, así como un desarrollo neuroconductuales neonatal subóptimo.

· Los fetos con retraso de crecimiento de aparición tardía con signos de redistribución hemodinámica cerebral presentan disrrupciones neurológicas que afectan a la neuroconducta neonatal.

De este modo, los objetivos establecidos serán los siguientes:

a) OBJETIVO PRINCIPAL
· Estudiar la evolución temporal de los parámetros Doppler en fetos con retraso de crecimiento de aparición tardía para evaluar su asociación con resultados perinatales adversos y neuroconductuales.

b) OBJETIVOS ESPECÍFICOS

· Describir al final del embarazo la tendencia de los índices de pulsatilidad longitudinal de Doppler de la arteria cerebral media, umbilical y materna arterias uterinas a finales de los fetos con retraso de crecimiento de aparición tardía

· Evaluar el desarrollo neuroconductual y los resultados perinatales de los fetos con un peso fetal estimado inferior al p10 y Doppler de la arteria umbilical normal.

· Evaluar el desarrollo neuroconductual y el resultado perinatal de los fetos con retraso de crecimiento de aparición tardía con signos de redistribución de intrauterina cerebral definido por el estudio Doppler de las arterias cerebrales anterior y media.

Los resultados de esta investigación se obtuvieron mediante un estudio longitudinal prospectivo de dos cohortes (Cohorte Caso y Cohorte Control), con un total de 116 pacientes en cada rama (tasa de aceptación: 90%) en la muestra inicial. El trabajo se realizó en la Unidad de Crecimiento Fetal del Materno-Fetal del Departamento de Medicina del Hospital Clínico de Barcelona entre noviembre de 2007 y agosto de 2009.
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Epstein, Douglas J. "Genetic control of the survival of murine trisomy 16 fetuses." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61876.

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Thayyil, S. S. "Post-mortem magnetic resonance imaging in fetuses, newborns and children." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/147674/.

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My thesis explores the feasibility and utility of whole body post-mortem magnetic resonance (MR) imaging as an alternative for conventional autopsy in fetuses, newborns and children. The thesis starts with a systematic review of the existing literature on post-mortem MR imaging to identify the knowledge gaps. This is followed by the development of an effective recruitment model and a comparative study on the accuracy of less invasive autopsy by post-mortem MR imaging with conventional autopsy in 200 fetuses, newborns and children. The cause of death was accurately identified in more than 90% of cases by less invasive autopsy, following a hospital death, unexplained stillbirth or an unexpected death under HM Coronial investigation. Post-mortem MR imaging of the brain had a very high negative predictive value for excluding major neuropathological lesions; opening of the head can be avoided if post-mortem MR imaging of the brain is normal. High-resolution, 3D post-mortem cardiac MR imaging accurately detected structural heart diseases in larger fetuses, newborns and children. However, the accuracy of post-mortem lung MR imaging was poor; renal lesions required histological examination for definitive diagnosis. Furthermore, post-mortem MR imaging cannot differentiate between the normal death process and ante-mortem hypoxic brain injury due to the changes in T1 and T2 relaxometry values occurring after death. The diagnostic utility and image quality at 1.5 Tesla MR imaging was poor in smaller fetuses, however high field MR imaging at 9.4 Tesla provided satisfactory MR images in this sub group. In addition, visceral organ weights were accurately estimated from post-mortem MR data sets and anatomical models of these organs reconstructed by rapid prototyping. My thesis concludes by demonstrating the proof of principle of MR guided percutaneous biopsy in a piglet model. In summary, less invasive autopsy by post-mortem MR imaging may be a satisfactory alternative to conventional autopsy; however accurate methods of percutaneous tissue sampling need to be developed and validated for adequate histological examination of visceral organs, particularly lungs, kidneys and heart.
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Books on the topic "Fetuses"

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Forrester, Mary Gore. Persons, Animals, and Fetuses. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3.

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Alain, Couture, and Baud C, eds. Gastrointestinal tract sonography in fetuses and children. Berlin: Springer, 2008.

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Alain, Couture, and Baud C, eds. Gastrointestinal tract sonography in fetuses and children. Berlin: Springer, 2008.

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L, Fowden A., and Rossdale Peter D, eds. Foetal maturation: Comparative aspects of normal and disturbed development. New Market, [England]: R & W Publications, 1993.

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Gilbert, Stephen G. Pictorial anatomy of the fetal pig. 2nd ed. Seattle: University of Washington Press, 1987.

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Rhee, Eleanor Hoon Joo. Superior mesenteric artery flow velocity waveforms in small for gestational age fetuses. [New Haven, Conn: s.n.], 1998.

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Chiasson, Robert B. Laboratory anatomy of the fetal pig. Dubuque, IA: Wm. C. Brown Publishers, 1995.

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Chiasson, Robert B. Laboratory anatomy of the fetal pig. Dubuque, IA: Wm. C. Brown Publishers, 1997.

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Marko, Milan. Differenzierung des Pansenepithels des Rindes während der intrauterinen Entwicklung: Licht- und elektronenmikroskopische Untersuchungen. Aachen: Shaker, 1992.

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Milan, Klima, and Schildger Bernd 1956-, eds. Embryology of dolphins: Staging and ageing of embryos and fetuses of some Cetaceans. Berlin: Springer, 2000.

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Book chapters on the topic "Fetuses"

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Piontelli, Alessandra. "Studying Fetuses." In Citizen Fetus, 173–84. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-17161-1_8.

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Piontelli, Alessandra. "Fetuses Become Unborn Citizens." In Citizen Fetus, 333–46. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-17161-1_16.

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Piontelli, Alessandra. "Fetuses from Other Worlds." In Citizen Fetus, 251–71. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-17161-1_12.

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Forrester, Mary Gore. "Introduction." In Persons, Animals, and Fetuses, 2–8. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_1.

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Forrester, Mary Gore. "How Animals Ought to be Treated." In Persons, Animals, and Fetuses, 115–28. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_10.

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Forrester, Mary Gore. "Animal Rights." In Persons, Animals, and Fetuses, 129–36. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_11.

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Forrester, Mary Gore. "What Do We Owe to Future Generations?" In Persons, Animals, and Fetuses, 137–46. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_12.

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Forrester, Mary Gore. "When Should We Bring New People into the World?" In Persons, Animals, and Fetuses, 147–55. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_13.

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Forrester, Mary Gore. "The Human Fetus: Introduction." In Persons, Animals, and Fetuses, 156–63. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_14.

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Forrester, Mary Gore. "Should Fetuses be Extended Persons?" In Persons, Animals, and Fetuses, 164–87. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1633-3_15.

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Conference papers on the topic "Fetuses"

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MOALIC, P., Y. GRUEL, P. FOLOPPE, B. DELAHOUSSE, G. BODY, and J. LEROY. "LEVELS AND DISTRIBUTION OF FREE AND C4b-BP-B0UND-PR0TEIN S IN HUMAN FETUSES AND FULL-TERM NEWBORNS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644266.

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Levels and plasmatic distribution of protein S were studied on umbilical cordplasmas from 25 normal full-term newborns (N) and 15 normal fetuses (F) between20 and 30 weeks of gestation. Samples from fetuses were collected for antenatal diagnosis by direct puncture of the umbilical vein under high resolution real-time ultrasound. Total protein S(PS) level was determined using Laurell rocket immuno-electrophoresis (Diagnostica Stago, Asnifcres-France). Free PS wasmeasured using this latter method, afterprecipitation of C4b-BP-bound-PS by polyethylene glycol (PEG). Normal pool plasma, treated as well, was considered as the reference curve. C4b-binding protein (C4b-BP) determinations were conducted by Laurell rocket immunoelectrophoresis. The qualitative distribution of free PS and C4b-BP-bound-PS in plasma was also assessed by crossed-immunoelectrophoresis(CIE). Results (mean - SD) were expressed in percentage, in relation to healthy adults values (n = 15). Low levels of total PS were obtained in all fetuses (16.4 ±4.2) and newborns (36.4 ±9.5) as compared to adults (91.6 ± 12.2). Free protein S level was also decreased both in fetuses (22.2 ±6.0) and newborns (48.5 ± 12.1 versus 89.4 ± 26.3 in adults). At these stages of development, the ratio Free PS / Total PS (both values were obtained according to a reference curve performed with a normal adult pool plasma untreated by PEG) was significantly higher as compared to normal adults (0.82 ±0.07 in F, 0.64 ±0.17 in N and 0.39 ±0.11 in A, p‹0.001, Student t test). The predominance of free PS was also visualized in the CIE patterns. These data may be explained by undetectable C4b-BP in 21-week old fetuses (‹2% in 10 cases). After the 26th week of gestation C4b-BP level was 7.8 ±7.4 ‹n=5) and reached a value of 19.2 ±15.6 in newborns (adults = 95.7 ±14.7). In human fetus and newborn, PS essentially circulates under free form and this might compensate the decrease of the total PS level.
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Oelmeier de Murcia, K., S. Heese, K. Hammer, M. Möllers, H. Köster, M. Falkenberg, M. Eveslage, J. Braun, W. Klockenbusch, and R. Schmitz. "Adrenal Gland size in growth restricted fetuses." In Interdisziplinärer Kongress | Ultraschall 2018 – 42. Dreiländertreffen SGUM | DEGUM | ÖGUM. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1670429.

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Snider, D., and Xiaowei Xu. "Mining fetal magnetocardiogram data for high-risk fetuses." In 2011 IEEE International Conference on Bioinformatics and Biomedicine Workshops (BIBMW). IEEE, 2011. http://dx.doi.org/10.1109/bibmw.2011.6112563.

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Roy, Debashish, Madhusudhana Gargesha, Eddie Sloter, Michiko Watanabe, and David Wilson. "Cryo-imaging in a toxicological study on mouse fetuses." In SPIE Medical Imaging, edited by Robert C. Molthen and John B. Weaver. SPIE, 2010. http://dx.doi.org/10.1117/12.845627.

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Daniele, Beniamino, Giulio Steyde, Edoardo Spairani, Giovanni Magenes, and Maria G. Signorini. "Discriminating Healthy and IUGR fetuses through Machine Learning models." In 2022 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2022. http://dx.doi.org/10.1109/bhi56158.2022.9926874.

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Barnea, O., O. Luria, and J. Bar. "Detection of growth-restricted fetuses using a patient-specific model." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609601.

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Wautier, J. L., Y. Gruel, B. Boizard, J. P. Caen, F. Daffos, and F. Forestier. "ANTENATAL DIAGNOSIS OF THROMBOPATHY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644271.

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We previously determined platelet antigens and glycoproteins in the human fetus after 19 weeks of intrauterine life (Blood 68, 488-92,1986). These results obtained in fetuses with normal platelets allowed us to do the first attempt of antenatal diagnosis in Glanzmann thrombasthenia. The fetal propositus was tested with monoclonal (AP2, AP3) or polyclonalantibodies (IgGL) directed against GPIIbllla or platelets antigen (PLA1, Leka). The foetus was found to be heterozygous for GT and similar results were foundafter his birth.Grey platelet syndrome is a rare congenital platelet defect caracterized by an alpha granule deficiency and is transmitted on the dominant feature. To be able to detect this abnormality before birth we have measured the platelet content of alpha granules.The amount of Beta thromboglobulin (gTG) at 18 weeks of intrauterine life was32±4.3 mg/109 platelets in normal platelets (adults 60 mg/10^ platelets). The foetus of the mother with grey platelet syndrome was sampled at 19 weeks when the mother was under platelet transfusion and the platelets were studied by electron microscopy and for their BTG content. The platelet morphologyshowed the presence of alpha granules and the $TG content was in the range of the control fetuses (42mg/109 platelets). The baby was born after artificial delivery under platelet transfusion. These results showed that the antenatal diagnosis of thrombopathies is feasible and can permit a therapy to avoid dramatic haemorrhage during pregnancy or delivery.
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Khandoker, Ahsan H., Chihiro Yoshida, Yoshiyuki Kasahara, Kiyoe Funamoto, Kana Nakanishi, Miyabi Fukase, Keiichi Kanda, Isra Haroun, Kyuichi Niizeki, and Yoshitaka Kimura. "Regulation of maternal-fetal heart rates and coupling in mice fetuses." In 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8513463.

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Neocleous, C. K., K. H. Nicolaides, K. C. Neokleous, and C. N. Schizas. "Artificial neural networks for non-invasive chromosomal abnormality screening of fetuses." In 2010 International Joint Conference on Neural Networks (IJCNN). IEEE, 2010. http://dx.doi.org/10.1109/ijcnn.2010.5596357.

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Gelashvili, O. A., I. R. Shalneva, T. P. Fedorenko, Ia M. Komleva, and V. D. Kuper. "Proportional correlations of the main anthropometric indices children and human fetuses." In Scientific achievements of the third millennium. SPC "LJournal", 2018. http://dx.doi.org/10.18411/scc-05-2018-08.

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Reports on the topic "Fetuses"

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Pirnareva, Elena. Monitoring of Fetuses with Intrauterine Growth Restriction: a Longitudinal Study. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, April 2018. http://dx.doi.org/10.7546/crabs.2018.03.17.

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Marchionni, Enrica, Daniele Guadagnolo, Gioia Mastromoro, and Antonio Pizzuti. Diagnostic yield of prenatal Exome Sequencing in fetal Central Nervous System Anomalies: systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2023. http://dx.doi.org/10.37766/inplasy2023.5.0003.

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Review question / Objective: The aim of this study is to assess the incremental diagnostic yield of prenatal exome sequencing analysis after inconclusive result of karyotype and Chromosomal Microarray Analysis in Central Nervous System fetal anomalies detected by ultrasound. Eligibility criteria: Inclusion criteria: papers describing fetuses with the indication to perform genome-wide sequencing studies based on prenatal imaging findings who underwent previous inconclusive karyotype and Chromosomal Microarray Analyses. The diagnostic yields of prenatal exome sequencing analysis OR prenatal genome sequencing analysis (with ≥20–30x depth of coverage and including only Single Nucleotide Variants) will be pooled in a meta-analysis. Exclusion Criteria: case reports and papers describing less than 5 cases; papers not describing the application of genome-wide sequencing studies based on prenatal imaging findings; papers describing genome-wide sequencing studies performed after negative targeted panels; papers describing fetuses with recurrent phenotypes as an explicitly selection criterium.
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Tse, Kai Yeung, Ilham Utama Surya, Rima Irwinda, Kwok Yin Leung, Yuen Ha Ting, Ye Cao, and Kwong Wai Choy. Diagnostic Yield of Exome Sequencing in Fetuses with Sonographic Features of Skeletal Dysplasias but Normal Karyotype or Chromosomal Microarray Analysis: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2023. http://dx.doi.org/10.37766/inplasy2023.5.0048.

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Baszler, Timothy, Igor Savitsky, Christopher Davies, Lauren Staska, and Varda Shkap. Identification of bovine Neospora caninum cytotoxic T-lymphocyte epitopes for development of peptide-based vaccine. United States Department of Agriculture, March 2006. http://dx.doi.org/10.32747/2006.7695592.bard.

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The goal of the one-year feasibility study was to identify specific cytotoxic T-lymphocyte (CTL) epitopes to Neosporacaninum in the natural bovine host in order to make progress toward developing an effective peptide-based vaccine against bovine neosporosis. We tested the hypothesis that: N. caninum SRS2 peptides contain immunogenicCTLepitope clusters cross-presented by multiple bovine MHC-I and MHC-IIhaplotypes. The specific objectives were: (1) Map bovine CTLepitopes of N. caninum NcSRS-2 and identify consensus MHC-I and class-II binding motifs; and (2) Determine if subunit immunization with peptides containing N. caninum-specificCTLepitopes cross-reactive to multiple bovine MHChaplotypes induces a CTL response in cattle with disparate MHChaplotypes. Neosporosis is a major cause of infectious abortion and congenital disease in cattle, persisting in cattle herds via vertical transmission.5 N. caninum abortions are reported in Israel; a serological survey of 52 Israeli dairy herds with reported abortions indicated a 31% infection rate in cows and 16% infection rate in aborted fetuses.9,14 Broad economic loss due to bovine neosporosis is estimated at $35,000,000 per year in California, USA, and $100,000,000 (Australian) per year in Australia and New Zealand.13 Per herd losses in a Canadian herd of 50 cattle are estimated more conservatively at $2,305 (Canadian) annually.4 Up to date practical measures to reduce losses from neosporosis in cattle have not been achieved. There is no chemotherapy available and, although progress has been made toward understanding immunity to Neospora infections, no efficacious vaccine is available to limit outbreaks or prevent abortions. Vaccine development to prevent N. caninum abortion and congenital infection remains a high research priority. To this end, our research group has over the past decade: 1) Identified the importance of T-lymphocyte-mediated immunity, particularly IFN-γ responses, as necessary for immune protection to congenital neosporosis in mice,1,2,10,11 and 2) Identified MHC class II restricted CD4+ CTL in Neosporainfected Holstein cattle,16 and 3) Identified NcSRS2 as a highly conserved surface protein associated with immunity to Neospora infections in mice and cattle.7,8,15 In this BARD-funded 12 month feasibility study, we continued our study of Neospora immunity in cattle and successfully completed T-lymphocyte epitope mapping of NcSRS2 surface protein with peptides and bovine immune cells,15 fulfilling objective 1. We also documented the importance of immune responses NcSRS2 by showing that immunization with native NcSRS2 reduces congenital Neospora transmission in mice,7 and that antibodies to NcSRS2 specifically inhibition invasion of placental trophoblasts.8 Most importantly we showed that T-lymphocyte responses similar to parasite infection, namely induction of activated IFN-γ secreting Tlymphocytes, could be induced by subunit immunization with NcSRS2 peptides containing the Neospora-specificCTLepitopes (Baszler et al, In preparation) fulfilling objective 2. Both DNA and peptide-based subunit approaches were tested. Only lipopeptide-based NcSRS2 subunits, modified with N-terminal linked palmitic acid to enhance Toll-like receptors 2 and 1 (TLR2-TLR1), stimulated robust antigen-specific T-lymphocyte proliferation, IFN-γ secretion, and serum antibody production across different MHC-IIhaplotypes. The discovery of MHC-II cross-reactive T-cellinducing parasite peptides capable of inducing a potentially protective immune response following subunit immunization in cattle is of significant practical importance to vaccine development to bovine neosporosis. In addition, our findings are more widely applicable in future investigations of protective T-cell, subunit-based immunity against other infectious diseases in outbred cattle populations.
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Castle, Manford C. Acute Effects of Organophosphorous Compounds on the Ovine Fetus. Fort Belvoir, VA: Defense Technical Information Center, June 2001. http://dx.doi.org/10.21236/ada389705.

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Teglia, Osvaldo, Noemí Borda, Alejandro García, and Rodolfo Notario. Bacteriemia recurrente debida a Campylobacter fetus en un paciente sin inmunodepresión. Buenos Aires: siicsalud.com, October 2014. http://dx.doi.org/10.21840/siic/143341.

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Perez-Perez, Guillermo I., Martin J. Blaser, and John H. Bryner. Lipopolysaccharide Structures of Campylobacter fetus are Related to Heat-Stable Serogroups. Fort Belvoir, VA: Defense Technical Information Center, January 1986. http://dx.doi.org/10.21236/ada265573.

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Zou, Juan, Huiling Chen, Shuqi Yang, Yuanchuan Zhang, Yazhou He, Xue Xiao, and Shanling Liu. Postnatal prognosis of fetus with mild and moderate isolated lateral ventriculomegaly: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0078.

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Perera, F. P. Molecular epidemiology of severe ambient air pollution on women and the developing fetus. Progress report, 15 September 1993--14 September 1994. Office of Scientific and Technical Information (OSTI), September 1994. http://dx.doi.org/10.2172/10183052.

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Perera, F. P. Molecular epidemiology of severe ambient air pollution on women and the developing fetus. Final report, September 15, 1993--September 14, 1996. Office of Scientific and Technical Information (OSTI), November 1997. http://dx.doi.org/10.2172/542067.

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