Relevant bibliographies by topics / Fetal nerve tissue

Academic literature on the topic 'Fetal nerve tissue'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 January 2023

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Journal articles on the topic "Fetal nerve tissue"

1

Anagnostou, Valsamo K., Ipatia Doussis-Anagnostopoulou, Dina G. Tiniakos, Despina Karandrea, Emmanouil Agapitos, Petros Karakitsos, and Christos Kittas. "Ontogeny of Intrinsic Innervation in the Human Thymus and Spleen." Journal of Histochemistry & Cytochemistry 55, no. 8 (April 4, 2007): 813–20. http://dx.doi.org/10.1369/jhc.6a7168.2007.

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The ontogeny of the innervation of human lymphoid organs has not been studied in detail. Our aim was to assess the nature and distribution of parenchymal nerves in human fetal thymus and spleen. We used the peroxidase immunohistochemical technique with antibodies specific to neuron-specific enolase (NSE), neurofilaments (NF), PGP9.5, S100 protein, and tyrosine hydroxylase (TH) and evaluated our results with image analysis. In human fetal thymus, NSE-, NF-, S100-, PGP9.5-, and TH-positive nerves were identified associated with large blood vessels from 18 gestational weeks (gw) onwards, increasing in density during development. Their branches penetrated the septal areas at 20 gw, reaching the cortex and the corticomedullary junction between 20 and 23 gw. Few nerve fibers were seen in the medulla in close association with Hassall's corpuscles. In human fetal spleen, NSE-, NF-, S100-, PGP9.5-, and TH-positive nerve fibers were localized in the connective tissue surrounding the splenic artery at 18 gw. Perivascular NSE-, NF-, S100-, PGP9.5-, and TH-positive nerve fibers were seen extending into the white pulp, mainly in association with the central artery and its branches, increasing in density during gestation. Scattered NSE-, NF-, S100-, PGP9.5-, and TH-positive nerve fibers and endings were localized in the red pulp from 18 gw onward. The predominant perivascular distribution of most parenchymal nerves implies that thymic and splenic innervation may play an important functional role during intrauterine life. (J Histochem Cytochem 55: 813–820, 2007)
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2

Bosse, A., K. Schwarz, E. Vollmer, and H. Kresse. "Divergent and co-localization of the two small proteoglycans decorin and proteoglycan-100 in human skeletal tissues and tumors." Journal of Histochemistry & Cytochemistry 41, no. 1 (January 1993): 13–19. http://dx.doi.org/10.1177/41.1.8417108.

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The core protein of a recently described small proteoglycan, proteoglycan-100, was localized in fetal human tissues by indirect immunocytochemistry and compared with the localization of known members of the small proteoglycan family. Co-localization of decorin and proteoglycan-100 was seen in bone tissue but decorin and proteoglycan-100 exhibited a substantially divergent distribution in fetal skin, cartilage and in the mineralization zone of the growth plate. Proteoglycan-100 was also found in striated muscle, nerve fibers, and synovial tissue. Immunostaining of a chondroblastic osteosarcoma demonstrated chondroid cells selectively expressing either proteoglycan-100 or decorin. Co-expression of both small proteoglycans was observed in sections from a chordoma. In fetal bone and in the two tumors, colocalization of proteoglycan-100 and of biglycan was also found. These results provide evidence of the wide and characteristic distribution of proteoglycan-100.
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Kjær, Inger. "Nerve tissue as an initiating factor in human fetal bone formation." Cell Differentiation and Development 27 (August 1989): 196. http://dx.doi.org/10.1016/0922-3371(89)90595-9.

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4

Panula, P., O. Häppölä, M. S. Airaksinen, S. Auvinen, and A. Virkamäki. "Carbodiimide as a tissue fixative in histamine immunohistochemistry and its application in developmental neurobiology." Journal of Histochemistry & Cytochemistry 36, no. 3 (March 1988): 259–69. http://dx.doi.org/10.1177/36.3.3343510.

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The object of this study was to develop an immunohistochemical method that could be used to study neuronal histamine, especially in nerve fibers and terminals where most previous methods have not been applicable. Three new antisera were produced in rabbits against conjugated histamine, and the fixative used in conjugation, 1-ethyl-3(3-diamethylaminopropyl)-carbodiimide (EDCDI), was used in tissue fixation and compared to paraformaldehyde. Specificity of the antisera was established with dot-blot tests on nitrocellulose, with blocking controls and affinity-purified antibodies. EDCDI appeared to be superior to paraformaldehyde as a fixative, and histamine-immunoreactive nerve cells were visualized in developing rat brain during late fetal development from embryonal day 12. By the second postnatal week, the distribution of histamine-immunoreactive neurons in rat brain had reached the adult pattern and immunoreactive nerve fibers were seen in many areas. Posterior hypothalamic neurons from newborn rat in vitro showed strong immunoreactivity for histamine and developed long varicose fibers, which covered the culture dish by the end of the fourth week in vitro. Fixation with EDCDI also allowed detection of histamine in gastric enterochromaffin-like cells and mast cells in rat. The results suggest that the histamine-containing neuron system in rat brain develops during the late fetal and early postnatal periods, and that immunoreactive neurons develop long fibers both in vivo and in vitro.
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5

Stromberg, Ingrid, Lars Björklund, and Petter Forander. "The Age of Striatum Determines the Pattern and Extent of Dopaminergic Innervation: a Nigrostriatal Double Graft Study." Cell Transplantation 6, no. 3 (May 1997): 287–96. http://dx.doi.org/10.1177/096368979700600311.

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In animal models of Parkinson's disease, transplanted fetal mesencephalic dopaminergic neurons can innervate the dopamine-depleted host brain, but it is unclear why large portions of the host striatum are left uninnervated. During normal development, the dopaminergic innervation first occurs in the form of a dense patchy pattern in the striatum, followed by a widespread nerve fiber network. Using intraocular double grafts we have investigated dopaminergic growth patterns initiated when ventral mesencephalic grafts innervate striatal targets. The fetal lateral ganglionic eminence was implanted into the anterior eye chamber. After maturation in oculo, fetal ventral mesencephalon was implanted and placed in contact with the first graft. In other animals the two pieces of tissue were implanted simultaneously. Tyrosine hydroxylase (TH) immunohistochemistry revealed a pattern of dense TH-positive patches throughout the total volume of the striatal grafts in simultaneously transplanted cografts, while a widespread, less dense, pattern was found when mature striatal transplants were innervated by fetal dopaminergic grafts. To investigate which type or types of growth patterns that developed after grafting to striatum in situ of an adult host, fetal ventral mesencephalic tissue was implanted into the lateral ventricle adjacent to the dopamine-lesioned striatum. After maturation of the mesencephalic graft, the fetal lateral ganglionic eminence was implanted into the reinnervated part of the host striatum. TH immunohistochemistry revealed a few nerve fibers within the striatal graft and the growth pattern was of the widespread type. In conclusion, grafted dopaminergic neurons preferably innervate mature striatum with a widespread sparse nerve fiber network, while the innervation of the immature striatum occurs in the form of dense patches. Furthermore, when the patchy pattern is formed, the total volume of the striatal target is innervated while growth of the widespread type terminates prior to reaching distal striatal parts. Thus, the growth pattern seems essential to the final volume that is innervated. Once the widespread growth pattern is initiated, the presence of immature striatum does not change the dopaminergic growth pattern.
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6

Nishiura, Hayate, Shino Jou, Toru Ogata, Hiroki Kondo, Toshihiro Ichijo, Jun Sasaki, and Kenji Ochiai. "Calving-related intradural avulsion injuries of the thoracolumbar spinal nerve roots in a calf." Journal of Veterinary Diagnostic Investigation 32, no. 6 (September 15, 2020): 968–71. http://dx.doi.org/10.1177/1040638720957640.

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Calving difficulty may lead to traumatic peripheral nerve injury. A male, 8-mo-old, Japanese Black calf with a history of secondary dystocia as a result of fetal gigantism had lameness and gait disturbance. At autopsy, multifocal dural thickening with adhesions to the adjacent spinal cord was observed at T12–13 and L4–5 vertebral levels. Microscopically, numerous traumatic neuroma-like fascicles of nerve twigs were embedded in the dura mater with abundant collagenous connective tissue. By immunohistochemistry, axons and Schwann cells were confirmed in each nerve fascicle. Our observations suggest that avulsion injuries in the preganglionic fibers of the spinal nerve roots, and secondary spinal cord compression, resulted in the development of neurologic signs.
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7

Fitzgibbon, T., and B. E. Reese. "Organization of retinal ganglion cell axons in the optic fiber layer and nerve of fetal ferrets." Visual Neuroscience 13, no. 5 (September 1996): 847–61. http://dx.doi.org/10.1017/s095252380000910x.

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AbstractPrevious authors have hypothesized that retinotopic projections may be influenced by ‘preordering’ of the axons as they grow towards their targets. In some nonmammalian species, axons are reorganized at or near the optic nerve head to establish a retinotopic order. Data are ambiguous concerning the retinotopy of the mammalian retinal nerve fiber layer and whether fibers become reorganized at the optic nerve head. We have examined this question in fetal and newborn ferrets (Mustela putorius furo) by comparing the arrangement of axons in the retinal nerve fiber layer with that in the optic nerve. Dil or DiA crystals were implanted into fixed tissue in the innermost layers of the retinal periphery, or at a location midway between the periphery and the optic nerve head. Fluorescence labelling was examined in 100–200 μm Vibratome sections, or the eyecup and nerve were photooxidized and 1–2 μm longitudinal or transverse sections were examined. Regardless of fetal age, eccentricity or quadrant of the implant site, a segregation of labelled peripheral axons from unlabelled central ones was not detected within the nerve fiber layer. Axons coursed into the nerve head along the margin of their retinal quadrant of origin, often entering the optic nerve as a radial wedge, thus preserving a rough map of retinal circumference. However, peripheral axons were in no way restricted to the peripheral (nor central) portions of the nerve head or nerve, indicating that the optic axons do not establish a map of retinal eccentricity. Our results demonstrate that (1) the nerve fiber layer is retinotopic only with respect to circumferential position and (2) optic axons are not actively reorganized to establish a retinotopic ordering at the nerve head. The present results suggest that any degree of order present within the optic nerve is a passive consequence of combining the fascicles of the retinal nerve fiber layer; optic axons are not instructed to establish, nor constrained to maintain, a retinotopic order within the optic nerve.
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8

Liu, Yihe. "Molecular Biology Research on the Information Coding Mechanism of Brain Nerve Cells." Highlights in Science, Engineering and Technology 2 (June 22, 2022): 166–71. http://dx.doi.org/10.54097/hset.v2i.570.

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The function of the brain is to integrate information and make behavioral decisions accordingly. The understanding of brain function is an important part of neuroscience, and it also provides inspiration for artificial intelligence. However, how information is represented and encoded in the brain, and how it is read by other downstream tissue links, these questions have not yet been clarified. To explore the regulation of information expression by molecular biology during the in vitro development of human fetal brain neurons. These cytokines all promote the expression of neural cells at the transcriptional level, laying the foundation for analyzing the molecular mechanism of their biological effects. The function of the brain is to integrate information and make behavioral decisions accordingly. The understanding of brain function is an important part of neuroscience, and it also provides inspiration for artificial intelligence. However, how information is represented and encoded in the brain, and how it is read by other downstream tissue links, these questions have not yet been clarified. To explore the regulation of information expression by molecular biology during the in vitro development of human fetal brain neurons. These cytokines all promote the expression of neural cells at the transcriptional level, laying the foundation for analyzing the molecular mechanism of their biological effects.
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9

Khmara, T. V., L. Ya Lopushniak, O. M. Boichuk, A. A. Halahdyna, L. М. Gerasym, and M. Yu Leka. "FETAL ANATOMICAL VARIABILITY OF STRUCTURES IN INFRAHYOID AREA." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 20, no. 3 (November 12, 2020): 164–69. http://dx.doi.org/10.31718/2077-1096.20.3.164.

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When performing myoplastic operations and surgical interventions on the thyroid gland, trachea and esophagus, information on the variant anatomy of the infrahyoid muscles, the features of their innervation and blood supply are of great clinical importance. Moreover, when additional muscles are attached to the thyroid gland, intraoperative bleeding can occur resulting in hematoma and tissue scarring in the postoperative period. There are fragmentary data in the literature on the variants of the structure and topography of the human infrahyoid area muscles. The specificity of branching nerves and blood vessels, their vascular-nervous relationships in a separate part of the sternohyoid, sternothyroid, thyrohyoid, and omohyoid muscles should be taken into account when performing rational incisions in the neck, moving both the flaps and the above muscles in plastic surgery. The purpose of study was to establish the anatomical variability and features of innervation and blood supply of the infrahyoid muscles of the neck in human foetuses of 4 – 10 gestational months age. Material and methods. The study was performed on 36 human foetuses, whose parieto-coccygeal length was 81.0 – 375.0 mm, without visible signs of anatomical abnormalities or anomalies in the cervical region. Thin sections of the structures from the anterior and lateral parts of the neck were prepared under the control of binocular magnifier, vascular injection technique, and morphometry. Foetal preparations weighing over 500.0 g were studied directly at Chernivtsi Regional Paediatric Pathological Bureau. Foetal preparations were taken from the Museum of M.G. Turkevich Human Anatomy Department, Bukovinian State Medical University. Results and discussion. The study demonstrated anatomical variability of the infrahyoid area muscles during the foetal period of human ontogenesis. Human foetuses were mainly found to have loose extending intramuscular branching of the nerves of the cervical loop in the infrahyoid muscles. The only exception is the inferior belly of the omohyoid muscle, where main nerve branching is found out. The distribution of nerves in the thickness of the infrahyoid muscles is uneven. Macroscopic examination revealed the smallest number of nerve branches was found within the middle third of the sternohyoid and upper third of the sternothyroid muscles. Arteries and nerves enter the sternothyroid and thyrohyoid muscles through the anterior surface, and the omohyoid and sternohyoid muscles enter mainly through the posterior surface of these muscles. The infrahyoid muscles are characterized by the main form of intramuscular branching of the arteries. The data on the peculiarities of intramuscular branching of arteries and nerves in the infrahyoid muscles we obtained, as well as the variant anatomy of the infrahyoid area muscles must be taken into account when performing a surgical access to the neck, or when operating on in the anterior cervical region, in particular myoplastic and reconstructive operations, in order to avoid muscle injuries.
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10

Stein, Donald G., and Marylou M. Glasier. "Some practical and theoretical issues concerning fetal brain tissue grafts as therapy for brain dysfunctions." Behavioral and Brain Sciences 18, no. 1 (March 1995): 36–45. http://dx.doi.org/10.1017/s0140525x00037250.

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AbstractGrafts of embryonic neural tissue into the brains of adult patients are currently being used to treat Parkinson's disease and are under serious consideration as therapy for a variety of other degenerative and traumatic disorders. This target article evaluates the use of transplants to promote recovery from brain injury and highlights the kinds of questions and problems that must be addressed before this form of therapy is routinely applied. It has been argued that neural transplantation can promote functional recovery through the replacement of damaged nerve cells, the reestablishment of specific nerve pathways lost as a result of injury, the release of specific neurotransmitters, or the production of factors that promote neuronal growth. The latter two mechanisms, which need not rely on anatomical connections to the host brain, are open to examination for nonsurgical, less intrusive therapeutic use. Certain subjective judgments used to select patients who will receive grafts and in assessment of the outcome of graft therapy make it difficult to evaluate the procedure. In addition, little long-term assessment of transplant efficacy and effect has been done in nonhuman primates. Carefully controlled human studies, with multiple testing paradigms, are also needed to establish the efficacy of transplant therapy.
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Dissertations / Theses on the topic "Fetal nerve tissue"

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Törnqvist, Nina. "On transplantation of fetal ventral mesencephalon with focus on dopaminergic nerve fiber formation /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-177-2/.

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2

Björklund, Lars. "Intracranial and intraocular grafting of fetal ventral mesencephalic and striatal tissues : neuronal survival and nerve growth characteristics /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980828bjor.

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Tollet, Cecilia Jenny. "The origin and early development of the intrinsic innervation in the foetal mouse lung." University of Western Australia. School of Biomedical and Chemical Sciences, 2003. http://theses.library.uwa.edu.au/adt-WU2004.0060.

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In this study, the origin and development of the intrinsic innervation in the foetal mouse lung is described and experimental evidence is provided to support the involvement of glial cell line-derived neurotrophic factor (GDNF) in the guidance of nerves and neuronal precursors in the developing lung. Antibodies were used to stain for neuronal precursors, neurones, nerve fibres, primordial epithelium and smooth muscle. These structures were revealed in whole mounts of foetal mouse lungs by immunofluorescence and confocal microscopy, and their spatial and temporal distribution was mapped from the onset of lung development and through the pseudoglandular period. The results showed that neuronal precursors, positive for neural crest cell markers, were present in the vagal tract of the foregut at embryonic day 10 (E10), the time of the evagination of the lung buds. These neural crest-derived cells (NCC) migrated into the lung at E11, along nerve processes directed from the vagus to the smooth musclecovered trachea and emerging lobar bronchi. During E11-E14, a network of nerves and ganglia became established along the dorsal trachea, and large ganglia formed a plexus at the ventral hilum. Nerve trunks issued from these ganglia, travelled along the smooth muscle-covered bronchi, providing a pathway for migrating NCC. To investigate the role of GDNF in the innervation of the lung, an in vitro model of left lung lobes was established. Lung growth and tubule branching was comparable to that in vivo, and neural tissue and smooth muscle continued to grow and thrive. A significant increase in nerve growth occurred when explants were cultured with GDNF compared to controls. Nerves extended, and NCC migrated towards GDNF-impregnated beads suggesting that GDNF may be the molecule guiding nerve fibres and NCC in the lung. The migrating NCC were negative for GDNF-family receptor α1 (GFRα1) during their migration into the lung while the nerves were positive. Since GDNF needs to be associated with its binding receptor, GFRα1, for cellular signalling, GDNF may induce the migration of the NCC if they migrate along the GFRα1-positive nerve fibres. It is concluded that neural tissue and smooth muscle become integral components of the lung shortly after the onset of lung development. The results show that the migration of neural crest-derived cells into the lung and the establishment of the innervation requires coordinated cross-talk between NCC, nerves and smooth muscle throughout development.
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Books on the topic "Fetal nerve tissue"

1

Transplantation of neural tissue into the spinal cord. 2nd ed. Georgetown, Tex: Landes Bioscience, 2006.

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C, Azmitia Efrain, Björklund Anders 1945-, and New York Academy of Sciences., eds. Cell and tissue transplantation into the adult brain. New York, N.Y: New York Academy of Sciences, 1987.

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3

Olle, Lindvall, ed. Restoration of brain function by tissue transplantation. Berlin: Springer-Verlag, 1993.

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Sanberg, Paul R. Cell transplantation for Huntington's disease. Austin: R.G. Landes, 1994.

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Sanberg, Paul R. Cell transplantation for Huntington's disease. Austin: R.G. Landes, 1994.

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Transplantation of neural tissue into the spinal cord. Austin: R.G. Landes, 1994.

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Gerta, Vrbová, ed. Transplantation of neural tissue into the spinal cord. Austin: R.G. Landes, 1994.

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B, Dunnett S., and Björklund Anders 1945-, eds. Neural transplantation: A practical approach. Oxford: IRL Press, 1992.

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Brundin, P., K. Wictorin, Olle Lindvall, and A. Björklund. Restoration of Brain Function by Tissue Transplantation. Springer London, Limited, 2013.

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1955-, Freeman Thomas B., and Widner Hakan, eds. Cell transplantation for neurological disorders: Toward reconstruction of the human central nervous system. Totowa, N.J: Humana Press, 1998.

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Book chapters on the topic "Fetal nerve tissue"

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Sørensen, J. Chr, A. J. Castro, E. J. Neafsey, and J. Zimmer. "Reduction of Post-lesional Atrophy by Transplants of Fetal Cerebral Cortical Tissue. Host-Transplant Nerve Connections and Trophic Factors." In Brain Repair, 133–43. London: Macmillan Education UK, 1990. http://dx.doi.org/10.1007/978-1-349-11358-3_10.

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