Relevant bibliographies by topics / Fetal effects

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Fetal effects'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Fetal effects"

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Chiriboga, Claudia A. "Fetal Effects." Neurologic Clinics 11, no. 3 (August 1993): 707–28. http://dx.doi.org/10.1016/s0733-8619(18)30147-6.

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ONGUN, Hakan, Kıymet ÇELİK, and Nihal OYGÜR. "Chorioamnionitis and Its Fetal Effects." Turkiye Klinikleri Journal of Pediatrics 29, no. 3 (2020): 175–86. http://dx.doi.org/10.5336/pediatr.2020-76142.

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Hanson, James W. "Fetal hydantoin effects." Teratology 33, no. 3 (June 1986): 349–53. http://dx.doi.org/10.1002/tera.1420330314.

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Goldman, Jacquelin. "Fetal Alcohol Syndrome and Fetal Alcohol Effects." Journal of Clinical Child Psychology 14, no. 1 (March 1985): 82. http://dx.doi.org/10.1207/s15374424jccp1401_14.

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Houme, H. Eugene, and Carol L. Clericuzio. "1295 FETAL PRIMIDONE EFFECTS." Pediatric Research 19, no. 4 (April 1985): 326A. http://dx.doi.org/10.1203/00006450-198504000-01319.

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Woodson, R. H. "Review of Fetal Alcohol Syndrome and Fetal Alcohol Effects." Contemporary Psychology: A Journal of Reviews 30, no. 7 (July 1985): 584. http://dx.doi.org/10.1037/023959.

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Alvear, J., S. Andreani, P. Vargas, F. Cortes, and P. De Valdivia. "Fetal Alcohol Syndrome and Fetal Alcohol Effects, Psicomotor Development20." Pediatric Research 42, no. 6 (December 1997): 923. http://dx.doi.org/10.1203/00006450-199712000-00053.

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Frost, Mackenzie S., Aqib H. Zehri, Sean W. Limesand, William W. Hay, and Paul J. Rozance. "Differential Effects of Chronic Pulsatile versus Chronic Constant Maternal Hyperglycemia on Fetal Pancreaticβ-Cells." Journal of Pregnancy 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/812094.

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Constant maternal hyperglycemia limits, while pulsatile maternal hyperglycemia may enhance, fetal glucose-stimulated insulin secretion (GSIS) in sheep. However, the impact of such different patterns of hyperglycemia on the development of the fetalβ-cell is unknown. We measured the impact of one week of chronic constant hyperglycemia (CHG,n=6) versus pulsatile hyperglycemia (PHG,n=5) versus controls (n=7) on the percentage of the fetal pancreas staining for insulin (β-cell area), mitotic and apoptotic indices and size of fetalβ-cells, and fetal insulin secretion in sheep. Baseline insulin concentrations were higher in CHG fetuses (P<0.05) compared to controls and PHG. GSIS was lower in the CHG group (P<0.005) compared to controls and PHG. PHGβ-cell area was increased 50% (P<0.05) compared to controls and CHG. CHGβ-cell apoptosis was increased over 400% (P<0.05) compared to controls and PHG. These results indicate that late gestation constant maternal hyperglycemia leads to significantβ-cell toxicity (increased apoptosis and decreased GSIS). Furthermore, pulsatile maternal hyperglycemia increases pancreaticβ-cell area but did not increase GSIS, indicating decreasedβ-cell responsiveness. These findings demonstrate differential effects that the pattern of maternal hyperglycemia has on fetal pancreaticβ-cell development, which might contribute to later life limitation in insulin secretion.
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Saji, Haruya, Michiko Yamanaka, Akiko Hagiwara, and Rieko Ijiri. "Losartan and fetal toxic effects." Lancet 357, no. 9253 (February 2001): 363. http://dx.doi.org/10.1016/s0140-6736(00)03648-5.

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Froelich, M. A., T. Y. Euliano, and D. Caton. "FETAL EFFECTS OF MATERNAL ANALGOSEDATION." Anesthesiology 96, no. 4 (April 1, 2002): NA. http://dx.doi.org/10.1097/00000542-200201000-00100.

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Dissertations / Theses on the topic "Fetal effects"

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Beckett, Cynthia Diane. "Navajo children and families living with fetal alcohol syndrome/fetal alcohol effects." Diss., The University of Arizona, 2002. http://hdl.handle.net/10150/280150.

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The aim of the study was to develop a culturally sensitive Grounded Theory of Navajo parenting for families who are living with Fetal Alcohol Syndrome (FAS)/Fetal Alcohol Effects (FAE). The research question was: What are the social and cultural factors and processes that Navajo families use to mange care for a child with FAS/FAE? The philosophical perspectives that guided the study were: the Navajo philosophy, or view of life; resilience (middle range theory); the Family Stress Theory; and the Resiliency Mode of Family Stress, Adjustment, and Adaptation. Resilience was used as the over arching conceptual perspective for the study. A Grounded Theory of Navajo Parenting emerged from the data. Key categories to support the emerging theory were identified. The core category was Versatility through Transcendence. The supporting categories were: Strategies for Managing Challenges; Transcendence in Parenting; Intergenerational Alcohol Abuse, Violence and Suffering; and Knowledge/Acquisition of Needs. The families described their stories of transcendence through substance abuse, suffering, and violence to be able to parent their children who were living with the primary and secondary challenges of prenatal alcohol exposures. Further research is needed to test and expand this emerging theory of Navajo parenting of children with FAS/FAE. The challenges that were related to FAS/FAE were more easily managed with patterns of resilience within the families. Factors that influenced family's abilities to parent will be disseminated to assist other families who are managing the problems associated with FAS/FAE.
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Webber, Troy A. "Fetal Testosterone: Developmental Effects on Externalizing Behavior." Scholar Commons, 2015. https://scholarcommons.usf.edu/etd/7376.

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Fetal testosterone (FT) exposure influences sexual differentiation and may promote well-established sex differences in externalizing (EXT) behavior. Although puberty may be a critical period for these effects, it is unknown how FT exposure influences EXT as a function of pubertal development. We used a longitudinal, multi-sample design to test the relationships between two proxy indices of FT exposure and EXT as a function of age and pubertal development (approximately ages 6, 9, 11, 14, and 16). Twin data were used to approximate FT exposure (TT-FT) because testosterone is thought to cross the intrauterine membrane and cause variability in co-twin gonadal hormone exposure, with increasing exposure for males and participants with male co-twins. Increasing number of older siblings may also approximate increasing FT exposure (SI-FT), although existing research has yet to disentangle possible postnatal socialization effects from potential FT exposure using this variable. Given that biologically related siblings share a fetal and social environment while non-biologically related siblings simply share a social environment, we tested the independent effect of SI-FT on EXT using a sibling adoption design. Across four independent samples, SI-FT and TT-FT predicted externalizing for males alone. SI-FT predicted EXT over-and-above socialization influences and interacted with pubertal development in two independent samples, with elevated EXT for those in mid-late puberty that were exposed to increased FT. TT-FT predicted EXT differentially as a function of developmental period. Our data are consistent with the notion that exposure to FT promotes sexually differentiated, sexually selected behavior during reproductively relevant periods.
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Conliffe, Phyllis R. (Phyllis Rowena). "Effects of maternal diabetes on fetal development in rats." Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=39344.

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The mechanisms underlying the high incidence of fetal abnormalities including fetal lung immaturity during maternal diabetes are not fully understood. Utilizing streptozotocin-diabetic rats as the model, I have examined the role of fetal hyperglycemia and hyperinsulinemia and other factors on fetal adrenal and lung functions in culture. Insulin and glucose did not alter fetal adrenal and lung cell proliferation and adrenal corticosterone output. On the other hand, a novel protein-bound, low molecular weight non-proteinaceous cytotoxic factor was detected in the serum of diabetic animals. In addition, a novel protein with cytostatic activity was found in fetal lungs, the concentration of which increased during diabetes. Partial amino acid sequence and Western Blot analysis revealed this protein to be similar to histone H2B. An extra-nuclear role is suggested for this protein because it appears to be present in the microsomal fraction of fetal lungs. It is concluded that fetal lung immaturity during diabetes may be contributed by cytotoxic and cytostatic factors contained in the serum and fetal lungs, respectively.
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Kelly, Mary Clare. "Maternal and fetal effects of intrathecal analgesia in obstetrics." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361287.

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Arkwright, P. D. "Effects of the human trophoblast on lymphocyte proliferation." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236250.

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Barrett, Robert Daniel. "Therapeutic hypothermia and its effects on the preterm fetal sheep." Thesis, University of Auckland, 2011. http://hdl.handle.net/2292/7200.

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There is compelling evidence that 72 h of moderate hypothermia initiated within 2 to 6 h after hypoxia-ischemia can protect against brain injury, disability and death in term newborn infants. Currently, there is no clinical treatment for hypoxic-ischemic encephalopathy for preterm infants. With the worldwide rates of preterm birth steadily increasing, there is much interest in using therapeutic hypothermia to treat preterm hypoxic-ischemic encephalopathy. The goal of this thesis was to investigate the effects and window of opportunity of therapeutic hypothermia on the brain and physiology after asphyxia in preterm fetal sheep. My first study showed that 68.5 h of selective head cooling, initiated 90 min after asphyxia, protected oligodendrocytes in the white matter (WM) and subventricular zone (SVZ) of the preterm fetal sheep brain at 3 days recovery from 25 min of umbilical cord occlusion. Overall proliferation of cells was not reduced by hypothermia in the WM or SVZ. The remainder of the studies focused on the use of 72 h of whole body hypothermia, and assessed effects at 7 days recovery from asphyxia. Two hypothermia protocols were examined, a 30 min onset after asphyxia protocol, and a clinically relevant, 5 h after asphyxia protocol. Whole body hypothermia was associated with mild bradycardia, mild changes in blood pressure and carotid blood flow and transitory suppression of EEG power. All physiological variables resolved to sham values by 96 h after asphyxia. Delayed hypothermia was associated with slower improvement of spectral edge frequency and EEG power than early onset hypothermia. The window of opportunity for SVZ protection was less than 5 h, with significant improvement in numbers of oligodendrocytes after only early onset but not delayed hypothermia. In contrast, there was no significant improvement in number of oligodendrocytes in the white matter tracts, with either early or late cooling. This was associated with reduced proliferation in the white matter, and no induction of microglia and caspase 3, which suggests that lack of replenishment of oligodendrocytes may have contributed to persistent reduction in numbers of oligodendrocytes after therapeutic hypothermia. Overall the studies in this thesis suggest that the window of opportunity for brain protection in the preterm infant is less than 5 hours and that synergistic treatment may be required to protect the WM after hypoxic-ischemic insults.
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Delorme, Danielle. "Effects of Interleukin-3 on murine fetal hemopoiesis in utero." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59630.

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The effect of interleukin-3 (IL-3), a candidate hemopoietic growth factor, on prenatal hemopoiesis is unclear. Microinjection of IL-3 directly into mouse fetuses (day 13) via the yolk sac, allowed us to evaluate the effects on morphogenetic events and more specifically on fetal liver populations using quantitative in vitro clonal assays for hemopoietic precursors. Control studies, required to distinguish stress effects of surgical laparotomy and microinjection, clearly revealed that the fetal liver is a sensitive organ responding with limited tissue disorganization, reduced cellularity and erythropoietic activity as identified 24 h after experimental intervention. Microinjection of 15 units of IL-3 promoted significant expansion of the depleted fetal liver hemopoietic cell populations and had stimulatory effects on connective tissue mast cells, absolute cell numbers including hemopoietic precursors (erythroid, granulocyte, macrophage, megakaryocyte) compared to controls. These studies suggest that fetal liver cells acquire a responsiveness to IL-3 early in development and that, IL-3 has a positive stimulatory effect on fetal liver cell populations, promoting the recovery of normal liver hemopoiesis.
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Samnakay, Naeem. "Antenatal bladder outflow obstruction : effects of morphology and apoptosis in the fetal kidney, and effects on fetal ACTH and cortisol levels in an ovine model." University of Western Australia. School of Women's and Infants' Health, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0151.

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Posterior urethral valves cause bladder outflow obstruction and damage to the developing fetal kidney. Posterior urethral valves affect 1 in 8000 new-born males. A third of these children develop end stage renal failure by adolescence, despite valve ablation in the early post-natal period, implying that majority of the damage to the kidneys occurs in utero. How does this damage occur, and should we intervene in utero? The answers to these questions require further research, and are the basis to this thesis. This thesis focused on the effect bladder outflow obstruction has on morphology and apoptosis in the fetal kidney in a fetal lamb model. It also looked at the effect of bladder outflow obstruction on fetal stress hormone levels. Bladder outflow obstruction was created surgically in fetal lambs at day 70 of gestation, and fetal kidneys were analysed at day 2, 5, 10, 20 and 30 after creation of obstruction. Controls undergoing sham surgery were used for comparison. Four aspects were investigated: - effects of bladder outflow obstruction on renal histology effects of bladder outflow obstruction on expression of pro-apoptosis gene Bax and anti-apoptosis gene Bcl-X - effects of bladder outflow obstruction on renal regional apoptosis effects of bladder outflow obstruction on serum fetal ACTH and cortisol levels. Bladder outflow obstruction resulted in sequential morphological change in the fetal kidney over time. By 2 days post-obstruction, cystic change was noted. In addition, patchy attenuation of the nephrogenic blastema was evident by 5 days post-obstruction, with more confluent blastemal attenuation as well as generalized renal architectural disorganization by 10 days post-obstruction. By 20 and 30 days post-obstruction, cystic renal dysplasia had developed. Bladder outflow obstruction resulted in an increase in the ratio of renal expression of pro-apoptosis gene Bax to anti-apoptosis gene Bcl-X. Regional apoptosis counts showed increased tubular apoptosis compared to controls at 2 days post-obstruction, and increased blastemal apoptosis compared to controls at 5 days post-obstruction. By 10 days post-obstruction, blastemal apoptosis counts were reduced compared to controls. There were no significant differences in fetal serum ACTH and cortisol levels between fetal lambs with bladder outflow obstruction and controls. In conclusion, the results of this thesis outline the spectrum of morphological change in the fetal kidney over 30 days of bladder outflow obstruction. They show that detectable changes in morphology occur within two days of bladder outflow obstruction. Likewise, detectable changes in gene expression occur within 2 days of bladder outflow obstruction. The increased ratio of expression of Bax to Bcl-X suggests a swing towards increased apoptosis in response to bladder outflow obstruction. Further research is required to ascertain if these changes are reversible. However, the early onset of these changes as shown in this thesis suggests that any fetal intervention to protect the fetal kidney from the effects of bladder outflow obstruction may need to be instituted very early in gestation
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Almeida, Nisha. "Measures of maternal tobacco smoke exposure and foetal growth." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112375.

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Objective. Most biomarker studies of maternal smoking have been based on a single blood or urinary cotinine value, which is inadequate in capturing maternal tobacco exposure over the entire pregnancy. This thesis used maternal hair biomarkers to investigate the association between maternal active and passive smoking, and birthweight for gestational age (BW for GA).
Methods. Subjects were 444 term controls drawn from 5,337 participants of a multi-centre nested case-control study of preterm birth in Montreal. Maternal hair, collected after delivery, was measured for average nicotine and cotinine concentration across the pregnancy, assuming hair growth of 1 cm/month. The BW for GA z-score used Canadian population-based standards. Multiple linear regression was used to assess effects on the z-score, after controlling for potential confounders.
Results. In regression models for maternal active smoking analysis, the addition of hair nicotine to models containing either self-report or hair cotinine or both self-report and cotinine explained significantly more variance in the BW for GA z-score (p=0.009, p=0.017, and p=0.033, respectively). In maternal passive smoking analysis, no significant effect of ETS on BW for GA was found using hair biomarkers.
Conclusion. These results indicate that hair biomarkers are sensitive tools capable of predicting reductions in birthweight for maternal active smoking. The stronger results obtained for nicotine are reflective of the fact that hair nicotine is a better measure of maternal smoking, but it could also suggest that nicotine plays an aetiologic role in affecting foetal growth.
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Heiberg, Ludvig. "An electrophoretic study of fetal mouse brain proteins after in vivo exposure to phenytoin and disulfiram." Master's thesis, University of Cape Town, 1990. http://hdl.handle.net/11427/27187.

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Although there have been two-dimensional electrophoretic studies on fetal brain tissue (for instance, Yoshida and Takahashi, 1980), the emphasis in most of this work has been on developmental changes in protein expression, and not on the effects that drugs have on fetal brain protein complement. Klose and co-workers (1977) did an early study using two-dimensional gel electrophoresis to determine the effects of various teratogens on whole embryos. No protein changes were found and that line of research was not continued. In this study two-dimensional gel electrophoresis is extensively used, in the belief that the usefulness of this technique to experimental teratology has not been fully evaluated. It is reasonable to suppose that a central nervous system teratogen administered during critical periods of susceptibility will led to perturbations of orderly brain development, and that these perturbations will be reflected as changes to the protein complement. The total brain protein complement of mice that have been exposed to drugs in utero will therefore be analysed, in the hope that any inductions or deletions of proteins as a result of drug exposure may provide a clue to the molecular events underlying drug injury to the fetus.
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Books on the topic "Fetal effects"

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Fetal alcohol syndrome. Oradell, N.J: Medical Economics Books, 1990.

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Malbin, Diane. Trying differently rather than harder: Fetal alcohol syndrome and fetal alcohol effects. [Salem, Or.]: State Office for Services to Children and Families, Oregon Dept. of Human Services, 1999.

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Anderson, Kim. Aboriginal approaches to fetal alcohol syndrome-effects. Edited by Ontario Federation of Indian Friendship Centres. Toronto, Ont: Ontario Federation of Indian Friendship Centres, 2002.

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Legge, Carole. Situational analysis: Fetal alcohol syndrome/fetal alcohol effects and the effects of other substance use during pregnancy. Ottawa: Health Canada, 2001.

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Roberts, Gary. Best practices: Fetal alcohol syndrome/fetal alcohol effects and the effects of other substance use during pregnancy. [Ottawa]: The Division, 2000.

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McCreight, Brenda. Recognizing and managing children with fetal alcohol syndrome/fetal alcohol effects: A guidebook. Washington, DC: CWLA Press, 1997.

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Fetal alcohol exposure and effects: A comprehensive bibliography. Westport, Conn: Greenwood Press, 1985.

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McTimoney, David Clifford. Your child! our future!: Fetal alcohol syndrome and fetal alcohol effects : educational package and resource kit. Oromocto, N.B: Oromocto Indian Nation, 1993.

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Fetal alcohol syndrome: An annotated bibliography. New York: Praeger, 1986.

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Canada, Canada Health. Best practices: Fetal alcohol syndrome : Fetal alchol effects and the effects of other substance use during pregnancy : prepared by Gary Roberts and Jo Nanson. Ottawa: Health Canada, 2001.

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Book chapters on the topic "Fetal effects"

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Schlotz, Wolff. "Fetal Effects." In Encyclopedia of Adolescence, 1022–33. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1695-2_333.

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Schlotz, Wolff. "Fetal Effects." In Encyclopedia of Adolescence, 1400–1413. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-33228-4_333.

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Pettoni, Ashley N. "Fetal Alcohol Effects." In Encyclopedia of Child Behavior and Development, 649–50. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_1125.

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Ward, Tracey, Raphael Bernier, Cora Mukerji, Danielle Perszyk, James C. McPartland, Ellen Johnson, Susan Faja, et al. "Fetal Alcohol Effects." In Encyclopedia of Autism Spectrum Disorders, 1277. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_100596.

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Dobson, Christine C., Parker J. Holman, Wendy Comeau, Tamara Bodnar, Vivian Lam, James F. Brien, James N. Reynolds, and Joanne Weinberg. "The Effects of Alcohol Exposure on Fetal Development." In Fetal Development, 331–64. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22023-9_17.

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Snow, M. H. L. "Environmental influences on fetal growth: effects and consequences." In Fetal Growth, 115–25. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1707-0_11.

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Ronca, April E., and Jeffrey R. Alberts. "Fetal and Birth Experiences: Proximate Effects, Developmental Consequences, Epigenetic Legacies." In Fetal Development, 15–42. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22023-9_2.

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Hanley, Gillian, Kaia Hookenson, Dan Rurak, and Tim F. Oberlander. "Fetal Effects of In Utero Serotonin Reuptake Inhibitor (SRI) Antidepressant Exposure." In Fetal Development, 365–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22023-9_18.

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Glover, Vivette, Yousra Ahmed-Salim, and Lauren Capron. "Maternal Anxiety, Depression, and Stress During Pregnancy: Effects on the Fetus and the Child, and Underlying Mechanisms." In Fetal Development, 213–27. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22023-9_12.

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Rush, D. "Effects of changes in maternal energy and protein intake during pregnancy, with special reference to fetal growth." In Fetal Growth, 203–29. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1707-0_21.

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Conference papers on the topic "Fetal effects"

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Maulina, Rufidah, Su-Chen Kuo, Chieh Yu Liu, and Yu-Ying Lu. "The Mediation Effect of Health Behavior on the Relationship Between Maternal Depression and Maternal-Fetal Attachment." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.02.40.

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Background: Numerous studies have shown the adverse effects of maternal depression, which impacts both mother and child as well as can lower the maternal-fetal attachment. However, during pregnancy, a pregnant woman tends to practice healthier behavior to improve her health and the baby. A gap remains in our understanding of the effect of health behavior as the variable which influences the relationship between depression and maternal-fetal attachment. This study aimed to investigate the mediating effect of healthy behavior on the relationship between maternal depression and maternal-fetal attachment. Subjects and Method: A cross sectional study was conducted at Community Health Centers in Surakarta, from July to September 2019. A sample of 224 pregnant women was selected for this study. The dependent variable was a healthy lifestyle. The independent variable was depression and maternal-fetal attachment. Depression was measured by Edinburgh Postpartum Depression Scale (EPDS). The data were analyzed by Hayes’ process mediation analysis. Results: Health-promoting lifestyle totally mediated the relationship between maternal depression and maternal-fetal attachment (b= -0.25; SE= 0.10; 95% CI= -0.47 to 0.05). Conclusion: Health-promoting lifestyle and behavior mediates the relationship between maternal depression and maternal-fetal attachment. Keywords: Nursing, midwife, maternal-fetal attachment, prenatal depression, health-promoting lifestyle Correspondence: Rufidah Maulina. National Taipei University of Nursing and Health Sciences. Taipei, Taiwan. Email: rufidahmaulina@gmail.com. Mobile: +6282221525673. DOI: https://doi.org/10.26911/the7thicph.02.40
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Manlin Wu, Yuhao Chen, Haixia Fu, Bingchun Liu, Xinjie Cui, Shuyu Li, and Zhigang Wang. "The novel mTOR inhibitor RAD001 (Eeverolimus) induces antiproliferative effects in goat fetal fibroblasts." In 2011 International Conference on Remote Sensing, Environment and Transportation Engineering (RSETE). IEEE, 2011. http://dx.doi.org/10.1109/rsete.2011.5964203.

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Crismale-Gann, Catina, Tiffany A. Polanco, Hillary Stires, and Wendie S. Cohick. "Abstract 3595: Effects of fetal alcohol exposure on tumor development and gene expression in rats." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3595.

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Li, Xiangrui, Jasmine Hect, Moriah Thomason, and Dongxiao Zhu. "Interpreting Age Effects of Human Fetal Brain from Spontaneous fMRI Using Deep 3D Convolutional Neural Networks." In 2020 IEEE 17th International Symposium on Biomedical Imaging (ISBI). IEEE, 2020. http://dx.doi.org/10.1109/isbi45749.2020.9098606.

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Raghunathan, Raksha, Chih-Hao Liu, Jessica Gutierrez, Manmohan Singh, Rajesh C. Miranda, and Kirill V. Larin. "Optical coherence angiography to assess the combined effects of alcohol and nicotine on fetal brain vasculature." In Optical Coherence Tomography and Coherence Domain Optical Methods in Biomedicine XXV, edited by Joseph A. Izatt and James G. Fujimoto. SPIE, 2021. http://dx.doi.org/10.1117/12.2583732.

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Liao, X. P., X. J. Zhou, C. H. Yan, W. L. Zhang, and X. D. Yu. "Notice of Retraction: The Effects of Season on Fetal Bone Development: A Study from Chinese Newborn Infants." In 2011 5th International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2011. http://dx.doi.org/10.1109/icbbe.2011.5781308.

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Brockmeyer, T., R. Williams, AB Knoll, S. Murray, HC Nielsen, and CE Dammann. "Effects of Fetal Rat Lung Type II Cells and Fibroblasts on Bone Marrow Mesenchymal Stem Cell Behavior." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3275.

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Gungor, Baris Doruk, Muhammed Kursad Ucar, and Ferda Bozkurt. "Statistical investigation of the effects of fetal heart rate (FHR) and uterine contractions (UC) signals on apgar score." In 2015 23th Signal Processing and Communications Applications Conference (SIU). IEEE, 2015. http://dx.doi.org/10.1109/siu.2015.7129858.

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Kowalski, William J., Berk M. Yigit, David J. R. Hutchon, and Kerem Pekkan. "Transition From the Fetal to Neonatal Circulation: Modeling the Effect of Umbilical Cord Clamping." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14431.

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The transition from fetal to neonatal circulation requires a concert of events to transfer gas exchange function from the placenta to the lungs and separate the pulmonary and systemic pathways. Pulmonary vascular resistance (PVR) rapidly decreases within the first minutes of extrauterine life and continues to gradually decrease during the first week, increasing pulmonary blood flow and reducing pulmonary pressure [1, 2]. Umbilical vessels constrict, removing the placental circulation and leading to closure of the ductus venosus (DV) [2]. The increased left atrial filling and reduced right atrial filling results in permanent closure of the flap of the foramen ovale, removing the R→L interatrial shunt. Closure of the ductus arteriosus (DA) completes the separation of the pulmonary and systemic circulations by 48 hours in 82% of term newborns and by 96 hours in 100% [3]. Removal of the placental circulation is routinely achieved by umbilical cord clamping (UCC) immediately after birth. This practice, however, has been called into question by many studies, which suggest that continued umbilical flow in the early neonate is beneficial, and immediate UCC can lead to infant anemia [4, 5]. Due to routine UCC, the effects of this practice on transitional flow patterns are largely unknown [1, 6]. We therefore developed a lumped parameter model (LPM) to study the role of UCC in the fetal to neonatal transition. Our model includes time-varying resistance functions that allow us to simulate the opening of the PVR and closure of the DA and umbilical vessels. This model demonstrates that UCC can lead to an earlier onset of DA flow reversal and slightly reduced cardiac output (CO).
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Jang, Su Hyun, In Joo Kim, and Sung Hee Lee. "Relationship Between Social Support and Maternal-Fetal Attachment Among Unmarried Pregnant Women in Korea: The Mediating Effects of Self-esteem." In Healthcare and Nursing 2015. Science & Engineering Research Support soCiety, 2015. http://dx.doi.org/10.14257/astl.2015.104.06.

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Reports on the topic "Fetal effects"

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Dave, Dhaval, and Muzhe Yang. Maternal and Fetal Health Effects of Working during Pregnancy. Cambridge, MA: National Bureau of Economic Research, October 2019. http://dx.doi.org/10.3386/w26343.

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McDonagh, Marian, Andrea C. Skelly, Amy Hermesch, Ellen Tilden, Erika D. Brodt, Tracy Dana, Shaun Ramirez, et al. Cervical Ripening in the Outpatient Setting. Agency for Healthcare Research and Quality (AHRQ), March 2021. http://dx.doi.org/10.23970/ahrqepccer238.

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Objectives. To assess the comparative effectiveness and potential harms of cervical ripening in the outpatient setting (vs. inpatient, vs. other outpatient intervention) and of fetal surveillance when a prostaglandin is used for cervical ripening. Data sources. Electronic databases (Ovid® MEDLINE®, Embase®, CINAHL®, Cochrane Central Register of Controlled Trials, and Cochrane Database of Systematic Reviews) to July 2020; reference lists; and a Federal Register notice. Review methods. Using predefined criteria and dual review, we selected randomized controlled trials (RCTs) and cohort studies of cervical ripening comparing prostaglandins and mechanical methods in outpatient versus inpatient settings; one outpatient method versus another (including placebo or expectant management); and different methods/protocols for fetal surveillance in cervical ripening using prostaglandins. When data from similar study designs, populations, and outcomes were available, random effects using profile likelihood meta-analyses were conducted. Inconsistency (using I2) and small sample size bias (publication bias, if ≥10 studies) were assessed. Strength of evidence (SOE) was assessed. All review methods followed Agency for Healthcare Research and Quality Evidence-based Practice Center methods guidance. Results. We included 30 RCTs and 10 cohort studies (73% fair quality) involving 9,618 women. The evidence is most applicable to women aged 25 to 30 years with singleton, vertex presentation and low-risk pregnancies. No studies on fetal surveillance were found. The frequency of cesarean delivery (2 RCTs, 4 cohort studies) or suspected neonatal sepsis (2 RCTs) was not significantly different using outpatient versus inpatient dinoprostone for cervical ripening (SOE: low). In comparisons of outpatient versus inpatient single-balloon catheters (3 RCTs, 2 cohort studies), differences between groups on cesarean delivery, birth trauma (e.g., cephalohematoma), and uterine infection were small and not statistically significant (SOE: low), and while shoulder dystocia occurred less frequently in the outpatient group (1 RCT; 3% vs. 11%), the difference was not statistically significant (SOE: low). In comparing outpatient catheters and inpatient dinoprostone (1 double-balloon and 1 single-balloon RCT), the difference between groups for both cesarean delivery and postpartum hemorrhage was small and not statistically significant (SOE: low). Evidence on other outcomes in these comparisons and for misoprostol, double-balloon catheters, and hygroscopic dilators was insufficient to draw conclusions. In head to head comparisons in the outpatient setting, the frequency of cesarean delivery was not significantly different between 2.5 mg and 5 mg dinoprostone gel, or latex and silicone single-balloon catheters (1 RCT each, SOE: low). Differences between prostaglandins and placebo for cervical ripening were small and not significantly different for cesarean delivery (12 RCTs), shoulder dystocia (3 RCTs), or uterine infection (7 RCTs) (SOE: low). These findings did not change according to the specific prostaglandin, route of administration, study quality, or gestational age. Small, nonsignificant differences in the frequency of cesarean delivery (6 RCTs) and uterine infection (3 RCTs) were also found between dinoprostone and either membrane sweeping or expectant management (SOE: low). These findings did not change according to the specific prostaglandin or study quality. Evidence on other comparisons (e.g., single-balloon catheter vs. dinoprostone) or other outcomes was insufficient. For all comparisons, there was insufficient evidence on other important outcomes such as perinatal mortality and time from admission to vaginal birth. Limitations of the evidence include the quantity, quality, and sample sizes of trials for specific interventions, particularly rare harm outcomes. Conclusions. In women with low-risk pregnancies, the risk of cesarean delivery and fetal, neonatal, or maternal harms using either dinoprostone or single-balloon catheters was not significantly different for cervical ripening in the outpatient versus inpatient setting, and similar when compared with placebo, expectant management, or membrane sweeping in the outpatient setting. This evidence is low strength, and future studies are needed to confirm these findings.
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Comstock, Sarah. Examining the Effect of Maternal High-Fat Diet Consumption on the Physiology and Pancreas Development of Fetal and Juvenile Nonhuman Primate Offspring. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.551.

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Haan, Matthew M., James R. Russell, Daniel G. Morrical, and Daryl R. Strohbehn. Effects of Grazing Management on Forage Sward Height, Mass, and Nutrient Concentrations and the Proportions of Fecal Cover and Bare Soil in Pastures. Ames (Iowa): Iowa State University, January 2007. http://dx.doi.org/10.31274/ans_air-180814-133.

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Burkett, Jeremy, Kenneth J. Stalder, Wendy J. Powers, Thomas J. Baas, and John W. Mabry. The Effect of Inorganic, Organic and No Trace Mineral Supplementation on Growth Performance, Fecal Excretion and Digestibility of Grow-Finish Swine. Ames (Iowa): Iowa State University, January 2006. http://dx.doi.org/10.31274/ans_air-180814-32.

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Baker, Ian. Yield strength anomaly and the environmental effect in FeAl. Final report for the period September 1, 1996 - August 31, 2000. Office of Scientific and Technical Information (OSTI), August 2000. http://dx.doi.org/10.2172/798488.

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Lenz, Mark. RV POSEIDON Fahrtbericht / Cruise Report POS536/Leg 1. GEOMAR, October 2020. http://dx.doi.org/10.3289/geomar_rep_ns_56_2020.

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DIPLANOAGAP: Distribution of Plastics in the North Atlantic Garbage Patch Ponta Delgada (Portugal) – Malaga (Spain) 17.08. – 12.09.2019 The expedition POS 536 is part of a multi-disciplinary research initiative of GEOMAR investigating the origin, transport and fate of plastic debris from estuaries to the oceanic garbage patches. The main focus will be on the vertical transfer of plastic debris from the surface and near-surface waters to the deep sea and on the processes that mediate this transport. The obtained data will help to develop quantitative models that provide information about the level of plastic pollution in the different compartments of the open ocean (surface, water column, seafloor). Furthermore, the effects of plastic debris on marine organisms in the open ocean will be assessed. The cruise will provide data about the: (1) abundance of plastic debris with a minimum size of 100 μm as well as the composition of polymer types in the water column at different depths from the sea surface to the seafloor including the sediment, (2) abundance and composition of plastic debris in organic aggregates (“marine snow”), (3) in pelagic and benthic organisms (invertebrates and fish) and in fecal pellets, (4) abundance and the identity of biofoulers (bacteria, protozoans and metazoans) on the surface of plastic debris from different water depths, (5) identification of chemical compounds (“additives”) in the plastic debris and in water samples.
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Effects of receiving-water quality and wastewater treatment on injury, survival, and regrowth of fecal-indicator bacteria and implications for assessment of recreational water quality. US Geological Survey, 1996. http://dx.doi.org/10.3133/wri964199.

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Effects of hydrologic, biological, and environmental processes on sources and concentrations of fecal bacteria in the Cuyahoga River, with implications for management of recreational waters in Summit and Cuyahoga Counties, Ohio. US Geological Survey, 1998. http://dx.doi.org/10.3133/wri984089.

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