Academic literature on the topic 'Fetal anoxia'
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Journal articles on the topic "Fetal anoxia"
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Sedmera, David, Pavel Kucera, and Eric Raddatz. "Developmental changes in cardiac recovery from anoxia-reoxygenation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 283, no. 2 (August 1, 2002): R379—R388. http://dx.doi.org/10.1152/ajpregu.00534.2001.
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The developing cardiovascular system is known to operate normally in a hypoxic environment. However, the functional and ultrastructural recovery of embryonic/fetal hearts subjected to anoxia lasting as long as hypoxia/ischemia performed in adult animal models remains to be investigated. Isolated spontaneously beating hearts from Hamburger-Hamilton developmental stages 14( 14HH), 20HH, 24HH, and 27HH chick embryos were subjected in vitro to 30 or 60 min of anoxia followed by 60 min of reoxygenation. Morphological alterations and apoptosis were assessed histologically and by transmission electron microscopy. Anoxia provoked an initial tachycardia followed by bradycardia leading to complete cardiac arrest, except for in the youngest heart, which kept beating. Complete atrioventricular block appeared after 9.4 ± 1.1, 1.7 ± 0.2, and 1.6 ± 0.3 min at stages 20HH, 24HH, and 27HH, respectively. At reoxygenation, sinoatrial activity resumed first in the form of irregular bursts, and one-to-one atrioventricular conduction resumed after 8, 17, and 35 min at stages 20HH, 24HH, and 27HH, respectively. Ventricular shortening recovered within 30 min except at stage 27HH. After 60 min of anoxia, stage 27HHhearts did not retrieve their baseline activity. Whatever the stage and anoxia duration, nuclear and mitochondrial swelling observed at the end of anoxia were reversible with no apoptosis. Thus the embryonic heart is able to fully recover from anoxia/reoxygenation although its anoxic tolerance declines with age. Changes in cellular homeostatic mechanisms rather than in energy metabolism may account for these developmental variations.
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Cybulsky, Andrey V., Tomoko Takano, Joan Papillon, Abdelkrim Khadir, Krikor Bijian, Chu-Chun Chien, Charles E. Alpers, and Hamid Rabb. "Renal expression and activity of the germinal center kinase SK2." American Journal of Physiology-Renal Physiology 286, no. 1 (January 2004): F16—F25. http://dx.doi.org/10.1152/ajprenal.00144.2003.
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Rat fetal kidney mRNA was analyzed by RT-PCR to identify protein kinases. This screening demonstrated expression of a protein kinase consistent with SK2, a group II germinal center kinase and homolog of human Ste20-like kinase (SLK). SK2 mRNA, protein expression, and kinase activity were increased in rat fetal kidney homogenates ( embryonic days 17–21) compared with adult controls. In adult kidneys subjected to cross-clamping of the renal artery, followed by reperfusion, SK2 mRNA, protein expression, and kinase activity were increased compared with untreated contralateral controls. By immunohistochemistry, SK2 expression was evident mainly in the cytoplasm of tubular epithelial cells in fetal and adult kidneys. There was also some expression in developing and mature podocytes, but staining of the interstitium was negative. In cultured renal tubular epithelial cells, SK2 kinase activity was increased after incubation with serum, or after exposure to chemical anoxia plus reexposure to glucose. Stable overexpression of SLK reduced cell proliferation and increased apoptosis and exacerbated apoptosis and necrosis after chemical anoxia plus reexposure to glucose. Thus SK2 is a renal epithelial protein kinase whose expression and activity are increased during development and recovery from acute renal failure, where tubular epithelial regeneration may recapitulate developmental processes. The actions of SK2 appear to be antiproliferative and may facilitate cell injury.
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Rabetsimamanga, Lyn Z. A., Hary F. Rabarikoto, Eddie B. Rekoronirina, and Hery R. Andrianampanalinarivo. "Spontaneous umbilical cord hematoma causing still birth: a case report in Madagascar." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 7, no. 5 (April 28, 2018): 2031. http://dx.doi.org/10.18203/2320-1770.ijrcog20181474.
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Spontaneous umbilical cord hematoma is uncommon complication during delivery. It is responsible for severe fetal distress or death. We report a case of a 28 year-old primigravida Malagasy woman at the 37th weeks of gestation. She was admitted in the delivery room for beat oscillating on the fetal heart monitoring at the beginning of labour. Cesarean section was indicated for acute fetal distress in monitoring at dilation 5cm of the cervix traduced by some decelerations. But she gives birth to a still born female fetus by vaginal way short time after. A 5,5 cm hematoma was discovered on umbilical cord. This still birth may be due to anoxia during acute compression of the umbilical vessels by the hematoma. So, placental and cord examinations in cases of unexplained fetal hypoxia and stillbirth are very important.
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RIDOUT, R., K. WILDENTHAL, and R. DECKER. "Lysosomal responses of fetal mouse hearts recovering from anoxia and substrate depletion." Journal of Molecular and Cellular Cardiology 18, no. 8 (August 1986): 853–65. http://dx.doi.org/10.1016/s0022-2828(86)80959-2.
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Turinsky, J., B. P. Bayly, and D. M. O'Sullivan. "1,2-Diacylglycerol and ceramide levels in rat liver and skeletal muscle in vivo." American Journal of Physiology-Endocrinology and Metabolism 261, no. 5 (November 1, 1991): E620—E627. http://dx.doi.org/10.1152/ajpendo.1991.261.5.e620.
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1,2-Diacylglycerol and ceramide levels were measured in liver and skeletal muscle of rats under the following four experimental conditions: 1) during rapid fetal growth in the second half of gestation and during postnatal aging, 2) during tissue anoxia lasting up to 10 min, 3) during fasting for up to 6 days, and 4) during stress induced by injection of endotoxin (lipopolysaccharide from Salmonella enteritidis). 1,2-Diacylglycerol and ceramide levels in fetal liver were 77 and 58% lower, respectively, than those of young rats after weaning. 1,2-Diacylglycerol and ceramide concentrations in liver of postnatal rats were not influenced by aging. Anoxia produced a 41–64% increase in liver 1,2-diacylglycerol levels and a 9–21% increase in liver ceramides. Fasting of adult rats for up to 6 days had no effect on 1,2-diacylglycerol and ceramide levels in liver. However, fasting increased skeletal muscle concentrations of 1,2-diacylglycerol and ceramides 88 and 44%, respectively. Injection of endotoxin had a biphasic effect on liver 1,2-diacylglycerol levels, causing a transient 52% increase at 1 h, followed by a 24–38% decrease below the control level at 17 h after endotoxin administration. Liver ceramide levels were increased 66, 52, and 27% at 1, 3, and 5 h after endotoxin, respectively, but did not differ from control at 17 h after injection. Endotoxin had no effect on muscle 1,2-diacylglycerol and ceramide concentrations at any interval.
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Gosseye, Serge, Marie-Claire Golaire, and Jeanne-Claudie Larroche. "Cerebral, Renal and Splenic Lesions due to Fetal Anoxia and their Relationship to Malformations." Developmental Medicine & Child Neurology 24, no. 5 (November 12, 2008): 510–18. http://dx.doi.org/10.1111/j.1469-8749.1982.tb13658.x.
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Intriago Balda, José Vicente, José Leonardo Castro Zambrano, Luís Carlos Demera Demera, and Valeria Soraya Franco Muñoz. "Sufrimiento fetal agudo en pacientes con preeclampsia con signos de severidad." RECIAMUC 6, no. 1 (January 23, 2022): 420–33. http://dx.doi.org/10.26820/reciamuc/6.(1).enero.2022.420-433.
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La preeclampsia-eclampsia es un trastorno multisistémico de causa desconocida, que incrementa la morbimortalidad materna y perinatal. En algunos países latinoamericanos, los problemas con mayor regularidad en las gestantes y que producen alteraciones fetales de diferente índole, son los trastornos hipertensivos. La placentación anormal que se produce en la preeclampsia se relaciona con poca o ausente remodelación de las arterias espirales maternas En la preeclampsia y la eclampsia los fetos se encuentran mal oxigenados (hipoxia crónica fetal) y desnutridos, son hipotróficos y algunos recién nacidos presentan lesiones neurológicas irreversibles. El Sufrimiento fetal agudo es cuando el feto presenta asfixia acelerada que puede provocar descomposición de una respuesta fisiológica o un daño que va afectar al sistema nervioso central, deficiencia múltiple de órganos y por ende llega a la muerte fetal. El diagnostico de concluye con los siguientes elementos: acidosis, anoxia, hipoxia, frecuencia cardiaca fetal, asfixia, liquido meconial. Para conseguir el objetivo palteado se ha realizado la búsqueda de artículos científicos, libros, guías, tesis, trabajos de grado y toda publicación de carácter académico derivada de repositorios médicos como PUBMed, Elseiver, Scielo y editoras académicas universitarias. Se ha utilizado el motor de búsqueda Google académico bajo las fórmulas “sufrimiento fetal”, “preeclampsia” y “sufrimiento fetal agudo en pacientes con preeclampsia “, tomando solo las publicaciones por relevancia que aporten una descripción global de los elementos que exponen de manera explícita la relación entre el diagnostico de sufrimiento fetal derivado de una preeclampsia materna, sin descartar las características y consideraciones para un diagnóstico. El sufrimiento Fetal está estrechamente ligado al desarrollo de enfermedades hipertensivas, sin embargo, dichas afecciones maternas afortunadamente tienen elementos preventivos que conllevan a un pronóstico favorable al momento de cuidar el desarrollo de nuevas vidas. Aunque el Sufrimiento Fetal engloba unas posibles consecuencias para el recién nacido, no necesariamente ligadas a la muerte, sino que es importante resaltar que la posibilidad de desencadenar efectos neurológicos a largo plazo invita a potenciar los mecanismos preventivos para evitarlo.
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Bissonnette, John M. "Mechanisms regulating hypoxic respiratory depression during fetal and postnatal life." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 278, no. 6 (June 1, 2000): R1391—R1400. http://dx.doi.org/10.1152/ajpregu.2000.278.6.r1391.
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Selected topics in the respiratory response to acute hypoxia in the fetus and newborn are reviewed. Peripheral chemoreceptors acting through ionotrophic glutamate receptors play an important role in affecting the initial augmentation phase. Whether fall off in peripheral chemoreceptor activity contributes to the secondary depressive phase remains controversial. A number of approaches including permanent electrolytic and reversible cooling lesions, Fos protein activation, and double-labeling immunohistochemistry has converged to show that an area in and around the locus ceruleus in the rostral pons affects the central depression. There is evidence that this is mediated by catecholamines acting at α2-adrenergic receptors. Tonic activity in early expiratory (postinspiratory) neurons may contribute to hypoxia-induced apneic episodes in the fetus and newborn. Desensitization of α-amino-3-hydroxy-5-methylisoxazole-4-proprionic acid receptors has been demonstrated in respiratory-related neurons both in vivo and in vitro. The role that this process might play in the depressive phase of the hypoxic ventilatory response has not been established. In vitro experiments with isolated brain stem-spinal cord preparations or transverse brain stem slices usually involve anoxia, whereas whole animal experiments use 8–15% O2. Therefore, caution must be exercised in attempting to construct a unifying framework from these two approaches.
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Pereira, Lívia Helena M., Juliana R. Machado, Janaínna G. P. Olegário, Laura P. Rocha, Marcos V. Silva, Camila S. O. Guimarães, Marlene A. Reis, Lúcio Roberto Castellano, Fernando S. Ramalho, and Rosana R. M. Corrêa. "Interleukin-6 and C-Reactive Protein Are Overexpressed in the Liver of Perinatal Deaths Diagnosed with Fetal Inflammatory Response Syndrome." Disease Markers 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/252780.
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Anatomopathologic studies have failed to define the fetal inflammatory response syndrome (FIRS) as a cause of fetal death. Here, liver fragments of perinatal autopsies were collected at a university hospital from 1990 to 2009 and classified according to the cause of death, perinatal stress, and gestational age (GA) of the fetus. IL-6, TNF-α, and C-reactive protein (CRP) expression were immunostained, respectively, with primary antibody. Cases with congenital malformation, ascending infection, and perinatal anoxia showed increased IL-6, CRP, and TNF-α, respectively. Prematures presented higher expression of IL-6 whereas term births showed higher expression of CRP. Cases classified as acute stress presented higher expression of IL-6 and TNF-αand cases with chronic stress presented higher expression of CRP. GA correlated negatively with IL-6 and positively with CRP and TNF-α. Body weight correlated negatively with IL-6 and positively with CRP and TNF-α. Despite the diagnosis of FIRS being clinical and based on serum parameters, the findings in the current study allow the inference of FIRS diagnosis in the autopsied infants, based on anin situliver analysis of these markers.
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Pedretti, S., and E. Raddatz. "F007 Modulation of the STAT3 pathway by transient anoxia and oxidant stress in a fetal heart model." Archives of Cardiovascular Diseases 102 (March 2009): S55. http://dx.doi.org/10.1016/s1875-2136(09)72260-0.
Full textDissertations / Theses on the topic "Fetal anoxia"
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Zavala, Coca Carlos Alberto. "Flujo venoso fetal e índice cerebro placentario como indicadores de hipoxia fetal en gestantes preeclámpticas severas." Doctoral thesis, Universidad Nacional Mayor de San Marcos, 2010. https://hdl.handle.net/20.500.12672/1343.
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Objetivo: Determinar el valor predictivo del Índice Cerebro Placentario y del flujo anormal del Ductus Venoso de Aranzio, medido por velocimetría Doppler, en pacientes con preeclampsia, en relación a un resultado perinatal adverso.
Materiales y métodos: Estudio prospectivo, no experimental, longitudinal, de tipo correlacional. Se realizaron exámenes ultrasonográficos Doppler para determinar el Índice Cerebro Placentario y el flujo anormal del Ductus Venoso de Aranzio, en los 7 días previos al parto, en 160 pacientes con diagnóstico de preeclampsia severa admitidas en la Unidad de Medicina Fetal y Diagnóstico Prenatal del Servicio de Obstetricia de Alto Riesgo del Hospital Guillermo Almenara Irigoyen – EsSalud.
El resultado perinatal adverso fue definido por los siguientes parámetros: Cesárea por SFA, APGAR menor 7 a los 5´, Líquido amniótico meconial, Oligohidramnios, pH de la arteria umbilical menor 7,2, Admisión en UCI neonatal, RCIU.
Se utilizó estadística descriptiva para la variable dependiente y estadística inferencial mediante el estadístico chi cuadrado (x²) y prueba exacta de Fisher, con un nivel de significancia de 0,05; confiabilidad del 95%. Además se calculó la sensibilidad, especificidad y valores predictivos positivo y negativo de la variable independiente.
Conclusiones: Se ha demostrado que la alteración del Índice Cerebro Placentario y del Flujo del Ductus Venoso de Aranzio medido por flujometría Doppler fetal, detecta a más del 65% de los recién nacidos con resultado perinatal adverso e hipoxia fetal y se asocia a la ocurrencia del mismo. Además esta es una prueba predictiva, estadísticamente significativa, de RCIU y de oligohidramnios, en pacientes con preeclampsia severa.
El presente estudio se realizó con un muestreo no aleatorio, por ende, este hecho de no aleatoriedad, pudiera plantear problemas de validez externa.Objective: To ascertain the value of cerebral-placental ratio and the abnormal fluxo of Aranzio´s Ductus Venous and for identifying newborns with neonatal morbidity in pregnancies complicated by severe preeclampsia. Study Design: A longitudinal and correlational study of 160 patients with severe preeclampsia (PA > 160/110, proteinuria 3+) was performed Doppler study done by one operador within 7 days before delivery. An abnormal cerebral-placental ratio and abnormal resistance and pulsabilility index of ductus venous were used to identificate fetal asphixia (cardiac insuficiency). The results belong 5 percentile were considered abnormal. These results were matched with perinatal results considered as abnormal. Results: Maternal characteristic were: age 33, parity 1, primigravid 45%, prenatal care 85%, gestational age at enrollment 35,1 weeks. The probability of detection IUGR is 65% and oligohydramnios 61,2%. Conclusion: The cerebral-placental ratio and abnormal fluxo of Aranzio´s Ductus venous identifies 65 % or more of the newborns with severe neonatal morbidity in pregnancies with severe preeclampsia.
Tesis
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Kolb, Rosana Marta. "Respostas metabolicas, moleculares e mobilização das reservas no desenvolvimento inicial de especies neotropicais sob anoxia." [s.n.], 2003. http://repositorio.unicamp.br/jspui/handle/REPOSIP/314851.
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Orientador: Angelo Luiz CortelazzoTese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
Made available in DSpace on 2018-08-03T15:27:42Z (GMT). No. of bitstreams: 1 Kolb_RosanaMarta_D.pdf: 10136146 bytes, checksum: 29e3b7de2d47c70d6ad0a1bd3fb98589 (MD5) Previous issue date: 2003
Resumo: O presente estudo objetivou examinar o metabolismo energético e as alterações ultraestruturais ocorridas sob anaerobiose, durante o estádio inicial pós-germinativo de quatro espécies: Sesbania vir gata, Sebastiania commersoniana, Erythrina speciosa, as quais ocorrem em áreas encharcadas ou alagadas e Schizolobium parahyba, que ocupa preferencialmente locais bem drenados. Para E. speciosa e S. parahyba também foi estudada a mobilização de reservas sob condições de normoxia e anoxia. Quando comparada às demais espécies, S. vir gata apresentou maiores níveis de A TP sob anaerobiose e suas raízes mostraram poucas alterações ultraestruturais, mesmo após 4 dias de anoxia. As modificações celulares não foram deletérias uma vez que todas as plântulas retomaram o crescimento após um período de reaeração. As raízes de S. commersoniana foram muito mais sensíveis à falta de oxigênio, com níveis de ATP menores do que para S. virgata. Suas células perderam a compartimentalização e suas plântulas não voltaram a crescer sob aerobiose. A mobilização das reservas foi menor na ausência de oxigênio. Tanto E. speciosa quanto S. parahyba utilizaram suas reservas de carboidrato sob anaerobiose, mas E. speciosa manteve níveis de ATP mais elevados do que S. parahyba. A manutenção dos compartimentos celulares em parte das plântulas de E. speciosa permitiu que as mesmas retomassem o crescimento após o tratamento anaeróbio, o que não ocorreu com as plântulas de S. parahyba. Entre as espécies estudadas, S. virgata e E. speciosa foram mais tolerantes à deficiência de oxigênio do que S. commersoniana e S. parahyba. A maior tolerância das espécies à anaerobiose, provavelmente está relacionada com a manutenção de um adequado metabolismo fermentativo, com conseqüente preservação da homeostase e da compartimentalização celular
Abstract: The aims of the present study were to examine the energy metabolism and the occurrence of ultrastructural alterations under anaerobiosis, during the early seedling development of four species: Sesbania virgata, Sebastiania commersoniana, Erythrina speciosa, which occur in waterlogged or flooded areas and Schizolobium parahyba, which occupies mainly well-drained areas. For E. speciosa and S. parahyba, the mobilization of reserves under normoxia and anoxia was also investigated. When compared with the other species, S. virgata presented higher ATP levels under anaerobiosis and its roots showed few ultrastructural alterations, even after 4 days of anoxia. The cellular modifications were not deleterious once all its seedlings resume growth after the reaeration period. The S. commersoniana roots were more sensitive to oxygen absence, with lower ATP levels than for S. virgata. The cellular compartmentalization was disrupted and its seedlings did not resume growth under aerobiosis. The mobilization of reserves was lower under anoxia. Both E. speciosa and S. parahyba were able to utilize their carbohydrate reserves under anaerobiosis, however, E. speciosa maintained higher ATP levels than S. parahyba. The maintenance of cellular compartments in part of the E. speciosa seedlings, was responsible for growth recovery after the anaerobic treatment. The same was not true for S. parahyba seedlings. Among the species studied, S. virgata and E. speciosa were more tolerant to oxygen deficiency than S. commersoniana and S. parahyba. The higher anaerobic tolerance of the species is probably related to the maintenance of an adequate fermentative energy metabolism that results in preservation of homeostasis and cell compartmentalization
Doutorado
Doutor em Biologia Vegetal
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Pennell, Craig Edward. "The role of lactate measurement in the prediction of fetal hypoxic-ischaemic brain injury during labour." University of Western Australia. School of Women's and Infants' Health, 2004. http://theses.library.uwa.edu.au/adt-WU2003.0037.
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[Truncated abstract] In this thesis the role of lactate measurement has been evaluated in intrapartum assessment of fetal wellbeing. Specifically, I have addressed the question of whether fetal lactate measurement is better than the assessment of fetal heart rate patterns or the measurement of pH at predicting fetal brain injury after intrapartum asphyxia. Using an ovine model of repeated umbilical cord occlusion designed to mimic events which may occur during human labour, I have shown that the measurement of fetal lactate levels after repeated cord occlusion is significantly associated with the severity of brain injury after the asphyxial insult. No significant associations were identified with fetal pH measurements or with the duration of decelerative or compound fetal heart rate patterns; however, this is the first study to describe an association between the duration of both increased fetal heart rate variability and fetal heart rate overshoot with the severity of subsequent brain injury. Although no significant association was identified between fetal arterial pressure measured between umbilical cord occlusions and the grade of brain injury, the studies performed in this thesis are the first to show a strong correlation between the duration of specific arterial pressure responses during cord occlusions and the grade of brain injury, accounting for approximately 90% of the variability seen in the severity of injury. The mechanism responsible for the improved ability of lactate measurement to predict fetal brain injury is unknown. It may be because fetal lactate levels are a more stable marker of anaerobic metabolism of glucose than fetal pH levels, which are influenced by both increasing levels of carbon dioxide and anaerobic metabolism of amino-acids and fatty acids. In addition fetal pH levels can be rapidly normalised through placental exchange of carbon dioxide whereas fetal lactate levels are slow to normalise across the placenta as they rely on facilitated diffusion.
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Allemandi, Wilma. "Estudo da imunorreatividade da proteína S100b no Hipocampo e Núcleo do Trato Solitário de ratos neonatos submetido à anóxia." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/42/42131/tde-22052012-103409/.
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Agressões nos períodos críticos do crescimento do sistema nervoso podem modificar os eventos de desenvolvimento. Entre os vários fatores nocivos está a anóxia. O organismo do neonato tem suprimento de energia anaeróbica relativamente rica, foi observado que a acidose ocorre com menor facilidade, propiciam sobrevivência. A proteína de astrócitos, S100b, exerce efeitos parácrinos e autócrinos em neurônios e glia. Sua estimulação promove sobrevivência e proteção neuronal, atuando como fator trófico e neurotrófico. Modelo animal de anóxia neonatal desenvolvido em nosso laboratório, nos revelou ativação neural pela expressão de Fos e alterações comportamentais, o que nos instigou a explorar os efeitos da anóxia nas células da glia no Hipocampo e Núcleo do Trato Solitário. Para sua exposição à anoxia, durante 25 minutos, foi utilizada câmara, saturada com nitrogênio gasoso 100%. Grupos P2 e P7 nas condições: Basal (B), sem estimulo; Sham (S) como controle experimental e Anóxia (A) com falta de oxigênio, foram analisados por S100b-IR com técnicas ABC/DAB e Western blot. Observamos significante diferença de S100b-IR no núcleo do trato solitário, somente no grupo P2 A 2 h em relação ao grupo P2 S 2 h. A reatividade glial de S100b na formação hipocampal (CA1, CA3+CA2 e DG), apresentou diferença significante no grupo anoxia de acordo com o estágio de maturação do animal. A técnica por Western blot em toda a formação hipocampal, apresentou aumento de S100b no grupo A em ambos P2 e P7, a avaliação de um todo foi diferente daquela de áreas especificas.Attacks to the nervous system at critical growth periods can modify developmental events. Among the various harmful factors at is anoxia. The high anaerobic energy supply to the newborn and a less easily acidosis occurrence provides survival. The astrocyte S100b protein exerts paracrine and autocrine effects on neurons and glia. Its stimulation promotes neuronal survival and protection, as a trophic and neurotrophic factor. An animal model of neonatal anoxia improved in our lab revealed neural activation by Fos expression and behavioral changes, which prompted us to explore the anoxia effects on glial cells in the Hypocampus and Nucleus of Solitary Tract. For their exposure to anoxia, a chamber, saturated with 100% nitrogen gas, for 25 minutes were used. Groups with P2 and P7, conditions: Baseline, without stimulation; Sham as the experimental control, and Anoxia with lack of oxygen, were evaluated by S100b-IR by ABC/DAB and Western blot techniques. The nucleus of solitary tract, significant different S100b-IR observed, only in the P2 A 2 h compared to P2 S 2 h. The glial S100b-IR at the hippocampal formation (CA1, CA2 + CA3 and DG) presented significant difference in the anoxic group according to the maturational stage of the animal. Western blot technique of the entire hippocampal formation, showed increase of S100b at the group A at both P2 and P7, the whole evaluation was different from of that of specific areas.
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Flores, Santin Josele R. "Cardiovascular Fetal Programming in Quail (Colinus virginianus), An Avian Comparative Model." Thesis, University of North Texas, 2016. https://digital.library.unt.edu/ark:/67531/metadc955048/.
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The consequences of early embryonic insults and how they affect subsequent life reflects the emerging concept of "fetal programming". The aim of this project is to study the effects of embryonic insults as they subsequently manifest themselves in adults, with emphasis on the heart and vasculature. My experiments establish that fetal programming operates on the bobwhite quail inducing similar changes as those observed in mammalians and other birds. The quail's fast development provides reliable data in a short period of time than other avian models (e.g. domestic chicken). Data on quail showed a correlation between egg mass and hatchling mass; where small eggs produce small hatchlings but a high mortality made it impractical as a stressor for this study. Hypoxia was used as a stressor during embryonic incubation, where it induced a low hatching weight in quail that was not observable in adult birds. Morphological measurements demonstrated an increased ventricular collagen content and reduced ventricular lumen in birds in adults incubated in hypoxia consistent with hypertension. The hematological analyzes showed few differences indicating organ remodeling instead of hematopoietic compensation. The assessment of vascular reactivity pointed out an impaired endothelium dependent relaxation commonly associated to hypertension in birds and mammals. Fetal programming could be a widespread response to an adverse prenatal environment in endotherms and the resulting data from this work contributes to our understanding of fetal programming in vertebrates and its long term consequences.
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Houzé, de l'Aulnoit Agathe. "Acquisition du rythme cardiaque fœtal et analyse de données pour la recherche de facteurs prédictifs de l’acidose fœtale." Thesis, Lille, 2019. http://www.theses.fr/2019LIL2S007.
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L’analyse visuelle du rythme cardiaque fœtal (RCF) est une excellente méthode de dépistage de l’hypoxie fœtale. Cette analyse visuelle est d’autre part sujette à une variabilité inter- et intra-individuelle importante. L’hypoxie fœtale au cours du travail s’exprime par des anomalies du RCF. La sous-évaluation de la gravité d’un RCF entraine une prise de risque indue pour le fœtus avec une augmentation de sa morbi-mortalité et sa surévaluation entraine un interventionnisme obstétrical inutile avec une augmentation du taux de césariennes. Ce dernier point pose par ailleurs en France un problème de santé publique.L’analyse automatisée du signal RCF permet de diminuer la variabilité inter- et intra-individuelle et d’accéder à d’autres paramètres calculés visant à augmenter la valeur diagnostique. Les critères d’analyse morphologiques du RCF (ligne de base, nombre d’accélérations, nombre et typage des ralentissements, variabilité à long terme (VLT)) ont été décrits ainsi que d’autres tels que les surfaces des ralentissements, les indices de variabilité à court terme (VCT) et les analyses fréquentielles. Il n’en demeure pas moins que la définition de la ligne de base, à partir de laquelle sont repérés les accélérations et les ralentissements reste, dans certains cas, difficile à établir.L’objectif principal de la thèse est d’établir un modèle prédictif de l’acidose fœtale à partir d’une analyse automatisée du RCF. L’objectif secondaire est de déterminer la pertinence des différents paramètres élémentaires classiques (CNGOF 2007) (fréquence de base, variabilité, accélérations, ralentissements) et celle d’autres paramètres inaccessible à l’œil (indices de variabilité à court terme, surfaces des ralentissements, analyse fréquentielle…). Par la suite, nous voulons identifier des critères de décision qui aideront à la prise en charge obstétricale.Nous proposons d’aborder l’analyse automatisée du RCF pendant le travail par l’intermédiaire d’une étude cas-témoins ; les cas étant des tracés RCF de nouveau-nés en acidose néonatale (pH artériel au cordon inférieur ou égal à 7,15) et les témoins, des tracés RCF de nouveau-nés sans acidose (pH artériel au cordon supérieur ou égal à 7,25). Il s’agit d’une étude monocentrique à la maternité de l’hôpital Saint Vincent de Paul, GHICL – Lille, sur notre base de données « Bien Naitre » (archivage numérique des tracés RCF depuis 2011), comptant un un nombre suffisant de cas sur ce seul centre. La maternité Saint Vincent de Paul (GHICL) présente depuis 2011 environ 70 cas par an d’acidose néonatale (pHa ≤ 7,10) (3,41%). Le logiciel R sera utilisé pour l’analyse statistiqueVisual analysis of the fetal heart rate FHR is a good method for screening for fetal hypoxia but is not sufficiently specific. The visual morphological analysis of the FHR during labor is subject to inter- and intra-observer variability – particularly when the FHR is abnormal. Underestimating the severity of an FHR leads to undue risk-taking for the fetus with an increase in morbidity and mortality and overvaluation leads to unnecessary obstetric intervention with an increased rate of caesarean section. This last point also induces a French public health problem.FHR automated analysis reduces inter and intra-individual variability and accesses other calculated parameters aimed at increasing the diagnostic value. The FHR morphological analysis parameters (baseline, number of accelerations, number and typing of decelerations, long-term variability (LTV)) were described as well as others such as the decelerations surfaces, short-term variability (STV) and frequency analyzes. Nevertheless, when attempting to analyze the FHR automatically, the main problem is computation of the baseline against which all the other parameters are determined.Automatic analysis provides information on parameters that cannot be derived in a visual analysis and that are likely to improve screening for fetal acidosis during labor.The main objective of the thesis is to establish a predictive model of fetal acidosis from a FHR automated analysis. The secondary objective is to determine the relevance of the classical basic parameters (CNGOF 2007) (baseline, variability, accelerations, decelerations) and that of other parameters inaccessible to the eye (indices of short-term variability, surfaces of decelerations, frequency analysis ...). Later, we want to identify decision criteria that will help in the obstetric care management.We propose to validate FHR automated analysis during labor through a case-control study; cases were FHR recordings of neonatal acidosis (arterial cord pH less than or equal to 7.15) and controls, FHR recordings of neonatal without acidosis (arterial cord pH upper than or equal to 7.25). This is a monocentric study at the maternity hospital of Saint Vincent de Paul Hospital, GHICL - Lille, on our « Well Born » database (digital archiving of RCF plots since 2011), with a sufficient number of cases on this only center. Since 2011, the Saint Vincent de Paul hospital (GHICL) has had about 70 cases per year of neonatal acidosis (pHa less than or equal to 7.10) (3.41%). The R software will be used for statistical analysis
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Martin, Nicolas. "Programmation foetale et plasticité cérébrale : conséquences d'une carence précoce en donneurs de méthyles chez le rat-impact à long terme d'un conditionnement hypoxique néonatal." Thesis, Nancy 1, 2011. http://www.theses.fr/2011NAN10091/document.
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Une altération du métabolisme de l'homocystéine constitue un facteur de risque pour la survenue des maladies neurodégénératives. Par ailleurs, alors que les effets délétères d'une hypoxie néonatale sévère sont bien connus, il a été récemment montré qu'un épisode hypoxique modéré exerçait une neuroprotection via une stimulation de la neurogenèse. Notre objectif fut l'étude des conséquences cérébrales d'une carence précoce en donneurs de méthyles (folates, vitamine B12) combinée ou non à une stimulation hypoxie modérée. Un modèle in vivo de rats nés de mères carencées en donneurs de méthyles fut utilisé. Il a été étudié les mécanismes impliqués dans un modèle de progéniteurs neuronaux carencés. Les résultats ont montré des atteintes de l'intégrité tissulaire et fonctionnelle de l'hippocampe et du cervelet associée à des déficits comportementaux, à différents stades de la vie chez les animaux carencés malgré le retour à une alimentation standard au sevrage. Ces perturbations sont liées aux processus épigénétiques et à l'homocystéinylation de protéines neuronales. De plus, un dimorphisme sexuel est apparu en lien avec le récepteur nucléaire ER alpha. La neurogenèse issue de l'hypoxie a engendré des conséquences bénéfiques à long terme sur le vieillissement cérébral des rats mâles, avec un maintien de l'intégrité hippocampique. Enfin, la combinaison de la carence et de l'hypoxie, a montré que le conditionnement hypoxique améliorait le devenir tissulaire et fonctionnel du cerveau des animaux carencés. Les mécanismes clés surviendraient au cours de périodes critiques de maturation des différentes structures cérébrales, soulignant l'importance des processus de la programmation foetaleThe alteration of homocysteine metabolism has been shown to constitute a risk factor for neurodegenerative diseases. Furthermore, whereas deleterious effects of severe neonatal hypoxia have been well documented, it was shown that a moderate episode of hypoxia can exert a neuroprotection with neurogenesis stimulation. Our main goal was to study the consequences on the brain of an early deficiency of methyl donors (folate, vitamin B12) with or without a hypoxia-related stimulation of neurogenesis. The effects of deficiency were investigated in rats born from dams fed a deficient diet until weaning. In vitro neuroprogenitors were additionally used for the study of cell mechanisms involved. Data showed alterations of tissue integrity in the hippocampus and the cerebellum, with associated behavioural deficits at various ages, despite a return to normal diet at weaning. Brain alterations were shown to be mainly related to epigenetic mechanisms and to homocysteinylation of specific neuronal proteins. Moreover, a sexual dimorphism was depicted, with the participation of ER alpha receptor. Neurogenesis induced in germinative zones by a brief neonatal hypoxia led to long term beneficial effects on brain aging in male rats, with preserved hippocampus integrity, in terms of cell density, synaptic plasticity, and related cognitive functions. Finally, the combination of deficiency and hypoxia revealed that brain conditioning by brief neonatal hypoxia was able to improve tissular and functional brain outcome in deficient rats. The key mechanisms involved would occur at critical periods during the maturation of the various brain structures, thus highlighting the role of fetal programming
Books on the topic "Fetal anoxia"
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Perinatal asphyxia. New York: Plenum Medical Book Co., 1986.
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Symposium on Perinatal Asphyxia (1988 Toronto, Ont.). Perinatal asphyxia: Its role in developmental deficits in children : papers and discussion based on a symposium held October 26, 1988, in Toronto, Ontario, Canada, jointly with the 42nd Annual Meeting of the American Academy for Cerebral Palsy and Developmental Medicine and sponsored by the Canadian Medical Protective Association. Ottawa, Ontario: Canadian Medical Protective Association, 1988.
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Workshop on Acute Perinatal Asphyxia in Term Infants (1993 Rockville, Md.). Report of the Workshop on Acute Perinatal Asphyxia in Term Infants: August 30-31, 1993, Rockville, Maryland. [Washington, D.C.?]: The Institute, 1996.
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L, Wright Linda, Merenstein Gerald B, Hirtz Deborah, National Institute of Child Health and Human Development (U.S.), National Institute of Neurological Disorders and Stroke (U.S.), American Academy of Pediatrics, and American College of Obstetricians and Gynecologists., eds. Report of the Workshop on Acute Perinatal Asphyxia in Term Infants: August 30-31, 1993, Rockville, Maryland. [Washington, D.C.?]: The Institute, 1996.
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F, Kubli, and International Federation of Gynecology and Obstetrics. Committee on Perinatal Mortality and Morbidity., eds. Perinatal events and brain damage in surviving children. Berlin: Springer-Verlag, 1988.
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(Editor), Ph Arbeille, ed. Fetal Hypoxia. Taylor & Francis, 1999.
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Lacoius-Petruccelli, Alberto. Perinatal Asphyxia. Vantage Press, 2006.
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J, Haddad, Saliba E. 1950-, and Arbeille Ph, eds. Perinatal asphyxia. Berlin: Springer-Verlag, 1993.
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Hronsky, Susan Lynn. The physiological, biochemical, and behavioral markers of oxygen deprivation in the human neonate. 1986.
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Oxygen: Basis of the regulation of vital functions in the fetus. Berlin: Springer-Verlag, 1992.
Find full textBook chapters on the topic "Fetal anoxia"
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Lacoius-Petruccelli, Alberto. "Types and Stages of Fetal Anoxia." In Perinatal Asphyxia, 57–63. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_8.
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Thatte, Shubhankar, Nick Grainger, and Judy McKay. "Understanding Feral IT Practices as Deviance." In Feral Information Systems Development, 189–208. IGI Global, 2014. http://dx.doi.org/10.4018/978-1-4666-5027-5.ch009.
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In this chapter, the authors discuss the concept of feral IT practices, which are defined as work practices of employees who use information technologies in ways that deviate from organizational norms. Such practices are in the main conducted beyond the control and/or knowledge of organizational IT management. The authors argue that feral practices can be looked at from many different perspectives, and thus may sometimes be viewed as innovative and beneficial for organizations, as opposed to being regarded as risky and potentially detrimental to the organization. However, such a view of feral practices is relatively under-theorized at present, and a challenge for researchers is to develop knowledge of how an organization can benefit from this unsanctioned innovation, while protecting itself through adequately managing the risks to which they may be exposed through these feral practices. Merton’s Theory of Anomie provides deeper insights into the phenomenon of non-compliance, and in this chapter, the authors aim to explore how the concept of deviance (non-compliance) can usefully be applied to offer new insights into the phenomenon of feral practices. A conceptual framework based on Merton’s theory is proposed that suggests that feral IT practices may arise from structural strain that inhibits the ability of an individual or group to achieve their goals and objectives. The organizational structure factors contributing to structural strain are discussed. The chapter concludes with a discussion of the potential contribution of Merton’s theory to understanding and ultimately managing feral practices in organizations such that risks are minimized and benefits maximized.
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Berga, Sarah L. "Disorders of gonadotropin secretion." In Oxford Textbook of Endocrinology and Diabetes, 1212–23. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.0838.
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Folliculogenesis and ovulation depend upon adequate gonadotropin stimulation, which in turn requires appropriate gonadotropinreleasing hormone (GnRH) input. There exists a group of related disorders in which GnRH drive to the pituitary is reduced, resulting in secondary diminution of follicle-stimulating hormone (FSH) and luteinizing hormone input to the ovary. Clinically, reduced GnRH drive results in a spectrum of ovarian compromise, ranging from luteal insufficiency to chronic anovulation. Variable menstrual patterns follow, including amenorrhoea, polymenorrhoea, with or without menorrhagia, and oligomenorrhoea, depending on the extent of follicular activity across time. Rarely, there is an organic or congenital cause for reduced GnRH drive, such as a brain tumour, coeliac disease, or migration of an insufficient number of GnRH neurons from the olfactory placode into the hypothalamus during fetal development. Typically, the cause is functional, that is, due to the endocrine consequences of certain psychological or behavioural variables. Anorexia nervosa provides the most dramatic example, but most women who develop functional hypothalamic anovulation do not meet criteria for an eating disorder and do not develop one subsequently. Because of the occult and heterogeneous nature of the behavioural variables that contribute to the genesis of this related group of disorders, a variety of names have been used to describe this syndrome, including exercise amenorrhoea, stress-related or stress-induced anovulation, functional hypothalamic amenorrhoea, functional hypothalamic chronic anovulation, and psychogenic amenorrhoea. Occasionally, psychiatric syndromes other than eating disorders such as depression coexist with functional hypothalamic anovulation, but unlike anorexia nervosa, in which amenorrhoea is almost universal, amenorrhoea is less common in women with bulimia and depression. Despite the multiplicity of names, the pathogenesis of anovulation in these diverse clinical settings is similar. In recognition of their common nature, I have chosen herein to refer to this group of disorders as ‘functional hypothalamic anovulation’ or FHA. As noted above, not all women have reduced gonadotropin secretion to the extent that they become amenorrhoeic or even persistently anovulatory, but most investigations have focused on subjects with the most complete expression of these related disorders, namely, those who are amenorrhoeic due to chronic anovulation. Generally, functional hypothalamic anovulation is considered to be a form of secondary amenorrhoea, but it can present as primary amenorrhoea. The diagnosis of functional hypothalamic anovulation is one of exclusion.
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Baldwin, Peter. "Introduction." In The Narcissism of Minor Differences. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195391206.003.0004.
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The Atlantic Gets Ever Wider. Not just in a physical sense, as oceans rise and coastlines recede, but also in ideological terms. Europe and America appear to be pitted against each other as never before. On one shore, capitalist markets, untempered by proper social policies, allow unbridled competition, poverty, pollution, violence, class divides, and social anomie. On the other side, Europe nurtures a social approach, a regulated labor market, and elaborate welfare networks. Possibly it has a less dynamic economy, but it is a more solidaristic and harmonious society. “Our social model,” as the voice of British left liberalism, the Guardian, describes the European way, “feral capitalism,” in the United States. With the collapse of communism, the European approach has been promoted from being the Third Way to the Second Way. The UK fl oats ambiguously between these two shores: “Janus Britain” in the phrase of the dean of transatlanticist observers, Timothy Garton Ash. It is part of Europe, says the British Left ; an Anglo-Saxon coconspirator, answer its continental counterparts. That major differences separate the United States from Europe is scarcely a new idea. But it has become more menacingly Manichaean over the last decade. Foreign policy disagreements fuel it: Iraq, Iran, Israel, North Korea. So does the more general question of what role the one remaining superpower should play while it still remains unchallenged. Robert Kagan has famously suggested that, when it comes to foreign policy, Americans and Europeans call different planets home. Americans wield hard power and face the nasty choices that follow in its wake. Europeans, sheltered from most geopolitical strife, enjoy the luxury of approaching conflict in a more conciliatory way: Martian unilateralism confronts Venusian multilateralism. But the dispute goes beyond diplomatic and military strategy. It touches on the nature of these two societies. Does having the strongest battalions change the country that possesses them? After all, America is not just militarily strong. It is also—compared to Europe—harsh, dominated by the market, crime-ridden, violent, unsolidaristic, and sharp-elbowed. Competition is an official part of the national ideology and violence the way it spills over into everyday life. Or so goes the argument: a major battle of worldviews and social practices separates America from Europe.
Conference papers on the topic "Fetal anoxia"
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Santos, Edson Moura dos, and Marcia Helena Braga Catroxo. "IDENTIFICAÇÃO DE PARTÍCULAS DE CORONAVÍRUS EM EQUINOS, ATRAVÉS DE TÉCNICA DE MICROSCOPIA ELETRÔNICA DE TRANSMISSÃO." In I Smart Congresso Nacional de Microscopia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2283.
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Introdução: O coronavírus equino (ECoV), vírus RNA de fita positiva, é classificado na família Coronaviridae, gênero Betacoronavirus, que juntamente com outros coronavírus são patogênicos para humanos. A infecção pelo ECoV acomete principalmente a mucosa do intestino delgado onde tende a danificar as microvilosidades levando a má absorção, diarreia aquosa e profusa, causando severa enterite. Outros sinais clínicos descritos para a infecção destacam-se febre, anorexia, apatia, depressão, letargia acarretando prejuízos na equinocultura. A rota fecal-oral é considerada a principal fonte de infecção para outros equinos. Objetivo: Detectar a presença de partículas de coronavírus em amostras de fezes ou de fragmentos de intestino delgado de equinos por microscopia eletrônica de transmissão. Material e métodos: No período de 2011 a 2021, aproximadamente 83 amostras de fezes ou fragmentos de intestino delgado de equinos, de casos clínicos foram enviadas ao Laboratório de Microscopia Eletrônica do Instituto Biológico de São Paulo, SP, Brasil, para diagnóstico viral. Os animais com idade variando entre 2 dias a 3 anos, apresentavam sinais clínicos diversos, entre eles, desconforto abdominal, febre, diarreia persistente amarelada ou sanguinolenta e pneumonia. Cerca de 10 animais morreram. As amostras foram processadas para microscopia eletrônica de transmissão utilizando a técnica de contrastação negativa (preparo rápido). Nesta técnica, as amostras são suspensas em tampão fosfato 0,1 M e pH 7,0, colocadas em contato com grades metálicas e contrastadas negativamente com molibdato de amônio a 2%. Resultados: Ao microscópio eletrônico de transmissão, foi visualizado um grande número de partículas com morfologia semelhante à coronavírus, pleomórficas, envelopadas, contendo projeções radiais típicas, em forma de coroa solar, medindo em média, 140 nm de diâmetro em 60 amostras (72,29%). Conclusão: A técnica utilizada foi eficiente para o diagnóstico rápido dos coronavírus equino.
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GIMENEZ, Elena Macedo, Lucas Augusto MARIOTTO, Lorrayne de Souza Araújo MARTINS, Antônio Campanha MARTINEZ, and Rodrigo Garcia MOTTA. "RELATO DE CASO - ACHADOS CLÍNICOS E ANATOMOPATOLÓGICOS DA ENTERITE LINFOPLASMOCITÁRIA EM OVINO." In I Congresso Brasileiro Online de Práticas Veterinárias: Uma abordagem para animais de grande porte e produção Animal. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/granvet-57.
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Introdução: As verminoses impactam em sérios prejuízos para a ovinocultura. São enfermidades com ciclo oro-fecal, em que os animais infectados eliminam os ovos do parasito pelas fezes, e no meio ambiente eclodem, e dão origem, as larvas infectantes. Na espécie ovina são raros os estudos com o diagnóstico da oesofagostomose pela histopatologia. Em cabanhas endêmicas, identificam-se: animais com histórico de emagrecimento progressivo, queda da produção de lã, anorexia, diarreia verde escura, anemia, hipoproteinemia e morte. Os principais achados anatomopatológicos: formação de nódulos no intestino e reação do tecido linfóide regional. Na microscopia enterite linfoplasmocitária nodular. Objetivo: relatar os achados clínicos e anatomopatológicos da enterite linfoplasmocitária em ovino. Relato de caso: Foi realizada a necropsia de uma ovelha, fêmea, 4 anos, mestiça, com histórico de emagrecimento progressivo há 4 meses, sem resposta terapêutica a vermifugação. Caquexia, mucosas hipocoradas, palidez visceral. Conteúdo intestinal com aspecto esverdeado, presença de múltiplos nódulos calcificados na serosa do rúmen, retículo, omaso e abomaso. Áreas de congestão intestinal e linfadenomegalia (linfonodos mesentérios). Fragmentos de mucosa intestinal e linfonodos mesentéricos foram fixados em formal 10% e colorados em Hematoxilina e Eosina (H&E). O exame microscópico identificou enterite, hiperplasia linfocitária, com aumento dos folículos no tecido linfóide e edema nodal. Resultados: A oesofagostomose é uma verminose, subdiagnosticada em ovinos, causa enterite linfoplasmocitária e nódulos intestinais, o que, em algumas situações, inviabiliza a ação dos antiparasitários. É importante, suspeitar desta enfermidade, em ovelhas que apresentam este histórico. Os achados clínicos e anatomopatológicos possibilitaram diagnosticar enterite linfoplasmocitária, causada por Oesophagostomum spp. em ovelha. Conclusão: O exame anatomopatológico possibilitou estabelecer o diagnóstico de enterite linfoplasmocitária, ressalta-se a importância dos exames complementares, na elucidação dos óbitos em animais de produção.
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Resende, Matheus Alex Araújo, and Rafaela Karin De Lima. "TERPENOS E OUTROS COMPOSTOS DERIVADOS PRESENTES EM QUEIJOS." In I Congresso Brasileiro de Biotecnologia Vegetal On-line. Revista Multidisciplinar de Educação e Meio Ambiente, 2021. http://dx.doi.org/10.51189/rema/2417.
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Introdução: O queijo é um rico produto alimentício que possuí diferentes voláteis em sua composição incluindo terpenóides. Visto a diversidade de produtos e de tipos desse alimento, a pesquisa buscou na literatura, artigos que utilizaram a Microextração em Fase Sólida acoplado a Cromatografia Gasosa (SPME-GC/MS) de queijos do tipo Minas Padrão, Minas Frescal, Coalho, Prato, Pecorino, Feta e Emmental no período de 2001 a 2021. Objetivo: Este resumo tem como objeto de estudo de terpenos e outros voláteis presentes nos queijos, buscando a análise de presença ou não destas moléculas em diferentes tipos de queijos com o objetivo de analisar como essas moléculas ajudam na particularidade dos aromas dos queijos. Material e Métodos: Os artigos para a revisão foram selecionados de acordo com sua relevância na plataforma Google Scholar dos últimos 20 anos, além da verificação do fator de impacto da revista da publicação. Assim, a base das palavras chaves utilizadas foram “Volatile compounds of cheeses”, “SPME analysis cheeses”, Minas chesses volatiles”, “Brazilian chesses volatiles” e “European cheeses volatile compunds”. Além de uma breve revisão da importância das moléculas voláteis e dos terpenos para os aromas presentes nos queijos, os artigos revisados também determinaram suas origens, que no caso dos terpenóides são relacionadas à alimentação dos ruminantes, de base vegetal. Também como método de identificação nas amostras, a Análise de Variância (ANOVA), o que permitiu uma interpretação dos compostos não somente qualitativamente, mas também quantitativamente. Resultados: Obteve-se uma tabela que apresenta voláteis identificados nos tipos de queijo em diversas amostras. Essa tabela se demonstrou útil para interpretar os diferentes tipos de queijo de acordo com os voláteis, incluindo terpenóides. Finalmente, tem-se uma breve discussão sobre pesquisas futuras nessa área e como os terpenos podem ser utilizados como marcadores moleculares nos tipos de queijos. Conclusão: Este trabalho mostra, através de uma revisão bibliográfica, que os compostos voláteis são um fator fundamental para o aroma e possuem uma grande diversidade de concentrações nos tipos de queijos estudados. Os resultados demonstraram a presença de diferentes moléculas de terpenos e outros voláteis, o que pode gerar sabores típicos de cada tipo de queijo.