Relevant bibliographies by topics / Feral ferrets

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  1. Journal articles
  2. Dissertations / Theses
  3. Conference papers

Academic literature on the topic 'Feral ferrets'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Feral ferrets"

1

Caley, Peter, L. M. McElrea, and Jim Hone. "Mortality rates of feral ferrets (Mustela furo) in New Zealand." Wildlife Research 29, no. 4 (2002): 323. http://dx.doi.org/10.1071/wr02004.

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Life-table data from feral ferret populations in New Zealand were analysed to estimate their mortality rates, and to test for any additive effect of Mycobacterium bovis infection on observed mortality rates. The observed instantaneous mortality rate was best estimated by modelling mortality as a 2-phase step model with different rates for juveniles (μ1 = 1.45 year–1, 95% C.I. 1.2–1.7 year–1) and adults (μ2 = 0.55 year–1, 95% C.I. 0.4–0.9 year–1). This corresponds to a survival probability of 0.25 during the first year of life, rising to 0.55 year–1 thereafter, and a life expectancy of 0.95 yea
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Medina‐Vogel, G., G. J. Hickling, and B. K. Clapperton. "Assessing spatial activity in captive feral ferrets,Mustela furoL. (Camivora: Mustelidae)." New Zealand Journal of Zoology 27, no. 2 (2000): 75–83. http://dx.doi.org/10.1080/03014223.2000.9518213.

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Byrom, Andrea E. "Dispersal and survival of juvenile feral ferrets Mustela furo in New Zealand." Journal of Applied Ecology 39, no. 1 (2002): 67–78. http://dx.doi.org/10.1046/j.1365-2664.2002.00689.x.

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Caley, P., J. Hone, and PE Cowan. "The relationship between prevalence ofMycobacterium bovisinfection in feral ferrets and possum abundance." New Zealand Veterinary Journal 49, no. 5 (2001): 195–200. http://dx.doi.org/10.1080/00480169.2001.36232.

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Henning, J., P. R. Davies, and J. Meers. "Seropositivity to rabbit haemorrhagic disease virus in non-target mammals during periods of viral activity in a population of wild rabbits in New Zealand." Wildlife Research 33, no. 4 (2006): 305. http://dx.doi.org/10.1071/wr03061.

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As part of a longitudinal study of the epidemiology of rabbit haemorrhagic disease virus (RHDV) in New Zealand, serum samples were obtained from trapped feral animals that may have consumed European rabbit (Oryctolagus cuniculus) carcasses (non-target species). During a 21-month period when RHDV infection was monitored in a defined wild rabbit population, 16 feral house cats (Felis catus), 11 stoats (Mustela erminea), four ferrets (Mustela furo) and 126 hedgehogs (Erinaceus europaeus) were incidentally captured in the rabbit traps. The proportions of samples that were seropositive to RHDV were
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Ogilvie, Shaun C., and Charles T. Eason. "Evaluation of iophenoxic acid and rhodamine B for marking feral ferrets(Mustela furo)." New Zealand Journal of Zoology 25, no. 2 (1998): 105–8. http://dx.doi.org/10.1080/03014223.1998.9518142.

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Gillies, Craig, and Matthew Brady. "Trialling monitoring methods for feral cats, ferrets and rodents in the Whangamarino wetland." New Zealand Journal of Zoology 45, no. 3 (2018): 192–212. http://dx.doi.org/10.1080/03014223.2017.1418756.

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8

Byrom, AE, P. Caley, BM Paterson, and G. Nugent. "Feral ferrets (Mustela furo) as hosts and sentinels of tuberculosis in New Zealand." New Zealand Veterinary Journal 63, sup1 (2015): 42–53. http://dx.doi.org/10.1080/00480169.2014.981314.

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9

Alterio, Nic. "Secondary poisoning of stoats (Mustela erminea), feral ferrets (Mustela furo),and feral house cats (Felis catus) by the anticoagulant poison, brodifacoum." New Zealand Journal of Zoology 23, no. 4 (1996): 331–38. http://dx.doi.org/10.1080/03014223.1996.9518092.

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10

Smith, G. P., J. R. Ragg, H. Moller, and K. A. Waldrup. "Diet of feral ferrets (Mustela furo) from pastoral habitats in Otago and Southland, New Zealand." New Zealand Journal of Zoology 22, no. 4 (1995): 363–69. http://dx.doi.org/10.1080/03014223.1995.9518054.

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Dissertations / Theses on the topic "Feral ferrets"

1

Morley, Craig Gordon. "The ecology and behaviour of feral ferrets (Mustela furo) in Canterbury farmland, New Zealand." Thesis, University of Canterbury. Zoology, 1999. http://hdl.handle.net/10092/5824.

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Ferret (Mustela furo) control programs, especially those attempting to prevent the spread of Mycobacterium bovis (Tb), would greatly benefit from practical knowledge of ferret ecology and behaviour. This study had two main objectives: firstly, to investigate ferret ecology by examining ferret abundance, survival, trapping success, and diet; and, secondly, to investigate ferret behaviour and activity patterns by using an acoustically sensitive transmitter system (ASTS). Abundance and survival estimates of ferrets tagged with passive integrated transponders (PITs), showed a marked seasonal varia
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2

Caley, Peter, and n/a. "Inference on the host status of feral ferrets (Mustela furo) in New Zealand for Mycobacterium bovis infection." University of Canberra. Resource, Environment & Heritage Studies, 2001. http://erl.canberra.edu.au./public/adt-AUC20050621.140940.

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This thesis is about making inference on the host status of feral ferrets in New Zealand for Mycobacterium bovis, the aetiological agent of bovine tuberculosis. The central question addressed is whether the rate of intra-specific transmission of M. bovis among ferrets is sufficient for the disease to persist in ferret populations in the absence of external, non-ferret sources of infection (inter-specific transmission). The question is tackled in three parts�firstly using model selection to identify suitable models for estimating the force of M. bovis infection in ferret populations; secondly a
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Paul, Arco P. "Overexpression of Serum Response Factor in Astrocytes Improves Neuronal Plasticity in a Model of Fetal Alcohol Spectrum Disorders." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/307.

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Neuronal plasticity deficits underlie many of the neurobehavioral problems seen in Fetal Alcohol Spectrum Disorders (FASD). Recently, we showed that third trimester alcohol exposure lead to a persistent disruption in ocular dominance (OD) plasticity. For instance, few days of monocular deprivation results in a robust reduction of cortical regions responsive to the deprived eye in normal animals, but not in ferrets exposed early to alcohol. This plasticity deficit can be reversed if alcohol-exposed animals are treated with a phosphodiesterase type 1 (PDE1) inhibitor during the period of monocul
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Conference papers on the topic "Feral ferrets"

1

Singh, S. K., and H. Ishiwara. "Bismuth ferrite Thin Films for Advanced FeRAM Devices." In 2005 International Conference on Solid State Devices and Materials. The Japan Society of Applied Physics, 2005. http://dx.doi.org/10.7567/ssdm.2005.h-8-3.

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2

Knutsen, Andrew K., Jason E. Hill, Jeffrey J. Neil, Terrie E. Inder, and Philip V. Bayly. "Quantification of Cortical Surface Convolution in the Developing Ferret and Human Infant." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205716.

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The human cerebral cortex undergoes folding from the 5th fetal month into the first post-natal year. Disturbances of folding have serious and lasting consequences, but the mechanism is not well understood. Van Essen [1] has hypothesized that axonal tension between strongly interconnected regions draws them together and induces outward folds. However, no direct measurements have confirmed this theory. As measures of shape, cortical curvature and sulcal depth changes during development can help provide insight into underlying mechanisms of growth.
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Knutsen, Andrew K., Christopher D. Kroenke, Larry A. Taber, Jeffrey J. Neil, and Philip V. Bayly. "MRI Measurement of Surface Strain Due to Growth in the Developing Ferret Brain." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176010.

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The human cerebral cortex undergoes folding from the 5th fetal month into the first post-natal year. Disturbances of folding have serious and lasting consequences, but the mechanism is not well understood. Van Essen [1] has hypothesized that axonal tension between strongly interconnected regions draws them together and induces outward folds. However, no direct measurements have confirmed this theory. Experimental data are needed, beginning with a quantitative description of the kinematics of cortical growth during folding.
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Journal articles
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