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Journal articles on the topic 'Family history of coronary heart disease'

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Published: 9 March 2023

Last updated: 10 March 2023

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1

Friedman, Gary D., and Arthur L. Kaltsky. "RE: FAMILY HISTORY OF CORONARY HEART DISEASE." American Journal of Epidemiology 126, no. 1 (July 1987): 159. http://dx.doi.org/10.1093/oxfordjournals.aje.a114655.

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2

Nasir, Khurram, Matthew J. Budoff, Nathan D. Wong, Maren Scheuner, David Herrington, Donna K. Arnett, Moyses Szklo, Philip Greenland, and Roger S. Blumenthal. "Family History of Premature Coronary Heart Disease and Coronary Artery Calcification." Circulation 116, no. 6 (August 7, 2007): 619–26. http://dx.doi.org/10.1161/circulationaha.107.688739.

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3

Nasir, Khurram, Erin Donnelly Michos, John A. Rumberger, Joel B. Braunstein, Wendy S. Post, Matthew J. Budoff, and Roger S. Blumenthal. "Coronary Artery Calcification and Family History of Premature Coronary Heart Disease." Circulation 110, no. 15 (October 12, 2004): 2150–56. http://dx.doi.org/10.1161/01.cir.0000144464.11080.14.

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4

O’Donnell, Christopher J. "Family History, Subclinical Atherosclerosis, and Coronary Heart Disease Risk." Circulation 110, no. 15 (October 12, 2004): 2074–76. http://dx.doi.org/10.1161/01.cir.0000145539.77021.ac.

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5

Crouch, Michael A., and Robert Gramling. "Family History of Coronary Heart Disease: Evidence-Based Applications." Primary Care: Clinics in Office Practice 32, no. 4 (December 2005): 995–1010. http://dx.doi.org/10.1016/j.pop.2005.09.008.

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6

Iqbal, Usman Javed, Majid Kaleem, and Muhammad Imran Hanif. "CORONARY ARTERY DISEASE." Professional Medical Journal 22, no. 05 (May 10, 2015): 532–35. http://dx.doi.org/10.29309/tpmj/2015.22.05.1261.

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Background: Development of heart failure is always secondary to presenceof risk factors like diabetes mellitus, hypertension, age, smoking and underlying coronaryartery disease. Objective: The objective of this study was to find the frequency of risk factorsand coronary artery involvement in patients of heart failure after myocardial infarction. StudyDesign: A non-randomized cross sectional study. Setting: Gulab Devi Chest Hospital. Period:Six months. Methods: 100 patients was done. Using non probability (purposive) samplingtechnique; all patients with ages between 20 to 80 years, irrespective of gender, diagnosedwith heart failure (with ejection fraction ≤ 40%) secondary to STEMI were included in thestudy. Patients that were not having heart failure secondary to STEMI and those that hadn’ttheir coronary angiography done were excluded from the study. The data were analyzed usingSPSS Version 20. Descriptive statistics was used to see analyze the data. Results: Mean ageof patients was 52.61±10.2years. There was an overall male predominance (81%). Commonrisk factors that we observed were smoking (65%), hypertension (62%), diabetes (58%) andpositive family history (38%). In this study 51% patients had triple vessels disease, 15% patientshad double vessels disease and 34% patients had single vessel disease. Most common lesionwas of LAD following LCX, RCA and LMS. Conclusion: Our study conclude that coronaryartery disease is the main causative factor for the development of heart failure in patients ofmyocardial infarction and even a single vessel disease can lead to heart failure with severesystolic dysfunction. Most common associated risk factor was smoking, hypertension being thesecond most common following diabetes and positive family history.

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Nadir, M. A., and A. D. Struthers. "Family history of premature coronary heart disease and risk prediction." Heart 97, no. 8 (March 1, 2011): 684. http://dx.doi.org/10.1136/hrt.2011.222265.

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8

Wrigley, Martha, and Judith Lathlean. "Family history of premature coronary heart disease: Discussing the evidence." British Journal of Cardiac Nursing 4, no. 12 (December 2009): 569–74. http://dx.doi.org/10.12968/bjca.2009.4.12.45680.

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9

Wrigley, Martha, and Judith Lathlean. "Family history of premature coronary heart disease: Exploring the experience." British Journal of Cardiac Nursing 5, no. 1 (January 2010): 40–45. http://dx.doi.org/10.12968/bjca.2010.5.1.46032.

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10

ZOLER, MITCHEL L. "Family History of Heart Disease Boosts Coronary Risk in Sisters." Internal Medicine News 38, no. 3 (February 2005): 64. http://dx.doi.org/10.1016/s1097-8690(05)71512-2.

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11

Prabhakaran, Dorairaj, and Panniyammakal Jeemon. "Should Your Family History of Coronary Heart Disease Scare You?" Mount Sinai Journal of Medicine: A Journal of Translational and Personalized Medicine 79, no. 6 (November 2012): 721–32. http://dx.doi.org/10.1002/msj.21348.

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12

Conroy, R. M., R. Mulcahy, N. Hickey, and L. Daly. "Is a family history of coronary heart disease an independent coronary risk factor?" Heart 53, no. 4 (April 1, 1985): 378–81. http://dx.doi.org/10.1136/hrt.53.4.378.

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13

Koulaouzidis, G., K. Wong, D. Charisopoulou, T. McArthur, P. J. Jenkins, and M. Y. Henein. "Coronary artery calcification is not related to coronary heart disease isolated family history." International Journal of Cardiology 190 (July 2015): 271–72. http://dx.doi.org/10.1016/j.ijcard.2015.04.147.

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14

Muratova, Viktorina N., Syed S. Islam, Emily C. Spangler, Ellen W. Demerath, and William A. Neal. "Cholesterol Screening in Children and Family History of Coronary Heart Disease." Circulation 103, suppl_1 (March 2001): 1369. http://dx.doi.org/10.1161/circ.103.suppl_1.9998-94.

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P94 Background: Selective blood cholesterol screening of children based upon National Cholesterol Education Program (NCEP) guidelines of family history of premature cardiovascular disease (CVD) or parental hypercholesterolemia is inadequate in a population with high prevalence of coronary heart disease (CHD), low levels of cholesterol screening, low socio-economic status (SES) and diminished access to preventive health care. We hypothesize that universal cholesterol screening of pre-pubertal school children may be effective in identifying children and their parents with abnormal lipid levels in this high risk rural population. Fifth grade school children from seven rural Appalachian counties participated in a school based cholesterol screening program. Data on family history of premature CHD, anthropometric and blood pressure measurements, tobacco smoke exposure, dietary history and physical activity levels were collected at the time of screening. Seven hundred and nine 5 th grade students ( mean age 10.8 years) participated in the program. One hundred seventy four (24.5%) were considered presumptively dyslipidemic after non-fasting finger- stick (FS) cholesterol screening. Thirty six percent of these dyslipidemic children had a fasting lipid profile done. Dyslipidemia was confirmed in 37(59%) of these children. FS cholesterol levels were significantly correlated with fasting TC (r=0.80 p < 0.0001). Among confirmed dyslipidemic children, family history was not a good predictor of dyslipidemia (sensitivity 21.6%). Seventy nine parents of dyslipidemic children participated in fasting lipid profile assessment. Fifty two parents (67%) were dyslipidemic, most of them (79%) did not have a family history of premature CHD or hypercholesterolemia. FS cholesterol levels were also correlated with fasting TC of fathers (r=0.46 p=0.01), and mothers (r=0.32 p=0.02). Conclusion: Significant correlation exists between non-fasting FS cholesterol levels of children and subsequent fasting lipid profile of children and their parents. Family history has low sensitivity in predicting children with elevated serum cholesterol concentrations.

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Tavares, Paula, Andreia Oliveira, and Carla Lopes. "Family history of coronary heart disease, health care and health behaviors." Revista Portuguesa de Cardiologia 30, no. 9 (September 2011): 703–10. http://dx.doi.org/10.1016/s0870-2551(11)70013-0.

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Tavares, Paula, Andreia Oliveira, and Carla Lopes. "Family history of coronary heart disease, health care and health behaviors." Revista Portuguesa de Cardiologia (English Edition) 30, no. 9 (September 2011): 703–10. http://dx.doi.org/10.1016/s2174-2049(11)70013-7.

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17

Kekäläinen, Päivi, Helena Sarlund, Kalevi Pyörälä, and Markku Laakso. "Family history of coronary heart disease is a stronger predictor of coronary heart disease morbidity and mortality than family history of non-insulin dependent diabetes mellitus." Atherosclerosis 123, no. 1-2 (June 1996): 203–13. http://dx.doi.org/10.1016/0021-9150(96)05808-x.

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18

Sun, Lianlian, Tingting Wu, Minyue Zhang, Saiyan Huang, Zhangfang Zeng, and Yujuan Wu. "Investigation on Family Support System and Willingness of Patients to Participate in Cardiac Rehabilitation after Percutaneous Coronary Intervention." Evidence-Based Complementary and Alternative Medicine 2022 (June 7, 2022): 1–6. http://dx.doi.org/10.1155/2022/3633188.

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Objective. We attempt to discuss the relationship between family support and willingness to participate in exercise rehabilitation in coronary heart disease patients after PCI to provide effective guidance for improving the quality of life of coronary heart disease patients after PCI. Methods. By convenient sampling, we selected 90 coronary heart disease patients in cardiology department from September 2021 to January 2022, using general information questionnaire, rehabilitation exercise knowledge, attitude, and behavior questionnaire of patients with coronary heart disease, and the social support scale to investigate the subjects. Results. The total score of knowledge, belief, and behavior in patients with coronary heart disease was 33.02 ± 6.28 points, the social support scale score was 39.63 ± 6.07 points, the multiple linear regression revealed that the educational level, history of cardiovascular disease, and the number of coronary stents of coronary heart disease patients after PCI are the main influencing factors that affect the willingness of coronary heart disease patients to participate in exercise rehabilitation. Conclusion. Rehabilitation exercise knowledge, belief, and behavior scores in coronary heart disease patients are low, and social support is negatively correlated with rehabilitation exercise in coronary heart disease patients.

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19

Chacko, Manas, P. Sankara Sarma, Sivadasanpillai Harikrishnan, Geevar Zachariah, and Panniyammakal Jeemon. "Family history of cardiovascular disease and risk of premature coronary heart disease: A matched case-control study." Wellcome Open Research 5 (April 16, 2020): 70. http://dx.doi.org/10.12688/wellcomeopenres.15829.1.

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Background: Self-reported family history of cardiovascular disease (CVD) is an independent risk factor for future coronary heart disease (CHD) events. However, inclusion of family history of CVD in the traditional risk scores failed to improve risk prediction of CHD. It is proposed that family history of CVD may substantially increase the risk of CHD among younger individuals. Methods: We conducted a matched case-control study with 170 hospital-based premature CHD patients (<55 years in men and <65 years in women) from a tertiary care centre in Thiruvananthapuram, Kerala and age and sex matched community-based controls in 1:1 ratio. Conditional logistic regression analysis was conducted to assess the independent association of family history of cardiovascular disease (CVD) and premature CHD. We estimated McNemar's odds ratios and their 95 percent confidence intervals. Results: The prevalence of any family history of CVD and CHD in the control population was 24% and 21%, respectively. The family history of CVD was independently associated with premature CHD (odds ratio (OR) = 9.0; 95% confidence interval (CI) 4.7–17.3). There was a dose-response relationship between family history and premature CHD as the risk increased linearly with increase in number of affected family members. Conclusions: Family history of CVD is an independent risk factor for premature CHD. The risk of premature CHD increases linearly with increase in number of affected family members. Collecting family history beyond parental history of CVD is important for risk stratification. Targeting young individuals with family history of CVD for intensive risk reduction interventions may help to prevent future events.

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Chacko, Manas, P. Sankara Sarma, Sivadasanpillai Harikrishnan, Geevar Zachariah, and Panniyammakal Jeemon. "Family history of cardiovascular disease and risk of premature coronary heart disease: A matched case-control study." Wellcome Open Research 5 (June 12, 2020): 70. http://dx.doi.org/10.12688/wellcomeopenres.15829.2.

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Background: Self-reported family history of cardiovascular disease (CVD) is an independent risk factor for future coronary heart disease (CHD) events. However, inclusion of family history of CVD in the traditional risk scores failed to improve risk prediction of CHD. It is proposed that family history of CVD may substantially increase the risk of CHD among younger individuals. Methods: We conducted a matched case-control study with 170 hospital-based premature CHD patients (<55 years in men and <65 years in women) from a tertiary care centre in Thiruvananthapuram, Kerala and age and sex matched community-based controls in 1:1 ratio. Conditional logistic regression analysis was conducted to assess the independent association of family history of cardiovascular disease (CVD) and premature CHD. We estimated McNemar's odds ratios and their 95 percent confidence intervals. Results: The prevalence of any family history of CVD and CHD in the control population was 24% and 21%, respectively. The family history of CVD was independently associated with premature CHD (odds ratio (OR) = 9.0; 95% confidence interval (CI) 4.7–17.3). There was a dose-response relationship between family history and premature CHD as the risk increased linearly with increase in number of affected family members. Conclusions: Family history of CVD is an independent risk factor for premature CHD. The risk of premature CHD increases linearly with increase in number of affected family members. Collecting family history beyond parental history of CVD is important for risk stratification. Targeting young individuals with family history of CVD for intensive risk reduction interventions may help to prevent future events.

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Patra, Tusharkanti, Prashant Kumar, Somnath Mukherjee, Anurag Passi, and S. K. Saidul Islam. "A comprehensive work up of various ventricular tachy-arrhythmias in relation to the underling cardiac disorder / status." International Journal of Research in Medical Sciences 5, no. 10 (September 28, 2017): 4292. http://dx.doi.org/10.18203/2320-6012.ijrms20174161.

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Background: Main objective of the study is details work up of the patients of ventricular tachy-arrhythmias and to find out its association with any structural heart disease.Methods: This institution based observational study was conducted in patients of documented sustained VT (ventricular tachycardia) with consecutive 102 patients.Results: The mean age of the VT patients was 56.7 years and the number of male patients were 70 (69%). In our study, among 102 patients 45 patients were diabetic, 64 patients were hypertensive, 30 patients were current smoker, family history of heart disease was present in 25 patients and family history of SCD (sudden cardiac death) was present in 5 patients. Among the patients who presented with symptoms of ventricular tachy arrhythmia, 25 patients had EF (ejection fraction) above 40%, 36 had EF between 31 to 40% and only 2 had EF below 30%. CAG (coronary angiography) done in 98 patients and 16 had normal coronaries. 20, 16 and 46 patients had single, double and triple vessel disease respectively. 80 patients had coronary heart disease (78%), 20 patients among them had acute ischemic events and 60 had chronic ischemic disease. 12 patients didn’t have any structural heart disease.Conclusions: Ischemic heart disease, acute or chronic, is the most common causes of ventricular tachyarrhythmia. male sex, diabetes mellitus, hypertension, smoking, family history of heart diseases or sudden cardiac death being the risk factors of coronary artery disease are also predisposing factors of ventricular tachyarrhythmia.

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22

Ahmad, Munir, Muhammad Yasir, Muhammad Hamid Saeed, Muhammad Saeed Ali Khan, Qasim Rauf, and Naeem Hameed. "Frequency of obstructive coronary artery disease in patients undergoing valvular heart disease surgery." Professional Medical Journal 27, no. 06 (June 10, 2020): 1297–303. http://dx.doi.org/10.29309/tpmj/2020.27.06.4675.

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Objectives: To determine the frequency of obstructive coronary artery disease in patients undergoing valvular heart disease surgery. Study Design: Cross-sectional study. Setting: Department of Cardiology, Faisalabad Institute of Cardiology, Faisalabad. Period: Six months, from 02 February, 2017 to 01 August, 2017. Material & Methods: After taking approval from hospital ethical committee, patients coming through outpatient department who fulfilled the inclusion criteria were enrolled and informed consent was taken from them. History of smoking, diabetes mellitus, renal dysfunction, dyslipidemia, hypertension and family history of coronary artery disease was assessed. Coronary angiography was performed by senior consultant interventional cardiologist for assessing obstructive coronary artery disease as per operational definition. All the information was collected on prespecified Performa. Results: In this study, out of 140 cases of valvular heart disease (VHD), 47.14%(n=66) were between 30-50 years of age whereas 52.86%(n=74) were between 51-70 years of age, mean ±sd was calculated as 51.71+9.09 years, 57.14%(n=80) were male while 42.86%(n=60) were female, frequency of obstructive coronary artery disease in patients undergoing valvular heart surgery was recorded as 29.3% (n=41) whereas 70.7% (n=99) had no such finding. Conclusion: The frequency of obstructive coronary artery disease was (29.3%) in patients undergoing valvular heart disease surgery. However, coronary artery disease was less frequent in rheumatic as compared to degenerative heart valve disease.

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23

Sumara, Retno, Nugroho Ari, and Indarti Indarti. "Identifikasi Faktor Kejadian Penyakit Jantung Koroner Terhadap Wanita Usia ≤ 50 Tahun di RSU Haji Surabaya." Jurnal Manajemen Asuhan Keperawatan 6, no. 2 (July 27, 2022): 53–59. http://dx.doi.org/10.33655/mak.v6i2.134.

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Coronary Heart Disease (CHD) has become the main cause of death in the world and in Indonesia. Many people have heart attacks without any symptoms. Men have a higher risk of developing coronary heart disease sooner than women. Women have a risk of coronary heart disease during menopause. Typical symptoms felt by patients with coronary heart disease are chest pain that radiates to the back, arms, neck. This study aims to identify the risk factors for coronary heart disease in women aged 50 years at RSU Haji Surabaya. The design of descriptive analysis was carried out on all patients with CHD as many as 30 patients with Consecutive Sampling with medical record data instruments (history of hypertension, obesity, cholesterol, smoking and diabetes mellitus). The results showed that CHD in women 50 years was dominantly caused by a history of hypertension 63%, history of high cholesterol and obesity 56.7%, history of DM 43.3%, family history 10%, history of smoking 6.7%. One of the main factors causing CHD is hypertension. Increased pressure of blood vessels against arterial walls can result in endothelial damage that triggers atherosclerosis.

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Delmi, Anwari, Dwi Laksono Adiputro, Dona Marisa, and Alfi Yasmina. "Prevalence and Determinant Factors of Coronary Heart Disease Status in Patients with Heart Failure." Berkala Kedokteran 17, no. 1 (March 2, 2021): 55. http://dx.doi.org/10.20527/jbk.v17i1.10249.

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Abstract: As a cardiovascular disease, heart failure is one of prime mortality cause in the world. Coronary heart disease has a role in development of heart failure so that it becomes one of heart failure’s major clinical risk factor. This study aimed to calculate the prevalence of and identify the factors that influence coronary heart disease status of heart failure patients at Alamanda ward of RSUD Ulin Banjarmasin in 2016. Factors that were studied consisted of age, sex, family history of heart disease, smoking, diabetes mellitus, and hypertension. This study used analytical observation with cross-sectional approach on medical records that were obtained by purposive sampling method. The result showed 46.4% of 263 heart failure patients had coronary heart disease status. Multivariate analysis inferred hypertension as the factor that influences the coronary heart disease status of heart failure patients (OR = 1.98, 95% CI = 1.06 – 3.71, P = 0.033). Keywords: cardiovascular disease, heart failure, coronary heart disease, hypertension

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25

Saito, Tomohiro, Toshihito Furukawa, Seiichiro Nanri, Ikuo Saito, and Shiro Nagano. "Importance of Age Factor in Assessing Family History of Coronary Heart Disease." Journal of Epidemiology 5, no. 3 (1995): 109–12. http://dx.doi.org/10.2188/jea.5.109.

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Yarnell, John, Shicheng Yu, Chris Patterson, François Cambien, Dominique Arveiler, Philippe Amouyel, Jean Ferrières, Gérald Luc, Alun Evans, and Pierre Ducimetière. "Family history, longevity, and risk of coronary heart disease: the PRIME Study." International Journal of Epidemiology 32, no. 1 (February 2003): 71–77. http://dx.doi.org/10.1093/ije/dyg038.

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PERKINS, KENNETH A. "FAMILY HISTORY OF CORONARY HEART DISEASE: IS IT AN INDEPENDENT RISK FACTOR?" American Journal of Epidemiology 124, no. 2 (August 1986): 182–94. http://dx.doi.org/10.1093/oxfordjournals.aje.a114377.

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28

Low, Kathryn Graff, Carl E. Thoresen, Jerry R. Pattillo, and Nancy Fleischmann. "Causal Attributions and Coronary Heart Disease in Women." Psychological Reports 73, no. 2 (October 1993): 627–36. http://dx.doi.org/10.2466/pr0.1993.73.2.627.

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The relationship between coronary heart-disease endpoints and attributional style in women has been previously unexamined. This study examined the attributions of 73 postmyocardial infarction (MI) women about their heart disease and explored the relationship between attributions and nonfatal coronary recurrence Women's primary causal attributions included personal behavior (9.6%), blaming others (19.3%), stress (28.8%), luck (12.3%), and family history (13.7%). The largest proportion of recurrences occurred in women attributing their infarcts to marital problems. Of the attributional ratings, ascriptions involving spouses were the only attributions that met entry criteria for logistic regression ( p = .019) after controlling for severity of first infarction.

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Rubiyaktho, David, and Cholid Tri Tjahjono. "Comparison of Predicted Significant Coronary Lesion by Duke Treadmill Score among Coronary Heart Disease Risk Factors in Patients with Positive Ischemic Response Treadmill Test." ACI (Acta Cardiologia Indonesiana) 4, no. 1 (July 6, 2018): 1. http://dx.doi.org/10.22146/aci.36632.

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Background: According to Framingham Study, independent risk factors for coronary heart disease (CHD) are diabetes, hypertension, smoking, dyslipidemia, family history of CHD and obesity. Previous study reported cut-off value of Duke Treadmill Score (DTS) < -0.5 represents a significant coronary lesion with positive predictive value 88.4%. Objective: To compare the incidence of predicted significant coronary lesions by DTS among various risk factors for coronary heart disease. Methods: A cross sectional study was done on 292 patients age 18 to 74 years old who had positive exercise testing for CAD screening during period of June 1st 2016 until May 30th 2017. DTS was calculated from treadmill test as: exercise time - (5 x ST deviation in mm) - (4 x exercise angina). A coronary lesion was predicted significant with DTS cut off value < -0.5. Results: Subjects mean age was 57 years old, male were 60.4%. The risk factors for CHD were found sequentially from the most frequent were hypertension 51.9%, smoking 35.3%, diabetes mellitus 23.1%, dyslipidemia 11.9%, obesity 4.2% and family history of CHD 6.3%. It was found that diabetes was significantly different from its effect on DTS value with p value = 0.021, while hypertension, obesity, dyslipidemia and family history CHD had no significant effect. Logistic regression found consistently that diabetes was significant (p=0.019). Conclusion: Predicted significant coronary lesions by DTS developed more frequent in diabetes compared to, hypertension, smoking, dyslipidemia, obesity and family history of coronary heart disease.

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Conkbayir, Cenk, Didem Melis Oztas, and Murat Ugurlucan. "Risk factors for Turkish Cypriot patients with coronary artery disease." Archives of Medical Science – Atherosclerotic Diseases 5, no. 1 (December 30, 2020): 332–34. http://dx.doi.org/10.5114/amsad.2020.103380.

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IntroductionIschemic heart disease is the leading cause of death worldwide. In this study, we evaluated the risk factors for Turkish Cypriot ischemic heart disease patients.Material and methodsIn this study we examined 7017 patients, retrospectively. 5.9% of the patients had myocardial infarction coronary artery disease (MICAD) and 94.1% of the patients were control patients. The mean age of the study population was 52, and 39.2% were female. The risk factors considered were: age, sex, hypertension (HT), diabetes mellitus (DM), family history of coronary artery disease, smoking, high total cholesterol levels and obesity.ResultsConsistently with the presumed risk factors, there was a significant difference between MICAD and control groups in our study for smoking, HT, DM and obesity. However, our study revealed no significant difference for high total cholesterol levels in contrast to the current literature. Assessing the risk factors among male and female patient groups separately, both men and women have the highest risk prevalence for hypertension. Smoking is the second most common risk factor among males, whereas it is the family history of coronary heart disease in the female group. HT, DM and obesity were found to be significant risk factors for both males and females in our study group, while family history was revealed to be significant only in women.ConclusionsModifiable risk factors such as smoking, hypertension, diabetes mellitus and obesity may be controlled by lifestyle changes and medical therapies, and should be approached with caution for preventing cardiac events. Non-modifiable risk factors such as age, gender, family history and ethnicity should be taken into account for disease prediction.

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Stetkiewicz, A., A. Goch, and A. Borkowska. "Depression in Coronary Heart Disease." European Psychiatry 24, S1 (January 2009): 1. http://dx.doi.org/10.1016/s0924-9338(09)70915-0.

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Background and aims:Current studies showed a significant association between depression and Coronary Heart Disease (CHD). Depression is a significant etiopathogenetic factor of CHD and caused with worsening of the course of the illness. The aim of this study was to asses the relationship between the intensity of depressed symptoms and the course of the illness and also demographic factors.Methods:The group of 111 CHD patients (89 male, 22 female) aged 41-65 years (mean 55±5,6). The individuals were assessed according to the following criteria: sex, job status (working, unemployed, retired), vascular diseases in family, myocardial infarction (MI) in history, number of MI, patients with acute MI were divided according to the duration of time from the onset of MI pain (patients who passed MI within a week and above a week from the onset of MI). The intensity of depressive symptoms were assessed by subjective Beck Depression Inventory (BDI) and objective Hamilton Depression Rating Scale (HDRS).Results:The intensity of depressed symptoms in BDI was 9,9±7,5 and in HDRS 6,6±4,5. Females showed significant higher level of depression compared with males.In patients who passed MI within a week before the evaluation as compared with those whose term after MI was longer than a week the less level of depression was observed. Also active workers presented significantly less degree of depressive symptoms than pensioners.Conclusion:The results obtained showed the association between the intensity of depression and the more severe course of the CHD, especially in female and pension patients.Acknowledgements:This research was supported by grant 502-16-653 Medical University of Lodz

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Deviana Widayanti and Chatarina Setya Widyastuti. "Faktor Risiko Terjadinya Penyakit Jantung Koroner pada Pasien di Unit Rawat Jalan Rumah Sakit Panti Rapih Yogyakarta." I Care Jurnal Keperawatan STIKes Panti Rapih 1, no. 1 (May 6, 2020): 21–30. http://dx.doi.org/10.46668/jurkes.v1i1.34.

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Background: Coronary Heart Disease (CHD) Is a condition when the arteries that supply blood to the heart wall experience hardening and narrowing. It is estimated that 30% of coronary heart disease causes death worldwide. Objective: This study aims to determine the risk factors for CHD in Panti Rapih Hospital. Methods: This descriptive study aims to determine the risk factors for CHD in outpatients at Panti Rapih Hospital. The population is patients who have been diagnosed with coronary heart disease and the sample was taken by 50 respondents with non-random accidental sampling technique. This research take the data use questionnaire and make univariat analysis. Results: Risk factors for CHD are a number of factors that cannot be changed: family history of 42%, age = 40 years 95% in men and 95% age = 65 years in women. Factors that can be changed are: Smoking 78%, history of hypertension 68%, history of diabetes mellitus 28%, dyslipidemic 90%, excess body weight42% and lack of exercise 38%. Conclusion: Risk factors for CHD that cannot be changed: family history of 42%, age = 40 years 95% in men and 95% age = 65 years in women. Factors that can be changed are: Smoking 78%, history of hypertension 68%, history of diabetes mellitus 28%, dyslipidemic 90%, excess body weight 42% and lack of exercise 38%. Keywords: coronary heart disease, risk factors

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Wadowski, Stephen J., Robert J. Karp, Renee Murray-Bachmann, and Carl Senft. "Family History of Coronary Artery Disease and Cholesterol: Screening Children in a Disadvantaged Inner-City Population." Pediatrics 93, no. 1 (January 1, 1994): 109–13. http://dx.doi.org/10.1542/peds.93.1.109.

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Background and purpose. Little information relating to cholesterol levels and screening for hypercholesterolemia in inner-city children exists. For this study, given the disrupted family backgrounds of many of our patients and the unreliability of family histories, our hypothesis was that in comparison with other samples, family history of coronary heart disease would be a poor screening tool for the identification of children with elevations in total serum cholesterol (TSC). Subjects and methods. During 15 months, more than 400 pediatric outpatients, 2 through 14 years old, were screened for a family history of atherosclerotic disease. These children were attending a clinic serving a disadvantaged black and Hispanic population at Kings County Hospital Center. Nonfasting TSC levels were measured in 300 children. Positive risk for coronary heart disease was determined by the presence of a family history of coronary heart disease (defined as angina, stroke, or myocardial infarction in any parent or grandparent) at less than 55 years age. Results. The mean TSC level was 4.27 mmol/L (SD ± 0.85) (165.0 mg/dL [SD ± 32.81]). The 29.4% of this population with a history suggestive of high risk for hypercholesterolemia had a mean TSC of 4.48 mmol/L (SD ± 0.971) (173.2 mg/dL [SD ± 37.5]), and those with no risk history had a mean TSC of 4.18 mmol/L (SD ± 0.750) (161.4 mg/dL [SD ± 29.91) (P < .005). Use of family history of coronary artery disease as a screening tool had a sensitivity of 39.3%, a specificity of 74.5%, and a positive predictive value of 39.8% for detection of moderate hypercholesterolemia (TSC ≥ 4.66 mmollL [180 mg/dL]). Conclusions. This population's mean TSC level did not differ (P > .10) from those obtained in multiple large studies of average North American populations, and the predictive value and sensitivity of family history as a screening tool was comparable, although the prevalence of a positive family history was greater. The findings may be due to a greater prevalence of coronary artery disease at a young age in these families. In this population, a positive risk history is an important indicator for further evaluation of these children.

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Pankow, James S., Aaron R. Folsom, Michael A. Province, D. C. Rao, John Eckfeldt, Gerardo Heiss, Eyal Shahar, and Kenneth K. Wu. "Family History of Coronary Heart Disease and Hemostatic Variables in Middle-Aged Adults." Thrombosis and Haemostasis 77, no. 01 (1997): 087–93. http://dx.doi.org/10.1055/s-0038-1655912.

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SummaryIndividuals with a family history of coronary heart disease (CHD) may be predisposed to atherothrombosis. To investigate this hypothesis, a family CHD risk score was computed for approximately 13,000 men and women aged 45 to 64; hemostatic variables (fibrinogen, factor VIIc, factor VIIIc, von Willebrand factor, antithrombin III, protein C) were also measured in plasma. After adjustment for age and ethnicity, there was a statistically significant, positive association between the family risk score and four of the six hemostatic variables (fibrinogen, factor VIIc, factor VIIIc, von Willebrand factor) in women and all six hemostatic variables in men. In general, these associations were weak and substantially attenuated after adjustment for constitutional, lifestyle, and biochemical covariates. These results indicate that mean levels of selected hemostatic variables, like traditional CHD risk factors, are higher in individuals with a family history of heart disease.

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Lockyer, Lesley. "Women's Interpretation of their Coronary Heart Disease Symptoms." European Journal of Cardiovascular Nursing 4, no. 1 (March 2005): 29–35. http://dx.doi.org/10.1016/j.ejcnurse.2004.09.003.

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Background: Men and women are known to delay in seeking medical attention when experiencing acute cardiac symptoms. However, women are more likely to have had chronic undifferentiated symptoms prior to an acute episode and then when experiencing an acute episode delay longer than do men. Studies have shown once women do present they tend to be further along the disease trajectory than men. Aims: The aim of this study was to explore women's own interpretation of the presenting symptoms they experienced. Methods: Twenty-nine women consented to take part in semi-structured interviews that focussed on their experience of coronary heart disease (CHD). The women's narrative around the initial disruption of their illness was analysed focusing on routine and reconstructed forms of narration. Results: The results showed that many failed to recognise and act on their symptoms. Most only sought medical attention after family or friends intervened. None of the women had seen themselves at risk of developing coronary heart disease despite many have a strong family history. Conclusion: The women's early symptoms of coronary heart disease were often undifferentiated and difficult to interpret by the women themselves. There is a need for further research to understand and recognise early symptoms of coronary heart disease in women. This will allow women and health professionals to assess risk and identify women with coronary heart disease early on in their disease trajectory so therapy to prevent the morbidity associated with coronary heart disease and acute life threatening episodes will be reduced.

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Barry, Timothy A., and Douglas R. Wassenaar. "An Investigation into the Relationship between Coronary Risk Factors and Coronary Heart Disease among the Pietermaritzburg Asian Population." South African Journal of Psychology 26, no. 1 (March 1996): 29–34. http://dx.doi.org/10.1177/008124639602600106.

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This study investigated the relationship between coronary heart disease and a number of established coronary risk factors among Indian males. Included were factors such as Type A behaviour pattern (sometimes referred to as the coronary-prone behaviour pattern). The study investigated whether the following coronary risk factors were able to distinguish those with myocardial infarction from controls: Type A behaviour pattern, stress levels, anger levels, diabetes, previous coronary heart disease, hypertension, obesity, family history of coronary heart disease, physical exercise, cigarette smoking and alcohol consumption. Statistical analysis indicated that none of the risk factors, except Type A behaviour pattern, were able to significantly distinguish the myocardial infarction group from controls.

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Silberberg, J. S., J. Wlodarczyk, J. Fryer, R. Robertson, and M. J. Hensley. "Risk Associated with Various Definitions of Family History of Coronary Heart Disease: The Newcastle Family History Study II." American Journal of Epidemiology 147, no. 12 (June 15, 1998): 1133–39. http://dx.doi.org/10.1093/oxfordjournals.aje.a009411.

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Bhosale, Aryan S., Manju Chandankhede, and Pratibha Dawande. "Review Article on Coronary Artery Disease." ECS Transactions 107, no. 1 (April 24, 2022): 15349–53. http://dx.doi.org/10.1149/10701.15349ecst.

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Coronary Artery Disease (CAD) is a dangerous condition caused by the formation of plaque in the blood vessels, which leads to a decrease in blood flow to the heart and causes chest pain. High blood pressure, smoking, diabetes, lack of exercise, alcoholism, high cholesterol, and poor diet are major causes. CAD is more common in older adults, and men are more likely to develop it than women. Heart attack, shortness of breath, and chest pain are all symptoms of CAD. Conclusion: CAD is a very serious and rapidly growing disease in India. It is most common in the elderly, men, smokers, and people with a family history. To prevent this condition, a healthy diet and regular exercise are recommended.

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Nasir, K., E. Donnelly Michos, and J. A. Rumberger. "Coronary artery calcification and family history of premature coronary heart disease. Sibling history is more strongly associated than parental history." ACC Current Journal Review 14, no. 2 (February 2005): 31. http://dx.doi.org/10.1016/j.accreview.2004.12.127.

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Djousse, Luc, Yuling Hong, Donna K. Arnett, University of Minnesota, Steven C. Hunt, Gerardo Heiss, and R. Curtis Ellison. "Birth weight and cardiovascular disease: The NHLBI Family Heart Study." Circulation 103, suppl_1 (March 2001): 1363. http://dx.doi.org/10.1161/circ.103.suppl_1.9998-66.

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P66 Epidemiologic studies have been inconsistent on the association between low birth weight and increased risk of cardiovascular disease (CVD). Little is known about potential mechanisms by which determinants of low birth weight affect CVD. We used data collected on 2,540 participants of the NHLBI Family Heart Study to assess the relation of birth weight to CVD, hypertension, and diabetes mellitus. Birth weight was obtained by self report. CVD was defined as positive history of myocardial infarction, coronary angioplasty, coronary bypass surgery, congestive heart failure, or stroke. Hypertension was defined as systolic blood pressure of at least 140 mm Hg, or diastolic pressure of at least 90 mm Hg, or use of antihypertensive medication. Diabetes mellitus was ascertained by medical history and pathologic blood sugar levels. We used generalized estimating equations to assess the prevalence odds ratios, adjusting for age, risk group, anthropometric, metabolic, and lifestyle factors. Prevalence odds ratios (95% CI) are presented below. Low birth weight was associated with increased prevalence odds of CVD compared with normal and large babies (p=0.0012). The trend was suggestive of increased prevalence odds of hypertension (p=0.05) and diabetes mellitus (p=0.0012) from the highest to the lowest category of birth weight. In conclusion, low birth weight was associated with increased prevalence odds ratio of CVD, diabetes mellitus, and hypertension. Low birth weight may add to other traditional risk factors in identifying subjects at low and high risk of CVD. Table 1.

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Hattu, Diana A. M., Pius Weraman, and Conrad L. H. Folamauk. "Hubungan Merokok dengan Penyakit Jantung Koroner di RSUD Prof. DR. W. Z. Johannes Kupang." Timorese Journal of Public Health 1, no. 4 (December 12, 2019): 157–63. http://dx.doi.org/10.35508/tjph.v1i4.2143.

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A coronary heart disease is a disease which occurs because of the damage of blood vessel walls due to several factors of risk, such as free radical substances contained in cigarettes and pollution, cholesterol, hypertension, and smoking habits. The purpose of this study is to determine the correlation of smoking with coronary heart diseases. The research is an analytic survey with case control design. The population of this study were people who smoke that suffer from coronary heart diseases and those who do not suffer from the diseases. The sampling technique used in this study was simple random sampling, which consisted of 86 respondents. The respondents were divided into case and control sample respondents with similar number of people (43) respectively. The statistical test used was contingency coefficient test (c). The research shows that there was no correlation between jobs and family members health history towards the coronary heart diseases(p>0.05), whereas a correlation was found between the type of cigarettes, smoking intensity, sexes, duration of smoking, and the early age when smoking began with coronary heart diseases (p˂0.05).

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Andriastuti, Murti, Sudigdo Sastroasmoro, and Agus Firmansyah. "Risk factors of coronary heart disease in children and young adults with parental history of premature coronary heart disease." Paediatrica Indonesiana 43, no. 2 (October 10, 2016): 51. http://dx.doi.org/10.14238/pi43.2.2003.51-8.

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Background Morbidity and mortality of coronary heart disease(CHD) are recently increasing. This is related to changes in lifestyle,such as lack of activity and high consumption of fatty diet. Themain cause of CHD is atherosclerosis. The development of ath-erosclerosis takes a long time, is asymptomatic, and might beginin childhood. The important risk factors that have roles in increas-ing the likelihood of atherosclerosis are family history of prematureCHD, hypertension, hyperlipidemia, obesity, smoking and irregu-lar activity.Objective The aim of this study was to find out the prevalence ofCHD risk factors in children and young adults who had parentalhistory of premature CHD.Methods This was a descriptive cross sectional study conductedon offspring of premature CHD patients who were admitted in theintensive cardiology care unit (ICCU) of Cipto MangunkusumoHospital between January 1999 to December 2001 and of prema-ture CHD patients who visited the Cardiology Clinic of the Depart-ment of Internal Medicine, Cipto Mangunkusumo Hospital duringMarch and April 2002. Subjects were aged 12 to 25 year-old.Results Among the subjects, 40% had hyperlipidemia, 8% hadhypertension, 11% were obese, 21% were active smokers, 41%were passive smokers, and 73% had irregular activity. Ninety-sevenpercents subjects had more than 1 risk factors.Conclusions The prevalence of hyperlipidemia, hypertension,obesity, passive smoker, active smoker and irregular activity inchildren and young adults with parental history of premature CHDin this study were higher than those in the normal population.Most had more than 1 risk factor, increasing the likelihood of CHD.A screening test should be performed on children with parentalhistory of premature CHD so that early preventive measures mightbe done to minimize the risk factors

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Behbudova, J. A. "INFLUENCE OF THE FACTOR FAMILY HISTORY ON THE RISK OF CORONARY HEART DISEASE." Eurasian heart journal, no. 1 (March 30, 2015): 18–22. http://dx.doi.org/10.38109/2225-1685-2015-1-18-22.

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For the purpose of investigating the correlation between incidence of myocardial infarction (MI) and family history of cardiovascular disease (CVD), the study of prevalence of CVD among the first-degree relatives of patients with a known MI episode has been conducted. Medico-genetic files of two groups of patients with evidence of MI on ECG at rest have been analyzed: first group comprising patients who had an episode of MI before 55 years of age and second group comprising patients who had an episode after 55 years of age. A fairly high percentage of CVD in families of majority of patients with a known episode of MI has been noticed. However, this study has demonstrated a relatively low prevalence of coronary artery disease (CAD) and MI, including MI episodes before the age of 55, among fathers of patients from both groups. Moreover, occurrence of CAD and MI in fathers of the subgroup of patients who suffered from MI before 50 years of age of these patients practically showed no difference from that of subgroups of patients with episodes of MI at an older age. A fairly large amount of patients in both groups had no history of CAD in their families (41,5% and 41,2%, respectively). Considering the results mentioned above, there is no evidence to suggest that a predisposition of patients to MI episode at a younger age increases with existence of this disease in fathers of such patients. Results of our study suggest that increased presence of CVD and risk factors in first-degree relatives increases the probability of MI CAD in most patients of both groups regardless of the age of development of an MI episode.

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Simovic, Stefan, Goran Davidovic, Jinsung Yoon, Sasko Kedev, Marija Zdravkovic, Zorana Vasiljevic, Davor Milicic, et al. "IS A FAMILY HISTORY OF CORONARY HEART DISEASE AN INDEPENDENT CARDIOVASCULAR RISK FACTOR?" Journal of the American College of Cardiology 79, no. 9 (March 2022): 1073. http://dx.doi.org/10.1016/s0735-1097(22)02064-2.

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Goldbourt, U. "RE:"FAMILY HISTORY OF CORONARY HEART DISEASE: IS IT AN INDEPENDENT RISK FACTOR?"." American Journal of Epidemiology 126, no. 2 (August 1, 1987): 359–60. http://dx.doi.org/10.1093/aje/126.2.359.

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Burke, Lora E. "Primary Prevention in Patients With a Strong Family History of Coronary Heart Disease." Journal of Cardiovascular Nursing 18, no. 2 (April 2003): 139–43. http://dx.doi.org/10.1097/00005082-200304000-00010.

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Hasanaj, Q., B. J. Wilson, J. Little, Z. Montazeri, and J. C. Carroll. "Family History: Impact on Coronary Heart Disease Risk Assessment beyond Guideline-Defined Factors." Public Health Genomics 16, no. 5 (2013): 208–14. http://dx.doi.org/10.1159/000353460.

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Magnussen, Costan G., Terence Dwyer, and Alison Venn. "Family history of premature coronary heart disease, child cardio-metabolic risk factors and left ventricular mass." Cardiology in the Young 24, no. 5 (October 10, 2013): 938–40. http://dx.doi.org/10.1017/s1047951113001571.

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AbstractIn a prospective cohort of 181 individuals followed up since childhood – when aged 9, 12 and 15 years – patients with a family history of premature coronary heart disease (n=18) had higher left ventricular mass index in adulthood – at mean age of 31 years – compared with those without (mean±standard error 39.1±1.9 versus 34.6±0.7 g/m2.7, p=0.04). The correlation between adult left ventricular mass index and child triglycerides (r=0.66, p=0.04 versus r=−0.03, p=0.75; pdiff=0.02) and diastolic blood pressure (r=0.65, p=0.02 versus r=0.16, p=0.07; pdiff=0.05) was stronger among those with a family history of coronary heart disease than in those without. Although preliminary, these data suggest that the higher left ventricular mass index among adults with a family history might be explained by their increased susceptibility to child cardio-metabolic risk factors.

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Hawe, E., P. J. Talmud, G. J. Miller, and S. E. Humphries. "Family History is a Coronary Heart Disease Risk Factor in the Second Northwick Park Heart Study." Annals of Human Genetics 67, no. 2 (March 2003): 97–106. http://dx.doi.org/10.1046/j.1469-1809.2003.00017.x.

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French, David P., Theresa M. Marteau, Victoria Senior, and John Weinman. "Perceptions of Multiple Risk Factors for Heart Attacks." Psychological Reports 87, no. 2 (October 2000): 681–87. http://dx.doi.org/10.2466/pr0.2000.87.2.681.

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There is increasing evidence that in some diseases such as coronary heart disease, risk factors may interact synergistically, resulting in greater likelihood of disease than would be produced by the sum of the risk factors. These two studies aimed to examine the extent to which 210 undergraduate students and 28 heart attack patients perceive risk factors to combine synergistically. Respondents read one of four vignettes, describing information about risk factors (with high and low smoking and family history), and estimated a man's likelihood of a heart attack in a hypothetical case. In both studies an interaction was found with either a family history or smoking eliciting ratings of high likelihood of heart attack, providing no evidence of synergistic models. This finding may reflect respondents' beliefs or the insensitivity of this paper-and-pencil method in detecting synergistic effects.

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