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Books on the topic 'Family history of coronary heart disease'

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Published: 9 March 2023
Last updated: 10 March 2023

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1

Preventing coronary heart disease: Prospects, policies, and politics. London: Routledge, 1991.

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2

Doina, Velican, ed. Natural history of coronary atherosclerosis. Boca Raton, Fla: CRC Press, 1989.

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3

Heart disease. Santa Barbara, Calif: Greenwood, an imprint of ABC-CLIO, 2012.

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4

Stendardo, Stef. Tertiary prevention in diabetes, CAD and stroke: A case-based approach. Leawood, KS: American Academy of Family Physicians, 2003.

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5

Favaloro, René G. The challenging dream of heart surgery: From the Pampas to Cleveland. Boston: Little, Brown, 1994.

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6

Town, University of Cape, ed. The shoulders of giants: The development of cardiothoracic surgery. Cape Town: Universityof Cape Town, 1990.

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7

Otterbacher, John. Sailing Grace: A true story of death, life, and the sea. Grand Rapids, Mich: Samadhi Press, 2007.

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8

Favaloro, René G. De la pampa a los Estados Unidos. 4th ed. Buenos Aires: Editorial Sudamericana, 1992.

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9

Li, Jie Jack. Triumph of the heart: The story of statins. New York: Oxford University Press, 2009.

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10

Triumph of the heart: The story of statins. New York: Oxford University Press, 2008.

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11

J, Novick William, ed. Healing the heart of Croatia. New York: Paulist Press, 1998.

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12

W, Albarran John, and Tagney Jenny, eds. Chest pain: Advanced assessment and management skills. Oxford, UK: Blackwell Pub., 2007.

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13

Public health and the risk factor: A history of an uneven medical revolution. Rochester, NY: University of Rochester Press, 2003.

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14

Fox, Lisa L. Type A behavior pattern: The role of family environment and family history of coronary heart disease. 1998.

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15

Coronary Heart Disease (BMA Family Doctor). Dorling Kindersley Publishers Ltd, 1999.

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16

Leibowitz, J. O. History of Coronary Heart Disease. University of California Press, 2021.

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17

Leibowitz, J. O. History of Coronary Heart Disease. University of California Press, 2021.

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18

Understanding Coronary Heart Disease (Family Doctor Series). Family Doctor Publications, 1998.

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19

Oz, Mehmet, and Lori Mosca. Heart to Heart: A Personal Plan for Creating a Heart - Healthy Family. HCI, 2005.

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20

Calnan, Michael. Preventing Coronary Heart Disease: Prospects, Policies, and Politics. Taylor & Francis Group, 2002.

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21

Calnan, Michael. Preventing Coronary Heart Disease: Prospects, Policies and Politics. Taylor & Francis Group, 1991.

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22

Calnan, Michael. Preventing Coronary Heart Disease: Prospects, Policies, and Politics. Taylor & Francis Group, 2002.

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23

Calnan, Michael. Preventing Coronary Heart Disease: Prospects, Policies, and Politics. Taylor & Francis Group, 2002.

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24

Rothstein, William G. Coronary Heart Disease Pandemic in the Twentieth Century. Taylor & Francis Group, 2021.

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25

Living With a Heart Condition (Ward Lock Family Health Guides). Ward Lock Ltd, 1995.

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26

Saving the Heart: The Battle to Conquer Coronary Disease. Oxford University Press, USA, 2000.

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27

Counselling the Coronary Patient and Partner (Royal College of Nursing Research Series). Elsevier, 1990.

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28

Rothstein, William G. Coronary Heart Disease Pandemic in the Twentieth Century: Emergence and Decline in Advanced Countries. Taylor & Francis Group, 2017.

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29

Coronary Heart Disease Pandemic in the Twentieth Century: Emergence and Decline in Advanced Countries. Taylor & Francis Group, 2017.

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30

Echocardiography In Acute Coronary Syndrome Diagnosis Treatment And Prevention. Springer, 2009.

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31

Otterbacher, John. Sailing Grace. Samadhi Press, 2007.

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32

Otterbacher, John. Sailing Grace. Samadhi Press, 2007.

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33

Sailing Grace. Bright Sky Pr, 2004.

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34

The Challenging Dream of Heart Surgery: From the Pampas to Cleveland. Little Brown and Company, 1995.

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35

McGee, Charles T. Heart Frauds: Uncovering the Biggest Health Scam in History. Piccadilly Books, 2001.

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36

Topol, Eric J., Michael D. Eisner, and Cleveland Clinic. Cleveland Clinic Heart Book: The Definitive Guide for the Entire Family from the Nation's Leading Heart Center. Hyperion, 2000.

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37

Forfar, Colin. Diagnosis and investigation in suspected heart disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0087.

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The past 20 years have seen significant changes in both the demographics and natural history of many cardiovascular diseases. Important reductions in case-fatality rates (such as in acute coronary syndromes) have resulted from improved diagnostics and treatment options and better understanding of natural history. For others (such as infective endocarditis), improvements have been limited and disappointing. While advances in therapy and the scientific evidence underpinning treatments have been crucial, the importance of accurate diagnosis has remained a key element for progress. Many of the principles needed for diagnosis are constant: the pre-eminence of a focused, accurate history, complete physical examination, and timely and relevant investigation endures. It is essential to have a secure knowledge of the strengths and limitations of interpretation of a frequently bewildering array of tests. Progress in this field has been rapid; advances in ultrasound, scintigraphy, and cardiac magnetic resonance stand out at the interface between structure and function central to good patient care.
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38

Farmakis, Dimitrios, John Parissis, George Papingiotis, and Gerasimos Filippatos. Acute heart failure. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0051_update_001.

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Acute heart failure is defined as the rapid development or change of symptoms and signs of heart failure that requires urgent medical attention and usually hospitalization. Acute heart failure is the first reason for hospital admission in individuals aged 65 or more and accounts for nearly 70% of the total health care expenditure for heart failure. It is characterized by an adverse prognosis, with an in-hospital mortality rate of 4–7%, a 2–3-month post-discharge mortality of 7–11%, and a 2–3-month readmission rate of 25–30%. The majority of patients have a previous history of heart failure and present with normal or increased blood pressure, while about half of them have preserved left ventricular ejection fraction. A high prevalence of cardiovascular or non-cardiovascular comordid conditions is further observed, including coronary artery disease, arterial hypertension, atrial fibrillation, diabetes mellitus, renal dysfunction, chronic lung disease, and anaemia.
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39

Bhopal, Raj S. Epidemic of Cardiovascular Disease and Diabetes. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198833246.001.0001.

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Coronary heart disease (CHD) and stroke, collectively cardiovascular disease (CVD), are caused by narrowing and blockage of the arteries supplying the heart and brain, respectively. In type 2 diabetes (DM2) insulin is insufficient to maintain normal blood glucose. South Asians have high susceptibility to these diseases. Drawing upon the scientific literature and discussions with 22 internationally recognized scholars, this book focuses on causal explanations and their implications for prevention and research. Genetically based hypotheses are considered together with the developmental origins of health and disease (DOHAD) family of hypotheses. The book then considers how CHD, stroke, and DM2 are closely linked to rising affluence and the accompanying changes in life-expectancy and lifestyles. The established causal factors are shown to be insufficient, though necessary, parts of a convincing explanation for the excess of DM2 and CVD in South Asians. In identifying new explanations, this book emphasizes glycation of tissues, possibly leading to arterial stiffness and microcirculatory damage. In addition to endothelial pathways to atherosclerosis an external (adventitial) one is proposed, i.e. microcirculatory damage to the network of arterioles that nourish the coronary arteries. In addition to the ectopic fat in their liver and pancreas as the cause of beta cell dysfunction leading to DM2, additional ideas are proposed, i.e. microcirculatory damage. The high risk of CVD and DM2 in urbanizing South Asians is not inevitable, innate or genetic, or acquired in early life and programmed in a fixed way. Rather, exposure to risk factors in childhood, adolescence, and most particularly in adulthood is the key. The challenge to produce focused, low cost, effective actions, underpinned by clear, simple, and accurate explanations of the causes of the phenomenon is addressed.
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40

Public Health and the Risk Factor: A History of an Uneven Medical Revolution (Rochester Studies in Medical History). University of Rochester Press, 2008.

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41

Rothstein, William G. Public Health and the Risk Factor: A History of an Uneven Medical Revolution. University of Rochester Press, 2003.

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42

Farmakis, Dimitrios, John Parissis, and Gerasimos Filippatos. Acute heart failure: epidemiology, classification, and pathophysiology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0051.

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Acute heart failure is defined as the rapid development or change of symptoms and signs of heart failure that requires urgent medical attention and usually hospitalization. Acute heart failure is the first reason for hospital admission in individuals aged 65 or more and accounts for nearly 70% of the total health care expenditure for heart failure. It is characterized by an adverse prognosis, with an in-hospital mortality rate of 4-7%, a 2-3-month post-discharge mortality of 7-11%, and a 2-3-month readmission rate of 25-30%. The majority of patients have a previous history of heart failure and present with normal or increased blood pressure, while about half of them have a preserved left ventricular ejection fraction. A high prevalence of cardiovascular or non-cardiovascular comordid conditions is further observed, including coronary artery disease, arterial hypertension, atrial fibrillation, diabetes mellitus, renal dysfunction, chronic lung disease, and anaemia. Different classification systems have been proposed for acute heart failure, reflecting the clinical heterogeneity of the syndrome; the categorization to acutely decompensated chronic heart failure vs de novo acute heart failure and to hypertensive, normotensive, and hypotensive acute heart failure are among the most widely used and clinically relevant classifications. The pathophysiology of acute heart failure involves several pathogenetic mechanisms, including volume overload, pressure overload, myocardial loss, and restrictive filling, while several cardiovascular and non-cardiovascular causes or precipitating factors lead to acute heart failure through a single of these mechanisms or a combination of them. Regardless of the underlying mechanism, peripheral and/or pulmonary congestion is the hallmark of acute heart failure, resulting from fluid retention and/or fluid redistribution. Myocardial injury and renal dysfunction are also involved in the precipitation and progression of the syndrome.
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43

Grundy, Scott M. Cholesterol-Lowering Therapy. Taylor & Francis Group, 2019.

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44

Grundy, Scott M. Cholesterol-Lowering Therapy: Evaluation of Clinical Trial Evidence. Taylor & Francis Group, 1999.

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45

Grundy, Scott M. Cholesterol-Lowering Therapy: Evaluation of Clinical Trial Evidence. Taylor & Francis Group, 1999.

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46

Cholesterol-Lowering Therapy: Evaluation of Clinical Trial Evidence. Informa Healthcare, 2000.

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47

Schmid, Jean-Paul, and Hugo Saner. Ambulatory preventive care: outpatient clinics and primary care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0023.

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Cardiac rehabilitation (CR) services aim to restore the physical, psychosocial, and vocational status of cardiac patients. The role of these services has evolved due to the progress of interventional cardiology with its prompt and effective treatment of acute coronary syndromes. The focus has moved from the restoration of a patient’s health following an acute event towards a more pronounced long-term targeted secondary prevention intervention. As a consequence, CR services have also expanded their indication in order to include not only patients after myocardial infarction or surgery but also a variety of ’non-acuteʼ cardiovascular disease (CVD) states like stable coronary heart disease and peripheral obstructive artery disease as well as asymptomatic patients with no history of CVD but with a constellation of cardiovascular risk factors, especially metabolic syndrome or diabetes mellitus. This chapter provides a wide-ranging summary of the issues concerning outpatients and primary care.
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48

Schmid, Jean-Paul, Hugo Saner, Paul Dendale, and Ines Frederix. Ambulatory preventive care: outpatient clinics and primary care. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0023_update_001.

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Cardiac rehabilitation (CR) services aim to restore the physical, psychosocial, and vocational status of cardiac patients. The role of these services has evolved due to the progress of interventional cardiology with its prompt and effective treatment of acute coronary syndromes. The focus has moved from the restoration of a patient’s health following an acute event towards a more pronounced long-term targeted secondary prevention intervention. As a consequence, CR services have also expanded their indication in order to include not only patients after myocardial infarction or surgery but also a variety of ’non-acuteʼ cardiovascular disease (CVD) states like stable coronary heart disease and peripheral obstructive artery disease as well as asymptomatic patients with no history of CVD but with a constellation of cardiovascular risk factors, especially metabolic syndrome or diabetes mellitus. This chapter provides a wide-ranging summary of the issues concerning outpatients and primary care.
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49

AlJaroudi, Wael. Risk Assessment Before Noncardiac Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0014.

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Perioperative risk assessment is essential in screening patients before noncardiac surgery. Cardiovascular complications such as fatal and non-fatal myocardial infarction (MI), ventricular arrhythmia, pulmonary edema, and stroke are important in-hospital causes of morbidity and mortality intra and post-operatively. The optimal approach is to identify patients at increased risk so that appropriate testing and therapeutic interventions are undertaken a priori to minimize such risk. The initial preoperative evaluation includes identification of surgery-specific risk, patient exercise functional capacity and clinical risk profile. Patients with major predictors of events such as acute coronary syndromes, recent MI, unstable arrhythmia, and severe valvular disease warrant further management and optimization that often lead to delaying surgery. Those with three or more predictors (history of ischemic heart disease, compensated heart failure, diabetes, renal insufficiency, or history of cerebrovascular disease) undergoing high- risk surgery often require stress testing. Although data from randomized prospective trials are lacking, numerous studies have demonstrated the utility of myocardial perfusion imaging (MPI) for determination of perioperative cardiac risk. The goal of this chapter is to review the use of MPI for preoperative risk assessment and the recommendations from the current guidelines. The focus will be on short-term and long-term prognosis including special groups such as after coronary stenting and before vascular surgery, liver and renal transplantation.
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50

Elliott, Perry, and Alexandros Protonotarios. Arrhythmogenic right ventricular cardiomyopathy: management of symptoms and prevention of sudden cardiac death. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0361.

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Patients with arrhythmogenic right ventricular cardiomyopathy (ARVC) have arrhythmia-related symptoms or are identified during screening of an affected family. Heart failure symptoms occur late in the disease’s natural history. As strenuous exercise has been associated with disease acceleration and worsening of ventricular arrhythmias, lifestyle modification with restricted athletic activities is recommended upon disease diagnosis or even identification of mutation carrier status. An episode of an haemodynamically unstable, sustained ventricular tachycardia or ventricular fibrillation as well as severe systolic ventricular dysfunction constitute definitive indications for implantable cardioverter defibrillator (ICD) implantation, which should also be considered following tolerated sustained or non-sustained ventricular tachycardia episodes, syncope, or in the presence of moderate ventricular dysfunction. Antiarrhythmic medications are used as an adjunct to device therapy. Catheter ablation is recommended for incessant ventricular tachycardia or frequent appropriate ICD interventions despite maximal pharmacological therapy. Amiodarone alone or in combination with beta blockers is most effective for symptomatic ventricular arrhythmias. Beta blockers are considered for use in all patients with a definite diagnosis but evidence for their prognostic benefit is sparse. Heart failure symptoms are managed using standard protocols and heart transplantation is considered for severe ventricular dysfunction or much less commonly uncontrollable ventricular arrhythmias.
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