Relevant bibliographies by topics / Family history of coronary heart disease

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Family history of coronary heart disease'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Family history of coronary heart disease"

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Friedman, Gary D., and Arthur L. Kaltsky. "RE: FAMILY HISTORY OF CORONARY HEART DISEASE." American Journal of Epidemiology 126, no. 1 (July 1987): 159. http://dx.doi.org/10.1093/oxfordjournals.aje.a114655.

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Nasir, Khurram, Matthew J. Budoff, Nathan D. Wong, Maren Scheuner, David Herrington, Donna K. Arnett, Moyses Szklo, Philip Greenland, and Roger S. Blumenthal. "Family History of Premature Coronary Heart Disease and Coronary Artery Calcification." Circulation 116, no. 6 (August 7, 2007): 619–26. http://dx.doi.org/10.1161/circulationaha.107.688739.

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Nasir, Khurram, Erin Donnelly Michos, John A. Rumberger, Joel B. Braunstein, Wendy S. Post, Matthew J. Budoff, and Roger S. Blumenthal. "Coronary Artery Calcification and Family History of Premature Coronary Heart Disease." Circulation 110, no. 15 (October 12, 2004): 2150–56. http://dx.doi.org/10.1161/01.cir.0000144464.11080.14.

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O’Donnell, Christopher J. "Family History, Subclinical Atherosclerosis, and Coronary Heart Disease Risk." Circulation 110, no. 15 (October 12, 2004): 2074–76. http://dx.doi.org/10.1161/01.cir.0000145539.77021.ac.

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Crouch, Michael A., and Robert Gramling. "Family History of Coronary Heart Disease: Evidence-Based Applications." Primary Care: Clinics in Office Practice 32, no. 4 (December 2005): 995–1010. http://dx.doi.org/10.1016/j.pop.2005.09.008.

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Iqbal, Usman Javed, Majid Kaleem, and Muhammad Imran Hanif. "CORONARY ARTERY DISEASE." Professional Medical Journal 22, no. 05 (May 10, 2015): 532–35. http://dx.doi.org/10.29309/tpmj/2015.22.05.1261.

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Background: Development of heart failure is always secondary to presenceof risk factors like diabetes mellitus, hypertension, age, smoking and underlying coronaryartery disease. Objective: The objective of this study was to find the frequency of risk factorsand coronary artery involvement in patients of heart failure after myocardial infarction. StudyDesign: A non-randomized cross sectional study. Setting: Gulab Devi Chest Hospital. Period:Six months. Methods: 100 patients was done. Using non probability (purposive) samplingtechnique; all patients with ages between 20 to 80 years, irrespective of gender, diagnosedwith heart failure (with ejection fraction ≤ 40%) secondary to STEMI were included in thestudy. Patients that were not having heart failure secondary to STEMI and those that hadn’ttheir coronary angiography done were excluded from the study. The data were analyzed usingSPSS Version 20. Descriptive statistics was used to see analyze the data. Results: Mean ageof patients was 52.61±10.2years. There was an overall male predominance (81%). Commonrisk factors that we observed were smoking (65%), hypertension (62%), diabetes (58%) andpositive family history (38%). In this study 51% patients had triple vessels disease, 15% patientshad double vessels disease and 34% patients had single vessel disease. Most common lesionwas of LAD following LCX, RCA and LMS. Conclusion: Our study conclude that coronaryartery disease is the main causative factor for the development of heart failure in patients ofmyocardial infarction and even a single vessel disease can lead to heart failure with severesystolic dysfunction. Most common associated risk factor was smoking, hypertension being thesecond most common following diabetes and positive family history.
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Nadir, M. A., and A. D. Struthers. "Family history of premature coronary heart disease and risk prediction." Heart 97, no. 8 (March 1, 2011): 684. http://dx.doi.org/10.1136/hrt.2011.222265.

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Wrigley, Martha, and Judith Lathlean. "Family history of premature coronary heart disease: Discussing the evidence." British Journal of Cardiac Nursing 4, no. 12 (December 2009): 569–74. http://dx.doi.org/10.12968/bjca.2009.4.12.45680.

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Wrigley, Martha, and Judith Lathlean. "Family history of premature coronary heart disease: Exploring the experience." British Journal of Cardiac Nursing 5, no. 1 (January 2010): 40–45. http://dx.doi.org/10.12968/bjca.2010.5.1.46032.

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ZOLER, MITCHEL L. "Family History of Heart Disease Boosts Coronary Risk in Sisters." Internal Medicine News 38, no. 3 (February 2005): 64. http://dx.doi.org/10.1016/s1097-8690(05)71512-2.

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Dissertations / Theses on the topic "Family history of coronary heart disease"

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Wrigley, Martha J. "Engaging families with a premature family history of heart disease : a primary prevention study for coronary heart disease." Thesis, University of Southampton, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485524.

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This thesis focuses upon a preventative approach for people with a familial history of premature coronary heart disease. The research study had two aims; firstIy to understand the experience of individuals when their parent or sibling had been diagnosed with heart disease; secondly to develop and evaluate a primary prevention health promotion programme for these people. The thesis provides unique insight into their personal experiences of living with tIris familial diagnosis and gives details of how this population can be identified and involved successfully in a health promotion programme. The study was conducted in a district general hospital; 28 people were recruited of whom 20 participants completed the two year study. Narrative interviews were conducted at the beginning and end of the study, providing insight into people's personal experience. Salutogenesis is the theoretical framework in which the narratives are discussed; from these health resources are identified which can enhance people's progress and maintenance for a healthy lifestyle. The health promotion programme has established, developed and evaluated a nurseled and doctor supported primary pjevention strategy. Education and support was provided in the programme for individuals and families. The participants' physical and behavioural changes were reviewed six monthly, for two years. People are aware of risk factors associated with heart disease, but still seek professional support and advice in relation to their own lifestyle and behaviour. Individual lifestyle changes were achieved by most participants, which translated into significant findings for blood pressure and alcohol consumption; positive changes were seen in physical activity, smoking, diet and psychosocial stress. There were no improvements in lipid profiles. The need for a preventatiye approach in health care, which includes primordial and primary prevention for heart disease, is discussed. The issues in this thesis are reflective upon current government focus to develop preventative health services which actively engage with people as integral to this process. The thesis discusses coronary heart disease, the roles of prevention and health promotion, and identifies health resources for people at high-risk of future cardiovascular problems.
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Ashton, William David. "Coronary risk factors in women in the United Kingdom." Thesis, University of Salford, 1997. http://usir.salford.ac.uk/42977/.

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Coronary heart disease (CHD) has traditionally been regarded as a male disease and, because of this, the magnitude of the problem in women is often overlooked. Yet, cardiovascular disease (CVD) and CHD in particular, remains, next to cancer, the leading cause of morbidity and mortality in women. The longstanding emphasis on the problem of CHD in men, has resulted in a widespread tendency to minimise the incidence and severity of the disease in women. Moreover, most epidemiologic studies examining morbidity and mortality from CHD have focused largely on men, producing a significant gender gap in the research. The lack of information on CHD risk factors and prevention of heart disease in women in Britain is of particular concern, given that British women have one of the highest rates of coronary disease in the world. The Marks and Spencer Coronary Risk Factor Study (MSCRFS) is a cross-sectional and prospective study of CHD risk factors in female employees of the Marks and Spencer retail organisation. The present study is confined to an analysis of cross-sectional data from 14,077 women screened between June 1988 and July 1991. The prevalence and distribution of a variety of lipid, lipoprotein, biochemical, anthropometric and lifestyle-related CHD risk factors among women in the United Kingdom is described, together with their key interrelationships. In addition, the metabolic impact of exogenous hormones, specifically oral contraceptives and postmenopausal hormone replacement, is described. This study - the largest of its kind in the UK - provides reference ranges for a wide range of CHD risk factors in women in the UK, and gives a unique insight into the impact of a variety of lifestyle-related factors on CHD risk. There is an enormous potential for reducing the very high risk of CHD among women in the UK, which needs to be addressed. Based on these data, health strategies designed to reduce morbidity and mortality from CHD can be planned and implemented more effectively.
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Hasanaj, Qendresa. "Family History in the Assessment of Risk for Common Complex Diseases: Current State of Evidence." Thesis, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/20682.

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Family history (FH) is a risk factor for many diseases. Disease guidelines often include family history as important in assessing chronic disease risks, but the empirical evidence base to inform the routine use of family history in primary care in practice appears largely lacking. An environmental scan of how family history is represented in prevention guidelines for five conditions showed that, while family history is often included in guidelines, there is variation in the definition used, recommendation given and evidence cited. A dataset on cardiovascular health in women was analyzed to examine whether family history offers useful discrimination value above standard risk factors. Regression results showed that family history is an independent risk predictor for coronary heart disease which improves discrimination beyond classical clinical factors. However, the absolute amount of discriminatory ability alone or with other factors is moderate at best, raising issues regarding clinical utility.
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O'Neil, John Nolen. "The association of family history of coronary heart disease, sex, psychosocial vulnerability, and hostility among college students /." The Ohio State University, 1999. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488192960170939.

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VERONESI, GIOVANNI. "Develoment, validation and clinical utility of a long-term cardiovascular disease risk prediction model in the italian population." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2014. http://hdl.handle.net/10281/50251.

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We illustrate the development of a 20-year prediction model of first major coronary or ischemic stroke event in a Northern Italian population of men and women aged 35 to 69 years at baseline. The model included age, blood lipids, systolic blood pressure, anti-hypertensive treatment, smoking habits and diabetes. The discrimination ability of the model was high as 0.736 in men and 0.801 in women. The model has been internally and externally validated using a different cohort study of subjects enrolled in Latina. Based on the external validation analysis, the risk score seems to be appropriate for long-term risk prediction in Italy and, more generally, in low-incidence populations. The clinical utility of the risk score in stratifying subjects in risk categories has been evaluated considering two strategies for the identification of “high-risk” subjects with contrasting public health goals, either to decrease the fraction of missed events or to decrease un-necessary treatment. These can be implemented by choosing threshold values for the predicted risk driven by either sensitivity or by specificity, respectively. The risk stratification based on 20-year absolute predicted risk had a higher clinical utility than any stratification based on the number of risk factors. Finally, we discussed from the statistical perspective the concept of “improvement” in risk prediction through the paradigmatic analysis of two indicators of disease heritability and social status, i.e. family history of coronary heart disease and educational level, added to the initial model. A new SAS package, Risk Estimation in Survival Analysis using SAS 9.2 [reSAS], detailed in the appendix, has been specifically developed from the author.
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Taraboanta, Catalin. "Impact of family history of premature coronary disease on carotid ultrasound and coronary calcium findings." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/721.

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First degree relatives (FDRs) of subjects with early onset of coronary heart disease (CHD) have higher risk of developing cardiovascular disease. We verified early CHD by angiography in the index patients and extensively phenotyped their FDRs to investigate the relationship of traditional and non-traditional cardiovascular risk factors to carotid ultrasound and coronary calcium scoring findings. B-mode carotid ultrasound was used to assess the combined intima-media thickness and plaque burden in 111 FDRs. The biochemical and anthropometrical characteristics of the FDRs were compared with those of healthy controls matched for sex, age, ethnicity and BMI. Odds ratios indicate that FDRs are more likely to have positive carotid ultrasound findings compared to controls; 2.23 (95% CI 1.14 – 4.37) for intima-media thickness and 2.3 (95% CI 1.22 - 4.35) for average total thickness. In multivariate analysis positive carotid ultrasound findings were higher in FDRs independent of age, gender, total cholesterol over HDL-c ratio, systolic blood pressure and smoking but not homocysteine which had higher values in FDRs compared to controls. In conclusion FDRs of patients with angiographically confirmed CHD have higher burden of subclinical atherosclerosis even when considered in the context of traditional risk factors. Coronary artery calcium scoring (CAC), assessed by 64-slice multi-detector computed tomography (MDCT), was used to assess burden of subclinical atherosclerosis in 57 FDRs compared to controls. FDRs have a two-fold increase in risk of having CAC positive findings; odds ratios for the 75th percentile was 1.96 (95%CI 1.04 – 3.67, p<0.05) while for the 90th percentile odds ratio was 2.59 (95% 1.232 – 5.473, p<0.05). In summary, the risk of significant CAC findings, measured by 64-slice MDCT, is two-fold higher in FDRs than controls. These findings correlate highly with carotid ultrasound findings in the same cohort. Different thresholds for CAC may be appropriate when assessing male versus female FDRs. Together increased carotid ultrasound findings and CAC scoring results in FDRs of patients with validated early onset of CHD suggest these imaging techniques as potentially useful tools in cardiovascular risk assessment that will go above and beyond the current diagnostic algorithms.
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Gunnell, David J. "Food, death and income - a follow-up study based on a survey of family diet and health in pre-war Britain (1937-9)." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336848.

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Ceballos, Mario E. "The effect of religion on the healing of coronary artery disease/hypertensive out patients in a family practice setting." Theological Research Exchange Network (TREN), 1999. http://www.tren.com.

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Mitchell, Jayne. "The association of selected coronary heart disease risk factor variables between family members, with specific reference to physical activity." Thesis, University of Exeter, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.292377.

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Cheng, Xi. "Prevalence, profile, predictors, and natural history of aspirin resistance measured by the ultegra rapid platelet function assay-asa in patients with coronary artery disease." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B33708708.

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Books on the topic "Family history of coronary heart disease"

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Preventing coronary heart disease: Prospects, policies, and politics. London: Routledge, 1991.

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Doina, Velican, ed. Natural history of coronary atherosclerosis. Boca Raton, Fla: CRC Press, 1989.

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Heart disease. Santa Barbara, Calif: Greenwood, an imprint of ABC-CLIO, 2012.

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Stendardo, Stef. Tertiary prevention in diabetes, CAD and stroke: A case-based approach. Leawood, KS: American Academy of Family Physicians, 2003.

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Favaloro, René G. The challenging dream of heart surgery: From the Pampas to Cleveland. Boston: Little, Brown, 1994.

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Town, University of Cape, ed. The shoulders of giants: The development of cardiothoracic surgery. Cape Town: Universityof Cape Town, 1990.

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Otterbacher, John. Sailing Grace: A true story of death, life, and the sea. Grand Rapids, Mich: Samadhi Press, 2007.

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Favaloro, René G. De la pampa a los Estados Unidos. 4th ed. Buenos Aires: Editorial Sudamericana, 1992.

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Li, Jie Jack. Triumph of the heart: The story of statins. New York: Oxford University Press, 2009.

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Triumph of the heart: The story of statins. New York: Oxford University Press, 2008.

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Book chapters on the topic "Family history of coronary heart disease"

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Genest, Jacques. "Homocysteine and Family History of Coronary Artery Disease." In Homocysteine and Vascular Disease, 203–16. Dordrecht: Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-017-1789-2_12.

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Schlant, R. C., Sandra Forman, J. Stamler, and P. L. Canner. "The Natural History of Coronary Heart Disease: Prognostic Factors After Recovery from Myocardial Infarction in 2789 Men." In Primary and Secondary Prevention of Coronary Heart Disease, 74–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70296-9_7.

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Burg, Matthew M. "Social support and the impact of coronary heart disease on the family." In Psychological treatment of cardiac patients., 121–36. Washington: American Psychological Association, 2018. http://dx.doi.org/10.1037/0000070-009.

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Pandey, Shivda, and Khurram Nasir. "Family History: An Index of Genetic and Environmental Predisposition to Coronary Artery Disease." In Asymptomatic Atherosclerosis, 169–78. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-179-0_12.

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Brown, Jonathan D., and Jorge Plutzky. "Cardiovascular Disease Prevention." In The Brigham Intensive Review of Internal Medicine, 860–69. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199358274.003.0083.

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This chapter focuses on how cardiovascular risk factors contribute to the initiation and progression of coronary heart disease (CHD). Cardiovascular risk factors are currently grouped into traditional and nontraditional types. Traditional risk factors include hyperlipidemia (or dyslipidemia), tobacco use, hypertension, diabetes mellitus, age, male gender, and family history, all of which have been linked to CHD, an association substantiated through multiple large prospective population studies. The categories can be further subdivided into modifiable and nonmodifiable factors: with dyslipidemia, tobacco use, hypertension, and diabetes comprising the former. In spite of their undeniable diagnostic and prognostic value, a portion of the population lacking these traditional risk factors remains at significant residual risk for CHD.
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Ernande, Laura. "Hypertension and diabetes." In ESC CardioMed, edited by Frank Flachskampf, 445–47. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0090.

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Both hypertension and diabetes mellitus (DM) are risk factors for coronary artery disease, resulting in a high risk of heart failure. They also directly impact the heart, leading to hypertensive heart disease and diabetic cardiomyopathy and also potentially causing heart failure. The hypertensive heart is characterized by left ventricular hypertrophy and frequently left atrial dilation. DM is also associated with an increased left ventricular mass. Left ventricular hypertrophy has an important prognostic value both in hypertensive and diabetic patients and may regress with antihypertensive medications. The differential diagnosis between hypertensive heart disease and hypertrophic cardiomyopathy can be sometimes challenging and requires a careful evaluation of the echocardiogram but also of the family history, electrocardiographic abnormalities, or the location of late gadolinium enhancement on cardiovascular magnetic resonance imaging. Hypertension and DM also influence ageing-related left ventricular remodelling. Diastolic dysfunction is frequent both in hypertensive and diabetic patients and is predictive of cardiac events and heart failure. However, diastolic dysfunction in diabetic patients seems to be more associated with age and co-morbidities such as obesity and hypertension than with DM itself. The presence of diastolic dysfunction predicts cardiac events and heart failure in those patients. Both hypertension and DM are risk factors for heart failure with preserved but also with reduced and mid-range ejection fraction. Therefore, left ventricular ejection fraction is a major parameter to evaluate in those patients. Myocardial strain is decreased both in hypertensive and in diabetic patients with normal left ventricular ejection fraction allowing the detection of subclinical systolic alteration and providing incremental prognostic value.
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Farne, Hugo, Edward Norris-Cervetto, and James Warbrick-Smith. "Chest pain." In Oxford Cases in Medicine and Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/oso/9780198716228.003.0015.

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A good way to come up with a list of causes is to visualize the anatomy of the affected area and think of what could go wrong. Thus, in chest pain, there may be pathology of the heart, aorta, lungs, pulmonary vessels, oesophagus, stomach, thoracic nerves, thoracic muscles, or ribs. The main causes of acute chest pain in an individual aged over 60 include are listed in Figure 9.1. A younger patient is less likely to be suffering from diseases of old age, such as: • Acute coronary syndrome • Stable angina • Myopericarditis (usually post-infarction) • Thoracic aortic dissection • Thoracic aortic aneurysm A younger female patient on the combined oral contraceptive pill is more likely to be suffering from: • PE (the combined oral contraceptive pill is thrombogenic) • Pneumothorax (especially if tall and thin) • Cocaine-induced coronary spasm (still rare, but particularly unusual in older people). The following diagnoses require immediate management and should be kept in mind: • Acute coronary syndrome (unstable angina, or myocardial infarction (MI)) • Aortic dissection • Pneumothorax • PE • Boerhaave’s perforation The key features of each are listed below. 1 Features of acute coronary syndrome ■ History of sudden-onset, central, crushing chest pain radiating to either/both arms, neck or jaw, usually lasting a few minutes to half an hour (longer if there is ongoing infarction). Have a higher index of suspicion in those with a previous history of angina on exertion or MI and/or cardiovascular risk factors (smoking, hypertension, hypercholesterolaemia, diabetes mellitus, family history). ■ Signs of hypercholesterolaemia: cholesterol deposits in small skin lumps on the back of the hand or bony prominences like elbows (xanthomata), in creamy spots around the eyelids (xanthelasma), or a creamy ring around the cornea (arcus). Note that arcus is a normal finding in older people. ■ Signs of peripheral (atherosclerotic) vascular disease: weak pulses, peripheral cyanosis, cool peripheries, atrophic skin, ulcers, bruits on auscultation of carotids. ■ Signs of brady- or tachyarrhythmia. An arrhythmia is relevant for two reasons.
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Rivera, Kathya, Kenneth Cusi, and Catherine Edwards. "Case 35: Cosecreting Adrenal Tumor Causing Severe Insulin Resistance." In Diabetes Case Studies: Real Problems, Practical Solutions, 124–29. American Diabetes Association, 2015. http://dx.doi.org/10.2337/9781580405713.35.

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A 38-year-old Caucasian woman was referred to our care with a presumed diagnosis of primary hyperaldosteronism and an associated large adrenal mass. She was found by her primary care physician (PCP) to have severe hypokalemia on routine lab work although she was completely asymptomatic. Past medical history included osteoporosis, hyperlipidemia, meningioma, seizure disorder, and hypothyroidism. Family history included type 2 diabetes (T2D), hypertension, dyslipidemia, and coronary heart disease. Medications at the time of endocrine evaluation included KCl 40 mEq p.o. q.i.d., diltiazem 60 mg p.o. q.i.d., losartan 25 mg p.o. daily, spirinolactone 50 mg p.o. daily, insulin glargine 45 units subcutaneously b.i.d., insulin aspart 26 units subcutaneously before meals, and a correction scale and pioglitazone 15 mg daily. On examination, blood pressure was 177/90 mmHg and pulse was 112 bpm. She was 4′ 9″ tall, weighed 36 kg and had a BMI of 17.2 kg/m2. Physical examination revealed a young female with a round face, bilateral clavicular fullness, and proximal muscle wasting of the extremities but no acanthosis nigricans, facial plethora, acne, bruises, hirsutism, central obesity, or purple striae.
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"Braunwald’s Heart Disease Family of Books." In Chronic Coronary Artery Disease, xix—xxii. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-42880-4.09001-4.

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Pottle, Alison. "Understanding Coronary Heart Disease." In Adult Nursing Practice. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199697410.003.0016.

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The aim of this chapter is to provide nurses with the knowledge to be able to assess, manage, and care for people with coronary heart disease (CHD) in an evidence-based and person-centred way. The chapter will provide a comprehensive overview of the causes, risk factors, and impact of CHD. In guiding you through patient assessment, the differences between acute coronary syndromes (ACS) and angina are established before exploring best practice to deliver care, as well as to prevent or to minimize further ill-health. Nursing assessments and priorities are highlighted throughout, and the nursing management of the symptoms and common health problems associated with coronary heart disease can be found in Chapters 15, 22, 24, and 25, respectively. CHD is defined as the failure of the coronary arteries to deliver adequate oxygen for myocardial work. It is almost always caused by atherosclerosis—a gradual build-up of fatty plaques within the artery wall that reduces blood flow. This failure to meet metabolic demands results in a range of clinical conditions sharing common pathological process (Baxendale, 1992), including ACS and angina. Chest pain is the symptom that informs clinical decision-making. It is classified based on history-taking and investigations such as the electrocardiogram (ECG). Angina was first described by Heberden in 1772 as a ‘painful and disagreeable sensation in the breast, which seems as if it would take their life away if it were to increase or continue.’ (cited by Fox et al., 2006). Stable angina is described as a clinical syndrome that is characterized by discomfort in the chest, jaw, shoulder, back, and arms, typically elicited by exertional emotional stress and relieved by rest or nitroglycerine (Fox et al., 2006). ACS is an umbrella term for several clinical presentations, including unstable angina, non-ST elevation myocardial infarction (NSTEMI), and ST elevation myocardial infarction (STEMI). The European Society of Cardiology defines ACS as ‘. . . a life threatening manifestation of atherosclerosis . . . caused by a ruptured atherosclerotic plaque . . . causing sudden complete or critical reduction in blood flow’ (Bassand et al., 2007).
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Conference papers on the topic "Family history of coronary heart disease"

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Ranganathan, D., C. O'Carroll-Loliat, R. Kinsella, A. Reynolds, and D. Ward. "64 Screening of patients with a family history of premature coronary artery disease." In Irish Cardiac Society Annual Scientific Meeting & AGM, Thursday October 5th – Saturday October 7th 2017, Millennium Forum, Derry∼Londonderry, Northern Ireland. BMJ Publishing Group Ltd and British Cardiovascular Society, 2017. http://dx.doi.org/10.1136/heartjnl-2017-ics17.63.

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Francis, R., D. Kawanishi, T. Baruch, P. Mahrer, S. Rahimtoola, and D. Feinstein. "IMPAIRED FIBRINOLYSIS IN CORONARY ARTERY DISEASE (CAD): INFLUENCE OF AGE AND FAMILY HISTORY (FH)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643028.

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We assessed fibrinolysis in 99 subjects with CAD documented by angiography. Tissue-type plasminogen activator (tpa) antigen and activity (act) were measured in plasma by ELISA and solid-phase bioimmunoassay, respectively, pre and post venous occlusion (VO) of the arm for 10 minutes. Pre-VO tpa inhibitor (PAI) was assayed by a modification of the method of Juhan-Vague (Thromb Pes 1984). Mean PAI was significantly higher, and mean post-VO tpa act significantly lower, in all CAD subjects than in 28 normals (no CAD by angiography). Mean increase in tpa antigen with VO (releasable tpa) was significantly lower than that of normals only in CAD subjects with age of onset <45. Mean releasable tpa and tpa act post-VO were lower in CAD subjects with age of onset <45 than in older subjects, lower in those with positive than negative FH of CAD in parents or siblings, and lowest in those with both FH and age of onset <45. In contrast, PAI was higher in older subjects. These data suggest that genetic factors may contribute to impaired fibrinolysis in CAD.
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Aznar, J., A. Estelles, G. Tormo, F. Espafia, and V. Torino. "FIBRINOLYTIC ALTERATIONS AS RISK FACTOR IN PATIENTS WITH CORONARY HEART DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643781.

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It has been reported that young survivors of myocardial infarction (MI) have elevated plasminogen activator inhibitor (PAI) levels. We have studied several fibrinolytic parameters (euglobulin lysis time, fibrin-plate lysis, tissue plasminogen activator (t-PA) antigen, t-PA activity, PAI activity, plasminogen, az antiplasmin and FDP/fdp in 55 patients with coronary heart disease (CHD), before and after an exercise test. The patients were classified in 4 groups:A) Patients with unstable angina (n=5); B) Patients with stable angina and previous history of MI (n=13); C) Patients with stable angina without previous history of MI (n=ll) and D) Patients with MI about 3 weeks before this study (n=26). All the groups were similars in age and life habits. Patients suffering from dislipemia and diabetes were excluded from the study. In basal conditions, PAI activity (U/ml) was high in the 4 patient groups (A:2.5±2.8; B:5.2 ± 4.9; C:2.8 ± 2.6; D: 4.6 ± 4.6) ascompared to a group of 10 healthy volunteers (0.46±0.5). In all the clinical groups there were a large number of patients (about 60%) whose PA inhibitor level was > 2 U/ml. t-PA antigen (ng/ml) was slightly elevated in all patient groups (A: 12.4+±4.6; B:12.4±5.6; C:12.5+4.0; D: 13.3+4.3) in comparison with control group (10.1+2.9). The release of t-PA antigen after the exercise test did not differ significantly from one group to another. However, this release was < 3 ng/ml in about 50% of patients in all clinical groups, as compared to the control group, in which the release of t-PA antigen was higher than 3 ng/ml in all the subjects. After the exercise PAI activity remained high in the patient groups. The increased level of t-PA inhibitor activity founded in the patients was partially inhibited by antiserum against PA inhibitor-1 but not by antiserum against PA inhibitor-2. The formation of a complex of about 115,000 daltons between the increased plasma PA-Inhibitor and purified single t-PA was observed by a zymographic fibrin technique. These findings demonstrate that CHD patients have a fibrinolytic hypofunction caused basically by an increase in t-PA inhibitor. This increase in PAI activity is more evident in patient with a previous history of MI.
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Schmidt, Márcia, Karine Schmidt, Vitória Silva, Filipa Waihrich, and Alexandre Quadros. "PERCEIVED STRESS IN PATIENTS WITH CORONARY ARTERY DISEASE DURING THE COVID-19 PANDEMIC." In International Psychological Applications Conference and Trends. inScience Press, 2021. http://dx.doi.org/10.36315/2021inpact023.

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"Introduction: The COVID-19 pandemic has brought many routine changes to the entire Brazilian population. On mental health, individuals were observed to be increasingly susceptible to developing symptoms such as psychological stress, anxiety, and depression due to social restrictions. The psychosocial factors exerted a fundamental role in the etiology and evolution of cardiovascular diseases (CVDs), with stress being one of these factors. Objective: To evaluate the self-perceived stress on arterial coronary disease patients during the COVID-19 pandemic. Methods: Coronary artery disease patients undergoing percutaneous coronary intervention in the period between February and May 2020 at a reference hospital in cardiology were considered eligible. The patients were interviewed about their self-perceived stress through phone calls from August to December 2020. The clinical characteristics were obtained through the electronic record. The stress was evaluated using a Perceived Stress Scale – PSS-10. Those patients with a score higher than the PSS-10 average (16 points) were considered stressed. The patients were divided into groups with and without stress. The categorical variables were expressed through the frequency and percentage and analyzed by the chi-square test, and the continuous variables were described by average and standard deviation, then compared using a Student's t test. Results: A total of 501 patients were assessed for eligibility, and 200 were included. Forty-nine percent of the patients presented stress symptoms. The stressed patients were frequently younger and had a family history of premature coronary artery disease. The women were more often stressed than the men, even though both groups had a higher prevalence of stressed patients than non-stressed patients. Concerning the PSS-10 questions, the patients with stress were more frequently upset with something unexpected; they felt more often unable to control important matters in their lives, more nervous, and irritated than those without stress. They also referred to the greater difficulty they faced in controlling irritations in their lives. Finally, 50% of the stressed patients also felt very frequently that their problems had accumulated in such a manner that they could not solve them anymore, in comparison to 0.98% in the without-stress group. Conclusion: The patients with arterial coronary disease and self-perceived stress were younger and presented a family history of premature coronary disease. They had less control over their irritations and important issues in their lives, feeling overloaded and incapable of solving their problems compared to those without stress."
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Gani, Azhari, and Munifah Abdat. "Prevalence of Coronary Heart Disease with Risk Factors History of Hypertension and Diabetes Mellitus at the Regional Government Hospital dr. Zainoel Abidin in Aceh." In 2nd Aceh International Dental Meeting 2021 (AIDEM 2021). Paris, France: Atlantis Press, 2022. http://dx.doi.org/10.2991/ahsr.k.220302.002.

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Koringer, C., R. Jäger, K. Huber, and K. lechner. "LEVELS OF PLASMINOGEN ACTIVATOR INHIBITOR IN PATIENTS WITH ANGINA PECTORIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644453.

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Several groups have shown that fibrinolytic capacity is impaired in survivors of myocardial infarction, due to increased levels of the fast-acting plasminogen activator inhibitor (PAI). In order to study the behaviour of PAI in patients with coronary heart disease, 180 patients with angina pectoris were investigated. They were 148 males and 32 females, ages ranging from 29 to 70 years (52.8 ± 8.2, mean ± S.D.). A sex- and age- matched normal population served as a control (n=105, age-range 30 to 69 years, 52.4 ± 7.9). PAI was determined by a functional titration assay, and its activity expressed as arbitrary units (AU). PAI levels were significantly (p <0.005) higher in patients with angina (24.3 ± 10.3 AU/ml, range 10.1 to 112.0 AU/ml) than in normals (20.4 ± 4.6 AU/ml, range 10.5 to 31.6 AU/ml). PAI levels were unrelated to sex or age, in both the patient and the control groups. As expected, plasma triglyceride levels were correlated to PAI in patients (r=0.19, p<0.01) and in normals (r=0.20, p<0.05). Patients with a history of previous myocardial infarction (n=114) had similar PAI levels as patients without infarction (24.2 ± 11.1 AU/ml as compared to 24.4 ± 9.6 AU/ml). It is concluded that PAI levels are elevated in patients with coronary heart disease, whether myocardial infarction has taken place or not.
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Poniewierski, M., M. Barthels, and H. Poliwoda. "THE SAFETY AND EFFICACY OF A LOW MOLECULAR WEIGHT HEPARIN (FRAGMIN) IN THE PREVENTION OF DEEP VEIN THROMBOSIS IN MEDICAL PATIENTS: A RANDOMIZED DOUBLE-BLIND TRIAL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643224.

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The safety and efficacy of 2500 anti-Factor Xa U of a low molecular weight heparin (Kabi 2165, Fragmin) subcutaneously once a day, and 5000 IU of standard unfractionated Heparin (KabiVitrum, Stockholm) subcutaneously twice daily as thromboprophylaxis was compared in 200 medical patients in a randomized double blind trial. According to the risk of DVT the patients were stratified before randomization in a high and low risk group. The high risk group consisted of 100 patients mainly with malignant diseases and/or previous history of thromboembolism, the low risk group of 100 patients with mainly myocardial infarction and/or coronary heart disease. The prophylaxis was given for seven to ten days. In 192 consecutive patients the clinical status and thermographic screening for DVT (leg temperature profiles, DeVeTherm) were daily evaluated. In two cases of suspected DVT and one case of suspected PE, the following phlebography or pulmonary scintigraphy were found to be negative. In the high risk group, one patient treated with Fragmin having a central venous catheter developed on day 10 symptoms of an arm vein thrombosis. There were no bleeding complications observed in either of the two treatment groups. Two patients with trombocytopenia (25.000 and 22.000/pl) due to chemotherapy and underlying malignant disease were successfully treated with Fragmin without developing any bleeding complications. In eight patients during Fragmin prophylaxis invasive diagnostic methods as heart catheterization, gastroscopy, bronchoscopy or spinal puncture were performed without noticing any bleeding events. 2500 anti-Factor Xa U of Fragmin gave plasma levels by anti-Factor Xa assay (S-2222, Kabi) of mean 0,1 U/ml when blood was sampled three to four hours after the subcutaneus application. There was no accumulation during the treatment periode observed.This study suggests that 2500 anti-Factor Xa U of Fragmin once daily is as safe and effective as 5000 IU of standard heparin twice daily in these medical patients. Especially in patients who need prophylaxis for a long time eg. with malignant disease, the once daily injection is welcomed.
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Reports on the topic "Family history of coronary heart disease"

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Li, Xiao, Fayang Ling, Wenchuan Qi, Sanmei Xu, Bingzun Yin, Zihan Yin, Qianhua Zheng, Xiang Li, and Fanrong Liang. Preclinical Evidence of Acupuncture on infarction size of Myocardial ischemia: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0044.

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Review question / Objective: Whether acupuncture is effective for infarction size on myocardial ischemia rat models. Condition being studied: Myocardial ischemia is a typical pathological condition of coronary heart disease (CHD), which has been a global issue with high incidence and mortality. Myocardial infarction caused by myocardial ischemia leads to cardiac dysfunction, and the size of myocardial infarction also determines the recovery and prognosis of cardiac function. Acupuncture, a long history of traditional Chinese medicine, is widely used to treat symptoms like thoracalgia and palpitation. Many researches based on rat experiments have shown that acupuncture affects infarction size, cardiac function, myocardial enzyme or arrhythmias severity on myocardial ischemia models; nevertheless, few literatures have systematically reviewed these studies, assessing the risk of bias, quality of evidence, validity of results, and summarizing potential mechanisms. A systematic review of animal studies can benefit future experimental designs, promote the conduct and report of basic researches and provide some guidance to translate the achievements of basic researches to clinical application in acupuncture for myocardial ischemia. Therefore, we will conduct this systematic review and meta analysis to evaluate effects of acupuncture on infarction size on myocardial ischemia rat models.
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