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Journal articles on the topic 'Familial aggregation'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Houwing-Duistermaat, Jeanine J., Bert H. F. Derkx, Frits R. Rosendaal, and Hans C. van Houwelingen. "Testing Familial Aggregation." Biometrics 51, no. 4 (December 1995): 1292. http://dx.doi.org/10.2307/2533260.

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2

SUSSER, EZRA, and MERVYN SUSSER. "FAMILIAL AGGREGATION STUDIES." American Journal of Epidemiology 129, no. 1 (January 1989): 23–30. http://dx.doi.org/10.1093/oxfordjournals.aje.a115119.

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3

Guo, S. W. "Familial Aggregation of Environmental Risk Factors and Familial Aggregation of Disease." American Journal of Epidemiology 151, no. 11 (June 1, 2000): 1121–31. http://dx.doi.org/10.1093/oxfordjournals.aje.a010156.

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4

RYBICKI, BENJAMIN A, MICHAEL C IANNUZZI, MARGARET M FREDERICK, BRUCE W THOMPSON, MILTON D ROSSMAN, EDDY A BRESNITZ, MICHAEL L TERRIN, et al. "Familial Aggregation of Sarcoidosis." American Journal of Respiratory and Critical Care Medicine 164, no. 11 (December 2001): 2085–91. http://dx.doi.org/10.1164/ajrccm.164.11.2106001.

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5

Wing, Yun-Kwok, Lei Chen, Siu-Ping Lam, Albert M. Li, Nelson L. S. Tang, Margaret H. L. Ng, Suk-Hang Cheng, et al. "Familial aggregation of narcolepsy." Sleep Medicine 12, no. 10 (December 2011): 947–51. http://dx.doi.org/10.1016/j.sleep.2011.05.007.

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6

Heun, Reinhard, and Sandra Hein. "Familial aggregation of depression, but no familial aggregation of individual depressive symptoms." European Psychiatry 22, no. 1 (January 2007): 16–21. http://dx.doi.org/10.1016/j.eurpsy.2006.09.001.

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AbstractBackgroundFamilial aggregation of major depression might indicate a genetic liability for the disorder. The complete disorder or, alternatively, only some individual symptoms might be inherited. Under the latter condition, an increased frequency of inherited symptoms might consecutively increase the likelihood to reach the threshold for depression in relatives and, thus, might cause the familial aggregation of depression. Up to now, no study investigated the possibility of a relationship between individual depressive symptoms and the familial aggregation of depression.MethodsThe familial aggregation of early-onset depression (age-at-onset < 60 years, EOD) but less so of late-onset depression (LOD) has been shown in this sample. To assess the hypothesis of an inheritance of individual depressive symptoms as a possible cause of the familial aggregation of depression, frequencies of symptoms were compared in relatives of depressed patients and of controls using forward logistic regression analyses.ResultsSome individual depressive symptoms showed clustering in relatives of patients with depression, but the pattern of inheritance was inconsistent, i.e. the clustering of symptoms was different between non-depressed and depressed relatives of patients with EOD and LOD, respectively. No intra-familial clustering of specific depressive symptoms within families of depressed subjects could be observed.ConclusionsDue to the inconsistencies in the clustering of individual symptoms in non-depressed and depressed relatives and the lack of intra-familial clustering, the familial aggregation of depression is unlikely to be caused by the aggregation of individual depressive symptoms. An inheritance of the vulnerability for complete depressive disorders influenced by environmental factors is more likely.

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7

BOLTON, DEREK, FRÜHLING RIJSDIJK, THOMAS G. O'CONNOR, SEAN PERRIN, and THALIA C. ELEY. "Obsessive–compulsive disorder, tics and anxiety in 6-year-old twins." Psychological Medicine 37, no. 1 (September 26, 2006): 39–48. http://dx.doi.org/10.1017/s0033291706008816.

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Background. Previous reports of genetic influences on obsessive–compulsive disorder (OCD) symptoms have suggested moderate heritability. Family history studies of co-morbidity have found familial aggregation with tics, especially for early-onset OCD, and familial aggregation with anxiety disorders.Method. Heritability of OCD and familial aggregation of OCD, tics and anxiety disorders were investigated in a community sample of 6-year-old twins using a two-phase design in which 4662 twin pairs were sampled and 854 pairs were assessed in the second phase by maternal-informant diagnostic interview using DSM-IV criteria.Results. In the multivariate model combined additive genetic and common environmental effects were estimated as 47% for sub-threshold OCD, and the model was unable to distinguish these sources of familial aggregation. There were strong familial aggregations between sub-threshold OCD and tics and between sub-threshold OCD and other anxiety disorders (80% and 97% respectively), although again specific sources could not be distinguished.Conclusions. The findings are consistent with the hypothesis of a tic-related early-onset OCD phenotype, but also with the hypothesis of an anxiety-related early-onset OCD phenotype.

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8

Kendler, Kenneth S. "Familial risk factors and the familial aggregation of psychiatric disorders." Psychological Medicine 20, no. 2 (May 1990): 311–19. http://dx.doi.org/10.1017/s0033291700017621.

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SynopsisAll major psychiatric disorders aggregate in families. For most disorders, both genes and environmental factors play an important role in this aggregation. While recent work has tended to concentrate on the importance of genetic factors, this report focuses on the potential importance of environmental risk factors which themselves aggregate in families. In particular, this article examines how much of the familial aggregation of a psychiatric disorder may result from the familial aggregation of a risk factor. The model is illustrated and then applied to putative familial risk factors for schizophrenia and depression. The results of the model suggest that if parental loss and exposure to pathogenic rearing practices are true risk factors for depression, then they could account for a significant proportion of the familial aggregation of depression. By contrast, the model predicts that even if obstetric injury and low social class are true risk factors for schizophrenia, they together would account for only a very small proportion of the tendency for schizophrenia to aggregate in families.

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9

Shakhtarina, Svetlana Vasilevna, A. A. Danilenko, and N. A. Falaleeva. "Familial Aggregation in Hodgkin’s Lymphoma." Clinical oncohematology 14, no. 2 (2021): 193–97. http://dx.doi.org/10.21320/2500-2139-2021-14-2-193-197.

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10

Page, Jessica M., Tsegaselassie Workalemahu, Nathan R. Blue, Alison Fraser, Michael W. Varner, Ware D. Branch, and Robert M. Silver. "1036 Familial aggregation of stillbirth." American Journal of Obstetrics and Gynecology 224, no. 2 (February 2021): S642—S643. http://dx.doi.org/10.1016/j.ajog.2020.12.1061.

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11

Suh, Il, Il Soon Kim, and Young Moon Chae. "Familial Aggregation of Blood Pressure." Yonsei Medical Journal 28, no. 3 (1987): 199. http://dx.doi.org/10.3349/ymj.1987.28.3.199.

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12

Cruz, Simao, Carolina Lemos, and Jose Maria Pereira Monteiro. "Familial aggregation of cluster headache." Arquivos de Neuro-Psiquiatria 71, no. 11 (November 2013): 866–70. http://dx.doi.org/10.1590/0004-282x20130170.

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Several studies suggest a strong familial aggregation for cluster headache (CH), but so far none of them have included subjects with probable cluster headache (PCH) in accordance with the International Classification of Headache Disorders. Objective To identify cases of probable cluster headache and to assess the familial aggregation of cluster headache by including these subjects. Method Thirty-six patients attending a headache consultation and diagnosed with trigeminal autonomic headaches were subjected to a questionnaire-based interview. A telephone interview was also applied to all the relatives who were pointed out as possibly affected as well as to some of the remaining relatives. Results Twenty-four probands fulfilled the criteria for CH or PCH; they had 142 first-degree relatives, of whom five were found to have CH or PCH, including one case of CH sine headache. The risk for first-degree relatives was observed to be increased by 35- to 46-fold. Conclusion Our results suggest a familial aggregation of cluster headache in the Portuguese population.

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13

Lee, April, and Chris Yang. "Familial aggregation of mandibular prognathism." Dentistry 3000 3, no. 1 (April 8, 2015): 13–15. http://dx.doi.org/10.5195/d3000.2015.32.

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Mandibular prognathism is a hereditary condition where there is an excess growth of the mandible in relation to the maxilla that can be associated with maxillary retrusion, mandibular protrusion, or both. Skeletal mandibular prognathism is most prevalent in Eastern Asian populations. This paper focuses on a Korean family with skeletal mandibular prognathism that was inherited through three generations. Apparently, neither mandible nor maxilla is retruded in the affected individuals, but there is a concave facial profile. The dentition has a class I occlusion with skeletal mandibular prognathism, and the only way to treat this case would be orthognathic surgery with the help of orthodontic appliances.

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14

Christophersen, Ingrid Elisabeth, Lasse Steen Ravn, Esben Budtz-Joergensen, Axel Skytthe, Stig Haunsoe, Jesper Hastrup Svendsen, and Kaare Christensen. "Familial Aggregation of Atrial Fibrillation." Circulation: Arrhythmia and Electrophysiology 2, no. 4 (August 2009): 378–83. http://dx.doi.org/10.1161/circep.108.786665.

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15

Liang, K.-Y., and T. H. Beaty. "Statistical designs for familial aggregation." Statistical Methods in Medical Research 9, no. 6 (December 1, 2000): 543–62. http://dx.doi.org/10.1191/096228000673742707.

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16

Freedson, Patty S., and Sherrie Evenson. "Familial Aggregation in Physical Activity." Research Quarterly for Exercise and Sport 62, no. 4 (December 1991): 384–89. http://dx.doi.org/10.1080/02701367.1991.10607538.

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17

NIU, TIANHUA, JOHN J ROGUS, CHANGZHONG CHEN, BINYAN WANG, JIANHUA YANG, ZHIAN FANG, SCOTT T WEISS, and XIPING XU. "Familial Aggregation of Bronchodilator Response." American Journal of Respiratory and Critical Care Medicine 162, no. 5 (November 2000): 1833–37. http://dx.doi.org/10.1164/ajrccm.162.5.9908127.

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18

Evenson, Sherrie K., and Patty S. Freedson. "FAMILIAL AGGREGATION AND PHYSICAL ACTIVITY." Medicine and Science in Sports and Exercise 21, Supplement (April 1989): S94. http://dx.doi.org/10.1249/00005768-198904001-00561.

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19

Chandran, V., C. T. Schentag, J. E. Brockbank, F. J. Pellett, S. Shanmugarajah, S. M. A. Toloza, P. Rahman, and D. D. Gladman. "Familial aggregation of psoriatic arthritis." Annals of the Rheumatic Diseases 68, no. 5 (June 4, 2008): 664–67. http://dx.doi.org/10.1136/ard.2008.089367.

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Objectives:The aim of this study was to determine the recurrence risk of psoriatic arthritis (PsA) and uncomplicated psoriasis in first-degree relatives (FDRs) of patients with PsA.Methods:All available FDRs (full siblings, parents and children) of 100 consecutive consenting patients attending a PsA clinic were evaluated for the presence of psoriasis and PsA using a standard protocol. The protocol included a screening questionnaire, physical examination by a rheumatologist, and radiographic and laboratory assessment. The prevalence of PsA and psoriasis in FDRs of the index cases was determined, and the recurrence risk ratio (λ) was calculated, assuming a population prevalence of PsA of 0.25%, and a population prevalence of psoriasis of 2%.Results:The 100 probands had 533 relatives. Eighty-four of them were deceased and 53 were unavailable (age <16 years). Of the remaining 396 FDRs, 107 did not participate (living too far away/did not consent). Thus, 289/396 (73%) of the available FDRs participated in the study. There were 130 siblings, 108 parents and 51 children. The prevalence of PsA and psoriasis among FDRs was 7.6% and 15.2%, respectively. The λ1 was 30.4 for PsA and 7.6 for psoriasis. The prevalence of PsA and psoriasis in siblings was 7.7% and 17.7%, respectively. The λS was 30.8 for PsA and 8.8 for psoriasis.Conclusions:The recurrence risk ratio for both PsA and psoriasis is high in FDRs and siblings of patients with PsA. These results confirm that both PsA and psoriasis have a strong heritable component.

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20

Liang, Kung-Yee, and Terri H. Beaty. "Statistical designs for familial aggregation." Statistical Methods in Medical Research 9, no. 6 (December 2000): 543–62. http://dx.doi.org/10.1177/096228020000900603.

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21

Breitner, JCS, J. M. Silverman, R. C. Mohs, and K. L. Davis. "Familial aggregation in Alzheimerʼs disease." Alzheimer Disease & Associated Disorders 2, no. 4 (1988): 385. http://dx.doi.org/10.1097/00002093-198802040-00020.

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22

Kujala, U. M., M. Ahotupa, T. J. Vasankari, J. Kaprio, and M. J. Tikkanen. "Familial aggregation of LDL oxidation." Scandinavian Journal of Clinical and Laboratory Investigation 57, no. 2 (January 1997): 141–46. http://dx.doi.org/10.1080/00365519709056382.

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23

Ilic, Milena, Miroslav Stojadinovic, and Zoran Milosavljevic. "Familial aggregation of bladder cancer." Vojnosanitetski pregled 68, no. 5 (2011): 447–51. http://dx.doi.org/10.2298/vsp1105447i.

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Background. Except for smoking and certain occupational exposures, the etiology of bladder cancer is largely unknown. Several case reports have described familial aggregation of transitional cell carcinoma of the bladder. Although the majority of patients with bladder cancer do not have family history of transitional cell carcinoma of the urinary tract, the study of familial transitional cell carcinoma may lead to the knowledge on the pathogenesis of this disease. The purpose of this study was to describe three cases of urinary bladder cancer in a single three-member family, i.e. in two generations (mother and son) and a family member related by marriage (the patient?s wife). Case report. Three cases of urinary bladder cancer occurred in a three-member family within the interval of 5 years. The following common characteristics were detected in our patients: old age (over 60), working as farmers for more than 50 years, negative personal medical history on relevant health disorders, place of birth - village, place of residence - village, the same water supply, similar nutrition, positive family history on urinary bladder cancer or other malignant tumors, the first sign of illness was macroscopic hematuria in all the patients and the same pathohistological type of cancer - carcinoma papillare transitiocellulare. Conclusion. The stated common characteristics in our cases indicate, above all, the impact of exposure to external surrounding factors on the occurrence of urinary bladder cancer.

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24

Ranthe, M. F., H. Bundgaard, M. Melbye, and H. A. Boyd. "Familial aggregation of aortic stenosis." European Heart Journal 34, suppl 1 (August 2, 2013): 1813. http://dx.doi.org/10.1093/eurheartj/eht308.1813.

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25

Stevens, M., C. M. van Duijn, W. Kamphorst, P. de Knijff, P. Heutink, W. A. van Gool, P. Scheltens, et al. "Familial aggregation in frontotemporal dementia." Neurology 50, no. 6 (June 1, 1998): 1541–45. http://dx.doi.org/10.1212/wnl.50.6.1541.

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26

Baharloo, Siamak, Susan K. Service, Neil Risch, Jane Gitschier, and Nelson B. Freimer. "Familial Aggregation of Absolute Pitch." American Journal of Human Genetics 67, no. 3 (September 2000): 755–58. http://dx.doi.org/10.1086/303057.

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27

WAHID, R., S. RICH, L. HADAWAY, and M. BLUMENTHAL. "937 Familial aggregation of BHR." Journal of Allergy and Clinical Immunology 97, no. 1 (January 1996): 417. http://dx.doi.org/10.1016/s0091-6749(96)81155-9.

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28

Fejzo, Marlena S., Sue Ann Ingles, Melissa L. Wilson, Wei Wang, Kimber W. Macgibbon, Roberto Romero, and T. Murphy Goodwin. "Familial aggregation of hyperemesis gravidarum." American Journal of Obstetrics and Gynecology 195, no. 6 (December 2006): S191. http://dx.doi.org/10.1016/j.ajog.2006.10.686.

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29

Schulte-Körne, Gerd, Wolfgang Deimel, Katarina Müller, Cornelius Gutenbrunner, and Helmut Remschmidt. "Familial Aggregation of Spelling Disability." Journal of Child Psychology and Psychiatry 37, no. 7 (October 1996): 817–22. http://dx.doi.org/10.1111/j.1469-7610.1996.tb01477.x.

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30

Zhang, Yafeng, Rita M. Cantor, Kimber MacGibbon, Roberto Romero, Thomas M. Goodwin, Patrick M. Mullin, and Marlena S. Fejzo. "Familial aggregation of hyperemesis gravidarum." American Journal of Obstetrics and Gynecology 204, no. 3 (March 2011): 230.e1–230.e7. http://dx.doi.org/10.1016/j.ajog.2010.09.018.

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31

Halder, Smita L. S., Meredythe A. McNally, G. R. Locke, Kristine A. Otto, Prabin Thapa, William S. Harmsen, Alan R. Zinsmeister, and Nicholas J. Talley. "Familial Aggregation of Functional Dyspepsia." American Journal of Gastroenterology 101 (September 2006): S487—S488. http://dx.doi.org/10.14309/00000434-200609001-01260.

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32

Beaty, T. H., C. R. Colyer, Y. C. Chang, K. Y. Liang, J. C. Graybeal, N. K. Muhammad, and L. S. Levin. "Familial Aggregation of Periodontal Indices." Journal of Dental Research 72, no. 2 (February 1993): 544–51. http://dx.doi.org/10.1177/00220345930720021201.

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33

Adams, Ted D., Steven C. Hunt, Lesley A. Mason, Maria E. Ramirez, A. Garth Fisher, and Roger R. Williams. "Familial Aggregation of Morbid Obesity." Obesity Research 1, no. 4 (July 1993): 261–70. http://dx.doi.org/10.1002/j.1550-8528.1993.tb00620.x.

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34

Blaumeiser, Bettina, Ineke van der Goot, Rolf Fimmers, Sandra Hanneken, Sibylle Ritzmann, Katia Seymons, Regina C. Betz, et al. "Familial aggregation of alopecia areata." Journal of the American Academy of Dermatology 54, no. 4 (April 2006): 627–32. http://dx.doi.org/10.1016/j.jaad.2005.12.007.

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35

PORTER, T. "The familial aggregation of prematurity." Journal of the Society for Gynecologic Investigation 3, no. 2 (March 1996): 353A. http://dx.doi.org/10.1016/1071-5576(96)83036-x.

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36

Grünig, E., L. Hamschmidt, H. Kücherer, T. Scheffold, A. Rempiss, E. Kuhn, HP Vosberg, and HA Katus. "Familial aggregation of dilated cardiomyopathy." Journal of Molecular and Cellular Cardiology 24 (August 1992): S11. http://dx.doi.org/10.1016/0022-2828(92)91546-h.

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37

Chakraborty, R. "Nonparametric Evaluations of Familial Aggregation." Biometrical Journal 30, no. 4 (January 19, 2007): 483–94. http://dx.doi.org/10.1002/bimj.4710300413.

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38

KHOURY, MUIN J., TERRI H. BEATY, and LIANG KUNG-YEE. "CAN FAMILIAL AGGREGATION OF DISEASE BE EXPLAINED BY FAMILIAL AGGREGATION OF ENVIRONMENTAL RISK FACTORS?" American Journal of Epidemiology 127, no. 3 (March 1988): 674–83. http://dx.doi.org/10.1093/oxfordjournals.aje.a114842.

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39

Louis, Elan D., Nora Hernandez, Lorraine N. Clark, and Ruth Ottman. "Familial Aggregation of Cranial Tremor in Familial Essential Tremor." Neuroepidemiology 41, no. 1 (2013): 48–53. http://dx.doi.org/10.1159/000348553.

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40

Kendler, KS. "Familial risk factors and the familial aggregation of psychiatric disorders." Alzheimer Disease & Associated Disorders 5, no. 3 (1991): 204–5. http://dx.doi.org/10.1097/00002093-199100530-00013.

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41

Louis, Elan D., Nora Hernandez, Karen P. Chen, Kelly V. Naranjo, Jemin Park, Lorraine N. Clark, and Ruth Ottman. "Familial Aggregation of the Cerebellar Signs in Familial Essential Tremor." Tremor and Other Hyperkinetic Movements 7 (January 13, 2017): 439. http://dx.doi.org/10.5334/tohm.335.

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42

Drake, Virginia E., Alexander Gelbard, Nara Sobriera, Elizabeth Wohler, Lynne L. Berry, Lena L. Hussain, and Alexander Hillel. "Familial Aggregation in Idiopathic Subglottic Stenosis." Otolaryngology–Head and Neck Surgery 163, no. 5 (June 30, 2020): 1011–17. http://dx.doi.org/10.1177/0194599820935402.

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Objective To evaluate inheritance patterns and define the familial clustering rate of idiopathic subglottic stenosis (iSGS). Study Design Retrospective observational study. Setting International multicenter collaborative of >30 tertiary care centers. Methods Patients with a clinically confirmed iSGS diagnosis within the North American Airway Collaborative’s iSGS1000 cohort consented between 2014 and 2018 were eligible for enrollment. Patient demographics and disease severity were abstracted from the collaborative’s iSGS longitudinal registry. Pedigrees of affected families were created. Results A total of 810 patients with iSGS were identified. Positive family history for iSGS was reported in 44 patients in 20 families. The rate of familial clustering in iSGS is 2.5%. Mean age of disease onset is 42.6 years. Of the 44 patients with familial aggregation of iSGS, 42 were female and 2 were male; 13 were mother-daughter pairs and 2 were father-daughter pairs. There were 3 sister-sister pairs. There was 1 niece-aunt pair and 2 groups of 3 family members. One pedigree demonstrated 2 affected mother-daughter pairs, with the mothers being first-degree paternal cousins. Inheritance is non-Mendelian, and anticipation is present in 11 of 13 (84%) parent-offspring pairs. The mean age of onset between parents (48.4 years) and offspring (36.1 years) was significantly different ( P = .016). Conclusion This study quantifies the rate of familial clustering of iSGS at 2.5%. Inheritance is non-Mendelian, and disease demonstrates anticipation. These data suggest that there may be a genetic contribution in iSGS.

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43

Wijsman, Ellen M., Nancy M. Robinson, Kathryn H. Ainsworth, Elisabeth A. Rosenthal, Ted Holzman, and Wendy H. Raskind. "Familial Aggregation Patterns in Mathematical Ability." Behavior Genetics 34, no. 1 (January 2004): 51–62. http://dx.doi.org/10.1023/b:bege.0000009476.33020.b9.

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44

Wang, Xiaobin, Binyan Wang, Changzhong Chen, Jianhua Yang, Zhian Fang, Barry Zuckerman, and Xiping Xu. "Familial Aggregation of Blood Pressure † 551." Pediatric Research 43 (April 1998): 96. http://dx.doi.org/10.1203/00006450-199804001-00572.

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45

Fisher, Naomi D. L., Shelley Hurwitz, Xavier Jeunemaitre, Paul N. Hopkins, Norman K. Hollenberg, and Gordon H. Williams. "Familial Aggregation of Low-Renin Hypertension." Hypertension 39, no. 4 (April 2002): 914–18. http://dx.doi.org/10.1161/01.hyp.0000013784.18175.51.

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46

Timberlake, Andrew T., and Derek M. Steinbacher. "Familial Aggregation of Plastic Surgical Procedures." Plastic and Reconstructive Surgery 142, no. 5 (November 2018): 782e—785e. http://dx.doi.org/10.1097/prs.0000000000004909.

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47

Johnson, Barbara A., David A. Brent, John Connolly, Jeff Bridge, James Matta, Doreen Constantine, Chris Rather, and Tina White. "Familial Aggregation of Adolescent Personality Disorders." Journal of the American Academy of Child & Adolescent Psychiatry 34, no. 6 (June 1995): 798–804. http://dx.doi.org/10.1097/00004583-199506000-00021.

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48

Simonen, R. L., T. Rankinen, L. P??russe, and C. Bouchard. "FAMILIAL AGGREGATION OF PHYSICAL ACTIVITY LEVELS." Medicine & Science in Sports & Exercise 33, no. 5 (May 2001): S277. http://dx.doi.org/10.1097/00005768-200105001-01563.

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49

Sengupta, Aditi, M. P. Sacheva, Col R. Sankar, and W. Selvamurthy. "Familial Aggregation Study of Hashimoto’s Thyroiditis." Anthropologist 3, no. 2 (April 2001): 75–79. http://dx.doi.org/10.1080/09720073.2001.11890692.

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50

Sahin-Yilmaz, Asli, Jayant M. Pinto, Marcy de Tineo, Samy Elwany, and Robert M. Naclerio. "Familial aggregation of nasal conditioning capacity." Journal of Applied Physiology 103, no. 3 (September 2007): 1078–81. http://dx.doi.org/10.1152/japplphysiol.00299.2007.

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In our previous studies on nasal conditioning, we observed a large variability among individuals to condition inspired air. Although we previously investigated various physiological parameters (age, sex, nasal mucosal temperature, heart rate, blood pressure, and nasal volume) that might underlie these differences, we have been unable to explain this variability. Many proteins and molecules, which are under genetic control and could affect nasal conditioning, are involved in water transport,. In this study, we hypothesized that familial factors may contribute to the differences in nasal conditioning capacity (NCC). We performed a prospective study of 47 sibling pairs. Cold dry air was delivered to the nose, and the total water gradient (TWG) was calculated to determine the NCC. We found a highly significant intraclass correlation of 0.53 ( P < 0.0001) between sibling pairs for the TWG. These results suggest that there is a familial basis for nasal conditioning and a large enough genetic component to search for genes explaining the observed correlation.

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