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Books on the topic 'Failure to prevent'

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Published: 9 March 2023
Last updated: 10 March 2023

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1

Smith, Tom. Heart attacks: Prevent and survive. London: Sheldon, 1995.

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2

Riezen, Rhonneke Dyann Van. Armourstone revetments: Will standard design criteria prevent failure? St. Catharines, Ont: Brock University, Dept. of Earth Sciences, 2005.

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3

Posner, Gerald L. Why America slept: The failure to prevent 9/11. New York: Random House, 2003.

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4

Grünfeld, Fred. The failure to prevent genocide in Rwanda: The role of bystanders. Leiden: Martinus Nijhoff, 2007.

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5

Raab, Philippe. Can endogenous group formation prevent coordination failure?: A theoretical and experimental investigation. Bonn, Germany: IZA, 2005.

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6

Great Britain. Health and Safety Executive., ed. Out of control: Why controls system go wrong and how to prevent failure. 2nd ed. Sudbury: HSE Books, 2003.

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7

executive, Health and safety. Out of control: Why control systems go wrong and how to prevent failure. Sudbury: HSE Books, 1995.

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8

United States. Congress. House. Committee on Government Operations. FDA's continuing failure to prevent deceptive health claims for food: Twenty-seventh report. Washington: U.S. G.P.O., 1990.

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9

University of Colorado at Denver. Center for Human Investment Policy. and Colorado Children's Trust Fund, eds. Child maltreatment in Colorado: The value of prevention and the cost of failure to prevent. Denver, Colo: Colorado Children's Trust Fund, 1995.

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10

author, Vermeulen Wessel N., and Krommendijk Jasper author, eds. Failure to prevent gross human rights violations in Darfur: Warnings to and responses by international decision makers (2003-2005). Leiden: Brill Nijhoff, 2014.

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11

Dadrian, Vahakn N. The Armenian genocide and the legal and political issues in the failure to prevent or to punish the crime. [Los Angeles]: University of West Los Angeles, School of Law, 1998.

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12

Mistaken for ADHD: How you can prevent mislabeling your child as a failure in life in the face of a looming ADHD misdiagnosis crisis. Bloominnton, IN: iUniverse, 2010.

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13

How to prevent lean implementation failures: 10 reasons why failures occur. Fort Wayne, Ind: WCM Associates, 2004.

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14

Rosengren, Eric S. Will greater disclosure and transparency prevent the next banking crisis? Boston: Federal Reserve Bank of Boston, 1998.

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15

BP pipeline failure: Hearing before the Committee on Energy and Natural Resources, United States Senate, One Hundred Ninth Congress, second session, to receive testimony relating to the effects of the BP pipeline failure in the Prudhoe Bay oil field on U.S. oil supply and to examine what steps may be taken to prevent a recurrence of such an event, September 12, 2006. Washington: U.S. G.P.O., 2007.

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16

A ticking time bomb: Counterterrorism lessons from the U.S. government's failure to prevent the Fort Hood Attack : hearing before the Committee on Homeland Security and Governmental Affairs, United States Senate, One Hundred Twelfth Congress, first session, February 15, 2011. Washington: U.S. G.P.O., 2011.

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17

United States. Congress. House. Committee on Oversight and Government Reform. Subcommittee on Health Care, District of Columbia, Census, and the National Archives. Examining the administration's failure to prevent and end Medicaid overpayment: Hearing before the Subcommittee on Health Care, District of Columbia, Census, and the National Archives of the Committee on Oversight and Government Reform, House of Representatives, One Hundred Twelfth Congress, second session, September 20, 2012. Washington: U.S. G.P.O., 2012.

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18

Mitrokhin, L. V. Failure of three missions: British diplomacy and intelligence in the efforts to overthrow Soviet government in Central Asia and Transcaucasia and prevent contacts between the Soviet state and the national liberation movements in Afghanistan, Iran and India, 1917-1921, drawing on materials in the National Archives of India in Delhi. Moscow: Progress Publishers, 1987.

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19

Help your family prevent kidney failure. [Bethesda, Md.?: National Institute of Diabetes and Digestive and Kidney Diseases, National Kidney Disease Education Program, 2004.

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20

Breaking Failure: A Guide to Prevent, Diagnose, or Mitigate Failure. Pearson Education, Limited, 2015.

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21

Gardner, Hall. Failure to Prevent World War I: The Unexpected Armageddon. Taylor & Francis Group, 2016.

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22

Posner, Gerald L. Why America Slept: The Failure to Prevent 9/11. Random House, 2003.

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23

Failure to Prevent World War I the Unexpected Armageddon. Taylor & Francis Group, 2015.

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24

Gardner, Hall. Failure to Prevent World War I: The Unexpected Armageddon. Taylor & Francis Group, 2016.

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25

Gardner, Hall. Failure to Prevent World War I: The Unexpected Armageddon. Taylor & Francis Group, 2016.

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26

SOLIVEN-QUEBEC, DR FRANCES G. PREVENT TYPE 2 DIABETES AND END-STAGE KIDNEY FAILURE. 2012.

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27

Posner, Gerald L. Why America Slept: The Failure to Prevent 9/11. Ballantine Books, 2004.

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28

Grünfeld, Fred, and Anke Huijboom. Failure to Prevent Genocide in Rwanda: The Role of Bystanders. Ebsco Publishing, 2007.

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29

Kashani, Kianoush B., and Amy W. Williams. Renal Failure. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0473.

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Renal failure is caused by acute kidney injury or chronic kidney disease. Acute kidney injury (AKI) is a common, devastating complication that increases mortality and morbidity among patients with various medical and surgical illnesses. Also known as acute renal failure, AKI is a rapid deterioration of kidney function that results in the accumulation of nitrogenous metabolites and medications and in electrolyte and acid-base imbalances. This chapter discusses the definition, epidemiology, pathophysiology, and etiology of AKI; the clinical approach to patients with AKI; and the management of AKI. Chronic kidney disease (CKD) has been categorized into 5 stages. When renal function decreases to stage 3, the complications of CKD become evident. These complications include hypertension, cardiovascular disease, lipid abnormalities, anemia, metabolic bone disease, and electrolyte disturbances. To prevent the progression of CKD, therapy must be directed toward preventing these complications and achieving adequate glucose control in diabetic patients with CKD.
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30

Collin, Alicia. Kidney diet: Find out how to eat healthy and prevent kidney failure. Cookmaster Publishing, 2021.

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31

Wijdicks, Eelco F. M., and Sarah L. Clark. Drugs Used to Prevent Complications. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0017.

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Comprehensive neurosciences nursing care goes far in providing optimal support, but the acute immobilization and anticipated prolonged bed rest requires the use of prophylactic drugs. Many options relate to failure to move limbs, failure to breathe adequately and placement of intravenous catheters This chapter covers the more critical preventive measures.Prevention of deep venous thrombosis, hyperglycemia, stress ulcers, ventilator-associated pneumonia, urinary tract infections, vascular access infections, ventriculitis, and post-craniotomy infections are discussed in this chapter. Pharmacists assist in effective stewardship and surveillance of critically ill patients by helping select the appropriate antibiotics, determining the need for drug levels, and initiating or stopping preventative medications.
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32

Hardt, Heidi. Lessons in Failure. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190672171.003.0001.

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Chapter 1 introduces the subject of institutional memory of strategic errors, discusses why it matters for international organizations (IOs) that engage in crisis management and reviews the book’s argument, competing explanations and methodological approach. One strategic error in the mandate or planning of an operation can increase the likelihood of casualties on the battlefield. Knowledge of past errors can help prevent future ones. The chapter explores an empirical puzzle; there remain key differences between how one expects IOs to learn and observed behavior. Moreover, scholars have largely treated institutional memory as a given without explaining how it develops. From relevant scholarship, the chapter identifies limitations of three potential explanations. The chapter then introduces a new argument for how IOs develop institutional memory. Subsequent sections describe research design and explain why NATO is selected as the domain of study. Last, the chapter identifies major contributions to literature and describes the book’s structure.
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33

The Failure to Prevent Genocide in Rwanda (International and Comparative Criminal Law Series). BRILL, 2007.

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34

Minority Rights Group International. Enforcing Mauritania's Anti-Slavery Legislation: The Continued Failure of the Justice System to Prevent... . Minority Rights Group, 2021.

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35

Choudhary, Rajiv, Kevin Shah, and Alan Maisel. Biomarkers in acute heart failure. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0037.

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Acute heart failure continues to be a worldwide medical problem, associated with frequent readmissions, high mortality, and a profound economic impact on national health care systems. In the past decade, biomarkers have shifted the way in which acute heart failure is managed by the cardiologist. The search for the ideal biomarker to aid in the diagnosis, prognosis, and treatment of acute heart failure is ongoing. The natriuretic peptides have proved extremely useful in determining whether acute dyspnoea has a cardiac aetiology. In addition, recent trials have demonstrated the use of natriuretic peptides in inpatient and outpatient prognosis, as well as in titrating medications in outpatients with chronic heart failure to prevent acute heart failure hospitalizations. Other emerging acute heart failure biomarkers include mid-regional pro-adrenomedullin, mid-regional proatrial natriuretic peptide, troponin, ST2, and neutrophil gelatinase-associated lipocalin.
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36

Choudhary, Rajiv, Kevin Shah, and Alan Maisel. Biomarkers in acute heart failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0037_update_001.

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Acute heart failure continues to be a worldwide medical problem, associated with frequent readmissions, high mortality, and a profound economic impact on national health care systems. In the past decade, biomarkers have shifted the way in which acute heart failure is managed by the cardiologist. The search for the ideal biomarker to aid in the diagnosis, prognosis, and treatment of acute heart failure is ongoing. The natriuretic peptides have proved extremely useful in determining whether acute dyspnoea has a cardiac aetiology. In addition, recent trials have demonstrated the use of natriuretic peptides in inpatient and outpatient prognosis, as well as in titrating medications in outpatients with chronic heart failure to prevent acute heart failure hospitalizations. Other emerging acute heart failure biomarkers include mid-regional pro-adrenomedullin, mid-regional proatrial natriuretic peptide, troponin, ST2, and neutrophil gelatinase-associated lipocalin.
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37

Choudhary, Rajiv, Kevin Shah, and Alan Maisel. Biomarkers in acute heart failure. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0037_update_002.

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Acute heart failure continues to be a worldwide medical problem, associated with frequent readmissions, high mortality, and a profound economic impact on national health care systems. In the past decade, biomarkers have shifted the way in which acute heart failure is managed by the cardiologist. The search for the ideal biomarker to aid in the diagnosis, prognosis, and treatment of acute heart failure is ongoing. The natriureticfc peptides have proved extremely useful in determining whether acute dyspnoea has a cardiac aetiology. In addition, recent trials have demonstrated the use of natriuretic peptides in inpatient and outpatient prognosis, as well as in titrating medications in outpatients with chronic heart failure to prevent acute heart failure hospitalizations. Other emerging acute heart failure biomarkers include mid-regional pro-adrenomedullin, mid-regional proatrial natriuretic peptide, troponin, ST2, and neutrophil gelatinase-associated lipocalin.
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38

Choudhary, Rajiv, Kevin Shah, and Alan Maisel. Biomarkers in acute heart failure. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0037_update_003.

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Acute heart failure continues to be a worldwide medical problem, associated with frequent readmissions, high mortality, and a profound economic impact on national health care systems. In the past decade, biomarkers have shifted the way in which acute heart failure is managed by the cardiologist. The search for the ideal biomarker to aid in the diagnosis, prognosis, and treatment of acute heart failure is ongoing. The natriureticfc peptides have proved extremely useful in determining whether acute dyspnoea has a cardiac aetiology. In addition, recent trials have demonstrated the use of natriuretic peptides in inpatient and outpatient prognosis, as well as in titrating medications in outpatients with chronic heart failure to prevent acute heart failure hospitalizations. Other emerging acute heart failure biomarkers include mid-regional pro-adrenomedullin, mid-regional proatrial natriuretic peptide, troponin, ST2, and neutrophil gelatinase-associated lipocalin.
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39

Swiatkowska, Ilona. Biomarkers of Hip Implant Function: Diagnostic Modalities to Prevent Chronic Periprosthetic Joint Infection and Implant Failure. Elsevier Science & Technology Books, 2022.

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40

Swiatkowska, Ilona. Biomarkers of Hip Implant Function: Diagnostic Modalities to Prevent Chronic Periprosthetic Joint Infection and Implant Failure. Elsevier Science & Technology, 2022.

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41

Arroyo, Vicente, Mónica Guevara, and Javier Fernández. Renal failure in cirrhosis. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0247.

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A major event in liver cirrhosis is the development of a progressive deterioration of circulatory function due to splanchnic arterial vasodilation and impairment in cardiac function. This feature determines a homeostatic activation of the renin–angiotensin–aldosterone system, sympathetic nervous system, and antidiuretic hormone. The splanchnic microcirculation is resistant to the vasoconstrictor effect of these systems. Therefore, the homeostasis of arterial pressure in cirrhosis occurs in the extrasplanchnic, mainly renal circulation. The activation of these systems produces renal fluid retention, which accumulates as ascites, and water retention and dilutional hyponatraemia. In the latest phase of cirrhosis, when circulatory dysfunction is severe, renal vasoconstriction is intense and patients develop type 2 hepatorenal syndrome (HRS) and refractory ascites.Type 1 HRS is an acute and rapidly progressive renal failure that occurs in the setting of a precipitating event, commonly an infection. Patients with type 1 HRS also present with rapid deterioration of liver function (encephalopathy, jaundice) and relative adrenal insufficiency. The mechanism of this multiorgan failure is an acute deterioration in circulatory function due to both an accentuation of arterial vasodilation and of cardiac dysfunction.There is no specific test for the diagnosis of HRS. The most accepted diagnostic criteria are those proposed by the International Ascites Club which are based on the exclusion of other types of renal failure. The course of renal failure following treatment of the precipitating event of HRS is another important diagnostic feature.The treatment of choice of tense ascites in cirrhosis is paracentesis associated with intravenous albumin infusion. Moderate sodium restriction and diuretics (spironolactone alone or associated with furosemide) are subsequently given to prevent re-accumulation of ascites. Diuretics are the treatment of choice in patients with moderate ascites. Patients with type 2 HRS and refractory ascites (not responding to diuretics) could be treated by frequent paracentesis or by the insertion of a transjugular intrahepatic portosystemic shunt (TIPS).Terlipressin plus albumin is the treatment of choice in type 1 HRS
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42

OWENS, Kimberly. Kidney Stone Cookbook for Dummies: Discover Several Recipes to Help You Manage Kidney Stones and Prevent Kidney Failure. Independently Published, 2021.

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43

Nagel, Dave. Effective Grading Practices for Secondary Teachers: Practical Strategies to Prevent Failure, Recover Credits, and Increase Standards-Based/Referenced Grading. Corwin Press, 2015.

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44

PENS, Lisa. New Cirrhosis Diet Control Cookbook: Doctors Approved Dietary Guide and Recipes to Reverse Liver Cirrhosis and Prevent Liver Failure. Independently Published, 2022.

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45

COLE, Colin. Heart Failure Treatment Guide: The Complete Guide to Learn, Understand, Treat, Prevent and Tips to Get Your Life Back. Independently Published, 2022.

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46

Inman, Daniel J. Presented at the 1995 Asme Design Engineering Technical Conferences--The 11th Biennale Conference on Reliability, Stress Analysis, and Failure Prevent (Htd). American Society of Mechanical Engineers, 1995.

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47

Scott, Gabriel. Healthy Body Organs Cookbook: Immune Boosting Diets Nutrition, Food Science, and Recipes and How to Prevent and Reverse Organ Failure and Everything You Need to Know. Independently Published, 2021.

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48

Lee, May. Kidney Failure Prevention Natural Healing and Cure: Know Early Signs and Symptoms of Failing Kidneys Ahead Before Your Life Is in Danger! Superfoods Naturally Prevent and Cure Kidney Diseases! Independently Published, 2016.

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49

Hatfield, Anthea. The kidney. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199666041.003.0022.

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Renal failure after surgery does occur and carries a high mortality. The immediate postoperative management of the patient’s fluids is an important part of their recovery. This chapter will teach you the physiology of kidney function, how and why things go wrong, and how to anticipate and prevent renal failure from developing. Conditions that can lead to renal problems are discussed and also the effects of the drugs given to the patients during and after the operation.
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50

Ostermann, Marlies, and Ruth Y. Y. Wan. Diuretics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0058.

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Fluid overload and chronic hypertension are the most common indications for diuretics. The diuretic response varies between different types and depends on underlying renal function. In patients with congestive heart failure, diuretics appear to reduce the risk of death and worsening heart failure compared with placebo, but their use in acute decompensated heart failure is questionable. Diuretics are also widely used in chronic kidney disease to prevent or control fluid overload, and treat hypertension. In acute kidney injury, there is no evidence that they improve renal function, speed up recovery, or change mortality. In patients with chronic liver disease and large volume ascites, paracentesis is more effective and associated with fewer adverse events than diuretic therapy, but maintenance treatment with diuretics is indicated to prevent recurrence of ascites. Mannitol has a role in liver patients with cerebral oedema and normal renal function. The use of diuretics in rhabdomyolysis is controversial and restricted to patients who are not fluid deplete. In conditions associated with resistant oedema (chronic kidney disease, congestive heart failure, chronic liver disease), combinations of diuretics with different modes of action may be necessary. Diuresis is easier to achieve with a continuous furosemide infusion compared with intermittent boluses, but there is no evidence of better outcomes. The role of combination therapy with albumin in patients with fluid overload and severe hypoalbuminaemia is uncertain with conflicting data.
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