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1

Eisner, Mark D. Environmental factors and asthma: What we learned from epidemiological studies. Philadelphia: Saunders, 2008.

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2

Harver, Andrew. Asthma, health, and society: A public health perspective. New York: Springer, 2010.

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3

Asthma, health, and society: A public health perspective. New York: Springer, 2010.

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4

American, Thoracic Society Workshop (1997 Montreal Canada). Immunobiology of asthma and rhinitis: Pathogenic factors and therapeutic options : American Thoracic Society Workshop summary : Montreal, Canada, June 1997. [New York, N.Y.]: American Thoracic Society, 1999.

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5

Asthma sourcebook: Basic consumer health information about allergic, exercise-induced, occupational, and other types of asthma, including facts about causes, risk factors, symptoms, and diagnostic tests and featuring details about treating asthma with medication and other therapies ... 3rd ed. Detroit, MI: Omnigraphics, Inc., 2011.

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6

Hodgson, Gabrielle. Statistical snapshots of people with asthma in Australia 2001. Canberra: Australian Institute of Health and Welfare, 2007.

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7

1946-, Stute W., and Seminar on Empirical Processes (1985 : Düsseldorf, Germany), eds. Seminar on Empirical Processes. Basel: Birkhäuser Verlag, 1987.

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8

Carol, Svec, ed. The Inflammation cure: How to combat the hidden factor behind heart disease, arthritis, asthma, diabetes, Alzheimer's disease, osteoporosis, and other diseases of aging. Chicago: Contemporary Books, 2004.

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9

Predine-Hug, François. L'odontologiste face à: Asthme, cirrhose, diabète, endocardite, épilepsie, grossesse, hémophilie, insuffisance cardiaque, AAP, AC, SIDA, toxicomanies : conduites à tenir. Paris: Éd. SID-Groupe EDP sciences, 2011.

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10

Cory, Stella. Prevalence of selected risk behaviors and chronic diseases and conditions: Steps communities, United States, 2006-2007. Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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11

Yeh, Fawn. Prevalence of asthma/asthma-like symptoms and asthma risk factors in Amerian Indian youth: Riverside asthma screening. [s.n.], 2001.

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12

W, Busse W., and Lemanske Robert F, eds. Asthma prevention. Boca Raton, FL: Taylor & Francis, 2005.

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13

Asthma: Social and Psychological Factors and Psychosomatic Syndromes. Muenchen: Karger, 2005.

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14

Brown, E. S., ed. Asthma: Social and Psychological Factors and Psychosomatic Syndromes. S. Karger AG, 2003. http://dx.doi.org/10.1159/isbn.978-3-318-00972-9.

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15

Lynde, Grant C. Asthma and Pregnancy. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0054.

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Asthma’s progression during pregnancy is highly variable. Improvement in symptoms can be seen in 18%–34% of patients, while worsening of symptoms can be seen in 20%–42% of patients. Acute exacerbations of asthma are most frequently seen late in the second trimester and are associated with a viral upper-respiratory infection. An acute exacerbation of asthma in the parturient can result in increased risk of maternal mortality, preterm delivery, and low-birth-weight infants. In patients with moderate to severe asthma, good control with inhaled corticosteroids, such as budesonide, is a cornerstone of reducing morbidity and mortality. The four components of care for the asthmatic patient are education, control of environmental factors, medications, and monitoring of symptoms.
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16

Immunobiology of asthma and rhinitis: Pathogenic factors and therapeutic options, Montreal, Canada, June, 1997. [S.l.]: American Thoracic Society, 1999.

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17

Cope, Shannon. Socioeconomic factors related to asthma control and health-related quality of life in children. 2007.

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18

Asthma sourcebook: Basic consumer health information about allergic, exercise-induced, occupational, and other types of asthma, including facts about causes, risk factors, symptoms, and diagnostic tests and featuring details about treating asthma with medication and other therapies .... Detroit, MI: Omnigraphics, Inc., 2016.

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19

Brown, E. S. Asthma: Social and Psychological Factors and Psychosomatic Syndromes : 10 figures and 16 tables, 2003 (Advances in Psychosomatic Medicine). S. Karger Publishers (USA), 2003.

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20

Weiss, Kevin B. Asthma's Impact on Society: The Social and Economic Burden (Lung Biology in Health and Disease, V. 138) (Lung Biology in Health and Disease, V. 138). Informa Healthcare, 1999.

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21

B, Weiss Kevin, Buist A. Sonia 1940-, and Sullivan Sean D. 1960-, eds. Asthma's impact on society: The social and economic burden. New York: M. Dekker, 2000.

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22

Alam, Saima, and Christopher Corrigan. Allergy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0001.

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Allergic disorders are on the increase. This chapter covers a range of common allergic problems encountered in patients with severe asthma which may contribute to symptomatology and exacerbations, including genetic and environmental factors, affecting the airways, and the diagnosis and management of atopic asthma in association with seasonal and perennial allergic rhinitis. The account elucidates the basic principles behind simple investigations, such as skin prick testing and in vitro allergen-specific IgE assays, as well as relatively novel tests, including component resolved diagnosis. Also covered are recent advances in managing patients with seasonal and perennial rhinosinusitis, with an explanation of the indications, and the protocols available for desensitization to pollens, house dust mites, and animal dander. Overall, this chapter should provide the reader with adequate knowledge to deal with day-to-day management dilemmas in a specialist asthma clinic.
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23

G, Holme, and Morley J. 1938-, eds. PAF in asthma. Academic Press, 1989.

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24

(Editor), J. Morley, ed. Paf in Asthma (Perspectives in Asthma Series, Vol 3). Academic Press, 1990.

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25

1952-, Schmitz-Schumann M., Menz G, and Page C. P, eds. PAF, platelets, and asthma. Basel: Birkhäuser, 1987.

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26

Michaud, Dominique, David Savitz, and Lorelei Mucci. Brain Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0024.

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cBrain tumors constitute an array of histologic types, the most common being meningioma and glioma. Unlike other cancers, both benign and malignant brain tumors are concerning for survival because of their anatomic location. Two-thirds of brain tumors are benign. The most well established risk factor is high dose ionizing radiation, based on studies of atomic bomb survivors as well as children treated for tinea capitis. In contrast, nonionizing radiation including from cellular telephones, is not a risk factor. Tobacco use does not appear to be associated with glioma or meningioma. There is fairly consistent evidence of an inverse association between allergies and asthma with risk of glioma, potentially through levels of IgE. Finally, occupational epidemiology studies suggest potential positive associations with specific exposures. The identification of modifiable risk factors for brain tumors has been challenging, due in part to the diversity of tumor subtypes.
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27

Cass, Lindy. Intraoperative Wheezing. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0020.

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Wheezing is an important sign for anesthesiologists. At the preoperative consult it usually indicates bronchospasm from poorly controlled asthma, though many other causes are possible. Intraoperative wheezing also has many potential causes, including bronchospasm, airway obstruction, anaphylaxis, or aspiration. Intraoperative wheezing is an anesthetic emergency that can lead to life-threatening respiratory and cardiac complications. Prompt action to maintain oxygenation, removal of any trigger factors, and, if indicated, bronchodilator administration will usually result in a safe outcome.
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28

Goldstein, Inge F., and Martin Goldstein. How Much Risk? Oxford University Press, 2002. http://dx.doi.org/10.1093/oso/9780195139945.001.0001.

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An excellent critical analysis and scientific assessment of the nature and actual level of risk leading environmental health hazards pose to the public. Issues such as radiation from nuclear testing, radon in the home, and the connection between electromagnetic fields and cancer, environmental factors and asthma, pesticides and breast cancer and leukemia clusters around nuclear plants are discussed, and how scientists assess these risks is illuminated. This book will enable readers to better understand environmental health issues, and with the proper scientific understanding, make informed, rational decisions about them.
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29

Schakett, Brent, and Kathleen Chen. Laryngospasm. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0013.

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Laryngospasm is a complication that all pediatric anesthesia providers must be able to successfully diagnosis and treat. The risk factors include but are not limited to recent upper respiratory infection, history of asthma, preschool-age child, airway surgery, and light anesthesia. Laryngospasm can be defined as either partial laryngospasm with residual opening of the glottis or complete laryngospasm where there is no air movement. Prevention is obtained by limiting risk factors; waiting 6 to 8 weeks after upper respiratory infection symptoms have resolved if possible, smoking cessation, suctioning of residual secretions, and maintaining an adequate depth of anesthesia during crucial times like intubation and extubation. Treatment includes jaw thrust with positive pressure and 100% oxygen, followed by a subhypnotic dose of propofol if the laryngospasm does not break, then finally succinylcholine if all other methods have failed. With treatments that depress respiratory drive, delirium can result and must be diagnosed correctly.
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30

1955-, Agosti Jan M., and Sheffer Albert L. 1929-, eds. Biotherapeutic approaches to asthma. New York: Dekker, 2002.

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31

Jeffrey, Andrew A. Wheeze. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0017.

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A wheeze is a high-pitched musical sound which reflects airflow obstruction. It may be monophonic or polyphonic, and may be heard during inspiration, expiration, or both phases of respiration. Stridor is the term used to describe wheeze which is louder over the neck than the chest, and may be audible without a stethoscope. Wheezing is typically associated with breathlessness, and may present as an acute or chronic problem. It is most often due to asthma or chronic obstructive pulmonary disease. In stable patients, a detailed history should be taken, with particular attention to the speed of onset of wheeze; trigger factors; and history of atopy. The clinical features, measurement of peak expiratory flow, and spirometry will usually differentiate between possible diagnoses. This chapter describes the approach to the diagnosis of patients with wheeze.
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32

Almond, Mark H., and Mark J. Griffiths. Swine ‘flu’ in pregnancy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0020.

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Influenza viruses are a significant cause of morbidity and mortality globally, resulting in severe illness in 3-5 million people and death in up to 500,000 during epidemic years. In March 2009, a novel H1N1 virus emerged in Mexico, spreading rapidly around the globe and achieving pandemic status within 3 months. Although it is now generally considered that the 2009 pandemic resulted in mild disease in most individuals, serious complications still occurred, with 12,000 deaths by mid-February 2010 in the United States alone. Risk factors for severe disease included asthma, cardiac disease, immunosuppression, pregnancy, diabetes mellitus, and obesity. The chapter outlines the case of a young pregnant female who presented with an influenza-like illness and subsequently developed acute respiratory distress syndrome requiring extracorporeal membrane oxygenation. The origins, presentation, diagnosis, complications, and management of pandemic influenza are discussed, in addition to a summary of the pulmonary physiology and pathology of pregnancy.
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33

Abhishek, Abhishek, and Michael Doherty. Placebo, nocebo, and contextual effects. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0027.

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Placebo effect is an example of ‘contextual’ effect and is the symptomatic improvement experienced by patients who have unknowingly received a placebo (inactive treatment) instead of an active drug. It occurs due to patient-specific factors such as expectation of improvement and is influenced by the context in which the treatment is delivered. Nocebo effect is the opposite of placebo effect and includes worsening of symptoms or incident adverse effects due to expectancy or negative contextual or practitioner influence. Placebo effect has been demonstrated in a range of musculoskeletal conditions, including osteoarthritis (OA), as well as other conditions such as Parkinson’s disease, irritable bowel syndrome, and asthma. In OA, the placebo effect is strongest for subjective outcomes like pain. In fact, the effect size (ES) of placebo analgesia in OA clinical trials (0.51) is clinically significant and higher than the ES (defined by the additional improvement above placebo) obtained from non-pharmacological (0.25) and pharmacological (0.39) treatments. A number of patient- and intervention-specific and contextual factors influence the magnitude of placebo-induced improvements. Placebo analgesia is real, not a ‘trick of the mind’, and results from central mechanisms that increase descending inhibition of pain. Contextual effects are an integral part of everyday clinical practice. While patient- and intervention-specific determinants cannot be changed easily, healthcare practitioners should optimize the physician-specific factors that enhance positive contextual response and minimize nocebo response. Such a strategy that will increase the overall improvement is particularly relevant for OA where there is no ‘cure’ and a predominance of negative beliefs.
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34

George, Holme, and Morley J, eds. PAF in asthma: Proceedings of a symposium held in Canada in June 1986. London: Academic Press, 1989.

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35

Svec, Carol, and William Joel Meggs. The Inflammation Cure : How to Combat the Hidden Factor Behind Heart Disease, Arthritis, Asthma, Diabetes, & Other Diseases. McGraw-Hill, 2003.

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36

Svec, Carol, and William Joel Meggs. The Inflammation Cure : How to Combat the Hidden Factor Behind Heart Disease, Arthritis, Asthma, Diabetes, & Other Diseases. McGraw-Hill, 2003.

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37

North, Crystal M., and David C. Christiani. Respiratory Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0025.

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This chapter describes the clinical presentation of commonly-encountered occupational and environmental respiratory disorders, including principles regarding disease recognition and prevention. Where appropriate, clinical cases are included to illustrate specific diseases. The chapter begins with a general introduction to the evaluation of individuals and populations, including important considerations from the history and physical examination as well as common findings on typical diagnostic tests such as chest X-rays and other imaging studies, and pulmonary function testing. Specific disease topics covered include (a) disorders due to irritant exposures (high-, moderate-, and low-solubility irritants); (b) disorders due to nonirritant exposures (carbon monoxide and indoor and ambient air pollution); (c) occupational airways diseases (work-related asthma and occupational chronic obstructive pulmonary disease); (d) hypersensitivity pneumonitis, byssinosis, and other diseases due to organic dust exposure; and (e) pneumoconiosis (including asbestosis, silicosis, and coal workers’ pneuconiosis). Childhood asthma is discussed as a risk factor for occupationally-related lung disease.
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38

Abi-Nahed, Genevieve. Asthma as a risk factor for dental caries and dental erosion in children and adolescents /cby Genevieve Abi-Nahed. 2007, 2007.

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39

Launois, Sandrine H., and Patrick Lévy. Pulmonary disorders and sleep. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0041.

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Sleep disorders and pulmonary diseases are closely associated, a fact clearly underestimated in routine patient care, despite evidence that these disorders interact to impact on quality of life as well as on morbidity and mortality. The prevalence of chronic insomnia, sleep-related breathing disorders, and restless leg syndrome is high in patients with chronic pulmonary disorders such as asthma, chronic obstructive pulmonary disease, cystic fibrosis, interstitial lung disease, chest wall and neuromuscular disorders, and chronic respiratory failure. This association may be fortuitous and reflect the impact of a chronic condition on sleep quality, or it may be due to specific sleep-related phenomena adversely affecting an underlying pulmonary disorder. Furthermore, obstructive sleep apnea has been implicated as a risk factor for pulmonary hypertension and pulmonary embolism. This chapter outlines the implications for both pulmonary and sleep specialists, in terms of clinical management and treatment strategies.
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