Relevant bibliographies by topics / Factors and asthma

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Factors and asthma'

Author: Grafiati
Published: 23 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Factors and asthma"

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Toskala, Elina, and David W. Kennedy. "Asthma risk factors." International Forum of Allergy & Rhinology 5, S1 (September 2015): S11—S16. http://dx.doi.org/10.1002/alr.21557.

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Vamos, Marina, and John Kolbe. "Psychological Factors in Severe Chronic Asthma." Australian & New Zealand Journal of Psychiatry 33, no. 4 (August 1999): 538–44. http://dx.doi.org/10.1080/j.1440-1614.1999.00591.x.

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Objective: Levels of psychological distress, social support factors, and emotional adjustment to illness were measured in a sample of patients with severe asthma. These were then examined in terms of their interrelationships and their ability to predict self-management knowledge. Method: A sample of 80 patients was recruited from a hospital-based asthma clinic designed for patients with severe asthma. Thirty-four percent of consecutive attenders approached took part. Morbidity and asthma management were recorded from case records. Anxiety, depression, social support, emotional adjustment to asthma and asthma knowledge were measured using self-report instruments selected for their acceptability and ease of administration. Results: Twenty-five percent of the sample had possible or definite caseness for anxiety; 10.3% had possible or definite caseness for depression. Twenty-five percent had inadequate social support in some way. Three independent attitudinal factors were found: emotional maladjustment to asthma, the doctor-patient relationship, and asthma-related stigma. Level of asthma knowledge was very low. None of the measures of psychosocial function chosen were predictive of asthma knowledge. Conclusions: Levels of asthma knowledge were dangerously low, despite apparently adequate educational intiatives. In addition, patients with severe asthma have high levels of distress, particularly of anxiety, even between attacks. Their attitudes to their illness are multifactorial, and are significantly correlated with emotional distress, morbidity indices and some demographic factors. While this may point the way to interventions designed to relieve patients' distress, the hypothesis that this might in turn relate to practical asthma knowledge was not confirmed.
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Bosley, C. M., Z. M. Corden, and G. M. Cochrane. "Psychosocial factors and asthma." Respiratory Medicine 90, no. 8 (September 1996): 453–57. http://dx.doi.org/10.1016/s0954-6111(96)90170-5.

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Mathison, David A., Donald D. Stevenson, and Ronald A. Simon. "Precipitating Factors in Asthma." Chest 87, no. 1 (January 1985): 50S—54S. http://dx.doi.org/10.1378/chest.87.1.50s.

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Kamal, Khalid M., and Lesley-Ann Miller. "Psychosocial Factors and Asthma." American Journal of Respiratory and Critical Care Medicine 169, no. 11 (June 2004): 1253–54. http://dx.doi.org/10.1164/ajrccm.169.11.952.

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Tauler, E., J. Llorens-Terol, A. Mur, and C. Leal. "ASTHMA AND ENVIRONMENTAL FACTORS." Pediatric Research 19, no. 10 (October 1985): 1120. http://dx.doi.org/10.1203/00006450-198510000-00290.

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Doull, l. J. M., and S. T. Holgate. "Asthma: early predisposing factors." British Medical Bulletin 53, no. 1 (January 1, 1997): 71–80. http://dx.doi.org/10.1093/oxfordjournals.bmb.a011607.

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Giardino, Nicholas D., and Karen B. Schmaling. "Behavioral factors in asthma." Behavior Analyst Today 3, no. 2 (2002): 203–12. http://dx.doi.org/10.1037/h0099970.

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Barnes, P. J., and I. M. Adcock. "Transcription factors and asthma." European Respiratory Journal 12, no. 1 (July 1, 1998): 221–34. http://dx.doi.org/10.1183/09031936.98.12010221.

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Van Lieshout, Ryan J., and Glenda MacQueen. "Psychological Factors in Asthma." Allergy, Asthma & Clinical Immunology 4, no. 1 (2008): 12. http://dx.doi.org/10.1186/1710-1492-4-1-12.

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Dissertations / Theses on the topic "Factors and asthma"

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Bédard, Annabelle. "NUTRITIONAL FACTORS IN ASTHMA." Thesis, Paris 11, 2015. http://www.theses.fr/2015PA11T004/document.

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L’objectif principal de la thèse était de comprendre le rôle complexe des facteurs nutritionnels dans l’asthme, avec une première partie portant sur l’alimentation et une seconde sur l’obésité. Alimentation – L’objectif était de comparer les typologies alimentaires obtenues par une analyse en composantes principales (ACP) à celles identifiées par une analyse factorielle confirmatoire (AFC) grâce aux données de l’étude E3N (étude épidémiologique auprès des femmes de la MGEN, 30589 femmes). En utilisant les ACP et les AFC, 3 typologies alimentaires ont été identifiées. Selon la méthode, des corrélations différentes étaient observées entre les typologies alimentaires et les groupes d’aliments, conduisant à des associations différentes avec les caractéristiques socio-économiques et l’incidence de l’asthme chez l’adulte. Obésité – Le premier objectif était de comprendre les rôles conjoints et indépendants, dépendants du temps, de l’activité physique et l’obésité sur l’asthme en utilisant des modèles marginaux structuraux chez 15352 femmes d’E3N. L’obésité était liée de façon causale à l’asthme actuel avec symptômes, indépendamment de l’activité physique. Le second objectif était de considérer le rôle modificateur du surpoids dans l’association entre l’exposition domestique aux sprays de nettoyage et l’asthme chez 304 femmes d’E3N. Parmi les femmes sans traitement anti-inflammatoire, le rôle délétère de l’utilisation de sprays sur l’asthme était plus fort chez les femmes en surpoids. L’AFC devrait être utilisée pour l’identification des typologies alimentaires en épidémiologie nutritionnelle. L’obésité pourrait causer de l’asthme indépendamment de l’activité physique, et pourrait augmenter l’effet délétère d’autres facteurs environnementaux de l’asthme
The general aim of the thesis was to assess the complex role of nutritional factors in asthma, with a first part focused on diet, and a second part focused on obesity. Diet – The objective was to compare dietary patterns based on principal component analysis (PCA) to patterns based on confirmatory factor analysis (CFA). The analysis was performed among 30,589 women from the E3N study (epidemiological cohort study among women of the Mutuelle Générale de l’Education Nationale). Whether PCA or CFA was used, 3 patterns were derived. For each pattern, we reported different correlations between food groups and dietary patterns when using PCA or CFA, leading to different associations between dietary patterns with socio-economic characteristics and adult-onset asthma. Obesity – The first objective was to assess the joint and independent, time-dependent, roles of physical activity and obesity on asthma using marginal structural models. Among 15,352 women from E3N, analyses showed that obesity was related causally to current asthma with symptoms independently of physical activity, whereas no association was reported for physical activity. The second objective was to assess the modifying role of overweight in the association between domestic exposure to cleaning sprays and current asthma. Among 304 women from E3N, analysis showed that among women without anti-inflammatory therapy, the effect of spray use on asthma was higher in overweight women. CFA should be considered for the assessment of dietary patterns in nutritional epidemiology. Obesity may lead to asthma independently of physical activity, and may increase the deleterious effect of other environmental factors of asthma
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Bédard, Annabelle 1986. "Nutritional factors in asthma = Facteurs nutritionels dans l'asthme." Doctoral thesis, Universitat Pompeu Fabra, 2015. http://hdl.handle.net/10803/460881.

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The general aim of the thesis was to assess the role of nutritional factors in asthma in the French E3N study. The first objective was to compare principal component (PCA) with confirmatory factor analysis (CFA) to identify dietary patterns. In comparison with PCA patterns, CFA patterns were more relevant in terms of correlations with food groups and socio-economic characteristics, and associations with adult-onset asthma. The second objective was to assess the joint roles of physical activity and obesity on asthma using marginal structural models. Analyses showed that obesity was related causally to current asthma with symptoms independently of physical activity, whereas no association was reported for physical activity. The third objective was to assess the modifying role of overweight in the association between domestic cleaning spray use and current asthma. Analyses showed that among women without anti-inflammatory therapy, the effect of spray use on asthma was higher in overweight women.
L’objectiu general de la tesi fou avaluar el paper dels factors nutricionals en l’asma. El primer objectiu fou comparar l’anàlisi de components principals (PCA) amb l’anàlisi factorial confirmatori (CFA) per identificar patrons de dieta. Comparat amb els patrons obtinguts amb PCA, els patrons obtinguts amb CFA mostraven major correlació amb grups d’aliments i característiques sociodemogràfiques, i aconseguien millor predicció de la incidència d’asma. El segon objectiu fou avaluar l’efecte conjunt de l’activitat física i l’obesitat en l’asma, utilitzant models marginals estructurals. Els resultats mostraren que l’obesitat es relaciona causalment amb els símptomes d’asma independentment de l’activitat física, mentre que no es va trobar cap efecte de l’activitat física. El tercer objectiu fou estudiar el paper modificador del sobrepès en l’associació entre l’ús de pulveritzadors de neteja domèstica i la presència d’asma. Les anàlisis mostraren que l’efecte de l’ús dels pulveritzadors era més gran en dones amb sobrepès i tractament antiinflamatori
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Turczynowicz, Leonid. "Asthma and risk factors in South Australia : an ecologic analysis." Title page, table of contents and abstract only, 2000. http://web4.library.adelaide.edu.au/theses/09MPM/09mpmt933.pdf.

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Includes CD-ROM inside back cover of volume 2. Bibliography: p. 178-222. Aims to identify current risk factors for asthma and to determine which of these factors, at the population level, is associated with asthma prevalence in children in South Australia. In addition, modelling techniques are used to determine which factors are significant predictors of asthma prevalence in 4 to 5 year old children in S.A. Study results show that at the population level, 9 risk factors are significantly associated with lifetime prevalence and 24 factors with period prevalence. Study findings are generally consistent with existing literature.
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Kinyanjui, Margaret. "Targeting Th2 transcription factors in experimental asthma." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=18717.

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Antigen specific CD4+ T cells adoptively transfer airway inflammation comprised mainly of lymphocytes and eosinophils. The ability of these transferred T cells to induce inflammation is dependent on the cytokines they express particularly Th2 cytokines. In order to better understand the mechanism by which adoptively transferred T cells induce airway inflammation, we chose to modulate the expression (GATA-3) and activity (STAT-6) of two key regulators of Th2 cytokine production. To modify expression of GATA-3, we used a bicistronic retroviral vector encoding GATA-3 and enhanced green fluorescent protein (EGFP). As a control, we used a retrovector encoding EGFP alone. By coupling in vitro antigen stimulation with retroviral transduction we generated antigen specific CD4+ T cells expressing EGFP alone or GATA-3 and EGFP. When transferred into naïve recipients that were subsequently challenged, these transduced CD4+ T cells induced lung inflammatory responses with an increase in both CD4+ lymphocytes and eosinophils. This antigen specific inflammatory response was enhanced in animals receiving T cells overexpressing GATA-3. Analysis of the infiltrating cells also revealed that the EGFP+ T cells were present in the lung following antigen challenge, comprising only a small fraction of the CD4+ T cells recruited to the lung during the antigen response. Thus, GATA-3 amplifies antigen-specific inflammatory responses in the airways by augmenting the ability of antigen specific T cells to recruit inflammatory cells to the lung following antigen challenge. To modify the activity of STAT-6 we used chimeric cell penetrating peptides containing a poly-arginine protein transduction domain (PTD) coupled to a sequence predicted to bind and inhibit STAT-6 activity (SIP-1). Using fluorescein-tagged SIP-1, we demonstrate that the poly-arginine PTD efficiently translocates to the cytoplasm within an hour. In vitro, antigen-induced IL-4 production was inhibited in SIP-1-treated spleno
Les cellules CD4+ T à antigènes spécifiques transfèrent par adoption l'inflammation pulmonaire constituées principalement de lymphocytes et d'éosinophiles. L'habileté de celles-ci à transférer des cellules T pour induire l'inflammation est dépendante de leur expression de cytokines Th2. De manière à mieux comprendre le mécanisme par lequel les cellules T transmises par adoption induisent l'inflammation pulmonaire, nous avons choisi de moduler l'expression de GATA-3) ou l'activité de (STAT-6) des deux régulateurs-clés de production de cytokine Th2. Afin de modifier l'expression de GATA-3 dans les cellules T destinées au transfert par adoption, nous avons utilisé un rétrovirus recombinant concentré avec une filtration par centrifugeuse. Ce procédé a dramatiquement augmenté leurs titres et ainsi leur habileté à transduire les cellules CD4+ T en culture primaire. Nous avons utilisé un rétrovirus recombinant qui encode la GATA-3 et / ou la protéine fluorescente verte (EGFP). En couplant in vitro la stimulation d'antigènes avec la transduction par vecteur viral, nous avons généré des cellules CD4+ T à antigènes spécifiques exprimant de l'EGFP seul ou bien de la GATA-3 et de l'EGFP. Lorsque transféré dans un rat qui avait subséquemment été provoqué avec des antigènes, ces cellules CD4+ T induisent une réaction aux inflammations pulmonaires avec une augmentation des lymphocytes et éosinophiles. Cette réaction inflammatoire fut accrue chez les animaux recevant les cellules T surexprimant la GATA-3. L'analyse des cellules infiltrantes a aussi révélé que bien que les cellules EGFP+ étaient présentes dans les poumons suivant la provocation par antigènes, elles étaient constituées seulement d'une petite fraction de cellules CD4+ T recrutées dans les poumons. Ainsi, la GATA-3 amplifie la réaction inflammatoire des poumons induite par antigènes en augmentant l'habileté des cellules T à antigènes spécifiques à recruter
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Gunnbjörnsdóttir, María Ingibjörg. "Asthma and respiratory symptoms in Nordic countries, environmental and personal risk factors /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7076.

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Matulonga, diakiese Bobette. "Particularité de l’asthme de la femme : incidence, évolution avec le vieillissement (ménopause) et facteurs de risque." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS255/document.

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IntroductionLes raisons de l’excès de risque d’asthme chez la femme ne sont pas établies. La réversion du sexe ratio autour de la puberté a fait évoquer le rôle des hormones sexuelles féminines dans l’asthme. Cependant l’histoire naturelle de l’asthme après 50 ans a été très peu étudiée. L’objectif général de cette thèse était d’apporter des nouveaux éléments de connaissance sur l’histoire naturelle de l’asthme de la femme. Les objectifs spécifiques étaient l’étude de l’incidence de l’asthme allergique et non-allergique de la femme et de son évolution avec le vieillissement (différences par rapport aux hommes après 50 ans, et association avec la ménopause) et l’étude de certains facteurs de risque associés à l’asthme de la femme.Matériels et méthodesL’incidence et l’évolution de l’asthme ont été étudiées chez 2 125 hommes et 2 298 femmes, initialement âgés de 20 à 44 ans et suivis pendant 20 ans dans la cohorte européenne sur la santé respiratoire « ECRHS ». Les associations entre la ménopause et l’asthme de la femme ont été étudiées dans un échantillon de 67 872 femmes, initialement âgées de 41 à 68 ans et suivies pendant 12 ans au sein de la cohorte E3N. Les données sur les expositions environnementales de l’Etude des facteurs Génétiques et Environnementaux de l’Asthme (EGEA) ont été utilisées pour étudier l’association entre l’usage domestique d’eau de Javel et l’asthme auprès de 607 femmes âgées de 44 ans en moyenne. L’asthme allergique est défini par la présence de tests cutanés positifs.RésultatsDans la cohorte ECRHS, les femmes étaient plus à risque que les hommes de développer de l’asthme non-allergique (Odds-Ratio (OR) ; Intervalle de confiance à IC95% : 4,0 [2,5-6,5]). Cette différence était moins marquée pour l’asthme allergique (OR 1,7 [1,0-2,7]), et on n’observait pas de différence dans la progression de l’asthme.L’analyse de la cohorte E3N ne montre pas de différence dans l’incidence de l’asthme entre les femmes pré-ménopausées et post-ménopausées de même âge, chez les femmes de corpulence normale (Hazard Ratio(HR) :1,1 [0,8-1,3]). Cependant, chez les femmes obèses, l’incidence de l’asthme augmentait lors de la transition vers la ménopause (aHR :1.9[1.0-3.7]) et en post-ménopause (HR :2,9 [1,1-4,1]). La ménopause chirurgicale était également associée à un plus grand risque d’asthme.Dans la cohorte EGEA, seuls 2% d’hommes contre 37% des femmes utilisaient hebdomadairement l’eau de Javel pour faire le ménage. Chez les femmes, l’usage fréquent d’eau de Javel était associé à un plus grand risque d’asthme non-allergique (OR :1,7 [IC :1,0-3,0]), mais il n’y a pas d’association avec l’asthme allergique. Chez les femmes asthmatiques, l’usage d’eau de javel était associé à une neutrophilie plus élevée.ConclusionA l’issue de ce travail, nous avons montré que le risque accru d’asthme chez la femme persistait au-delà de la période reproductive, et que la chute des hormones sexuelles féminines à la ménopause n’était pas indépendamment associée à l’incidence de l’asthme. D’autres facteurs, dont l’interaction entre les facteurs métaboliques et hormonaux, et les expositions genre-dépendantes pourraient expliquer l’excès de risque d’asthme non-allergique chez la femme
IntroductionThe reason for the higher asthma incidence in women during the reproductive years is still unknown. The gender switch in asthma incidence around puberty has been put forward to suggest a role of female sex hormones in asthma. However, there are limited findings in the natural history of asthma after age of 50 and whether the transition through menopause is associated with change in asthma incidence is unknown.The objective of this thesis was to investigate the natural history of allergic and non-allergic asthma in women in term of incidence, evolution with aging (menopause) and risk factors of asthma in women with a focus on the gender differences.Materials and methodsThe gender differences in allergic and non-allergic asthma was assess in 2.125 men and 2.298 women free of asthma at baseline (aged 20-44) followed for 20 years as a part of the European Community Respiratory Health Survey (ECRHS). The association between asthma incidence and menopausal status was assessed in 67,872 women free of asthma at baseline (aged 41-68 years) and regularly followed-up as a part of the French E3N cohort. Association between the domestic use of cleaning bleach and allergic and non-allergic asthma have been assess in 607 women (mean age 44 years) participating in the first follow-up of the Epidemiological Study on the Genetics and Environment of Asthma (EGEA). Allergy was define by a positive skin prick test.ResultsWith data from ECRHS we showed that the incidence of non-allergic asthma was higher in women as compared to men (Odds Ratio (OR): 4.0 [95% Confident Interval (CI95%): 2.5-6.5]. The female predominance was less marked for allergic asthma (OR :1.7 [IC :1.0-2.7]). However, in asthmatics there was no gender differences in the progression of asthma. With data from E3N we showed that among normal-weight women, there was no difference in asthma incidence between premenopausal and naturally postmenopausal women of the same age (Hazard Ratio(HR) :1,1 [0,8-1,3]). However, in obese women, the transition through menopause (HR: 1.9[1.0-3.7]) and the later period after menopause (HR:2.1 [1.1-4.1]) were associated with a higher asthma incidence. Surgically post-menopausal women were also at increased risk of developing asthma (HR:1.33[95%CI 1.01-1.75]).In EGEA, only 2% of men and 37% of women reported using bleach weekly for home cleaning. Women using bleach were at increased risk for non-allergic asthma (OR 3.3; 95%CI 1.5-7.1), and more particularly for non-allergic adult-onset asthma (OR 4.9; 95%CI 2.0-11.6). No association was observed for allergic asthma. Among women with asthma, frequent use of bleach was significantly associated with higher blood neutrophil cell counts.ConclusionGender differences in asthma incidence still persist in older adults however, changes in female sex hormones level occurring around natural menopause was not independently associated with change in asthma incidence in women. Our findings do not support the hypothesis that female sex hormones are independently associated with asthma incidence. The interaction between metabolic and hormonal factors and the gender difference in environment factor such as bleach use might explain the gender switch in asthma incidence around puberty and the female predominance in asthma more particularly for non-allergic asthma in adulthood
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Ferreira, Manuel A. R. "Genetic risk factors for allergic asthma in Australian families /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19164.pdf.

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McCants, Kellie M. "Factors affecting treatment regimen adherence in children and adolescents with asthma." Connect to this title online, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1041866923.

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Thesis (Ph. D.)--Ohio State University, 2004.
Title from first page of PDF file. Document formatted into pages; contains xv, 134 p.; also includes graphics Includes bibliographical references (p. 101-108). Available online via OhioLINK's ETD Center
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Deverell, Marie. "Risk factors for persistent asthma in adolescents : a community based longitudinal birth cohort." University of Western Australia. School of Paediatrics and Child Health, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0171.

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[Truncated abstract] Asthma is a chronic and complex disorder and despite our increase in the understanding of the genetics, pathology and mechanisms underlying asthma a gold standard definition of asthma does not exist. A criterion for recognising and diagnosing asthma in epidemiological studies is crucial in order to determine risk factors for disease. Prospective longitudinal birth cohort studies have increased our understanding of the natural history and risk factors for asthma, yet we are still not able to accurately predict which children will go on to have asthma as adults. It is during the transition from childhood to adolescence where factors underlying asthma change and the prevalence of asthma shifts between the sexes. There are inconsistencies regarding risk factors for the development and persistence of disease during this transitional period. Risk factors predicting the development and persistence of asthma and intermediate phenotypes (BHR, airway inflammation and atopy) may be influenced by gender and risk factors predicting disease may differ between childhood and adolescence. Aims 1. To identify risk factors for Asthma, BHR and Atopy at 14yrs of age. 2. To determine risk factors for persistence of asthma between 6 and 14 years. 3. To examine the influence of gender on risk factors during adolescence. Method The West Australian Pregnancy Cohort is a longitudinal birth cohort. The cohort initially consisted of 2868 live births with follow-ups at 1, 2, 3, 6, 8, 10 and 14 years of V age. ... Strong associations were seen with BHR and new diagnosis of wheeze and asthma in VI teenagers. Interestingly having either a cat or dog inside was protective for persistence of disease; in particular stronger associations were seen in teenage girls not in boys. During this transitional period the risk factors for asthma and intermediate phenotypes differ between the sexes. Different mechanisms are likely to be involved in determining asthma in boys and girls during adolescence and shed new light on the recognised switch in the gender balance in asthma prevalence from the male predominance in childhood to the female predominance in adult life. Our understanding of the natural course of disease from the prenatal period to adulthood and the identification of the various asthma phenotypes has the potential to change prognosis and planning of therapeutic strategies. Identifying those at high risk for persistence of disease in the early stages of life will allow therapeutic interventions to be more appropriately targeted.
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Uddenfeldt, Monica. "A Longitudinal Study of Asthma : Risk Factors and Prognosis." Doctoral thesis, Uppsala universitet, Arbets- och miljömedicin, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-133218.

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The aim of this thesis was to identify risk factors for the onset of adult asthma. Other objectives were to study determinants of smoking habits and the association between sensitization and outcome of asthma. In 1990, a questionnaire was distributed to 12,732 individuals from three age groups (16, 30-39 and 60-69 years) in two counties of Sweden. In a second phase, 2538 subjects who had reported respiratory symptoms and 600 controls were invited to clinical investigations, 81% participated. At the follow-up in 2003 subjects of the remaining cohort (11,282) were re-invited. Analyses are based on the 67% (n=7563) who responded to both questionnaires 1990 and 2003. In 2003, 17.2% of the young adults, 11.4% of the middle-aged and 10.3% of the elderly reported having, or having had, asthma. A total of 791 subjects reported onset of asthma during the 13-year study period. Lifestyle factors such as smoking, obesity, hard physical training and a low consumption of fruit and fish were constant risk factors for onset of asthma after adjusting for socioeconomic group. A smoker’s risk of asthma onset was increased by 37%. The impact of risk factors differed between the age-groups. BMI had a significantly higher impact in the middle-aged and elderly. In subjects participating in the clinical investigations in 1990, sensitization to pets, were determinants of both persistent asthma and onset of asthma in 2003. The risk for persistent asthma was threefold. The risk for onset of asthma was more than doubled. Smoking at baseline in 1990 was the strongest determinant of being a smoker in 2003. Allergic sensitization and clinically verified asthma were not associated with smoking habits in 2003. No differences in changing smoking habits could be identified between smokers with or without asthma. In conclusion, modifiable lifestyle factors are important risk factors for adult onset asthma. The co-occurrence and interplay between asthma and cigarette smoking is still puzzling.
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Books on the topic "Factors and asthma"

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Eisner, Mark D. Environmental factors and asthma: What we learned from epidemiological studies. Philadelphia: Saunders, 2008.

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Harver, Andrew. Asthma, health, and society: A public health perspective. New York: Springer, 2010.

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Asthma, health, and society: A public health perspective. New York: Springer, 2010.

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American, Thoracic Society Workshop (1997 Montreal Canada). Immunobiology of asthma and rhinitis: Pathogenic factors and therapeutic options : American Thoracic Society Workshop summary : Montreal, Canada, June 1997. [New York, N.Y.]: American Thoracic Society, 1999.

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Asthma sourcebook: Basic consumer health information about allergic, exercise-induced, occupational, and other types of asthma, including facts about causes, risk factors, symptoms, and diagnostic tests and featuring details about treating asthma with medication and other therapies ... 3rd ed. Detroit, MI: Omnigraphics, Inc., 2011.

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Hodgson, Gabrielle. Statistical snapshots of people with asthma in Australia 2001. Canberra: Australian Institute of Health and Welfare, 2007.

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1946-, Stute W., and Seminar on Empirical Processes (1985 : Düsseldorf, Germany), eds. Seminar on Empirical Processes. Basel: Birkhäuser Verlag, 1987.

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Carol, Svec, ed. The Inflammation cure: How to combat the hidden factor behind heart disease, arthritis, asthma, diabetes, Alzheimer's disease, osteoporosis, and other diseases of aging. Chicago: Contemporary Books, 2004.

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Predine-Hug, François. L'odontologiste face à: Asthme, cirrhose, diabète, endocardite, épilepsie, grossesse, hémophilie, insuffisance cardiaque, AAP, AC, SIDA, toxicomanies : conduites à tenir. Paris: Éd. SID-Groupe EDP sciences, 2011.

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Cory, Stella. Prevalence of selected risk behaviors and chronic diseases and conditions: Steps communities, United States, 2006-2007. Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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Book chapters on the topic "Factors and asthma"

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Bianco, Sebastiano, Mariella Robuschi, Adriano Vaghi, Gabriella Gambaro, Stefano Spagnotto, Maria Grazia Pieroni, Rosa Metella Refini, and Piersante Sestini. "Osmotic and Ionic Factors in Bronchial Responsiveness." In Asthma Treatment, 145–54. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3446-4_12.

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Cookson, William. "Genetic Factors in Asthma." In Advances in Experimental Medicine and Biology, 55–60. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5855-2_8.

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Bossi, R., P. Banfi, and F. Berni. "Psychogenic Factors in Asthma." In Methods in Asthmology, 77–85. Milano: Springer Milan, 1993. http://dx.doi.org/10.1007/978-88-470-2263-8_6.

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Jeebhay, Mohamed F., Paul K. Henneberger, Nicole Le Moual, Jean-Luc Malo, and Susan M. Tarlo. "Disease Occurrence and Risk Factors." In Asthma in the Workplace, 15–34. 5th ed. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003000624-3.

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Heederik, Dick, and Torben Sigsgaard. "Epidemiology and risk factors of occupational respiratory asthma and occupational sensitization." In Occupational Asthma, 17–32. Basel: Birkhäuser Basel, 2010. http://dx.doi.org/10.1007/978-3-7643-8556-9_2.

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Martinez, Fernando D. "Maternal Risk Factors in Asthma." In Ciba Foundation Symposium 206 - The Rising Trends in Asthma, 233–43. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470515334.ch15.

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Peden, David B. "Genetic and Environmental Factors in Asthma." In Asthma, Health and Society, 43–57. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-78285-0_3.

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Shah, Tejas, and Kalpana Joshi. "Analysis of FOXO3a Gene Polymorphism Associated with Asthma." In FOXO Transcription Factors, 259–66. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8900-3_22.

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Hew, Mark, and Liam G. Heaney. "The contribution of comorbidities, psychosocial factors and adherence to the presentation of severe asthma." In Severe Asthma, 30–47. Sheffield, United Kingdom: European Respiratory Society, 2019. http://dx.doi.org/10.1183/2312508x.10022718.

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Goodwin, R. D. "Asthma and Anxiety Disorders." In Asthma: Social and Psychological Factors and Psychosomatic Syndromes, 51–71. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000073780.

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Conference papers on the topic "Factors and asthma"

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van der Veer, Tjeerd, Marleen Bakker, Lieke Kamphuis, Alain Vincent François Dubois, Rogier A. S. Hoek, Joachim G. J. V. Aerts, and Menno E. Van Der Eerden. "Bronchiectasis: prevalance of asthma diagnosis and asthma-associated factors." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3329.

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Backman, Helena, Caroline Stridsman, Sven-Arne Jansson, Linnea Hedman, Hannu Kankaanranta, Anne Lindberg, Bo Lundback, and Eva Rönmark. "Risk factors for severe asthma among adults with asthma." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.4642.

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Maio, Sara, Sandra Baldacci, Sonia Cerrai, Giuseppe Sarno, Megon Bresciani, Manuela Latorre, Nicola Murgia, et al. "Late-asthma onset and associated factors." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4580.

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Dos Santos Carvalho, Filipe Gonçalves, Clara Padró Casas, María Basagaña Torrento, Marisa Rivera Ortun, María Del Mar Martínez Colls, Carlos Pollan Guisasola, Alicia Francoso Vicente, et al. "Prognostic factors in severe eosinophilic asthma." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa5064.

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Corsico, Angelo Guido, Francesca Locatelli, Lucia Cazzoletti, Amelia Grosso, Fedrica Albicini, Erica Gini, Vanessa Ronzoni, Eti Maria Giulia Di Vincenzo, and Isa Cerveri. "Asthma control during pregnancy and associated factors." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4593.

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de Nijs, Selma B., M. Amelink, B. vd Berg, F. H. Krouwels, Peter J. Sterk, E. J. Weersink, and Elisabeth H. D. Bel. "Risk Factors Of Asthma Onset In Adulthood." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3968.

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Diab, N., S. Riley, and M. B. Stanbrook. "Factors Associated with Exacerbations in Patients with Asthma, COPD and Asthma-COPD Overlap." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4541.

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Pochini, Alma, Ben M. Williams, Hasanboy M. Isomitdinov, and Gongzhu Hu. "A Data Mining Analysis of Asthma Risk Factors." In 2015 3rd International Conference on Applied Computing and Information Technology/2nd International Conference on Computational Science and Intelligence (ACIT-CSI). IEEE, 2015. http://dx.doi.org/10.1109/acit-csi.2015.101.

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Martinez Rivera, Carlos, Clara Padró Casas, Maria Basagaña Torrentó, Marisa Rivera Ortún, Carlos Pollán Guisasola, Jose Tomas Navarro Ferrando, Eva Martinez Caceres, Aina Teniente Serra, Alicia Francoso Vicente, and Maria Del Mar Martinez Colls. "Prognostic factors in severe eosinophilic asthma in children." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa1290.

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Wong, Katy, Jing Li, Ting Fan Leung, N. S. Zhong, and Gary Wong. "Protective factors against childhood asthma in rural China." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.oa1475.

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Reports on the topic "Factors and asthma"

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Gindi, Renee. Health, United States, 2019. Centers for Disease Control and Prevention (U.S.), 2021. http://dx.doi.org/10.15620/cdc:100685.

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Abstract:
Health, United States, 2019 is the 43rd report on the health status of the nation and is submitted by the Secretary of the Department of Health and Human Services to the President and the Congress of the United States in compliance with Section 308 of the Public Health Service Act. This report was compiled by the National Center for Health Statistics (NCHS) of the Centers for Disease Control and Prevention (CDC). The Health, United States series presents an annual overview of national trends in key health indicators. The 2019 report presents trends and current information on selected measures of morbidity, mortality, health care utilization and access, health risk factors, prevention, health insurance, and personal health care expenditures in a 20-figure chartbook. The Health, United States, 2019 Chartbook is supplemented by several other products including Trend Tables, an At-a-Glance table, and Appendixes available for download on the Health, United States website at: https://www.cdc.gov/nchs/hus/ index.htm. The Health, United States, 2019 Chartbook contains 20 figures and 20 tables on health and health care in the United States. Examining trends in health informs the development, implementation, and evaluation of health policies and programs. The first section (Figures 1–13) focuses on health status and determinants: life expectancy, infant mortality, selected causes of death, overdose deaths, suicide, maternal mortality, teen births, preterm births, use of tobacco products, asthma, hypertension, heart disease and cancer, and functional limitations. The second section (Figures 14–15) presents trends in health care utilization: use of mammography and colorectal tests and unmet medical needs. The third section (Figures 16–17) focuses on health care resources: availability of physicians and dentists. The fourth section (Figures 18–20) describes trends in personal health care expenditures, health insurance coverage, and supplemental insurance coverage among Medicare beneficiaries. The Highlights section summarizes major findings from the Chartbook. Suggested citation: National Center for Health Statistics. Health, United States, 2019. Hyattsville, MD. 2021.
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