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Journal articles on the topic 'Facial flushing'

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Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2022

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1

Curran, Charles F. "Doxorubicin-Associated Facial Flushing." Archives of Dermatology 128, no. 10 (October 1, 1992): 1408. http://dx.doi.org/10.1001/archderm.1992.01680200120029.

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2

Zalzal, George H. "Congenital gustatory facial flushing." Otolaryngology–Head and Neck Surgery 104, no. 6 (June 1991): 878–80. http://dx.doi.org/10.1177/019459989110400620.

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3

Curran, C. F. "Doxorubicin-associated facial flushing." Archives of Dermatology 128, no. 10 (October 1, 1992): 1408b—1408. http://dx.doi.org/10.1001/archderm.128.10.1408b.

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4

Salleras i Redonnet, Montserrat, and Nuria Lamas Doménech. "Tratamiento del rubor facial (flushing)." Piel 29, no. 9 (November 2014): 587–91. http://dx.doi.org/10.1016/j.piel.2014.03.013.

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5

Mc Cormack, Orla, and John V. Reynolds. "Intermittent Facial Flushing and Diarrhea." New England Journal of Medicine 371, no. 3 (July 17, 2014): 260. http://dx.doi.org/10.1056/nejmicm1314969.

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6

Dizon, Maria Victoria C. "Localized Facial Flushing in Infancy." Archives of Dermatology 133, no. 9 (September 1, 1997): 1143. http://dx.doi.org/10.1001/archderm.1997.03890450093011.

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7

Tur, E., K. S. Ryatt, and H. I. Maibach. "Idiopathic Recalcitrant Facial Flushing Syndrome." Dermatology 181, no. 1 (1990): 5–7. http://dx.doi.org/10.1159/000247849.

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8

Cobb, Alistair R. M., Michael Vourvachis, Jahangir Ahmed, Michelle Wyatt, David Dunaway, and Richard Hayward. "Aberrant facial flushing following monobloc fronto-facial distraction." Journal of Cranio-Maxillofacial Surgery 43, no. 8 (October 2015): 1511–15. http://dx.doi.org/10.1016/j.jcms.2015.07.005.

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9

Arpornsuwan, Manote, and Matinun Arpornsuwan. "Invisible Facial Flushing in Two Cases of Dengue Infection and Influenza Detected by PC Program and Smartphone App: Decorrelation Stretching and K-Means Clustering." Case Reports in Infectious Diseases 2020 (February 13, 2020): 1–6. http://dx.doi.org/10.1155/2020/8790130.

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We report the two cases of dengue infection and influenza with invisible facial flushing. The invisible facial flushing can be detected and visible by the Manote and Matinun (M&M) technique using PC program and smartphone app (decorrelation stretching and K-Means clustering). The unique patterns of facial flushing in the patients with high fever provide a clue to the diagnosis of dengue infection and influenza. This new innovative method could detect dengue infection and influenza earlier in the patients with high fever.
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10

Ezzo, Danielle C., and Priti N. Patel. "Facial flushing associated with duloxetine use." American Journal of Health-System Pharmacy 64, no. 5 (March 1, 2007): 495–96. http://dx.doi.org/10.2146/ajhp060069.

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11

Kasha, Edwin E., and Arthur L. Norins. "Scombroid fish poisoning with facial flushing." Journal of the American Academy of Dermatology 18, no. 6 (June 1988): 1363–65. http://dx.doi.org/10.1016/s0190-9622(88)80119-1.

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12

DRUMMOND, PETER D. "Facial flushing during provocation in women." Psychophysiology 36, no. 3 (May 1999): 325–32. http://dx.doi.org/10.1017/s0048577299980344.

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13

Lübbe, Jann, and Maria Milingou. "Tacrolimus Ointment, Alcohol, and Facial Flushing." New England Journal of Medicine 351, no. 26 (December 23, 2004): 2740. http://dx.doi.org/10.1056/nejmicm040139.

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14

Lipsett, Susan C. "Young Boy With Unilateral Facial Flushing." Annals of Emergency Medicine 69, no. 6 (June 2017): 688–736. http://dx.doi.org/10.1016/j.annemergmed.2016.12.008.

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15

Boling, Bryan, Christopher Key, Justin Wainscott, and Annette Rebel. "Harlequin Syndrome as a Complication of Epidural Anesthesia." Critical Care Nurse 34, no. 3 (June 1, 2014): 57–61. http://dx.doi.org/10.4037/ccn2014870.

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Harlequin syndrome is a rare neurological condition that results in unilateral facial flushing and sweating. Although the syndrome is generally a benign condition with complete resolution if appropriate treatment is initiated, unilateral facial flushing can be a sign of several serious conditions and should be thoroughly investigated. Sudden onset of facial flushing related to harlequin syndrome developed in a patient who had bilateral lung transplant with postoperative epidural anesthesia for pain control. Differential diagnosis includes neurovascular disease (acute stroke), malignant neoplasm of brain or lung, Horner syndrome, idiopathic hyperhidrosis, and Frey syndrome. Harlequin syndrome is often easily treated by discontinuing the anesthetic or adjusting placement of the epidural catheter. (Critical Care Nurse. 2014;34[3]:57–61)
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16

Goadsby, Peter J., Lars Edvinsson, and Rolf Ekman. "Cutaneous Sensory Stimulation Leading to Facial Flushing and Release of Calcitonin Gene-Related Peptide." Cephalalgia 12, no. 1 (February 1992): 53–56. http://dx.doi.org/10.1046/j.1468-2982.1992.1201053.x.

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A patient is described with a 17-year history of intractable left-sided facial pain. The pain occurred daily in 5 sec spasms to a maximum of one every 2–3 min and was restricted to the left upper face. It was associated with rhinorrhoea on the left and often with ipsilateral facial flushing. Conventional therapy, including carbamazepine, baclofen and three posterior fossa explorations, had not provided lasting relief. Local facial stimulation by tapping a painful trigger point led to both pain and flushing of the face ipsilaterally. During this flushing, blood was collected and assayed using sensitive radioimmunoassays for several neuropeptides (neuropeptide Y, substance P, vasoactive intestinal polypeptide and calcitonin gene-related peptide). A marked (119%) increase in calcitonin gene-related peptide was noted in the external jugular vein blood ipsilaterally during the flushing with no change in the other peptides measured. To quantitate the effect of calcitonin gene-related peptide on human extracranial vessels, standard pharmacological procedures were used to examine the potency of the peptide as a vasodilator of human facial artery. The 1C50 of calcitonin gene-related peptide for the prostaglandin F2a -precontracted human facial artery was 10-9 mol/1. The relevance of these observations to the clinical problem of migraine is considered.
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17

Saito, H. "Congenital Horner's syndrome with unilateral facial flushing." Journal of Neurology, Neurosurgery & Psychiatry 53, no. 1 (January 1, 1990): 85–86. http://dx.doi.org/10.1136/jnnp.53.1.85.

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18

Lolar, Sara, and Paul Chrobak. "What is causing this manʼs facial flushing?" Journal of the American Academy of Physician Assistants 30, no. 2 (February 2017): 55–57. http://dx.doi.org/10.1097/01.jaa.0000511801.98996.ff.

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19

Lam, Joseph, and Julie Milligan. "Case 2: Unilateral facial flushing with eating." Paediatrics & Child Health 14, no. 6 (July 1, 2009): 371–73. http://dx.doi.org/10.1093/pch/14.6.371a.

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20

Ganti, Somshekar, Richard Milton, Leslie Davidson, and Andrew Thorpe. "Facial Flushing Due to Recurrent Bronchial Carcinoid." Annals of Thoracic Surgery 83, no. 3 (March 2007): 1196–97. http://dx.doi.org/10.1016/j.athoracsur.2006.07.033.

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21

Humphrey, John, Gregory Black, and Laurianne Wild. "Facial Flushing with Food: The Auriculotemporal Syndrome." Journal of General Internal Medicine 28, no. 3 (August 10, 2012): 475–76. http://dx.doi.org/10.1007/s11606-012-2175-5.

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22

Hoskins, P. J., P. G. Wiles, H. P. Volkmann, and D. A. Pyke. "Chlorpropamide alcohol flushing: A normal response?" Clinical Science 73, no. 1 (July 1, 1987): 77–80. http://dx.doi.org/10.1042/cs0730077.

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1. The relationship between chlorpropamide alcohol flushing and non-insulin dependent diabetes remains uncertain. It is known, however, that the frequency of facial flushing with alcohol and the temperature response depend upon both the plasma level of chlorpropamide and the starting facial temperature [10]. 2. We tested 23 young adult non-diabetic subjects with 8 g of ethanol after a dose of chlorpropamide 250 mg twice daily for 2 days or a placebo, in a double blind, cross-over manner. Previously, nine other subjects had participated in a pilot study to assess the safety of the chlorpropamide dose and to ensure that adequate plasma chlorpropamide levels were achieved. 3. No subject was negative for chlorpropamide alcohol flushing, as defined by the following criteria: (1) facial temperature rise of 35% or more of maximum possible rise, (2) observer assessment or (3) subject assessment. In 26 of the total 32 subjects, all three criteria were fulfilled. 4. Thus, among young, healthy non-diabetic adults chlorpropamide alcohol flushing would appear to be a normal phenomenon.
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23

Yokoyama, Akira, Tetsuji Yokoyama, Mitsuru Kimura, Sachio Matsushita, and Masako Yokoyama. "Combinations of alcohol-induced flushing with genetic polymorphisms of alcohol and aldehyde dehydrogenases and the risk of alcohol dependence in Japanese men and women." PLOS ONE 16, no. 7 (July 26, 2021): e0255276. http://dx.doi.org/10.1371/journal.pone.0255276.

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Objective The risk of alcohol dependence (AD) in Japanese men and women was evaluated according to combinations of alcohol flushing and aldehyde dehydrogenase-2 (ALDH2, rs671) and alcohol dehydrogenase-1B (ADH1B, rs1229984) genotypes, all of which are known to determine AD susceptibility in Asians. Previous studies have focused on men, since women account for a smaller proportion of AD subjects. Methods Case control studies were conducted between 3721 male and 335 female AD Japanese and 610 male and 406 female controls who were asked about their current or former tendency to experience facial flushing after drinking a glass of beer and underwent ALDH2 and ADH1B genotyping. The time at which alcohol-induced facial flushing tendencies had disappeared in former-flushing AD subjects was also evaluated. Results Current alcohol flushing, the inactive ALDH2*1/*2 genotype, and the fast-metabolizing ADH1B*2 allele were less frequently found in the AD groups. Although alcohol flushing was strongly influenced by the ALDH2 and ADH1B genotypes, multiple logistic model showed that never or former flushing and the genotype combinations were independent strong risk factors of AD in men and women. Never or former flushing (vs. current flushing) markedly increased the odds ratios of AD in carriers of each of the ALDH2 and ADH1B genotype combinations. The temporal profiles for drinking and flushing in former-flushing AD subjects revealed that the flushing response disappeared soon after or before the start of habitual drinking during young adulthood, regardless of the ALDH2 genotype. Conclusion Although alcohol flushing is influenced by the ALDH2 and ADH1B genotypes, constitutional or acquired flushing tolerance is an independent susceptibility trait for AD. The combination of the alcohol flushing status and the ALDH2 and ADH1B genotypes can provide a better new strategy for AD risk assessment than the alcohol flushing status alone or the genotypes alone in Asian men and women.
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24

de Coo, IF, LA Wilbrink, J. Haan, MD Ferrari, and GM Terwindt. "Evaluation of the new ICHD-III beta cluster headache criteria." Cephalalgia 36, no. 6 (September 22, 2015): 547–51. http://dx.doi.org/10.1177/0333102415607856.

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Aim In the revised criteria of the International Classification of Headache Disorders (ICHD-III beta) the following items are added to the diagnostic criteria of cluster headache: ipsilateral sensation of fullness in the ear and ipsilateral forehead/facial flushing. We evaluated the possible additional value of these symptoms for diagnosing cluster headache. Methods In this cross-sectional cohort study of (potential) cluster headache patients we investigated these additional symptoms using a Web-based questionnaire. Patients not fulfilling the ICHD-II criteria for cluster headache but fulfilling the ICHD-III beta criteria were interviewed. Results Response rate was 916/1138 (80.5%). Of all 573 patients with cluster headache according to ICHD-II criteria, 192 (33.5%) reported ipsilateral ear fullness and 113 (19.7%) facial flushing during attacks. There was no difference in reporting ipsilateral ear fullness and facial flushing between patients who received a diagnosis of cluster headache and patients who did not. None of the patients who did not fulfill all ICHD-II criteria could be categorized as cluster headache according to the ICHD-III beta criteria. Conclusion The results of this study do not support the addition of ear fullness and facial flushing to the new ICHD-III beta criteria.
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25

Deng, Min, and Susan Nedorost. "Facial Flushing: An Uncommon Presentation of Serotonin Toxicity." Dermatitis 20, no. 5 (September 2009): 296–97. http://dx.doi.org/10.2310/6620.2009.09016.

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26

Pattrick, M., and M. Doherty. "Facial flushing after intra-articular injection of steroid." BMJ 295, no. 6610 (November 28, 1987): 1380. http://dx.doi.org/10.1136/bmj.295.6610.1380.

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27

Kang, Jennifer H., Muhammad Shahzad Zafar, and Klaus-Georg E. Werner. "Child Neurology: An infant with episodic facial flushing." Neurology 91, no. 6 (August 6, 2018): 278–81. http://dx.doi.org/10.1212/wnl.0000000000005949.

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Congenital harlequin syndrome is rare dysautonomia of the face most often reported in adults and rarely in infants and children. It is a diagnosis of exclusion and a seemingly benign condition. We report a case of a 6-month-old girl with episodic unilateral and bilateral facial flushing provoked upon awakening and resolved with sleeping with associated autonomic features consistent with harlequin syndrome. This is followed by a review of cases identified regarding this condition in infants and children.
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28

Dhar, S., and A. J. Kanwar. "Facial Flushing – A Side Effect of Pulse Therapy." Dermatology 188, no. 4 (1994): 332. http://dx.doi.org/10.1159/000247180.

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29

Yuraitis, Melanie, and Carolyn I. Jacob. "Botulinum Toxin for the Treatment of Facial Flushing." Dermatologic Surgery 30, no. 1 (January 2004): 102–4. http://dx.doi.org/10.1111/j.1524-4725.2004.30017.x.

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30

YURAITIS, MELANIE, and CAROLYN I. JACOB. "Botulinum Toxin for the Treatment of Facial Flushing." Dermatologic Surgery 30, no. 1 (January 2004): 102–4. http://dx.doi.org/10.1097/00042728-200401000-00028.

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31

Negro, Juan José, José Hernán Sarasola, Fernando Fariñas, and Irene Zorrilla. "Function and occurrence of facial flushing in birds." Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 143, no. 1 (January 2006): 78–84. http://dx.doi.org/10.1016/j.cbpa.2005.10.028.

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32

Scala, Jacopo, Aleksandra Vojvodic, Petar Vojvodic, Tatjana Vlaskovic-Jovicevic, Zorica Peric-Hajzler, Dusica Matovic, Sanja Dimitrijevic, et al. "Botulin Toxin Use in Rosacea and Facial Flushing Treatment." Open Access Macedonian Journal of Medical Sciences 7, no. 18 (August 30, 2019): 2985–87. http://dx.doi.org/10.3889/oamjms.2019.784.

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Botulinum toxin (BTX) is a neurotoxin derived from the Clostridium botulinum bacterium that inhibits the release of acetylcholine at the neuromuscular junction level whose effects has been used for many years to treat a variety of muscular/neuromuscular conditions and more recently also for cosmetic use. BTX has experimented in some dermatological conditions, which include Rosacea and facial flushing treatment with good results. The complex mechanism underlying those results is not completely understood but was proposed a release inhibition of acetylcholine from peripheral autonomic nerves of the cutaneous vasodilatory system combined with the blockade substance P and calcitonin gene-related peptide (CGRP) thus modulating blood vessel dilatation. We analysed the published data on BTX off label applications rosacea and flushing retrieved from PubMed.
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33

Touch, Khun. "Facial flushing and blood pressure among Cambodians with T2D." Diabetes Research and Clinical Practice 120 (October 2016): S86. http://dx.doi.org/10.1016/s0168-8227(16)31122-6.

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34

Golding, D. N. "Points: Facial flushing after intra-articular injection of steroid." BMJ 296, no. 6616 (January 16, 1988): 213. http://dx.doi.org/10.1136/bmj.296.6616.213-g.

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35

Bae, Y., J. Kim, S. Kim, J. Jung, S. Yoon, and Y. Seo. "Relationship between alcohol-related facial flushing and carotid atherosclerosis." Atherosclerosis 275 (August 2018): e244. http://dx.doi.org/10.1016/j.atherosclerosis.2018.06.775.

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36

Dizon, M. V. "Localized facial flushing in infancy. Auriculotemporal nerve (Frey) syndrome." Archives of Dermatology 133, no. 9 (September 1, 1997): 1143–45. http://dx.doi.org/10.1001/archderm.133.9.1143.

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37

Lee, Sami, Jong-Sung Kim, Jin-Gyu Jung, Mi-Kyeong Oh, Tae-Heum Chung, and Jihan Kim. "Korean Alcohol Guidelines for Moderate Drinking Based on Facial Flushing." Korean Journal of Family Medicine 40, no. 4 (July 20, 2019): 204–11. http://dx.doi.org/10.4082/kjfm.19.0059.

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38

Wilkin, Jonathan K. "Why is flushing limited to a mostly facial cutaneous distribution?" Journal of the American Academy of Dermatology 19, no. 2 (August 1988): 309–13. http://dx.doi.org/10.1016/s0190-9622(88)70177-2.

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39

Drummond, Peter D. "Correlates of facial flushing and pallor in anger-provoking situations." Personality and Individual Differences 23, no. 4 (October 1997): 575–82. http://dx.doi.org/10.1016/s0191-8869(97)00077-9.

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40

de la Torre, C. "Alcohol Intolerance With Facial Flushing Due to Topical Pimecrolimus Treatment." Actas Dermo-Sifiliográficas (English Edition) 99, no. 5 (2008): 422–23. http://dx.doi.org/10.1016/s1578-2190(08)70282-8.

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41

Bowman, Nena, and Donna Seger. "Facial flushing and rash without urticaria after N-acetylcysteine administration." Visual Journal of Emergency Medicine 15 (April 2019): 100561. http://dx.doi.org/10.1016/j.visj.2019.100561.

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42

Zhang, Jing, Sunfu Zhang, Yanlin Song, Guangzhi Ma, Yu Meng, Zengpanpan Ye, Xuelei Ma, and Ming Liu. "Facial flushing after alcohol consumption and the risk of cancer." Medicine 96, no. 13 (March 2017): e6506. http://dx.doi.org/10.1097/md.0000000000006506.

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43

DRUMMOND, PETER D., and PHILIP M. FINCH. "REFLEX CONTROL OF FACIAL FLUSHING DURING BODY HEATING IN MAN." Brain 112, no. 5 (1989): 1351–58. http://dx.doi.org/10.1093/brain/112.5.1351.

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44

Sabir, H., F. Babor, B. C. Kieseier, E. Mayatepek, and B. Assmann. "Unilateral Facial Flushing and Sweating After Physical Exercise: Harlequin Syndrome." Klinische Pädiatrie 223, no. 02 (January 26, 2011): 90–91. http://dx.doi.org/10.1055/s-0030-1269925.

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45

Drummond, P. D., and R. H. Edis. "Loss of facial sweating and flushing in Holmes-Adie syndrome." Neurology 40, no. 5 (May 1, 1990): 847. http://dx.doi.org/10.1212/wnl.40.5.847.

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46

DRUMMOND, PETER D. "The effect of adrenergic blockade on blushing and facial flushing." Psychophysiology 34, no. 2 (March 1997): 163–68. http://dx.doi.org/10.1111/j.1469-8986.1997.tb02127.x.

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47

Barbara, Giomi, Cardinali Carla, Caproni Marzia, and Fabbri Paolo. "Persistent Erythema, Telangiectases, and Facial Flushing After Carcinoid Tumor Excision." SKINmed: Dermatology for the Clinician 2, no. 4 (July 2003): 259–63. http://dx.doi.org/10.1111/j.1540-9740.2003.02245.x.

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48

Suwaki, H., and H. Ohara. "Alcohol-induced facial flushing and drinking behavior in Japanese men." Journal of Studies on Alcohol 46, no. 3 (May 1985): 196–98. http://dx.doi.org/10.15288/jsa.1985.46.196.

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49

DeSio, John M., Cynthia H. Kahn, and Carol A. Warfield. "Facial Flushing and/or Generalized Erythema After Epidural Steroid Injection." Anesthesia & Analgesia 80, no. 3 (March 1995): 617–19. http://dx.doi.org/10.1097/00000539-199503000-00034.

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50

DRUMMOND, PETER D., and JAMES W. LANCE. "FACIAL FLUSHING AND SWEATING MEDIATED BY THE SYMPATHETIC NERVOUS SYSTEM." Brain 110, no. 3 (1987): 793–803. http://dx.doi.org/10.1093/brain/110.3.793.

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