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Journal articles on the topic 'Evoked potentials'

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Published: 4 June 2021

Last updated: 29 July 2024

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1

Jacot, E., and S. Wiener-Vacher. "Potential value of vestibular evoked myogenic potentials in paediatric neuropathies." Journal of Vestibular Research 18, no. 4 (December 1, 2008): 231–37. http://dx.doi.org/10.3233/ves-2008-18406.

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Purpose: Showing the interest of vestibular evoked myogenic potentials in paediatric neurological vestibulospinal pathology detection and followup. Materials and methods: The vestibular evoked myogenic potentials testing apparatus presented is now commonly used in ENT clinics for patients from 1 month of age. Our system and protocol permits control to evoke and select the best EMG level and makes possible a comparison of data from different sides or level of stimulation or different sessions. Normal vestibular evoked myogenic potentials latencies obtained with tone bursts were remarkably stable (P: 13 +/− 0.8 ms, N: 19.6 +/− 1.6 ms). The reported case illustrates abnormal vestibular evoked myogenic potentials latencies in neuropathy. Results: A 6 y.o. child with progressive imbalance was referred to the ENT department for vestibular functional evaluation. Abnormally long latencies in the vestibular evoked myogenic potentials and neurological examination oriented the diagnosis towards Guillain-Barre syndrome and immediate referral to a neurology department. Vestibular evoked myogenic potentials also helped to monitor the neurological recovery. Conclusion: The present case shows the potential value of vestibular evoked myogenic potentials in diagnosis and evaluation of descending brainstem pathways in neuropathies like Guillain-Barre syndrome in complement to neurological evaluation.

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2

Štětkářová, Ivana. "Evoked potentials." Neurologie pro praxi 21, no. 4 (September 8, 2020): 268–74. http://dx.doi.org/10.36290/neu.2020.037.

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3

PhD, T. Allison. "Evoked Potentials." Neurology 40, no. 3, Part 1 (March 1, 1990): 565. http://dx.doi.org/10.1212/wnl.40.3_part_1.565-a.

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4

Marshall, Lawrence F. "Evoked potentials." Critical Care Medicine 19, no. 11 (November 1991): 1337. http://dx.doi.org/10.1097/00003246-199111000-00004.

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5

Lew, Henry L. "Evoked potentials." Physical Medicine and Rehabilitation Clinics of North America 15, no. 1 (February 2004): xiii—xiv. http://dx.doi.org/10.1016/s1047-9651(03)00127-x.

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6

Kraft, George H. "Evoked potentials." Physical Medicine and Rehabilitation Clinics of North America 15, no. 1 (February 2004): xi—xii. http://dx.doi.org/10.1016/s1047-9651(03)00128-1.

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7

REGAN, D. "Evoked Potentials." American Journal of Ophthalmology 102, no. 6 (December 1986): 807–8. http://dx.doi.org/10.1016/0002-9394(86)90427-7.

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8

Salvi, Richard J., William Ahroon, Samuel S. Saunders, and Sally A. Arnold. "Evoked Potentials." Ear and Hearing 8, no. 3 (June 1987): 151–56. http://dx.doi.org/10.1097/00003446-198706000-00004.

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9

Jacobson, Gary. "Evoked Potentials." Cephalalgia 12, no. 4 (August 1992): 186–87. http://dx.doi.org/10.1046/j.1468-2982.1992.1204185-2.x.

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10

Galloway, N. R. "Evoked Potentials." British Journal of Ophthalmology 71, no. 8 (August 1, 1987): 642. http://dx.doi.org/10.1136/bjo.71.8.642.

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11

Hughes, John R. "Evoked potentials." Electroencephalography and Clinical Neurophysiology/Evoked Potentials Section 68, no. 1 (January 1987): 79–80. http://dx.doi.org/10.1016/0168-5597(87)90073-6.

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12

Walker, Francis O. "Evoked potentials." Surgical Neurology 32, no. 6 (December 1989): 478. http://dx.doi.org/10.1016/0090-3019(89)90017-7.

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13

Whalen, L. R., D. W. Wheeler, R. A. LeCouteur, J. V. Yovich, L. C. Boggie, J. L. Grandy, and R. A. Kainer. "Sensory nerve conduction velocity of the caudal cutaneous sural and medial cutaneous antebrachial nerves of adult horses." American Journal of Veterinary Research 55, no. 7 (July 1, 1994): 892–97. http://dx.doi.org/10.2460/ajvr.1994.55.07.892.

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Summary Maximal conduction velocities of compound action potentials evoked by stimuli of 2 times threshold in the caudal cutaneous sural (ccsn) and medial cutaneous antebrachial (mcan) nerves were determined by averaging potentials evoked and recorded through percutaneous needle electrodes. Mean maximal conduction velocities of compound action potentials were: ccsn = 61.3 ± 2.0 meters/second (m/s) and mcan = 56.4 ± 2.8 m/s. To confirm accuracy of our percutaneous recordings, compound action potentials were recorded through bipolar chlorided silver electrodes from the exposed surfaces of fascicles of the ccsn and the mcan. The maximal conduction velocities of these potentials were in agreement with the conduction velocities of compound action potentials that were evoked and recorded through percutaneous needle electrodes. The specificity of stimulating and recording sites was verified by recording before and after section of the nerves. Stimuli from 3 to 5 times threshold evoked a second, longer latency, compound action potential that consisted of a variable number of components in the ccsn and mcan. The configurations and conduction velocities of the shorter latency potentials were the same as those of the single compound action potentials evoked by stimuli of 2 times threshold. Mean conduction velocities of the longer latency potentials were: ccsn = 24.4 ± 2.6 m/s and mcan = 24.5 ± 2.2 m/s. Needle electrode and direct stimulation of either the ccsn or the mcan at 3 to 5 times threshold failed to evoke contractions of limb muscles. Therefore, action potentials that contributed to the evoked compound potentials recorded in these horses arose, most likely, from afferent nerve fibers.

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14

Murray, N. "Evoked Potentials III: The Third International Evoked Potentials Symposium." Journal of Neurology, Neurosurgery & Psychiatry 52, no. 6 (June 1, 1989): 815–16. http://dx.doi.org/10.1136/jnnp.52.6.815-b.

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15

Sedgwick, E. "Evoked Potentials II. The Second International Evoked Potentials Symposium." Journal of Neurology, Neurosurgery & Psychiatry 48, no. 12 (December 1, 1985): 1312. http://dx.doi.org/10.1136/jnnp.48.12.1312.

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16

Celesia, G. G. "Evoked visual potentials." Cleveland Clinic Journal of Medicine 52, no. 2 (June 1, 1985): 221–22. http://dx.doi.org/10.3949/ccjm.52.2.221.

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17

Aoyagi, Masaru. "Auditory Evoked Potentials." Equilibrium Research 69, no. 3 (2010): 113–26. http://dx.doi.org/10.3757/jser.69.113.

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18

Noordeen, M. H. H., and B. A. Taylor. "Somatosensory Evoked Potentials." Journal of Bone and Joint Surgery-American Volume 82, no. 10 (October 2000): 1517–18. http://dx.doi.org/10.2106/00004623-200010000-00042.

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19

Weinstein, Stuart L. "Somatosensory Evoked Potentials." Journal of Bone and Joint Surgery-American Volume 82, no. 10 (October 2000): 1518. http://dx.doi.org/10.2106/00004623-200010000-00043.

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20

Hyde, Martyn L. "Auditory evoked potentials." Current Opinion in Otolaryngology & Head and Neck Surgery 2 (April 1994): 177–85. http://dx.doi.org/10.1097/00020840-199404000-00015.

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21

Bajalan, A. "Advanced Evoked Potentials." Journal of Neurology, Neurosurgery & Psychiatry 52, no. 10 (October 1, 1989): 1220. http://dx.doi.org/10.1136/jnnp.52.10.1220-a.

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22

Rönnberg, Jerker, Stig Arlinger, Björn Lyxell, and Catharina Kinnefors. "Visual Evoked Potentials." Journal of Speech, Language, and Hearing Research 32, no. 4 (December 1989): 725–35. http://dx.doi.org/10.1044/jshr.3204.725.

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This study investigated the putative relationship between visual evoked potentials (VEPs) and specific aspects of speechreading. The nature and constraints of the relationship between VEPs and cognitive functioning was also examined. The original finding of Shepherd, DeLavergne, Frueh, and Clobridge (1977) that visual-neural speed (VN 130) predicts speechreading skill was not replicated. However, the picture is rather complex in that we find significant correlations for some context-free word discrimination and sign-alphabet testing conditions. These correlations occur only for the VN 130/P 200 peak-to-peak amplitude measure, not for neural speed. Nevertheless, visual-neural speed (VN 130 and P 200) was relevant to certain aspects of long-term memory access (i.e., letter matching, Posner & Mitchell, 1967) and to complex short-term memory function (i.e., reading span, Baddeley, Logie, Nimmo-Smith, & Brereton, 1985).

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23

Colebatch, James G. "Vestibular evoked potentials." Current Opinion in Neurology 14, no. 1 (February 2001): 21–26. http://dx.doi.org/10.1097/00019052-200102000-00004.

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24

Nehamkin, Sheryl, Michael Windom, and Tanvir U. Syed. "Visual Evoked Potentials." American Journal of Electroneurodiagnostic Technology 48, no. 4 (December 2008): 233–48. http://dx.doi.org/10.1080/1086508x.2008.11079688.

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25

Celesia, Gastone G. "Somatosensory Evoked Potentials." Journal of Clinical Neurophysiology 2, no. 1 (January 1985): 77–82. http://dx.doi.org/10.1097/00004691-198501000-00005.

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26

Gilmore, Robin L. "Evoked Potentials III." Journal of Clinical Neurophysiology 7, no. 4 (October 1990): 568. http://dx.doi.org/10.1097/00004691-199010000-00013.

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27

Aminoff, Michael J., and Douglas S. Goodin. "Visual Evoked Potentials." Journal of Clinical Neurophysiology 11, no. 5 (September 1994): 493–99. http://dx.doi.org/10.1097/00004691-199409000-00004.

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28

Sohn, Young H., and Mark Hallett. "Motor evoked potentials." Physical Medicine and Rehabilitation Clinics of North America 15, no. 1 (February 2004): 117–31. http://dx.doi.org/10.1016/s1047-9651(03)00105-0.

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29

Nicholas, James F., and Satwant K. Samra. "Sensory evoked potentials." Seminars in Anesthesia, Perioperative Medicine and Pain 16, no. 1 (March 1997): 14–27. http://dx.doi.org/10.1016/s0277-0326(97)80004-9.

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30

Kalkman, Cor J. "Motor evoked potentials." Seminars in Anesthesia, Perioperative Medicine and Pain 16, no. 1 (March 1997): 28–35. http://dx.doi.org/10.1016/s0277-0326(97)80005-0.

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31

Ruth, Roger A., and Paul R. Lambert. "Auditory Evoked Potentials." Otolaryngologic Clinics of North America 24, no. 2 (April 1991): 349–70. http://dx.doi.org/10.1016/s0030-6665(20)31143-9.

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32

Aunon, J. I. "Evoked potentials research." IEEE Engineering in Medicine and Biology Magazine 11, no. 1 (March 1992): 67–68. http://dx.doi.org/10.1109/51.136135.

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33

Barber, Colin, and Thomas Blum. "Evoked Potentials III." Ear and Hearing 9, no. 4 (August 1988): 228. http://dx.doi.org/10.1097/00003446-198808000-00043.

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34

Picton, T. W. "Auditory evoked potentials." Electroencephalography and Clinical Neurophysiology 87, no. 2 (August 1993): S10. http://dx.doi.org/10.1016/0013-4694(93)90894-2.

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35

Plourde, G. "Auditory evoked potentials." Best Practice & Research Clinical Anaesthesiology 20, no. 1 (March 2006): 129–39. http://dx.doi.org/10.1016/j.bpa.2005.07.012.

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36

Gugino, Verne, and Robert J. Chabot. "SOMATOSENSORY EVOKED POTENTIALS." International Anesthesiology Clinics 28, no. 3 (1990): 154–64. http://dx.doi.org/10.1097/00004311-199002830-00005.

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37

Borges, Lawrence F. "MOTOR EVOKED POTENTIALS." International Anesthesiology Clinics 28, no. 3 (1990): 170–73. http://dx.doi.org/10.1097/00004311-199002830-00007.

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38

Nuwer, Marc R. "Somatosensory evoked potentials." Pediatric Neurology 4, no. 2 (March 1988): 129. http://dx.doi.org/10.1016/0887-8994(88)90060-4.

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39

Allison, T. "Evoked potentials. II." Electroencephalography and Clinical Neurophysiology/Evoked Potentials Section 62, no. 4 (July 1985): 317. http://dx.doi.org/10.1016/0168-5597(85)90009-7.

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40

Schramm, Johannes. "Somatosensory evoked potentials." Surgical Neurology 25, no. 4 (April 1986): 406. http://dx.doi.org/10.1016/0090-3019(86)90219-3.

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41

DeVault, Kenneth R., Sabrina Beacham, Leopold J. Streletz, and Donald O. Castell. "Cerebral evoked potentials." Digestive Diseases and Sciences 38, no. 12 (December 1993): 2241–46. http://dx.doi.org/10.1007/bf01299903.

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42

กวีวงศ์ประเสริฐ, สมัย. "Evoked Potentials ในเวชปฏิบัติทั่วไป." Chulalongkorn Medical Journal 29, no. 2 (February 1985): 239–74. http://dx.doi.org/10.58837/chula.cmj.29.2.10.

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43

Fecchio, Matteo, Simone Russo, Sara Parmigiani, Alice Mazza, Alessandro Viganò, Adenauer G. Casali, Renzo Comolatti, Ezequiel Mikulan, Marcello Massimini, and Mario Rosanova. "Spatiotemporal specificity of TMS-evoked potentials versus sensory evoked potentials." Brain Stimulation 14, no. 6 (November 2021): 1688. http://dx.doi.org/10.1016/j.brs.2021.10.320.

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44

Brantberg, Krister, and Arne Tribukait. "Vestibular evoked myogenic potentials in response to laterally directed skull taps." Journal of Vestibular Research 12, no. 1 (November 1, 2002): 35–45. http://dx.doi.org/10.3233/ves-2002-12104.

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In recent years it has been demonstrated that loud clicks generate short latency vestibular evoked myogenic potentials (VEMP). It has also been demonstrated that midline forehead skull tap stimulation evokes similar VEMP. In the present study, the influence of skull tap direction on VEMP was studied in 13 normal subjects and in five patients with unilateral vestibular loss. Gentle skull taps were delivered manually above each ear on the side of the skull. The muscular responses were recorded over both sternocleidomastoid muscles using skin electrodes. Among the normals, laterally directed skull taps evoked “coordinated contraction-relaxation responses”, i.e. skull taps on one side evoked a negative-positive “inverted” VEMP on that side and a positive-negative "normal" VEMP on the other side. Among patients with unilateral vestibular function loss, skull taps above the lesioned ear evoked similar coordinated contraction-relaxation responses. However, skull taps above the healthy ear did not evoke that type of response. These findings suggest that laterally directed skull taps activate mainly the contralateral labyrinth.

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45

Donzel-Raynaud, Christine, Christian Straus, Michela Bezzi, Stefania Redolfi, Mathieu Raux, Marc Zelter, Jean-Philippe Derenne, and Thomas Similowski. "Upper airway afferents are sufficient to evoke the early components of respiratory-related cortical potentials in humans." Journal of Applied Physiology 97, no. 5 (November 2004): 1874–79. http://dx.doi.org/10.1152/japplphysiol.01381.2003.

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Repeated inspiratory occlusions in humans elicit respiratory-related cortical potentials, the respiratory counterpart of somatosensory-evoked potentials. These potentials comprise early components (stimulus detection) and late components (cognitive processing). They are considered as the summation of several afferent activities from various part of the respiratory system. This study assesses the role of the upper airway as a determinant of the early and late components of the potentials, taking advantage of the presence of a tracheotomy in patients totally or partially deafferented. Eight patients who could breathe either through the mouth or through a tracheotomy orifice (whole upper airway bypassed) were studied (4 quadriplegic patients with phrenic pacing, 4 patients with various sources of inspiratory pump dysfunction). Respiratory-related evoked potentials were recorded in CZ-C3 and CZ-C4. They were consistently present after mouth occlusions, with a first positive P1 and a first negative N1 components of normal latencies (P1: 40.4 ± 6.1 ms in CZ-C3 and 47.6 ± 7.6 ms in CZ-C4; N1: 84.4 ± 27.1 ms in CZ-C3 and 90.2 ± 17.4 ms in CZ-C4) and amplitudes. Tracheal occlusions did not evoke any cortical activity. Therefore, in patients with inspiratory pump dysfunction, the activation of upper airway afferents is sufficient to produce the early components of the respiratory-related evoked cortical potentials. Per contra, in this setting, pulmonary afferents do not suffice to evoke these components.

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46

Schwindt, Peter C., and Wayne E. Crill. "Synaptically Evoked Dendritic Action Potentials in Rat Neocortical Pyramidal Neurons." Journal of Neurophysiology 79, no. 5 (May 1, 1998): 2432–46. http://dx.doi.org/10.1152/jn.1998.79.5.2432.

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Schwindt, Peter C. and Wayne E. Crill. Synaptically evoked dendritic action potentials in rat neocortical pyramidal neurons. J. Neurophysiol. 79: 2432–2446, 1998. In a previous study iontophoresis of glutamate on the apical dendrite of layer 5 pyramidal neurons from rat neocortex was used to identify sites at which dendritic depolarization evoked small, prolonged Ca2+ spikes and/or low-threshold Na+ spikes recorded by an intracellular microelectrode in the soma. These spikes were identified as originating in the dendrite. Here we evoke similar dendritic responses by electrical stimulation of presynaptic elements near the tip of the iontophoretic electrode with the use of a second extracellular electrode. In 9 of 12 recorded cells, electrically evoked excitatory postsynaptic potentials (EPSPs) above a minimum size triggered all-or-none postsynaptic responses similar to those evoked by dendritic glutamate iontophoresis at the same site. Both the synaptically evoked and the iontophoretically evoked depolarizations were abolished reversably by blockade of glutamate receptors. In all recorded cells, the combination of iontophoresis and an EPSP, each of which was subthreshold for the dendritic spike when given alone, evoked a dendritic spike similar to that evoked by a sufficiently large iontophoresis. In one cell tested, dendritic spikes could be evoked by the summation of two independent subthreshold EPSPs evoked by stimulation at two different locations. We conclude that the dendritic spikes are not unique to the use of glutamate iontophoresis because similar spikes can be evoked by EPSPs. We discuss the implications of these results for synaptic integration and for the interpretation of recorded synaptic potentials.

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47

Shine, Timothy S. J., Barry A. Harrison, Martin L. De Ruyter, Julia E. Crook, Michael Heckman, Jasper R. Daube, Wolf H. Stapelfeldt, et al. "Motor and Somatosensory Evoked Potentials." Anesthesiology 108, no. 4 (April 1, 2008): 580–87. http://dx.doi.org/10.1097/aln.0b013e318168d921.

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Background Paraplegia is a devastating complication for patients undergoing repair of thoracoabdominal aortic aneurysms. A monitor to detect spinal cord ischemia is necessary if anesthesiologists are to intervene to protect the spinal cord during aortic aneurysm clamping. Methods The medical records of 60 patients who underwent thoracoabdominal aortic aneurysm repair with regional lumbar epidural cooling with evoked potential monitoring were reviewed. The authors analyzed latency and amplitude of motor evoked potentials, somatosensory evoked potentials, and H reflexes before cooling and clamping, after cooling and before clamping, during clamping, and after release of aortic cross clamp. Results Twenty minutes after the aortic cross clamp was placed, motor evoked potentials had 88% sensitivity and 65% specificity in predicting spinal cord ischemia. The negative predictive value of motor evoked potentials at 20 min after aortic cross clamping was 96%. Conclusions Rapid loss of motor evoked potentials or H reflexes after application of the aortic cross clamp identifies a subgroup of patients who are at high risk of developing spinal cord ischemia and in whom aggressive anesthetic and surgical interventions may be justified.

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48

FERBERT, A., H. BUCHNER, and H. BRÜCKMANN. "BRAINSTEM AUDITORY EVOKED POTENTIALS AND SOMATOSENSORY EVOKED POTENTIALS IN PONTINE HAEMORRHAGE." Brain 113, no. 1 (1990): 49–63. http://dx.doi.org/10.1093/brain/113.1.49.

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49

Riemslag, F. C. C., G. L. Van Der Heijde, and M. M. M. M. Van Dongen. "Are eye movement evoked potentials different from pattern reversal evoked potentials?" Documenta Ophthalmologica 66, no. 4 (August 1987): 279–89. http://dx.doi.org/10.1007/bf00213656.

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50

Furlong, P. L., Q. Azis, K. D. Singh, G. F. A. Harding, and D. G. Thompson. "Oesophageal evoked potentials and evoked magnetic fields." Electroencephalography and Clinical Neurophysiology 98, no. 2 (February 1996): P4. http://dx.doi.org/10.1016/0013-4694(96)84526-3.

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