Relevant bibliographies by topics / Erythrocytes

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Erythrocytes'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Erythrocytes"

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Walker, Britta, Syeda T. Towhid, Evi Schmid, Sascha M. Hoffmann, Majed Abed, Patrick Münzer, Sebastian Vogel, et al. "Dynamic adhesion of eryptotic erythrocytes to immobilized platelets via platelet phosphatidylserine receptors." American Journal of Physiology-Cell Physiology 306, no. 3 (February 1, 2014): C291—C297. http://dx.doi.org/10.1152/ajpcell.00318.2013.

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Glucose depletion of erythrocytes triggers suicidal erythrocyte death or eryptosis, which leads to cell membrane scrambling with phosphatidylserine exposure at the cell surface. Eryptotic erythrocytes adhere to endothelial cells by a mechanism involving phosphatidylserine at the erythrocyte surface and CXCL16 as well as CD36 at the endothelial cell membrane. Nothing has hitherto been known about an interaction between eryptotic erythrocytes and platelets, the decisive cells in primary hemostasis and major players in thrombotic vascular occlusion. The present study thus explored whether and how glucose-depleted erythrocytes adhere to platelets. To this end, adhesion of phosphatidylserine-exposing erythrocytes to platelets under flow conditions was examined in a flow chamber model at arterial shear rates. Platelets were immobilized on collagen and further stimulated with adenosine diphosphate (ADP, 10 μM) or thrombin (0.1 U/ml). As a result, a 48-h glucose depletion triggered phosphatidylserine translocation to the erythrocyte surface and augmented the adhesion of erythrocytes to immobilized platelets, an effect significantly increased upon platelet stimulation. Adherence of erythrocytes to platelets was blunted by coating of erythrocytic phosphatidylserine with annexin V or by neutralization of platelet phosphatidylserine receptors CXCL16 and CD36 with respective antibodies. In conclusion, glucose-depleted erythrocytes adhere to platelets. The adhesive properties of platelets are augmented by platelet activation. Erythrocyte adhesion to immobilized platelets requires phosphatidylserine at the erythrocyte surface and CXCL16 as well as CD36 expression on platelets. Thus platelet-mediated erythrocyte adhesion may foster thromboocclusive complications in diseases with stimulated phosphatidylserine exposure of erythrocytes.
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Klotz, F. W., J. D. Chulay, W. Daniel, and L. H. Miller. "Invasion of mouse erythrocytes by the human malaria parasite, Plasmodium falciparum." Journal of Experimental Medicine 165, no. 6 (June 1, 1987): 1713–18. http://dx.doi.org/10.1084/jem.165.6.1713.

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Plasmodium falciparum malaria merozoites require erythrocyte sialic acid for optimal invasion of human erythrocytes. Since mouse erythrocytes have the form of sialic acid found on human erythrocytes (N-acetyl neuraminic acid), mouse erythrocytes were tested for invasion in vitro. The Camp and 7G8 strains of P. falciparum invaded mouse erythrocytes at 17-45% of the invasion rate of human erythrocytes. Newly invaded mouse erythrocytes morphologically resembled parasitized human erythrocytes as shown on Giemsa-stained blood films and by electron microscopy. The rim of parasitized mouse erythrocytes contained the P. falciparum 155-kD protein, which is on the rim of ring-infected human erythrocytes. Camp but not 7G8 invaded rat erythrocytes, indicating receptor heterogeneity. These data suggest that it may be possible to adapt the asexual erythrocytic stage of P. falciparum to rodents. The development of a rodent model of P. falciparum malaria could facilitate vaccine development.
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Murakami, K., and K. Tanabe. "An antigen of Plasmodium yoelii that translocates into the mouse erythrocyte membrane upon entry into the host cell." Journal of Cell Science 73, no. 1 (February 1, 1985): 311–20. http://dx.doi.org/10.1242/jcs.73.1.311.

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Monoclonal antibodies against the rodent malaria parasite, Plasmodium yoelii, have been prepared and characterized by indirect immunofluorescence on acetone-fixed infected mouse erythrocytes. The antibody of clone K2 reacted strongly with late trophozoites and schizonts, whereas it did so weakly and diffusely with ring forms and early trophozoites. Strong fluorescence was confined to granular structures in schizonts and merozoites. Parasites that invaded erythrocytes in vitro lost the strong fluorescence. Instead, immunofluorescence appeared in the membranes of erythrocytes infected in vitro with merozoites. Erythrocytes infected with more than one merozoite had intensified immunofluorescence in their membranes. Staining of the invaded erythrocytes with 4′,6-diamidino-2-phenylindole (DAPI) hydrochloride demonstrated that membranes of all the invaded erythrocytes acquired the P. yoelii antigen. These results suggest that the P. yoelii antigen in merozoites is translocated into erythrocyte membranes upon entry into the host cell. Immunofluorescence continued to appear in membranes of infected erythrocytes throughout the intra-erythrocytic parasite growth. Staining of unfixed infected erythrocytes with the K2 antibody failed to detect the parasite antigen. In contrast, immunofluorescence was present in unfixed membranes of erythrocyte ghosts, which had been spontaneously formed after rupture of schizont-infected erythrocytes by merozoite release. No immunofluorescence appeared in either acetone-fixed or unfixed ghosts of normal erythrocytes. These results suggest the antigenic determinant of the P. yoelii antigen is exposed at the cytoplasmic surface of the infected erythrocyte membrane. Immunoprecipitation has revealed that the K2 antibody recognizes a 160 X 10(3) Mr P. yoelii antigen.
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Reno, Paul W., Katherine Kleftis, Stuart W. Sherburne, and Bruce L. Nicholson. "Experimental Infection and Pathogenesis of Viral Erythrocytic Necrosis (VEN) in Atlantic Cod, Gadus morhua." Canadian Journal of Fisheries and Aquatic Sciences 43, no. 5 (May 1, 1986): 945–51. http://dx.doi.org/10.1139/f86-117.

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Mature Atlantic cod (Gadus morhua) were experimentally infected with viral erythrocytic necrosis (VEN) by inoculation with washed erythrocytes or ceil-free homogenates of erythrocytes from naturally infected fish. Approximately one third of the animals exposed exhibited active infections. The temporal pattern of infection was similar between naturally infected and experimentally infected fish. One to two months after infection, immature erythrocytes began to show clear evidence of VEN followed by a rapid increase in the proportion of infected immature erythrocytes, frequently reaching 100%. A subsequent dramatic drop in infection of immature erythrocytes occurred, coinciding with an increase of infection in mature erythrocytes. Significant erythroblastosis occurred when the overall erythrocyte infection rate reached approximately 10%, but none of the newly generated erythrocytes appeared infected. The peak infection rate (40–60% of erythrocytes infected) declined slowly and the infection, in most instances, was completely resolved.
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Hsiao, L. L., R. J. Howard, M. Aikawa, and T. F. Taraschi. "Modification of host cell membrane lipid composition by the intra-erythrocytic human malaria parasite Plasmodium falciparum." Biochemical Journal 274, no. 1 (February 15, 1991): 121–32. http://dx.doi.org/10.1042/bj2740121.

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The phospholipid and fatty acid compositions of the host infected erythrocyte plasma membrane (IEPM) have been determined for erythrocytes infected with the human malaria parasite Plasmodium falciparum. IEPM were prepared by selective lysis of the host erythrocyte (but not of the parasite membranes) with 0.1% saponin, followed by differential centrifugation. The purity of the IEPM was determined by measuring the membrane-specific enzyme markers acetylcholinesterase, glutamate dehydrogenase and lactate dehydrogenase, and by immunoelectron microscopy using monoclonal antibodies specific for human erythrocyte glycophorin A (4E7) and for a 195 kDa parasite membrane glycoprotein (Pf6 3B10.1). Both approaches demonstrated that the host erythrocyte plasma membrane preparation was free from contamination by parasite membranes. During intra-erythrocytic development of the parasite, the phospholipid composition of the erythrocyte membrane was strikingly altered. IEPM contained more phosphatidylcholine (38.7% versus 31.7%) and phosphatidylinositol (2.1% versus 0.8%) and less sphingomyelin (14.6% versus 28.0%) than normal uninfected erythrocytes. Similar alterations in phospholipid composition were determined for erythrocyte membranes of parasitized cells isolated by an alternative method utilizing polycationic polyacrylamide microbeads (Affigel 731). The total fatty acid compositions of the major phospholipids in IEPM were determined by g.l.c. The percentage of polyunsaturated fatty acids in normal erythrocyte phospholipids (39.4%) was much higher than in phospholipids from purified parasites (23.3%) or IEPM (24.0%). The unsaturation index of phospholipids in IEPM was considerably lower than in uninfected erythrocytes (107.5 versus 161.0) and was very similar to that in purified parasites (107.5 versus 98.5). Large increases in palmitic acid (C16:0) (from 21.88% to 31.21%) and in oleic acid (C18:1) (from 14.64% to 24.60%), and major decreases in arachidonic acid (C20:4) (from 17.36% to 7.85%) and in docosahexaenoic acid (C22:6) (from 4.34% to 1.8%) occurred as a result of infection. The fatty acid profiles of individual phospholipid classes from IEPM resembled in many instances the fatty acid profiles of parasite phospholipids rather than those of uninfected erythrocytes. Analysis of IEPM from P. falciparum-infected erythrocytes (trophozoite stage) revealed that, during intra-erythrocytic maturation of the parasite, the host erythrocyte phospholipid composition was markedly refashioned. These alterations were not dependent on the method used to isolate the IEPM, with similar results obtained using either a saponin-lysis method or binding to Affigel beads. Since mature erythrocytes have negligible lipid synthesis and metabolism, these alterations must occur as a result of parasite-directed metabolism of erythrocyte lipids and/or trafficking of lipids between the parasite and erythrocyte membranes.
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Lowe-Jinde, L. "Hematological changes in Cryptobia-infected rainbow trout (Salmo gairdneri)." Canadian Journal of Zoology 64, no. 6 (June 1, 1986): 1352–55. http://dx.doi.org/10.1139/z86-201.

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Rainbow trout, Salmo gairdneri, were experimentally infected with Cryptobia salmositica and total and differential erythrocyte and leucocyte abundances were examined. Total erythrocyte counts were profoundly reduced and were lowest at 4 weeks postinoculation during the peak of infection. Differential erythrocyte number showed significant reduction in mature erythrocytes, degenerating erythrocytes, and erythroblasts. The reduction in mature erythrocytes number was observed from 3 through 8 weeks postinoculation, while erythroblast reduction occurred at 1 through 5 weeks postinoculation. Subsequently, the number of erythroblasts increase and reach control values at 6, 7, and 8 weeks postinoculation. Although the total leucocyte count was not significantly changed, the differential leucocyte abundance showed that granulocytes were significantly increased. Similarly, the rarely seen, circulating, immature granuloblasts were increased. Both granuloblast and granulocyte numbers returned to control values by 8 weeks postinoculation. Thus, the pattern of differential erythrocytic and leucocytic changes reflect an acute parasitemia and a subsequent partial recovery of the infected fish to the parasitemia.
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Orwa, Titus Okello, Rachel Waema Mbogo, and Livingstone Serwadda Luboobi. "Mathematical Model for Hepatocytic-Erythrocytic Dynamics of Malaria." International Journal of Mathematics and Mathematical Sciences 2018 (July 2, 2018): 1–18. http://dx.doi.org/10.1155/2018/7019868.

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Human malaria remains a major killer disease worldwide, with nearly half (3.2 billion) of the world’s population at risk of malaria infection. The infectious protozoan disease is endemic in tropical and subtropical regions, with an estimated 212 million new cases and 429,000 malaria-related deaths in 2015. An in-host mathematical model ofPlasmodium falciparummalaria that describes the dynamics and interactions of malaria parasites with the host’s liver cells (hepatocytic stage), the red blood cells (erythrocytic stage), and macrophages is reformulated. By a theoretical analysis, an in-host basic reproduction numberR0is derived. The disease-free equilibrium is shown to be locally and globally asymptotically stable. Sensitivity analysis reveals that the erythrocyte invasion rateβr, the average number of merozoites released per bursting infected erythrocyteK, and the proportion of merozoites that cause secondary invasions at the blood phaseζare the most influential parameters in determining the malaria infection outcomes. Numerical results show that macrophages have a considerable impact in clearing infected red blood cells through phagocytosis. Moreover, the density of infected erythrocytes and hence the severity of malaria are shown to increase with increasing density of merozoites in the blood. Concurrent use of antimalarial drugs and a potential erythrocyte invasion-avoidance vaccine would minimize the density of infected erythrocytes and hence malaria disease severity.
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TUFTS, B. L., R. A. FERGUSON, and R. G. BOUTILIER. "In Vivo and In Vitro Effects of Adrenergic Stimulation on Chloride/Bicarbonate Exchange in Rainbow Trout Erythrocytes." Journal of Experimental Biology 140, no. 1 (November 1, 1988): 301–12. http://dx.doi.org/10.1242/jeb.140.1.301.

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In vitro and in vivo experiments were carried out to determine the effect of catecholamines on erythrocytic chloride/bicarbonate exchange in the rainbow trout. A further modified boat assay is described and was used to measure bicarbonate flux through intact erythrocytes. Catecholamines had no significant effect on the bicarbonate flux in vitro. The erythrocytes were sensitive to adrenergic stimulation, however, since the agonists used caused a decrease in the pH gradient across the erythrocyte membrane. Exhaustive exercise was associated with an increase in bicarbonate flux through the intact erythrocytes. The mechanism for this increase is not clear, but it is evidently not adrenergic in origin.
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SHARMA, A. K., S. N. S. RANDHAWA, S. K. UPPAL, and R. RANJAN. "Changes in haematology, blood mineral profile, ultra structure and superoxide dismutase activities in erythrocytes in hypophosphetemic buffaloes given excess molybdenum." Indian Journal of Animal Sciences 84, no. 5 (May 13, 2014): 516–19. http://dx.doi.org/10.56093/ijans.v84i5.40653.

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Phosphorus deficiency in buffaloes resulted into development of macrocytic, normochromic anaemia with significant decline in haemoglobin, packed cell volume and total erythrocyte count. Erythrocytic superoxide dismutase activity was markedly decreased in hypophosphataemic animals given excess molybdenum (T2) in comparison to hypophosphataemic animals not given molybdenum (T3). Scanning electron microscopy of erythrocytes in both T2 and T3 animals showed presence of spherocytes, discocytes and slight membrane corrugations; while erythrocytes in healthy control (T1) appeared biconcave with mild poikilocytosis. From the present study it can be concluded that phosphorus deficiency induces ultrastructural abnormalities in erythrocyte, which is further aggravated by excess molybdenum supplementation. Increased oxidative damage may contribute structural changes in phosphorus deficiency and molybdenum toxicity.
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Muenster, Stefan, Arkadi Beloiartsev, Binglan Yu, E. Du, Sabia Abidi, Ming Dao, Gregor Fabry, et al. "Exposure of Stored Packed Erythrocytes to Nitric Oxide Prevents Transfusion-associated Pulmonary Hypertension." Anesthesiology 125, no. 5 (November 1, 2016): 952–63. http://dx.doi.org/10.1097/aln.0000000000001294.

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Abstract Background Transfusion of packed erythrocytes stored for a long duration is associated with increased pulmonary arterial pressure and vascular resistance. Prolonged storage decreases erythrocyte deformability, and older erythrocytes are rapidly removed from the circulation after transfusion. The authors studied whether treating stored packed ovine erythrocytes with NO before transfusion could prevent pulmonary vasoconstriction, enhance erythrocyte deformability, and prolong erythrocyte survival after transfusion. Methods Ovine leukoreduced packed erythrocytes were treated before transfusion with either NO gas or a short-lived NO donor. Sheep were transfused with autologous packed erythrocytes, which were stored at 4°C for either 2 (“fresh blood”) or 40 days (“stored blood”). Pulmonary and systemic hemodynamic parameters were monitored before, during, and after transfusion. Transfused erythrocytes were labeled with biotin to measure their circulating lifespan. Erythrocyte deformability was assessed before and after NO treatment using a microfluidic device. Results NO treatment improved the deformability of stored erythrocytes and increased the number of stored erythrocytes circulating at 1 and 24 h after transfusion. NO treatment prevented transfusion-associated pulmonary hypertension (mean pulmonary arterial pressure at 30 min of 21 ± 1 vs. 15 ± 1 mmHg in control and NO–treated packed erythrocytes, P < 0.0001). Washing stored packed erythrocytes before transfusion did not prevent pulmonary hypertension. Conclusions NO treatment of stored packed erythrocytes before transfusion oxidizes cell-free oxyhemoglobin to methemoglobin, prevents subsequent NO scavenging in the pulmonary vasculature, and limits pulmonary hypertension. NO treatment increases erythrocyte deformability and erythrocyte survival after transfusion. NO treatment might provide a promising therapeutic approach to prevent pulmonary hypertension and extend erythrocyte survival.
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Dissertations / Theses on the topic "Erythrocytes"

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Gonzalez, Laurie J. "The influence of membrane lipid order on cell shape and microvesiculation in human erythrocytes /." Diss., CLICK HERE for online access, 2006. http://contentdm.lib.byu.edu/ETD/image/etd1615.pdf.

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Persson, Sylvi. "Erythrocyte deformability studies by viscometry and filtrometry." Lund : Dept. of Medicine, University Hospital of Lund, Lund University, 1994. http://catalog.hathitrust.org/api/volumes/oclc/39207611.html.

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Flatt, Joanna Frances. "A study of human erythrocyte membrane structure and function using variant erythrocytes." Thesis, University of Bristol, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.560498.

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Human erythrocytes are highly specialised cells boasting numerous features to maximise gas carriage, exchange and delivery around the body. The role of the highly proteinaceous red cell membrane in these processes is vital. Some membrane proteins such as the Rh-associated glycoprotein (RhAG) and aquaporin-1 (AQP1) are postulated to form gas channels, and disorders affecting membrane proteins can have extensive effects on normal red cell function. In this work, the role and interactions of Rh proteins are probed using rare variant Rh-deficient erythrocytes. The RhCE polypeptide is required for normal expression of other members of the Rh complex. Lack of RhCE is associated with depressed expression of CD44, an adhesion molecule, and may alter expression of proteins involved in complement such as decay acceleration factor and Iymphocyte function-associated antigen 3. Absence of RhAG prevents Rh complex expression and is found to affect band 3 macrocomplex proteins GPA and protein 4.2, highlighting the important role for RhAG in the macrocomplex. AQP1 is increased in the absence of RhAG, which supports the hypothesis that they share similar functions. The hereditary stomatocytoses are disorders that affect the ion permeability of red cell membranes. This work comprises a study of the pleiotropic disorder stomatin-deficient cryohydrocytosis (sdCHC), which is caused by mutations in the red cell glucose transporter, glut1. The mutant proteins show minimal glucose transport and increased permeability to cations when expressed heterologously in Xenopus laevis oocytes - consistent with the disease phenotype. sdCHC erythrocytes have very reduced amounts of stomatin, a monotopic membrane protein, a feature shared by other very leaky red cells. This is accompanied by a concomitant increase in stomatin-like protein 2, whose function in red cells is currently unknown. The fates of stomatin proteins in normal and leaky cells are investigated throughout erythropoiesis and found to differ between cation-leaky phenotypes.
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Salles, Christine. "Erythrocytes et hémodialyse." Bordeaux 2, 1992. http://www.theses.fr/1992BOR2B004.

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HOLTZCLAW, JOHN DAVID. "CHARACTERIZATION OF LIGHT SICKLE ERYTHROCYTES DERIVED FROM DENSE ERYTHROCYTES IN VITRO." University of Cincinnati / OhioLINK, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=ucin990632508.

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Janzen, Johan. "Fibrinogen adsorption to human erythrocytes." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/25820.

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Fibrinogen adsorption to human erythrocytes has been implicated as the mechanism responsible for the reversible aggregation of red cells. The object of this thesis was to measure the binding of fibrinogen to human erythrocytes and to compare the binding with aggregation behaviour. The binding of isolated ¹²⁵I-fibrinogen to red cells was estimated from the amount of radiolabel removed from solution upon addition of cells, from the amount of radiolabel retained in cell pellets produced by ultracentrifugation and corrected for intracellular radiolabel, and from radiolabel retained by cells after exhaustive washing in protein-free buffer. Binding of human albumin to red cells was determined by the same methods. The free energy of formation of red cell/red cell contacts due to isolated fibrinogen was determined by the method of Evans and Buxbaum (Biophys. J. 3̲4̲, 1-12, 1981). The solution depletion method required corrections for dilution effects and was not sensitive enough for detailed determination of binding of these proteins to red cells. Binding estimated from cell pellets and corrected for intracellular radiolabel increased linearily with protein concentration to 12 mg/mL for fibrinogen and 50 mg/mL for albumin. The binding at 1 mg/mL was between 50 and 250 molecules per cell for fibrinogen and between 1000 and 1400 molecules per cell for albumin. Binding determined from wash analyses was similar to pellet binding corrected for free radiolabel. The surface affinity of red cells for red cell beads was determined to be 1.8 ± 0.5 x 10⁻³ erg/cm² (mean ± SEM, n = 4) and 2.8 ± 0.8 x 10⁻³ erg/cm² (mean ± SEM, n = 28) at 4.4 and 8.8 mg/ml fibrinogen respectively. Adhesion between red cells and red cell beads occurred at 2.2 mg/mL fibrinogen, but was too weak to allow estimation of the surface affinity. The surface affinities and binding estimates allow calculation of the mean binding energy for fibrinogen to the red cell surface. The surface affinity per molecule, assuming 250 molecules per cell at 1 mg/mL, was 30 ± 10 and 24 ± 7 kilocalories/mole at 4.4 and 8.8 mg/mL respectively. The difference was not significant. This result poses a problem as binding energies of this order suggest that the adhesion would be irreversible. If fibrinogen binding were 1500 molecules per cell at 1 mg/mL a binding energy of 4 ± 1 kilocalories/mole would be implied. These results are interpreted as indicating that fibrinogen bound to red cells may be displaced during ultracentrifugation.
Medicine, Faculty of
Pathology and Laboratory Medicine, Department of
Graduate
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Goding, Linda M. "Macrophage Recognition of Xenogeneic Erythrocytes." University of Toledo Health Science Campus / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=mco1197469419.

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Dodson, Walter R. "Dynamics of erythrocytes and microcapsules." College Park, Md. : University of Maryland, 2008. http://hdl.handle.net/1903/8153.

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Thesis (Ph. D.) -- University of Maryland, College Park, 2008.
Thesis research directed by: Fischell Dept. of Bioengineering. Title from t.p. of PDF. Includes bibliographical references. Published by UMI Dissertation Services, Ann Arbor, Mich. Also available in paper.
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BOUSCH, WAHL MARIE-ROSE. "Les erythrocytes vecteurs de medicaments." Strasbourg 1, 1993. http://www.theses.fr/1993STR15083.

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Rowe, Jane Alexandra. "Rosetting of Plasmodium falciparium infected erythrocytes." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260775.

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Books on the topic "Erythrocytes"

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1949-, Agre Peter, and Parker John C. 1935-, eds. Red blood cell membranes: Structure, function, clinical implications. New York: Dekker, 1989.

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Lang, Florian, and Michael Föller. Erythrocytes: Physiology and pathophysiology. London: Imperial College Press, 2012.

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Mauro, Magnani, and De Flora Antonio, eds. Red blood cell aging. New York: Plenum Press, 1991.

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1941-, Eaton John Wallace, Konzen Diana K, and White James G, eds. Cellular and molecular aspects of aging: The red cell as a model : proceedings of a conference held in Minneapolis, Minnesota, September 8-11, 1984. New York: Liss, 1985.

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Sprandel, Ulrich, and James L. Way, eds. Erythrocytes as Drug Carriers in Medicine. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-0044-9.

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U, Sprandel, Way J. L, and International Society for the Use of Resealed Erythrocytes. Meeting, eds. Erythrocytes as drug carriers in medicine. New York: Plenum Press, 1997.

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Roth, Stephen Joel. The effect of anti-glycophorin A monoclonal antibodies and FAB fragments on Plasmodium falciparum erythrocyte invasion in vitro. [New Haven: s.n.], 1986.

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R, Bridges Kenneth, and Pearson Howard A, eds. Anemias and other red cell disorders. New York: McGraw-Hill, 2007.

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Haro, Ana Maria Rollan. Active erythrocytes, drug delivery and related studies. [S.l: The Author], 2000.

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Westhoff, Connie M., Jerald E. Mullersman, and Gary E. Carnahan. Guidelines for transfusion of erythrocytes containing sickle hemoglobin. Bethesda, Md: AABB, 2004.

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Book chapters on the topic "Erythrocytes"

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Proske, Uwe, David L. Morgan, Tamara Hew-Butler, Kevin G. Keenan, Roger M. Enoka, Sebastian Sixt, Josef Niebauer, et al. "Erythrocytes." In Encyclopedia of Exercise Medicine in Health and Disease, 303. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2361.

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Sedlmayr, Peter. "Fetal Erythrocytes." In Cellular Diagnostics, 680–86. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000209186.

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Shilpi, Satish, Kapil Khatri, Umesh Dhakad, Neelesh Kumar Mehra, and Arvind Gulbake. "Resealed Erythrocytes." In Micro- and Nanotechnologies-Based Product Development, 163–76. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003043164-10.

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Baker, Julien S., Fergal Grace, Lon Kilgore, David J. Smith, Stephen R. Norris, Andrew W. Gardner, Robert Ringseis, et al. "Production of Erythrocytes." In Encyclopedia of Exercise Medicine in Health and Disease, 726. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4469.

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Stecca, C., and M. Duverger van Bogaert. "N-Demethylation Reactions in Intact Erythrocytes and Erythrocyte Supernatant." In Archives of Toxicology, 291–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74117-3_52.

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Knauf, Philip A. "Anion Transport in Erythrocytes." In Membrane Physiology, 191–220. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1943-6_12.

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Knauf, Philip A. "Anion Transport in Erythrocytes." In Physiology of Membrane Disorders, 191–220. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2097-5_12.

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Parker, John C. "Genetic Variants of Erythrocytes." In Molecular Biology of Membrane Transport Disorders, 507–17. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1143-0_25.

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Andisi, Kivisi C., and Abdirahman I. Abdi. "Analysis of var Gene Transcription Pattern Using DBLα Tags." In Methods in Molecular Biology, 173–84. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2189-9_14.

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AbstractThe Plasmodium falciparum erythrocyte membrane protein 1 (PfEMP1) antigens, which are encoded by a multigene family called var genes, are exported and inserted onto the surface of the infected erythrocytes. PfEMP1 plays a key role in the pathogenesis of severe malaria and are major targets of naturally acquired immunity. Studying the expression pattern of var genes in P. falciparum clinical isolates is crucial for understanding disease mechanism and immunity to malaria. However, var genes are highly variable, which makes it difficult to study their expression in clinical isolates obtained directly from malaria patients. In this chapter, we describe an approach for analysis of var gene expression that targets a region referred to as DBLα tag, which is relatively conserved in all var genes.
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Hadley, Terence J., and Louis H. Miller. "Invasion of Erythrocytes by Malaria Parasites: Erythrocyte Ligands and Parasite Receptors." In Chemical Immunology and Allergy, 49–71. Basel: KARGER, 1988. http://dx.doi.org/10.1159/000318613.

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Conference papers on the topic "Erythrocytes"

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Sauliutė, Gintarė, Milda Stankevičiūtė, Gintaras Svecevičius, Janina Baršienė, and Roberta Valskienė. "Assessment of heavy metals bioconcentration factor (BCF) and genotoxicity response induced by metal mixture in Salmo salar tissues." In Environmental Engineering. VGTU Technika, 2017. http://dx.doi.org/10.3846/enviro.2017.043.

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The aim of this study was to evaluate metals bioconcentration factor (BCF) in gills, liver, kidneys and muscle in relation with genotoxicity effects of metal mixture in peripheral blood, kidneys, gills and liver erythrocytes of the Atlantic salmon (Salmo salar). Fish were exposed to maximum-permissible waterborne concentrations of Zn – 0.1, Cu – 0.01, Ni – 0.01, Cr – 0.01, Pb – 0.005 and Cd – 0.005 mg/L, respectively for 7 and 14 days. Genotoxicity was studied using the micronucleus test. In addition, erythrocyte nuclear abnormalities (ENAs) were analysed. Our study indicates that metal BCF in Atlantic salmon is tissue-dependent. Based on the BCF classification scale, the relatively low values of metals bioconcentration were assessed, except for Zn (gills) and Cu (liver) (359.6 and 594.0, respectively). Zn intensively concentrated in fish tissues, while Pb – least of all. Overall, metals were concentrated mostly in the liver, least – in the muscle. Significant differences among BCF values of Pb in gills and muscle and Cd in gills were measured between 7 and 14 d exposure groups. Treatment with metal mixture significantly increased micronucleus frequencies after 7 d of exposure in liver and peripheral blood erythrocytes. Significant genotoxicity response was not observed after 14 d treatment. The erythrocytic nuclei abnormalities determined in S. salar blood were nuclear bud on filament (NBf), nuclear bud (NB), blebbed (BL), kidney shaped, vacuolated (VacNuc), 8-shaped nuclei and fragmented-apoptotic (FA) erythrocytes. Significant elevation in total ENAs level was detected in kidneys and liver erythrocytes after 7 d treatment, while after 14 d – in gills and kidneys erythrocytes. No significant differences among analysed responses were measured between 7 and 14 d exposure groups, except total ENAs level in liver erythrocytes.
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Hashimoto, Shigehiro. "Deformation of Erythrocyte in Couette Type of Pulsatile Shear Field." In ASME 2020 Fluids Engineering Division Summer Meeting collocated with the ASME 2020 Heat Transfer Summer Conference and the ASME 2020 18th International Conference on Nanochannels, Microchannels, and Minichannels. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/fedsm2020-20074.

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Abstract The experimental methodology has been designed for evaluation of the cyclic deformation of an erythrocyte in the pulsatile shear field in vitro. To observe the deformation of the suspended erythrocytes in the Couette type of the shear flow, a rheoscope system has been manufactured. The system consists of a pair of counter-rotating parallel disks and an inverted phase-contrast microscope. The human erythrocytes were suspended in the dextran aqueous solution of high viscosity. The rotating speed varies sinusoidally to make the pulsatile shear field. The deformation of each erythrocyte was measured at the video image of the rheoscope. The experimental results show that the system is available to measure the following behavior of an erythrocyte. The ellipsoidal shape of each erythrocyte varies cyclically to follow the pulsatile cyclic shear field. The delay of deformation phase of each erythrocyte in the cycle is related to the frequency of the cyclic variation of the shear field. The delay is also related to deformability of the erythrocyte.
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Mori, Tatsuya, and Takashi Saito. "Estimation of Erythrocyte Deformability Using a Viscoelastic Model With Two Degrees of Freedom." In ASME 2011 International Mechanical Engineering Congress and Exposition. ASMEDC, 2011. http://dx.doi.org/10.1115/imece2011-63840.

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An erythrocyte is represented using a viscoelastic model with two degrees of freedom, where a spring was connected parallel to a dashpot to estimate quantitatively the dynamic deformability. To identify such parameters as the elastic modulus and viscous damping coefficient of the model, erythrocytes were deformed using a periodical shear flow field. Displacement of the center of gravity ΔG and the deformation quantity of major axis ΔL were obtained from time series of images. In this paper, erythrocytes from a normal subjects were treated with glutaraldehyde to create variations in hardness and deformability. Both of the data series are periodic on movement and deformation of erythrocytes. Based on the experimental data, parameters of the viscoelastic model were identified. The estimation method of dynamic deformability of erythrocytes was examined.
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Hoser, M., and G. F. Savidge. "DIFFERENCES IN PEPTIDE MAPS OF a POLYMERS FROM FIBRIN PRODUCED IN THE PRESENCE AND ABSENCE OF ERYTHROCYTES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643320.

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α chain polymerisation during clot formation is accelerated in the presence of erythrocytes. This effect is abrogated if the erythrocytes are obtained from patients with various haemoglo-binopathies. Enzyme digests of the α polymers produced in the presence or absence of erythrocytes were prepared to further define any differences between them.Clots were produced from citrate/EACA plasma samples or plasma/erythrocyte mixtures by the addition of thrombin and calcium. After five hours, clots were washed iii 8 M urea until traces of haemoglobin were removed. After reduction and alkylation clots were dissolved in 0.5% SDS. a polymers were purified on sephacryl S-300 and protein concentrations were adjusted to 0.5 mg/ml. These were digested with S. Aureus V8 protease (150 mg/ml), papain (50 mg/ml) or chymotrypsin (100 mg/ml) at 37°C at sequential time intervals.After the addition of 2% SDS samples were analysed on 15% SDS polyacrylamide gels.In all cases digestion of a polymers from clots formed in the absence of erythrocytes took place more rapidly and contained peptide bands not apparent in other digests.The observations suggest that α polymers formed in the presence or absence of erythrocytes exhibit differing kinetics of response to proteolytic cleavage and indicate that erythrocytes may influence the primary and/or quartenary structure of the polymers studied.
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Улитина, Анна Сергеевна, Мария Владимировна Химина, Андрей Анатольевич Колесов, and Ольга Васильевна Сироткина. "STUDY OF THE DONOR ERYTHROCYTES WITH DIFFERENT STORAGE TIME USING ATOMIC FORCE MICROSCOPY." In Актуальные проблемы современной науки: сборник статей международной научной конференции (Санкт-Петербург, Октябрь 2023). Crossref, 2023. http://dx.doi.org/10.37539/231011.2023.93.38.002.

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Образцы размороженных отмытых донорских эритроцитов со сроком хранения более 10 лет (n=20) и свежие образцы донорских эритроцитов. (n=20) были изучены с помощью атомно-силовой микроскопии (АСМ), а также с помощью гематологического анализатора. Получены данные о потенциальной пригодности декриоконсервированной донорской эритроцитной взвеси длительного хранения для трансфузии реципиенту. 20 samples of thawed washed erythrocytes with storage time more than 10 years and 20 samples of fresh erythrocytes were assessed with atomic force microscopy (AFM) and hematology analyzer. Obtained data show the potential applicability of decryopreserved erythrocyte suspension of prolonged storage for transfusion to recipient.
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Arko, Matevž, Anna Romolo, Vid Šuštar, and Veronika Kralj-Iglič. "Role of Erythrocyte Sedimentation Rate (ESR) in preparation of Platelet and Extracellular Vesicles Rich Plasma." In Socratic Lectures 7. University of Lubljana Press, 2022. http://dx.doi.org/10.55295/psl.2022.d23.

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Uses of platelet and extracellular vesicles rich plasma (PVRP) are in many fields of medicine as it was found that PVRP has regenareative properties. Preparation of PVRP is performed by sep-aration of erythrocytes from the liquid they are immersed in. Erythrocytes are the most numerous blood cells; they are also relatively large and dense as they are filled with haemoglobin. Therefore they sediment due to gravitation or systemic centrifugation force thereby pushing plasma that carries smaller particles in the opposite direction. This mechanism that takes place during pro-cessing determines the composition of plasma. Its tuning is therefore key in acquiring a prepara-tion with desired properties. In particular, it is of interest to study the effect of erythrocyte sedi-mentation rate (ESR) on composition and volume of acquired plasma. Different animals have different ranges of ESR values which may enable insight into mechanisms of plasma preparation. In this contribution we present the basic mechanisms of plasma preparation and properties of blood of different animals. Keywords: Erythrocyte Erythrocyte sedimentation rate; Platelet rich plasma; Horse blood, Cattle blood, Goat blood, Sheep blood
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Maciaszek, Jamie L., and George Lykotrafitis. "Sickle Cell Trait Erythrocytes are Thrice as Stiff as Healthy Erythrocytes." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-37779.

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Atomic force microscopy (AFM) allows for high-resolution topography studies of biological cells and measurement of their mechanical properties in physiological conditions. In this work, AFM was employed to measure the stiffness of abnormal human red blood cells (RBCs) from patients with the genotype for sickle cell trait. The determined Young’s modulus was compared with that obtained from measurements of erythrocytes from healthy subjects. The results showed that the Young’s modulus of pathological erythrocytes was approximately three times higher than in normal cells. Observed differences indicate the effect of hemoglobin S as well as possible changes in the organization of the cell cytoskeleton associated with the sickle cell trait.
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Kudryavtsev, V. A., and A. V. Shatrov. "Visualization of Blood Erythrocytes Sedimentation Process on Transitive Layer ”Erythrocytes - Plasma”." In 2021 IEEE Ural-Siberian Conference on Computational Technologies in Cognitive Science, Genomics and Biomedicine (CSGB). IEEE, 2021. http://dx.doi.org/10.1109/csgb53040.2021.9496034.

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Hentschel, Gesine, Sabrina Küspert, Ligia de Souza, Florian Pape, Birgit Glasmacher, and Florian Rummel. "Developing Rheological Characterization Methods for Bovine Blood and Hydrogel-Based Artificial Blood." In The Nordic Rheology Conference. University of Stavanger, 2024. http://dx.doi.org/10.31265/atnrs.782.

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Graphical abstract: Rheological understanding of blood is relevant for the design of cardiovascular medical devices and implants. Erythrocytes are represented by hydrogel beads dispersed in a continuous glycerol phase. The model system is characterized by aims of optical and rheological characterization. The erythrocyte schematic has been taken from 1. The deformable nature of the hydrogel particles can be seen from the pictures taken during the squeezing experiment.
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UddhavraoKatkar, Bhagirath, and Vaishali Barkade. "Prediction of Isolated Erythrocytes Formation from Compound Erythrocytes in Blood Cells Images." In 2017 International Conference on Computing, Communication, Control and Automation (ICCUBEA). IEEE, 2017. http://dx.doi.org/10.1109/iccubea.2017.8463823.

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Reports on the topic "Erythrocytes"

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Viksna, Ludmila, Oksana Kolesova, Aleksandrs Kolesovs, Ieva Vanaga, and Seda Arutjunana. Clinical characteristics of COVID-19 patients (Latvia, Spring 2020). Rīga Stradiņš University, December 2020. http://dx.doi.org/10.25143/fk2/hnmlhh.

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Data include following variables: Demographics, epidemiological history, comorbidities, diagnosis, complications, and symptoms on admission to the hospital. Also, body’s temperature and SpO2. Blood cells: white cells count (WBC), neutrophils (Neu), lymphocytes (Ly), eosinophils (Eo) and monocytes (Mo), percentages of segmented and banded neutrophils, erythrocytes (RBC), platelet count (PLT), hemoglobin (Hb), and hematocrit (HCT); Inflammatory indicators: erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP); Tissue damage indicators: alanine aminotransferase (ALT), lactate dehydrogenase (LDH), and troponin T (TnT); Electrolytes: potassium and sodium concentration; Renal function indicators: creatinine and glomerular filtration rate (GFR); Coagulation tests: D-dimer, prothrombin time, and prothrombin index on admission to the hospital.
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Momchilova, Albena, Zlatan Tsonchev, Mariana Hadzhilazova, Rumiana Tzoneva, Alexander Alexandrov, Dimitar Nikolakov, Viktoria Ilieva, and Roumen Pankov. Sphingolipid Metabolism Is Dysregulated in Erythrocytes from Multiple Sclerosis Patient. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, March 2020. http://dx.doi.org/10.7546/crabs.2020.03.17.

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Zuckerman, Kenneth S. Reparative Medicine: Production of Erythrocytes & Platelets from Human Embryonic Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada566171.

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Shore, Karen, and Paul Kirby. In Vivo Test for Chemical Induction of Micronucleated Polychromatic Erythrocytes in Mouse Bone Marrow Cells, Test Article: Ethylenediamine Dinitrate (EDDN). Fort Belvoir, VA: Defense Technical Information Center, August 2009. http://dx.doi.org/10.21236/ada518233.

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McElwain, Terry, Eugene Pipano, Guy Palmer, Varda Shkap, Stephen Hines, and Douglas Jasmer. Protection of Cattle Against Babesiosis: Immunization with Recombinant DNA Derived Apical Complex Antigens of Babesia bovis. United States Department of Agriculture, June 1995. http://dx.doi.org/10.32747/1995.7612835.bard.

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Bovine babesiosis caused by Babesia bovis continues to be a significant deterrent to global livestock production. Current control methods have both biological and technical drawbacks that have stimulated research on improved methods of vaccination. This BARD project has focused on characterization of candidate Babesia bovis vaccine antigens located in the apical complex, a unique group of subcellular organelles - including rhoptries, micronemes, and spherical bodies - involved in the invation of erythrocytes. Spherical bodies and rhoptries were partially purified and their contents characterized using monoclonal antibodies. Existing and newly developed monoclonal antibodies bound to antigens in the spherical body, rhoptry, merozoite membrane, and infected erythrocyte membrane. In an initial immunization study using biologically cloned strains, it was demonstrated that strain-common epitopes are important for inducing immune protection against heterologous challenge. Rhoptry-associated antigen 1 (RAP-1) had been demonstrated previously to induce partial immune protection, fulfilled criteria of broad interstrain B and T cell epitope conservation, and thus was further characterized. The RAP-1 gene family consists of at least two gene copies, is homologous to the RAP-1 gene family in B. bigemina, and contains significant sequence similarity to other erythroparasitic protozoan candidate vaccine antigens, including the apical membrane antigen of Plasmodium falciparum. A new RAP-1 monoclonal antibody was developed that inhibits merozoite growth in vitro, demonstrating the presence of a RAP-1 neutralization sensitive domain. Based on these observations, cattle were immunized with Mo7 (Mexico) strain recombinant RAP-1 representing one of the two gene copies. All cattle responded with variable levels of serum antibodies inhibitory to heterologous Israel strain merozoite growth in vitro, and RAP-1 specific T lymphocytes that proliferated when stimulated with either homologous or heterologous native parasite antigen. Minimal protection from clinical disease was present after virulent Israel (heterologous) strain B. bovis challenge. In total, the results support the continued development of RAP-1 as a vaccine antigen, but indicate that additional information about the native structure and function of both RAP-1 gene copies, including the relationship of conserved and polymorphic sequences to B and T cell lepitopes relevant for protection, is necessary for optimization of RAP-1 as a vaccine component.
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Sowers, Arthur E. The Electrofusion Mechanism in Erythrocyte Ghost Membranes. Fort Belvoir, VA: Defense Technical Information Center, November 1988. http://dx.doi.org/10.21236/ada203041.

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Bitensky, Mark W., and Tatsuro Yoshida. Use of an Erythrocyte Platform to Deploy Technology for Infection Detection. Fort Belvoir, VA: Defense Technical Information Center, September 2004. http://dx.doi.org/10.21236/ada426850.

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Pandolf, K. B., A. J. Young, M. N. Sawka, J. L. Kenney, and M. W. Sharp. Does Erythrocyte Infusion Improve Two-Mile Run Performance at High Altitude? Fort Belvoir, VA: Defense Technical Information Center, January 1995. http://dx.doi.org/10.21236/ada360256.

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Peltier, Tanner. Plasmodium falciparum protein export and erythrocyte remodeling in blood-stage malaria infections. Ames (Iowa): Iowa State University, January 2021. http://dx.doi.org/10.31274/cc-20240624-1130.

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Young, Andrew J., Michael N. Sawka, Stephen R. Muza, Robert Boushel, and Timothy Lyons. Autologous Erythrocyte Infusion May Not Attenuate the Decrement in VO2MAX at High Altitude,. Fort Belvoir, VA: Defense Technical Information Center, January 1995. http://dx.doi.org/10.21236/ada360197.

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