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Relevant bibliographies by topics / Erwin frink

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Academic literature on the topic 'Erwin frink'

Author: Grafiati

Published: 29 July 2024

Last updated: 30 July 2024

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Journal articles on the topic "Erwin frink"

1

Frank, Nelita. "Erwin Heinrich Frank: o antropólogo alemão que escolheu a Amazônia para viver." Boletim do Museu Paraense Emílio Goeldi. Ciências Humanas 5, no. 1 (April 2010): 147–52. http://dx.doi.org/10.1590/s1981-81222010000100010.

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Nota biográfica sobre Erwin Heinrich Frank (1950-2008), antropólogo alemão que trabalhou no Peru, Equador e Brasil. Neste último país, Frank foi professor na Universidade Federal do Pará e na Universidade Federal de Roraima. Escrita pela esposa do cientista, a nota apresenta elementos da formação, do trabalho de campo e da obra do antropólogo, incluindo o projeto de pesquisa que deu origem ao texto inédito publicado neste mesmo número da revista, sobre a etnografia de Theodor Koch-Grünberg.

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Bren, Frank. "Ripple Effect: the Theatrical Life of Max Linder." New Theatre Quarterly 25, no. 3 (August 2009): 241–54. http://dx.doi.org/10.1017/s0266464x09000426.

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By 1909 the French actor, playwright, and director Max Linder was probably the most popular male film star of his time, and his success as an innovative writer-actor of variety and revue continued until the outbreak of the First World War. But this followed five years of frustration in stage-ornament roles on the professional, ‘legitimate’ stage, and only after success in the cinema did his playlets, integrating filmed and live action, further enhance his fame in variety venues across Europe. After the war, and Linder's stints in Hollywood, his long descent into bouts of manic depression tragically began. But his theatrical spirit survives in the cine-stage works of the Prague theatre, Laterna Magika, and Frank Bren also discusses here his possible influence on the work of Erwin Piscator, and more surely on the spectacular Paris music-hall production, Jour de fête à l'Olympia, created by and starring Jacques Tati in 1961. This was plainly modelled on Linder's cinema-theatre creations of 1910–1914, with Tati and Pierre Etaix the outstanding successors to Max in French film comedy. Australian actor-author Frank Bren is currently writing a biography of Pierre Etaix, whose classic film comedies of the sixties are now being restored for international re-release – two of them paying discreet homage to Max Linder. Bren has written or co-written histories of Polish and Chinese cinema and theatre as well as articles for diverse international periodicals.

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Minteer, Christopher J., Kyra Thrush, Peter Niimi, Joel Rozowsky, Jason Liu, Mor Frank, Thomas McCabe, et al. "Abstract PR009: Revisiting the bad luck hypothesis: Cancer risk and aging are linked to replication-driven changes to the epigenome." Cancer Research 83, no. 2_Supplement_1 (January 15, 2023): PR009. http://dx.doi.org/10.1158/1538-7445.agca22-pr009.

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Abstract Aging is the leading risk factor for cancer. While some have proposed that the age-related accumulation of somatic mutations drives this relationship, it is likely not the full story. Here, we show that both aging and cancer share a common epigenetic replication signature, which we modeled from DNA methylation data in extensively passaged immortalized human cells in vitro and tested on clinical tissues. This epigenetic signature of replication – termed CellDRIFT – increased with age across multiple tissues, distinguished tumor from normal tissue, and was escalated in normal breast tissue from cancer patients. Additionally, within-person tissue differences were correlated with both predicted lifetime tissue-specific stem cell divisions and tissue-specific cancer risk. Overall, our findings suggest that age-related replication drives epigenetic changes in cells, potentially pushing them towards a more tumorigenic state. Citation Format: Christopher J. Minteer, Kyra Thrush, Peter Niimi, Joel Rozowsky, Jason Liu, Mor Frank, Thomas McCabe, Erin Hofstatter, Mariya Rozenblit, Lajos Pusztai, Kenneth Beckman, Mark Gerstein, Morgan E. Levine. Revisiting the bad luck hypothesis: Cancer risk and aging are linked to replication-driven changes to the epigenome [abstract]. In: Proceedings of the AACR Special Conference: Aging and Cancer; 2022 Nov 17-20; San Diego, CA. Philadelphia (PA): AACR; Cancer Res 2022;83(2 Suppl_1):Abstract nr PR009.

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Bakalov, A. S. "ON THE FORMATION OF GERMAN REALISM." Izvestiya of the Samara Science Centre of the Russian Academy of Sciences. Social, Humanitarian, Medicobiological Sciences 23, no. 77 (2021): 81–90. http://dx.doi.org/10.37313/2413-9645-2021-23-77-81-90.

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The relevance of research. In German literary criticism, there is no unambiguous definition of the phenomenon of literary realism, however, at the empirical level, it is understood as a literary system based on a mimetic-oriented depiction of reality, often critically comprehended and subjectively colored due to the norms and ideas that are taking shape in society. Research methodology. Complex and systematic methods of literature analysis are applied. In this article, the author comes to the conclusion that the realism of the turn of the XIX - early XX centuries. retains its main principles of artistic comprehension of the world, and at the same time the signs that do not allow talking about its dissolution in the eclectic picture of the emerging modernity. The main thing remains the disclosure of "the essence of life phenomena through their individualized generalization (typification)", analysis and specific historical logic of presentation Realism at the turn of the 19th - early 20th centuries. closely associated with such phenomena as regional literature, "new business-like", historical novel. On its basis, workers' and proletarian-revolutionary literature developed in many ways. In German literature of the twentieth century. realistic tendencies intensified in the times following the historical and political catastrophes, primarily after the two world wars lost by Germany. Realism played a significant role in the literature of the Weimar Republic (the works of E.M. Remarque, L. Feuchtwanger, L. Frank and others), while in contact with modernist and avant-garde trends (for example, with "new business-like"). Realism turned out to be no less significant after 1945, having equally influenced the formation of the literatures of West and East Germany (writers of the "group of 47", Erwin Strittmatter, "socialist realism", etc.). German realism, which emerged in the middle of the 19th century, was able to demonstrate its flexibility and ability to enter into alliances with other natural artistic directions, without losing its main specificity - the desire for materiality, the authenticity of personal and collective experience, as well as symbolizing the "obvious" with the goal of approaching the "true".

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Clarke, Colin. "H.E. Erwin Walther - H.E. ERWIN WALTHER Chamber Music: Neun Stücke für Klarinette und Klavier1; Rotationen [Entwurf] Version A2; Katenarien3; Schwebende Klänge4; Katenaria [Audiogramm]5; Rotationen [Entwurf] Version B2. 1,2Ib Hausmann (cl), 2,3,4Peter Bruns (vlc), 1,2,4,5Frank Gutschmidt (pno). NEOS 11209. - H.E. ERWIN WALTHER Vocal Music: Vier Lieder nach Spanischen Texten1; Drei Gesänge2; Sechs Lieder3; Zwei Lieder2; Vier heitere Lieder3; 12 Sprechlieder2. 1Wolfram Tessmer (bar), 2Joachim Vogt (ten), 3Yvonne Friedli (sop), Frank Gutschmidt (pno). NEOS 11210." Tempo 67, no. 266 (October 2013): 114–15. http://dx.doi.org/10.1017/s0040298213001137.

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Braithwaite, Dejana, Shama Karanth, Christiaan Leeuwenburgh, Todd M. Manini, Meghann Wheeler, Danting Yang, Livingstone Aduse-Poke, et al. "Abstract B001: Elevated all-cause and cardiovascular mortality in cancer survivors with sarcopenia." Cancer Research 83, no. 2_Supplement_1 (January 15, 2023): B001. http://dx.doi.org/10.1158/1538-7445.agca22-b001.

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Abstract Abstract: Sarcopenia, a condition characterized by the loss of muscle mass, strength and function with age, is highly prevalent in cancer survivors. The relationship between sarcopenia and prognosis among cancer survivors is not well understood. Methods: From the Third National Health and Nutrition Examination Survey (NHANES III), we identified 946 participants who were diagnosed with cancer (mean age 60.6 years); the most common disease sites were breast, prostate, colon and melanoma. We assembled an age, sex and race-matched cohort of 1,857 participants without cancer (mean age, 60.2 years). Sarcopenia was defined by appendicular lean mass and body height (men <7.26 kg/m2, women <5.45 kg/m2). Proportional-hazard models were used to assess whether sarcopenia was associated with all cause and CVD-specific time to death after taking into account baseline age, sex, race/ethnicity, smoking status, and energy intake for each cohort. In the cancer survivor cohort, models were additionally adjusted for a history of having more than one cancer and time since diagnosis. Mortality was ascertained from the National Center for Health Statistics Linked Mortality Files. Results: There were 321 deaths among cancer survivors (33.9% of the cohort) during a median follow-up of 10.5 years versus 495 deaths among participants without cancer (26.7% of the cohort) during a median follow-up of 10.9 years. Deaths from cardiovascular disease (CVD) were observed in 58 (6.1%) cancer survivors versus 122 (6.6%) participants without cancer. Overall, sarcopenia was more prevalent among cancer survivors versus the matched cohort (22.2% versus 19.7% respectively). Rates of CVD death were more than twice higher among cancer survivors with versus without sarcopenia (adjusted hazard ratio, 2.17, 95% confidence interval [CI], 1.16 to 4.05). All-cause mortality was 79% higher (adjusted hazard ratio, 1.79; 95% CI, 1.36 to 2.36) among cancer survivors with versus without sarcopenia. No significant associations were seen between sarcopenia and rates of death from all causes and CVD specifically among participants without cancer. Conclusion: Cancer survivors with sarcopenia have an increased risk of all-cause and CVD-specific mortality. In contrast, sarcopenia was not a major predictor of these outcomes in the matched cohort without cancer. Citation Format: Dejana Braithwaite, Shama Karanth, Christiaan Leeuwenburgh, Todd M. Manini, Meghann Wheeler, Danting Yang, Livingstone Aduse-Poke, Stephen Anton, Frank Penedo, Erin M. Siegel, Jonathan D. Licht, Dongyu Zhang. Elevated all-cause and cardiovascular mortality in cancer survivors with sarcopenia [abstract]. In: Proceedings of the AACR Special Conference: Aging and Cancer; 2022 Nov 17-20; San Diego, CA. Philadelphia (PA): AACR; Cancer Res 2022;83(2 Suppl_1):Abstract nr B001.

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Lin, Benjamin, Julia Ziebro, Kasey R. Skinner, Abigail Shelton, Erin Smithberger, Ryan Bash, Frank B. Furnari, and Ryan Miller. "Abstract 1125: Elucidating the transcriptomic response to EGFR-targeted therapy in EGFR-driven glioblastoma." Cancer Research 82, no. 12_Supplement (June 15, 2022): 1125. http://dx.doi.org/10.1158/1538-7445.am2022-1125.

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Abstract Glioblastoma (GBM) is the most common malignant brain tumor in adults with a dismal 15-month median survival. Standard therapy consisting of surgical resection, radiation, and temozolomide has been unsuccessful in meaningfully extending survival and preventing recurrence; thus, novel therapeutics are urgently needed. One proposed targeted treatment strategy for GBM involves using small molecule inhibitors against common genetic mutations. Epidermal growth factor receptor (EGFR) is the most commonly overexpressed oncogene in GBM (~56%). While EGFR tyrosine kinase inhibitors (TKI) have shown promise in other cancers, GBM clinical trials with EGFR TKI have failed. One reason for this failure is the development of adaptive therapeutic resistance. Understanding the mechanisms behind drug resistance is essential for the development of novel, effective therapeutics for GBM. To better understand adaptive resistance in GBM, we utilized two genetically engineered mouse astrocyte lines harboring common GBM mutations: Cdkn2a-/-, EGFRvIII (CEv3) and Cdkn2a-/-, Pten-/-, EGFRvIII (CEV3P). CDKN2A and PTEN are commonly deleted or otherwise inactivated tumor suppressor genes in GBM while the vIII variant of EGFR is the single most common oncogene mutation, making it an attractive therapeutic target. Cell lines CEv3 and CEv3P are both sensitive to neratinib, an irreversible second-generation EGFR TKI, at IC50 of 0.24μM and 0.13µM, respectively. To better understand adaptive response to neratinib treatment, we profiled the transcriptome with RNA sequencing at 0, 4, 24, and 48 hours. Our data shows that kinome rewiring is detectable after just 4 hours of treatment and sustained through 48 hours, with differential expression of 70% or more of the expressed kinome. We propose that differentially expressed kinases in response to neratinib can potentially activate alternative signaling pathways that bypass EGFR inhibition, which ultimately confers resistance to EGFR targeted therapy. Furthermore, we hypothesize that the epigenome is directly responsible for this adaptive kinome response through BRD4 dependent enhancer remodeling. Because dual therapy against EGFR and BRD4 has shown promising results in other cancers, targeting the epigenome through BRD4 represents a potential combination therapy with EGFR TKI in GBM. To profile BRD4-associated epigenomic changes, we used Cleavage Under Targets and Release Using Nuclease (CUT&RUN) to interrogate several regulatory marks (H3K4me1, K3K4me3, H3K27ac) in addition to BRD4. We seek to integrate RNA sequencing and CUT&RUN data to determine if kinases differentially expressed following neratinib treatment correlate with epigenetic marks for their respective enhancer(s). This work will provide insight into the adaptive resistance mechanism of EGFR driven GBM. Citation Format: Benjamin Lin, Julia Ziebro, Kasey R. Skinner, Abigail Shelton, Erin Smithberger, Ryan Bash, Frank B. Furnari, Ryan Miller. Elucidating the transcriptomic response to EGFR-targeted therapy in EGFR-driven glioblastoma [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 1125.

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Abi-Dargham, Anissa, Christer Allgulander, O. Gureje, Rachel Jenkins, R. N. Kalaria, Brian Leonard, F. Njenga, et al. "CINP 2005 Regional Meeting, 20-22 April 2005." South African Journal of Psychiatry 11, no. 1 (April 1, 2005): 10. http://dx.doi.org/10.4102/sajpsychiatry.v11i1.92.

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List of abstract titles and authors:1. Antipsychotics across the spectrum: An overview of their mechanisms of actionAnissa Abi-Dargham2. Recent advances in the treatment of common anxiety disordersChrister Allgulander3. Psychiatry in Africa: The myths, the realities and the exoticO Gureje4. Mental Health policy developmet in Kenya and Tanznia - A DFID funded projectRachel Jenkins, David Kima, Joseph Mbatia, Frank Njenga5. Vascular factors in Alzheimer's diseaseR N Kalaria6. Depression as an immunologically based Neurodegenerative disorderBrian Leonard7. Eight years of progress in Arican PsychiatryF Njenga8. Treatment of Depression: Present and futureDr R.M. Pinder9. Imaging the Serotinergic system in impulsive aggressive personality disorder patientsLarry J Siever, Antonia S. New, Mari Goodman, Monte Buchsbaum, Erin Hazlett, Karen O'Flynn, Anissa Abi-argham, Marc Lauelle10. Mode of action of Atypical antipsychotic rugs: Focus on A2 AdrnoceptorsT.H. SvenssonNeuroscience: Selected Abstracts11. Chemical odulato of Fronto-execuitive functions: Neropsychiatric implicationsTrevor W Robbins12. Neural mechanisms of recognition memory and of social atacntProf. G Horn13. Estrogen signling after estrogen receptor ß (ERß)Jan-Ake Gustafsson14. Getting Lost: Hippocampal contributions to agerelated memory dysfunctionCarol BarnesMetals and the brain: Selected abstracts15. Modeling the contributin of iron mismanagement to Neurological disordersProf. J R C Connor16. Aluminium-triggered fibrillogenesis of B-AmyloidsProf. PZ Zatta, Dr D Drago, Mr G Tognon, Dr F RicchelliPsychiatry in Africa:17. Psychosocal aspects of Khat use among the youth of NairobiMs T M Khamis18. PTSD among motor vehicle accident survivors, KenyaDr F A Ongecha19. Psychiatric relities within African context - The Kenyan case StudyProf. D M N Ndetei20. Adolescent-parenta interactions from infancy, Nairobi KenyaDr L K Ksakhala, Prof. D M N Ndetei21. Alcohol use ong young persons: A focus group study in Southwest NigeriaO A Obeijide22. Personality disorders and personality traits among tyoe 2 Diabetic patientsProf. O El Rufaie, Dr M Sabosy, Dr M S Abuzeid23. Association of traumatic experiences with depression among Nigerian adolescentsDr O Omigbodun, Dr K BakareMs O B Yusuf, Dr O Esan24. Prevalence of depression among women attending outpatient clinics in MalawiDr M Tugumisirize, Prof. Agn, Dr Musisi25. Non-fatal suicidalbehaviour at the Johannesburg General HospitalDr M Y H Moosa, Prof. F Y Jeenah, Dr A Pillay, Pof. M Vorstere, Dr R Liebenberg26. Integrating mental health into general primary health care - Uganda's experienceDr N Kigozi27. Depression among Nigerian survivors of stroke:Prevalance and associated factorsDr F.O Fatoye Dr M A Komolafe, Dr A. O Adewuya, Dr B.A. Eegunranti Prof. M.A. Lawal28. NGO Involvement mental health care -The way forwardDr Basangwa29. Prevalen of Attenton Deficit Hyperactivity sorder among African school childrenDr E KashalaProf. T Tylleskar, Dr I Elgen, Dr K Sommerfelt30. Barriers to effective mental health care in NigeriaMs L. Kola31. Quay of life evaluation in patients with HIV-I infection with respect to the impact of Phyttherapy (Traditional Herb in Zimbabwe)M B Sebit, S K Chandiwaa, A S Latif, E Gomo, S W Acuda, F Makoni, J Vushe

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Shelton, Abigail K., Erin Smithberger, Madison Butler, Allie Stamper, Ryan E. Bash, Steve P. Angus, Michael P. East, et al. "Abstract 3248: Acquired resistance to targeted inhibitors in EGFR-driven glioblastoma: Identification of dual kinase targets." Cancer Research 82, no. 12_Supplement (June 15, 2022): 3248. http://dx.doi.org/10.1158/1538-7445.am2022-3248.

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Abstract Glioblastoma (GBM) is a devastating primary brain tumor with <5% 5-year survival. CDKN2A deletion (~60%) and EGFR amplification (~55%) mutations frequently co-occur in these tumors. EGFR is an attractive therapeutic target due to its mutational frequency and availability of brain-penetrant tyrosine kinase inhibitors (TKI). Several EGFR TKI have failed clinically, due in part to acquired resistance. To mechanistically examine this type of resistance, we used a panel of ten genetically engineered mouse astrocyte lines harboring Cdkn2a deletion and EGFRvIII, a common (~30%) activating mutation. Resistant cells were generated via long-term exposure to gefitinib or erlotinib, either in vitro or in vivo. Both transcriptomic (RNAseq) and proteomic (multiplexed inhibitor beads with mass spectrometry, MIB-MS) experiments showed that cell lines clustered primarily by resistance phenotype and secondarily by method of resistance development when analyzed using principal component analysis and unsupervised hierarchical clustering. Kinases involved in proliferation and differentiation signaling pathways (ex: Pdgfrb, Pdk2, Tnik, Mapk3, Fgfr2) were upregulated in both RNAseq and MIB-MS datasets and thus represent putative druggable targets for dual kinase inhibition. Analysis of commonly upregulated kinases and their commercially available inhibitors revealed dovitinib and dasatinib, two brain-penetrant drugs approved for other cancer indications, as candidates for dual inhibition with an EGFR TKI. Resistant cell lines were all more sensitive to dovitinib than their drug-naïve parents; however, sensitivity to dasatinib varied. BLISS analysis of dual treatment with EGFR TKI neratinib and dasatinib or dovitinib revealed synergistic drug interactions in most lines. Additionally, drug-naïve cells displayed a robust, acute proteomic response to EGFR TKI afatinib over 48h, while the response of resistant lines was significantly blunted. This model system can also be used to examine acute vs. long-term kinome response to EGFR TKI. Acute response was examined by treating drug-naïve cells with afatinib over 48h, and long-term kinome rewiring was observed by comparing untreated cells to gefitinib- and erlotinib-resistant cell lines. Combing both RNAseq datasets for kinases upregulated in both drug-naïve cells over a 48h EGFR TKI treatment course and in resistant cell lines compared to their sensitive parents reveals 21 and 13 common kinases, respectively, at p<0.001. Eight of these kinases (Cdk19, Ddr1, Kalrn, Khk, Mapk3, Pink1, Tnik, Ulk2) appear in both the in vitro and in vivo datasets, indicating a conserved kinome response regardless of method of resistance generation. Overall, integrated kinome profiling in GBM models with defined mutational profiles provides a powerful framework to identify novel therapeutic targets that could significantly alter current treatment paradigms. Citation Format: Abigail K. Shelton, Erin Smithberger, Madison Butler, Allie Stamper, Ryan E. Bash, Steve P. Angus, Michael P. East, Gary L. Johnson, Michael E. Berens, Frank B. Furnari, Ryan Miller. Acquired resistance to targeted inhibitors in EGFR-driven glioblastoma: Identification of dual kinase targets [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 3248.

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Smithberger, Erin, Abigail K. Shelton, Ryan E. Bash, Madison K. Butler, Alex R. Flores, Allie Stamper, Steven P. Angus, et al. "Abstract 1857: Glioblastoma growth is suppressed dual inhibition of EGFR and CDK6 kinases." Cancer Research 82, no. 12_Supplement (June 15, 2022): 1857. http://dx.doi.org/10.1158/1538-7445.am2022-1857.

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Abstract Glioblastoma (GBM) is a malignant brain tumor that has proven difficult to treat, despite expressing promising targets such as EGFRvIII. EGFRvIII, a mutant version of the epidermal growth factor receptor (EGFR), is constitutively active and not present in normal brain cells. The tumor specificity of EGFRvIII and the frequent EGFR amplification seen in GBM make EGFR a potentially attractive therapeutic target; however, clinical studies have shown little to no efficacy for EGFR tyrosine kinase inhibitors (TKI). One reason for this lack of efficacy may be adaptive resistance. We used RNA sequencing and multiplexed inhibitor beads with mass spectrometry (MIB-MS) to study the transcriptomes and kinomes of genetically engineered mouse astrocytes to investigate this resistance and identify potential targets for dual inhibition. Out of 329 kinases detected by MIB-MS, 76 were differentially expressed between cells with Cdkn2a deletion (“C”) and cells that also overexpressed EGFRvIII (“CEv3”). Thirty-four of these kinases were overexpressed in the CEv3 cells relative to the parental C cells (log2 fold change of 5.6, p<1x105). One of these kinases, Cdk6, is also significantly overexpressed in CEv3 cells versus cells that have a further loss of function mutation of Pten (“CEv3P”) (log2 fold change of 5.6, p<1x105). Despite this significant differential expression at the protein level, RNA expression of Cdk6 was similar between cell lines. When these cells were treated with the CDK6 inhibitor abemaciclib, CEv3 cells were found to be significantly more sensitive to inhibition than C and CEv3P cells (IC50 of 0.10 μM vs. 0.18 μM and 0.23 μM, respectively). Similarly, when cells were treated with abemaciclib in combination with the EGFR inhibitor neratinib, there was significantly higher synergy in CEv3 cells than C or CEv3P cells. Genotypically-matched patient-derived xenograft (PDX) cells were assayed for EGFR-CDK6 inhibitor synergy and showed a similar pattern of greater synergy in cells with EGFRvIII overexpression and functional PTEN than cells with EGFRvIII overexpression and PTEN loss. CEv3 and CEv3P cells were orthotopically implanted into mice and treated with neratinib, abemaciclib, or a combination. In CEv3-injected mice, combination treatment led to significantly longer survival than either single agent or control treatment. However, in CEv3P-injected mice, no survival difference was seen between any of the treatment arms. Taken together, these data provide strong evidence that CDK6 is a promising target for combination treatment with EGFR inhibitors in glioblastoma. Citation Format: Erin Smithberger, Abigail K. Shelton, Ryan E. Bash, Madison K. Butler, Alex R. Flores, Allie Stamper, Steven P. Angus, Michael P. East, Gary L. Johnson, Michael E. Berens, Frank B. Furnari, Ryan Miller. Glioblastoma growth is suppressed dual inhibition of EGFR and CDK6 kinases [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 1857.

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Dissertations / Theses on the topic "Erwin frink"

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Wieland, Frank [Verfasser], Rainer [Akademischer Betreuer] Willmann, Thomas [Akademischer Betreuer] Hörnschemeyer, Erwin [Akademischer Betreuer] Bergmeier, and Michael [Akademischer Betreuer] Hoppert. "The phylogenetic system of Mantodea (Insecta: Dictyoptera) / Frank Wieland. Gutachter: Thomas Hörnschemeyer ; Erwin Bergmeier ; Michael Hoppert. Betreuer: Rainer Willmann." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2012. http://d-nb.info/1042846898/34.

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Kubitza, Erwin [Verfasser], Frank [Akademischer Betreuer] Schulz-Nieswandt, and Wolfgang [Akademischer Betreuer] Leidhold. "Organisationskultur und Genossenschaften. Unter besonderer Berücksichtigung des Genossenschaftsgründers Friedrich Wilhelm Raiffeisen und seines Werkes / Erwin Kubitza. Gutachter: Frank Schulz-Nieswandt ; Wolfgang Leidhold." Köln : Universitäts- und Stadtbibliothek Köln, 2013. http://d-nb.info/1047666464/34.

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Kubitza, Erwin Verfasser], Frank [Akademischer Betreuer] [Schulz-Nieswandt, and Wolfgang [Akademischer Betreuer] Leidhold. "Organisationskultur und Genossenschaften. Unter besonderer Berücksichtigung des Genossenschaftsgründers Friedrich Wilhelm Raiffeisen und seines Werkes / Erwin Kubitza. Gutachter: Frank Schulz-Nieswandt ; Wolfgang Leidhold." Köln : Universitäts- und Stadtbibliothek Köln, 2013. http://nbn-resolving.de/urn:nbn:de:hbz:38-54687.

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Frank, Katja [Verfasser], Erwin [Akademischer Betreuer] Schadel, and Andrea [Akademischer Betreuer] Bartl. "Existenzialistische Absurdität und kein Ausweg? Rausch und Kunst von der französischen Décadence bis zur Literatur der Moderne / Katja Frank. Betreuer: Erwin Schadel ; Andrea Bartl." Bamberg : University of Bamberg Press, 2012. http://d-nb.info/1058948032/34.

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Erkin, Aras Verfasser], Daniel [Akademischer Betreuer] [Wentzel, and Frank T. [Akademischer Betreuer] Piller. "The boundaries of co-production : how the interplay of self-printing and branding affects product valuation / Aras Erkin ; Daniel Wentzel, Frank Thomas Piller." Aachen : Universitätsbibliothek der RWTH Aachen, 2018. http://d-nb.info/1170394264/34.

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Erkin, Aras [Verfasser], Daniel [Akademischer Betreuer] Wentzel, and Frank T. [Akademischer Betreuer] Piller. "The boundaries of co-production : how the interplay of self-printing and branding affects product valuation / Aras Erkin ; Daniel Wentzel, Frank Thomas Piller." Aachen : Universitätsbibliothek der RWTH Aachen, 2018. http://d-nb.info/1170394264/34.

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Fronk, Sebastian [Verfasser], Dieter [Akademischer Betreuer] Senk, Wolfgang [Akademischer Betreuer] Bleck, and Erwin A. T. [Akademischer Betreuer] Wosch. "Beitrag zur Charakterisierung von Seigerungen sowie zum Reinheitsgrad von Stahlproben mittels optischer Emissionsspektrometrie (PDA-OES) und laserinduzierter Spektralanalytik / Sebastian Fronk ; Dieter Georg Senk, Wolfgang Peter Bleck, Erwin A. T. Wosch." Aachen : Universitätsbibliothek der RWTH Aachen, 2019. http://nbn-resolving.de/urn:nbn:de:101:1-2020052506412689933688.

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Fronk, Sebastian Verfasser], Dieter [Akademischer Betreuer] [Senk, Wolfgang [Akademischer Betreuer] Bleck, and Erwin A. T. [Akademischer Betreuer] Wosch. "Beitrag zur Charakterisierung von Seigerungen sowie zum Reinheitsgrad von Stahlproben mittels optischer Emissionsspektrometrie (PDA-OES) und laserinduzierter Spektralanalytik / Sebastian Fronk ; Dieter Georg Senk, Wolfgang Peter Bleck, Erwin A. T. Wosch." Aachen : Universitätsbibliothek der RWTH Aachen, 2019. http://d-nb.info/1210862751/34.

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Books on the topic "Erwin frink"

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1960-, Aurich Rolf, and Spaich Herbert 1950-, eds. Erwin Goelz alias Frank Maraun: Filmkritiker. [München]: Edition Text + Kritik, 2006.

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Chell, Erwin Frank. Life narrative of Erwin Frank Chell, or, Betts and Erv. Aurora, Colo. (13799-A E. Marina Dr., Aurora 80014): E.F. Chell, 1993.

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Otto, Frank, Gülch R. W, Jacob Ruthard, and International Erwin-Riesch-Symposium (1988 : Tübingen, Germany), eds. Evaluation of cardiac contractility: Based on the International Erwin-Riesch-Symposium dedicated to Otto Frank, Tübingen, June 2, 1988. Stuttgart: Gustav Fischer, 1990.

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Eva, König. "Para quê serve o conhecimento se eu não posso dividi-lo?": "Was nützt alles Wissen, wenn man es nicht teilen kann?" : Gedenkschrift für Erwin Heinrich Frank. Berlin: Gebr. Mann Verlag, 2013.

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5

United States. Congress. Senate. Committee on Armed Services. Nominations before the Senate Armed Services Committee, second session, 112th Congress: Hearings before the Committee on Armed Services, United States Senate, One Hundred Twelfth Congress, second session, on nominations of Adm. Samuel J. Locklear III, USN ; Ltg. Thomas P. Bostick, USA ; Hon. Frank Kendall III ; Hon. James N. Miller, Jr. ; Hon. Erin C. Conaton ; Mrs. Jessica L. Wright ; Mrs. Katharina G. McFarland ; Ms. Heidi Shyu ; Dr. Kathleen H. Hicks ; Mr. Derek H. Chollet ; Gen. Mark A. Welsh III, USAF ; Lt. Gen. John F. Kelly, USMC ; Ltg. Frank J. Grass, ARNG ; and Gen. Joseph F. Dunford, Jr., USMC ; February 9 ; March 29 ; April 26 ; July 19 ; November 15, 2012. Washington: U.S. G.P.O., 2013.

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Book chapters on the topic "Erwin frink"

1

Reece, Bob. "Frank the Poet." In Exiles from Erin, 151–83. London: Palgrave Macmillan UK, 1991. http://dx.doi.org/10.1007/978-1-349-21557-7_8.

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"13. Frank Erwin and UT Take On the Rag." In Once Upon a Time in Texas, 125–37. University of Texas Press, 2002. http://dx.doi.org/10.7560/771185-014.

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