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Books on the topic 'Epithelial mesenchyme transition'

Author: Grafiati
Published: 11 March 2023

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1

Pierre, Savagner, ed. Rise and fall of epithelial phenotype: Concepts of epithelial-mesenchymal transition. Georgetown, Tex., U.S.A: Landes Bioscience/Eurekah.com, 2005.

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2

Sŏnggyun'gwan Taehakkyo (Seoul, Korea). Sanhak Hyŏmnyŏktan. TGF-beta e ŭihan oncogenic Epithelial-Mesenchymal Transition (EMT) yubal inja rosŏ hangsanhwa tanbaekchildŭl ŭi yŏkhal kyumyŏng kwa kijŏn yŏn'gu =: Molecular characterization of distinct roles of anti-oxidative proteins and their signaling mechanism in the oncogenic epithelial-mesenchymal transition by TGF-beta. [Seoul]: Pogŏn Pokchi Kajokpu, 2009.

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3

Muthuswamy, Senthil, John Douglas Haley, and Raghu Kalluri. Abstracts of papers presented at the 2008 meeting on epithelial-mesenchymal transition: March 17-March 20, 2008. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 2008.

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4

Campbell, Kyra, and Eric Theveneau, eds. The Epithelial-to Mesenchymal Transition. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0779-4.

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5

Thompson, Erik W. Epithelial-mesenchymal transitions: new advances in development, fibrosis and cancer: 9 tables. Basel: Karger, 2011.

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6

Cheung, Albert. Pax3 and PAX3/FKHR induces cell aggregation and morphogenic mesenchymal-epithelial transition. Ottawa: National Library of Canada, 2002.

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7

Sivasubramaniyan, Kavitha. A novel antibody directed against SSEA-4 defines spontaneous epithelial-to-mesenchymal cell transition in human prostate cancer. [S.l: s.n.], 2014.

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8

Savagner, Pierre. Rise and Fall of Epithelial Phenotype: Concepts of Epithelial-Mesenchymal Transition (Molecular Biology Intelligence Unit). Springer, 2005.

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9

Savagner, Pierre. Rise and Fall of Epithelial Phenotype: Concepts of Epithelial-Mesenchymal Transition. Springer, 2010.

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10

Savagner, Pierre. Rise and Fall of Epithelial Phenotype: Concepts of Epithelial-Mesenchymal Transition. Springer London, Limited, 2008.

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11

Campbell, Kyra, and Eric Theveneau. Epithelial-To Mesenchymal Transition: Methods and Protocols. Springer, 2021.

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12

Campbell, Kyra, and Eric Theveneau. Epithelial-To Mesenchymal Transition: Methods and Protocols. Springer, 2020.

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13

Margret, Dr Jerlin, Dr Raghu Dhanapal, and Dr Goma Kumar. EPITHELIAL MESENCHYMAL TRANSITION IN ORAL SQUAMOUS CELL CARCINOMA”. LAP LAMBERT Academic Publishing, 2021.

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14

(Editor), E. W. Thompson, and Pierre Savagner (Editor), eds. Advances in Epithelial-mesenchymal Transitions (Cells Tissues Organs). Not Avail, 2007.

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15

The Epithelialto- Mesenchymal Transition (EMT) in Cancer. MDPI, 2018. http://dx.doi.org/10.3390/books978-3-03842-794-0.

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16

Ilter, Didem. The Role of ERK2 in Regulating Epithelial-Mesenchymal Transition. 2014.

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17

Newgreen, D. Epithelial-Mesenchymal Transitions: Part I (Journal - Acta Anatomica , Vol 154, No 1). S Karger Pub, 1996.

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18

Battistelli, Cecilia, Marc Diederich, Timothy Joseph Keane, Pilar Sandoval, Sergio Valente, and Raffaele Strippoli, eds. Molecular Mechanisms and New Therapeutic Targets in Epithelial to Mesenchymal Transition (EMT) and Fibrosis. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-502-3.

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19

Sheng, Guojun, and Erik W. Thompson. Epithelial-Mesenchymal Transition 2020 : Special Topic Issue: Cells Tissues Organs 2022, Vol. 211, No. 2. Karger AG, S., 2022.

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20

Sheng, Guojun, and Erik W. Thompson. Epithelial-Mesenchymal Transition 2020 : Special Topic Issue: Cells Tissues Organs 2022, Vol. 211, No. 2. Karger AG, S., 2022.

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21

Newgreen, D. F. Recent Progress in Epithelial-Mesenchymal Transitions: Development - Cancer - Pathology (Cells Tissues Organs (Formerly Acta Anatomica)). Edited by D. F. Newgreen. S. Karger A. G., 2005.

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22

Battistelli, Cecilia, Marc Diederich, Timothy Joseph Keane, Pilar Sandoval, Sergio Valente, and Raffaele Strippoli, eds. Molecular Mechanisms and New Therapeutic Targets in Epithelial to Mesenchymal Transition (EMT) and Fibrosis, Volume II. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-83250-256-3.

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23

Dettman, Robert, Juan Antonio Guadix, Elena Cano, Rita Carmona, and Ramón Muñoz-Chápuli. The multiple functions of the proepicardial/epicardial cell lineage in heart development. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0020.

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Abstract:
The epicardium is the outer cell layer of the vertebrate heart. In recent years, both the embryonic and adult epicardium have revealed unsuspected peculiarities and functions, which are essential for cardiac development. In this chapter we review the current literature on the epicardium, and describe its evolutionary origin, the mechanisms leading to the induction of its extracardiac progenitor tissue, the proepicardium, and the way in which the proepicardium is transferred to the heart to form the epicardium. We also describe the epicardial epithelial–mesenchymal transition from which mesenchymal cells originate, and the developmental fate of these cells, which contribute to the vascular, interstitial, valvular, and adipose tissue. Finally, we review the molecular interactions established between the epicardium and the myocardium, which are key for myocardial development and can also play a role in cardiac homeostasis. This chapter highlights how the epicardium has become a major protagonist in cardiac biology.
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24

Hastie, Nick, and Eve Miller-Hodges. WT1 and its disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0329_update_001.

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Abstract:
Mutations in the Wilms tumour suppressor gene, WT1, are associated with Wilms tumour in childhood. However, in addition WT1 has a key role in renal development, emerging roles in podocyte function, and a potential role in tissue regeneration. An understanding of WT1 is of increasing importance to clinical practice. WT1 is a complex gene with multiple isoforms. It is crucial for normal embryonic development, especially kidney development, where it is necessary for mesenchymal-to-epithelial transition to form the nephron. WT1 mutations lead to abnormalities in renal and genitourinary development, causing diseases such as Denys–Drash syndrome and Frasier syndrome as well as Wilms tumour. Recently, WT1 mutations have been recognized as a significant cause of isolated steroid-resistant nephrotic syndrome in children and young adults, without other associated syndromic features. WT1 continues to be expressed in adult podocytes, where it acts as a transcriptional activator of many podocyte genes. However, the specific role of WT1 in adult podocyte function remains poorly understood.
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