Relevant bibliographies by topics / Epithelial cells

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Epithelial cells'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Epithelial cells"

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Jin, Shiying. "Bipotent stem cells support the cyclical regeneration of endometrial epithelium of the murine uterus." Proceedings of the National Academy of Sciences 116, no. 14 (March 14, 2019): 6848–57. http://dx.doi.org/10.1073/pnas.1814597116.

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The endometrial epithelium of the uterus regenerates periodically. The cellular source of newly regenerated endometrial epithelia during a mouse estrous cycle or a human menstrual cycle is presently unknown. Here, I have used single-cell lineage tracing in the whole mouse uterus to demonstrate that epithelial stem cells exist in the mouse uterus. These uterine epithelial stem cells provide a resident cellular supply that fuels endometrial epithelial regeneration. They are able to survive cyclical uterine tissue loss and persistently generate all endometrial epithelial lineages, including the functionally distinct luminal and glandular epithelia, to maintain uterine cycling. The uterine epithelial stem cell population also supports the regeneration of uterine endometrial epithelium post parturition. The 5-ethynyl-2′-deoxyuridine pulse-chase experiments further reveal that this stem cell population may reside in the intersection zone between luminal and glandular epithelial compartments. This tissue distribution allows these bipotent uterine epithelial stem cells to bidirectionally differentiate to maintain homeostasis and regeneration of mouse endometrial epithelium under physiological conditions. Thus, uterine function over the reproductive lifespan of a mouse relies on stem cell-maintained rhythmic endometrial regeneration.
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Sun, Tung-Tien. "Altered phenotype of cultured urothelial and other stratified epithelial cells: implications for wound healing." American Journal of Physiology-Renal Physiology 291, no. 1 (July 2006): F9—F21. http://dx.doi.org/10.1152/ajprenal.00035.2006.

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The differentiation of cultured stratified epithelial cells can deviate significantly from that of normal epithelium, leading to suggestions that cultured cells undergo abnormal differentiation, or a truncated differentiation. Thus cultured epidermal and corneal epithelial cells stop synthesizing their tissue-specific keratin pair K1/K10 and K3/K12, respectively. The replacement of these keratins in the suprabasal compartment by K6/K16 keratins that are made by all stratified squamous epithelia during hyperplasia rules out a truncated differentiation. Importantly, the keratin pattern of in vivo corneal epithelium undergoing wound repair mimics that of cultured rabbit corneal epithelial cells. Although cultured urothelial cells continue to synthesize uroplakins, which normally form two-dimensional crystalline urothelial plaques covering almost the entire apical urothelial surface, these proteins do not assemble into crystals in cultured cells. Cultured epithelial cells can, however, rapidly regain normal differentiation on the removal of mitogenic stimuli, the use of a suitable extracellular matrix, or the transplantation of the cells to an in vivo, nonmitogenic environment. These data suggest that cultured epithelial cells adopt altered differentiation patterns mimicking in vivo regenerating or hyperplastic epithelia. Blocking the synthesis of tissue-specific differentiation products, such as the K1 and K10 keratins designed to form extensive disulfide cross-links in cornified cells, or the assembly of uroplakin plaques allows epithelial cells to better migrate and proliferate, activities that are of overriding importance during wound repair. Cultured urothelial and other stratified epithelial cells provide excellent models for studying the regulation of the synthesis and assembly of differentiation products, a key cellular process during epithelial wound repair.
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Son, Joung A., Joung A. Son, Taizo Hogetsu, Joung A. Son, Taizo Hogetsu, and Yil-Sung Moon. "The process of epithelial cell death in Pinus thunbergii caused by the pine wood nematode, Bursaphelenchus xylophilus." Nematology 16, no. 6 (2014): 663–68. http://dx.doi.org/10.1163/15685411-00002795.

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This study describes a new technique to investigate how the pine wood nematode (PWN), Bursaphelenchus xylophilus, kills pine epithelial cells. After inoculating PWN into 20-cm-long Pinus thunbergii stem cuttings and incubating for 1, 3 or 7 days, the cuttings were split into 2.5 cm segments. The segments were tangentially cut so that the epithelia of several cortical resin canals were exposed, and these were stained with Evans Blue for the detection of dead epithelial cells. While almost no dead epithelial cells were found in the cortical resin canals of non-PWN-inoculated control cuttings up to day 7 of the experiment, dead epithelial cells were distributed sparsely in the epithelium of cortical resin canals throughout pine cuttings inoculated with PWN 1, 3 and 7 days after inoculation. The sparse and sporadic distribution of dead pine cells in the epithelium suggested that individual PWN attacked one epithelial cell at a time with its stylet and migrated between attacks.
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Ramos, Tiago, Deborah Scott, and Sajjad Ahmad. "An Update on Ocular Surface Epithelial Stem Cells: Cornea and Conjunctiva." Stem Cells International 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/601731.

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The human ocular surface (front surface of the eye) is formed by two different types of epithelia: the corneal epithelium centrally and the conjunctival epithelium that surrounds this. These two epithelia are maintained by different stem cell populations (limbal stem cells for the corneal epithelium and the conjunctival epithelial stem cells). In this review, we provide an update on our understanding of these epithelia and their stem cells systems, including embryology, new markers, and controversy around the location of these stem cells. We also provide an update on the translation of this understanding into clinical applications for the treatment of debilitating ocular surface diseases.
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Lin, Y., L. Xia, J. D. Turner, and X. Zhao. "Morphologic observation of neutrophil diapedesis across bovine mammary gland epithelium in vitro." American Journal of Veterinary Research 56, no. 2 (February 1, 1995): 203–7. http://dx.doi.org/10.2460/ajvr.1995.56.02.203.

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SUMMARY Neutrophils are present in milk of cows as a means of suppressing invading pathogens during mastitis. However, the manner by which neutrophils traverse the secretory epithelia is still not clear: do they diapedese between epithelial cells or do they kill epithelial cells to gain entry into milk? We investigated the process of bovine neutrophil diapedesis across bovine mammary gland epithelium in vitro. The bovine mammary epithelial cell line mac-t, grown on collagen-coated filters, formed a confluent monolayer with characteristic tight junctions, basal-apical polarity, and functional barriers to the dye trypan blue. Neutrophils added on the apical surface of the monolayer were stimulated to diapedese across the epithelium by the addition of Staphylococcus aureus (107 colony-forming units/ml) to the basal compartment. Light and transmission electron microscopy revealed the series of events for neutrophil transmigration: accumulation of neutrophils on the surface of epithelial monolayer; projection of pseudopods into intercellular junctions and movement of neutrophils between adjacent epithelial cells; and reapproximation of the lateral epithelial cell membranes and reformation of the apical tight junctions after neutrophils crossed the epithelium. Morphologically, epithelial cell damage caused by neutrophil diapedesis was not evident. This in vitro model provides a two-dimensional epithelial sheet by which neutrophil diapedesis can be qualitatively studied under defined conditions. Results of the study suggest a major mode by which bovine neutrophils diapedese across the alveolar epithelia into milk during mastitis.
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Ohmoto, Makoto, Shugo Nakamura, Hong Wang, Peihua Jiang, Junji Hirota, and Ichiro Matsumoto. "Maintenance and turnover of Sox2+ adult stem cells in the gustatory epithelium." PLOS ONE 17, no. 9 (September 2, 2022): e0267683. http://dx.doi.org/10.1371/journal.pone.0267683.

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Continuous turnover of taste bud cells in the oral cavity underlies the homeostasis of taste tissues. Previous studies have demonstrated that Sox2+ stem cells give rise to all types of epithelial cells including taste bud cells and non-gustatory epithelial cells in the oral epithelium, and Sox2 is required for generating taste bud cells. Here, we show the dynamism of single stem cells through multicolor lineage tracing analyses in Sox2-CreERT2; Rosa26-Confetti mice. In the non-gustatory epithelium, unicolored areas populated by a cluster of cells expressing the same fluorescent protein grew over time, while epithelial cells were randomly labeled with multiple fluorescent proteins by short-term tracing. Similar phenomena were observed in gustatory epithelia. These results suggest that the Sox2+ stem cell population is maintained by balancing the increase of certain stem cells with the reduction of the others. In the gustatory epithelia, many single taste buds contained cells labeled with different fluorescent proteins, indicating that a single taste bud is composed of cells derived from multiple Sox2+ stem cells. Our results reveal the characteristics of Sox2+ stem cells underlying the turnover of taste bud cells and the homeostasis of taste tissues.
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Ferguson, C. A., A. S. Tucker, and P. T. Sharpe. "Temporospatial cell interactions regulating mandibular and maxillary arch patterning." Development 127, no. 2 (January 15, 2000): 403–12. http://dx.doi.org/10.1242/dev.127.2.403.

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The cellular origin of the instructive information for hard tissue patterning of the jaws has been the subject of a long-standing controversy. Are the cranial neural crest cells prepatterned or does the epithelium pattern a developmentally uncommitted population of ectomesenchymal cells? In order to understand more about how orofacial patterning is controlled we have investigated the temporal signalling interactions and responses between epithelium and mesenchymal cells in the mandibular and maxillary primordia. We show that within the mandibular arch, homeobox genes that are expressed in different proximodistal spatial domains corresponding to presumptive molar and incisor ectomesenchymal cells are induced by signals from the oral epithelium. In mouse, prior to E10, all ectomesenchyme cells in the mandibular arch are equally responsive to epithelial signals such as Fgf8, indicating that there is no pre-specification of these cells into different populations and suggesting that patterning of the hard tissues of the mandible is instructed by the epithelium. By E10.5, ectomesenchymal cell gene expression domains are still dependent on epithelial signals but have become fixed and ectopic expression cannot be induced. At E11 expression becomes independent of epithelial signals such that removal of the epithelium does not affect spatial ectomesenchymal expression. Significantly, however, the response of ectomesenchyme cells to epithelial regulatory signals was found to be different in the mandibular and maxillary primordium. Thus, whereas both mandibular and maxillary arch epithelia could induce Dlx2 and Dlx5 expression in the mandible and Dlx2 expression in the maxilla, neither could induce Dlx5 expression in the maxilla. Reciprocal cell transplantations between mandibular and maxillary arch ectomesenchymal cells revealed intrinsic differences between these populations of cranial neural crest-derived cells. Research in odontogenesis has shown that the oral epithelium of the mandibular and maxillary primordia has unique instructive signaling properties required to direct odontogenesis, which are not found in other branchial arch epithelia. As a consequence, development of jaw-specific skeletal structures may require some prespecification of maxillary ectomesenchyme to restrict the instructive influence of the epithelial signals and allow development of maxillary structures distinct from mandibular structures.
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Farman, N., C. R. Talbot, R. Boucher, M. Fay, C. Canessa, B. Rossier, and J. P. Bonvalet. "Noncoordinated expression of alpha-, beta-, and gamma-subunit mRNAs of epithelial Na+ channel along rat respiratory tract." American Journal of Physiology-Cell Physiology 272, no. 1 (January 1, 1997): C131—C141. http://dx.doi.org/10.1152/ajpcell.1997.272.1.c131.

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Na+ reabsorption from the epithelial surface of the respiratory tract plays a fundamental role in respiratory physiology. As in the epithelia of the renal collecting tubule and distal colon, Na+ enters across the luminal surface of respiratory epithelial cells via a recently cloned amiloride-sensitive multisubunit (alpha, beta, gamma) epithelial Na+ channel. We have examined the cellular expression at the mRNA level of the alpha-, beta-, and gamma-subunits of rat epithelial Na+ channel (rENaC) in the rat lung and upper airway epithelial cells using in situ hybridization. A large prevalence of alpha- and gamma-rENaC subunit expression (over beta) was found in tracheal epithelium, in a subpopulation of alveolar cells, presumably type II pneumocytes, and in nasal and tracheal gland acini. In contrast, equivalent levels of expression of all three subunits were detected in bronchiolar epithelium and in rat nasal gland ducts. This diversity of expression may reflect cell-specific functions of the amiloride-sensitive Na+ channel along the respiratory tract.
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Vermeer, Paola D., Lacey Panko, Philip Karp, John H. Lee, and Joseph Zabner. "Differentiation of human airway epithelia is dependent on erbB2." American Journal of Physiology-Lung Cellular and Molecular Physiology 291, no. 2 (August 2006): L175—L180. http://dx.doi.org/10.1152/ajplung.00547.2005.

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A clinical case documented a reversible change in airway epithelial differentiation that coincided with the initiation and discontinuation of trastuzumab, an anti-erbB2 antibody. This prompted the investigation into whether blocking the erbB2 receptor alters differentiation of the airway epithelium. To test this hypothesis, we treated an in vitro model of well-differentiated human airway epithelia with trastuzumab or heregulin-α, an erbB ligand. In addition, coculturing with human lung fibroblasts tested whether in vivo subepithelial fibroblasts function as an endogenous source of ligands able to activate erbB receptors expressed by the overlying epithelial cells. Epithelia were stained with hematoxylin and eosin and used for morphometric analysis. Trastuzumab treatment decreased the ciliated cell number by 49% and increased the metaplastic, flat cell number by 640%. Heregulin-α treatment increased epithelial height and decreased the number of metaplastic and nonciliated columnar cells, whereas it increased the goblet cell number. We found that normal human lung fibroblasts express transforming growth factor-α, heparin-binding epidermal-like growth factor, epiregulin, heregulin-α, and amphiregulin, all of which are erbB ligands. Cocultures of airway epithelia with primary fibroblasts increased epithelial height comparable to that achieved following heregulin-α treatment. These data show that erbB2 stimulation is required for maintaining epithelial differentiation. Furthermore, the mesenchyme underlying the airway epithelium secretes a variety of erbB ligands that may direct various pathways of epithelial differentiation.
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Yu, Qian, Liang-Chun Wang, Sofia Di Benigno, Daniel C. Stein, and Wenxia Song. "Gonococcal invasion into epithelial cells depends on both cell polarity and ezrin." PLOS Pathogens 17, no. 12 (December 1, 2021): e1009592. http://dx.doi.org/10.1371/journal.ppat.1009592.

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Neisseria gonorrhoeae (GC) establishes infection in women from the cervix, lined with heterogeneous epithelial cells from non-polarized stratified at the ectocervix to polarized columnar at the endocervix. We have previously shown that GC differentially colonize and transmigrate across the ecto and endocervical epithelia. However, whether and how GC invade into heterogeneous cervical epithelial cells is unknown. This study examined GC entry of epithelial cells with various properties, using human cervical tissue explant and non-polarized/polarized epithelial cell line models. While adhering to non-polarized and polarized epithelial cells at similar levels, GC invaded into non-polarized more efficiently than polarized epithelial cells. The enhanced GC invasion in non-polarized epithelial cells was associated with increased ezrin phosphorylation, F-actin and ezrin recruitment to GC adherent sites, and the elongation of GC-associated microvilli. Inhibition of ezrin phosphorylation inhibited F-actin and ezrin recruitment and microvilli elongation, leading to a reduction in GC invasion. The reduced GC invasion in polarized epithelial cells was associated with non-muscle myosin II-mediated F-actin disassembly and microvilli denudation at GC adherence sites. Surprisingly, intraepithelial GC were only detected inside epithelial cells shedding from the cervix by immunofluorescence microscopy, but not significantly in the ectocervical and the endocervical regions. We observed similar ezrin and F-actin recruitment in exfoliated cervical epithelial cells but not in those that remained in the ectocervical epithelium, as the luminal layer of ectocervical epithelial cells expressed ten-fold lower levels of ezrin than those beneath. However, GC inoculation induced F-actin reduction and myosin recruitment in the endocervix, similar to what was seen in polarized epithelial cells. Collectively, our results suggest that while GC invade non-polarized epithelial cells through ezrin-driven microvilli elongation, the apical polarization of ezrin and F-actin inhibits GC entry into polarized epithelial cells.
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Dissertations / Theses on the topic "Epithelial cells"

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Liu, Ke. "Role of second messengers in controlling growth patterns of corneal epithelial cells /." View thesis, 2002. http://library.uws.edu.au/adt-NUWS/public/adt-NUWS20030718.102224/index.html.

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Thesis (Ph. D.)--University of Western Sydney, 2002.
"This thesis is submitted in fulfilment of the requirements of the degree of Doctor of Philosophy to the University of Western Sydney School of Biological Sciences."t.p. Includes bibliographical references (leaves 138-150).
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Wu, Ka-kei. "Effects of polysaccharides on gastric epithelial cells." Click to view the E-thesis via HKUTO, 2003. http://sunzi.lib.hku.hk/hkuto/record/B31971386.

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胡嘉麒 and Ka-kei Wu. "Effects of polysaccharides on gastric epithelial cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31971386.

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Samadikuchaksaraei, Ali. "Derivation of pulmonary epithelial cells from stem cells." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422341.

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Hedengran, Faulds Malin. "Estrogen receptor signalling in mammary epithelial cells /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-936-6/.

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Robson, Ewan John Douglas. "Characterisation of epithelial-mesenchymal transition in murine mammary epithelial cells." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.616130.

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Kalair, Waseem. "Isolation and transplantation of murine intestinal stem cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0016/MQ59179.pdf.

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Tan, Chong Da. "Metabolic regulation of epithelial sodium channels in human airway epithelial cells." Thesis, St George's, University of London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.546781.

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Liu, Mengfei, and 刘梦菲. "Epithelial morphogenesis in three-dimensional cell culture system." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208611.

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In human body, the most common structures formed by epithelial cells are hollow cysts or tubules. The key feature of the cysts and tubules is the central lumen, which is lined by epithelial cell sheets. The central lumen allows material exchange, thus it is indispensable for the proper function of the epithelial tissue. In order to understand the way that the epithelial cells form highly specialized structure, an in vitro three-dimensional (3D) culture system was established. The Caco-2 cells were embedded in reconstituted basement membrane termed matrigel, whose biochemical constitution and physical properties were similar with the in vivo environment. The Caco-2 cells in matrigel spontaneously formed spherical multi-cell cysts, which could continuously expand. The confocal imaging and reconstruction technique helped understand the cyst structure and its formation process. The cysts developed central lumen surrounded by a layer of polarized cells. The apical domain of the cells faced the lumen, while the basal domain attached to the extracellular matrix. In the mature cysts, fluid was secreted by the cells around the lumen at the apical domain, and accumulated in the central lumen. The laser burning experiment showed that the intraluminal pressure was higher than the outer environment. The intact cell sheet was required to keep the engorged morphology of the cysts. The tension of the cell layer balanced with the intraluminal pressure. To investigate the effect of pressure on cyst development, the cysts were treated with cholera toxin, which could increase intraluminal pressure through promoting apical secretion. The time-lapse images showed that under cholera toxin treatment, the expansion of the cysts was accelerated. The high intraluminal pressure led to shape change of thecells, followed by increase in cell proliferation rate. Cholera toxin itself could not promote cell growth. In the3D cultured cysts, it was the increased intraluminal pressure that directly induced the acceleration of cell proliferation. It indicated that not only biochemical signals, but also mechanical force, contributed to epithelial morphogenesis. The mechanical stimulation could be converted into biochemical signals, further affect cell behavior. In response to mechanical stimulation, the focal adhesion kinase was activated in the cells around the cyst lumen. Furthermore, the microarray analysis suggested that multiple signaling pathways were altered under intraluminal pressure stimulation, including the pathways related to cytoskeleton organization, cell cycle and cell adhesion. Taken together, comparing with the conventional two-dimensional cell culture on rigid surface, the three-dimensional culture system provided the cells a more physiological environment. The 3D culture system allows the epithelial cells to form well-organized hollow structure. It is a convenient model for investigating the process and mechanism of epithelial morphogenesis.
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Biochemistry
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Doctor of Philosophy
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Ellis, Steven E. "Xenogeneic transplantation of immortalized bovine mammary epithelial cells." Thesis, Virginia Tech, 1994. http://hdl.handle.net/10919/46164.

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The focus of this research was to investigate the use of an immortalized bovine mammary epithelial cell line as a starting material for xenogeneic transplantation into the mammary glands of immunocompetent recipients. PSG-5 cells (a clonal derivative of the MAC-T cell line engineered to express ovine IGF-I ) were transplanted into the cleared mammary fat pads of recipient mice. Following transplantation, spheroidal cell structures were observed in the cleared mammary fat pads of immunocompetent control mice and in mice exposed to PSG-5 cells during fetal development. Spheroidal cell structures were not observed in glands that had not received cell transplants. The success of this xenogeneic transplantation prompted the development of a MAC-T cell clone expressing a bacterial β-galactosidase (βMAC-T's) for use as a histological marker protein. Studies were then performed to determine the most appropriate age and location for cell transplantation into ovine recipients. Between three and 12 weeks of age, parenchymal volume in the mammary glands collected from e\ves in this study (n=4/age group: 3,6,9, and 12 weeks) increased nearly lO-fold (3.8cm3 to 34.5cm3 ). Total gland volume increased approximately 5-fold (67.3cm3 to 316.2cm3 ). Based on these determinations of parenchymal and glandular volumes, we determined that transplantation should begin with lambs at about three weeks of age. This data provides a starting point to begin trials using βMAC-T cells, which have been engineered to express a histological marker protein, for transplantation into intact ovine and murine mammary glands.


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Books on the topic "Epithelial cells"

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1948-, Taub Mary, ed. Tissue culture of epithelial cells. New York: Plenum Press, 1985.

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S, Matlin Karl, and Valentich John D, eds. Functional epithelial cells in culture. New York: Liss, 1989.

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Ian, Freshney R., ed. Culture of epithelial cells. New York: Wiley-Liss, 1992.

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Freshney, R. Ian, and Mary G. Freshney, eds. Culture of Epithelial Cells. New York, USA: John Wiley & Sons, Inc., 2002. http://dx.doi.org/10.1002/0471221201.

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Taub, Mary, ed. Tissue Culture of Epithelial Cells. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4814-6.

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Gregory, Bock, Clark Sarah, and Ciba Foundation, eds. Junctional complexes of epithelial cells. Chichester: Wiley, 1987.

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Ghannoum, Mahmoud A. Candida adherence to epithelial cells. Boca Raton, Fla: CRC Press, 1990.

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Clare, Wise, ed. Epithelial cell culture protocols. Totowa, NJ: Humana Press, 2002.

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Gerencser, George A. Epithelial transport physiology. New York: Humana Press, 2010.

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Randell, Scott H., and M. Leslie Fulcher. Epithelial cell culture protocols. 2nd ed. New York: Humana Press, 2012.

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Book chapters on the topic "Epithelial cells"

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Hackett, Tillie-Louise, Stephanie Warner, Dorota Stefanowicz, and Darryl Knight. "Epithelial Cells." In Inflammation and Allergy Drug Design, 139–48. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444346688.ch10.

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Hoffman, Jill M., and Charalabos Pothoulakis. "Epithelial Cells." In Inflammation - From Molecular and Cellular Mechanisms to the Clinic, 437–56. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2017. http://dx.doi.org/10.1002/9783527692156.ch18.

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Saucedo, Leslie. "Epithelial Cells." In Getting to Know Your Cells, 19–24. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-30146-9_4.

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Devalia, Jagdish Laxman, Jia Hua Wang, and Robert James Davies. "Airway epithelial cells." In Cellular Mechanisms in Airways Inflammation, 245–62. Basel: Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8476-1_9.

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Rabsilber, Tanja M., and Gerd U. Auffarth. "Lens Epithelial Cells." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-35951-4_373-4.

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Draheim, Kyle M., and Stephen Lyle. "Epithelial Stem Cells." In Methods in Molecular Biology, 261–74. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-145-1_18.

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Rabsilber, Tanja M., and Gerd U. Auffarth. "Lens Epithelial Cells." In Encyclopedia of Ophthalmology, 1050–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_373.

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Olmo-Fontánez, Angélica M., and Jordi B. Torrelles. "Alveolar Epithelial Cells." In Advances in Host-Directed Therapies Against Tuberculosis, 247–55. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56905-1_16.

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Mizuguchi, Yoshiaki, Susan Specht, Kumiko Isse, John G. Lunz, and Anthony J. Demetris. "Biliary Epithelial Cells." In Molecular Pathology Library, 27–51. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7107-4_4.

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Karin, Norman J., Min I. N. Zhang, E. Radford Decker, and Roger O’Neil. "Signaling pathways regulating ion transport in polarized cells." In Epithelial Transport, 256–74. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1495-7_12.

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Conference papers on the topic "Epithelial cells"

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Roshal, D. S., K. K. Fedorenko, K. Azzag, S. B. Roshal, and S. Bagdighyan. "ANALYSIS AND MODELING OF THE TOPOLOGY OF CANCEROUS, HEALTHY AND NON-PROLIFERATIVE EPITHELIUM." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-210.

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The topological features of epithelial monolayers with different rates of cell division are analyzed, using monolayers of HeLa, HCerEpiC, COS cells, ascidian epithelium. It is shown that the topological defectiveness of the monolayer increases with an increase in the rate of cell division. Modeling of the structure and growth processes of the epithelia was carried out.
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Futterman, Matthew, and Evan A. Zamir. "A Model for Epithelial Migration and Wound Healing in the Avian Embryo." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19565.

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It is increasingly clear that (collective) migration of epithelia plays an important role in morphogenesis and wound healing [6]. One of the interesting phenomena about epithelial migration is that the leading edge of the epithelia displays characteristics of both epithelia and cells undergoing EMT (epithelial-to-mesenchymal transition), so-called “partial” EMT. Developmental models in Drosophila and zebrafish have become important for studying signaling pathways involved in epithelial migration in recent years, but it is difficult to study the biomechanics of these systems. [2] Here, we revisit a little-used developmental model originally characterized by Chernoff [3] over two decades ago, which uses the area opaca (AO) of the chick embryo, an extraembryonic epithelium in birds which normally functions to spread across and encompass the nutritive yolk in a process called epiboly. We believe this model will be useful for studying epithelial migration because it is easily accessible and can be separated from the embryo to control the biomechanical environment.
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Molladavoodi, Sara, John B. Medley, Maud Gorbet, and H. J. Kwon. "Mechanotransduction in Corneal Epithelial Cells." In ASME 2013 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/imece2013-65406.

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Mechanical properties of the cornea can be affected by diseases such as keratoconus. In keratoconus, a decrease in both thickness and rigidity of the cornea is observed. It is currently not clear whether and how changes in mechanical properties of the cornea are associated with corneal epithelial cell behavior. In the present study, polyacrylamide (PAA) gels with different elastic moduli have been prepared and human corneal epithelial cells (HCECs) have been cultured on them. To investigate the effect that changes in elastic modulus may have on adhesion and migration of corneal epithelial cells, actin filament organization and expression of adhesion molecules were characterized. It was found that HCECs actin filament organization improves with increasing substrate stiffness and integrin α3 expression significantly increases on more compliant substrates.
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Koo, J., C. Kim, J. Lee, K. Kim, and J. Yoon. "Regeneration of Airway Epithelium Using Autologous Epithelial Cells." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2012.

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Yamauchi, Y., T. Kohyama, S. Kamitani, S. Kawasaki, M. Desaki, K. Takami, H. Takizawa, and T. Nagase. "Epithelial Mesenchymal Transition Modulates the Cell Proliferation of Lung Epithelial Cells." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5306.

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Chen, Wen-Ling, Yi-Shan Chung, Yu-Wei Chiou, Ming-Jer Tang, and Ming-Long Yeh. "Adhesion strengths of normal epithelial cells and epithelial mesenchymal transition cells by using single-cell force spectroscopy." In 2011 IEEE International Conference on Nano/Micro Engineered and Molecular Systems (NEMS). IEEE, 2011. http://dx.doi.org/10.1109/nems.2011.6017402.

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Brockmeyer, T., L. Pham, K. Zscheppang, S. Murray, Z. Borok, H. Nielsen, and C. Dammann. "Epithelial-Mesenchymal Transition in Fetal Type-II Epithelial Cells." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5299.

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Tsarik, A. A., M. A. Kokhnyuk, P. V. Alkhovik, and M. Yu Yurkevich. "METHODOLOGICAL APPROACHES FOR ALVEOLAR EPITHELIAL CELL PRIMARY CULTURES OBTAINING." In SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2021. http://dx.doi.org/10.46646/sakh-2021-2-135-138.

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Evaluation of various proteolytic enzymes efficiency for disaggregation of lung tissue is carried out and an optimized method for alveolar epithelial cells isolation is presented. This method includes mechanical disaggregation of tissue followed by processing of explanations with 0.25% trypsin solution in combination with filtration of the cell suspension through pores with a diameter of 100 gm and 50 gm. The obtained cell cultures were characterized by high viability (more than 91%) and morphological heterogeneity. Along with actively dividing rounded cells, differentiated alveolar epithelial cells with cuboid or polygonal morphology, characterized by high secretory activity, were visualized.
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Li, Ping, Jing Wang, Chengxiang Dai, Yingxuan Shi, Suke Li, and Tony Liu. "Cell therapeutic potential of human amniotic epithelial cells." In INTERNATIONAL SYMPOSIUM ON THE FRONTIERS OF BIOTECHNOLOGY AND BIOENGINEERING (FBB 2019). AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5110810.

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Zielinski, Rachel, Cosmin Mihai, and Samir Ghadiali. "Multi-Scale Modeling of Cancer Cell Migration and Adhesion During Epithelial-to-Mesenchymal Transition." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53511.

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Cancer is a leading cause of death in the US, and tumor cell metastasis and secondary tumor formation are key factors in the malignancy and prognosis of the disease. The regulation of cell motility plays an important role in the migration and invasion of cancer cells into surrounding tissues. The primary modes of increased motility in cancerous tissues may include collective migration of a group of epithelial cells during tumor growth and single cell migration of mesenchymal cells after detachment from the primary tumor site [1]. In epithelial cancers, metastasizing cells lose their cell-cell adhesions, detach from the tumor mass, begin expressing mesenchymal markers, and become highly motile and invasive, a process known as epithelial-to-mesenchymal transition (EMT) (Fig. 1) [2]. Although the cellular and biochemical signaling mechanisms underlying EMT have been studied extensively, there is limited information about the biomechanical mechanisms of EMT. In particular, it is not known how changes in cell mechanics (cell stiffness, cell-cell adhesion strength, traction forces) influence the detachment, migration and invasion processes that occur during metastasis.
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Reports on the topic "Epithelial cells"

1

Reed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, July 1996. http://dx.doi.org/10.21236/ada315811.

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Reed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, July 1995. http://dx.doi.org/10.21236/ada300387.

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Reed, Steven I. Cell Cycle in Normal and Malignant Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, July 1998. http://dx.doi.org/10.21236/ada354074.

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Eirew, Peter D. Characterization of Human Mammary Epithelial Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2008. http://dx.doi.org/10.21236/ada501896.

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Eirew, Peter D. Characterization of Human Mammary Epithelial Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada516902.

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Baum, Linda G. Elucidation of a Novel Cell Death Mechanism in Prostate Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, December 2004. http://dx.doi.org/10.21236/ada432559.

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Baum, Linda G. Elucidation of a Novel Cell Death Mechanism in Prostate Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, December 2002. http://dx.doi.org/10.21236/ada413351.

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Baum, Linda G. Elucidation of a Novel Cell Death Mechanism in Prostate Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, December 2003. http://dx.doi.org/10.21236/ada421357.

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Sukumar, Saraswati. Cellular Plasticity of Epithelial Cells-Cause of Metastasis. Fort Belvoir, VA: Defense Technical Information Center, September 2005. http://dx.doi.org/10.21236/ada448411.

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Thorburn, Andrew M. Gene Targeting in Normal Human Breast Epithelial Cells. Fort Belvoir, VA: Defense Technical Information Center, December 2004. http://dx.doi.org/10.21236/ada432980.

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