Academic literature on the topic 'Epilepsy'
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Journal articles on the topic "Epilepsy"
Budiman, Marshen, Praevilia M. Salendu, and Johnny L. Rompis. "Pengaruh Riwayat Kejang Demam terhadap Kejadian Epilepsi pada Anak." e-CliniC 11, no. 1 (October 10, 2022): 19. http://dx.doi.org/10.35790/ecl.v11i1.37740.
Full textTri Utami, Melinda, Refani Adisti Rahmadini, Adelia Putri, Damayanti, Sri Ana, Rian Dzikrianto, and Yulia Hermawati. "REVIEW ARTIKEL : PENGGUNAAN OBAT ANTI EPILEPSI DI RUMAH SAKIT." Medimuh : Jurnal Kesehatan Muhammadiyah 4, no. 2 (December 28, 2023): 113–18. http://dx.doi.org/10.37874/mh.v4i2.632.
Full textBudiman, Marshen, Praevilia M. Salendu, and Johnny L. Rompis. "Pengaruh Riwayat Kejang Demam terhadap Kejadian Epilepsi pada Anak." e-CliniC 11, no. 1 (October 10, 2022): 19–26. http://dx.doi.org/10.35790/ecl.v11i1.44268.
Full textKurnia, Bonar, Edward Nangoy, and Jimmy Posangi. "Diet Ketogenik untuk Penyakit Epilepsi Resisten Obat." Jurnal Biomedik:JBM 13, no. 3 (April 10, 2021): 282. http://dx.doi.org/10.35790/jbm.13.3.2021.31946.
Full textSari, Putu Ratih Padmarini Gantari, Dewi Sutriani Mahalini, I. Nyoman Budi Hartawan, and Dyah Kanya Wati. "Tingkat kualitas hidup pasien anak epilepsi dengan QOLCE-16." Intisari Sains Medis 14, no. 3 (December 30, 2023): 1249–53. http://dx.doi.org/10.15562/ism.v14i3.1908.
Full textSert, Havva, Serap Çetinkaya, Ahmet Seven, and Meryem Pelin. "Knowledge levels of senıor nursing students about epilepsyHemşirelik son sınıf öğrencilerinin epilepsi hakkındaki bilgi düzeyleri." Journal of Human Sciences 14, no. 2 (May 30, 2017): 1966. http://dx.doi.org/10.14687/jhs.v14i2.4532.
Full textRamadhan, Muhammad Fitra, Prastiya Indra Gunawa, and Muhammad Arifin Parenrengi. "Faktor-Faktor Risiko Pasien Epilepsi Intraktabel pada Anak." Malahayati Nursing Journal 4, no. 12 (December 1, 2022): 3321–34. http://dx.doi.org/10.33024/mnj.v4i12.7518.
Full textPutri, Derby Ayudhia Utami Iskandar, and Riri Gusnita Sari. "KARAKTERISTIK DAN TATALAKSANA SINDROM EPILEPSI: SEBUAH TINJAUAN LITERATUR." JURNAL RISET KESEHATAN POLTEKKES DEPKES BANDUNG 16, no. 1 (February 29, 2024): 309–16. http://dx.doi.org/10.34011/juriskesbdg.v16i1.2525.
Full textSaputra, Dedyanto Henky. "Peran Diet Ketogenik dalam Tata Laksana Epilepsi." Cermin Dunia Kedokteran 49, no. 11 (November 1, 2022): 629–34. http://dx.doi.org/10.55175/cdk.v49i11.318.
Full textFitriyani, Fitriyani, Firly Windiyani, Tusy Triwahyuni, and Dharmawita Dharmawita. "TINGKAT PENGETAHUAN TENTANG PENYAKIT EPILESPI DAN PERTOLONGAN PERTAMA SAAT SERANGAN PADA MAHASISWA FAKULTAS KEDOKTERAN UNIVERSITAS MALAHAYATI ANGKATAN 2019 DAN 2021." Jurnal Ilmu Kedokteran dan Kesehatan 10, no. 6 (July 10, 2023): 2083–89. http://dx.doi.org/10.33024/jikk.v10i6.9427.
Full textDissertations / Theses on the topic "Epilepsy"
Madeira, Bianca Cecchele. "Avaliação de uma ferramenta online para indicar pacientes candidatos à cirurgia da epilepsia." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2015. http://hdl.handle.net/10183/139953.
Full textBackground: Epilepsy is one of the most commons neurological conditions in practice. Its prevalence in the general population is significant (about 2%). Among these patients, there are those that are drug-resistant (those who do not achieve control of their seizures with two suitable drugs optimally), which correspond to about 20-40%. These patients have inadequate control of their chronic condition and long-term treatment becomes unsatisfactory and expensive, regardless of the reduced quality of life of patients, the psychosocial impact, disability and increased risk of death. In addition, the fact that they often need caregivers usually familiar, which, therefore, also fail to produce. It is therefore imperative that cost-effective alternative treatments are available for these patients. The epilepsy surgery is a suitable option for selected patients, since it can substantially improve the quality of life of patients, in many cases making even the patient seizure free, in addition to reducing costs in the long term. The big problem is before surgery: lies in the fact that there is some difficulty in identifying and referring these patients for proper evaluation, either by ignorance on the part of physicians in general, and the lack of formal recommendations. Objectives: Evaluate an online tool to nominate patients candidates for epilepsy surgery, thus contributing to a larger facility for the purposes of clinical decision-making with better identification of these patients. Methods: We conducted a cross-sectional study evaluating the surgical indication in patients with focal epilepsy, as testing of an online tool available for this purpose. This work was performed with consecutive outpatients attending at the Epilepsy Clinic of the Hospital de Clínicas de Porto Alegre, in the period of January-April 2014. All patients included in the study had a diagnosis of epilepsy and their information was taken from the review of their medical records using a standardized questionnaire answered by researchers. The informations who were not registered, were collected during a routine visit. In total, 211 patients were evaluated. Results: In our study, first we conducted a prevalence analysis, finding a result of 56.9% of patients referred for surgical evaluation. In addition, we conducted an evaluation of variables that contribute to the indication or not of surgical treatment. At this stage, we found the frequency of seizures, the number of tested drugs and the presence of side effects such as variables with greater statistical significance for indication for surgical treatment. Conclusion: We believe that this work has great clinical relevance because it is a tool that can help in decision making for treatment, thus benefiting patients and reducing health care costs in the medium and long term.
Räty, Lena. "Living with epilepsy : young people with uncomplicated epilepsy and adults with newly debuted epilepsy /." Linköping : Univ, 2002. http://www.bibl.liu.se/liupubl/disp/disp2003/med764s.pdf.
Full textOliveira, Elton Pallone de. "Estudo crítico dos modelos experimentais em epilepsia espontânea do tipo ausência." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052011-135103/.
Full textEpilepsy is a very commom neurological disorders in world population. It is a chronicle condition highly disabling that affects both genera male and female independent of your age with a soft predominance in men and is more frequent in child under 2 years old and adult above 65 years old. The morbidity and mortality consequences of this disorder have many negative repercussions at society and global economy consequently. It is estimated about 60 to 100 millions of people around the world present any epileptic condition during their lives. According some researchers the epilepsy incidence varies about 11 to 131/100 thousand habitants for year and the prevalence between 1.5 to 30/1000 habitants for year, about this statics the higher values are found in developing countries, Latin America and Africa particularly. The Idiopathic Generalized Epilepsy (IGE) are about a third of all others kinds of epilepsies and are 15 to 20% more frequent tha n others types of epilepsies. The absences IGEs are strictly related with childhood and adolescence age group and sometimes can affect patients (2.8 5.7 of cases) with age higher than 15 years old. The physiopathology as the real causes of to occur and to reoccur of absences crises in adult age are not completely enlightened and represent a important challenge to epileptlogists. The IGEs (genetic etiology) are classified in: a) typical absence seizures, b) atypical absence seizures, c) absence seizures with special factors, d) mioclonics seizures, f) tonic mioclonic seizures, g) clone seizures, h) tonic seizures and i) atonic seizures. The treatment commonly is pharmacologic and seizures are controlled in major parts of cases although about a third of patients are refratory to anticonvulsants drugs. Having as principal finality the elucidation of basic mechanisms and help of development of effectiv e therapeutical approaches to these patients, researchers around the world spend many efforts to develop experimental models able to reproduce the phenomena that want to reproduce. Among the principal experimental models of IGEs, it is possible to cite: (1) the general epilepsy model induced by penicillin in cats; (2) the models of investigation of bicuculin; (3) induction by electrical stimulation; (4) Genetic Absence Epilepsy Rats of Strasbourg (GAERS); (5) cepa WAG/Rij; (6) the model of gamma-hydroxybutyric (GHB) and (7) mutant rats. These experimental models have promoted ways to researchers can to evaluate and quantify adequately the neuronal alterations that occur during epileptigenes process both in vitro or in vivo, making possible important advances in development of new therapeutical approaches and improvement in quality of life of epilepsy carriers
Hendriks, Marc P. H. "Memory deficits in epilepsy patients referred to tertiary epilepsy centres." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 2005. http://arno.unimaas.nl/show.cgi?fid=6293.
Full textSprissler, Ryan Steven, and Ryan Steven Sprissler. "Genetics of Epilepsy." Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/625894.
Full textCourville, Aaron. "Chaosmakers for epilepsy." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0003/MQ45985.pdf.
Full textNeri, Marina Liberalesso 1980. "Avaliação neuropsicológica de crianças com epilepsia rolândica = funções executivas." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309169.
Full textTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A epilepsia benigna da infância com pontas centrotemporais (EBIPCT) ou rolândica (ER) é a forma mais freqüente de epilepsia na infância e é classificada como sendo focal, genética e de evolução benigna. Apesar de não haver déficit intelectual, essas crianças podem apresentar alterações cognitivas específicas. O presente estudo teve como objetivos: identificar e descrever alterações de funções executivas em crianças com ER e verificar a influência de variáveis clínicas da epilepsia nas funções executivas. Os participantes foram submetidos à aplicação de testes de funções executivas. Foram incluídas crianças com diagnóstico clínico e eletroencefalográfico de ER. A maioria dos pacientes estava controlada e muitos deles encontravam-se sem medicação. Os resultados obtidos foram comparados com os de um grupo de crianças de um grupo-controle (GC) constituído de indivíduos normais, com idade e nível sócio-educacional semelhantes ao das crianças com epilepsia. Os dados coletados nos dois grupos foram analisados e comparados através dos testes estatísticos: Mann-Whitney, teste Qui-quadrado e teste Exato de Fisher. Os resultados mostraram que quanto aos dados numéricos crianças com ER obtiveram pior desempenho em cinco das seis categorias consideradas do Wisconsin Card Sorting Test (WCST): nº de erros, nº de erros perseverativos, nº de respostas perseverativas, nº de categorias completadas e nº de fracassos em manter o set. Considerando-se resultados categóricos crianças com ER obtiveram pior desempenho no Trail Making Test (TMT) parte B, no Teste de fluência verbal FAS e em três categorias do WCST: nº de erros, nº de erros perseverativos e nº de respostas perseverativas. Em relação à variável idade de início da epilepsia, crianças com início mais precoce apresentaram pior desempenho em um dos instrumentos utilizados quando comparadas com crianças com início mais tardio da epilepsia; em relação às demais variáveis da epilepsia, não foi possível qualquer tipo de análise; quanto às variáveis das crises, não houve diferença nos resultados obtidos pelos pacientes divididos em grupos. Diante desses resultados concluímos que: as crianças com ER apresentaram déficit em funções executivas quando comparadas com o grupo-controle; a bateria utilizada mostrou-se adequada para detecção das disfunções apresentadas pelas crianças com ER; em relação à variável da epilepsia idade de início, crianças com início mais precoce apresentaram pior desempenho do que crianças com início mais tardio da epilepsia; em relação às demais variáveis da epilepsia e às variáveis das crises, não houve diferença entre os dois grupos. Assim, nosso estudo segue a linha de raciocínio de que o termo benigno deve ser usado com cautela uma vez que nossos pacientes apresentaram déficits neuropsicológicos em funções executivas, independentemente da fase ativa da epilepsia e do uso de medicações
Abstract: The benign childhood epilepsy with centrotemporal spikes (EBIPCT) or rolandic epilepsy (RE) is the most common type of childhood epilepsy and is considered to be a focal, genetic and benign epilepsy. Although there is no intellectual deficit, these children may have specific cognitive impairments. This study aimed: to identify and describe changes in executive functions in children with RE and to verify the influence of clinical variables of epilepsy in executive functions. Participants were evaluated with tests to assess executive functions. We included children with clinical and EEG features of RE. Most patients were controlled and several were without medication. The results were compared with those of a control group (CG) comprised of normal subjects with age and socioeconomic level similar to that of the RE group. The data collected from both groups were analyzed and compared using statistical tests: Mann-Whitney, Chi-square and Fisher exact tests. The results showed that concerning the numerical data, children with RE had the worst performance in five of the six categories of the WCST: number of errors, number of perseverative errors, number of perseverative answers, number of completed categories and number of failures to maintain the set. Considering categorical results, children with RE had worse performance in Trail Making Test part B, Verbal fluency test FAS and three categories of the WCST: number of errors, number of perseverative errors and number of perseverative answers. Concerning the variable age of onset of epilepsy, children with earlier onset of epilepsy had a worse performance in one of the tools when compared with children with later onset of epilepsy; the other variables of epilepsy did not allow any analysis; considering seizure variables, there was no difference between the two groups. We conclude that: children with RE showed deficits in executive functions when compared with the control group; the set of tests was adequate to detect the dysfunctions presented by children with RE; in relation to the variable age of onset of epilepsy, children with earlier onset had a worse performance than children with later onset of epilepsy; concerning other epilepsy and seizure variables, there was no difference between the two groups. Thus, our study is in keeping with the idea that the term benign should be cautiously used since our patients had neuropsychological deficits in executive functions regardless of the active phase of epilepsy and the use of medications
Doutorado
Ciencias Biomedicas
Doutor em Ciências Médicas
Rocha, Renata Pina. "Epilepsia reflexa evacuatória: revisão de literatura e descrição de caso clínico com registro de crise ao vídeo-eletroencefalograma." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17160/tde-25042018-161435/.
Full textBackground: Reflex epilepsy is a condition in which all seizures are triggered by a specific stimulus. There are many known stimulus, including defecation which has rarely been reported. In the majority of cases, it presents with refractory seizures. Objective: review the literature on reflex epilepsy, as well as identify defecation as a possible trigger and report a case of a patient with reflex seizures triggered by defecation documented by video-electroencephalogram monitoring. Methods: literature review on reflex epilepsies and review of medical records of a patient with reflex epilepsy triggered by defecation. Results: we present a 10-year-old-boy patient, left-handed, whose epilepsy onset was at the age of four. Its seizures were characterized by episodes of loss of consciousness and atonia during defecation. Video-electroencephalogram monitoring had shown a seizure triggered by defecation, characterized by right head deviation, left upper limb automatisms, associated with consciousness impairment and afterwards nose wiping with the left hand. The ictal EEG demonstrated theta rhythmic activity in the left temporal lobe. The brain magnetic resonance was normal. The interictal SPECT identified left anterior temporal lobe mild hypoperfusion. The patient became seizure free after the treatment with valproic acid and carbamazepine. Neuropsychological and quality of life assessments suggested global impairment, before and after seizure control. Conclusion: this is the third case of epilepsy induced by defecation, and the second with seizures documented by videoelectroencephalogram. Similar to this case, both had suggested lateralization and localization over the left temporal region, suggesting this region as a potential epileptogenic zone in patients with this type of reflex epilepsy. In our patient, the treatment with valproic acid and carbamazepine was effective for the seizure control and may be an option to other patients with this condition.
Kemper, Birgit. "Neuropsychologische Untersuchung bei Frontallappenepilepsien ein Vergleich kognitiver Leistungen zwischen Patienten mit Frontal- und Temporallappenepilepsie im Rahmen der prächirurgischen Diagnostik /." Münster : Universität Münster, 1995. http://catalog.hathitrust.org/api/volumes/oclc/64528329.html.
Full textSantos, Renato Oliveira dos. "Investigando o papel de genes candidatos na epilepsia do lobo temporal mesi = genes PTPRM e IL1B = Investigating candidates genes in mesial temporal lobe epilepsy : PTPRM and IL1B genes." [s.n.], 2015. http://repositorio.unicamp.br/jspui/handle/REPOSIP/312709.
Full textTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: As epilepsias formam um grupo de doenças neurológicas crônicas caracterizadas por crises epilépticas, as quais podem ser definidas como um distúrbio intermitente do sistema nervoso causado por descarga elétrica anormal, súbita e sincronizada dos neurônios cerebrais. A epilepsia de lobo temporal (ELT) é a mais frequente, representando aproximadamente 50% dos casos em adultos e tem como manifestação típica, a crise parcial complexa. Além disso, é frequentemente refratária ao tratamento medicamentoso. Os principais sintomas gerados pela ELT são predominantemente pelo acometimento das estruturas mediais do lobo temporal, sendo a ELT mesial (ELTM), a forma mais comum de ELT. Atualmente é ainda controversa a participação de fatores genéticos contribuindo na etiologia das epilepsias, principalmente da ELTM, que não teve até hoje nenhum gene inequivocamente associado a sua predisposição. O objetivo deste trabalho foi investigar o papel de dois genes candidatos: o PTPRM e o IL1B na predisposição à ELTM. Para tanto utilizamos as seguintes modalidades de estudo em pacientes com ELTM (i) estudo de associação genética através da genotipagem de polimorfismos de nucleotídeo único (SNPs) localizados nos referidos genes candidatos (esta etapa do estudo foi realizada durante o mestrado); (ii) quantificação dos transcritos de ambos os genes, por PCR em tempo real em tecido das estruturas mediais do lobo temporal (principalmente hipocampo) que foi obtido através da realização de cirurgia para tratamento das crises refratárias. (iii) Para o PTPRM, foi também realizada a localização do transcrito pela técnica de hibridação "in situ" em tecido hipocampal de pacientes e de controle. Além disso, como existem evidências do envolvimento do PTPRM em etapas importantes do desenvolvimento cerebral, e pouco se conhece da função específica desse gene no cérebro realizamos (iv) a quantificação do transcrito de PTPRM durante o desenvolvimento em cérebro de camundongos. (v) Finalmente, com o objetivo específico de avaliar se o aumento de expressão de IL1B no tecido hipocampal se refletia também na circulação realizamos a quantificação do transcrito e proteica do IL1B no plasma de pacientes com ELTM. Nossos resultados revelaram associação genética entre SNPs localizados em ambos os genes investigados e o fenótipo estudado. No entanto, em nenhum dos estudos uma variante funcional pode ser identificada. A quantificação dos transcritos em tecido hipocampal dos pacientes com ELTM indicou que ambos os genes PTPRM e IL1B estão hiper-regulados em pacientes quando comparados ao tecido controle. Não identificamos variação significativa na expressão do transcrito de PTPRM no cérebro de camundongos nas diferentes etapas de desenvolvimento. Não identificamos variação significativa na quantificação do transcrito e proteica de IL1B no plasma dos pacientes com ELTM quando comparados aos controles. Em conclusão, nossos resultados dos estudos de associação indicam um papel de PTPRM e de IL1B na predisposição à ELTM, porém não fomos capazes de encontrar uma variante funcional associada ao fenótipo. Corroborando o papel de ambos os genes nosso estudo de expressão gênica no tecido acometido indicou um aumento de expressão de ambos os genes. No entanto, o aumento de expressão de IL-1beta no tecido hipocampal não se traduziu pelo aumento no plasma dos pacientes. Finalmente, nosso estudo do perfil de expressão de PTPRM durante o desenvolvimento cerebral não aponta para um papel desse gene em etapas específicas do desenvolvimento
Abstract: The epilepsies are a group of chronic neurological disorders characterized by seizures, which can be defined as an intermittent disorder caused by an abnormal and sudden electrical discharge of neurons in the brain. Temporal lobe epilepsy (TLE) is the most frequent form, representing approximately 50% of cases in adults, and it is often refractory to drug treatment. The main symptoms in TLE are generated by the involvement of the medial temporal lobe structures, characterizing mesial TLE (MTLE). The contribution of genetic factor to MTLE it is still controversial and to date, no gene has been unequivocally associated with the predisposition to MTLE. Therefore, the aim of this study was to investigate the role of two candidate genes: PTPRM and IL1B in the predisposition to MTLE. To achieve this we use the following type study modalities in patients with MTLE (i) genetic association study by genotyping of single nucleotide polymorphisms (SNPs) located in these two candidate genes; (ii) quantification of the transcripts of both genes by real-time PCR in hippocampal tissue obtained from epilepsy surgery for the treatment of refractory seizures. (iii) For PTPRM we also performed in situ hybridization experiments in order to localize the transcript in hippocampal tissue from patients and controls. Furthermore, since there is evidence that PTPRM could be involved in key stages of brain development and little is known about the specific role of this gene in the brain, we performed (iv) quantification of its transcript during development in mouse brain. (v) Finally, with the specific objective of assessing whether the increase of IL1B expression in hippocampal tissue was also seen outside the central nervous system we quantified IL1B transcript and protein in plasma of patients with MTLE. Our results revealed genetic association between SNPs located in both genes and the phenotype. The quantification of transcripts in hippocampal tissue of patients with MTLE indicated that both genes are hyper-regulated when compared to control tissue. We did not find any significant variation in transcript expression of PTPRM in mouse brain during developed. In addition, no difference in transcript expression and protein levels of IL1B was observed in plasma of patients with MTLE. In conclusion, our results indicate an involvement of PTPRM and IL1B in the predisposition to MTLE; however, we are unable to find a functional variant associated with the phenotype. Corroborating the role of both genes in MTLE gene expression in affected tissue (hippocampus) indicated an up-regulation of both genes. However, the increase in IL1B expression in hippocampal tissue was not reflected by an increase of transcript or protein in plasma of patients with MTLE. Finally, our expression profile of PTPRM during brain development does not point to a role for this gene in specific stages of development
Doutorado
Fisiopatologia Médica
Doutora em Ciências
Books on the topic "Epilepsy"
Parkinson, Gill. Epilepsy. New York, NY: Continuum, 2006.
Find full textBazil, Carl W. Epilepsy. New York: Oxford University Press, 2011.
Find full textMcGowen, Tom. Epilepsy. New York: F. Watts, 1989.
Find full textKrishnamoorthy, Ennapadam S., Simon Shorvon, and Steven Schachter, eds. Epilepsy. Cambridge: Cambridge University Press, 2017. http://dx.doi.org/10.1017/9781139547918.
Full textMiller, John W., and Howard P. Goodkin, eds. Epilepsy. Oxford: John Wiley & Sons, 2014. http://dx.doi.org/10.1002/9781118456989.
Full textMcCandless, David W. Epilepsy. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-0361-6.
Full textService, Royal College of General Practitioners Library and Information. Epilepsy. London: The College, 1986.
Find full textParks, Peggy J. Epilepsy. San Diego, CA: ReferencePoint Press, 2009.
Find full textHopkins, Anthony. Epilepsy. New York, NY: Demos Publications Inc, 1987.
Find full textJohnstone, Gary. Epilepsy. London: Boxtree, 1995.
Find full textBook chapters on the topic "Epilepsy"
Berg, Charles. "Epilepsy and Hystero-Epilepsy." In Clinical Psychology, 195–208. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003251514-16.
Full textÖztürk, Şerefnur. "Epilepsy." In Neurological Disorders in Clinical Practice, 25–31. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23168-6_4.
Full textReilly, Colin, and Christopher Gillberg. "Epilepsy." In Comorbid Conditions Among Children with Autism Spectrum Disorders, 235–56. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19183-6_10.
Full textMahmoud, Sherif Hanafy. "Epilepsy." In Patient Assessment in Clinical Pharmacy, 225–34. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11775-7_17.
Full textHilton, David A., and Aditya G. Shivane. "Epilepsy." In Neuropathology Simplified, 113–17. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-14605-8_8.
Full textGilbert, Patricia. "Epilepsy." In The A-Z Reference Book of Childhood Conditions, 70–76. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-7098-5_16.
Full textHattier, Megan A., Lauryn M. Toby, and Lindsey W. Williams. "Epilepsy." In Comorbid Conditions in Individuals with Intellectual Disabilities, 195–236. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15437-4_7.
Full textEl-Hagrassy, Mirret M., Ana C. Texeira-Santos, and Felipe Fregni. "Epilepsy." In Neuromethods, 445–517. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7880-9_13.
Full textLee, Dong Soo, So Won Oh, and Myung-Chul Lee. "Epilepsy." In Clinical PET and PET/CT, 139–50. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0802-5_12.
Full textLehtimäki, Kai, and Jukka Peltola. "Epilepsy." In Fundamentals and Clinics of Deep Brain Stimulation, 235–47. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36346-8_15.
Full textConference papers on the topic "Epilepsy"
Sethi, P. K., and Nitin K. Sethi. "Epilepsy and Armed Forces." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694888.
Full textAgarwal, Ekta, S. Rajadhyaksha, K. Srivastava, and V. Kulkarni. "Three Staged Epidemiological Survey of Pediatric Epilepsy in a Rural Suburb of Pune." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694857.
Full textC., Soumya V., and Ramshekhar N. Menon. "Evolution of Electroclinical Characteristics of West Syndrome: A Hospital Based Retrospective Study." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694858.
Full textU., Saraf U., Asranna A., Menon R. N., Radhakrishnan A., Manju P., Vibina V. P., Cherian A., and Thomas S. V. "Electroclinical Predictors of Cognitive and Seizure Outcome in Children with Epileptic Encephalopathy Due to Electrical Status Epilepticus in Sleep (ESES)." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694859.
Full textChawla, Archita, Rajinder Bansal, Suman Sharma, Namita Bansal, Gagandeep Singh, Chirag Gupta, and Karan Chouhan. "An Ethnographic and Structured Assessment of Treatment-Seeking Attitudes and Behaviors of People with Epilepsy in the Community." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694860.
Full textSrivastava, Arpna, Aparna Dixit, Ramesh Dodamanni, Jyotirmoy Banerjee, Manjari Thipathi, and P. Sarat Chandra. "Expression Profile of Histone Deacetylases in Patients with Hippocampal Sclerosis." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694861.
Full textKulkarni, Chanda, Ranjana G., and G. R. K. Sarma. "An Evaluation of Factors Influencing Adherence to Antiepileptic Medications (AEDs): A Cross-sectional Hospital-Based Study—An Overview and Recommendations to Improve." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694862.
Full textD., Chavan M., Karamthoti M. B., and Kurra S. B. "Third Generation Cardioselective Beta Blocker Exhibits Significant Anticonvulsant Properties in Pentylenetetrazole Model in Wistar Albino Rats." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694863.
Full textD., Chavan M., Karamthoti M. B., and Kurra S. B. "Na+-K+-2Cl– Cotransport Inhibitors and their Effect on Induced Seizure Tests in Experimental Models." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694864.
Full textGupta, Chirag, Gagandeep Singh, Karan Chouhan, Archita Chawla, R. K. Setia, Suman Sharma, and Namita Bansal. "Correlation between Distance from Health Centre and Adherence to Clinic Appointments and Medication Procurement among People with Epilepsy in the Community." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694865.
Full textReports on the topic "Epilepsy"
Jiang, Huabei. Photoacoustic Imaging of Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, April 2014. http://dx.doi.org/10.21236/ada607157.
Full textLin, Ching-Yi. Magnetic Stimulation and Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada611602.
Full textJiang, Huabei. Photoacoustic Imaging of Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, April 2010. http://dx.doi.org/10.21236/ada613888.
Full textJiang, Huabei. Photoacoustic Imaging of Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, April 2013. http://dx.doi.org/10.21236/ada576914.
Full textJiang, Huabei. Photoacoustic Imaging of Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, April 2012. http://dx.doi.org/10.21236/ada587641.
Full textJiang, Huabei. Photoacoustic Imaging of Epilepsy. Fort Belvoir, VA: Defense Technical Information Center, April 2011. http://dx.doi.org/10.21236/ada549242.
Full textGirisankar Prema, Abinaya, Iyshwarya Bhaskar Kalarani, and Ramakrishnan Veerabathiran. Genetic aspects of epilepsy. Peeref, November 2022. http://dx.doi.org/10.54985/peeref.2211p2734634.
Full textArtac, Kaitlin. Catamenial Epilepsy: A Review. Ames (Iowa): Iowa State University, May 2023. http://dx.doi.org/10.31274/cc-20240624-345.
Full textPhongsavanh, Assua. Epilepsy: an uncurable disease? Ames (Iowa): Iowa State University, January 2021. http://dx.doi.org/10.31274/cc-20240624-631.
Full textDichter, Marc A. Preventing Epilepsy After Traumatic Brain Injury. Fort Belvoir, VA: Defense Technical Information Center, February 2008. http://dx.doi.org/10.21236/ada485727.
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