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Books on the topic 'Epigenetic biology'

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Published: 10 March 2023

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1

Epigenetic principles of evolution. London: Elsevier, 2012.

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2

Cabej, Nelson R. Epigenetic Principles of Evolution. Burlington: Elsevier Science, 2011.

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3

Torday, John, and William Miller. Cellular-Molecular Mechanisms in Epigenetic Evolutionary Biology. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-38133-2.

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4

C, Goodwin Brian, and Saunders P. T. 1939-, eds. Theoretical biology: Epigenetic and evolutionary order from complex systems. Baltimore: Johns Hopkins University Press, 1992.

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5

Hurd, Paul J., Adele Murrell, and Ian C. Wood. Epigenetic mechanisms in development and disease. London: Portland Press Limited, 2013.

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6

J, Lamb Marion, ed. Epigenetic inheritance and evolution: The Lamarckian dimension. Oxford: Oxford University Press, 1995.

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7

Evolution in four dimensions: Genetic, epigenetic, behavioral, and symbolic variation in the history of life. Cambridge, MA: MIT Press, 2004.

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8

1925-, Johnen A. G., and Albers B. 1953-, eds. The epigenetic nature of early chordate development: Inductive interaction and competence. Cambridge [Cambridgeshire]: Cambridge University Press, 1985.

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9

Epigenomics, from chromatin biology to therapeutics. Cambridge: Cambridge University Press, 2012.

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10

Manel, Esteller, ed. Epigenetics in biology and medicine. Boca Raton: Taylor & Francis, 2008.

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11

Epigenetics: The ultimate mystery of inheritance. New York: W.W. Norton, 2011.

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12

Müller-Sievers, Helmut. Self-generation: Biology, philosophy, and literature around 1800. Stanford, Calif: Stanford University Press, 1997.

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13

David, Epel, ed. Ecological developmental biology: Integrating epigenetics, medicine, and evolution. Sunderland, Mass., U.S.A: Sinauer, 2009.

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14

Epigenetics of aging. New York: Springer, 2010.

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15

Rollo, C. David. Phenotypes: Their epigenetics, ecology and evolution. London: Chapman & Hall, 1994.

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16

Phenotypes: Their epigenetics, ecology and evolution. London: Chapman & Hall, 1994.

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17

Melekhova, O. P. Svobodnoradikalʹnye prot︠s︡esy v ėpigenomnoĭ reguli︠a︡t︠s︡ii razvitii︠a︡. Moskva: Nauka, 2010.

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18

Heard, Edith. Épigénétique et mémoire cellulaire. Paris]: Collège de France, 2013.

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19

Cabej, Nelson R. Epigenetic Principles of Evolution. Academic Press, 2018.

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20

Cabej, Nelson R. Epigenetic Principles of Evolution. Elsevier Science & Technology Books, 2018.

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21

Jr, William Miller, and Professor John Torday. Cellular-Molecular Mechanisms in Epigenetic Evolutionary Biology. Springer International Publishing AG, 2020.

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22

Torday, John, and William Miller Jr. Cellular-Molecular Mechanisms in Epigenetic Evolutionary Biology. Springer International Publishing AG, 2021.

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23

Cabej, Nelson R. Building the Most Complex Structure on Earth: An Epigenetic Narrative of Development and Evolution of Animals. Elsevier, 2013.

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24

Cabej, Nelson R. Building the Most Complex Structure on Earth: An Epigenetic Narrative of Development and Evolution of Animals. Elsevier Science & Technology Books, 2013.

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25

Epigenetic Alterations in Oncogenesis Advances in Experimental Medicine and Biology. Springer, 2012.

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26

(Editor), Peter Saunders, ed. Theoretical Biology: Epigenetic and Evolutionary Order from Complex Systems. Edinburgh University Press, 1990.

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27

Goodwin, Brian. Theoretical Biology: Epigenetic and Evolutionary Order from Complex Systems. The Johns Hopkins University Press, 1992.

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28

Epigenetic Approaches to Allergy Research. Springer, 2013.

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29

Pascual, Marién, and Sergio Roa. Epigenetic Approaches to Allergy Research. Springer, 2013.

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30

Mastroeni, Diego F. An Epigenetics Perspective on Diseases of the Central Nervous System. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0011.

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In the next two decades epigenetics could revolutionize understanding and treatment of diseases of the central nervous system. New research already demonstrates that manipulation of epigenetic mechanisms in vivo and in vitro can ameliorate a host of pathogenic processes associated with neurodegenerative disorders such as Alzheimer’s disease (AD), Parkinson’s (PD), amyotrophic lateral sclerosis (ALS), Huntington’s (HD), and multiple sclerosis (MS), among others. These advances have come relatively rapidly for a field that is still in its infancy compared to the much longer history of epigenetics in developmental biology. Epigenetic modifications are all-encompassing, from nucleotides to amino acids. They are capable of altering transcriptional to biochemical activity in a consistent manner across thousands of genes and hundreds of biologic pathways, yet they can do so differentially even in individuals or cells with identical gene codes. As such, epigenetic modifications are likely to touch on virtually all the mechanisms described in this book.
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31

Lamb, Marion J., Kim Sterelny, Eva Jablonka, Wilson Robert A, and Anna Zeligowski. Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life. MIT Press, 2014.

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32

Lamb, Marion J., Eva Jablonka, and Anna Zeligowski. Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life. MIT Press, 2014.

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33

Lamb, Marion J., Kim Sterelny, Eva Jablonka, Wilson Robert A, and Anna Zeligowski. Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life. MIT Press, 2014.

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34

Genie in Your Genes: Epigenetic Medicine and the New Biology of Intention. Energy Psychology Press, 2014.

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35

The Genie in Your Genes: Epigenetic Medicine and the New Biology of Intention. Elite Books, 2007.

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36

The Genie In Your Genes Epigenetic Medicine And The New Biology Of Intention. Energy Psychology Press, 2009.

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37

Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life. MIT Press, 2014.

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38

Singh, Sandeep, and Dhiraj Kumar, eds. Protocols used in Molecular Biology. BENTHAM SCIENCE PUBLISHERS, 2020. http://dx.doi.org/10.2174/97898114393151200101.

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Protocols used in Molecular Biology is a compilation of several examples of molecular biology protocols. Each example is presented with a concise introduction, materials and chemicals required, a step-by-step procedure and troubleshooting tips. Information about the application of the protocol is also provided. The techniques included in this book are essential to research in the fields of proteomics, genomics, cell culture, epigenetic modification and structural biology. The protocols can also be used by clinical researchers (neuroscientists and oncologists, for example) for medical applications (diagnostics, therapeutics and multidisciplinary projects).
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39

Dean, Michael, and Karobi Moitra. Biology of Neoplasia. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0002.

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The term “cancer” encompasses a large heterogeneous group of diseases that involve uncontrolled cell growth, division, and survival, culminating in local invasion and/or distant metastases. Cancer is fundamentally a genetic disease at the cellular level. Tumors occur because clones of abnormal cells acquire multiple lesions in DNA, nearly always involving mutations, chromosomal rearrangements, and extensive alteration of the epigenome. Up to 10% of cancers also involve inherited germline mutations that are moderately to highly penetrant. Cancers begin as localized growths or premalignant lesions that may regress or disappear spontaneously, or progress to a malignant primary tumor. The somatic changes that drive abnormal growth involve activating mutations of specific oncogenes, inactivation of tumor suppressor genes, and/or disruption of epigenetic controls. The latter can result from methylation or the modification of histones and other proteins that affect the remodeling of chromosomes. Numerous non-inherited factors can cause cancer by accelerating these events.
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40

Divan, Aysha, and Janice A. Royds. 7. Molecular biology in the clinic. Oxford University Press, 2016. http://dx.doi.org/10.1093/actrade/9780198723882.003.0007.

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Environmental agents can cause genetic and epigenetic changes to DNA, the consequences of which lead to deregulation of cellular processes and pathways that cause disease. Genetic variation can either be inherited if acquired through the germline or non-heritable when the DNA changes occur in somatic (body) cells. ‘Molecular biology in the clinic’ discusses two key contemporary areas of clinical research that have benefited from an improved knowledge of their molecular basis: ageing and cancer. It shows that we are now better able to predict disease risk and design drugs that have higher clinical efficacy by targeting specific molecular pathways.
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41

Michel, Jean-Baptiste. Biology of vascular wall dilation and rupture. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0016.

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Arterial pathologies, important causes of death and morbidity in humans, are closely related to modifications in the circulatory system during evolution. With increasing intraluminal pressure and arterial bifurcation density, the arterial wall becomes the target of interactions with blood components and outward convection of plasma solutes and particles, including plasma zymogens and leukocyte proteases. Abdominal aortic aneurysms of atherothrombotic origin are characterized by the presence of an intraluminal thrombus (ILT), a major source of proteases, including plasmin, MMP-9, and elastase. Saccular cerebral aneurysms are characterized by the interaction of haemodynamics and arterial bifurcation defects, of either genetic or congenital origin. They also develop an intrasaccular thrombus, implicated in rupture. Aneurysms of the ascending aorta (TAAs) are not linked to atherothrombotic disease, and do not develop an ILT. The most common denominator of TAAs, whatever their aetiology, is the presence of areas of mucoid degeneration, and increased convection and vSMC-dependent activation of plasma zymogens within the wall, causing extracellular matrix proteolysis. TAA development is also associated with an epigenetic phenomenon of SMAD2 overexpression and nuclear translocation, potentially linked to chronic changes in mechanotransduction. Aortic dissections share common aetiologies and pathology (areas of mucoid degeneration) with TAAs, but differ by the absence of any compensatory epigenetic response. There are main experimental animal models of aneurysms, all characterized by the cessation of aneurysmal progression after interruption of the exogenous stimuli used to induce it. These new pathophysiological approaches to aneurysms in humans pave the way for new diagnostic and therapeutic tools.
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42

Dooves, Stephanie, Dwayne Holmes, Judith Wagner, and Vivi M. Heine. Induced Pluripotent Stem Cells in Brain Diseases: Understanding the Methods, Epigenetic Basis, and Applications for Regenerative Medicine. Springer London, Limited, 2012.

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43

Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life (Life and Mind: Philosophical Issues in Biology and Psychology). The MIT Press, 2006.

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44

Fabris, Flavia. Waddington’s Processual Epigenetics and the Debate over Cryptic Variability. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198779636.003.0012.

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This chapter reappraises Waddington’s processual theory of epigenetics and examines its implications for contemporary evolutionary biology. It focuses in particular on the ontological difference between two conflicting assumptions that have been conflated in the recent debate over the nature of cryptic variability: a substance view that is consistent with the modern synthesis and construes variability as a preexisting pool of random genetic variation; and a processual view, which derives from Waddington’s conception of developmental canalization and understands variability as an epigenetic process. The chapter also discusses how these opposing interpretations fare in their capacity to explain the genetic assimilation of acquired characters.
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45

Tollefsbol, Trygve. Epigenetics Methods. Elsevier Science & Technology, 2020.

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46

Tollefsbol, Trygve. Epigenetics Methods. Elsevier Science & Technology Books, 2020.

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47

Lamb, Marion J., and Eva Jablonka. Evolution in Four Dimensions: Genetic, Epigenetic, Behavioral, and Symbolic Variation in the History of Life (Life and Mind: Philosophical Issues in Biology and Psychology). The MIT Press, 2005.

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48

Political Biology: Science and Social Values in Human Heredity from Eugenics to Epigenetics. Palgrave Macmillan, 2016.

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49

Ringrose, Leonie. Epigenetics and Systems Biology. Elsevier Science & Technology Books, 2017.

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50

Epigenetics and Systems Biology. Elsevier Science & Technology Books, 2017.

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