Log in

Relevant bibliographies by topics / Eosinophilic meningoencephalitis / Journal articles

Journal articles on the topic 'Eosinophilic meningoencephalitis'

To see the other types of publications on this topic, follow the link: Eosinophilic meningoencephalitis.

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Eosinophilic meningoencephalitis.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Bansal, Sharad, Mukesh Gupta, Deepak Sharma, and Shweta Bansal. "A Rare Case of Ibuprofen-Induced Eosinophilic Meningitis in a 13-Year-Old Girl." Clinical Medicine Insights: Pediatrics 8 (January 2014): CMPed.S13829. http://dx.doi.org/10.4137/cmped.s13829.

Full text

Abstract:

Eosinophilic meningoencephalitis is based on clinical manifestations and microscopic identification of eosinophils present in cerebrospinal fluid (CSF). It is caused by a variety of helminthic infections with most common being angiostrongyliasis, gnathostomiasis, toxocariasis, cysticercosis, schistosomiasis, baylisascariasis, and paragonimiasis. Many case reports are there in which parasites have been found responsible, but there are rare reports of CSF eosinophilia associated with the use of drugs. We report a case of drug-induced (ibuprofen) eosinophilic meningitis in a healthy female who presented to us with severe headache and improved dramatically after drug withdrawal.

APA, Harvard, Vancouver, ISO, and other styles

2

Graeff-Teixeira, Carlos, Ana Cristina Arámburu da Silva, and Kentaro Yoshimura. "Update on Eosinophilic Meningoencephalitis and Its Clinical Relevance." Clinical Microbiology Reviews 22, no. 2 (April 2009): 322–48. http://dx.doi.org/10.1128/cmr.00044-08.

Full text

Abstract:

SUMMARY Eosinophilic meningoencephalitis is caused by a variety of helminthic infections. These worm-specific infections are named after the causative worm genera, the most common being angiostrongyliasis, gnathostomiasis, toxocariasis, cysticercosis, schistosomiasis, baylisascariasis, and paragonimiasis. Worm parasites enter an organism through ingestion of contaminated water or an intermediate host and can eventually affect the central nervous system (CNS). These infections are potentially serious events leading to sequelae or death, and diagnosis depends on currently limited molecular methods. Identification of parasites in fluids and tissues is rarely possible, while images and clinical examinations do not lead to a definitive diagnosis. Treatment usually requires the concomitant administration of corticoids and anthelminthic drugs, yet new compounds and their extensive and detailed clinical evaluation are much needed. Eosinophilia in fluids may be detected in other infectious and noninfectious conditions, such as neoplastic disease, drug use, and prosthesis reactions. Thus, distinctive identification of eosinophils in fluids is a necessary component in the etiologic diagnosis of CNS infections.

APA, Harvard, Vancouver, ISO, and other styles

3

Goldman-Yassen, Adam E., Anna Derman, Rebecca Pellett Madan, and Alireza Radmanesh. "A Worm’s Tale or Why to Avoid the Raccoon Latrine: A Case of Baylisascaris procyonis Meningoencephalitis." Case Reports in Radiology 2022 (August 21, 2022): 1–6. http://dx.doi.org/10.1155/2022/5199863.

Full text

Abstract:

The raccoon roundworm Baylisascaris procyonis (B. procyonis) may infect humans to cause severe or fatal meningoencephalitis, as well as ocular and visceral larva migrans. Young children are at greater risk for cerebral larva migrans with severe meningoencephalitis, and early empiric therapy may improve outcomes. Familiarity with characteristic brain imaging findings may prompt earlier diagnosis, particularly in the setting of CSF eosinophilia. We report a case of a 19-month-old boy who presented with truncal ataxia and was found to have peripheral and CSF eosinophilia. MRI demonstrated symmetric, confluent T2 hyperintense signal in the cerebral and cerebellar deep white mater, which helped differentiate B. procyonis meningoencephalitis from other infectious and non-infectious causes of eosinophilic meningoencephalitis. Early recognition and treatment of B. procyonis meningoencephalitis are important for improved outcomes, and careful review of neuroimaging can play a critical role in suggesting the diagnosis.

APA, Harvard, Vancouver, ISO, and other styles

4

Dorta-Contreras, Alberto Juan, Piotr Lewczuc, Elena Noris-García, María Teresa Interián-Morales, María Esther Magraner Tarrau, Bárbara Padilla-Docal, and Xiomara Escobar-Pérez. "sICAM-1 in meningoencephalitis due to Angiostrongylus cantonensis." Arquivos de Neuro-Psiquiatria 64, no. 3a (September 2006): 589–91. http://dx.doi.org/10.1590/s0004-282x2006000400011.

Full text

Abstract:

INTRODUCTION: Angiostrongylus cantonensis meningoencephalitis is an emergent disease in the Americas. METHOD: Twelve children suffering from eosinophilic meningoencephalitis due to this parasite aged between 6-10 years were studied. Cerebrospinal fluid (CSF) and serum samples were taken simultaneously in the first diagnostic puncture at admission. RESULTS: All cases showed typical findings on the routine CSF and serum analysis: increased CSF total protein, increased Q (CSF/serum) albumin accompanied by eosinophilia in CSF. No intrathecal synthesis of immunoglobulins was found. Mean serum and CSF sICAM-1 values were 337.4 and 3.97 ng/mL. Qalbumin and QsICAM-1 mean values were 4.1 and 6.2 respectively. In 50% of the patients an increased brain-derived fraction of sICAM-1 was found. CONCLUSION: It may be suggested that a dynamic of the sICAM-1 brain derived fraction is perhaps associated to the immune response in the evolution of the disease.sICAM-1 may be an agent in negative feedback for eosinophils passage through the blood-CSF barrier into the inflammatory brain response.

APA, Harvard, Vancouver, ISO, and other styles

5

Prociv, Paul, and James R. Tiernan. "Eosinophilic meningoencephalitis with permanent sequelae." Medical Journal of Australia 147, no. 6 (September 1987): 294–95. http://dx.doi.org/10.5694/j.1326-5377.1987.tb133461.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Mimoso, M. G., M. C. Pereira, M. H. Estevão, A. A. Barroso, and H. C. Mota. "Eosinophilic meningoencephalitis due toToxocara canis." European Journal of Pediatrics 152, no. 9 (September 1993): 783–84. http://dx.doi.org/10.1007/bf01954007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Du, Wen-Yuan, Jiunn-Wang Liao, Chia-Kwung Fan, and Kua-Eyre Su. "Combined Treatment with Interleukin-12 and Mebendazole Lessens the Severity of Experimental Eosinophilic Meningitis Caused by Angiostrongylus cantonensis in ICR Mice." Infection and Immunity 71, no. 7 (July 2003): 3947–53. http://dx.doi.org/10.1128/iai.71.7.3947-3953.2003.

Full text

Abstract:

ABSTRACT Angiostrongylus cantonensis is the major cause of eosinophilic meningoencephalitis cases in Taiwan. Mice were orally infected with 35 infective larvae. One group of mice were given a single dose of mebendazole (20 mg/kg of body weight) per os at various times and examined at 14 days postinfection (dpi) for worm recovery rate and pathological studies. A 94 to 97% reduction in worm recovery was observed when medication was given at 4 to 5 dpi. Sections of the brains revealed that untreated infected mice developed typical severe eosinophilic meningoencephalitis. Meninges of these mice were thickened by massive infiltration of eosinophils, whereas only moderate pathological change was observed in the brains of mice that were treated with mebendazole at 4 dpi. Infected mice that received daily injections of 10 ng of interleukin-12 (IL-12) only for various numbers of days also exhibited moderate pathological changes in the brain. Eosinophil infiltration in the brains of these mice was low, and severe mechanical injuries in the parenchyma were observed. Treatment with mebendazole in combination with IL-12, however, resulted in low levels of worm recovery and dramatic lessening of the eosinophilic meningitis. A reverse transcriptase PCR assay of mRNA expression in the brain also revealed that the use of IL-12 had shifted the immune response of the mouse from Th2 type to Th1 type. This study could be used in developing strategies for the treatment of human angiostrongylosis.

APA, Harvard, Vancouver, ISO, and other styles

8

Phan, Hai Thanh, Kiem Hao Tran, and Huu Son Nguyen. "Eosinophilic Meningitis due to Angiostrongylus cantonensis in Children." Case Reports in Neurology 13, no. 1 (March 19, 2021): 184–89. http://dx.doi.org/10.1159/000512809.

Full text

Abstract:

Meningoencephalitis is not a rare disease in children. However, eosinophilic meningitis due to <i>Angiostrongylus cantonensis</i> is unusual in the pediatric population. We describe the case of a 12-year-old girl from the central area of Vietnam with eosinophilic meningitis due to <i>A. cantonensis</i>. The patient lived in a rural area, where farming is widespread, and presented with fever and headache. Laboratory results showed peripheral eosinophilia, a cerebrospinal fluid white blood cell count of 730/mm<sup>3</sup> with 65% eosinophils. Cerebrospinal fluid ELISA was positive for <i>A. cantonensis</i>, and blood ELISA was positive for <i>A. cantonensis</i>. The presentation was consistent with a diagnosis of <i>A. cantonensis</i> eosinophilic meningitis. The patient recovered fully after administration of albendazole (200 mg/day for 2 weeks), as well as intravenous dexamethasone (0.6 mg/kg/day every 8 h) and mannitol (1.5 g/kg/day every 8 h) for the first 3 days, followed by 5 days of oral prednisolone (2 mg/kg/day).

APA, Harvard, Vancouver, ISO, and other styles

9

Hong, David S., Marc Bernstein, Candice Smith, Hayley Gans, and Richard J. Shaw. "Eosinophilic Meningoencephalitis: Psychiatric Presentation and Treatment." International Journal of Psychiatry in Medicine 38, no. 3 (September 2008): 287–95. http://dx.doi.org/10.2190/pm.38.3.e.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Ferreira, Lucas Fernandes, Filipe Tupinamba Di Pace, and Guilherme Diogo Silva. "A Woman With Eosinophilic Brainstem Meningoencephalitis." JAMA Neurology 79, no. 2 (February 1, 2022): 198. http://dx.doi.org/10.1001/jamaneurol.2021.4861.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Chen, C. Y., C. C. Kuo, C. P. Lo, M. Y. Huang, Y. M. Wang, and W. Y. Wang. "Eosinophilic meningoencephalitis caused by Angiostrongylus cantonesis." QJM 107, no. 7 (December 23, 2011): 573–75. http://dx.doi.org/10.1093/qjmed/hcr261.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Batmanian, J. J., and J. H. O'Neill. "Eosinophilic meningoencephalitis with permanent neurological sequelae." Internal Medicine Journal 34, no. 4 (April 2004): 217–18. http://dx.doi.org/10.1111/j.1444-0903.2004.00561.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Narasimhan, Manisha, Omar Shum, Robert Stevens, and Lynette Masters. "Eosinophilic meningoencephalitis due to angiostronglyus infection." Journal of Neurology, Neurosurgery & Psychiatry 88, no. 5 (May 2017): e1.4-e1. http://dx.doi.org/10.1136/jnnp-2017-316074.101.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Grosse, Pascal, Klaus Schmierer, and Joachim Schulz. "Diagnostic pitfalls in eosinophilic cryptococcal meningoencephalitis." Lancet Neurology 2, no. 8 (August 2003): 512. http://dx.doi.org/10.1016/s1474-4422(03)00488-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Shih, P. C., H. H. Lee, S. C. Lai, K. M. Chen, S. T. Jiang, Y. F. Chen, and S. J. Shiow. "Efficacy of curcumin therapy against Angiostrongylus cantonensis-induced eosinophilic meningitis." Journal of Helminthology 81, no. 1 (March 2007): 1–5. http://dx.doi.org/10.1017/s0022149x07182353.

Full text

Abstract:

AbstractAngiostrongylus cantonensis can invade the central nervous system, leading to human eosinophilic meningitis or eosinophilic meningoencephalitis. Curcumin is a natural product which has the effects of anti-inflammation, anti-oxidation and anti-carcinogensis, while the administration of curcumin has been reported to possibly relieve the symptoms of meningitis. The present study tested the potential efficacy of curcumin in A. cantonensis-induced eosinophilic meningitis of BALB/c mice. Assay indicators for the therapeutic effect included the larvicidal effect, eosinophil counts and matrix metalloproteinase-9 (MMP-9) activity in angiostrongyliasis. Eosinophils were mildly reduced in treatment groups compared with infected-untreated mice. However, there were no significant differences in larvicidal effects or MMP-9 activity. This study suggests that anti-inflammatory treatment with curcumin alone has low efficacy, but the treatment does not interfere with MMP-9 expression and is not useful for larvicidal effects. The possible reasons include low curcumin across the blood–brain barrier and also those larvae that survive stimulate MMP-9 production, which promotes blood–brain barrier damage, with leukocytes then crossing the blood–brain barrier to cause meningitis. Further studies will be required to test these possibilities.

APA, Harvard, Vancouver, ISO, and other styles

16

Kang, SangJoon, Jaeyoung Park, Hoe Jong Jeong, Jae-Jeong Joo, and Seungmin Kim. "A Neurotoxocariasis Case Manifesting Multiple Cerebral Infarction and Eosinophilic Meningoencephalitis." Journal of the Korean Neurological Association 39, no. 4 (November 1, 2021): 331–35. http://dx.doi.org/10.17340/jkna.2021.4.10.

Full text

Abstract:

Although Toxocara canis is known to cross the blood-brain barrier, central nervous system involvement is uncommon. Clinical manifestations vary and include cerebral infarction, meningoencephalitis, myelitis, vasculitis or seizure. However cerebral infarction and meningoencephalitis rarely occur simultaneously. We report a case of multiple cerebral infarction combined with eosinophilic meningoencephalitis in a patient with neurotoxocariasis. After control of increased intracranial pressure and treatment with albendazole and steroid, the patient’s clinical symptoms improved markedly.

APA, Harvard, Vancouver, ISO, and other styles

17

Chiong, Fabian, Andrew R. Lloyd, and Jeffrey J. Post. "Severe Eosinophilic Meningoencephalitis Secondary to Suspected Neuroangiostrongyliasis with a Good Clinical Outcome." Case Reports in Infectious Diseases 2019 (May 26, 2019): 1–4. http://dx.doi.org/10.1155/2019/4037196.

Full text

Abstract:

Angiostrongylus cantonensishas caused sporadic cases of eosinophilic meningoencephalitis in Sydney, Australia. We describe a 36-year-old man who presented subacutely with fevers, reduced level of consciousness, confusion, ophthalmoplegia, and urinary incontinence. He was diagnosed with severe eosinophilic meningoencephalitis secondary to suspectedAngiostrongylus cantonensisbased on clinical, serological, and radiological findings. The patient was treated with albendazole and prednisolone with full neurological recovery. Management of neuroangiostrongyliasis with anthelminthic is controversial as it is thought to cause worsened outcomes through inciting an inflammatory response as a result of parasite killing. We managed to successfully treat our patient using albendazole and prednisolone and achieved a good outcome.

APA, Harvard, Vancouver, ISO, and other styles

18

Parameswaran, K. "Case series of eosinophilic meningoencephalitis from South India." Annals of Indian Academy of Neurology 9, no. 4 (2006): 217. http://dx.doi.org/10.4103/0972-2327.29203.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Higuchi, Masanori, Yumiko Sakai, Miyuki Koyanagi, Yasuo Tsuda, and Seiji Motomura. "Eosinophilic meningoencephalitis in a case of rheumatoid arthritis." Japanese Journal of Clinical Immunology 21, no. 5 (1998): 198–205. http://dx.doi.org/10.2177/jsci.21.198.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Loibl, J., D. Thaller, R. van den Hoven, and B. Schwarz. "Idiopathic eosinophilic meningoencephalitis in a Dutch Warmblood gelding." Equine Veterinary Education 25, no. 4 (March 20, 2012): 166–70. http://dx.doi.org/10.1111/j.2042-3292.2012.00391.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Vidana, B., T. Floyd, C. Brena, R. Lyle, A. Carson, C. M. Hamilton, and M. Wessels. "First Case of Idiopathic Eosinophilic Meningoencephalitis in Sheep." Journal of Comparative Pathology 174 (January 2020): 169. http://dx.doi.org/10.1016/j.jcpa.2019.10.095.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Morassutti, Alessandra L., and Carlos Graeff-Teixeira. "Interface Molecules ofAngiostrongylus cantonensis: Their Role in Parasite Survival and Modulation of Host Defenses." International Journal of Inflammation 2012 (2012): 1–6. http://dx.doi.org/10.1155/2012/512097.

Full text

Abstract:

Angiostrongylus cantonensisis a nematode parasite that causes eosinophilic meningoencephalitis in humans. Disease presents following the ingestion of third-stage larvae residing in the intermediate mollusk host and disease manifests as an acute inflammation of the meninges characterized by eosinophil infiltrates which release a battery of proinflammatory and cytotoxic agents in response to the pathogen. As a mechanism of neutralizing these host defenses,A. cantonensisexpresses different molecules with immunomodulatory properties that are excreted or secreted (ES). In this paper we discuss the role of ES proteins on disease exacerbation and their potential use as therapeutic targets.

APA, Harvard, Vancouver, ISO, and other styles

23

Furugen, Makoto, Shin Yamashiro, Maki Tamayose, Yui Naha, Kazuya Miyagi, Chikara Nakasone, Teruhito Uchihara, et al. "Elsberg Syndrome with Eosinophilic Meningoencephalitis Caused by Angiostrongylus cantonensis." Internal Medicine 45, no. 22 (2006): 1333–36. http://dx.doi.org/10.2169/internalmedicine.45.1871.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Xie, Mei, Zhen Zhou, Suhua Guo, Zengqing Li, Hui Zhao, and Jiusheng Deng. "Next-generation sequencing specifies Angiostrongylus eosinophilic meningoencephalitis in infants." Medicine 98, no. 35 (August 2019): e16985. http://dx.doi.org/10.1097/md.0000000000016985.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Vidaña, B., T. Floyd, C. Brena, R. Lyle, A. Carson, C. M. Hamilton, and M. Wessels. "First Case of Idiopathic Eosinophilic Meningoencephalitis in a Sheep." Journal of Comparative Pathology 174 (January 2020): 58–62. http://dx.doi.org/10.1016/j.jcpa.2019.10.194.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Stroh, Gregory, and Cassandra Braun. "CHRONIC EOSINOPHILIC PNEUMONIA IN A PATIENT WITH AUTOIMMUNE MENINGOENCEPHALITIS." Chest 156, no. 4 (October 2019): A185. http://dx.doi.org/10.1016/j.chest.2019.08.252.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Dorta-Contreras, Alberto Juan, and Hansotto Reiber. "Intrathecal Synthesis of Immunoglobulins in Eosinophilic Meningoencephalitis Due to Angiostrongylus cantonensis." Clinical Diagnostic Laboratory Immunology 5, no. 4 (July 1, 1998): 452–55. http://dx.doi.org/10.1128/cdli.5.4.452-455.1998.

Full text

Abstract:

ABSTRACT Eosinophilic meningoencephalitis due to the nematodeAngiostrongylus cantonensis, which is endemic to Cuba, occurs in children and is due to accidental contact with soil snails. The course is less often fatal than in adult patients in southeastern Asia. Cerebrospinal fluid (CSF) and serum samples from 24 pediatric patients were analyzed and evaluated in CSF/serum quotient diagrams (Reiber graphs) to characterize the neuroimmunological response and the blood-CSF barrier dysfunction that occur in the course of the disease. At the time of the first diagnostic lumbar puncture, together with eosinophilic pleocytosis (1,920 ± 400 cells/μl), intermediate blood-CSF barrier dysfunction (i.e., an increased CSF/serum albumin quotient) with no intrathecal immunoglobulin G (IgG), IgA, and IgM class response was observed in all cases. Seven days later, at the time of early clinical recovery, the blood-CSF barrier dysfunction was normalized in 75% of the patients, but meanwhile, intrathecal immunoglobulin synthesis emerged in all cases, as either a two-class response (IgG and IgA in 85% of the patients) or a three-class response (IgG, IgA, and IgM; 30%). The fraction of eosinophilic cells (40%) remained large despite a decreasing total cell count. The neuroimmunological pattern of this inflammatory response to the parasite and its toxins is discussed with regard to the CSF patterns of other infectious diseases caused by bacteria or viruses.

APA, Harvard, Vancouver, ISO, and other styles

28

Henke, D., M. Vandevelde, D. Gorgas, J. Lang, and A. Oevermann. "Eosinophilic Granulomatous Meningoencephalitis in 2 Young Belgian Tervueren Shepherd Dogs." Journal of Veterinary Internal Medicine 23, no. 1 (January 2009): 206–10. http://dx.doi.org/10.1111/j.1939-1676.2009.0247.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Li, Hua, Xiao-Guang Chen, Jin-Bao Gu, and Feng Xu. "A Severe Eosinophilic Meningoencephalitis Caused by Infection of Angiostrongylus cantonensis." American Journal of Tropical Medicine and Hygiene 79, no. 4 (October 1, 2008): 568–70. http://dx.doi.org/10.4269/ajtmh.2008.79.568.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Ahn, Ari, Yong-Joon Choe, Jeonghyun Chang, Duckhee Kim, Heungsup Sung, Mi-Na Kim, Seok Ho Hong, Jina Lee, Mi-Sun Yum, and Tae-Sung Ko. "Chronic Eosinophilic Meningoencephalitis by Prototheca Wickerhamii in an Immunocompetent Boy." Pediatric Infectious Disease Journal 36, no. 7 (July 2017): 687–89. http://dx.doi.org/10.1097/inf.0000000000001552.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

BENNETT, PF, FJ ALLAN, WG GUILFORD, AF JULIAN, and CG JOHNSTON. "Idiopathic eosinophilic meningoencephalitis in Rottweiler dogs: three cases (1992–1997)." Australian Veterinary Journal 75, no. 11 (November 1997): 786–89. http://dx.doi.org/10.1111/j.1751-0813.1997.tb15651.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Bolender, E., MA Worsham-Frye, and M. Tandy. "Baylisacaris procyonis: A rare cause of eosinophilic meningoencephalitis in children." American Journal of the Medical Sciences 365 (February 2023): S225. http://dx.doi.org/10.1016/s0002-9629(23)00429-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Hawkins, Ian Keith, Daniel R. Rissi, Anibal G. Armién, Rebecca Penrose Wilkes, and Eman Anis. "Rabies-like neuronal inclusions in a dog with meningoencephalomyelitis and neuritis of unknown origin." Veterinary Record Case Reports 7, no. 2 (June 2019): e000759. http://dx.doi.org/10.1136/vetreccr-2018-000759.

Full text

Abstract:

A four-year-old intact female pointer dog from a hunting plantation in South Georgia, USA, developed acute neurological signs. The referring veterinarian suspected rabies or pseudorabies; the dog was euthanased and submitted to the Tifton Veterinary Diagnostic and Investigational Laboratory for necropsy. No gross anatomical changes were present. Portions of brain were submitted for rabies virus fluorescent antibody test with a negative result. Histopathology revealed a marked lymphoplasmacytic meningoencephalitis with numerous intracytoplasmic eosinophilic inclusions within neurons, still raising concerns for rabies meningoencephalitis. Rabies immunohistochemistry was then performed on the brain, yielding a negative result. Brain samples were also negative for canine distemper virus and herpesvirus (pan-herpesvirus primers) PCR. Electron microscopy did not reveal viral particles within the inclusions. Similar intraneuronal pseudoinclusions have been previously reported in non-rabid dogs and cats. Such inclusions are a diagnostic challenge, especially in animals with central neurological signs and/or meningoencephalitis.

APA, Harvard, Vancouver, ISO, and other styles

34

EL-BAHNASAWY, MAMDOUH, MOHAMMAD EL FEKY, AYMAN MORSY, MOUSA ISMAIL, and TOSSON MORSY. "EGYPTIAN EOSINOPHILIC AND INFECTIOUS MENINGOENCEPHALITIS AND THEIR IMPACT ON PSYCHOLOGICAL ASPECTS." Journal of the Egyptian Society of Parasitology 46, no. 1 (April 1, 2016): 67–80. http://dx.doi.org/10.21608/jesp.2016.88945.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Diaz, James H. "Recently Reemerging Helminthic Infections Causing Eosinophilic Meningoencephalitis: Neuroangiostrongyliasis, Baylisascariasis, and~Gnathostomiasis." Journal of Neuroparasitology 1 (2010): 1–14. http://dx.doi.org/10.4303/jnp/n100503.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

El-Bahnasawy, Mamdouh M. M., Mohammad Reda El Feky, and Ayman T. A. Morsy. "Egyptian Eosinophilic and Infectious Meningoencephalitis and Their Impact on Psychological Aspects." Journal of the Egyptian Society of Parasitology 46, no. 1 (April 2016): 67–80. http://dx.doi.org/10.12816/0026151.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Chen, An-Chih, Ling-Yuh Shyu, Yi-Chieh Lin, Ke-Min Chen, and Shih-Chan Lai. "Proteasome serves as pivotal regulator in Angiostrongylus cantonensis-induced eosinophilic meningoencephalitis." PLOS ONE 14, no. 8 (August 15, 2019): e0220503. http://dx.doi.org/10.1371/journal.pone.0220503.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

VIDAL, JOSE E., ANTONIO CARLOS SEGURO, and JAQUES SZTAJNBOK. "EOSINOPHILIC MENINGOENCEPHALITIS DUE TO TOXOCARA CANIS: CASE REPORT AND REVIEW OF THE LITERATURE." American Journal of Tropical Medicine and Hygiene 69, no. 3 (September 1, 2003): 341–43. http://dx.doi.org/10.4269/ajtmh.2003.69.341.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

Padilla-Docal, Barbara, Alberto J. Dorta-Contreras, Raisa Bu-Coifiu-Fanego, Hermes Hernández, Jesús Barroso, and Consuelo Sanchez-Martinez. "Intrathecal synthesis of IgE in children with eosinophilic meningoencephalitis caused by Angiostrongylus cantonensis." Cerebrospinal Fluid Research 5, no. 1 (2008): 18. http://dx.doi.org/10.1186/1743-8454-5-18.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Fox, Amy S., Kevin R. Kazacos, Nevenka S. Gould, Peter T. Heydemann, Chinnamma Thomas, and Kenneth M. Boyer. "Fatal Eosinophilic Meningoencephalitis and Visceral Larva Migrans Caused by the Raccoon AscaridBaylisascaris procyonis." New England Journal of Medicine 312, no. 25 (June 20, 1985): 1619–23. http://dx.doi.org/10.1056/nejm198506203122507.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Ko, R. C., S. W. Chan, K. W. Chan, K. Lam, M. Farrington, H. W. Wong, and P. Yuen. "Four documented cases of eosinophilic meningoencephalitis due to Angiostrongylus cantonensis in Hong Kong." Transactions of the Royal Society of Tropical Medicine and Hygiene 81, no. 5 (September 1987): 807–10. http://dx.doi.org/10.1016/0035-9203(87)90039-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Salvadori, C., M. Baroni, M. Arispici, and C. Cantile. "Magnetic resonance imaging and pathological findings in a case of canine idiopathic eosinophilic meningoencephalitis." Journal of Small Animal Practice 48, no. 8 (August 2007): 466–69. http://dx.doi.org/10.1111/j.1748-5827.2007.00400.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

JIN, Er-hu, Qiang MA, Da-qing MA, Wen HE, Ai-ping JI, and Cheng-hong YIN. "Magnetic resonance imaging of eosinophilic meningoencephalitis caused by Angiostrongylus cantonensis following eating freshwater snails." Chinese Medical Journal 121, no. 1 (January 2008): 67–72. http://dx.doi.org/10.1097/00029330-200801010-00013.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Dongol, Srijana, Shreema Shrestha, Narayan Shrestha, and J. Adhikari. "Clinical Profile and Outcome of Acute Encephalitis Syndrome in Dhulikhel Hospital of Nepal." Journal of Nepal Paediatric Society 32, no. 3 (February 23, 2013): 201–5. http://dx.doi.org/10.3126/jnps.v32i3.6683.

Full text

Abstract:

Introduction: Acute encephalitis syndrome (AES) is a constellation of clinical signs and or symptoms i.e. acute fever with acute change in mental status. AES may be present as encephalitis, meningoencephalitis or meningitis. It can be associated with severe complication, including impaired consciousness, seizure, limb paresis or death. Materials and Methods: Study consisted of retrospective analysis of hospital records of children up to 16 years of age admitted with diagnosis of AES in the department of Paediatrics Dhulikhel Hospital, Kathmandu University Teaching Hospital, Dhulikhel Kavre from January 2010 to December 2011. Results: During the two years (January 2010 to December 2011), 47 patients of AES were admitted. Among the admitted cases there were 34 male and 13 female patients. Meningitis cases were 29, encephalitis cases were 14 and 4 meningoencephalitis cases. Among the meningitis cases, viral meningitis accounted for 12, bacterial meningitis accounted for 15 and 1 tubercular meningitis.One was eosinopilic meningitis in which the causative organism was found to be fasciolosis by ELISA. Viral encephalitis was found to be the most common cause of encephalitis. Sensorineural hearing loss was seen in 3 cases, subdural effusion in 1 and hydrocephalus in 1. One patient had intracranial hemorrhage with hemiparesis as a complication of eosinophilic meningitis. Conclusion: Acute encephalitis syndrome is one of the most common causes of PICU admission in Dhulikhel hospital. Bacterial meningitis was common among the acute encephalitis syndrome followed by viral meningitis. One case of eosinophilic meningitis with intracranical hemorrhage and hemiparesis was found. Sensorineural hearing loss was found to be commonest complication. DOI: http://dx.doi.org/10.3126/jnps.v32i3.6683 J. Nepal Paediatr. SocVol.32(3) 2012 201-205

APA, Harvard, Vancouver, ISO, and other styles

45

Mayhew, IG, KE Hill, Y. Ahn, and BR Jones. "Can drug-induced aseptic meningitis account for some cases of eosinophilic meningitis/meningoencephalitis in dogs?" New Zealand Veterinary Journal 70, no. 3 (October 25, 2021): 184–85. http://dx.doi.org/10.1080/00480169.2021.1990154.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

MORASSUTTI, ALESSANDRA L., LISA N. RASCOE, SUKWAN HANDALI, ALEXANDRE J. DA SILVA, PATRICIA P. WILKINS, and CARLOS GRAEFF-TEIXEIRA. "Cross-reactivity of the 31 kDa antigen of Angiostrongylus cantonensis – Dealing with the immunodiagnosis of meningoencephalitis." Parasitology 144, no. 4 (November 21, 2016): 459–63. http://dx.doi.org/10.1017/s0031182016001918.

Full text

Abstract:

SUMMARYThe primary causative agent of eosinophilic meningoencephalitis (EoM) in endemic regions is the nematode Angiostrongylus cantonensis. The occurrence of EoM was previously restricted to countries in Southeast Asia and the Pacific Islands; however, more recently, it has been reported from other regions, including Brazil. The commonly used diagnosis is detection of specific antibody reactivity to the 31 kDa antigen, which is derived from female worm somatic extracts. Here we report the occurrence of cross-reactivity to this antigen in sera from other parasitic infections, especially those that may cause EoM, such as gnathostomiasis, toxocariasis, hydatidosis and strongyloidiasis. We also demonstrated that the cross-reactivity, in part, is dependent of the concentration of antigen used in Western blot assays. We discuss the importance of these findings on the interpretation of this test.

APA, Harvard, Vancouver, ISO, and other styles

47

Gardiner, C. H., Andrew E. Gutter, Donald K. Nichols, Daniel C. Anderson, Susan Wells, Lisa Fitzgerald, and Richard K. Harris. "Eosinophilic Meningoencephalitis Due to Angiostrongylus Cantonensis as the Cause of Death in Captive Non-Human Primates." American Journal of Tropical Medicine and Hygiene 42, no. 1 (January 1, 1990): 70–74. http://dx.doi.org/10.4269/ajtmh.1990.42.70.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Cardy, T. J. A., and I. Cornelis. "Clinical presentation and magnetic resonance imaging findings in 11 dogs with eosinophilic meningoencephalitis of unknown aetiology." Journal of Small Animal Practice 59, no. 7 (March 30, 2018): 422–31. http://dx.doi.org/10.1111/jsap.12837.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Sakaguchi, Kanako, Kenneth Kim, Ingeborg Langohr, Annabel G. Wise, Roger K. Maes, Gordon Pirie, Tokuma Yanai, Mohie Haridy, Lorrie Gaschen, and Fabio Del Piero. "Zebra-borne neurotropic equid herpesvirus 1 meningoencephalitis in a Thomson’s gazelle (Eudorcas thomsonii)." Journal of Veterinary Diagnostic Investigation 29, no. 4 (April 20, 2017): 548–56. http://dx.doi.org/10.1177/1040638717707000.

Full text

Abstract:

We describe the histopathologic, immunohistochemical, and molecular features of a case of meningoencephalitis in a Thomson’s gazelle ( Eudorcas thomsonii) naturally infected with zebra-borne equid herpesvirus 1 (EHV-1) and the implications for the molecular detection of zebra-borne EHV-1. A 4-y-old female Thomson’s gazelle was submitted for postmortem examination; no gross abnormalities were noted except for meningeal congestion. Microscopic evaluation demonstrated multifocal nonsuppurative meningoencephalitis with intranuclear eosinophilic and amphophilic inclusion bodies and EHV-9 antigen in neurons. PCR demonstrated the presence of a herpesvirus with a nucleotide sequence 99–100% identical to the corresponding sequences of zebra-borne EHV-1 and of EHV-9 strains. To determine whether EHV-1 or EHV-9 was involved, a PCR with a specific primer set for EHV-9 ORF59/60 was used. The sequence was identical to that of 3 recognized zebra-borne EHV-1 strains and 91% similar to that of EHV-9. This isolate was designated as strain LM2014. The partial glycoprotein G ( gG) gene sequence of LM2014 was also identical to the sequence of 2 zebra-borne EHV-1 strains (T-529 isolated from an onager, 94-137 from a Thomson’s gazelle). The histologic lesions of encephalitis and antigen localization in this gazelle indicate prominent viral neurotropism, and lesions were very similar to those seen in EHV-1– and EHV-9–infected non-equid species. Histologic lesions caused by EHV-9 and zebra-borne EHV-1 are therefore indistinguishable.

APA, Harvard, Vancouver, ISO, and other styles

50

Suriapperuma, TNP, V. Koculen, C. Udagedara, and KP Weerasekara. "Eosinophilic meningoencephalitis due to neurotoxocariasis presenting as gross motor developmental regression in an infant: A Case Report." Sri Lankan Journal of Infectious Diseases 11, no. 2 (October 28, 2021): 107. http://dx.doi.org/10.4038/sljid.v11i2.8342.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!