Academic literature on the topic 'Eosinophilic meningoencephalitis'
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Journal articles on the topic "Eosinophilic meningoencephalitis"
Bansal, Sharad, Mukesh Gupta, Deepak Sharma, and Shweta Bansal. "A Rare Case of Ibuprofen-Induced Eosinophilic Meningitis in a 13-Year-Old Girl." Clinical Medicine Insights: Pediatrics 8 (January 2014): CMPed.S13829. http://dx.doi.org/10.4137/cmped.s13829.
Full textGraeff-Teixeira, Carlos, Ana Cristina Arámburu da Silva, and Kentaro Yoshimura. "Update on Eosinophilic Meningoencephalitis and Its Clinical Relevance." Clinical Microbiology Reviews 22, no. 2 (April 2009): 322–48. http://dx.doi.org/10.1128/cmr.00044-08.
Full textGoldman-Yassen, Adam E., Anna Derman, Rebecca Pellett Madan, and Alireza Radmanesh. "A Worm’s Tale or Why to Avoid the Raccoon Latrine: A Case of Baylisascaris procyonis Meningoencephalitis." Case Reports in Radiology 2022 (August 21, 2022): 1–6. http://dx.doi.org/10.1155/2022/5199863.
Full textDorta-Contreras, Alberto Juan, Piotr Lewczuc, Elena Noris-García, María Teresa Interián-Morales, María Esther Magraner Tarrau, Bárbara Padilla-Docal, and Xiomara Escobar-Pérez. "sICAM-1 in meningoencephalitis due to Angiostrongylus cantonensis." Arquivos de Neuro-Psiquiatria 64, no. 3a (September 2006): 589–91. http://dx.doi.org/10.1590/s0004-282x2006000400011.
Full textProciv, Paul, and James R. Tiernan. "Eosinophilic meningoencephalitis with permanent sequelae." Medical Journal of Australia 147, no. 6 (September 1987): 294–95. http://dx.doi.org/10.5694/j.1326-5377.1987.tb133461.x.
Full textMimoso, M. G., M. C. Pereira, M. H. Estevão, A. A. Barroso, and H. C. Mota. "Eosinophilic meningoencephalitis due toToxocara canis." European Journal of Pediatrics 152, no. 9 (September 1993): 783–84. http://dx.doi.org/10.1007/bf01954007.
Full textDu, Wen-Yuan, Jiunn-Wang Liao, Chia-Kwung Fan, and Kua-Eyre Su. "Combined Treatment with Interleukin-12 and Mebendazole Lessens the Severity of Experimental Eosinophilic Meningitis Caused by Angiostrongylus cantonensis in ICR Mice." Infection and Immunity 71, no. 7 (July 2003): 3947–53. http://dx.doi.org/10.1128/iai.71.7.3947-3953.2003.
Full textPhan, Hai Thanh, Kiem Hao Tran, and Huu Son Nguyen. "Eosinophilic Meningitis due to Angiostrongylus cantonensis in Children." Case Reports in Neurology 13, no. 1 (March 19, 2021): 184–89. http://dx.doi.org/10.1159/000512809.
Full textHong, David S., Marc Bernstein, Candice Smith, Hayley Gans, and Richard J. Shaw. "Eosinophilic Meningoencephalitis: Psychiatric Presentation and Treatment." International Journal of Psychiatry in Medicine 38, no. 3 (September 2008): 287–95. http://dx.doi.org/10.2190/pm.38.3.e.
Full textFerreira, Lucas Fernandes, Filipe Tupinamba Di Pace, and Guilherme Diogo Silva. "A Woman With Eosinophilic Brainstem Meningoencephalitis." JAMA Neurology 79, no. 2 (February 1, 2022): 198. http://dx.doi.org/10.1001/jamaneurol.2021.4861.
Full textDissertations / Theses on the topic "Eosinophilic meningoencephalitis"
Lunn, Julian Alexander. "Canine Neural Angiostrongyliasis." Thesis, The University of Sydney, 2006. http://hdl.handle.net/2123/2077.
Full textWEN-YUAN, DU, and 杜文圓. "Studies on the treatment of eosinophilic meningoencephalitis and the cerebral apoptosis in mice angiostrongylosis." Thesis, 2003. http://ndltd.ncl.edu.tw/handle/47123177434200889163.
Full text國立臺灣大學
微生物學研究所
91
Angiostrongylus cantonensis is the major cause of eosinophilic meningoencephalitis cases in Taiwan. Mice were orally infected with 35 infective larvae. One group of mice were given single dose mebendazole (20mg/kg) per os at various times and examined at 14 days postinfection (dpi) for worm recovery rate and pathological studies. A 94-97% reduction in worm recovery was observed if medication was given 4-5 dpi. Sections of the brains revealed that untreated infected mice developed typical severe eosinophilic meningoencephalitis. Meninges of these mice were thickened by massive infiltration of eosinophils, whereas only moderate pathological change was observed in the brains of mice that were treated with mebendazole at 4 dpi. Infected mice that received daily injections of 10 ng interleukin-12 (IL-12) for varying days only also exhibited moderate pathological change in the brains. Eosinophil infiltration in the brains of these mice were low and severe mechanical injuries in the parenchyma were observed. Treatment with mebendazole in combination with IL-12, however, resulted in low worm recovery and dramatic lessening of the eosinophilic meningitis. The reverse transcriptase-PCR assay on mRNA expression in the brain also revealed that the use of IL-12 had shifted the immune response of the mouse from Th2-type into Th1-type. In situ staining of the sections with TUNEL assay, anti-Akt or anti-caspase antibodies, revealed that infiltrated eosinophils in the meninges of infected but nontreated mice were resistant to apoptosis. Some of the infiltrated eosinophils in the meninges of various treatment groups, however, were undergoing programmed cell death.
Chen, In-Wen, and 陳映雯. "Effects of different treatments on acute eosinophilic meningoencephalitis caused by Angiostrongylus cantonensis in ICR mice." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/28431492989376304509.
Full textPei-Chun and 味佩君. "Matrix metalloproteinase-12 in eosinophilic meningitis or meningoencephalitis of BALB/c mice caused by Angiostrongylus cantonensis." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/26962458180633756893.
Full text中山醫學大學
醫學研究所
96
Angiostrongylus cantonensis, the rat lugworm, is the principal cause of eosinophilic meningitis or meningoencephalitis. In the previous study, matrix metalloproteinase-9 (MMP-9) has been proved involved in the pathogenesis of eosinophilic meningitis or meningoencephalitis. In this study, we used 5-week BALB/c male mice infected 50 A. cantonensis larvae. The results showed that MMP-12 is found in the brain tissues and cerebral spinal fruid (CSF) of infected mice and significantly increases from day 5 PI. Similarly, the expression of elastin, the substrate of MMP-12 is also increased. By immunohistochemistry, confirmed that MMP-12 distributed in leukocytes that infiltrated into subarachnoid space and mainly expressed in macrophages and eosinophils. The mice treated with albendazole (10mg/kg per day) alone, doxycycline (30mg/kg per day) alone, or a combination of albendazole (10mg/kg per day) and doxycycline (30mg/kg per day) for 7 consecutive days on day 5 and 15 post-inoculation (PI), respectively. The treatment of albendazole-doxycycline co-therapy on day 5 PI significantly reduced the infiltration of leukocytes and the expression of MMP-12 in infected mice. These results suggested that MMP-12 may contribute to the develop of eosinophilic meningitis or meningoencephalitis caused by A. cantonensis. This new approach may be beneficial to treat the parasitic meningitis or meningoencephalitis.
Lunn, Julian Alexander. "Canine Neural Angiostrongyliasis." 2007. http://hdl.handle.net/2123/2077.
Full textSummary Canine Neural Angiostrongyliasis (CNA) is caused by the obligatory neural migration of Angiostrongylus cantonensis larvae in dogs. Characteristically, cases are juvenile dogs with progressive CNS dysfunction characterised by hyperaesthesia and often associated with eosinophilic pleocytosis of the CSF. In Australia, most cases occur between March and June. The rat lungworm, A cantonensis was first described by Chen in 1935 in Canton, China. While initially called Pulmonema cantonensis the parasite was later reclassified as A cantonensis. A disease diagnosed as eosinophilic meningoencephalitis was first described in 1944 in Taiwan. The same disease was reported in 1948 in the East Caroline Islands but it was not until 1961 that A cantonensis was confirmed as the aetiological agent when a patient in a Hawaiian mental institution, who had died of eosinophilic meningoencephalitis, had A cantonensis larvae recovered from the brain and spinal cord. The first reports of animals infected with A cantonensis were made by Mason in 1976 when he described a syndrome occurring in puppies in the Brisbane area, characterised by urinary incontinence, hind limb paresis and hyperaesthesia, often associated with eosinophilic pleocytosis of the CSF. Reports of infection in other species followed including macropods, bats, horses, primates and birds. Twenty-two cases of suspected CNA were collected prospectively to compare with those previously described, including 37 cases published by Mason in 1983, and to examine the accuracy of an ELISA used to diagnose human neural angiostrongyliasis in Australia. Samples were collected from two control populations in an attempt to validate the ELISA results. In the prospective series of cases, there was a significantly older subpopulation of dogs in addition to “classical” young dogs, suggesting that this syndrome can occur at any age and should be considered a differential in any dog with progressive neurological disease. The mortality rate in the prospective group was lower than in the published group, which is a reflection of the severity of the disease in younger animals as is the case with human patients. Definitive diagnosis of neural angiostrongyliasis in human patients has been achieved by identifying A cantonensis larvae within the CSF or aqueous humour. In dogs, the only definitive way to diagnose CNA has been via necropsy. While many cases of CNA are characteristic and presumptive diagnosis can be made based on typical history, signalment, clinical signs, CSF analysis and response to glucocorticoids, there appear to be an increasing number of cases occurring in older dogs, that displaying focal, atypical clinical signs or that develop permanent sequelae. Serology has been a useful tool in diagnosing neural angiostrongyliasis in humans. In its current form the ELISA is not sensitive or specific enough to allow a definitive diagnosis of CNA to be made using serum but is useful when applied to CSF specimens. Further refinement of the antigen or using monoclonal rather than polyclonal antibodies may improve the accuracy of the serology. Alternatively, methods such as Western Blot, Immuno-PCR or dot-blot ELISA, which have been successfully used to diagnoses angiostrongyliasis in humans, may be worthy of investigation The major differential diagnosis for CNA is neosporosis. Other differential diagnoses include idiopathic eosinophilic meningoencephalitis, parasitic infections including Toxoplasma gondii, Taenia solium, Gnathostoma spinigerum, visceral larval migrans (Toxocara canis) and schistosomiasis, fungal, bacterial, viral and rickettsial infections as well as neoplasia, trauma, drug reactions and toxicities. Treatment of CNA has been limited to glucocorticoids, however there may be adjunct therapies including anthelmintices, cyclosporine, and matrix metalloproteinase inhibitors. In Mason’s series of cases the use of anthelmintics significantly worsened the clinical outcome for patients. It does not appear, however, that the use of these agents in species other than the dog exacerbates clinical signs. Acquired immunity is short lived in rats and mice, which would suggest the same is true in dogs. Routine heartworm and intestinal parasite prophylaxis appears to have no influence on the occurrence of CNA.
Book chapters on the topic "Eosinophilic meningoencephalitis"
Hsu, Hao-Ting, and Po-Yen Chen. "Case 10. A 1-Year-Old Boy with Fever, Progressive Consciousness Change, and Seizure: Eosinophilic Meningoencephalitis." In Paediatric Infectious Diseases, 49–52. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7276-8_10.
Full text"Eosinophilic Meningoencephalitis." In Encyclopedia of Parasitology, 955. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_1083.
Full textALICATA, J. E. "BIOLOGY AND DISTRIBUTION OF THE RAT LUNGWORM, ANGIOSTRONGYLUS CANTONENSIS, AND ITS RELATION TO EOSINOPHILIC MENINGOENCEPHALITIS AND OTHER NEUROLOGICAL DISORDERS OF MAN AND ANIMALS." In Proceedings of the First International Congress of Parasitology, 826–28. Elsevier, 1999. http://dx.doi.org/10.1016/b978-0-08-011427-9.50179-7.
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