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Books on the topic 'Enzyme interaction'

Author: Grafiati
Published: 10 September 2022
Last updated: 18 September 2022

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1

Nycander, Maria. Interaction of cystatins with cysteine proteinases. Uppsala: Sveriges Lantbruksuniversitet, 1998.

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2

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions. Vienna: Springer Vienna, 2015. http://dx.doi.org/10.1007/978-3-7091-1402-5.

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3

Pang, K. Sandy, A. David Rodrigues, and Raimund M. Peter, eds. Enzyme- and Transporter-Based Drug-Drug Interactions. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-0840-7.

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4

Cozza, Kelly L. Concise guide to the cytochrome P450 system: Drug interaction principles for medical practice. Washington, DC: American Psychiatric Pub., 2001.

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5

Pang, K. Sandy. Enzyme- and transporter-based drug-drug Interactions: Progress and future challenges. New York: Springer, 2010.

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6

Invertebrate-microbial interactions: Ingested fungal enzymes in arthropod biology. Ithaca: Comstock Pub. Associates, 1987.

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7

Randall, Richard Christopher. Protein and enzyme interactions in solution and at the silica-water interface. Salford: University of Salford, 1988.

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8

Mekras, Christos Ioannis. A study of the interactions of enzymes with polyions in aqueous solutions. Salford: University of Salford, 1985.

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9

Raaska, Laura. Cultivation and spawn production of the wood-decaying fungus, shiitake (Lentinula edodes): Optimization of spawn growth, production of degradative enzymes and interaction with wood inhabitants. Espoo: Technical Research Centre of Finland, 1993.

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10

Giralt, Ernest, Mark Peczuh, and Xavier Salvatella. Protein surface recognition: Approaches for drug discovery. Chichester, West Sussex: John Wiley & Sons, 2011.

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11

Busacca, Maurizio, and Roberto Paladini. Collaboration Age. Venice: Fondazione Università Ca’ Foscari, 2020. http://dx.doi.org/10.30687/978-88-6969-424-0.

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Recently, public policies of urban regeneration have intensified and multiplied. They are being promoted with the aim to start social and economic dynamics within the local context which is subject to intervention. From the empirical analysis, we realise that such activities are mainly implemented by three subjects or by mixed coalitions (public institutions, actors of the third sector and companies). Within them, each player is moved by a multiplicity of interests and goals that go beyond their own nature – public interest, market and mutualism – and tend to redefine themselves, thus becoming hybrid forms of production of value (social, economic, cultural). By studying a number Italian and Catalan cases, this essay deals with the theory that, under specific conditions and configurations, a collaborative direction – of organization, production and design – would give life to successful procedures, even without the identification of a one-best-way. The collaboration is not simply a choice of operation, but a real production method which mobilises social resources to create hybrid solutions – between state, market and society – to complex issues that could not be faced solely with the use of the rationale of action of one among the three actors. In this framework, the systems of relations and interactions between players and shared capital become an essential condition for the success of every initiative of urban redevelopment, or failure thereof. Such initiatives are brought to life by the strategic role of individuals who foster connections as well as the dissemination of non-redundant information between social networks, and collective and individual actors which would otherwise be separated and barely able to communicate and collaborate with each other. In addition to the functions carried out by knowledge brokers, that have been extensively described in organisational studies and economic sociology, the aforementioned figures act as real social enzymes, that is to say, they handle the available information and function as catalysts of social processes of production of knowledge. Moreover, they increase the reaction speed, working on mechanisms which control the spontaneity.
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12

Abdelnour-Esquivel, Ana. Interaction of cytokinins and phenethylamines in cell-free enzyme systems and plant callus cultures. 1990.

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13

United States. National Aeronautics and Space Administration., ed. Interaction of light and ethylene on stem gravitropism: Grant #: NAGW 3859, final report. [Washington, DC: National Aeronautics and Space Administration, 1996.

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14

Cozza, Kelly L., C. M. D. Armstrong Scott, and R. M. D. Oesterheld Jessica. Concise Guide to Drug Interaction Principles for Medical Practice: Cytochrome P450s, Ugts, P-Glycoproteins (Concise Guides). 2nd ed. American Psychiatric Publishing, Inc., 2003.

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15

MD, Kelly L. Cozza, and Scott C. Armstrong MD. Concise Guide to the Cytochrome P450 System: Drug Interaction Guidelines for Medical Practice. 4th ed. American Psychiatric Association, 2001.

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16

Markley, John L., Dexter B. Northrop, and Catherine A. Royer, eds. High Pressure Effects in Molecular Biophysics and Enzymology. Oxford University Press, 1996. http://dx.doi.org/10.1093/oso/9780195097221.001.0001.

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High Pressure Effects in Molecular Biophysics and Enzymology is designed to acquaint biochemists, biophysicists, and graduate students with advances in the application of high pressure in connection with spectroscopy as a research tool in the study of biomolecules. The 23 chapters written by leading authorities present an overview of current approaches to the use of high pressure in research on enzyme kinetics, protein folding and structure, lipid bilayer structure and organization, lipid-protein interaction, and DNA structure. This important, timely volume is the first devoted exclusively to high-pressure effects in biochemistry and will be the definitive reference in its subject for the next several years.
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17

Kim, Sung-Kun (Sean), Dong-Woo Lee, and Ki Duk Park, eds. Interactions Between Small Molecule Ligands and Target Enzymes. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-685-0.

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18

Selzer, Paul M., Gottfried Unden, Anja Schuffler, and Eckhard Thines. Host - Pathogen Interaction: Microbial Metabolism, Pathogenicity and Antiinfectives. Wiley & Sons, Limited, John, 2016.

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19

Rodrigues, A. David, K. Sandy Pang, and Raimund M. Peter. Enzyme- and Transporter-Based Drug-Drug Interactions: Progress and Future Challenges. Springer, 2014.

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20

Rodrigues, A. David, K. Sandy Pang, and Raimund M. Peter. Enzyme- and Transporter-Based Drug-Drug Interactions: Progress and Future Challenges. Springer, 2010.

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21

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Robert T. Sauer (Editor), eds. Protein-DNA Interactions, Volume 208: Volume 208: Protein - Dna Interactions (Methods in Enzymology). Academic Press, 1991.

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22

Sliz, Piotr. Structure, function and interactions of enzyme IIA from the phosphoenolpyruvate: Lactose phosphotransferase system. 2000.

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23

Martin, Michael M. Invertebrate-Microbial Interactions: Ingested Fungal Enzymes in Arthropod Biology (A Comstock Book). Comstock Publishing, 1988.

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24

Olkkola, Klaus T., Hugo E. M. Vereecke, and Martin Luginbühl. Drug interactions in anaesthetic practice. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0021.

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When two or more drugs are administered simultaneously, the pharmacological response may be greater or less than the sum of the effects of the individual drugs. One drug may antagonize or potentiate the effects of the other and there may be also qualitative differences in response. Although some drug interactions increase the toxicity or result in loss of therapeutic effect, others are beneficial. Indeed, modern anaesthetic techniques depend on beneficial drug interactions. A sound combination of drugs helps clinicians to increase both the efficacy and safety of drug treatment. Drugs may interact on a pharmaceutical, pharmacodynamic, or pharmacokinetic basis. Many pharmacodynamic interactions are predictable and can be avoided by the use of common sense. However, it is much more difficult to predict the likelihood of pharmacokinetic and pharmaceutical interactions despite good prior knowledge of pharmacokinetics and chemical properties of individual drugs. Pharmaceutical drug interactions usually occur before the drug is given to the patient and they are caused by chemical (such as acid–base, salt formation, oxidation–reduction, hydrolysis, or epimerization) or physical (such as adsorption/absorption or emulsion breaking) reactions. When drugs have a pharmacokinetic interaction, one drug alters the absorption, distribution, or the elimination of the other drug. Many pharmacokinetic drug interactions are due to inhibition or induction of cytochrome P450 enzymes. Pharmacodynamic drug interactions are caused by drugs having an effect on the same receptors or the same physiological system. This chapter gives anaesthetists an overview of clinically relevant perioperative drug interactions.
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25

Young, James Patrick. Enzyme associations in deoxyribonucleotide biosynthesis: Anti-idiotypic antibodies as probes for direct protein-protein interactions. 1992.

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26

Lambert, David G. Mechanisms and determinants of anaesthetic drug action. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0013.

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This chapter is broken into two main sections: a general description of the principles of ligand receptor interaction and a discussion of the main groups of ‘targets’; and explanation of some common pharmacological interactions in anaesthesia, critical care, and pain management. Agonists bind to and activate receptors while antagonists bind to receptors and block the effects of agonists. Antagonists can be competitive (most common) or non-competitive/irreversible. The main classes of drug target are enzymes, carriers, ion channels, and receptors with examples of anaesthetic relevance interacting with all classes. There are many examples in anaesthesia where multiple interacting drugs are co-administered—polypharmacology. To give an example: neuromuscular blockade. Rocuronium is a non-depolarizing neuromuscular blocker acting as a competitive antagonist at the nicotinic acetylcholine receptor. Rocuronium competes with endogenous acetylcholine to shift the concentration–response curve for contraction to the right. The degree of contractility is less for a given concentration of acetylcholine (agonist) in the presence of rocuronium. Using the same principle, the rightward shift can be compensated by increasing the amount of acetylcholine (as long as the amount of rocuronium presented to the receptor as an antagonist remains unchanged, its action can be overcome by increased agonist). Acetylcholine at the effect site is increased by acetylcholinesterase inhibition with neostigmine. One of the side-effects of neostigmine is that it acts as an indirect parasympathomimetic. In the cardiovascular system this would lead to muscarinic receptor-mediated bradycardia; these effects are routinely reversed by the competitive muscarinic antagonist glycopyrrolate.
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27

Invertebrate-Microbial Interactions: Ingested Fungal Enzymes in Arthropod Biology (Explorations in Chemical Ecology). Cornell University Press, 1988.

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28

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions: Selected Topics in the Theory and Diagnosis of Inhibition and Activation Mechanisms. Springer, 2016.

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29

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions: Selected Topics in the Theory and Diagnosis of Inhibition and Activation Mechanisms. Baici Antonio, 2015.

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30

Lexi-Comp's drug interactions handbook: The new standard for drug and herbal interactions featuring a complete guide to cytochrome P450 enzyme substrates, inducers, and inhibitors. Hudson, OH: Lexi-Comp, 2004.

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31

A, Bachmann Kenneth, ed. Lexi-Comp's drug interactions handbook: The new standard for drug and herbal interactions featuring a complete guide to cytochrome P450 enzyme substrates, inducers, and inhibitors. 2nd ed. Hudson, Ohio: Lexi-Comp, 2004.

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32

A, Bachmann Kenneth, ed. Lexi-Comp's drug interactions handbook: The new standard for drug and herbal interactions : featuring a complete guide to cytochrome P450 enzyme substrates, inducers, and inhibitors. Hudson, Ohio: Lexi-Comp, 2003.

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33

Davis, Ralph Eugene. Vaccinia virus ribonucleotide reductase: Gene sequencing, intracellular localization, and interaction with a DNA-binding protein. 1992.

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34

Neil B., M.d. Sandson. Drug Interactions Casebook: The Cytochrome P450 System and Beyond. American Psychiatric Publishing, 2003.

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35

SmallMolecule Inhibitors of ProteinProtein Interactions Current Topics in Microbiology and Immmunology. Springer, 2011.

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36

Zuppa, Athena. Pediatric Critical Care Pharmacology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199918027.003.0018.

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This chapter includes essential information about the basic principles of pharmacology and relates them to unique characteristics of critically ill children. The author provides a succinct summary of fundamentals of pharmacokinetics and pharmacodynamics, including absorption, distribution, metabolism, and elimination. First- and zero-order kinetics are reviewed, along with examples of drugs commonly used in the intensive care unit that follow those patterns of metabolism. The chapter also includes crucial information about how development from birth affects the various aspects of pharmacology. The effects on drug metabolism of shock, and renal and hepatic dysfunction are provided, along with drug–drug interactions, including commonly used drugs in critical care that induce or inhibit enzyme activity.
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37

J, Mulder Gerard, ed. Conjugation reactions in drug metabolism: An integrated approach : substrates, co-substrates, enzymes and their interactions in vivo and in vitro. London: Taylor & Francis, 1990.

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38

Iversen, Les. 2. How drugs work. Oxford University Press, 2016. http://dx.doi.org/10.1093/actrade/9780198745792.003.0002.

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‘How drugs work’ outlines the basic mechanisms of pharmacology. Drugs are chemicals that can be naturally occurring or man-made, and which can be administered in a variety of ways. They can act on receptors—often highly specific proteins in cells which can up-regulate or down-regulate processes—or on other targets, such as DNA or enzymes. The molecular action of drugs can be investigated in a lab, but the effects on the whole organism are more important. Effective doses need to be determined, taking into account metabolic rates, drug interactions, and side effects. Prolonged drug use can cause tolerance and substance addiction.
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39

C, Lee Y., and Lee Reiko T, eds. Recognition of carbohydrates in biological systems: Edited by Yuan C. Lee, Reiko T. Lee. San Diego, Calif: Academic Press, 2003.

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40

Lee, Y. C., and Reiko T. Lee. Recognition of Carbohydrates in Biological Systems, Part A : General Procedures, Volume 362 (Methods in Enzymology). Academic Press, 2003.

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41

Lee, Y. C., and Reiko T. Lee. Recognition of Carbohydrates in Biological Systems, Part A : General Procedures, Volume 362 (Methods in Enzymology). Academic Press, 2003.

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42

(Editor), Jonathan B. Chaires, and Michael J. Waring (Editor), eds. Drug-Nucleic Acid Interactions (Methods in Enzymology, Volume 340) (Methods in Enzymology). Academic Press, 2001.

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43

Ahsan, Husain, Graham Robert M, and Victor Chang Cardiac Research Institute., eds. Drugs, enzymes, and receptors of the renin-angiotensin system: Celebrating a century of discovery. Amsterdam: Harwood Academic Publishers, 2000.

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44

A, Burleigh Barbara, and Soldati Dominique, eds. Molecular mechanisms of parasite invasion. Austin, Tex: Landes Bioscience, 2008.

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45

Graham, Robert M., and Ahsan Husain. Drugs, Enzymes and Receptors of the Renin-Angiotensin System: Celebrating a Century of Discovery (The Victor Chang Molecular Cardiology Series, V. 1). CRC, 2000.

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46

Giralt, Ernest, Mark Peczuh, and Xavier Salvatella. Protein Surface Recognition: Approaches for Drug Discovery. Wiley & Sons, Incorporated, John, 2011.

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47

Giralt, Ernest, Mark Peczuh, and Xavier Salvatella. Protein Surface Recognition: Approaches for Drug Discovery. Wiley & Sons, Incorporated, John, 2010.

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48

Giralt, Ernest, Mark Peczuh, and Xavier Salvatella. Protein Surface Recognition: Approaches for Drug Discovery. Wiley & Sons, Incorporated, John, 2011.

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49

Protein Surface Recognition: Approaches for Drug Discovery. Wiley, 2010.

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50

Chinoy, Hector, and Robert G. Cooper. Polymyositis and dermatomyositis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0124.

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Polymyositis (PM), dermatomyositis (DM), and inclusion body myositis (IBM) form part of the idiopathic inflammatory myopathies (IIM), a heterogeneous group of rare autoimmune diseases characterized by an acquired proximal muscle weakness, raised muscle enzymes (including creatine kinase), inflammatory cell infiltrates in muscle biopsy tissue, electrophysiological abnormalities, and presence of circulating myositis-specific/myositis-associated autoantibodies. The underlying aetiology of IIM is poorly understood, but likely involves interactions between environmental and genetic risk factors. Myositis may also manifest in association with other connective tissue disorders. The predominant clinical presentation of IIM is skeletal muscle weakness, but many extramuscular features can also occur. Access to good neuropathological support is essential in securing an accurate IIM diagnosis and excluding non-inflammatory myopathies, although IBM is often difficult to distinguish from PM. Antibody testing can help define IIM clinical subtypes, including cancer-associated myositis, predict prognosis, and help in optimizing treatment decisions. MRI can be invaluable for differentiating disease activity from damage, and detecting treatment-induced interval changes. Therapeutic effectiveness of new and existing treatments (where the evidence base remains poor) depends on making a prompt diagnosis and initiating early and appropriately aggressive treatment to prevent establishment of muscle damage. This chapter attempts to summarize the salient features of IIM and update the reader about currently used diagnostics and treatment paradigms in this rare and understudied disease.
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