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Journal articles on the topic 'ENO'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Aziza, Noor Laili, Noorkomala Sari, and Sofiya Irsalina. "Aktivitas Antagonistik Cendawan Endofit Asal Bunga Bawang Dayak (Eleutherine bulbosa (Mill.) Urb.) terhadap Fusarium sp. yang Menginfeksi Tanaman Cabai." Jurnal Fitopatologi Indonesia 17, no. 5 (January 21, 2022): 210–15. http://dx.doi.org/10.14692/jfi.17.5.210-215.

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Penyakit layu yang disebabkan oleh Fusarium spp. merupakan penyakit penting dalam budi daya cabai. Salah satu cara potensial untuk mengendalikan penyakit tanaman ialah menggunakan agens hayati seperti cendawan endofit dari tanaman obat. Bawang dayak termasuk tanaman obat yang bersifat antibakteri, anticendawan, antiinflamasi, dan antioksidan. Penelitian bertujuan menentukan aktivitas antagonistik cendawan endofit yang berasal dari bunga bawang dayak terhadap cendawan patogen Fusarium spp. Sebanyak tujuh belas isolat cendawan endofit berhasil diperoleh dari bunga bawang dayak, yaitu isolat EnA, EnB, EnC, EnD, EnE, EnF, EnG, EnH, EnI, EnJ, EnK, EnL, EnM, EnN, EnO, EnP, dan EnQ. Lima dari tujuh belas isolat, yaitu EnA, EnF, EnI, EnJ, dan EnK digunakan untuk uji antagonisme terhadap Fusarium spp. dengan metode dual kultur. Penghambatan pertumbuhan koloni Fusarium spp. yang disebabkan oleh isolat EnA, EnF, EnI, EnJ, dan EnK berturut-turut sebesar 67.6%, 53.15%, 77.25%, 70,42% dan 67.1%.

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2

Fjordholm, Ulrik S., Siddhartha Mishra, and Eitan Tadmor. "ENO Reconstruction and ENO Interpolation Are Stable." Foundations of Computational Mathematics 13, no. 2 (March 28, 2012): 139–59. http://dx.doi.org/10.1007/s10208-012-9117-9.

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3

Li, Jiancheng, Xiaobin Fu, and Jie Fu. "Exhaled Nitric Oxide Is Useful in Symptomatic Radioactive Pneumonia: A Retrospective Study." Mediators of Inflammation 2017 (2017): 1–6. http://dx.doi.org/10.1155/2017/5840813.

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The aim was to defect the exhaled nitric oxide (eNO) prediction value of symptomatic radioactive pneumonia (SRP). 64 cases of lung cancer or esophagus cancer, who had the primary radiotherapy (intensity-modulated radiation therapy), were included from 2015 June to 2016 January. During the following, the patients were divided: the symptomatic radiation pneumonia group (SRP, with the CTCAE v4.0 score > 2) and the asymptomatic radiation pneumonia group (ASRP, with CTCAE v4.0 score ≤ 1). All the patients were measured eNO before and at the end of thoracic radiotherapy and gain the posttherapy eNO value and the eNO ratio (posttherapy eNO value/pretherapy eNO value), then the predictive values of eNO toward SRP were measured using the receiver-operating characteristic (ROC). 17 cases were included in the SRP group and the other 47 were included in the ASRP group. The posttherapy eNO was 29.35 (19~60) bbp versus 20.646 (11~37) (P<0.001), and the ratio was 1.669 (0.61~3.5) versus 0.920 (0.35~1.5) (P<0.01) (symptomatic versus asymptomatic). ROC showed that the cutoff value of SRP was 19.5 bbp (posttherapy eNO, area under concentration-time curve (AUC) = 0.879) and 1.305 (eNO ratio, AUC = 0.774), which meant that posttherapy eNO and eNO ratio were useful in finding SRP.

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4

Barker, M., C. Lex, A. Möller, Angela Zacharasiewicz, and F. Horak Jr. "Exhaliertes Stickstoffmonoxid (eNO)." Monatsschrift Kinderheilkunde 155, no. 6 (June 2007): 560–62. http://dx.doi.org/10.1007/s00112-007-1538-1.

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5

Zhao, Chuanliang, Mali Qin, Ling Jin, Ju Lai, Yang Wang, Shuangxi Liu, and Shaoqing Yu. "Significance of Exhaled Nitric Oxide and Carbon Monoxide in Auxiliary Diagnosis and Evaluation of Allergic Rhinitis." Mediators of Inflammation 2022 (November 23, 2022): 1–10. http://dx.doi.org/10.1155/2022/2083057.

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Objective. The concentration of exhaled NO and CO is considered as a candidate marker of respiratory inflammatory disease. This report discusses the exhaled NO and CO in the auxiliary diagnosis and evaluation of allergic rhinitis (AR). Methods. 60 AR patients from October 2017 to March 2019, compared with 30 healthy controls. The severity of AR disease was distinguished by symptom score. Both groups were tested for exhaled nitric oxide through the nose or mouth and exhale carbon monoxide through the mouth. AR patients received glucocorticoid nasal spray for 1 month and were tested again for nNO, eNO, eCO, and symptom score. Results. Before treatment, all the nNO, eNO, and eCO of the AR group were higher than the control group. There were differences in the severe and moderate subgroup: severe > moderate > mild . eCO was not significantly different between the mild and control groups. The nNO, eNO, and eCO levels were positively correlated with symptom score. After treatment, nNO decreased significantly in the three subgroups; eNO and eCO in the severe AR group decreased significantly. Drawing the ROC curve, the area under curve (AUC) of nNO is 0.978. The AUC of eNO and eCO was 0.786 and 0.577, respectively. Conclusion. The nNO, eNO, and eCO in the AR group are higher than healthy people, which positively correlated with the severity of AR symptoms. The detection of nNO, eNO, and eCO can monitor the changes of AR. The detection of nNO level as an indicator of AR auxiliary diagnosis has high accuracy.

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6

Lassmann‐Klee, Paul G., Lauri Lehtimäki, Tuula Lindholm, Leo Pekka Malmberg, Anssi R. A. Sovijärvi, and Päivi Liisa Piirilä. "Converting F ENO by different flows to standard flow F ENO." Clinical Physiology and Functional Imaging 39, no. 5 (June 3, 2019): 315–21. http://dx.doi.org/10.1111/cpf.12574.

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7

Serna, Susana, and Antonio Marquina. "Power ENO methods: a fifth-order accurate Weighted Power ENO method." Journal of Computational Physics 194, no. 2 (March 2004): 632–58. http://dx.doi.org/10.1016/j.jcp.2003.09.017.

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8

Shu, Chi-Wang. "Numerical experiments on the accuracy of ENO and modified ENO schemes." Journal of Scientific Computing 5, no. 2 (June 1990): 127–49. http://dx.doi.org/10.1007/bf01065581.

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9

Steele, Sean. "Brian Eno: Oblique Music." Popular Music and Society 44, no. 3 (February 24, 2021): 344–46. http://dx.doi.org/10.1080/03007766.2021.1891657.

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10

Wygrecka, Malgorzata, Leigh M. Marsh, Rory E. Morty, Ingrid Henneke, Andreas Guenther, Juergen Lohmeyer, Philipp Markart, and Klaus T. Preissner. "Enolase-1 promotes plasminogen-mediated recruitment of monocytes to the acutely inflamed lung." Blood 113, no. 22 (May 28, 2009): 5588–98. http://dx.doi.org/10.1182/blood-2008-08-170837.

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Abstract Cell surface–associated proteolysis plays a crucial role in the migration of mononuclear phagocytes to sites of inflammation. The glycolytic enzyme enolase-1 (ENO-1) binds plasminogen at the cell surface, enhancing local plasmin production. This study addressed the role played by ENO-1 in lipopolysaccharide (LPS)–driven chemokine-directed monocyte migration and matrix invasion in vitro, as well as recruitment of monocytes to the alveolar compartment in vivo. LPS rapidly up-regulated ENO-1 cell-surface expression on human blood monocytes and U937 cells due to protein translocation from cytosolic pools, which increased plasmin generation, enhanced monocyte migration through epithelial monolayers, and promoted matrix degradation. These effects were abrogated by antibodies directed against the plasminogen binding site of ENO-1. Overexpression of ENO-1 in U937 cells increased their migratory and matrix-penetrating capacity, which was suppressed by overexpression of a truncated ENO-1 variant lacking the plasminogen binding site (ENO-1ΔPLG). In vivo, intratracheal LPS application in mice promoted alveolar recruitment of monocytic cells that overexpressed ENO-1, but not of cells overexpressing ENO-1ΔPLG. Consistent with these data, pneumonia-patients exhibited increased ENO-1 cell-surface expression on blood monocytes and intense ENO-1 staining of mononuclear cells in the alveolar space. These data suggest an important mechanism of inflammatory cell invasion mediated by increased cell-surface expression of ENO-1.

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11

Meneses, J. "PRESENTACIÓN XV ENO –VI CANCOA." Optica Pura y Aplicada 51, no. 3 (October 5, 2018): i—ii. http://dx.doi.org/10.7149/opa.51.3.i.

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12

Eno, Rick. "A Conversation with Rick Eno." Industrial Biotechnology 13, no. 6 (December 2017): 282–84. http://dx.doi.org/10.1089/ind.2017.29109.ren.

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13

Chirico, Miriam. "The Plot by Will Eno." Theatre Journal 72, no. 3 (2020): 366–67. http://dx.doi.org/10.1353/tj.2020.0075.

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14

Aràndiga, Francesc, and Ana Marı́a Belda. "Weighted ENO interpolation and applications." Communications in Nonlinear Science and Numerical Simulation 9, no. 2 (April 2004): 187–95. http://dx.doi.org/10.1016/s1007-5704(03)00107-2.

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15

NELSON, ROXANNE. "eNO Analysis Aids Asthma Diagnosis." Pediatric News 41, no. 6 (June 2007): 1–5. http://dx.doi.org/10.1016/s0031-398x(07)70348-x.

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16

Highfield, Roger. "One minute interview: Brian Eno." New Scientist 203, no. 2716 (July 2009): 25. http://dx.doi.org/10.1016/s0262-4079(09)61832-7.

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17

Harten, Ami. "ENO schemes with subcell resolution." Journal of Computational Physics 83, no. 1 (July 1989): 148–84. http://dx.doi.org/10.1016/0021-9991(89)90226-x.

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18

Wilcoxson, Mark, and Vasilios Manousiouthakis. "On an Implicit ENO Scheme." Journal of Computational Physics 115, no. 2 (December 1994): 376–89. http://dx.doi.org/10.1006/jcph.1994.1203.

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19

Bauer, Robert Bruce. "A Hybrid Adaptive ENO Scheme." Journal of Computational Physics 136, no. 1 (September 1997): 180–96. http://dx.doi.org/10.1006/jcph.1997.5766.

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20

Zakrzewicz, Dariusz, Miroslava Didiasova, Anna Zakrzewicz, Andreas C. Hocke, Florian Uhle, Philipp Markart, Klaus T. Preissner, and Malgorzata Wygrecka. "The interaction of enolase-1 with caveolae-associated proteins regulates its subcellular localization." Biochemical Journal 460, no. 2 (May 13, 2014): 295–307. http://dx.doi.org/10.1042/bj20130945.

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We found that ENO-1 localizes to caveolae and interacts with caveolin-1 and annexin 2. The association of ENO-1 with caveolar proteins and localization within caveolae are required for ENO-1 cell surface localization and ENO-1-dependent cell migration and invasion.

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21

Yan, Yujian, Yuyi Ou, Boyi Yang, Yanyan Jia, Lianpeng Sun, and Hui Lu. "Insight into the Removal of Enoxacin in an Anaerobic Sulfur-Mediated Wastewater Treatment System: Performance, Kinetics and Mechanisms." Water 14, no. 18 (September 16, 2022): 2896. http://dx.doi.org/10.3390/w14182896.

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The removal of enoxacin (ENO), a broad-spectrum fluoroquinolone antibiotic, was firstly examined in a sulfate-reducing up-flow sludge bed (SRUSB) bioreactor over a long-term operation (366 days). Over 94% of the ENO was removed in the SRUSB bioreactor via adsorption and biodegradation at different initial ENO concentrations (i.e., 25–1000 μg/L). Based on the results of the batch tests, the sulfate-reducing sludge exhibited a high ENO adsorption capacity within a kd of 22.7–28.9 L/g-SS. The adsorption of ENO by the sulfate-reducing sludge was a spontaneous (ΔG° < 0 KJ/mol) and exothermic (ΔH° < 0 KJ/mol) process including physisorption and chemisorption (absolute value of ΔH° = 51.882 KJ/mol). Moreover, ENO was effectively biodegraded by the sulfate-reducing sludge within specific rates of 2.5–161.3 μg/g-SS/d. The ENO biodegradation process in the sulfate-reducing sludge system was most accurately described by the first-order kinetic model. Collectively, our findings provide insight into the applicability of a sulfate-reducing sludge system for ENO-contaminated wastewater treatment.

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22

Santiago, Krystal R., Alfonso M. Duran, Carlos A. Casiano, Frankis G. Almaguel, and Bhaskar Das. "Abstract C063: Enolase-1 as a candidate theranostics target for neuroendocrine prostate cancer." Cancer Epidemiology, Biomarkers & Prevention 32, no. 1_Supplement (January 1, 2023): C063. http://dx.doi.org/10.1158/1538-7755.disp22-c063.

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Abstract Prostate cancer (PCa) is the second most common cancer in American men, with men of African ancestry (AA) having twice the mortality of men of European ancestry (AA). Although taxane-based chemotherapy is the last line of defense in men with advanced PCa, it ultimately fails due to chemoresistance. The protein-specific membrane antigen (PSMA) has been an effective target for the imaging and therapy of advanced PCa. Specifically, PSMA radioligand therapy (PSMA-RLT) is a theranostics (therapy + diagnostics) option for men with advanced PCa. However, about 30% of men with advanced PCa have limited response to PSMA-RLT due to the presence of neuroendocrine-like PCa (NEPC), which lacks PSMA expression. A promising alternative to PSMA targeting is the glycolytic enzyme enolase (ENO), which localizes to the cell surface in advanced tumors. There are three enolases with similar functions, with ENO-1 and ENO-2 implicated in PCa. Previously, we showed that AA and EA men with PCa show differential serum antibody reactivity to ENO. While circulating serum antibodies from EA-PCa patients recognized ENO in both docetaxel (DTX)-sensitive and -resistant PCa cell lines, sera from AA-PCa men only recognized ENO in DTX-sensitive cells, with loss of immunoreactivity in the resistant cells. Here, we demonstrate, using immunoblotting and specific monoclonal antibodies, that chemosensitive NEPC cells express both ENO-1 and ENO-2; however, DTX-resistant cells only express ENO-1 with a clear loss of ENO-2. This pattern is identical to that produced by the AA-PCa serum antibodies, suggesting the possibility that AA-PCa patients may generate a preferential antibody response to ENO-2, with EA-PCa patients preferentially targeting ENO-1. This differential immunoreactivity may reflect differences between AA and EA PCa patients in tumor immunobiology, consistent with emerging literature. The loss of ENO-2 in chemoresistant PCa cells also generates a metabolic vulnerability due to the loss of ENO redundancy. We hypothesize that ENO-1 is expressed on the surface of NEPC cell lines and can be targeted with small molecule inhibitors (SMIs) that could be used as potential theranostics agents. Current studies are identifying ENO-1 surface expression on NEPC cell lines using monoclonal antibodies as well as AA and EA patient antibodies, via immunofluorescence microscopy, membrane fractionation analysis, and flow cytometry. We also initiated the testing of small molecule inhibitors (SMIs) targeting ENO-1 in chemoresistant NEPC cells. Our long-term goal is to identify an alternative treatment for patients with NEPC by establishing ENO-1 as a novel theranostics target. This could benefit AA-PCa patients, which may not mount a strong antitumor ENO-1 immune response. Citation Format: Krystal R. Santiago, Alfonso M. Duran, Carlos A. Casiano, Frankis G. Almaguel, Bhaskar Das. Enolase-1 as a candidate theranostics target for neuroendocrine prostate cancer [abstract]. In: Proceedings of the 15th AACR Conference on the Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; 2022 Sep 16-19; Philadelphia, PA. Philadelphia (PA): AACR; Cancer Epidemiol Biomarkers Prev 2022;31(1 Suppl):Abstract nr C063.

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23

Kang, Min-goo, and Seung-woo Park. "A Finite Volume Model Using ENO Scheme for 2D Unsteady flows." Journal of Korea Water Resources Association 36, no. 1 (February 1, 2003): 1–11. http://dx.doi.org/10.3741/jkwra.2003.36.1.001.

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24

Ahmad, Tanveer, Ulaganathan Mabalirajan, Duraisamy Arul Joseph, Lokesh Makhija, Vijay Pal Singh, Balaram Ghosh, and Anurag Agrawal. "Exhaled nitric oxide estimation by a simple and efficient noninvasive technique and its utility as a marker of airway inflammation in mice." Journal of Applied Physiology 107, no. 1 (July 2009): 295–301. http://dx.doi.org/10.1152/japplphysiol.00235.2009.

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Allergic airway inflammation (AI) is commonly associated with enhanced exhaled nitric oxide (ENO) in both humans and mice. Since mouse models are being used to understand various mechanisms of asthma, a noninvasive, simple, and reproducible method to determine ENO in mice is required for serial nonterminal assessment that can be used independent of environmental situations in which the ambient air contains substantial amounts of NO as a contaminant. The aim of this study was to noninvasively measure ENO in individual mice and to test its utility as a marker of AI in different models of allergic AI. We modified the existing ENO measuring methods by incorporating flushing and washout steps that allowed simple but reliable measurements under highly variable ambient NO conditions (1–100 ppb). This method was used to serially follow ENO in acute and chronic models of allergic AI in mice. ENO was reproducibly measured by this modified method and was positively correlated to AI in both acute and chronic models of asthma but was not independently related to airway remodeling. Resolution of AI and other related parameters in dexamethasone-treated mice resulted in reduction of ENO, further confirming this association. Restriction of allergen challenge to pulmonary but not nasal airways was associated with a smaller increase in ENO compared with allergen challenge to both. Hence, ENO can now be reliably measured in mice independent of ambient NO levels and is a valid biomarker for AI. However, nasal and pulmonary airways are likely to be independent sources of ENO, and any results must be interpreted as such.

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25

Montecinos, Gino I., and Eleuterio F. Toro. "ENO-ET: a reconstruction scheme based on extended ENO stencil and truncated highest-order term." Applied Mathematics and Computation 442 (April 2023): 127742. http://dx.doi.org/10.1016/j.amc.2022.127742.

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26

Shu, Chi-Wang. "Essentially non-oscillatory and weighted essentially non-oscillatory schemes." Acta Numerica 29 (May 2020): 701–62. http://dx.doi.org/10.1017/s0962492920000057.

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Essentially non-oscillatory (ENO) and weighted ENO (WENO) schemes were designed for solving hyperbolic and convection–diffusion equations with possibly discontinuous solutions or solutions with sharp gradient regions. The main idea of ENO and WENO schemes is actually an approximation procedure, aimed at achieving arbitrarily high-order accuracy in smooth regions and resolving shocks or other discontinuities sharply and in an essentially non-oscillatory fashion. Both finite volume and finite difference schemes have been designed using the ENO or WENO procedure, and these schemes are very popular in applications, most noticeably in computational fluid dynamics but also in other areas of computational physics and engineering. Since the main idea of the ENO and WENO schemes is an approximation procedure not directly related to partial differential equations (PDEs), ENO and WENO schemes also have non-PDE applications. In this paper we will survey the basic ideas behind ENO and WENO schemes, discuss their properties, and present examples of their applications to different types of PDEs as well as to non-PDE problems.

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27

Huang, Xiaoquan, Souksavanh Thansamay, Kaiqi Yang, Tiancheng Luo, and Shiyao Chen. "Measurement of Exhaled Nitric Oxide in Cirrhotic Patients with Esophageal and Gastric Varices." BioMed Research International 2019 (November 3, 2019): 1–6. http://dx.doi.org/10.1155/2019/9673162.

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Background and aims. This study aimed to detect exhaled nitric oxide (eNO) level in cirrhotic patients and explore the correlation between eNO levels and the severity of cirrhosis. Methods. Patients were enrolled to analyze the relationship of eNO with noncirrhosis, cirrhosis, and complications of decompensated cirrhosis. We explored the potential predictive values of eNO in different states of cirrhosis. Results. The eNO levels were significantly increased in cirrhotic patients compared with noncirrhotic patients (14 (10–18) vs 8 (6–13) ppb, P<0.001). The eNO level was increased in those with ascites (15 (14–22) vs 13 (10–18) ppb, P=0.026), with portal vein thrombosis (19.5 (11.75–22) vs 13.5 (10–17) ppb, P=0.032), or with the mucosal red-color sign of esophageal and gastric varices (EGV) (16.5 (10–21.75) vs 13 (10–14.75) ppb, P=0.041). Among cirrhotic patients undergoing hepatic venous pressure gradient (HVPG) measurement, the eNO level was significantly increased in the high-HVPG group (HVPG >12 mm Hg) compared with the low-HVPG group (6 mm Hg ≤ HVPG ≤ 12 mm Hg) (15 (11.75–19.25) vs 10 (8–14) ppb, P=0.011). Conclusions. The eNO level was increased in cirrhotic patients, especially in those complicated with ascites, portal vein thrombosis, mucosal red-color sign of varices, and high HVPG.

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28

Chang, Daniel, Weiguo Yao, Christina J. Tiller, Jeffrey Kisling, James E. Slaven, Zhangsheng Yu, Mark H. Kaplan, and Robert S. Tepper. "Exhaled nitric oxide during infancy as a risk factor for asthma and airway hyperreactivity." European Respiratory Journal 45, no. 1 (September 26, 2014): 98–106. http://dx.doi.org/10.1183/09031936.00034614.

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Childhood asthma is often characterised by elevated exhaled nitric oxide (eNO), decreased lung function, increased airway reactivity and atopy; however, our understanding of when these phenotypic airway characteristics develop remains unclear. This study evaluated whether eNO, lung function, airway reactivity and immune characteristics during infancy are risk factors of asthma at age 5 years.Infants with eczema, enrolled prior to wheezy illness (n=116), had eNO, spirometry, airway reactivity and allergen sensitisation assessed at entry to the study and repeated at age 5 years (n=90).Increasing eNO at entry was associated with an increased risk of asthma (p=0.037) and increasing airway reactivity (p=0.015) at age 5 years. Children with asthma at 5 years of age had a greater increase in eNO between infancy and age 5 years compared with those without asthma (p=0.002). Egg sensitisation at entry was also associated with an increased risk of asthma (p=0.020), increasing eNO (p = 0.002) and lower forced expiratory flows (p=0.029) as a 5 year-old.Our findings suggest that, among infants at high risk for developing asthma, eNO early in life may provide important insights into the subsequent risk of asthma and its airway characteristics.

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29

Hesthaven, Jan S., and Fabian Mönkeberg. "Hybrid high-resolution RBF-ENO method." Journal of Computational Physics: X 12 (September 2021): 100089. http://dx.doi.org/10.1016/j.jcpx.2021.100089.

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30

Getreuer, Pascal, and François G. Meyer. "ENO Multiresolution Schemes with General Discretizations." SIAM Journal on Numerical Analysis 46, no. 6 (January 2008): 2953–77. http://dx.doi.org/10.1137/060663763.

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31

Jiang, Guang-Shan, and Chi-Wang Shu. "Efficient Implementation of Weighted ENO Schemes." Journal of Computational Physics 126, no. 1 (June 1996): 202–28. http://dx.doi.org/10.1006/jcph.1996.0130.

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32

Abgrall, R., S. Lantéri, and T. Sonar. "ENO Approximations for Compressible Fluid Dynamics." ZAMM 79, no. 1 (January 1999): 3–28. http://dx.doi.org/10.1002/(sici)1521-4001(199901)79:1<3::aid-zamm3>3.0.co;2-1.

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33

Choi, Hyun-Ho, and Jung-Ryun Lee. "Energy-Neutral Operation Based on Simultaneous Wireless Information and Power Transfer for Wireless Powered Sensor Networks." Energies 12, no. 20 (October 10, 2019): 3823. http://dx.doi.org/10.3390/en12203823.

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For energy-neutral operation (ENO) of wireless sensor networks (WSNs), we apply a wireless powered communication network (WPCN) to a WSN with a hierarchical structure. In this hierarchical wireless powered sensor network (WPSN), sensor nodes with high harvesting energies and good link budgets have energy remaining after sending their data to the cluster head (CH), whereas the CH suffers from energy scarcity. Thus, we apply the simultaneous wireless information and power transfer (SWIPT) technique to the considered WPSN so that the sensor nodes can transfer their remaining energy to the CH while transmitting data in a cooperative manner. To maximize the achievable rate of sensing data while guaranteeing ENO, we propose a novel ENO framework, which provides a frame structure for SWIPT operation, rate improvement subject to ENO, SWIPT ratio optimization, as well as clustering and CH selection algorithm. The results of extensive simulations demonstrate that the proposed ENO based on SWIPT significantly improves the achievable rate and reduces the energy dissipated in the network while guaranteeing ENO, in comparison with the conventional schemes without SWIPT.

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34

Lai, Chun Sing, Mengxuan Yan, Xuecong Li, Loi Lei Lai, and Yang Xu. "Coordinated Operation of Electricity and Natural Gas Networks with Consideration of Congestion and Demand Response." Applied Sciences 11, no. 11 (May 28, 2021): 4987. http://dx.doi.org/10.3390/app11114987.

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This work presents a new coordinated operation (CO) framework for electricity and natural gas networks, considering network congestions and demand response. Credit rank (CR) indicator of coupling units is introduced, and gas consumption constraints information of natural gas fired units (NGFUs) is given. Natural gas network operator (GNO) will deliver this information to an electricity network operator (ENO). A major advantage of this operation framework is that no frequent information interaction between GNO and ENO is needed. The entire framework contains two participants and three optimization problems, namely, GNO optimization sub-problem-A, GNO optimization sub-problem-B, and ENO optimization sub-problem. Decision sequence changed from traditional ENO-GNO-ENO to GNO-ENO-GNO in this novel framework. Second-order cone (SOC) relaxation is applied to ENO optimization sub-problem. The original problem is reformulated as a mixed-integer second-order cone programming (MISOCP) problem. For GNO optimization sub-problem, an improved sequential cone programming (SCP) method is applied based on SOC relaxation and the original sub-problem is converted to MISOCP problem. A benchmark 6-node natural gas system and 6-bus electricity system is used to illustrate the effectiveness of the proposed framework. Considering pipeline congestion, CO, with demand response, can reduce the total cost of an electricity network by 1.19%, as compared to −0.48% using traditional decentralized operation with demand response.

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35

Ogasa, Toshiyuki, Hitoshi Nakano, Hiroshi Ide, Yasushi Yamamoto, Nobuhiko Sasaki, Shinobu Osanai, Yuji Akiba, Kenjiro Kikuchi, and Jun Iwamoto. "Flow-mediated release of nitric oxide in isolated, perfused rabbit lungs." Journal of Applied Physiology 91, no. 1 (July 1, 2001): 363–70. http://dx.doi.org/10.1152/jappl.2001.91.1.363.

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The effects of changing perfusate flow on lung nitric oxide (NO) production and pulmonary arterial pressure (Ppa) were tested during normoxia and hypoxia and after N G-monomethyl-l-arginine (l-NMMA) treatment during normoxia in both blood- and buffer-perfused rabbit lungs. Exhaled NO (eNO) was unaltered by changing perfusate flow in blood-perfused lungs. In buffer-perfused lungs, bolus injections of ACh into the pulmonary artery evoked a transient increase in eNO from 67 ± 3 (SE) to 83 ± 7 parts/billion with decrease in Ppa, whereas perfusate NO metabolites (pNOx) remained unchanged. Stepwise increments in flow from 25 to 150 ml/min caused corresponding stepwise elevations in eNO production (46 ± 2 to 73 ± 3 nl/min) without changes in pNOx during normoxia. Despite a reduction in the baseline level of eNO, flow-dependent increases in eNO were still observed during hypoxia.l-NMMA caused declines in both eNO and pNOx with a rise in Ppa. Pulmonary vascular conductance progressively increased with increasing flow during normoxia and hypoxia. However,l-NMMA blocked the flow-dependent increase in conductance over the range of 50–150 ml/min of flow. In the more physiological conditions of blood perfusion, eNO does not reflect endothelial NO production. However, from the buffer perfusion study, we suggest that endothelial NO production secondary to increasing flow, may contribute to capillary recruitment and/or shear stress-induced vasodilation.

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Fissan, Heinz, Hans-Georg Horn, Burkhard Stahlmecke, and Jing Wang. "From nanoobject release of (Bio)nanomaterials to exposure." BioNanoMaterials 14, no. 1-2 (September 1, 2013): 37–47. http://dx.doi.org/10.1515/bnm-2013-0004.

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AbstractAn increasing variety of different nanostructured materials including bionanomaterials are used. During synthesis, but also during use of nanostructured materials along their life-cycle, nanostructured materials and engineered nano-objects (ENO) – may be released into the environment. They will follow different exposure pathways and create an exposure concentration at the point of different biological systems, especially human beings. The inhalation pathway is of greatest importance with regard to health issues. The exposure concentration together with the breathing conditions integrated over time leads to the dose of the deposited material, which is of greatest interest for different effect studies. We discuss in this paper the kind of nanostructured material released from bionanomaterials into the environment. A large part of existing exposure studies in the literature is critically considered. A strategy is proposed to investigate in a more effective way the ENO-release from nanostructured materials as the first step of the exposure pathway. The release – exposure relationship as well as exposure – dose relationship for the case of inhalation is described leading to the possibility of tracing and ideally a complete balancing from ENO-release to dose. In the end the still needed activities for ENO-control methods in the environment are summarized.

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Hamdan, Mager, Luis Rojas, Ysbelia Obregón, Rosa Aparicio, Alida Pérez, Yndra Cordero, Clara Díaz, Jossblerys Da Silva, and Alfredo Usubillaga. "Derivados hemisintéticos del ent-kaurenol y evaluación de su actividad antimicrobiana." Revista de la Facultad de Farmacia 64, no. 1 (2022): 20–28. http://dx.doi.org/10.53766/refa/2022.64.01.03.

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La obtención de seis nuevos derivados hemisintéticos a partir de ent-kaurenol (I), se realizó mediante la reacción de esterificación de Steglich empleando los ácidos p-cloro-fenil-acético, o-cloro-fenil-acético, m-cloro-fenil-acético, p-bromo-fenil-acético, nicotínico y salicílico; utilizando la combinación de diciclohexilcarbodiimida (DCC, agente de acoplamiento), 4-dimetilaminopiridina (DMAP, catalizador nucleofílico) y como solvente el diclorometano. Todos los compuestos fueron caracterizados mediante técnicas espectroscópicas de IR y RMN uni y bidimensionales, lográndose identificar como ent-kaur-19-O-[2’-(p-cloro-fenil)-carboximetil]-16-eno (II), ent-kaur-19-O-[2´-(o-cloro-fenil)-carboximetil]-16-eno (III), ent-kaur-19-O-[2’-(m-cloro-fenil)-carboximetil]-16-eno (IV), ent-kaur-19-O-[2’-(p-bromo-fenil)-carboximetil]-16-eno (V), ent-kaur-19-O-(m-piridil-carboxil)-16-eno (VI) y ent-kaur-19-O-(o-hidroxi-fenil-carboxil)-16-eno (VII). Estos compuestos no presentaron actividad antimicrobiana mediante el método de difusión en agar en pozo frente a cepas ATCC de Staphylococcus aureus, Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, Pseudomonas aeruginosa y Candida albicans a una concentración de 2 mg/mL.

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Boucheneb, Hanifa, and John Mullins. "Analyse des réseaux temporels. Calcul des classes enO(n2)et des temps de chemin enO(m ×n)." Techniques et sciences informatiques 22, no. 4 (April 1, 2003): 435–59. http://dx.doi.org/10.3166/tsi.22.435-459.

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MANISCALCO, Mauro, Alessandro VATRELLA, George CREMONA, Luigi CARRATÙ, and Matteo SOFIA. "Exhaled nitric oxide after inhalation of isotonic and hypotonic solutions in healthy subjects." Clinical Science 101, no. 6 (November 20, 2001): 645–50. http://dx.doi.org/10.1042/cs1010645.

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Airway nitric oxide (NO) homoeostasis is influenced by chemical and mechanical stimuli in humans; airway epithelium, which is an important site of NO production, is sensitive to osmotic challenge. The effect of inhaled hypotonic solutions on exhaled NO (eNO) is not known. In this study we evaluated the effect of ultrasonically nebulized distilled water (UNDW), a hypotonic indirect stimulus, on eNO levels. A total of 10 non-smoking healthy subjects were enrolled in the study. eNO was detected by chemiluminescence, and specific airway conductance (sGaw) was measured by plethysmography. Bronchial challenges with UNDW and with an isotonic solution were performed according to a double-blind experimental design. Baseline levels of eNO were 28.1±14.7p.p.b. UNDW did not cause any significant change in sGaw (from 0.190±0.029 to 0.181±0.036cmH2Oċs-1). With respect to baseline values, the eNO concentration decreased significantly after inhalation of 8 or 16ml of UNDW (from 26.0±13.1 to 17.2±8.5 and 16.6±7.7p.p.b. respectively; P < 0.001, n = 10). After bronchial challenge with UNDW, eNO was significantly reduced in comparison with after inhalation of the isotonic solution. In five subjects, pretreatment with NG-nitro-l-arginine methyl ester (l-NAME), an inhibitor NO synthesis, decreased NO levels from 21.7±8.5 to 10.0±3.3p.p.b. Subsequent inhalation of 16ml of UNDW did not cause any further decrease in NO levels (10.1±3.7p.p.b.; not significant compared with l-NAME). We conclude that inhalation of aqueous solutions decreases eNO levels in healthy subjects, and that this effect is not associated with any significant change in airway calibre. The UNDW-induced decrease in eNO is not enhanced by pretreatment with the NO synthase inhibitor l-NAME, suggesting that inhaled solutions may interfere with the airway NO pathway in humans.

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Ratnawati, Ratnawati, V. Nguyen, J. Morton, R. L. Henry, and P. S. Thomas. "Exhaled nitric oxide in acute exacerbation of pediatric asthma." Paediatrica Indonesiana 48, no. 2 (May 1, 2008): 64. http://dx.doi.org/10.14238/pi48.2.2008.64-70.

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Background Measurement of exhaled nitric oxide (eNO) is a non-invasive and easy method to monitor airway inflammation.Objectives To compare the levels of eNO during and after anexacerbation of asthma, to evaluate the effect of glucocor-ticosteroids (GCS) on the levels of eNO and to correlate eNOwith other markers of inflammation such as symptom scores, FEV 1and sputum eosinophils.Methods The observational study was performed over 24 monthsat a tertiary paediatric hospital. Subjects underwent eNOmeasurement, spirometry and sputum induction during an asthmaexacerbation and then two weeks later. A symptom score wasrecorded everyday for two weeks. All subjects were treated withß 2 -adrenergic agonists and an oral glucocorticosteroid (GCS).Results Fifteen subjects with acute asthma exacerbation aged 5and 16 years old participated in the study. The mean level ofeNO during the acute exacerbation was significantly higher thaneNO levels at the follow-up visit, 11.2 (95%CI 9.2;13.2) vs. 8.0(95%CI 5.0;11.1) ppb, P=0.03. In the acute exacerbation, eNOcorrelated with sputum eosinophils (P=0.04), but no correlationcould be found between eNO and the other markers ofinflammation during exacerbation or follow up.Conclusions eNO level increased during asthma exacerbation anddecreased after two weeks of glucocorticosteroid therapy.Measurement of eNO is a practical monitoring method inemergency management of asthmatic children.

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41

Dewanto, F. Bethavianus Bayu, and Setyo Prasiyanto Cahyono. "Semiotics Analysis on Historical Memes in Eno Bening’s Tweet Replies." Applied Linguistics, Linguistics, and Literature (ALLURE) Journal 1, no. 1 (July 31, 2021): 33–52. http://dx.doi.org/10.26877/allure.v1i1.9291.

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This research is entitled Semiotics Analysis on Historical Memes in Eno Bening’s Tweet Replies. The study concerns with all the way the researchers explore the meaning of representamen/signs on either non-verbal and verbal data of historical memes in Eno Bening’s tweet replies. The method used in this paper is descriptive qualitative analysis where the researchers describe the meaning of the memes. The researchers also use semiotics analysis proposed by Pierce’s semiotic analysis (1994) in order to discover the interpretation of meaning implied in the memes. From the analysis, the researchers conclude that the historical memes in Eno Bening’s tweet replies mostly used to retell a story of historical events that happened in the past. Furthermore, memes are also used to criticize, give an opinion and also satirize a historical event.

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McLaughlin, Noel. "Another Green World?: Eno, Ireland and U2." Popular Music History 9, no. 2 (December 21, 2015): 173–94. http://dx.doi.org/10.1558/pomh.v9i2.29457.

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Chan, Tony F., and H. M. Zhou. "ENO-Wavelet Transforms for Piecewise Smooth Functions." SIAM Journal on Numerical Analysis 40, no. 4 (January 2002): 1369–404. http://dx.doi.org/10.1137/s0036142900370915.

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Abboy, Chandar, and Peter Spiegler. "Unsure Benefits of eNO to Manage Asthma." Clinical Pulmonary Medicine 16, no. 2 (March 2009): 107. http://dx.doi.org/10.1097/cpm.0b013e31819b339a.

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Aràndiga, Francesc, Pep Mulet, and Vicent Renau. "Non-separable two-dimensional weighted ENO interpolation." Applied Numerical Mathematics 62, no. 8 (August 2012): 975–87. http://dx.doi.org/10.1016/j.apnum.2012.03.005.

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Lin, Chi-Tien, and Xu-Dong Liu. "Convex ENO Schemes for Hamilton–Jacobi Equations." Journal of Scientific Computing 31, no. 1-2 (January 27, 2007): 195–211. http://dx.doi.org/10.1007/s10915-006-9121-9.

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47

Jiang, Guang-Shan, and Danping Peng. "Weighted ENO Schemes for Hamilton--Jacobi Equations." SIAM Journal on Scientific Computing 21, no. 6 (January 2000): 2126–43. http://dx.doi.org/10.1137/s106482759732455x.

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48

Zhang, Xuliang, Lixin Xu, Xiaokai Song, Xiangrui Li, and Ruofeng Yan. "Molecular cloning of enolase from Trichinella spiralis and the protective immunity in mice." Acta Parasitologica 63, no. 2 (June 26, 2018): 252–60. http://dx.doi.org/10.1515/ap-2018-0029.

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Abstract Trichinella spiralis, the main pathogen of trichinosis, infects a wide range of mammalian hosts and is one of the most widespread parasites worldwide. For parasites, glycolysis is the most important way to generate energy. Previous studies showed that some enzymes involved in the glycolytic pathway play roles in regulation the host immunity. In this paper, enolase from T. spiralis was cloned and the protective potentials were studied. One hundred and sixty ICR mice were divided into four groups and vaccinated with recombinant enolase (pET-ENO), eukaryotic recombinant plasmid encoding enolase (pVAX1-ENO) and negative controls (pVAXl and PBS), respectively. Two weeks after the second immunization, each mouse was challenged orally with 200 muscle larvae (MLs) of T. spiralis. Results showed that mice vaccinated with pET-ENO and pVAX1-ENO induced specific antibodies of IgG, IgA, IgM, but no IgE. Subclasses of IgG antibodies showed that mice immunized with recombinant protein and recombinant plasmids induced a Th1/Th2 immune response. Concentrations of serum cytokines were detected and showed significant increase of IFN-γ, IL-4 and TGFβ1, while IL-17 in each group was not significantly different. Flow cytometric analysis showed significant increase of CD4+ and CD8+ T lymphocytes in the groups immunized with recombinant protein and recombinant plasmids. Challenge infection demonstrated that immunized groups had a reduced number of worm burdens. The reductions of larvae per gram muscle (LPG) in pET-ENO and pVAX1-ENO group were 17.7% and 15.8% when compared with PBS control.

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FORREST, Ian A., Therese SMALL, and Paul A. CORRIS. "Effect of nebulized epoprostenol (prostacyclin) on exhaled nitric oxide in patients with pulmonary hypertension due to congenital heart disease and in normal controls." Clinical Science 97, no. 1 (June 1, 1999): 99–102. http://dx.doi.org/10.1042/cs0970099.

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Inhaled epoprostenol (prostacyclin) may be used in the treatment of severe pulmonary hypertension, improving oxygenation and reducing pulmonary artery pressures. We have observed symptomatic benefits of epoprostenol in patients with congenital heart disease that extend beyond acute haemodynamic effects of the drug, which has a short biological half-life. The aim of this study was to examine the effects of epoprostenol in patients and normal subjects on exhaled nitric oxide (eNO), based on the hypothesis that the drug may alter the resting vasoconstrictor/vasodilator balance. Nine patients with pulmonary hypertension complicating left-to-right cardiac shunts and nine healthy controls received 100 µg of nebulized epoprostenol. Exhaled eNO was measured, using a chemiluminescence method, before, immediately after and 18 h after nebulization. There was no significant difference between the two groups in baseline eNO or eNO immediately following nebulized epoprostenol. Epoprostenol produced a delayed elevation in eNO 18 h after nebulization in patients, but not in normal controls. This study supports the concept that epoprostenol, while having no effect on the normal pulmonary circulation, acts on the hypertensive circulation via a mechanism that may result in a delayed alteration of vasoconstrictor/vasodilator balance.

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Kadioglu, Samet Y. "An Essentially Nonoscillatory Spectral Deferred Correction Method for Hyperbolic Problems." International Journal of Computational Methods 13, no. 03 (May 31, 2016): 1650017. http://dx.doi.org/10.1142/s0219876216500171.

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We present a computational method based on the spectral deferred corrections (SDC) time integration technique and the essentially nonoscillatory (ENO) finite volume method for hyperbolic problems. The SDC technique is used to advance the solutions in time with high-order of accuracy. The ENO method is used to define high-order cell edge quantities that are then used to evaluate numerical fluxes. The coupling of the SDC method with a high-order finite volume method (piece-wise parabolic method (PPM)) is first carried out by Layton et al. [J. Comput. Phys. 194(2) (2004) 697]. Issues about this approach have been addressed and some improvements have been added to it in Kadioglu et al. [J. Comput. Math. 1(4) (2012) 303]. Here, we investigate the implications when the PPM method is replaced with the well-known ENO method. We note that the SDC-PPM method is fourth-order accurate in time and space. Therefore, we kept the order of accuracy of the ENO procedure as fourth-order in order to be able to make a consistent comparison between the two approaches (SDC-ENO versus SDC-PPM). We have tested the new SDC-ENO technique by solving smooth and nonsmooth hyperbolic problems. Our numerical results indicate that the fourth-order of accuracy in both space and time has been achieved for smooth problems. On the other hand, the new method performs very well when it is applied to nonlinear problems that involve discontinuous solutions. In other words, we have obtained highly resolved discontinuous solutions with essentially no-oscillations at or around the discontinuities.

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