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Books on the topic 'Endonucleasi'

Author: Grafiati

Published: 9 March 2023

Last updated: 10 March 2023

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1

Edgell, David R., ed. Homing Endonucleases. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-968-0.

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2

Pingoud, Alfred M., ed. Restriction Endonucleases. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18851-0.

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3

Homing endonucleases: Methods and protocols. New York: Humana Press, 2014.

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4

Belfort, Marlene, David W. Wood, Barry L. Stoddard, and Victoria Derbyshire, eds. Homing Endonucleases and Inteins. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/3-540-29474-0.

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5

G, Chirikjian Jack, ed. Restriction endonucleases and methylases. New York: Elsevier, 1987.

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6

Bolton, Bryan John. Class ii restriction endonucleases: Screening, purification and characterization. Salford: University of Salford, 1988.

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7

Cross, Stephen R. H. Restriction endonuclease map variation and natural selection in populations of Drosophila melanogaster. Birmingham: University of Birmingham, 1985.

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8

Price, Rebecca Clare. The effects of restriction endonucleases on mammalian cells of different radiosensitivity. Manchester: University of Manchester, 1994.

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9

Fraser, Murray J. Endo-exonucleases. Austin, Tex: R.G. Landes Co., 1996.

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10

Cartwright, Nicola. Detection and typing of human papillomavirus using semi-nested PCR and restriction endonuclease analysis with respect to vulval carcinoma. [s.l.]: typescript, 1996.

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11

1955-, Zenkova Marina A., ed. Artificial nucleases. New York: Springer-Verlag, 2004.

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12

1932-, Eckstein Fritz, and Lilley, David M. J. 1948-, eds. Nucleic acids and molecular biology. Berlin: Springer-Verlag, 1987.

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13

Pingoud, Alfred. Restriction Endonucleases. Springer London, Limited, 2012.

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14

Pingoud, Alfred. Restriction Endonucleases. Springer, 2012.

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15

Edgell, David R. Homing Endonucleases: Methods and Protocols. Humana Press, 2016.

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16

Belfort, Marlene, Barry L. Stoddard, David W. Wood, and Vicky Derbyshire. Homing Endonucleases and Inteins. Springer, 2008.

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17

David W. Wood,Marlene Belfort,Barry L. Stoddard. Homing Endonucleases and Inteins. Springer, 2008.

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18

Wah, David Alan. Structural study of the type IIs restriction endonuclease FokI. 1998.

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19

Pingoud, Alfred M. Restriction Endonucleases (Nucleic Acids and Molecular Biology). Springer, 2004.

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20

Rigby, Susan. The role of DNA secondary structure on restriction endonuclease activity. 1987.

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21

Hawkes, Tracey-Elaine. Use of restriction endonucleases for typing of adenovirus isolates. 1995.

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22

Homing Endonucleases and Inteins (Nucleic Acids and Molecular Biology Book 16). Springer, 2006.

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23

Population structure of the Atlantic bottlenose dolphin as determined by restriction endonuclease analysis of mitochondrial dna. [Washington, D.C.?: Marine Mammal Commission, 1992.

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24

Romano, Patrick Robert. Restriction endonuclease mapping of rRNA genes and the origin of the tetraploid tree frog Hyla versicolor. 1985.

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25

(Editor), Marlene Belfort, Barry L. Stoddard (Editor), David W. Wood (Editor), and Victoria Derbyshire (Editor), eds. Homing Endonucleases and Inteins (Nucleic Acids and Molecular Biology) (Nucleic Acids and Molecular Biology). Springer, 2005.

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26

Nuclease Methods and Protocols. Humana Press, 2001.

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27

Grimme, Stephan. Immunhistochemischer Nachweis von Apurinic/Apyrimidinic Endonuclease (APE) in benachbarten Schnitten von Ovarialkarzinomen und die Korrelation zu bekannten klinisch pathologischen Prognoseparametern und Überlebenszeit. 2010.

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28

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Abstract:

Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.

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29

Modern Prometheus: Editing the Human Genome. University of Cambridge ESOL Examinations, 2016.

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30

Krecic, Annette Marie. Nuclear and cytoplasmic functions of a yeast heterogeneous nuclear ribonucleoprotein. 1998.

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31

Kozubek, Jim. Modern Prometheus: Editing the Human Genome with Crispr-Cas9. Cambridge University Press, 2018.

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