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Relevant bibliographies by topics / Endogenous opioids

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Academic literature on the topic 'Endogenous opioids'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

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Journal articles on the topic "Endogenous opioids"

1

Herz, Albert. "Opioid reward mechanisms: a key role in drug abuse?" Canadian Journal of Physiology and Pharmacology 76, no. 3 (1998): 252–58. http://dx.doi.org/10.1139/y98-017.

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There is increasing evidence to implicate the mesolimbic dopamine system in the rewarding effects of drugs of abuse such as opioids, psychostimulants, and alcohol, and in addition endogenous opioids may play a key role in the underlying adaptive mechanisms. Opioid agonists with affinity for µ and delta opioid receptors are rewarding, whereas opioid agonists with affinity for kappa receptors are aversive. These opposing motivational effects are paralleled by an increase and decrease, respectively, of dopamine release in the nucleus accumbens. Opposite effects are induced in response to selectiv

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2

Smith, Howard. "Peripherally-Acting Opioids." Pain Physician 2s;11, no. 3;2s (2008): S121—S132. http://dx.doi.org/10.36076/ppj.2008/11/s121.

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Opioids are broad-spectrum analgesics with potent pain-relieving qualities but also with potential adverse effects related to both short-term and long-term therapy. Researchers have attempted to alter existing opioid analgesics, utilize different routes/ formulations, or combine opioid analgesics with other compounds in efforts to improve analgesia while minimizing adverse effects. Exogenous opioids, administered in efforts to achieve analgesia, work by mimicking the actions of endogenous opioids. Endogenous opioids and their receptors are located in the brain (supraspinal areas), spinal cord,

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3

Bovill, James G. "Endogenous opioids and opioid receptors." Current Opinion in Anaesthesiology 6, no. 4 (1993): 668–72. http://dx.doi.org/10.1097/00001503-199308000-00014.

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Corder, Gregory, Daniel C. Castro, Michael R. Bruchas, and Grégory Scherrer. "Endogenous and Exogenous Opioids in Pain." Annual Review of Neuroscience 41, no. 1 (2018): 453–73. http://dx.doi.org/10.1146/annurev-neuro-080317-061522.

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Opioids are the most commonly used and effective analgesic treatments for severe pain, but they have recently come under scrutiny owing to epidemic levels of abuse and overdose. These compounds act on the endogenous opioid system, which comprises four G protein–coupled receptors (mu, delta, kappa, and nociceptin) and four major peptide families (β-endorphin, enkephalins, dynorphins, and nociceptin/orphanin FQ). In this review, we first describe the functional organization and pharmacology of the endogenous opioid system. We then summarize current knowledge on the signaling mechanisms by which

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Santiago, T. V., and N. H. Edelman. "Opioids and breathing." Journal of Applied Physiology 59, no. 6 (1985): 1675–85. http://dx.doi.org/10.1152/jappl.1985.59.6.1675.

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This review summarizes recent developments on the effects of opiate drugs and the various endogenous opioid peptides on breathing. These developments include demonstration of receptors and site-specific effects of application of opioids in the pons and medulla, demonstration of variable tolerance of respiratory responses in addicted individuals as well as their offspring, and demonstration of an endogenous opioid influence on breathing in early neonatal life and in certain physiological settings and disease states. The validity and limitations of using naloxone as a tool to uncover postulated

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Rees, John M. H. "Endogenous opioids." Baillière's Clinical Rheumatology 1, no. 1 (1987): 27–56. http://dx.doi.org/10.1016/s0950-3579(87)80028-6.

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Gintzler, PhD, Alan R., and Nai-Jiang Liu, MD, PhD. "Harnessing endogenous opioids for pain relief: Fantasy vs reality." Journal of Opioid Management 16, no. 1 (2020): 67–72. http://dx.doi.org/10.5055/jom.2020.0552.

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Objective: To review evidence demonstrating efficacy and feasibility of harnessing the activity of endogenous opioid analgesic systems for pain management.Methods: The authors sought to summarize a wealth of data that establish proof of concept that the analgesic activity of endogenous opioids can be exploited to clinically benefit from the enormous pain-relieving abilities of these peptides without contributing to the current crisis of death by synthetic opioid overdose.Results: There is a plethora of studies demonstrating that not only can endogenous opioids mediate placebo-induced antinocic

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Liren, A.-L., and G. Feuerstein. "The Opioid System in Circulatory Control." Physiology 7, no. 1 (1992): 26–30. http://dx.doi.org/10.1152/physiologyonline.1992.7.1.26.

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Opioid peptides and multiple opioid receptors are found in brain cardiovascular nuclei, autonomic ganglia, the heart, and blood vessels, and opioids induce potent cardiovascular changes. The role of endogenous opioids in normal cardiovascular homeostasis is unclear;however, current data suggest opioid involvement in stress.

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9

Ismail, Zahinoor, and Nady el-Guebaly. "Nicotine and Endogenous Opioids: Toward Specific Pharmacotherapy." Canadian Journal of Psychiatry 43, no. 1 (1998): 37–42. http://dx.doi.org/10.1177/070674379804300103.

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Objective: To address the theoretical framework behind opioid receptor antagonism for the treatment of nicotine abuse. The current literature is reviewed with a focus on opioid–nicotine interactions in animals and humans. Furthermore, previous studies addressing the effect of opioid antagonism on smoking behaviour are reviewed critically with a focus on suggestions and implications for future trials. Method: Computerized data bases and reference lists of existing articles were searched for prior publications in 3 areas: 1) the association between nicotine and endogenous opioids, 2) nicotine an

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Meier, Isabell M., Marie Eikemo, and Siri Leknes. "The Role of Mu-Opioids for Reward and Threat Processing in Humans: Bridging the Gap from Preclinical to Clinical Opioid Drug Studies." Current Addiction Reports 8, no. 2 (2021): 306–18. http://dx.doi.org/10.1007/s40429-021-00366-8.

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Abstract Purpose of Review Opioid receptors are widely expressed in the human brain. A number of features commonly associated with drug use disorder, such as difficulties in emotional learning, emotion regulation and anhedonia, have been linked to endogenous opioid signalling. Whereas chronic substance use and misuse are thought to alter the function of the mu-opioid system, the specific mechanisms are not well understood. We argue that understanding exogenous and endogenous opioid effects in the healthy human brain is an essential foundation for bridging preclinical and clinical findings rela

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Dissertations / Theses on the topic "Endogenous opioids"

1

Ghule, Aishwarya [Verfasser]. "The importance of endogenous opioids in feeding behavior / Aishwarya Ghule." Bonn : Universitäts- und Landesbibliothek Bonn, 2018. http://d-nb.info/1183093942/34.

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Stagg, Nicola Jane. "Reversal of Neuropathic Pain with Exercise is Mediated by Endogenous Opioids." Diss., The University of Arizona, 2007. http://hdl.handle.net/10150/194833.

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Exercise is often prescribed for patients with chronic pain, but there is little objective evidence supporting this recommendation. Therefore, we tested the effect of moderate aerobic exercise on the sensory hypersensitivity produced in an animal model of neuropathic pain. Male rats that underwent unilateral ligation of the L5 and L6 spinal nerves (SNL) were divided into exercise-trained or sedentary groups. Exercise training was performed using a treadmill, beginning 7 days after surgery, and continued 5 days a week for 5 weeks. Animals were exercised 30 min/day, at a speed of 14-16 m/mi

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Robinson, Kathleen Clare. "Cutaneous Biology and Endogenous Opioids: How the Skin Modulates Pain and Addiction." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11215.

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The Proopiomelanocortin gene, (POMC), produces many biologically active peptides including the endogenous opioid, β-endorphin, and the melanocortins: α-Melanocyte Stimulating Hormone, (αMSH), γMSH, βMSH and Adrenocorticotropic Hormone, (ACTH). βendorphin is released by the brain in response to stress or injury and is a potent analgesic. Melanocortins are well known for regulating pigmentation, metabolism, and cortisol levels. Additionally, opioids and melanocortins are known to have opposing actions in several settings including the regulation of pain and met

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au, Ash_Frew@yahoo com, and Ashley Kim Frew. "The influence of discouragement, anxiety and anger on pain: An examination of the role of endogenous opioids." Murdoch University, 2005. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20050930.111852.

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Animal research suggests that exposure to inescapable stressors can lead to an endogenous opioid-mediated form of pain inhibition, known as stress-induced analgesia (SIA). Similar results have been found with humans, although the literature is much less extensive and at times contradictory where uncontrollable stressors have led to an increase, rather than a decrease in pain. More recently, there has been some suggestion that emotions play an important role in pain modulation, and that particular negative moods are associated with opioid-mediated hypoalgesia. This research aimed to clarify the

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Gustafsson, Lisa. "Endogenous Opioids and Voluntary Ethanol Drinking : Consequences of Postnatal Environmental Influences in Rats." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7776.

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Daoura, Loudin. "Early Environment and Adolescent Ethanol Consumption : Effects on Endogenous Opioids and Behaviour in Rats." Doctoral thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-198670.

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Excessive and compulsive ethanol drinking is one of the most serious public health issues. Therefore, it is vital to increase the knowledge about risks and protection for alcohol use disorders (AUD) to optimize prevention and treatment strategies. Ethanol consumption commonly initiates during adolescence when extensive neuronal maturation and development also occurs. Early exposure to ethanol is a risk factor for AUD, but the effects of adolescent drinking and the basis for the individual susceptibility to AUD are not fully understood. The interactions between genotype and environmental factor

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Olson, Keith Mathew, and Keith Mathew Olson. "Atypical Opioid Interactions – Development of Selective Mu-Delta Heterodimer Antagonists, Clinical Opioids at Non-Mu Pain Targets and Endogenous Biased Signaling." Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/626669.

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Most clinical opioids produce analgesia through the Mu Opioid Receptor (MOR) providing the only effective treatment for chronic pain patients. These studies explore three pre-clinical strategies to improve MOR analgesia and minimize side effects: 1) compounds that target G-protein Coupled Receptors (GPCRs) heterodimers, such as heterodimerization between the Delta Opioid Receptor (DOR) and MOR (MDOR); 2) multi-functional compounds that target multiple receptor systems for synergistic effects, such as a MOR agonist and a the serotonin reuptake transporter (SERT) inhibitor; or 3) biased agonists

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Schwellnus, Martin Peter. "The role of the endogenous opioid system in thermoregulation during exercise." Master's thesis, University of Cape Town, 1988. http://hdl.handle.net/11427/27169.

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In man the metabolic heat produced during physical exercise stresses the thermoregulatory system, particularly if hot, humid environmental conditions prevail. It has recently been postulated that endogenous opioids may play a role in regulating body temperature at rest and because it has also been shown that blood levels of these substances increase during exercise, the possibility exists that endogenous opioids may play a role in thermoregulation during exercise. A study was conducted in two parts to determine the thermoregulatory response during exercise with and without pharmacologic blocka

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Frew, Ashley. "The influence of discouragement, anxiety and anger on pain : an examination of the role of endogenous opioids /." Access via Murdoch University Digital Theses Project, 2004. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20050930.111852.

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Frew, Ashley Kim. "The influence of discouragement, anxiety and anger on pain: an examination of the role of endogenous opioids." Thesis, Frew, Ashley Kim (2005) The influence of discouragement, anxiety and anger on pain: an examination of the role of endogenous opioids. PhD thesis, Murdoch University, 2005. https://researchrepository.murdoch.edu.au/id/eprint/55/.

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Animal research suggests that exposure to inescapable stressors can lead to an endogenous opioid-mediated form of pain inhibition, known as stress-induced analgesia (SIA). Similar results have been found with humans, although the literature is much less extensive and at times contradictory where uncontrollable stressors have led to an increase, rather than a decrease in pain. More recently, there has been some suggestion that emotions play an important role in pain modulation, and that particular negative moods are associated with opioid-mediated hypoalgesia. This research aimed to clarify the

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Books on the topic "Endogenous opioids"

1

Holaday, John W. Endogenous opioids and their receptors. Upjohn, 1985.

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2

Marcello, Negri, Lotti G, and Grossman Ashley, eds. Clinical perspectives of endogenous opioids production. J. Wiley, 1992.

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3

Browning, Andrew J. F. Endogenous opioid peptides and human reproduction. University of Birmingham, 1985.

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4

Salansky, Norman. Endogenous opioid peptide level changes under electrostimulation and their assessment by the EEG. [s.n.], 1994.

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Sommer, Claudia. Endogenous opioids mediate stress-induced analgesia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0031.

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This chapter reviews the landmark paper published in 1990 by Stein et al. and entitled ‘Opioids from immunocytes interact with receptors on sensory nerves to inhibit nociception in inflammation’. Opioids, besides acting centrally as analgesics, may act peripherally upon opioid receptors located on axons and on immune cells. In the publication by Stein et al., it was shown for the first time that endogenous opioid peptides released from immune cells mediate stress-induced analgesia, potentially through opioid receptors on peripheral nerve endings. This finding has led to numerous follow-up stud

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Dickenson, Tony. Endogenous opioids in the CNS. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0019.

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This short and concise paper was the first to unequivocally reveal that there were endogenous opioids in the central nervous system (CNS), identify their peptide nature and sequence, and show that they exerted physiological inhibitory effects. The idea that there were natural opioids fitted with concurrent reports of opiate-binding sites, and this led to the description of multiple receptors with their own families of peptide transmitters. No truly novel opioid drugs have emerged since, and attempts to protect and manipulate the enkephalins for pain control have yet to be successful. This does

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Taylor, Véronique A., and Pierre Rainville. Endogenous opioids in placebo-induced analgesia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0011.

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Placebos achieve scientifically proven pain-relieving effects yet are inactive substances for the treatment of pain. Levine, Gordon, and Fields were the first to demonstrate the role of endogenous opioids in placebo-induced analgesia during dental post-operative pain. Several studies using pharmacological manipulations and/or neuroimaging techniques confirmed their findings that placebo analgesia is reversible by naloxone, and also identified brain pathways involved in opioidergic neurotransmission during placebo analgesia (prefrontal regions rich in opioid receptors such as the anterior cingu

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Bird, Mark F., and David G. Lambert. Deorphanization of ORL-1/LC132 by reverse pharmacology in two landmark studies. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0026.

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Deorphanization of ORL-1/LC132 in 1995 by reverse pharmacology in two simultaneously published landmark studies added a new member to the opioid family of G-protein coupled receptors. Meunier and Reinscheid used cells expressing recombinant ORL-1 (human) or LC132 (rat) and the presumed intracellular inhibition of cyclic AMP formation to ‘fish’ for endogenous peptide ligands in rat whole-brain and pig hypothalamic extracts. Both studies reported the isolation of a 17-amino-acid peptide, which was named nociceptin and orphanin FQ by the two authors, respectively. The behaviour of the isolated pe

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Bromley, Lesley. The molecular basis for the placebo effect. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0041.

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The landmark paper discussed in this chapter is ‘The neurobiology of placebo analgesia: From endogenous opioids to cholecystokinin’, published by Benedetti and Amanzio in 1997. This major review considered the placebo and nocebo effect in a more scientific framework compared to previous treatise of a nebulous concept whose only role is to act as a comparator for controlled trials. By expounding robust evidence, Benedetti and Amanzio added credence to the placebo effect, with not just psychological but also physiological data, acknowledging it as an effective therapeutic action. Furthermore, th

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Simon, Eric J., and Hiroshi Takagi. Advances in Endogenous and Exogenous Opioids: Proceedings of the International Narcotic Research Conference Held in Kyoto, Japan on July 26-30 1981. Elsevier, 2013.

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Book chapters on the topic "Endogenous opioids"

1

Berezniuk, Iryna, and Lloyd D. Fricker. "Endogenous Opioids." In The Opiate Receptors. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-993-2_5.

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Spector, S., and J. Donnerer. "Presence of Endogenous Opiate Alkaloids in Mammalian Tissues." In Opioids. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77460-7_13.

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Khachaturian, H., M. K. H. Schaefer, and M. E. Lewis. "Anatomy and Function of the Endogenous Opioid Systems." In Opioids. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77460-7_20.

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Blatteis, C. M., and A. A. Romanovsky. "Endogenous Opioids and Fever." In Thermal Balance in Health and Disease. Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-7429-8_61.

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McCubbin, James A. "Endogenous Opioids/Endorphins/Enkephalin." In Encyclopedia of Behavioral Medicine. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_247.

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LaCaille, Lara, Anna Maria Patino-Fernandez, Jane Monaco, et al. "Endogenous Opioids/Endorphins/Enkephalin." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_247.

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McCubbin, James A. "Endogenous Opioids/Endorphins/Enkephalin." In Encyclopedia of Behavioral Medicine. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-6439-6_247-2.

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Carr, D. B., and S. M. Fishman. "EXERCISE AND ENDOGENOUS OPIOIDS." In Exercise Endocrinology, edited by K. Fotherby and S. B. Pal. De Gruyter, 1985. http://dx.doi.org/10.1515/9783110866483-010.

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Papini, Mauricio R., and Leonardo A. Ortega. "Endogenous Opioids, Opioid Receptors, and Incentive Processes." In Handbook of Behavior, Food and Nutrition. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-92271-3_66.

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Faden, A. I. "Role of Endogenous Opioids in Central Cardiovascular Regulation and Dysregulation." In Opioids II. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77540-6_8.

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Conference papers on the topic "Endogenous opioids"

1

Gifford, Alex H., Laurie A. Waterman, Joseph Ward, John Baird, and Donald A. Mahler. "Neuromodulatory Effect Of Endogenous Opioids On The Intensity And Unpleasantness Of Breathlessness In COPD." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5806.

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Ekstrom, M. P., T. Hunt, S. Louw, et al. "The Effect of Endogenous Opioids on the Peripheral Modulation of Breathlessness in People with Moderate to Severe COPD." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5741.

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Mangani, Davide, Linglin Huang, Meromit Singer, et al. "964 Dynamic immune landscapes during melanoma progression reveal a role for endogenous opioids in driving T cell dysfunction." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0964.

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Olson, Keith M., Justin Lavigne, John Streicher, Frank Porreca, and Victor J. Hruby. "Differential Potencies for Endogenous Dynorphins Indicate Functional Selectivity at the Delta Opioid Receptor." In The 24th American Peptide Symposium. Prompt Scientific Publishing, 2015. http://dx.doi.org/10.17952/24aps.2015.223.

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Nazarova, G. A., N. G. Bogdanova, E. V. Alekseeva, and S. K. Sudakov. "INTERMITTENT USE OF SOLUTIONS OF GLUCOSE, SODIUM CHLORIDE AND MONOSODIUM GLUTAMATE CAUSES ENDOGENOUS OPIOID DEPENDENCE." In MODERN PROBLEMS IN SYSTEMIC REGULATION OF PHYSIOLOGICAL FUNCTIONS. NPG Publishing, 2019. http://dx.doi.org/10.24108/5-2019-confnf-59.

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Fabri, Júlia Campos, Maria Julia Filgueiras Granato, Maria Clara Lopes Rezende, Maria Luiza Franco de Oliveira, and Leandro de Souza Cruz. "The impact of genetic polymorphism in pain mechanisms." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.708.

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Background:Variations in genes codifying target structures in the nociceptive pathway can result in pain attenuation or increase.Objective:Investigate the genetic polymorphism influence in the individual pain threshold. Methods: Search on PubMed with the terms “genetic”, “pain” and its synonyms published in the last 10 years. Results:The subjective and individual mechanisms of pain aren’t completely understood, but genetic susceptibility is one of the hypothesis to explain these differences.The KCNK18 gene influences the synaptic transmission by producing potassium channel protein that equaliz

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