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Journal articles on the topic 'Endocrinology; Type-2 diabetes mellitus'

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Published: 4 June 2021
Last updated: 15 February 2022

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1

Shivaprasad, C., and Sanjay Kalra. "Bromocriptine in type 2 diabetes mellitus." Indian Journal of Endocrinology and Metabolism 15, no. 5 (2011): 17. http://dx.doi.org/10.4103/2230-8210.83058.

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2

Lastra, Guido, Sofia Syed, L. Romayne Kurukulasuriya, Camila Manrique, and James R. Sowers. "Type 2 Diabetes Mellitus and Hypertension." Endocrinology and Metabolism Clinics of North America 43, no. 1 (March 2014): 103–22. http://dx.doi.org/10.1016/j.ecl.2013.09.005.

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3

Udawat H., Harsh, and R. K. Goyal R.K. "Dyslipidaemia in type 2 diabetes mellitus." International Journal of Diabetes and Metabolism 8, no. 3 (2000): 101–10. http://dx.doi.org/10.1159/000497528.

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4

Willi, Steven M., and Leonard E. Egede. "Type 2 diabetes mellitus in adolescents." Current Opinion in Endocrinology & Diabetes 7, no. 2 (April 2000): 71–76. http://dx.doi.org/10.1097/00060793-200004000-00005.

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5

Willi, Steven M., and Leonard E. Egede. "Type 2 diabetes mellitus in adolescents." Current Opinion in Endocrinology & Diabetes 7, no. 2 (April 2000): 71–76. http://dx.doi.org/10.1097/00075197-200004000-00005.

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6

Pohl, MD, John H., Janice A. Greer, RN, and Khalid S. Hasan, MD. "TYPE 2 DIABETES MELLITUS IN CHILDREN." Endocrine Practice 4, no. 6 (November 1998): 413–16. http://dx.doi.org/10.4158/ep.4.6.413.

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7

Dedov, Ivan I., Marina V. Shestakova, Alexander Y. Mayorov, Minara S. Shamkhalova, Olga Yu Sukhareva, Gagik R. Galstyan, Alla Y. Tokmakova, et al. "Diabetes mellitus type 2 in adults." Diabetes mellitus 23, no. 2S (June 22, 2020): 4–102. http://dx.doi.org/10.14341/dm12507.

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8

Udovichenko, Oleg Viktorovich. "Comments to: ?Type 2 diabetes mellitus ? time to change the concept?" Diabetes mellitus 17, no. 1 (March 29, 2014): 85–86. http://dx.doi.org/10.14341/dm2014185-86.

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Authors of the paper ?Diabetes mellitus ? time to change the concept? (Diabetes Mellitus, 2013; №1: 91) address weight-reduction treatment modalities (including bariatric surgery) as the pivotal approach to type 2 diabetes management. While acknowledging the importance of weight loss in patients with type 2 diabetes mellitus (T2DM), this short letter aims to advise the endocrinology community against viewing bariatric surgery as the ultimate solution of all challenges of T2DM.
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9

Sharma, Arya M., and David C. W. Lau. "Obesity and Type 2 Diabetes Mellitus." Canadian Journal of Diabetes 37, no. 2 (April 2013): 63–64. http://dx.doi.org/10.1016/j.jcjd.2013.03.360.

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10

Malecki, Maciej T. "Genetics of type 2 diabetes mellitus." Diabetes Research and Clinical Practice 68 (June 2005): S10—S21. http://dx.doi.org/10.1016/j.diabres.2005.03.003.

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11

Chang, Sang Ah. "Smoking and Type 2 Diabetes Mellitus." Diabetes & Metabolism Journal 36, no. 6 (2012): 399. http://dx.doi.org/10.4093/dmj.2012.36.6.399.

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12

Trence, Dace L., and Irl B. Hirsch. "HYPERGLYCEMIC CRISES IN DIABETES MELLITUS TYPE 2." Endocrinology and Metabolism Clinics of North America 30, no. 4 (December 2001): 817–31. http://dx.doi.org/10.1016/s0889-8529(05)70217-6.

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13

Philipson, Louis H. "Harnessing heterogeneity in type 2 diabetes mellitus." Nature Reviews Endocrinology 16, no. 2 (December 12, 2019): 79–80. http://dx.doi.org/10.1038/s41574-019-0308-1.

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14

Velloso, Lício A., Decio L. Eizirik, and Miriam Cnop. "Type 2 diabetes mellitus—an autoimmune disease?" Nature Reviews Endocrinology 9, no. 12 (July 9, 2013): 750–55. http://dx.doi.org/10.1038/nrendo.2013.131.

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15

Inzucchi, Silvio E., and Robert S. Sherwin. "The Prevention of Type 2 Diabetes Mellitus." Endocrinology and Metabolism Clinics of North America 34, no. 1 (March 2005): 199–219. http://dx.doi.org/10.1016/j.ecl.2004.11.008.

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16

Afrin, Sajeda, Chandra Rani Sarkar, ATM Zoadur Rahim Zahid, and Neaz Ahmed. "Thyroid function in type 2 diabetes mellitus." Journal of Bangladesh Society of Physiologist 12, no. 2 (January 22, 2018): 61–64. http://dx.doi.org/10.3329/jbsp.v12i2.35424.

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Background: Type 2 Diabetes Mellitus (T2DM) and thyroid disorder are common endocrine disorders that affect major population worldwide. Subclinical hypothyroidism is common among T2DM.Objectives: To observe thyroid stimulating hormone (TSH), total thyroxine (TT4), free thyroxine (FT4), total triiodothyronine (TT3), free triiodothyronine (FT3) levels in newly diagnosed T2DM.Methods: This cross sectional study was conducted from July 2014 to June 2015. For this, 50 newly diagnosed T2DM patients aged 30 to 50 years of both sexes were enrolled from the Out Patient Department of Endocrinology, Rangpur Medical College and Diabetic Association, Rangpur. Fifty age matched non-diabetic healthy subjects of both sexes constituted control. Serum TSH, TT4 , FT4, TT3, FT3 levels were estimated to observe thyroid function. All these hormones were estimated by ELISA method. For statistical analysis independent sample “t” test was performed.Results: Serum TSH was significantly higher (p<0.001) and serum TT4, FT4, FT3 levels were significantly lower (p<0.001) in T2DM compared to control. Eight (16%) of T2DM were hypothyroid.Conclusion: From this study it can be concluded that altered thyroid status leading to hypothyroidism may be associated with T2DM.Bangladesh Soc Physiol. 2017, December; 12(2): 61-64
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17

Groop, Leif, and Valeriya Lyssenko. "Genes and type 2 diabetes mellitus." Current Diabetes Reports 8, no. 3 (June 2008): 192–97. http://dx.doi.org/10.1007/s11892-008-0033-y.

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18

De Galan, B. E., and J. B. L. Hoekstra. "Glucose counterregulation in Type 2 diabetes mellitus." Diabetic Medicine 18, no. 7 (July 2001): 519–27. http://dx.doi.org/10.1046/j.1464-5491.2001.00610.x.

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19

Jacob, A. N., K. Salinas, B. Adams-Huet, and P. Raskin. "Weight gain in type 2 diabetes mellitus." Diabetes, Obesity and Metabolism 9, no. 3 (May 2007): 386–93. http://dx.doi.org/10.1111/j.1463-1326.2006.00622.x.

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20

Bondar', Irina Arkad'evna, and Olesya Yur'evna Shabel'nikova. "Genetic framework of type 2 diabetes mellitus." Diabetes mellitus 16, no. 4 (December 18, 2013): 11–16. http://dx.doi.org/10.14341/dm2013411-16.

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More than 100 genes associated with the risk of type 2 diabetes mellitus (T2DM) are now established. Most of them affect insulin secretion, adipogenesis and insulin resistance, but the exact molecular mechanisms determining their involvement in the pathogenesis of T2DM are not understood completely.
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21

Ratner, Robert E. "Type 2 diabetes mellitus: the grand overview." Diabetic Medicine 15, S4 (December 1998): S4—S7. http://dx.doi.org/10.1002/(sici)1096-9136(1998120)15:4+3.0.co;2-1.

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22

Ratner, Robert E. "Type 2 diabetes mellitus: the grand overview." Diabetic Medicine 15, S4 (December 1998): S4—S7. http://dx.doi.org/10.1002/(sici)1096-9136(1998120)15:4+3.3.co;2-t.

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23

Wolford, Johanna K., and Barbora Vozarova de Courten. "Genetic Basis of Type 2 Diabetes Mellitus." Treatments in Endocrinology 3, no. 4 (2004): 257–67. http://dx.doi.org/10.2165/00024677-200403040-00007.

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24

Zozulinska, Dorota, and Bogna Wierusz-Wysocka. "Type 2 diabetes mellitus as inflammatory disease." Diabetes Research and Clinical Practice 74, no. 2 (November 2006): S12—S16. http://dx.doi.org/10.1016/j.diabres.2006.06.007.

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25

Isaia, G. C., P. Ardissone, M. Di Stefano, D. Ferrari, V. Martina, M. Porta, M. Tagliabue, and G. M. Molinatti. "Bone metabolism in type 2 diabetes mellitus." Acta Diabetologica 36, no. 1-2 (July 1, 1999): 35–38. http://dx.doi.org/10.1007/s005920050142.

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26

Asfandiyarova, Nailya S., Olga V. Dashkevich, Natalya V. Doroshina, and Ekaterina I. Suchkova. "Type 2 diabetes mellitus and multiple chronic diseases." Diabetes mellitus 21, no. 6 (February 18, 2019): 455–61. http://dx.doi.org/10.14341/dm9605.

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Background: Recently, there has been an increase in the number of patients with multiple chronic diseases (MCD), particularly due to obesity and ageing. The role of type 2 diabetes mellitus (T2DM) in the development of MCD, however, is still unclear. Aims: This study aimed to determine the incidence of T2DM in the structure of polymorbidity considering sex and age-related characteristics. Materials and methods: Patients with MCD (n = 2,254; 769 men/1,485 women; aged, 1899 years) were examined. The incidence of type 2 diabetes among patients with MCD considering age and sex was determined. Results: Type 2 DM was detected in 407 patients with MCD (18.1%; male:female, 1:2.53). The polymorbidity index in male patients with type 2 diabetes was 1.52.0 times higher than that in male patients without diabetes. The rate of polymorbidity index increase was similar in both groups; however, its high initial value in patients with diabetes at a young age determined the burden of the comorbidity at a later age. In type 2 diabetes, hypertension was the predominant comorbidity at 1859 years of age (p0.05), whereas other cardiovascular diseases and liver and kidney diseases were predominant at 4574 years of age (p0.001) and hemiplegia at 4589 years of age (p0.05). Between 60 and 74 years, oncological diseases were found to be more common in patients without diabetes (p0.001). Obesity, regardless of the presence of diabetes, was associated with a greater disease burden (p0.05). Sex-related difference considering MCD in patients with type 2 DM was only observed for the higher incidence of myocardial infarction (p0.001) and peptic ulcer disease in males (p0.01). Females were more likely to have obesity, liver steatosis at a young age, or osteoarthritis than males in the general group (p0.05); no differences were noted with respect to other diseases. Conclusions: In this study, type 2 diabetes was present in 18.1% of patients with MCD; moreover, a high initial polymorbidity index in patients with T2DM at young age was associated with a higher incidence of chronic diseases later in life than that in patients without diabetes. Based on these results, type 2 diabetes, along with ageing and obesity, can be considered as a risk factor in the development of MCD.
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27

Bogatyrev, Sergey N. "Physical activity and type 2 diabetes mellitus risk: population studies review." Diabetes mellitus 19, no. 6 (December 2, 2016): 486–93. http://dx.doi.org/10.14341/dm8030.

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Physical activity is one of the most important components of a healthy lifestyle. Regular physical activity helps to maintain normal blood glucose levels and reduce the risk of type 2 diabetes mellitus. This review presents population studies investigating physical activity as a factor for type 2 diabetes mellitus risk. A search using the keywords ‘physical activity’, ‘type 2 diabetes mellitus’ and ‘risk’ identified more than 40 relevant original studies and meta-analyses, which are presented in this review. Different types of physical activity have positive protective effects on type 2 diabetes mellitus risk and reduce the risk of death in patients with type 2 diabetes mellitus.
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28

Holst, Jens Juul. "Incretin therapy for diabetes mellitus type 2." Current Opinion in Endocrinology & Diabetes and Obesity 27, no. 1 (February 2020): 2–10. http://dx.doi.org/10.1097/med.0000000000000516.

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29

Angiolillo, Dominick J. "Antiplatelet therapy in type 2 diabetes mellitus." Current Opinion in Endocrinology, Diabetes and Obesity 14, no. 2 (April 2007): 124–31. http://dx.doi.org/10.1097/med.0b013e32807f2ad9.

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30

Jiang, Xinli, Huijie Ma, Yan Wang, and Yan Liu. "Early Life Factors and Type 2 Diabetes Mellitus." Journal of Diabetes Research 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/485082.

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Type 2 diabetes mellitus (T2DM) is a multifactorial disease, and its aetiology involves a complex interplay between genetic, epigenetic, and environmental factors. In recent years, evidences from both human and animal experiments have correlated early life factors with programming diabetes risk in adult life. Fetal and neonatal period is crucial for organ development. Many maternal factors during pregnancy may increase the risk of diabetes of offsprings in later life, which include malnutrition, healthy (hyperglycemia and obesity), behavior (smoking, drinking, and junk food diet), hormone administration, and even stress. In neonates, catch-up growth, lactation, glucocorticoids administration, and stress have all been found to increase the risk of insulin resistance or T2DM. Unfavorable environments (socioeconomic situation and famine) or obesity also has long-term negative effects on children by causing increased susceptibility to T2DM in adults. We also address the potential mechanisms that may underlie the developmental programming of T2DM. Therefore, it might be possible to prevent or delay the risk for T2DM by improving pre- and/or postnatal factors.
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31

Hamdy, Osama, Laurie J. Goodyear, and Edward S. Horton. "DIET AND EXERCISE IN TYPE 2 DIABETES MELLITUS." Endocrinology and Metabolism Clinics of North America 30, no. 4 (December 2001): 883–907. http://dx.doi.org/10.1016/s0889-8529(05)70220-6.

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32

Marcovecchio, M., A. Mohn, and F. Chiarelli. "Type 2 diabetes mellitus in children and adolescents." Journal of Endocrinological Investigation 28, no. 11 (December 2005): 853–63. http://dx.doi.org/10.1007/bf03347581.

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33

Richards, Lisa. "Genetic risk factors in type 2 diabetes mellitus." Nature Reviews Endocrinology 5, no. 8 (August 2009): 414. http://dx.doi.org/10.1038/nrendo.2009.117.

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34

Opriş, Simona, and Gianina-Ioana Constantin. "Dyslipidemia Prevalence, Atherogenicity and Type 2 Diabetes Mellitus." Metabolism 104 (March 2020): 154056. http://dx.doi.org/10.1016/j.metabol.2019.12.002.

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35

Cryer, Philip E. "Severe iatrogenic hypoglycemia in type 2 diabetes mellitus." Nature Clinical Practice Endocrinology & Metabolism 3, no. 1 (January 2007): 4–5. http://dx.doi.org/10.1038/ncpendmet0355.

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36

Karamitri, Angeliki, and Ralf Jockers. "Melatonin in type 2 diabetes mellitus and obesity." Nature Reviews Endocrinology 15, no. 2 (December 7, 2018): 105–25. http://dx.doi.org/10.1038/s41574-018-0130-1.

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37

Regensteiner, Judith G. "Type 2 Diabetes Mellitus and Cardiovascular Exercise Performance." Reviews in Endocrine and Metabolic Disorders 5, no. 3 (August 2004): 269–76. http://dx.doi.org/10.1023/b:remd.0000032416.13070.01.

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38

Pinto, Miguel, Jaime Villena, and Arturo Villena. "Diabetic Ketoacidosis in Peruvian Patients with type 2 Diabetes Mellitus." Endocrine Practice 14, no. 4 (May 2008): 442–46. http://dx.doi.org/10.4158/ep.14.4.442.

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39

Levit, Shmuel, Yury Ivanovich Filippov, and A. S. Gorelyshev. "Type 2 diabetes mellitus: time to change the concept." Diabetes mellitus 16, no. 1 (March 15, 2013): 91–102. http://dx.doi.org/10.14341/2072-0351-3603.

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Diabetes mellitus is a heterogeneous group of diseases that, although unified by a number of characteristics, require a differential thera- peutic approach. Current review discusses key pathogenic features of type 2 diabetes mellitus that determine therapy goals and options in management. We further enunciate and pathogenetically substantiate a new "gravicentric" concept for treatment of type 2 diabetes mellitus that differs in many ways from the common contemporary approach.
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40

Mogensen, Carl Erik. "Diabetic Renal Disease in Patients with Type 2 Diabetes Mellitus." Treatments in Endocrinology 1, no. 1 (2002): 3–11. http://dx.doi.org/10.2165/00024677-200201010-00001.

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41

Starostina, E. G. "Psychological aspects of diet therapy in type 2 diabetes mellitus." Obesity and metabolism 5, no. 2 (June 15, 2008): 7–10. http://dx.doi.org/10.14341/omet200827-10.

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The importance of studying the psychological aspects of nutrition diet is, perhaps, the most commonly used words in the lexicon of Endocrinology and Diabetology and most unpleasant - for patients with diabetes mellitus (DM). Any restrictions on certain aspects of nutrition in patients with diabetes create a negative attitude to the disease, since they require non-established habits and tastes, a significant change in lifestyle, often - constant "struggle" with gusto. And if type 1 diabetes decrease dietary restrictions for patient education - the so-called liberalized diet - allows much to make life easier to patient without compromising compensation diabetes [3, 11], the rules for type 2 diabetes, especially in combination with obesity, are fundamentally based on constraints.
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42

Hayden, Melvin R., and James R. Sowers. "Hypertension in type 2 diabetes mellitus." Insulin 1, no. 1 (January 2006): 22–37. http://dx.doi.org/10.1016/s1557-0843(06)80005-9.

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43

Martin, P., K. K. Hampton, C. Walton, H. Tindall, and J. A. Davies. "Microproteinuria in Type 2 Diabetes Mellitus from Diagnosis." Diabetic Medicine 7, no. 4 (May 1990): 315–18. http://dx.doi.org/10.1111/j.1464-5491.1990.tb01396.x.

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44

Tan, K. C. B. "Reverse cholesterol transport in type 2 diabetes mellitus." Diabetes, Obesity and Metabolism 11, no. 6 (June 2009): 534–43. http://dx.doi.org/10.1111/j.1463-1326.2008.01012.x.

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45

Linfoot, P., C. Bergstrom, and E. Ipp. "Pathophysiology of ketoacidosis in Type 2 diabetes mellitus." Diabetic Medicine 22, no. 10 (October 2005): 1414–19. http://dx.doi.org/10.1111/j.1464-5491.2005.01660.x.

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46

Hardy, K., and S. McNulty. "Prescription charge exemption and Type 2 diabetes mellitus." Diabetic Medicine 25, no. 1 (January 10, 2008): 113–14. http://dx.doi.org/10.1111/j.1464-5491.2007.02343.x.

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47

Schaper, N. C., M. H. Nabuurs-Franssen, and M. S. P. Huijberts. "Peripheral vascular disease and Type 2 diabetes mellitus." Diabetes/Metabolism Research and Reviews 16, S1 (2000): S11—S15. http://dx.doi.org/10.1002/1520-7560(200009/10)16:1+<::aid-dmrr112>3.0.co;2-v.

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48

Marcus, SaraRani, KN Kalaivanam, and Mala Dharmalingam. "Lipid peroxidation in type 2 diabetes mellitus." International Journal of Diabetes in Developing Countries 26, no. 1 (2006): 30. http://dx.doi.org/10.4103/0973-3930.26889.

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49

Panchu, Pallavi. "Auditory acuity in type 2 diabetes mellitus." International Journal of Diabetes in Developing Countries 28, no. 4 (2008): 114. http://dx.doi.org/10.4103/0973-3930.45270.

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50

Campbell, Ian W. "Type 2 diabetes mellitus: ?the silent killer?" Practical Diabetes International 18, no. 6 (2001): 187–91. http://dx.doi.org/10.1002/pdi.230.

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