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Journal articles on the topic 'Endocrine'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 March 2023

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1

Sánchez, P., M. Zanabria, S. Latorre, J. Calvache, A. Coy, and W. Rojas. "Disruptores endocrinos y su camino hacia el desequilibrio metabólico." Revista Colombiana de Endocrinología, Diabetes & Metabolismo 7, no. 1 (April 24, 2020): 38–42. http://dx.doi.org/10.53853/encr.7.1.567.

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El presente artículo de revisión tiene como objetivo presentar, de forma resumida, la evidencia que existe sobre las repercusiones metabólicas a nivel de obesidad y diabetes, que se genera como consecuencia de la exposición a sustancias químicas exógenas, denominadas disruptores endocrinos (DE), a las cuales nos exponemos de forma cotidiana y que afectan nuestra salud y la de nuestra descendencia. Adicionalmente, con la presente revisión hacemos un llamado no solo a la comunidad médica, sino a los sectores involucrados en la producción, distribución y reglamentación del uso de estas sustancias, pues cada vez hay más evidencia de los efectos nocivos que pueden generar y debemos evitar su uso. Los datos se obtuvieron de estudios clínicos aleatorizados y de una revisión en idioma español e inglés de los últimos 15 años, que incluyó los términos DeCS: disruptores endocrinos, con alternativa DeCS: sustancias disruptoras endocrinas y efecto disruptor endocrino, así como términos MeSH: endocrine disruptors y alternativas MeSH: disruptors, endocrine; endocrine disrupting chemicals; chemicals, endocrine disrupting; endocrine disruptor effect; disruptor effect, endocrine; effect, endocrine disruptor; endocrine disruptor effects; disruptor effects, endocrine; effects, endocrine disruptor.

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2

Batrinos, Menelaos. "The aging of the endocrine hypothalamus and its dependent endocrine glands." HORMONES 11, no. 3 (July 15, 2012): 241–53. http://dx.doi.org/10.14310/horm.2002.1354.

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3

Escoresca Suárez, I., and A. Utrera González. "Endocrine surgery." Cirugía Andaluza 31, no. 3 (August 7, 2020): 216. http://dx.doi.org/10.37351/2020313.1.

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4

Imura, Hiroo, and Jun-ichi Fukata. "Endocrine–paracine interaction in communication between the immune and endocrine systems. Activation of the hypothalamic-pituitary-adrenal axis in inflammation." European Journal of Endocrinology 130, no. 1 (January 1994): 32–37. http://dx.doi.org/10.1530/eje.0.1300032.

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Imura H, Fukata J. Endocrine–paracrine interaction in communication between the immune and endocrine systems. Activation of the hypothalamic-pituitary-adrenal axis in inflammation. Eur J Endocrinol 1994;130:32–7. ISSN 0804–4643 There are bidirectional communications between the immune and endocrine systems. Cytokines produced in inflammatory foci cause changes in the endocrine system, including activation of the hypothalamic-pituitary-adrenal (HPA) axis. Hormones produced in the endocrine system, especially glucocorticoids, affect the immune system to modulate its function. This is an important endocrine system for the defence mechanism. In addition, bacterial lipopolysaccharide produces cytokines in the brain and endocrine organs which are considered to act through the paracrine mechanism to regulate the HPA axis. Endocrine–paracrine interaction is important for the defence mechanism of the organism. Hiroo Imura, Kyoto University School of Medicine, Yoshida Honmachi Sakyo-ku, Kyoto 606-01, Japan

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5

Bosman, Fred T. "Endocrine cells in non-endocrine tumours." Journal of Pathology 159, no. 3 (November 1989): 181–82. http://dx.doi.org/10.1002/path.1711590302.

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6

Wright, Nicholas A. "Endocrine cells in non-endocrine tumours." Journal of Pathology 161, no. 1 (May 1990): 85–87. http://dx.doi.org/10.1002/path.1711610114.

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7

Borba, Victoria Zeghbi Cochenski, Tatiana Lemos Costa, Carolina Aguiar Moreira, and Cesar Luiz Boguszewski. "MECHANISMS OF ENDOCRINE DISEASE: Sarcopenia in endocrine and non-endocrine disorders." European Journal of Endocrinology 180, no. 5 (May 2019): R185—R199. http://dx.doi.org/10.1530/eje-18-0937.

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Objective This paper reviews the main mechanisms, diagnostic criteria, treatment options and available data on sarcopenia in endocrine and non-endocrine disorders. The literature notes the presence of sarcopenia as a comorbid condition or a complication of another clinical situation and not a disease that only affects elderly patients. Method We performed a literature review, focusing on the following: mechanisms related to sarcopenia in elderly patients, and sarcopenia as it presents in the context of chronic and endocrine diseases; diagnostic tools and methods; aspects of sarcopenia and treatment options specific to chronic diseases and endocrine disorders respectively. Results Sarcopenia in chronic and endocrine disorders shares many mechanisms with sarcopenia affecting elderly patients, but certain diseases can have a predominant aspect that leads to sarcopenia. The prevalence of sarcopenia varies, depending on different diagnostic criteria, from around 12 to 60% in chronic illnesses and 15 to 90% in endocrine disorders. The interplay between sarcopenia, chronic diseases and elderly patients requires further study, to clarify the impact of each, in terms of prognosis and mortality. Conclusion Awareness of the presentation of sarcopenia in the context of other diseases and ages (and not just the elderly) is fundamental to ensure that preventive measures can be deployed.

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8

Heath, D. "Pulmonary Endocrine Pathology, Endocrine Cells and Endocrine Tumours of the Lung." Journal of Clinical Pathology 46, no. 7 (July 1, 1993): 687. http://dx.doi.org/10.1136/jcp.46.7.687-b.

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9

Akopova, R. A., and T. V. Kokoreva. "ENDOCRINE ASPECTS OF MALE INFERTILITY." Bulletin "Biomedicine and sociology" 3, no. 4 (December 30, 2018): 17–19. http://dx.doi.org/10.26787/nydha-2618-8783-2018-3-4-17-19.

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10

Gore, Andrea. "Neuroendocrine targets of endocrine disruptors." HORMONES 9, no. 1 (January 15, 2010): 16–27. http://dx.doi.org/10.14310/horm.2002.1249.

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11

Ghemigian, A., I. Popescu, E. Petrova, and A. Buruiană. "Endocrine secondary hypertension." Romanian Medical Journal 62, no. 3 (September 30, 2015): 222–28. http://dx.doi.org/10.37897/rmj.2015.3.2.

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The need to evaluate patients for secondary hypertension is common in clinical practice. Suspicion of endocrine hypertension occurs when the disease starts suddenly at young age, in cases with loss of blood pressure control in a patient with previously well-controlled blood pressure or labile blood pressure cases. Careful medical history and physical examination are very important to rule out other factors responsible for these patterns of hypertension (certain medications, alcohol, lack of compliance to treatment or dietary salt restriction, panic attacks etc). Pheochromocytoma and Cushing syndrome – the major endocrine diseases associated with secondary hypertension – are rare in clinical practice. Instead, primary hyperaldosteronism is becoming more frequently identified. It can go unnoticed because of nonspecific clinical presentation and of the fact that hypokalaemia, classically described, is actually rare in practice.

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12

GHEMIGIAN, A., I. POPESCU, E. PETROVA, and A. BURUIANĂ. "Diabetes secondary to endocrine diseases." Romanian Journal of Medical Practice 10, no. 3 (September 30, 2015): 264–68. http://dx.doi.org/10.37897/rjmp.2015.3.9.

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Diabetes mellitus may occur as a complication of endocrine disorders and it’s name is, according to current classification (American Diabetes Association): „other specific types of diabetes“. This particular endocrine diseases associate sustained and excessive hormonal secretion, which interferes insulin secretion or action. Over time, glucose intolerance and then diabetes occur. Generally, there are middle forms with no ketosis because of the persistence of endogenous insulin secretion. More then that, hyperglycemia is usually reversible with the treatment of the underlying endocrinopathy. Main endocrine diseases that cause diabetes are reviewed.

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13

Broglio, Fabio, Cristina Gottero, Emanuela Arvat, and Ezio Ghigo. "Endocrine and Non-Endocrine Actions of Ghrelin." Hormone Research in Paediatrics 59, no. 3 (2003): 109–17. http://dx.doi.org/10.1159/000069065.

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14

Casals-Casas, Cristina, and Béatrice Desvergne. "Endocrine Disruptors: From Endocrine to Metabolic Disruption." Annual Review of Physiology 73, no. 1 (March 17, 2011): 135–62. http://dx.doi.org/10.1146/annurev-physiol-012110-142200.

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15

Osamura, R. Yoshiyuki, Toshiki Iwasaka, and Shinobu Umemura. "Endocrine System and Endocrine Disrupting Chemicals(EDCs)." Journal of Toxicologic Pathology 14, no. 1 (2001): 59–64. http://dx.doi.org/10.1293/tox.14.59.

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16

Fabri, Peter J. "Editorial: The Endocrine Surgeon and Endocrine Neoplasms." Cancer Control 4, no. 1 (January 1997): 9. http://dx.doi.org/10.1177/107327489700400113.

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17

Orlando, C., and S. Gelmini. "Telomerase in endocrine and endocrine-dependent tumors." Journal of Steroid Biochemistry and Molecular Biology 78, no. 3 (September 2001): 201–14. http://dx.doi.org/10.1016/s0960-0760(01)00101-7.

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18

Coyne, Mary D. "The pineal: Endocrine and non-endocrine function." Trends in Endocrinology & Metabolism 1, no. 6 (July 1990): 324–25. http://dx.doi.org/10.1016/1043-2760(90)90074-d.

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19

López-Cantarero García-Cervantes, M. "Training in Endocrine Surgery." Cirugía Andaluza 33, no. 3 (August 8, 2022): 365–468. http://dx.doi.org/10.37351/2022333.10.

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20

Petros, Beyene. "Neuro Endocrine Physiology: Pineal Gland Development." Endocrinology and Disorders 2, no. 3 (April 5, 2018): 01–02. http://dx.doi.org/10.31579/2640-1045/023.

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21

Oxley, Dwight K. "Endocrine Diseases." Archives of Pathology & Laboratory Medicine 127, no. 3 (March 1, 2003): 381–82. http://dx.doi.org/10.5858/2003-127-0381-ed.

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22

West, Robert Lee. "Endocrine Pathology." Archives of Pathology & Laboratory Medicine 127, no. 7 (July 1, 2003): 897–98. http://dx.doi.org/10.5858/2003-127-897b-ep.

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23

Takeda, Ryoyu. "Endocrine Hypertension." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 30, no. 3 (1993): 182–87. http://dx.doi.org/10.3143/geriatrics.30.182.

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24

Osborn, Irene. "Endocrine Abnormalities." Anesthesiology Clinics of North America 5, no. 3 (September 1987): 521–29. http://dx.doi.org/10.1016/s0889-8537(21)00331-x.

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25

Komatsu, Kazuhiro. "Endocrine Disruptors." Journal for the Integrated Study of Dietary Habits 10, no. 1 (1999): 2–6. http://dx.doi.org/10.2740/jisdh.10.2.

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26

Reasner, Charles A., and William L. Isley. "Endocrine emergencies." Postgraduate Medicine 101, no. 3 (March 1997): 231–42. http://dx.doi.org/10.3810/pgm.1997.03.185.

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27

Wallace, Kathleen. "Endocrine problems." Postgraduate Medicine 104, no. 1 (July 1998): 41. http://dx.doi.org/10.3810/pgm.1998.07.528.

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28

Fackelmayer, Oliver J., James X. Wu, and Michael W. Yeh. "Endocrine Surgery." Surgical Clinics of North America 101, no. 5 (October 2021): 767–84. http://dx.doi.org/10.1016/j.suc.2021.05.019.

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29

Newell-Price, John, and Anthony P. Weetman. "Endocrine emergencies." Acute Medicine Journal 1, no. 2 (April 1, 2002): 4–8. http://dx.doi.org/10.52964/amja.0006.

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Endocrine emergencies are uncommon and require a high index of suspicion if they are to be managed appropriately. This is especially pertinent to physicians accepting emergency admissions to hospital. Treatment needs to be started prior to diagnostic confirmation. We outline the management of the following conditions that may present acutely: Thyroid emergencies – Myxoedema coma – Thyroid Storm Pituitary apoplexy Phaeochromocytoma crisis Acute adrenocortical insufficiency.

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30

Sherlock, Mark, and Tara McDonnell. "Endocrine hypertension." Medicine 49, no. 8 (August 2021): 502–6. http://dx.doi.org/10.1016/j.mpmed.2021.05.011.

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31

Johanson, Norman A. "Endocrine Arthropathies." Clinics in Rheumatic Diseases 11, no. 2 (August 1985): 297–323. http://dx.doi.org/10.1016/s0307-742x(21)00543-9.

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32

Chowdhury, Subhankar, and Deep Dutta. "Endocrine Labomas." Indian Journal of Endocrinology and Metabolism 16, no. 8 (2012): 275. http://dx.doi.org/10.4103/2230-8210.104059.

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33

Cook, B. "Endocrine asymmetry." Journal of Endocrinology 113, no. 3 (June 1987): 331–32. http://dx.doi.org/10.1677/joe.0.1130331.

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34

KS, Shyam. "Endocrine emergencies." Journal of Medical and Scientific Research 1, no. 2 (June 1, 2013): 88–94. http://dx.doi.org/10.17727/jmsr.2013/1-016.

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35

Kishi, R., and H. Arito. "Endocrine disruptors." SANGYO EISEIGAKU ZASSHI 41, Special (1999): 96–97. http://dx.doi.org/10.1539/sangyoeisei.kj00001990879.

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36

Kang, Chang Gun, Seung Hwan Lee, and Eui Kyung Kim. "Endocrine Disruptors." Journal of the Korean Medical Association 50, no. 4 (2007): 359. http://dx.doi.org/10.5124/jkma.2007.50.4.359.

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37

Sistrunk, John W. "Endocrine Physiology." Mayo Clinic Proceedings 75, no. 11 (November 2000): 1226. http://dx.doi.org/10.4065/75.11.1226-a.

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38

Kleer, Celina G. "Endocrine Diseases." Mayo Clinic Proceedings 78, no. 2 (February 2003): 255. http://dx.doi.org/10.4065/78.2.254.

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39

Kalra, Sanjay. "Endocrine covfefe." Indian Journal of Endocrinology and Metabolism 21, no. 5 (2017): 787. http://dx.doi.org/10.4103/ijem.ijem_235_17.

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40

Galoiu, S. "Endocrine TUMORS." Acta Endocrinologica (Bucharest) 9, no. 1 (2013): 145–48. http://dx.doi.org/10.4183/aeb.2013.145.

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41

Perrais, David, Justin Taraska, and Wolfhard Almers. "Endocrine granules." Physiology News, Spring 2005 (April 1, 2005): 33–35. http://dx.doi.org/10.36866/pn.58.33.

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42

Macdonald, John S. "Endocrine tumors." Current Opinion in ONCOLOGY 2, no. 1 (February 1990): 71–72. http://dx.doi.org/10.1097/00001622-199002000-00011.

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43

&NA;. "Endocrine tumors." Current Opinion in ONCOLOGY 2, no. 1 (February 1990): 199–208. http://dx.doi.org/10.1097/00001622-199002000-00029.

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44

Macdonald, John S. "Endocrine tumors." Current Opinion in Oncology 3, no. 1 (February 1991): 101–2. http://dx.doi.org/10.1097/00001622-199102000-00014.

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45

&NA;. "Endocrine tumors." Current Opinion in Oncology 3, no. 1 (February 1991): 220–28. http://dx.doi.org/10.1097/00001622-199102000-00022.

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46

Macdonald, John S. "Endocrine tumors." Current Opinion in Oncology 4, no. 1 (February 1992): 87–88. http://dx.doi.org/10.1097/00001622-199202000-00011.

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47

&NA;. "Endocrine tumors." Current Opinion in Oncology 4, no. 1 (February 1992): 191–96. http://dx.doi.org/10.1097/00001622-199202000-00023.

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48

Macdonald, John S. "Endocrine tumors." Current Opinion in Oncology 6, no. 1 (January 1994): 51–52. http://dx.doi.org/10.1097/00001622-199401000-00007.

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49

Nobari, Hadi, Elena Mainer-Pardos, José Carmelo Adsuar, Juan Manuel Franco-García, Jorge Rojo-Ramos, Marco Antonio Cossio-Bolaños, Luis Urzua Alul, and Jorge Pérez-Gómez. "Association Between Endocrine Markers, Accumulated Workload, and Fitness Parameters During a Season in Elite Young Soccer Players." Frontiers in Psychology 12 (August 31, 2021). http://dx.doi.org/10.3389/fpsyg.2021.702454.

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The purpose of this study was to analyze differences between endocrine markers in soccer players, based on playing positions, and correlations between endocrine markers (testosterone, cortisol, growth hormone, and insulin-like growth factor-1), with accumulated workload training and fitness parameters [maximal oxygen uptake (VO2max), countermovement jump (CMJ), and isometric maximal strength (1-RM) of the knee for hamstring (ISH) and quadriceps (ISQ) muscles] during early-, mid-, and end-seasons. Twenty-four elite soccer players under 17 participated in this study. The results showed that there was no difference between levels of the endocrine markers among the different positions of the players. Significant correlations were observed between endocrines parameters and fitness performance (ISQ, ISH, VO2max, and CMJ). Regression analysis showed that 1-RM and VO2max were the best predictors of endocrine markers. These findings demonstrated that the activity profiles of youth soccer players were not influenced by endocrine markers. Also, it may be assumed that endocrines levels can be used to better explain the physical capacities of this population. Finally, endocrines markers may help to predict changes in 1-RM and VO2max.

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50

"Endocrine." Laboratory Investigation 87, S1 (February 2007): 100–105. http://dx.doi.org/10.1038/sj.labinvest.3700571.

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