Relevant bibliographies by topics / Endocrine

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Endocrine'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 March 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Endocrine.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Endocrine"

1

Sánchez, P., M. Zanabria, S. Latorre, J. Calvache, A. Coy, and W. Rojas. "Disruptores endocrinos y su camino hacia el desequilibrio metabólico." Revista Colombiana de Endocrinología, Diabetes & Metabolismo 7, no. 1 (April 24, 2020): 38–42. http://dx.doi.org/10.53853/encr.7.1.567.

Full text
Abstract:
El presente artículo de revisión tiene como objetivo presentar, de forma resumida, la evidencia que existe sobre las repercusiones metabólicas a nivel de obesidad y diabetes, que se genera como consecuencia de la exposición a sustancias químicas exógenas, denominadas disruptores endocrinos (DE), a las cuales nos exponemos de forma cotidiana y que afectan nuestra salud y la de nuestra descendencia. Adicionalmente, con la presente revisión hacemos un llamado no solo a la comunidad médica, sino a los sectores involucrados en la producción, distribución y reglamentación del uso de estas sustancias, pues cada vez hay más evidencia de los efectos nocivos que pueden generar y debemos evitar su uso. Los datos se obtuvieron de estudios clínicos aleatorizados y de una revisión en idioma español e inglés de los últimos 15 años, que incluyó los términos DeCS: disruptores endocrinos, con alternativa DeCS: sustancias disruptoras endocrinas y efecto disruptor endocrino, así como términos MeSH: endocrine disruptors y alternativas MeSH: disruptors, endocrine; endocrine disrupting chemicals; chemicals, endocrine disrupting; endocrine disruptor effect; disruptor effect, endocrine; effect, endocrine disruptor; endocrine disruptor effects; disruptor effects, endocrine; effects, endocrine disruptor.
APA, Harvard, Vancouver, ISO, and other styles
2

Batrinos, Menelaos. "The aging of the endocrine hypothalamus and its dependent endocrine glands." HORMONES 11, no. 3 (July 15, 2012): 241–53. http://dx.doi.org/10.14310/horm.2002.1354.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Escoresca Suárez, I., and A. Utrera González. "Endocrine surgery." Cirugía Andaluza 31, no. 3 (August 7, 2020): 216. http://dx.doi.org/10.37351/2020313.1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Imura, Hiroo, and Jun-ichi Fukata. "Endocrine–paracine interaction in communication between the immune and endocrine systems. Activation of the hypothalamic-pituitary-adrenal axis in inflammation." European Journal of Endocrinology 130, no. 1 (January 1994): 32–37. http://dx.doi.org/10.1530/eje.0.1300032.

Full text
Abstract:
Imura H, Fukata J. Endocrine–paracrine interaction in communication between the immune and endocrine systems. Activation of the hypothalamic-pituitary-adrenal axis in inflammation. Eur J Endocrinol 1994;130:32–7. ISSN 0804–4643 There are bidirectional communications between the immune and endocrine systems. Cytokines produced in inflammatory foci cause changes in the endocrine system, including activation of the hypothalamic-pituitary-adrenal (HPA) axis. Hormones produced in the endocrine system, especially glucocorticoids, affect the immune system to modulate its function. This is an important endocrine system for the defence mechanism. In addition, bacterial lipopolysaccharide produces cytokines in the brain and endocrine organs which are considered to act through the paracrine mechanism to regulate the HPA axis. Endocrine–paracrine interaction is important for the defence mechanism of the organism. Hiroo Imura, Kyoto University School of Medicine, Yoshida Honmachi Sakyo-ku, Kyoto 606-01, Japan
APA, Harvard, Vancouver, ISO, and other styles
5

Bosman, Fred T. "Endocrine cells in non-endocrine tumours." Journal of Pathology 159, no. 3 (November 1989): 181–82. http://dx.doi.org/10.1002/path.1711590302.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Wright, Nicholas A. "Endocrine cells in non-endocrine tumours." Journal of Pathology 161, no. 1 (May 1990): 85–87. http://dx.doi.org/10.1002/path.1711610114.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Borba, Victoria Zeghbi Cochenski, Tatiana Lemos Costa, Carolina Aguiar Moreira, and Cesar Luiz Boguszewski. "MECHANISMS OF ENDOCRINE DISEASE: Sarcopenia in endocrine and non-endocrine disorders." European Journal of Endocrinology 180, no. 5 (May 2019): R185—R199. http://dx.doi.org/10.1530/eje-18-0937.

Full text
Abstract:
Objective This paper reviews the main mechanisms, diagnostic criteria, treatment options and available data on sarcopenia in endocrine and non-endocrine disorders. The literature notes the presence of sarcopenia as a comorbid condition or a complication of another clinical situation and not a disease that only affects elderly patients. Method We performed a literature review, focusing on the following: mechanisms related to sarcopenia in elderly patients, and sarcopenia as it presents in the context of chronic and endocrine diseases; diagnostic tools and methods; aspects of sarcopenia and treatment options specific to chronic diseases and endocrine disorders respectively. Results Sarcopenia in chronic and endocrine disorders shares many mechanisms with sarcopenia affecting elderly patients, but certain diseases can have a predominant aspect that leads to sarcopenia. The prevalence of sarcopenia varies, depending on different diagnostic criteria, from around 12 to 60% in chronic illnesses and 15 to 90% in endocrine disorders. The interplay between sarcopenia, chronic diseases and elderly patients requires further study, to clarify the impact of each, in terms of prognosis and mortality. Conclusion Awareness of the presentation of sarcopenia in the context of other diseases and ages (and not just the elderly) is fundamental to ensure that preventive measures can be deployed.
APA, Harvard, Vancouver, ISO, and other styles
8

Heath, D. "Pulmonary Endocrine Pathology, Endocrine Cells and Endocrine Tumours of the Lung." Journal of Clinical Pathology 46, no. 7 (July 1, 1993): 687. http://dx.doi.org/10.1136/jcp.46.7.687-b.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Akopova, R. A., and T. V. Kokoreva. "ENDOCRINE ASPECTS OF MALE INFERTILITY." Bulletin "Biomedicine and sociology" 3, no. 4 (December 30, 2018): 17–19. http://dx.doi.org/10.26787/nydha-2618-8783-2018-3-4-17-19.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Gore, Andrea. "Neuroendocrine targets of endocrine disruptors." HORMONES 9, no. 1 (January 15, 2010): 16–27. http://dx.doi.org/10.14310/horm.2002.1249.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Endocrine"

1

Monges-Ranchin, Geneviève. "Les cellules endocrines de l'estomac et leur pathologie." Aix-Marseille 2, 1987. http://www.theses.fr/1987AIX21917.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Sharma, Vinay. "Endocrine and non-endocrine factors affecting the outcome of assisted conception." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244126.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Delgado, Verdugo Alberto. "Genetic Aspects of Endocrine Tumorigenesis : A Hunt for the Endocrine Neoplasia Gene." Doctoral thesis, Uppsala universitet, Endokrinkirurgi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-224111.

Full text
Abstract:
Endocrine tumors arise from endocrine glands. Most endocrine tumors are benign but malignant variants exist. Several endocrine neoplasms display loss of parts of chromosome 11 or 18, produce hormones and responds poorly to conventional chemotherapeutics. The multiple endocrine neoplasia syndromes are mainly confined to endocrine tumors. This opens the question if there exists a single or several endocrine tumor genes. The aim of the study was to describe genetic derangements in endocrine tumors. Paper I: Investigation of mutational status of SDHAF2 in parathyroid tumors. SDHAF2 is located in the proximity of 11q13, a region that frequently displays loss in parathyroid tumors. We established that mutations in SDHAF2 are infrequent in parathyroid tumors. Paper II: Study of SDHAF2 gene expression in a cohort of benign pheochromocytomas (PCC) (n=40) and malignant PCC (n=10). We discovered a subset of  benign PCC (28/40) and all malignant PCC (10/10) with significantly lower SDHAF2 expression. Benign PCC with low SDHAF2 expression and malignant tumors consistently expressing low levels of SDHAF2 were methylated in the promoter region. SDHAF2 expression was restored in vitro after treatment with 5- aza-2-deoxycytidine. Paper III: HumanMethylation27 array (Illumina) covering 27578 CpG sites spanning over 14495 genes were analyzed in a discovery cohort of 10 primary small neuroendocrine tumors (SI-NETs) with matched metastases. 2697 genes showed different methylation pattern between the primary tumor and its metastasis. We identified several hypermethylated genes in key regions. Unsupervised clustering of the tumors identified three distinct clusters, one with a highly malignant behavior. Paper IV: Loss of chromosome 18 is the most frequent genetic aberration in SI-NETs. DNA from SI-NETs were subjected to whole exome capture sequencing and high resolution SNP array. Genomic profiling revealed loss of chromosome 18 in 5 out of 7 SI-NETs. No tumor-specific somatic mutation on chromosome 18 was identified which suggests involvement of other mechanisms than point mutations in SI-NET tumorigenesis. Paper V: The cost for diagnostic genetic screening of common susceptibility genes in PCC is expensive and labor intensive. Three PCC from three patients with no known family history were chosen for exome capture sequencing. We identified three variants in known candidate genes. We suggest that exome-capture sequencing is a quick and cost-effective tool.
APA, Harvard, Vancouver, ISO, and other styles
4

Симоненко, Наталія Олександрівна, Наталия Александровна Симоненко, Nataliia Oleksandrivna Symonenko, and N. Vasko. "Endocrine system and disorders." Thesis, Вид-во СумДУ, 2009. http://essuir.sumdu.edu.ua/handle/123456789/16782.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Kohler, Romain. "Collection de cas d'auto-enseignement en imagerie des glandes endocrines (thyroïde, parathyroïdes, surrénales, pancréas endocrine) /." Genève : [s.n.], 2006. http://www.unige.ch/cyberdocuments/theses2006/KohlerR/these.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Martínez, Rodríguez María Ángeles. "Endocrine disruption assessment: exposure, biomonitoring and potential activity of widespread endocrine disruptors during pregnancy and early stages of life." Doctoral thesis, Universitat Rovira i Virgili, 2020. http://hdl.handle.net/10803/669420.

Full text
Abstract:
Els disruptors endocrins (DEs) són substàncies o mescles exògenes que alteren les funcions de sistema endocrí i, en conseqüència, causen efectes adversos per a la salut en un organisme intacte, o la seva progènie, o (sub) poblacions. Entre aquests DEs, hi ha un grup que es caracteritza per estar àmpliament estès i present en envasos i materials en contacte amb aliments. L'envasament protegeix de les alteracions, però es poden donar migracions de les substàncies químiques als aliments. Tenint en compte això, s'han seleccionat els següents compostos químics: bisfenols (BPA, BPS i BPF); plastificants ftalats i no ftalats; substàncies perfluorooalquiladas (PFOS i PFOA) i tetrabromobisfenol A (TBBPA). La hipòtesi general d'aquesta tesi és que hi ha exposició durant les etapes primerenques de la vida. Aquesta exposició pot causar efectes adversos. Per tant, l'objectiu general d'aquesta tesi és avaluar els nivells d'exposició a diferents DEs en una població de dones embarassades (EXHES) a Tarragona, Espanya. A més a més, estudiar com aquests químics poden afectar el desenvolupament cel·lular. Per abordar-lo, es va estimar l'exposició prenatal als DEs mitjançant el model integrat de dosimetria externa i interna (PBPK). Seguit per la biomonitorització d'aquests químics en diferents matrius (orina, sang, llet materna), i de la reconstrucció i simulació de l'exposició. finalment, es van realitzar estudis in vitro per avaluar l'activitat potencial endocrina de DE àmpliament distribuïts. Els resultats d'aquesta tesi confirmen que l'exposició a DEs pot observar-se en poblacions adultes i en el període prenatal. Aquesta exposició pot causar efectes adversos. Per aquesta raó, han d'establir-se alternatives segures als DEs. Seguit d’unes regulacions concises respecte a l'exposició de la població més vulnerable.
Los disruptores endocrinos (DEs) son sustancias o mezclas exógenas que alteran las funciones del sistema endocrino y, en consecuencia, causan efectos adversos para la salud en un organismo intacto, o su progenie, o (sub)poblaciones. Entre estos DEs, hay un grupo que se caracteriza por estar ampliamente extendido y presentes en envases y materiales en contacto con alimentos. El envasado protege al producto de las alteraciones, pero se pueden dar migraciones de las substancias químicas a los alimentos. En base a esto, se han seleccionado los siguientes compuestos químicos: bisfenoles (BPA, BPS y BPF); plastificantes ftalatos y no ftalatos; sustancias perfluorooalquiladas (PFOS y PFOA) y tetrabromobisfenol A (TBBPA). La hipótesis general de esta tesis es que existe exposición a DE en etapas tempranas de la vida. Esta exposición puede causar efectos adversos. Por lo tanto, el objetivo general de esta tesis es evaluar los niveles de exposición a diferentes DE en una población de mujeres embarazadas (EXHES) en Tarragona, España, y a su vez, estudiar cómo estos químicos pueden afectar al desarrollo celular. Para abordarlo, se estimó la exposición prenatal a los DEs mediante el modelo integrado de dosimetría externa e interna (PBPK). Seguido por el biomonitoreo de estos químicos en diferentes matrices (orina, sangre, leche materna), y de la reconstrucción y simulación de la exposición. finalmente, se realizaron estudios in vitro para evaluar la actividad potencial endocrina de DE ampliamente distribuidos. Los resultados de esta tesis confirman que la exposición a DEs puede observarse en poblaciones adultas y en el periodo prenatal. Esta exposición puede causar efectos adversos. Por esa razón, se necesitan alternativas seguras a estos DE. Deben de establecerse unas regulaciones concisas respecto a la exposición de la población más vulnerable.
Endocrine disruptors (EDs) are exogenous substances or mixtures that alters function(s) of the endocrine system and consequently causes adverse health effects in an intact organism, or its progeny, or (sub) populations. Among these EDs, there is a group that takes an important place due to their widespread distribution and their presence in food-packaging or food contact materials. Packaging protects food from alterations. However, the packaging could be a source of contamination as migration can occur from these materials with the transfer of chemicals into the food. According to that, some bisphenols (BPA, BPS and BPF); phthalates and non-phthalate plasticizers; perfluorooalkilated substances (PFOS and PFOA) and tetrabromobisphenol A (TBBPA) were selected in this thesis. The general hypothesis of this thesis is that there is EDs exposure during prenatal and early stages of life and this exposure can cause adverse outcomes. Therefore, the general objective of this thesis is to assess the exposure levels to different EDs from a pregnant women EXHES cohort in Tarragona, Spain and to study how these chemicals can affect normal cellular development. To address that, prenatal exposure to EDs was estimated by integrated external and internal dosimetry modelling, physiologically based pharmacokinetic (PBPK) model. Followed by biomonitoring of these chemicals in different matrices (urine, blood, breast milk), reconstructed exposure and reconstructed-PBPK simulations and finally, in vitro studies to assess the endocrine potential activity for different widespread EDs were performed. Results from this thesis confirm that exposure to EDs can be observed in adult populations and in the early stages of life. This exposure can cause adverse effects. For that reason, safe alternatives to these EDs are needed. Regulations regarding the most vulnerable exposure population should be established in the endocrine disruption context.
APA, Harvard, Vancouver, ISO, and other styles
7

Bruïne, Adriaan Pieter de. "Endocrine differentiation in colorectal cancer." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1993. http://arno.unimaas.nl/show.cgi?fid=6579.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Bensing, Sophie. "Pituitary autoantibodies in endocrine disorders /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-478-3/.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Olsson, Tommy. "Endocrine studies in stroke patients." Doctoral thesis, Umeå universitet, Medicin, 1989. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-101772.

Full text
Abstract:
There are a number of links between the endocrine system and the nervous system. In this study, the impact of ischemic stroke on the endocrine system was investigated. Elderly volunteers were studied because data regarding the influence of advanced age on endocrine parameters were lacking. Only small differences in pituitary-thyroid and pituitary-adrenal hormone axes were found between two groups of elderly patients, 60 and 80 years of age. The 80-year-old age group had a lower thyrotropin response to thyrotropin releasing hormone (TRH) and a decline in dopamine excretion. Patients with acute ischemic stroke showed a pronounced hypercortisolism studied by the dexamethasone test and urine free cortisol measurements. In multiple regression analyses, postdexamethasone cortisol levels were positively correlated to proximity of the lesion to the frontal pole of the brain and disorientation. Urine cortisol levels were predicted by limb paresis, disorientation and body temperature. High cortisol excretion was associated with a worse functional outcome. Norepinephrine excretion was correlated to urine cortisol levels and to motor impairment. Patients with acute stroke had elevated free thyroxin indices. A paradoxical growth hormone response to TRH was found in the majority of stroke patients. In a multiple regression model disorientation was negatively correlated to thyrotropin response after TRH and positively correlated to prolactin response. Growth hormone response to TRH was associated with extensive paresis. In a cohort study diabetic and non-diabetic patients were prospectively studied after an initial stroke. Diabetes mellitus adversely influenced survival, the risk for a recurrent stroke and myocardial infarction.

S. 1-66: sammanfattning, s. 69-190: 6 uppsatser


digitalisering@umu
APA, Harvard, Vancouver, ISO, and other styles
10

Hidayat, Egi. "On identification of endocrine systems." Licentiate thesis, Uppsala universitet, Avdelningen för systemteknik, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-227630.

Full text
Abstract:
System identification finds nowadays application in various areas of biomedical research as a tool of empiric mathematical modeling and model individualization. Hormone regulation is a classical example of biological feedback where control theories, in general, and system identification, in particular, are indispensable in unraveling the regulation mechanisms and explicating the complex dynamical phenomena arising in endocrine systems. The main function of endocrine feedback regulation is to maintain the hormone levels within a particular physiological range as well as to sustain an appropriate hormone secretion pattern. Therefore, a natural operating mode of a closed-loop endocrine system is a stable periodic cycle. This property significantly reduces the repertoire of readily available identification techniques, not least due to the fact that the regulation (input) signal is immeasurable in many practical cases. There are two approaches to blind identification of hormone dynamics presented in this thesis. The first one is based on constrained nonlinear least-squares method. Weighting functions play an important role in satisfying the biological conditions on the identified model. The second approach is derived from a novel time-delay system identification method in Laguerre domain. In the latter, the time delay appears due to a specific input signal model and is estimated along with the finite-dimensional dynamics of hormone kinetics.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Endocrine"

1

D, Colby Howard, and Goodman Robert L, eds. Clinical endocrine physiology. Philadelphia: Saunders, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Gosney, John R. Pulmonary endocrine pathology: Endocrine cells and endocrine tumors of the lung. Oxford: Butterworth-Heinemann, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

1933-, Thomas J. A., Korach Kenneth S, and McLachlan John A, eds. Endocrine toxicology. New York: Raven Press, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Endocrine physiology. St. Louis, Mo: Mosby, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Lloyd, Ricardo V. Endocrine Pathology. New Jersey: Humana Press, 2003. http://dx.doi.org/10.1385/1592594034.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Loriaux, Lynn, and Chaim Vanek, eds. Endocrine Emergencies. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-67455-7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Mishra, Anand Kumar, Amit Agarwal, Rajeev Parameswaran, and Kul Ranjan Singh. Endocrine Surgery. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429197338.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Endocrine physiology. 3rd ed. New York: McGraw-Hill Medical, 2010.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Hubbard, Johnathan, William B. Inabnet, and Chung-Yau Lo, eds. Endocrine Surgery. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84628-881-4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Bland, Kirby I., Michael G. Sarr, Markus W. Büchler, Attila Csendes, Oliver James Garden, and John Wong, eds. Endocrine Surgery. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-447-0.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Endocrine"

1

Merrick, Malcolm V. "Endocrine." In Essentials of Nuclear Medicine, 149–75. London: Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-0907-5_6.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Suskind, David L., and Polly Lenssen. "Endocrine." In Pediatric Nutrition Handbook, 61–64. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118785034.ch3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Holliman, John H., Daniel L. Feeback, and Nancy K. Hall. "Endocrine." In Oklahoma Notes, 142–68. New York, NY: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-0322-0_12.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Jacocks, M. Alex. "Endocrine." In Oklahoma Notes, 136–42. New York, NY: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4684-0454-8_26.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Jarolim, Dala R. "Endocrine." In Oklahoma Notes, 218–33. New York, NY: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4684-0458-6_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Jacocks, M. Alex. "Endocrine." In Oklahoma Notes, 151–58. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2372-6_26.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

El-Radhi, A. Sahib. "Endocrine." In Paediatric Symptom and Sign Sorter, 163–71. Second edition. | Boca Raton, FL : CRC Press, Taylor & Francis Group, [2019] | Series: Pediatric diagnosis and management | Preceded by Paediatric symptom sorter / A. Sahib El-Radhi ; foreword by James Carroll. 2011.: CRC Press, 2019. http://dx.doi.org/10.1201/9780429455155-13.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Fry, John. "Endocrine." In The Beecham Manual for Family Practice, 245–61. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-011-6361-3_20.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Varghese, Jeena, and Conor Best. "Endocrine." In Managing Immunotherapy Related Organ Toxicities, 59–79. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-00241-0_4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Imam, Ibrahim. "Endocrine." In 700 Essential Neurology Checklists, 400–401. New York: CRC Press, 2021. http://dx.doi.org/10.1201/9781003221258-126.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Endocrine"

1

"Uroradiology GU / Endocrine." In Proceedings of UK Radiological Conference 2020. The British Institute of Radiology, 2020. http://dx.doi.org/10.1259/conf-pukrc.2020.posters-f-gu-uro.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Dooper, Marten. "Chemo-endocrine therapy worse for cognition than endocrine therapy alone." In SABCS 2022, edited by Stefan Rauh. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/2ec9a13b.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Chirita, Anca Livia, Mihaela Popescu, Veronica Calborean, Victor Gheorman, and Ion Udristoiu. "PSYCHIATRIC DISORDERS ASSOCIATED WITH ENDOCRINE DYSFUNCTIONS." In The European Conference of Psychiatry and Mental Health "Galatia". Archiv Euromedica, 2023. http://dx.doi.org/10.35630/2022/12/psy.ro.25.

Full text
Abstract:
Introduction: Psychiatric disorders occurring during endocrine dysfunction and, conversely, endocrine dysfunctions associated with mental disorders were the emergence of a new discipline, psychoendocrinology. Psychiatric disorders correlated with endocrine diseases are defined as psychopathological manifestations of variable intensity and clinical symptomatology, determined by complex psycho-neuro-endocrinological interrelationships. Defining elements consist of the association between diagnosis of mental disorders and specific symptoms for endocrine dysfunction. Methods: We conducted a prospective one-year study (January 2018 - December 2018 on 112 patients hospitalized in the Clinic of Psychiatry who also had an endocrinological comorbidity. We investigated the frequency and severity of psychoendocrinological associations by studying a number of demographic and clinical items. Results: The results showed that the highest incidence belongs to thyroid disorder - 55.36%, followed by gonadal disorders - 24.11%, and, rarely, pituitary diseases and diabetes. Hyperthyroidism was associated most frequently with manic episodes, while unipolar depression prevailed in patients with hypothyroidism. In gonadal disorders, present in majority in female patients (secondary amenorrhea, menopause or erectile dysfunction in males), depression accompanied by anxiety, often severe in intensity, was the most frequent psychiatric diagnosis. Psychotic disorders were met in a smaller number of cases, especially in patients with long history of endocrine disorders and instability of biological constants. Conclusions: We may state that affective disorders are the most frequent nosologically category in patients with endocrine dysfunctions. It requires a better collaboration between specialists in endocrinology and psychiatry, to highlight the determinants which contribute to the development of psychopathological manifestations in endocrine diseases and to individualize the treatment depending on cases’ particularities.
APA, Harvard, Vancouver, ISO, and other styles
4

Rotar, C. "Endocrine Control Evolutionary Algorithm." In 12th International Symposium on Symbolic and Numeric Algorithms for Scientific Computing (SYNASC 2010). IEEE, 2010. http://dx.doi.org/10.1109/synasc.2010.27.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Gonza´lez, Camille, Anai´s Va´zquez, Angel Morales, Liz Di´az, Carlos R. Cabrera, and Kai Griebenow. "Development of a Peroxidase Biosensor for the Detection of Endocrine Disrupting Chemicals (EDCs)." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13059.

Full text
Abstract:
Endocrine disruptor compounds are able to mimic or antagonize the effects of endogenous hormones. Phenolic compounds are one of the most abundant classes of endocrine disruptors due to their presence in a broad range of chemical manufacturing processes. The detection of such compounds in food, medicine, and the environment (i.e., water) is crucial to ensure their quality. To prevent the noxious effects of endocrine disruptors an efficient monitoring system is required in order for immediate remediation to be activated. The long-term goal of the project is to develop a robust and stable amperometric enzyme based biosensor able to determine the concentration of phenolic endocrine disruptors. This type of biosensor can be useful to monitor endocrine distruptors in biological fluids and environmental samples as for example the spacecraft drinking water, to ensure the health of the astronauts in space.
APA, Harvard, Vancouver, ISO, and other styles
6

Wei, Cui, Sheng Qiang, and X. Gao. "Artificial Endocrine System and Applications." In 2006 Chinese Control Conference. IEEE, 2006. http://dx.doi.org/10.1109/chicc.2006.280709.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Chen, S., S. Masri, H. Chan, K. Petrossian, S. Phung, Y. Yuan, H. Li, and C. Kowolik. "Molecular Characterization of Endocrine Resistance." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-407.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Hiscox, S., P. Barnfather, E. Hayes, P. Bramble, J. Christensen, R. Nicholson, and P. Barrett-Lee. "Inhibition of Focal Adhesion Kinase Suppresses Adverse Features of Endocrine-Resistant Breast Cancer Cells and Improves Endocrine Response in ER+, Endocrine-Sensitive Cells." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-3130.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Timokhina, Ekaterina Petrovna, Nataliya Valentinovna Yaglova, Sergey Stanislavovich Obernikhin, Valentin Vasilyevich Yaglov, Svetlana Vladimirovna Nazimova, and Dibakhan Aslanbekovna Tsomartova. "SENSITIVITY OF CATHEHOLAMINE-PRODUCING STRUCTURES TO DISRUPTIVE ACTION OF DDT." In International conference New technologies in medicine, biology, pharmacology and ecology (NT +M&Ec ' 2020). Institute of information technology, 2020. http://dx.doi.org/10.47501/978-5-6044060-0-7.11.

Full text
Abstract:
The results of the research revealed that norepinephrine-produsing formations demonstrate higher sensitivity to low-dose exposure to endocrine disrupting chemical DDT than epinephrine-producing ones. Differences in sensitivity to endocrine disruption remains unchanged whatever onset of exposure is prenatal or postnatal period of ontogeny.
APA, Harvard, Vancouver, ISO, and other styles
10

Qurbanova R.Sh., Asadova Sh.Sh., Vahabova Sh.B., Qurbanova R. Sh ,. Asadova Sh Sh ,. Vahabova Sh B. "CLİNİCAL AND MORPHOLOGİCAL PATTERNS OF ENDOCCRİNE-CELL CARSİNOMA OF UTERİ." In THE FIRST INTERNATIONAL SCIENTIFIC – PRACTICAL VIRTUAL CONFERENCE IN MODERN & SOCIAL SCIENCES: NEW DIMENSIONS, APPROACHES AND CHALLENGES. IRETC, 2022. http://dx.doi.org/10.36962/mssndac-01-11.

Full text
Abstract:
The relevance of the problem. The endicrine-cell carsinoma of corpus uteri and of the cervex uteri remains insufficiently studied, from clinical and proqnosis point of view insignificant group of malignant epithelial organ tumors. The purpose of the study. Taking into consideration all stated above, as the goal of current investigation has been appeared complex statistic, clinic, laboratory-instrumental and morphological study of this cancer with comparative analysis of results in different types of treatment, and also including progression and prognosis of the process. Material and methods. 405 cases of endocrine cell carcinoma of corpus uteri were identified within 2016- 2021. Complaints, anamnesis and clinical stages of the process approximately didn’t differ from the analogous groups of comparison of patient by the moment of appeal for the specialized aid. The obtained results and their discussion. It was determined that the fraquancy of countering of the endocrine cell carcinoma of corpus uteri is 26,1%, but the cancer of cervix uteri 12,7% from total number patients, respectively. It was proved that there exists the direct correlation between the frequency of endocrine cell carcinoma of corpus uteri and anatomical section of organ. In 56,0% od cases primary tumor origin is situated in lower segment of corpus, circus cervix or proximal part of cervical canal. “Carcinoids” and “Small cell homomorphous solid squamous cell and glandular carcinoids” were encountered most frequently among histotypes and the versions. Morphologically in tumor tissue were revealed mono and double specialized apudocytes, producing olygopeptides and amines with paraendocrine effects. Proceeding from secretory status of the endocrine tumor component were distinguished following clinical-morphological subgroups of endocrine cell carcinoma of corpus utseri: 1) with prevailing secretion of peptides (excluding the somatostatin); 2) with prevailing secretion of amines and somatostatin; 3) with similar secretion of peptides and amines. It was proved that, there is direct close correlation between prevailing hormonal status and clinical progression of endocrine-cell carcinoma of corpus uteri. Despite of somatostatin predominant secretion of oligopeptides by the tumor accelerates clinical progression, and brings to early and multidirectional metastases. On the contrary, with prevailing secretion of amines (serotonin, melatonin) and somatostatin the process doesn’t improving, and metastases, also, recurrences after radical therapy isn’t seen. It was revealed, that when choosing the tactic of radical treatment, side by side with the other factors, must be taken into consideration the hormonal status of endocrine-cell carcinoma of corpus uteri. High prevalence of secretion of oligopeptides in endocrine-cell carcinoma of corpus uteri can be useful in direct indication for choosing of complex or combinative treatment with necessary use radical surgical operation. Keywords: distinguished following clinical-morphological subgroups.
APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Endocrine"

1

NMR Publicering. Suspected endocrine disrupting substances. Nordisk Ministerråd, October 2013. http://dx.doi.org/10.6027/na2013-921.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Clarke, Robert S. Endocrine Therapy of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada443230.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Clarke, Robert. Endocrine Therapy of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2008. http://dx.doi.org/10.21236/ada492475.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Clarke, Robert. Endocrine Therapy of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2006. http://dx.doi.org/10.21236/ada463407.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Clarke, Robert. Endocrine Therapy of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2007. http://dx.doi.org/10.21236/ada472777.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Risbridger, Gail. Endocrine Disruption and Human Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2008. http://dx.doi.org/10.21236/ada485058.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Clarke, Robert. XBP1, Unfolded Protein Response, and Endocrine Responsiveness. Fort Belvoir, VA: Defense Technical Information Center, May 2009. http://dx.doi.org/10.21236/ada516632.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Clarke, Robert. XBP1, Unfolded Protein Response, and Endocrine Responsiveness. Fort Belvoir, VA: Defense Technical Information Center, May 2012. http://dx.doi.org/10.21236/ada563383.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Clarke, Robert. XBP1, Unfolded Protein Response, and Endocrine Responsiveness. Fort Belvoir, VA: Defense Technical Information Center, May 2011. http://dx.doi.org/10.21236/ada550804.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Klun, Jerome Anthony, Ada Rafaeli, and Ashok Raina. Endocrine Control of Pheromone Production in Arthopods. United States Department of Agriculture, October 1985. http://dx.doi.org/10.32747/1985.7566575.bard.

Full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Endocrine' for particular source types:
Journal articles Dissertations / Theses Books
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography