Academic literature on the topic 'End stage kidney disease'
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Journal articles on the topic "End stage kidney disease"
Crawford, Gregory B., Julie A. Robinson, Amy E. Z. Baker, and Susan M. Crail. "End-Stage Kidney Disease." American Journal of Hospice and Palliative Medicine® 31, no. 3 (April 11, 2013): 331–37. http://dx.doi.org/10.1177/1049909113484383.
Full textLanewala, AliAsghar. "Chronic kidney disease – End-stage kidney disease group." Asian Journal of Pediatric Nephrology 4, no. 1 (2021): 3. http://dx.doi.org/10.4103/ajpn.ajpn_24_21.
Full textKuehn, Bridget M. "End-stage Kidney Disease Doubles." JAMA 327, no. 16 (April 26, 2022): 1540. http://dx.doi.org/10.1001/jama.2022.5342.
Full textKuehn, Bridget M. "End-stage Kidney Disease Doubles." JAMA 327, no. 16 (April 26, 2022): 1540. http://dx.doi.org/10.1001/jama.2022.5342.
Full textGupta, Ryan, Karen Woo, and Jeniann A. Yi. "Epidemiology of end-stage kidney disease." Seminars in Vascular Surgery 34, no. 1 (March 2021): 71–78. http://dx.doi.org/10.1053/j.semvascsurg.2021.02.010.
Full textFarmakis, Christopher, and Roberto Collazo-Maldonado. "Hypertension in end-stage kidney disease." Hypertension Journal 7, no. 1 (2021): 14–18. http://dx.doi.org/10.15713/ins.johtn.0215.
Full textMoss, Alvin H., Nancy Armistead, and Dale Lupu. "End-Stage Kidney Disease Without Dialysis." Health Affairs 34, no. 11 (November 2015): 2005. http://dx.doi.org/10.1377/hlthaff.2015.1225.
Full textWong, Jonathan, Enric Vilar, and Ken Farrington. "Endotoxemia in End-Stage Kidney Disease." Seminars in Dialysis 28, no. 1 (July 13, 2014): 59–67. http://dx.doi.org/10.1111/sdi.12280.
Full textHylek, Elaine M. "Apixaban for End-Stage Kidney Disease." Circulation 138, no. 15 (October 9, 2018): 1534–36. http://dx.doi.org/10.1161/circulationaha.118.036449.
Full textKrishnan, Amrish, Yogeshni Chandra, Joji Malani, Shilpanjali Jesudason, Shaundeep Sen, and Angus G. Ritchie. "End‐stage kidney disease in Fiji." Internal Medicine Journal 49, no. 4 (April 2019): 461–66. http://dx.doi.org/10.1111/imj.14108.
Full textDissertations / Theses on the topic "End stage kidney disease"
Webster, Angela C. "Immunosuppression and malignancy in end stage kidney disease." Connect to full text, 2006. http://hdl.handle.net/2123/1186.
Full textTitle from title screen (viewed 21 May 2007). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Public Health, Faculty of Medicine. Includes bibliographical references. Also available in print form.
Webster, Angela Claire. "Immunosuppression and malignancy in end stage kidney disease." Thesis, The University of Sydney, 2006. http://hdl.handle.net/2123/1186.
Full textWebster, Angela Claire. "Immunosuppression and malignancy in end stage kidney disease." University of Sydney, 2006. http://hdl.handle.net/2123/1186.
Full textIntroduction Kidney transplantation confers both survival and quality of life advantages over dialysis for most people with end-stage kidney disease (ESKD). The mortality rate on dialysis is 10-15% per year, compared with 2-4% per year post-transplantation. Short-term graft survival is related to control of the acute rejection process, requiring on-going immunosuppression. Most current immunosuppressive algorithms include one of the calcineurin inhibitors (CNI: cyclosporin or tacrolimus), an anti-metabolite (azathioprine or mycophenolate) and corticosteroids, with or without antibody induction agents (Ab) given briefly peri-transplantation. Despite this approach, between 15-35% of recipients undergo treatment for an episode of acute rejection (AR) within one year of transplantation. Transplantation is not without risk, and relative mortality rates for kidney recipients after the first post-transplant year remain 4-6 times that of the general population. Longer-term transplant and recipient survival are related to control of chronic allograft nephropathy (rooted in the interplay of AR, non-immunological factors, and the chronic nephrotoxicity of CNI) and limitation of the complications of chronic ESKD and long-term immunosuppression: cardiovascular disease, cancer and infection, which are responsible for 22%, 39% and 21% of deaths respectively. This thesis is presented as published works on the theme of immunosuppression and cancer after kidney transplantation. The work presented in the first chapters of this thesis has striven to identify, evaluate, synthesise and distil the entirety of evidence available of new and established immunosuppressive drug agents through systematic review of randomised trial data, with particular emphasis on quantifying harms of treatment. The final chapters use inception cohort data from the Australian and New Zealand Dialysis and Transplant Registry (ANZDATA), which is first validated then used to explore the risk of cancer in more detail than was possible from trial data alone. Interleukin 2 receptor antagonists Interleukin-2 receptor antagonists (IL2Ra, commercially available as basiliximab and daclizumab) are humanised or chimeric IgG monoclonal antibodies to the alpha subunit of the IL2 receptor present only on activated T lymphocytes, and the rationale for their use has been as induction agents peri-transplantation. Introduced in the mid-1990s, IL2Ra use has increased globally, and by 2003 38% of new kidney transplant recipients in the United States and 25% in Australasia received an IL2Ra. This study aimed to systematically identify and synthesise the evidence of effects of IL2Ra as an addition to standard therapy, or as an alternative to other induction agents. We identified 117 reports from 38 randomised trials involving 4893 participants. Where IL2Ra were compared with placebo (17 trials; 2786 patients), graft loss was not different at one (Relative Risk -RR 0.84; 0.64 to 1.10) or 3 years (RR 1.08; 0.71 to1.64). AR was reduced at 6 months (RR 0.66; 0.59 to 0.74) and at 1 year (RR 0.66; 0.59 to 0.74) but cytomegalovirus (CMV) disease (RR 0.82; CI 0.65 to 1.03) and malignancy (RR 0.67; 0.33 to1.36) were not different. Where IL2Ra were compared with other antibody therapy no significant differences in treatment effects were demonstrated, but IL2Ra had significantly fewer side effects. Given a 40% risk of rejection, 7 patients would need treatment with IL2Ra in addition to standard therapy, to prevent 1 patient having rejection, with no definite improvement in graft or patient survival. There was no apparent difference between basiliximab and daclizumab. Tacrolimus versus cyclosporin for primary immunosuppression There are pronounced global differences in CNI use; 63% of new kidney transplant recipients in the USA but only 22% in Australia receive tacrolimus as part of the initial immunosuppressive regimen. The side effects of CNI differ: tacrolimus is associated more with diabetes and neurotoxicity, but less with hypertension and dyslipidaemia than cyclosporin, with uncertainty about equivalence of nephrotoxicity or how these relate to patient and graft survival, or impact on patient compliance and quality of life. This study aimed to systematically review and synthesise the positive and negative effects of tacrolimus and cyclosporin as initial therapy for renal transplant recipients. We identified 123 reports from 30 randomised trials involving 4102 participants. At 6 months graft loss was reduced in tacrolimus-treated recipients (RR 0•56; 0•36 to 0•86), and this effect persisted for 3 years. The relative reduction in graft loss with tacrolimus diminished with higher levels of tacrolimus (P=0.04), but did not vary with cyclosporin formulation (P=0.97) or cyclosporin level (P=0.38). At 1 year, tacrolimus patients suffered less AR (RR 0•69; 0•60 to 0•79), and less steroid-resistant AR (RR 0•49; 0•37 to 0•64), but more insulin-requiring diabetes (RR 1•86; 1•11 to 3•09), tremor, headache, diarrhoea, dyspepsia and vomiting. The relative excess in diabetes increased with higher levels of tacrolimus (P=0.003). Cyclosporin-treated recipients experienced significantly more constipation and cosmetic side-effects. We demonstrated no differences in infection or malignancy. Treating 100 recipients with tacrolimus instead of cyclosporin for the 1st year post-transplantation avoids 12 suffering acute rejection and 2 losing their graft but causes an extra 5 to become insulin dependent diabetics, thus optimal drug choice may vary among patients. Target of rapamycin inhibitors for primary immunosuppression Target of rapamycin inhibitors (TOR-I) are among the newest immunosuppressive agents and have a novel mode of action but uncertain clinical role. Sirolimus is a macrocyclic lactone antibiotic and everolimus is a derivative of sirolimus. Both prevent DNA synthesis resulting in arrest of the cell cycle. Animal models suggested TOR-I would provide synergistic immunosuppression when combined with CNI, but early clinical studies demonstrated synergistic nephrotoxicity. Since then diverse trials have explored strategies that avoid this interaction and investigated other potential benefits. The aim of this study was to systematically identify and synthesise available evidence of sirolimus and everolimus when used in initial immunosuppressive regimens for kidney recipients. We identified 142 reports from 33 randomised trials involving 7114 participants, with TOR-I evaluated in four different primary immunosuppressive algorithms: as replacement for CNI, as replacement for antimetabolites, in combination with CNI at low and high dose, and with variable dose of CNI. When TOR-I replaced CNI (8 trials, 750 participants), there was no difference in AR (RR 1.03; 0.74 to 1.44), but creatinine was lower (WMD -18.31 umol/l; -30.96 to -5.67), and bone marrow more suppressed (leucopoenia RR 2.02; 1.12 to 3.66, thrombocytopenia RR 6.97; 2.97 to 16.36, anaemia RR 1.67; 1.27 to 2.20). When TOR-I replaced antimetabolites (11 trials, 3966 participants), AR and CMV were reduced (RR 0.84; 0.71 to 0.99 and RR 0.49; 0.37 to 0.65) but hypercholesterolaemia was increased (RR 1.65; 1.32 to 2.06). When low was compared to high-dose TOR-I, with equal CNI dose (10 trials, 3175 participants), AR was increased (RR 1.23; 1.06 to 1.43) but GFR higher (WMD 4.27 ml/min; 1.12 to 7.41). When low-dose TOR-I and standard-dose CNI were compared to higher-dose TOR-I and reduced CNI AR was reduced (RR 0.67; 0.52 to 0.88), but GFR also reduced (WMD -9.46 ml/min; -12.16 to -6.76). There was no significant difference in mortality, graft loss or malignancy risk demonstrated for TOR-I in any comparison. Generally surrogate endpoints for graft survival favoured TOR-I (lower risk of acute rejection and higher GFR) and surrogate endpoints for patient outcomes were worsened by TOR-I (bone marrow suppression, lipid disturbance). Long-term hard-endpoint data from methodologically robust randomised trials are still needed. Monoclonal and polyclonal antibody therapy for treating acute rejection Strategies for treating AR include pulsed steroids, an antibody (Ab) preparation, the alteration of background immunosuppression, or combinations of these options. In 2002, in the USA 61.4% of patients with AR received steroids, 20.4% received Ab and 18.2% received both. The Ab available for AR are not new: horse and rabbit derived polyclonal antibodies (ATG and ALG) have been used for 35 years, and a mouse monoclonal antibody (muromonab-CD3) became available in the late 1980s. These preparations remove the functional T-cell population from circulation, producing powerful saturation immunosuppression which is useful for AR but which may be complicated by immediate toxicity and higher rates of infection and malignancy. The aim of this study was to systematically evaluate and synthesise all evidence available to clinicians for treating AR in kidney recipients. We identified 49 reports from 21 randomised trials involving 1394 participants. Outcome measures were inconsistent and incompletely defined across trials. Fourteen trials (965 patients) compared therapies for 1st AR episodes (8 Ab versus steroid, 2 Ab versus another Ab, 4 other comparisons). In treating first rejection, Ab was better than steroid in reversing AR (RR 0.57; CI 0.38 to 0.87) and preventing graft loss (RR 0.74; CI 0.58 to 0.95) but there was no difference in preventing subsequent rejection (RR 0.67; CI 0.43 to 1.04) or death (RR 1.16; CI 0.57 to 2.33) at 1 year. Seven trials (422 patients) investigated Ab treatment of steroid-resistant rejection (4 Ab vs another Ab, 1 different doses Ab, 1 different formulation Ab, 2 other comparisons). There was no benefit of muromonab-CD3 over ATG or ALG in reversing rejection (RR 1.32; CI 0.33 to 5.28), preventing subsequent rejection (RR 0.99; CI 0.61 to 1.59), graft loss (RR 1.80; CI 0.29 to 11.23) or death (RR 0.39; CI 0.09 to 1.65). Given the clinical problem caused by AR, comparable data are sparse, and clinically important differences in outcomes between widely used interventions have not been excluded. Standardised reproducible outcome criteria are needed. Validity of cancer data in an end stage kidney disease registry Registries vary in whether the data they collect are given voluntarily or as a requirement of law, the completeness of population coverage, the breadth of data collected and whether data are assembled directly or indirectly through linkage to other databases. Data quality is crucial but difficult to measure objectively. Formal audit of ANZDATA cancer records has not previously taken place. The aim of this study was to assess agreement of records of incident cancer diagnoses held in ANZDATA (voluntary reporting system) with those reported under statute to the New South Wales (NSW) state Central Cancer Registry (CCR), to explore the strengths and weaknesses of both reporting systems, and to measure the impact of any disagreement on results of cancer analyses. From 1980-2001, 9453 residents received dialysis or transplantation in NSW. Records from ANZDATA registrants were linked to CCR using probabilistic matching and agreement between registries for patients with 1 or more cancers, all cancers and site-specific cancer was estimated using the kappa-statistic (κ). ANZDATA recorded 867 cancers in 779 (8.2%) registrants; CCR 867 cancers in 788 (8.3%), with κ =0.76. ANZDATA had sensitivity 77.3% (CI 74.2 to 80.2), specificity 98.1% (CI 97.7 to 98.3) if CCR records were regarded as the reference standard. Agreement was similar for diagnoses whilst receiving dialysis (κ =0.78) or after transplantation (κ =0.79), but varied by cancer type. Melanoma (κ =0.61) and myeloma (κ =0.47) were less good; lymphoma (κ =0.80), leukaemia (κ =0.86) and breast cancer (κ =0.85) were very good. Artefact accounted for 20.8% non-concordance but error and misclassification did occur in both registries. Cancer risk did not differ in any important way whether estimated using ANZDATA or CCR records. Quality of cancer records in ANZDATA are high, differences largely explicable, and seem unlikely to alter results of analyses. Risk of cancer after kidney transplantation Existing data on the magnitude of excess risk of cancer across different kidney recipient groups are sparse. Quantifying an individual transplant candidate’s cancer risk informs both pre-transplant counselling, treatment decisions and has implications for monitoring, screening and follow-up after transplantation. The aims of this study were firstly to establish the risk of cancer in the post-transplant population compared to that experienced by the general population, and secondly to quantify how excess risk varied within the transplanted population, seeking to establish meaningful absolute risk estimates for post-transplant cancer based on unalterable recipient characteristics known a priori at the time of transplantation. 15,183 residents of Australia and New Zealand had a transplant between 1963 and 2004, and were followed for a median of 7.2 years (130,186 person-years), with 1642 (10.8%) developing cancer. Overall, kidney recipients had 3 times the cancer risk, with risk inversely related to age (Standardised Incidence Ratio of 15 to 30 in children reducing to 2 in people > 65 years). Female recipients aged 25 -29 had rates of cancer (779.2/100,000) equivalent to women aged 55 - 59 from the general population. The risk pattern of lymphoma, colorectal and breast cancer was similar to the overall age trend, melanoma showed less variability across ages and prostate cancer showed no risk increase. Within the transplanted population cancer risk was affected by age differently for each sex (P=0.007), and was elevated for recipients with prior non-skin malignancy (Hazard Ratio: HR 1.40; 1.03 to 1.89), of white race (HR 1.36; 1.12 to 1.89), but reduced for those with diabetic ESKD (HR 0.67; 0.50 to 0.89) Rates of cancer in kidney recipients were similar to non-transplanted people 20 -30 years older, but risk differed across patient groups. Men aged 45 - 54 at transplantation with graft function at 10 years had a risk of cancer that varied from 1 in 13 (non-white, diabetic ESKD, no prior cancer) to 1 in 5 (white, prior cancer, ESKD from other causes).
O’Lone, Emma. "Cardiovascular disease: priorities and outcomes in end stage kidney disease." Thesis, The University of Sydney, 2020. https://hdl.handle.net/2123/22326.
Full textMetcalfe, Wendy. "End stage renal disease : outcomes and standards of care." Thesis, University of Aberdeen, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251850.
Full textJassal, Sarbjit Vanita. "Kidney transplantation in elderly patients with end-stage renal disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0005/MQ40712.pdf.
Full textWyld, Melanie Lisa Romola. "Health outcomes and costs in Chronic and End-Stage Kidney Disease." Thesis, The University of Sydney, 2018. http://hdl.handle.net/2123/20419.
Full textNicholas, Pauline. "Impaired cognition in end stage kidney disease: Prevalence, predictors and differences between treatment." Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/203098/1/Pauline_Nicholas_Thesis.pdf.
Full textAlashek, Wiam Abdulaziz. "Epidemiology of dialysis-treated end-stage kidney disease in adults in Libya." Thesis, University of Nottingham, 2013. http://eprints.nottingham.ac.uk/28388/.
Full textTynkevich, Elena. "Muscle Wasting in Non-end Stage Chronic Kidney Disease : Determinants and Outcomes." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T086.
Full textMainly described in patients on dialysis, muscle wasting has received little attention in early stage chronic kidney disease (CKD). We used 24-hour creatininuria to assess determinants of low muscle mass and its putative associations with CKD outcomes, using data from the NephroTest cohort, including 1429 non-dialysis patients with CKD stages 1 to 5. Kidney function was assessed with both measured (mGFR, by 51Cr-EDTA renal clearance) and estimated glomerular filtration rate (eGFR, by CKD-EPI equation). End-stage renal disease (ESRD) and pre-ESRD death were the main studied outcomes. The mean baseline creatininuria decreased from 15.3±3.1 to 12.1±3.3 mmol/24 h in men and from 9.6±1.9 to 7.6±2.5 in women, when mGFR fell from ≥ 60 to < 15 mL/min/1.73 m2. Other determinants of low creatininuria were an older age, diabetes, a lower body mass index, a lower level of proteinuria or protein intake. A fast annual decline in mGFR of 5 mL/min/1.73 m2 was linked with a 2-fold decrease in creatininuria, independent of changes in protein intake and other determinants of muscle mass. Over a median follow-up of 3.6 years, 229 patients developed ESRD and 113 patients died before ESRD. After adjustment for confounders, patients with low muscle mass showed a significantly higher risk for pre-ESRD death (HR 1.6, 95% CI 0.88-2.9), but a lower risk for ESRD (HR 0.60, 95% CI 0.39-0.91). The latter was reversed (HR 1.5, 95% CI 1.01-2.4) when mGFR was replaced by eGFR. Decrease in 24-hour creatininuria may appear early in CKD patients, is related to pre-ESRD death. The lower risk for ESRD may reflect later dialysis start due to overestimation of true GFR by eGFR in patients with low muscle mass
Books on the topic "End stage kidney disease"
State University of New York at Albany. School of Public Health. End Stage Renal Disease in New York State. Albany, NY: Health Research, Inc., 1991.
Find full textJoseph, Loscalzo, and London Gérard M, eds. Cardiovascular disease in end-stage renal failure. Oxford: Oxford University Press, 2000.
Find full textFriedman, Eli A. Present and future therapies for end-stage renal disease. New Jersey: World Scientific, 2010.
Find full text1933-, Friedman Eli A., and Mallappallil Mary C, eds. Present and future therapies for end-stage renal disease. New Jersey: World Scientific, 2010.
Find full textJassal, Sarbjit Vanita. Kidney transplantation in elderly patients with end-stage renal disease. Ottawa: National Library of Canada, 1998.
Find full textLibrary of Congress. Congressional Research Service, ed. Medicare's end stage renal disease program. [Washington, D.C.]: Congressional Research Service, Library of Congress, 1992.
Find full text1938-, Hardy Mark A., and Kutscher Lillian G, eds. Positive approaches to living with end stage renal disease: Psychosocial and thanatologic aspects. New York: Praeger, 1986.
Find full textFederal-Provincial Advisory Committee on Institutional and Medical Services (Canada). Subcommittee on Institutional Program Guidelines. End-stage renal disease program: Report. Ottawa: Health and Welfare Canada, 1986.
Find full textHalper, Thomas. The misfortunes of others: End-stage renal disease in the United Kingdom. Cambridge: Cambridge University Press, 1989.
Find full textUnited States. Agency for Health Care and Policy Research. Laboratory tests in end-stage renal disease patients undergoing dialysis. Rockville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1994.
Find full textBook chapters on the topic "End stage kidney disease"
Wightman, Aaron, and Michael Freeman. "Ethical Issues in End Stage Kidney Disease." In Pediatric Kidney Disease, 1703–18. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-11665-0_62.
Full textLantos, John D. "Ethical Issues in End Stage Kidney Disease (ESKD)." In Pediatric Kidney Disease, 1625–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_61.
Full textGhosn, Muriel, and Fuad N. Ziyadeh. "Treatment of the Patient with End-Stage Diabetic Nephropathy." In Diabetes and Kidney Disease, 215–31. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118494073.ch16.
Full textAl Khunaizi, Ahd, and Ahsan Alam. "End-Stage Renal Disease in Patients with Autosomal Dominant Polycystic Kidney Disease." In Polycystic Kidney Disease, 229–41. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7784-0_14.
Full textMartinusen, Dan. "Chronic Kidney Disease and End Stage Renal Disease." In Renal Medicine and Clinical Pharmacy, 45–115. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37655-0_4.
Full textCantarin, Maria Paula Martinez, and Jerry McCauley. "Epidemiology of End-Stage Renal Disease and Kidney Transplantation." In Contemporary Kidney Transplantation, 1–7. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-14779-6_24-1.
Full textParajuli, Sandesh, and Patrick K. Reville. "End Stage Renal Disease – Treatment Options: Dialysis Versus Transplant." In Kidney Transplant Management, 5–16. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-00132-2_2.
Full textCantarin, Maria P. Martinez, and Jerry McCauley. "Epidemiology of End-Stage Renal Disease and Kidney Transplantation." In Contemporary Kidney Transplantation, 335–41. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-19617-6_24.
Full textSchaefer, Franz. "Hypertension in End-Stage Kidney Disease: Dialysis." In Pediatric Hypertension, 499–513. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-06231-5_48.
Full textSeeman, Tomáš. "Hypertension in End-Stage Kidney Disease: Transplantation." In Pediatric Hypertension, 515–32. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-06231-5_49.
Full textConference papers on the topic "End stage kidney disease"
Ho, Samuel Chung-sum, Eugene Yu-hin Chan, Ellen Yu, Matthew Hon-lam Lee, Yuet-ling Tung, and Alison Lap-tak Ma. "335 Fracture burden in paediatric end stage kidney disease." In RCPCH Conference Singapore. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjpo-2021-rcpch.185.
Full textBulhões Calheiros, Marília, Vani Abreu de Souza Filho, Camille Constanza Codogno Postigo Castro, Aloma Guinami Scabora, Alisson Aliel Vigano Pugliesi, and Manoel de Barros Bertolo. "METHOTREXATE AND END-STAGE KIDNEY DISEASE: A DANGEROUS COMBINATION." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17502.
Full textRebouças Filho, Pedro Pedrosa, Suane Pires Pinheiro Da Silva, Jefferson Silva Almeida, Elene Firmeza Ohata, Shara Shami Araujo Alves, and Francisco Dos Santos Hercules Silva. "An Approach to Classify Chronic Kidney Diseases using Scintigraphy Images." In XXXII Conference on Graphics, Patterns and Images. Sociedade Brasileira de Computação - SBC, 2019. http://dx.doi.org/10.5753/sibgrapi.est.2019.8318.
Full textBotros, M., J. Kenneth, P. Singh, and B. Keller. "Early Postoperative Acute Kidney Injury and End Stage Renal Disease in Lung Transplant Recipients." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2832.
Full textKhattar, G., K. El Gharib, P. Ngowari, J. Kotys, V. R. Kandala, K. Salman, H. El Hage, et al. "Unravelling the Fluid Paradox in End Stage Kidney Disease and Sepsis: A Meta-analysis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5479.
Full textHuang, Zhongping, Jie Ren, and Anilchandra Attaluri. "Experimental Study of a Hybrid Renal Replacement System." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14326.
Full textGordeeva, Olga, Vladislav Hergert, Irina Lyadova, and Natalya Karpina. "Quantiferon test in the complex diagnosis of tuberculosis in patients with end-stage kidney disease." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1457.
Full textSellars, M., J. Clayton, K. Detering, A. Tong, D. Power, and R. Morton. "OP56 The cost-effectiveness of advance care planning for older adults with end-stage kidney disease." In ACP-I Congress Abstracts. British Medical Journal Publishing Group, 2019. http://dx.doi.org/10.1136/spcare-2019-acpicongressabs.56.
Full textBoukhechba, Mehdi, Mingyue Tang, Brendan Bowman, Jamie Zoellner, and Emaad Abdel-Rahman. "A Smartwatch Based system for Monitoring Fluid Consumption of End Stage Kidney Patients." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002101.
Full textThomas, CB, AP Wheeler, GR Bernard, and TW Rice. "Morbidity and Mortality of End Stage Renal Disease vs. Acute Kidney Injury in Patients with Acute Lung Injury." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4644.
Full textReports on the topic "End stage kidney disease"
Han, Mei, JiaHui Cao, Zixuan Chen, Ying Wang, Lily LAI, and Jianping Liu. Hematuria and subsequent long-term risk of end-stage kidney disease: a systematic review and meta-analysis of cohort study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2023. http://dx.doi.org/10.37766/inplasy2023.6.0041.
Full textWala, Kamila, and Jacek Szepietowski. Difelikefalin in the treatment of chronic kidney disease-associated pruritus: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0154.
Full textStoumpos, Sokratis, Patrick Mark, David Kingsmore, Giles Roditi, and Aleksandra Radjenovic. Use of Ferumoxytol enhanced Magnetic Resonance Angiography for cardiovascular assessment in late-stage chronic kidney disease. University of Glasgow, April 2020. http://dx.doi.org/10.36399/gla.pubs.215112.
Full textZhang, Fan, Ying Zhang, Liuyan Huang, and Wenqin Zhou. Interventions for promoting physical activity in patients with end stage renal disease receiving hemodialysis. INPLASY - International Platform of Registered Systematic Review Protocols, March 2020. http://dx.doi.org/10.37766/inplasy2020.3.0013.
Full textDor, Avi, Mark Pauly, Margaret Eichleay, and Philip Held. End-stage Renal Disease and Economic Incentives: The International Study of Health Care Organization and Financing. Cambridge, MA: National Bureau of Economic Research, May 2007. http://dx.doi.org/10.3386/w13125.
Full textBishop, Haley, Hannah White, Matthew Wilhaucks, Ashton Weaver, Christin Simpson, and Margaret Harvey. Medicinal Treatment versus Dialysis in End Stage Renal Disease Patients with Perioperative Hyperkalemia: A Scoping Review. University of Tennessee Health Science Center, April 2024. http://dx.doi.org/10.21007/con.dnp.2024.0087.
Full textLiu, Jenny. Inclusive Planning to Evaluate Improved Non-Emergency Medical Transportation Services for Patients With End Stage Renal Disease. Portland State University Library, December 2014. http://dx.doi.org/10.15760/trec.41.
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